Making Sense of All the Post Emergent Herbicide Options in Turf
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2,4-Dichlorophenoxyacetic Acid
2,4-Dichlorophenoxyacetic acid 2,4-Dichlorophenoxyacetic acid IUPAC (2,4-dichlorophenoxy)acetic acid name 2,4-D Other hedonal names trinoxol Identifiers CAS [94-75-7] number SMILES OC(COC1=CC=C(Cl)C=C1Cl)=O ChemSpider 1441 ID Properties Molecular C H Cl O formula 8 6 2 3 Molar mass 221.04 g mol−1 Appearance white to yellow powder Melting point 140.5 °C (413.5 K) Boiling 160 °C (0.4 mm Hg) point Solubility in 900 mg/L (25 °C) water Related compounds Related 2,4,5-T, Dichlorprop compounds Except where noted otherwise, data are given for materials in their standard state (at 25 °C, 100 kPa) 2,4-Dichlorophenoxyacetic acid (2,4-D) is a common systemic herbicide used in the control of broadleaf weeds. It is the most widely used herbicide in the world, and the third most commonly used in North America.[1] 2,4-D is also an important synthetic auxin, often used in laboratories for plant research and as a supplement in plant cell culture media such as MS medium. History 2,4-D was developed during World War II by a British team at Rothamsted Experimental Station, under the leadership of Judah Hirsch Quastel, aiming to increase crop yields for a nation at war.[citation needed] When it was commercially released in 1946, it became the first successful selective herbicide and allowed for greatly enhanced weed control in wheat, maize (corn), rice, and similar cereal grass crop, because it only kills dicots, leaving behind monocots. Mechanism of herbicide action 2,4-D is a synthetic auxin, which is a class of plant growth regulators. -
USDA, Forest Service Forest Health Protection GSA Contract No
SERA TR 02-43-13-03b Triclopyr - Revised Human Health and Ecological Risk Assessments Final Report Prepared for: USDA, Forest Service Forest Health Protection GSA Contract No. GS-10F-0082F USDA Forest Service BPA: WO-01-3187-0150 USDA Purchase Order No.: 43-1387-2-0245 Task No. 13 Submitted to: Dave Thomas, COTR Forest Health Protection Staff USDA Forest Service Rosslyn Plaza Building C, Room 7129C 1601 North Kent Street Arlington, VA 22209 Submitted by: Patrick R. Durkin Syracuse Environmental Research Associates, Inc. 5100 Highbridge St., 42C Fayetteville, New York 13066-0950 Telephone: (315) 637-9560 Fax: (315) 637-0445 E-Mail: [email protected] Home Page: www.sera-inc.com March 15, 2003 TABLE OF CONTENTS LIST OF APPENDICES ...................................................... iv LIST OF WORKSHEETS ...................................................... v LIST OF ATTACHMENTS .................................................... v LIST OF TABLES ............................................................ v LIST OF FIGURES ......................................................... viii ACRONYMS, ABBREVIATIONS, AND SYMBOLS .............................. ix COMMON UNIT CONVERSIONS AND ABBREVIATIONS ......................... xi CONVERSION OF SCIENTIFIC NOTATION .................................... xii EXECUTIVE SUMMARY ................................................... xiii 1. INTRODUCTION ........................................................ 1-1 2. PROGRAM DESCRIPTION ................................................ 2-1 2.1. OVERVIEW -
Hell - Another Great Deception
Hell - Another Great Deception (Copyright ©1truth1law.com 2015) The existence of a literal place called “hell,” where sinners suffer various forms of torment forever, has been ingrained in the minds of people for centuries and, in most cases, those who hold this belief accept it without much thought or study. Religious authorities have used this doctrine to control and manipulate their adherents by instilling fear and blind obedience in them. After all, who wants to be tormented for eternity? It’s better to listen to your church’s directives than turn your back on that authority and end up in hell as a result. Although it is not always stated in exactly these terms, a religious organization can make implications to this effect and its followers will accept and believe the lie. One of the problems associated with this false teaching is that it pictures Almighty God as an unjust and cruel tyrant who seems to enjoy seeing people suffer. In reality, it is “the god of this present evil age,” who gets satisfaction from the misfortunes of others, Grace to you and peace from God the Father and our Lord Jesus Christ, who gave himself for our sins, that he might deliver us from this present evil age… (Gal. 1:3-4a; NKJV used throughout unless otherwise stated; emphasis added). But even if our gospel is veiled, it is veiled to those who are perishing, whose minds the god of the age (Satan) has blinded … (2Cor. 4:3-4; Ed. notes in parenthesis; emphasis added). Those who see you (Satan) will gaze at you, and consider you, saying: ‘Is this this man (Satan when he is judged) who made the earth tremble (cf. -
Herbicide Mode of Action Table High Resistance Risk
Herbicide Mode of Action Table High resistance risk Chemical family Active constituent (first registered trade name) GROUP 1 Inhibition of acetyl co-enzyme A carboxylase (ACC’ase inhibitors) clodinafop (Topik®), cyhalofop (Agixa®*, Barnstorm®), diclofop (Cheetah® Gold* Decision®*, Hoegrass®), Aryloxyphenoxy- fenoxaprop (Cheetah®, Gold*, Wildcat®), fluazifop propionates (FOPs) (Fusilade®), haloxyfop (Verdict®), propaquizafop (Shogun®), quizalofop (Targa®) Cyclohexanediones (DIMs) butroxydim (Factor®*), clethodim (Select®), profoxydim (Aura®), sethoxydim (Cheetah® Gold*, Decision®*), tralkoxydim (Achieve®) Phenylpyrazoles (DENs) pinoxaden (Axial®) GROUP 2 Inhibition of acetolactate synthase (ALS inhibitors), acetohydroxyacid synthase (AHAS) Imidazolinones (IMIs) imazamox (Intervix®*, Raptor®), imazapic (Bobcat I-Maxx®*, Flame®, Midas®*, OnDuty®*), imazapyr (Arsenal Xpress®*, Intervix®*, Lightning®*, Midas®* OnDuty®*), imazethapyr (Lightning®*, Spinnaker®) Pyrimidinyl–thio- bispyribac (Nominee®), pyrithiobac (Staple®) benzoates Sulfonylureas (SUs) azimsulfuron (Gulliver®), bensulfuron (Londax®), chlorsulfuron (Glean®), ethoxysulfuron (Hero®), foramsulfuron (Tribute®), halosulfuron (Sempra®), iodosulfuron (Hussar®), mesosulfuron (Atlantis®), metsulfuron (Ally®, Harmony®* M, Stinger®*, Trounce®*, Ultimate Brushweed®* Herbicide), prosulfuron (Casper®*), rimsulfuron (Titus®), sulfometuron (Oust®, Eucmix Pre Plant®*, Trimac Plus®*), sulfosulfuron (Monza®), thifensulfuron (Harmony®* M), triasulfuron (Logran®, Logran® B-Power®*), tribenuron (Express®), -
Exposure to Herbicides in House Dust and Risk of Childhood Acute Lymphoblastic Leukemia
Journal of Exposure Science and Environmental Epidemiology (2013) 23, 363–370 & 2013 Nature America, Inc. All rights reserved 1559-0631/13 www.nature.com/jes ORIGINAL ARTICLE Exposure to herbicides in house dust and risk of childhood acute lymphoblastic leukemia Catherine Metayer1, Joanne S. Colt2, Patricia A. Buffler1, Helen D. Reed3, Steve Selvin1, Vonda Crouse4 and Mary H. Ward2 We examine the association between exposure to herbicides and childhood acute lymphoblastic leukemia (ALL). Dust samples were collected from homes of 269 ALL cases and 333 healthy controls (o8 years of age at diagnosis/reference date and residing in same home since diagnosis/reference date) in California, using a high-volume surface sampler or household vacuum bags. Amounts of agricultural or professional herbicides (alachlor, metolachlor, bromoxynil, bromoxynil octanoate, pebulate, butylate, prometryn, simazine, ethalfluralin, and pendimethalin) and residential herbicides (cyanazine, trifluralin, 2-methyl-4- chlorophenoxyacetic acid (MCPA), mecoprop, 2,4-dichlorophenoxyacetic acid (2,4-D), chlorthal, and dicamba) were measured. Odds ratios (OR) and 95% confidence intervals (CI) were estimated by logistic regression. Models included the herbicide of interest, age, sex, race/ethnicity, household income, year and season of dust sampling, neighborhood type, and residence type. The risk of childhood ALL was associated with dust levels of chlorthal; compared to homes with no detections, ORs for the first, second, and third tertiles were 1.49 (95% CI: 0.82–2.72), 1.49 (95% CI: 0.83–2.67), and 1.57 (95% CI: 0.90–2.73), respectively (P-value for linear trend ¼ 0.05). The magnitude of this association appeared to be higher in the presence of alachlor. -
Triclopyr Human Health and Ecological Risk Assessment Corrected Final Report
SERA TR-052-25-03c Triclopyr Human Health and Ecological Risk Assessment Corrected Final Report Submitted to: Paul Mistretta, COR USDA/Forest Service, Southern Region 1720 Peachtree RD, NW Atlanta, Georgia 30309 USDA Forest Service Contract: AG-3187-C-06-0010 USDA Forest Order Number: AG-43ZP-D-09-0034 SERA Internal Task No. 52-25 Submitted by: Patrick R. Durkin Syracuse Environmental Research Associates, Inc. 8125 Solomon Seal Manlius, New York 13104 Fax: (315) 637-0445 E-Mail: [email protected] Home Page: www.sera-inc.com May 24, 2011 October 20, 2011 (Minor Correction) July 9, 2016 (Corrections) Error Notes October 20, 2011 In the original release of the final report (SERA TR-052-25-03a dated May 24, 2011), Tables 2 and 22 incorrectly listed the water solubility of TCP as 100 mg/L. As indicated in Table 1, the correct value, from Knuteson (1999), is 49,000 mg/L. This error was noted by Dr. K. King (U.S. Fish and Wildlife Service). The error has been corrected. While the Gleams-Driver runs were made using the 100 mg/L water solubility, re-runs using the water solubility of 49,000 mg/L yielded results that are indistinguishable from the original runs. Thus, the appendices have not been change. Water solubility is not a sensitive parameter in GLEAMS unless the soil water is saturated. This did not occur in the Gleams-Driver modeling. July 9, 2016 During an audit of WorksheetMaker (Version 6.00.15), it was noted that the chronic toxicity values of TCP to aquatic invertebrates had been entered incorrectly into the WorksheetMaker database and the aquatic toxicity values of TCP for algae had been omitted. -
MC(' Potential Exposure of Humans to 2
(MC( FUNDAMENTAL AND APPLIED TOXICOLOGY 1:3 3 9-3 4 6 (1981) Potential Exposure of Humans to 2,4,5-T and TCDD in the Oregon Coast Ranges MICHAEL NEWTON" and LOGAN A. NORRISB "Professor of Forest Ecology, Oregon State University, Corvallis; BChief Research Chemist, USDA Forest Service, Corvallis, Oregon ABSTRACT Potential Exposure of Humans to 2,4,5-T and TCDD in Humans may be exposed to herbicides through drift; inges- the Oregon Coast Ranges. Newton, M. and Norris, L.A. tion of wild and domestic meat, vegetables, and fruit; con- (1981). F.undam. AppL Toxicol. 1:339-346. Research on the sumption of water; and dermal contact while handling the use of 2,4,5-trichlorophenoxyacetic acid (2,4,5-T) contami- chemicals, equipment, and treated vegetation. The range of -8 nated with 2.5 X 10 parts 2,3,7,8-tetrachlorodibenzo-p- potential exposure extends from zero, if there is no encounter dioxin (TCDD) in forests of the Oregon Coast Ranges per- with the herbicide, to the worst situation where the person has mits estimates of human exposures for both compounds. encountered the highest levels of water contamination, drift Estimated total exposure of nearby ( ^ 1/8 mile distant) resi- exposure, meat contamination, and dermal exposure simul- dents during the first week after application is 0.0039 mg/kg taneously. We have brought estimates of all sources together of 2,4,5-T for a 70-kg adult. Exposure to TCDD in the same to determine the possible range of total exposure from episode would be 1.9 X 10 b ° mg/kg. -
INDEX to PESTICIDE TYPES and FAMILIES and PART 180 TOLERANCE INFORMATION of PESTICIDE CHEMICALS in FOOD and FEED COMMODITIES
US Environmental Protection Agency Office of Pesticide Programs INDEX to PESTICIDE TYPES and FAMILIES and PART 180 TOLERANCE INFORMATION of PESTICIDE CHEMICALS in FOOD and FEED COMMODITIES Note: Pesticide tolerance information is updated in the Code of Federal Regulations on a weekly basis. EPA plans to update these indexes biannually. These indexes are current as of the date indicated in the pdf file. For the latest information on pesticide tolerances, please check the electronic Code of Federal Regulations (eCFR) at http://www.access.gpo.gov/nara/cfr/waisidx_07/40cfrv23_07.html 1 40 CFR Type Family Common name CAS Number PC code 180.163 Acaricide bridged diphenyl Dicofol (1,1-Bis(chlorophenyl)-2,2,2-trichloroethanol) 115-32-2 10501 180.198 Acaricide phosphonate Trichlorfon 52-68-6 57901 180.259 Acaricide sulfite ester Propargite 2312-35-8 97601 180.446 Acaricide tetrazine Clofentezine 74115-24-5 125501 180.448 Acaricide thiazolidine Hexythiazox 78587-05-0 128849 180.517 Acaricide phenylpyrazole Fipronil 120068-37-3 129121 180.566 Acaricide pyrazole Fenpyroximate 134098-61-6 129131 180.572 Acaricide carbazate Bifenazate 149877-41-8 586 180.593 Acaricide unclassified Etoxazole 153233-91-1 107091 180.599 Acaricide unclassified Acequinocyl 57960-19-7 6329 180.341 Acaricide, fungicide dinitrophenol Dinocap (2, 4-Dinitro-6-octylphenyl crotonate and 2,6-dinitro-4- 39300-45-3 36001 octylphenyl crotonate} 180.111 Acaricide, insecticide organophosphorus Malathion 121-75-5 57701 180.182 Acaricide, insecticide cyclodiene Endosulfan 115-29-7 79401 -
U.S. EPA, Pesticide Product Label, QUINCLORAC 75 SWF, 02/06/2006
u.s. ENVIRONM£~J7AL PkO';EC';ION N:JENCY EPfI, Req. Cate ~f Issuance: Office of t'esticide "roqram.<; Number: Keqistratlon Divlsion (7:'()SC) 1200 Pennsylvania Ave., N.W. WashingtrJII, D.::'. ?046D 42750- 131 FEE - 6 2006 NOTICE OF PESTICIDE: Term of Issuance: ~ Registration Conditional __ Reregistration : under r: fRJ'l., as am'?nd'O'd: Name of ~esticide Product: Quinclorac 75DF SWF Name and Address of RegIstrant ,include ZIP Codej: Albaugh, Inc. P.O. Box 2127 Valdosta, GA 31604-2127 Note: Changes in .LabEoli~fg differing in suostance from that accepted in connection with this registration must be submitted to and accepted by the Registratlon Division prior to use of the label in commerce. In any correspondence on this product always refer to the above EPA registration number. em ::he bas':s of lnformat':on f ~rr,lshed by the reg:s'Crant, the above llamed pesticide ':5 hereby roegisterea/reregJ.3terec under :::--.e federa; =nsec::':c:ide, fungicide and Rodenticide Act. Reg':straL,on is ir. no (.Jay tc be ::onstrued as an endorsement or recommendation of this product by the Agency. =n urder to protect hea~ttJ and tr.e ",nvil"onment, ::he Admir.istrator, on his motion, may at any time suspend or cancel the regi3t:::ation of a pesticide in accoraance with the Act. The acceptance of any name 1.n connection with the registration of a product under this Act is not to be construed as giving the registrant a right to -=xclusive use of the r,ame or to its use if it has been coveroed by others. -
US EPA, Pesticide Product Label, QUINCLORAC 4L AG,03/04/2019
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Well Water Testing Clinic—June
S ALCHA - D ELTA S OIL & W ATER C ONSERVATION D ISTRICT VOLUME 33,ISSUE 2 2ND QUARTER 2013 DATES TO REMEMBER May 27 WELL WATER TESTING CLINIC—JUNE 3-5 Memorial Day, Office Closed When is the last time you had your water testing kit includes a test for June 1 household well water tested? It’s likely arsenic, coliform, and nitrates+nitrites, Well-water Testing Kits that the majority of homeowners have along with easy-to-follow sampling available during Friendly never tested their household drinking instructions. Frontier Days, Sullivan Roadhouse area water. Drinking water can contain Following collection, samples can be contaminants that are invisible to the June 3, 4, 5 dropped off at the District office. The Well-water kits available, naked eye and harmful to human health. District will cover the cost of delivery samples due by 12:30 pm at Arsenic is a highly toxic contaminant to the lab for analysis. Results will then SWCD office and can occur naturally. It is a hazardous be mailed from the testing laboratory June 4 material and a possible carcinogen. to both the homeowner and District Board Meeting, 8:30 am Coliform bacteria are an indicator of office. July 2 the presence of fecal contamination. We are offering this Board Meeting, 8:30 am These bacteria can present a very serious kit at a reduced July 4 health risk that can cause illness and rate. For a District Independence Day, Office could signal a threat to your drinking cooperator (a pro- Closed water supply. ducer of renewable August 6 Nitrates+nitrites, over a relatively short resources and Board Meeting, 8:30 am following a con- period of time, can be very dangerous, Board meetings are usually causing serious illness to infants. -
Corn Herbicides to Control Glyphsoate-Resistant Canola Volunteers
AGRONOMY SCIENCES RESEARCHRESEARCH UPDAUPDATETE Corn Herbicides to Control Glyphosate-Resistant Canola Volunteers 2013 Background Table 1. Herbicide treatments. • With the rapid adoption of glyphosate-resistant corn in Western Treatment Application Rate/Acre Canada, glyphosate-resistant canola volunteers have become a major weed concern to corn producers in the region. 1 Gly Only (Check) 1 L/acre (360g ae) • The Pest Management Regulatory Agency now allows 2 Gly + Dicamba 1 L/acre + 0.243 L/acre herbicides to be tank-mixed if they have individual registrations 3 Gly + 2,4-D 1 L/acre + 0.4 L/acre (600g/L) on the crop and have a common application timing. 4 Gly + MCPA Amine 1 L/acre + 0.45L/acre • Several herbicide options are available to control glyphosate 5 Gly + Bromoxynil 1 L/acre + 0.48 L/acre tolerant canola volunteers in corn; however, some herbicides may have undesirable effects on corn. Gly / Bromoxynil 6 1 L/acre + 0.48 L/acre (Split Application*) Objectives * Glyphosate and bromoxynil applied separately at V3 stage. • Assess crop injury and yield effects of various herbicides tank- mixed with glyphosate to control glyphosate-resistant canola • Herbicide injury scores were recorded at: volunteers in glyphosate-resistant corn. • 3-5 days after treatment • 7-10 days after treatment • Identify the most suitable post-emergence strategy for • 21-24 days after treatment managing glyphosate-resistant canola volunteers in glyphosate-resistant corn. • Other recorded observations include: • Brittle snap counts Study Description • Yield (bu/acre) & moisture (%) • Test weight (lbs/bu) • The study compared the crop response of four industry leading hybrids (Pioneer® brand and competitive) with five different Results herbicide treatments (Table 1).