Prevalence and Treatment Approaches of Impacted
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Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper -
Non-Syndromic Occurrence of True Generalized Microdontia with Mandibular Mesiodens - a Rare Case Seema D Bargale* and Shital DP Kiran
Bargale and Kiran Head & Face Medicine 2011, 7:19 http://www.head-face-med.com/content/7/1/19 HEAD & FACE MEDICINE CASEREPORT Open Access Non-syndromic occurrence of true generalized microdontia with mandibular mesiodens - a rare case Seema D Bargale* and Shital DP Kiran Abstract Abnormalities in size of teeth and number of teeth are occasionally recorded in clinical cases. True generalized microdontia is rare case in which all the teeth are smaller than normal. Mesiodens is commonly located in maxilary central incisor region and uncommon in the mandible. In the present case a 12 year-old boy was healthy; normal in appearance and the medical history was noncontributory. The patient was examined and found to have permanent teeth that were smaller than those of the average adult teeth. The true generalized microdontia was accompanied by mandibular mesiodens. This is a unique case report of non-syndromic association of mandibular hyperdontia with true generalized microdontia. Keywords: Generalised microdontia, Hyperdontia, Permanent dentition, Mandibular supernumerary tooth Introduction [Ullrich-Turner syndrome], Chromosome 13[trisomy 13], Microdontia is a rare phenomenon. The term microdontia Rothmund-Thomson syndrome, Hallermann-Streiff, Oro- (microdentism, microdontism) is defined as the condition faciodigital syndrome (type 3), Oculo-mandibulo-facial of having abnormally small teeth [1]. According to Boyle, syndrome, Tricho-Rhino-Phalangeal, type1 Branchio- “in general microdontia, the teeth are small, the crowns oculo-facial syndrome. short, and normal contact areas between the teeth are fre- Supernumerary teeth are defined as any supplementary quently missing” [2] Shafer, Hine, and Levy [3] divided tooth or tooth substance in addition to usual configuration microdontia into three types: (1) Microdontia involving of twenty deciduous and thirty two permanent teeth [7]. -
On the Genetics of Hypodontia and Microdontia: Synergism Or Allelism of Major Genes in a Family with Six Affected Members
JMed Genet 1996;33:137-142 137 On the genetics of hypodontia and microdontia: synergism or allelism of major genes in a family J Med Genet: first published as 10.1136/jmg.33.2.137 on 1 February 1996. Downloaded from with six affected members S P Lyngstadaas, H Nordbo, T Gedde-Dahl Jr, P S Thrane Abstract and epigenetic factors.7 In a large study oftooth Familial severe hypodontia of the per- number and size in British schoolchildren, ex- manent dentition is a rare condition. The cluding patients with more widespread ab- genetics ofthis entity remains unclear and normalities, Brook3 favoured a multifactorial several modes of inheritance have been model with a continuous spectrum, related to suggested. We report here an increase in tooth number and size, with thresholds, and the number of congenitally missing teeth where position on the scale depends upon the after the mating of affected subjects from combination of numerous genetic and en- two unrelated Norwegian families. This vironmental factors, each with a small effect. condition may be the result of allelic mut- In this study the proportion ofaffected relatives ations at a single gene locus. Alternatively, varied with the severity of the condition in the incompletely penetrant non-allelic genes probands and an association between hy- may show a synergistic effect as expected podontia and microdontia was noted. for a multifactorial trait with interacting Other causes of hypodontia have been sug- gene products. This and similar kindreds gested and include an evolutionary trend to- may allow identification of genes involved wards fewer teeth,28 infections during in growth and differentiation of dental tis- pregnancy and early childhood, hormonal dys- sues by linkage and haplotype association function, which itself may be inherited, and analysis. -
Oral Manifestations in Patients with Glycogen Storage Disease: a Systematic Review of the Literature
applied sciences Review Oral Manifestations in Patients with Glycogen Storage Disease: A Systematic Review of the Literature 1, 1, 1 2 Antonio Romano y, Diana Russo y , Maria Contaldo , Dorina Lauritano , Fedora della Vella 3 , Rosario Serpico 1, Alberta Lucchese 1,* and Dario Di Stasio 1 1 Multidisciplinary Department of Medical-Surgical and Dental Specialties, University of Campania “L. Vanvitelli”, Via Luigi De Crecchio 6, 80138 Naples, Italy; [email protected] (A.R.); [email protected] (D.R.); [email protected] (M.C.); [email protected] (R.S.); [email protected] (D.D.S.) 2 Department of Medicine and Surgery, Centre of Neuroscience of Milan, University of Milano-Bicocca, 20126 Milan, Italy; [email protected] 3 Interdisciplinary Department of Medicine, University of Bari “A. Moro”, 70124 Bari, Italy; [email protected] * Correspondence: [email protected]; Tel.: +39-0815667670 Contributed equally to this article, so they are co-first authors. y Received: 28 August 2020; Accepted: 21 September 2020; Published: 25 September 2020 Abstract: (1) Background: Glycogen storage disease (GSD) represents a group of twenty-three types of metabolic disorders which damage the capacity of body to store glucose classified basing on the enzyme deficiency involved. Affected patients could present some oro-facial alterations: the purpose of this review is to catalog and characterize oral manifestations in these patients. (2) Methods: a systematic review of the literature among different search engines using PICOS criteria has been performed. The studies were included with the following criteria: tissues and anatomical structures of the oral cavity in humans, published in English, and available full text. -
Esthetic Treatment of Anterior Spacings in a Patient with Localized
pISSN, eISSN 0125-5614 Case Report M Dent J 2019; 39 (2) : 53-63 Esthetic treatment of anterior spacings in a patient with localized microdontia using no-prep veneers combined with periodontal surgery: A clinical report Pajaree Limothai, Chalermpol Leevailoj Esthetic Restorative and Implant Dentistry, Faculty of Dentistry, Chulalongkorn University Objectives: This case report describes the treatment of a patient with maxillary anterior spacings, resulting from microdontia, using a multidisciplinary approach to improve her esthetic appearance. Materials and Methods: A 23-year-old Thai female patient had multiple spaces between her maxillary anterior teeth with a high smile line and an unsymmetrical gingival level. The Recurring Esthetic Dental (RED) proportion was used to determine the widths of maxillary teeth and Bolton’s analysis was used to confirm the RED results, after that a diagnostic wax-up model was fabricated. Esthetic crown lengthening was performed from the right maxillary canine to the left maxillary canine to reduce excess gingival exposure and increase the length of the teeth according to the proportion acquired from the calculation. After complete gingival healing, no-prep ceramic veneers were placed on the maxillary anterior teeth using the IPS Empress® Esthetic ceramic system. Results: The no-prep veneers preserved all tooth structures and gave a satisfactory esthetic result. The patient was satisfied with the outcome. The final restorations closed the spaces with the natural appearance the patient desired. The function and occlusion of the restorations were good. The veneers and the periodontal tissues were in good condition at the 1-year recall. Conclusion: The multidisciplinary approach and no-prep ceramic veneers used in this case restored the maxillary anterior spacing and provided an excellent esthetic outcome. -
Oral Manifestations of Red Blood C Research Article
z Available online at http://www.journalcra.com INTERNATIONAL JOURNAL OF CURRENT RESEARCH International Journal of Current Research Vol. 10, Issue, 11, pp.75681-75686, November, 2018 DOI: https://doi.org/10.24941/ijcr.33312.11.2018 ISSN: 0975-833X RESEARCH ARTICLE ORAL MANIFESTATIONS OF RED BLOOD CELL DISORDERS: A RECENT ANATOMIZATION 1Swatantra Shrivastava, 2Sourabh Sahu, 3Pavan Kumar Singh, 4Rajeev Kumar Shrivastava, 5, *Soumendu Bikash Maiti and 6Stuti Shukla 1Department of Oral Medicine and Radiology, New Horizon Dental College and Research Institute, Bilaspur, India 2MGM Medical College and Hospital, Aurangabad, India 3Department of Public Health Dentistry, Vyas Dental College and Hospital, India 4Master of Dental Surgery , Department of Prosthodontics and Crown and Bridge, New Horizon Dental College and Research Institute, Bilaspur, Udaipur, India 5Senior Lecturer, Department of Oral Medicine and Radiology, Pacific Dental College and Research Center, India 6Post Graduate, Department of Oral Medicine and Radiology, New horizon Dental College and Research center, Bilaspur India ARTICLE INFO ABSTRACT Article History: Primary objective of the literature is recognize and evaluate the wide array of oral manifestations Received 17th August, 2018 associated with red blood cell disorders which would eventually aid in diagnosis of the lesions Received in revised form associated with the disorders for the practitioners. It starts with petechiae, spontaneous gingival 03rd September, 2018 bleeding, herpetic infection in aplastic anaemia to hunter’s glossitis in pernicious anaemia. Literature Accepted 26th October, 2018 also includes enamel hypoplasia associated with erythroblastis fetalis, atrophic glossitis in iron th Published online 30 November, 2018 deficiency anemia with symptom of glossodynia in megaloblastic anemia. Marked manifestations of pharyngo-esophageal ulcerations and esophageal webs seen in plummer Vinson syndrome and Key Words: periodontitis, taurodontism, agenesia, supernumerary teeth to be seen in fanconi’s anemia. -
Associated Dental Anomalies: the Orthodontist Decoding the Genetics Which Regulates the Dental Development Disturbances
T ÓPICO E SP E CIAL Anomalias dentárias associadas: o ortodontista decodificando a genética que rege os distúrbios de desenvolvimento dentário Daniela Gamba Garib*, Bárbara Maria Alencar**, Flávio Vellini Ferreira***, Terumi Okada Ozawa**** Resumo O presente trabalho versa sobre o diagnóstico e a abordagem ortodôntica das anomalias dentárias, enfatizando os aspectos etiológicos que definem tais irregularidades de desenvolvimento. Parece existir uma inter-relação genética na determinação de algumas dessas anomalias, considerando-se a alta frequência de associações. Um mesmo defeito genético pode originar diferentes manifestações fenotípicas, incluindo agenesias, microdontias, ectopias e atraso no desenvolvimento dentário. As implicações clínicas das anomalias dentárias associadas são muito relevantes, uma vez que o diag- nóstico precoce de uma determinada anomalia dentária pode alertar o clínico sobre a possibilidade de desenvolvimento de outras anomalias associadas no mesmo paciente ou em outros membros da família, permitindo a intervenção ortodôntica em época oportuna. Palavras-chave: Genética. Anomalias dentárias. Agenesia. Etiologia. Ortodontia. INTRODUÇÃO anomalias dentárias. As anomalias expressam-se Caracterizada por complexos e precisos pro- com distintos graus de severidade. Da manifesta- cessos biológicos de substituição de dentes decí- ção mais branda para a mais severa – representa- duos por dentes permanentes, a dentadura mista das, respectivamente, desde o atraso cronológico representa uma das manifestações de perfeição na -
Description Concept ID Synonyms Definition
Description Concept ID Synonyms Definition Category ABNORMALITIES OF TEETH 426390 Subcategory Cementum Defect 399115 Cementum aplasia 346218 Absence or paucity of cellular cementum (seen in hypophosphatasia) Cementum hypoplasia 180000 Hypocementosis Disturbance in structure of cementum, often seen in Juvenile periodontitis Florid cemento-osseous dysplasia 958771 Familial multiple cementoma; Florid osseous dysplasia Diffuse, multifocal cementosseous dysplasia Hypercementosis (Cementation 901056 Cementation hyperplasia; Cementosis; Cementum An idiopathic, non-neoplastic condition characterized by the excessive hyperplasia) hyperplasia buildup of normal cementum (calcified tissue) on the roots of one or more teeth Hypophosphatasia 976620 Hypophosphatasia mild; Phosphoethanol-aminuria Cementum defect; Autosomal recessive hereditary disease characterized by deficiency of alkaline phosphatase Odontohypophosphatasia 976622 Hypophosphatasia in which dental findings are the predominant manifestations of the disease Pulp sclerosis 179199 Dentin sclerosis Dentinal reaction to aging OR mild irritation Subcategory Dentin Defect 515523 Dentinogenesis imperfecta (Shell Teeth) 856459 Dentin, Hereditary Opalescent; Shell Teeth Dentin Defect; Autosomal dominant genetic disorder of tooth development Dentinogenesis Imperfecta - Shield I 977473 Dentin, Hereditary Opalescent; Shell Teeth Dentin Defect; Autosomal dominant genetic disorder of tooth development Dentinogenesis Imperfecta - Shield II 976722 Dentin, Hereditary Opalescent; Shell Teeth Dentin Defect; -
Oral Manifestations of Ehlers-Danlos Syndrome
C LINICAL P RACTICE Oral Manifestations of Ehlers-Danlos Syndrome • Yves Létourneau, DMD • • Rénald Pérusse, DMD, MD • • Hélène Buithieu, DMD, MSD • Abstract Ehlers-Danlos syndrome is a rare hereditary disease of the connective tissue which can present oral manifestations. A brief history of the disease is presented along with the epidemiology and characteristics of the 8 main phenotypes of the syndrome. The article also describes the case of a 12-year-old patient presenting with hypermobility of the temporo-mandibular joint and capillary fragility, and highlights the precautions to take when treating patients with this syndrome. MeSH Key Words: case report; dental care for chronically ill; Ehlers-Danlos syndrome © J Can Dent Assoc 2001; 67:330-4 This article has been peer reviewed. hlers-Danlos syndrome (EDS) is a hereditary collagen adc/jcda/vol-67/issue-6/330.html). Only 4 types of EDS, disease presenting primarily as dermatological and namely types IV, VI, VII6 and X5 can be confirmed by E joint disorders. The first description of the syndrome biochemical and molecular tests. Since humans possess 19 in the literature was of a young Spaniard who was able to types of collagen, it is especially difficult to establish a stretch the skin overlying his right pectoral muscle over to the precise diagnosis.7 Even when there is a bleeding disorder left angle of his mandible.1 In 1901, Ehlers described the associated with the syndrome, blood analyses are not diag- condition as a hyperelasticity of the skin and a strong tendency nostically useful, in that no correlation has been made to bruising. -
Esthetic Treatment of Anterior Spacings in a Patient with Localized
pISSN, eISSN 0125-5614 Case Report M Dent J 2019; 39 (2) : 115-125 Esthetic treatment of anterior spacings in a patient with localized microdontia using no-prep veneers combined with periodontal surgery: A clinical report Pajaree Limothai, Chalermpol Leevailoj Esthetic Restorative and Implant Dentistry, Faculty of Dentistry, Chulalongkorn University Objectives: This case report describes the treatment of a patient with maxillary anterior spacings, resulting from microdontia, using a multidisciplinary approach to improve her esthetic appearance. Materials and Methods: A 23-year-old Thai female patient had multiple spaces between her maxillary anterior teeth with a high smile line and an unsymmetrical gingival level. The Recurring Esthetic Dental (RED) proportion was used to determine the widths of maxillary teeth and Bolton’s analysis was used to confirm the RED results, after that a diagnostic wax-up model was fabricated. Esthetic crown lengthening was performed from the right maxillary canine to the left maxillary canine to reduce excess gingival exposure and increase the length of the teeth according to the proportion acquired from the calculation. After complete gingival healing, no-prep ceramic veneers were placed on the maxillary anterior teeth using the IPS Empress® Esthetic ceramic system. Results: The no-prep veneers preserved all tooth structures and gave a satisfactory esthetic result. The patient was satisfied with the outcome. The final restorations closed the spaces with the natural appearance the patient desired. The function and occlusion of the restorations were good. The veneers and the periodontal tissues were in good condition at the 1-year recall. Conclusion: The multidisciplinary approach and no-prep ceramic veneers used in this case restored the maxillary anterior spacing and provided an excellent esthetic outcome. -
Cornea and External Disease Robert Cykiert, M.D
Cornea and External Disease Robert Cykiert, M.D. I. Basics papilla vascular response if giant, the differential includes atopy, vernal, GPC, prosthesis, suture follicles lymphatic response acute chronic EKC, pharygoconjunctival fever adult inclusion conjunctivitis medicamentosa (epinephrine, neosynephrine) toxic Parinaud's oculoglandular,syndrome r/o sarcoid HSV primary conjunctivitis r/o GPC, vernal conjunctivitis Newcastle's conjunctivitis with acute follicles, always check lid margin for HSV vesicles, ulcers membranes conjunctivitis ocular cicatricial pemphigoid erythema multiforme Stevens Johnson syndrome Srogrens syndrome atopy Symblepharon scieroderrna burns radiation burns trachoma EKC sarcoid drugs filaments exposure (keratoconjunctivitis sicca, neurotrophic, patching recurrent erosion) bullous keratopathy HSV meds superior limbic keratoconjunctivitis psoriasis aerosol keratitis diabetes mellitus radiation retained FB Thygeson's SPK ptosis Enlarged Corneal nerves MEN TIIb icthyosis Hanson's Kconus Refsums Fuchs corneal dystrophy old age failed PKP congenital glaucoma trauma neurofibromatosis MEN TIIb AD with thick corneal nerves, medullary thyroid cancer, pheochromocytoma, mucosal neuromas, and marfanoid habitus thickened lid margin with rostral lashes, thick lips, epibulbar neuromas cafe au lait spots, periungual, lingual neuromas often confused with NFI often die early from amyloid producing thyroid cancer in 10-20 year old with distant mets at dx thick nerves precede the cancer! corneal edema whenever epithelium disrupted, -
DH 318 General and Oral Pathology
DH 248 General and Oral Pathology Spring 2014 Meeting Times: Tuesday & Thursday 10:00 - 11:50 a.m. CASA Mortuary Science Room 70 Credits: 4 credit hours Faculty: Sherri Lukes, RDH, MS, Associate Professor, Room 129 Office: 453-7289 Cell: 521-3392 E-mail: [email protected] Office Hours: Monday 1:00 p.m. - 4:00 p.m. Tuesday 1:00-4:00 Other office hours by appointment COURSE DESCRIPTION: This course has been designed to integrate oral pathology and general pathology. Students will study principles of general pathology with emphasis on the relationships to oral diseases. Pathologic physiology is included such as tissue regeneration, the inflammatory process, immunology and wound healing. Clinical appearance, etiology, location and treatment options of general system diseases is presented, along with the oral manifestations. Special attention will be placed on common pathological conditions of the oral cavity and early recognition of these conditions. DH Competencies addressed in the course: PC.1 Systematically collect analyze, and record data on the general, oral, and psychosocial health status of a variety of patients/clients using methods consistent with medico-legal principles. PC.2 Use critical decision making skills to reach conclusions about the patient’s/client’s dental hygiene needs based on all available assessment data. PC.3 Collaborate with the patient / client, and/or other health professionals, to formulate a com- prehensive dental hygiene care plan that is patient / client-centered and based on current scientific evidence. PC.4 Provide specialized treatment that includes preventive and therapeutic services designed to achieve and maintain oral health. Assist in achieving oral health goals formulated in collaboration with the patient / client.