The Cambridge Historical Dictionary of Disease

Total Page:16

File Type:pdf, Size:1020Kb

The Cambridge Historical Dictionary of Disease P1: FCH/SPH P2: FCH/SPH QC: FCH/SPH T1: FCH CB518-FM CB518-Kiple-V1.cls March 4, 2003 15:23 THE CAMBRIDGE HISTORICAL DICTIONARY OF DISEASE Edited by KENNETHKENNETH F.F. KIPLEKIPLE Bowling Green State University iii P1: FCH/SPH P2: FCH/SPH QC: FCH/SPH T1: FCH CB518-FM CB518-Kiple-V1.cls March 4, 2003 15:23 PUBLISHED BY THE PRESS SYNDICATE OF THE UNIVERSITY OF CAMBRIDGE The Pitt Building, Trumpington Street, Cambridge, United Kingdom CAMBRIDGE UNIVERSITY PRESS The Edinburgh Building, Cambridge CB2 2RU, UK 40 West 20th Street, New York, NY 10011-4211, USA 477 Williamstown Road, Port Melbourne, VIC 3207, Australia Ruiz de Alarcon´ 13, 28014 Madrid, Spain Dock House, The Waterfront, Cape Town 8001, South Africa http://www.cambridge.org C Cambridge University Press 2003 This book is in copyright. Subject to statutory exception and to the provisions of relevant collective licensing agreements, no reproduction of any part may take place without the written permission of Cambridge University Press. First published 2003 Printed in the United States of America Typefaces Poppl-Pontifex 8.75/12 pt. and Poppl-Laudatio System LATEX2ε [TB] A catalog record for this book is available from the British Library. Library of Congress Cataloging in Publication Data The Cambridge historical dictionary of disease / edited by Kenneth F. Kiple. p. cm. Includes bibliographical references and index. ISBN 0-521-80834-0 – ISBN 0-521-53026-1 (pbk.) 1. Diseases – History – Dictionaries. I. Kiple, Kenneth F., 1939– RC41 .C365 2003 616 .009 – dc21 2002031368 ISBN 0 521 80834 0 hardback ISBN 0 521 53026 1 paperback iv P1: FCH/SPH P2: FCH/SPH QC: FCH/SPH T1: FCH CB518-FM CB518-Kiple-V1.cls March 4, 2003 15:23 Contents Preface I xiii 15 Beriberi I 44 Melinda S. Meade 1 Acquired Immune Deficiency Syndrome (AIDS) I 1 16 Black Death I 49 Allan M. Brandt Katharine Park 2 African Trypanosomiasis (Sleeping 17 Black and Brown Lung Disease I 52 Sickness) I 7 Daniel M. Fox Maryinez Lyons 18 Bleeding Disorders I 54 3 Ainhum I 14 Oscar D. Ratnoff Donald B. Cooper 19 Botulism I 57 4 Alzheimer’s Disease I 14 William H. Barker Joseph A. Kwentus 20 Brucellosis (Malta Fever, Undulant Fever) 5 Amebic Dysentery I 19 I 58 K. David Patterson Lise Wilkinson 6 Anemia I 21 21 Bubonic Plague I 60 Alfred Jay Bollet and Audrey K. Brown Ann G. Carmichael 7 Anorexia Nervosa I 26 22 Cancer I 63 Heather Munro Prescott Thomas G. Benedek and Kenneth F. Kiple 8 Anthrax I 29 23 Carrion’s Disease (Oroya Fever) I 67 Lise Wilkinson Oscar Urteaga-Ballon 9 Apoplexy and Stroke I 31 24 Catarrh I 69 Jacques Poirier and Christian Derouesne Roger K. French 10 Arboviruses I 34 25 Cestode Infection I 70 Wilbur G. Downs K. David Patterson 11 Arenaviruses I 38 26 Chagas’ Disease I 70 Wilbur G. Downs Marvin J. Allison 12 Arthritis (Rheumatoid) I 39 27 Chlorosis I 71 Howard Duncan and James C. C. Leisen Robert P. Hudson 13 Ascariasis I 42 28 Cholera I 74 K. David Patterson Reinhard S. Speck 14 Bacillary Dysentery I 43 29 Cirrhosis I 79 K. David Patterson Thomas S. N. Chen and Peter S. Y. Chen vii P1: FCH/SPH P2: FCH/SPH QC: FCH/SPH T1: FCH CB518-FM CB518-Kiple-V1.cls March 4, 2003 15:23 Contents 30 Clonorchiasis I 81 48 Enterobiasis I 116 K. David Patterson K. David Patterson 31 Croup I 81 49 Epilepsy I 116 James D. Cherry Jerrold E. Levy 32 Cystic Fibrosis I 83 50 Ergotism I 120 Thomas G. Benedek John S. Haller, Jr. 33 Cytomegalovirus Infection I 84 51 Erysipelas I 121 R. H. Kampmeier Ann G. Carmichael 34 Dengue I 85 52 Fascioliasis I 123 James McSherry K. David Patterson I 35 Diabetes I 88 53 Fasciolopsiasis 123 Leslie Sue Lieberman K. David Patterson I 36 Diarrheal Diseases (Acute) I 92 54 Favism 123 Herbert L. DuPont Peter J. Brown 55 Filariasis I 125 37 Diphtheria I 94 Todd L. Savitt Ann G. Carmichael 56 Fungus Infections (Mycoses) I 128 38 Down Syndrome I 96 Geoffrey C. Ainsworth Christine E. Cronk 57 Fungus Poisoning I 132 39 Dracunculiasis I 98 Geoffrey C. Ainsworth Donald R. Hopkins 58 Gallstones (Cholelithiasis) I 134 40 Dropsy I 100 R. Ted Steinbock J. Worth Estes 59 Gangrene I 136 I 41 Dysentery 105 Diane Quintal and Robert Jackson K. David Patterson 60 Genetic Disease I 138 I 42 Dyspepsia 105 Eric J. Devor James Whorton 61 Giardiasis I 144 I 43 Ebola Virus Disease 107 K. David Patterson Wilbur G. Downs 62 Glomerulonephritis (Bright’s Disease) I 44 Echinococcosis (Hydatidosis) I 110 144 K. David Patterson Donald M. Larson 45 Eclampsia I 110 63 Goiter I 146 Sally McMillen Clark T. Sawin 46 Emphysema I 112 64 Gonorrhea I 150 Ronald J. Knudson Richard B. Rothenberg 47 Encephalitis Lethargica I 114 65 Gout I 153 R. T. Ravenholt Thomas G. Benedek viii P1: FCH/SPH P2: FCH/SPH QC: FCH/SPH T1: FCH CB518-FM CB518-Kiple-V1.cls March 4, 2003 15:23 Contents 66 Heart-Related Diseases I 156 83 Leprosy (Hansen’s Disease) I 192 Joel D. Howell Ann G. Carmichael 67 Herpes Simplex I 161 84 Leptospirosis I 195 R. H. Kampmeier Otto R. Gsell 68 Herpesviruses I 162 85 Leukemia I 196 R. H. Kampmeier Gordon J. Piller I 69 Histoplasmosis I 163 86 Lupus Erythematosus 199 Scott F. Davies Thomas G. Benedek 87 Lyme Borreliosis (Lyme Disease) I 201 70 Hookworm Infection I 165 Robert D. Leff John Ettling 88 Malaria I 203 71 Huntington’s Disease (Chorea) I 168 Frederick L. Dunn Eric J. Devor 89 Marburg Virus Disease I 207 I 72 Hypertension 169 Wilbur G. Downs Thomas W. Wilson and Clarence E. Grim 90 Mastoiditis I 208 I 73 Infectious Hepatitis 171 John L. Kemink, John K. Niparko, and Steven A. Francis L. Black Telian 74 Infectious Mononucleosis I 174 91 Measles I 211 R. H. Kampmeier Robert J. Kim-Farley 75 Inflammatory Bowel Disease (Crohn’s 92 Meningitis I 214 Disease, Ulcerative Colitis) I 175 K. David Patterson Joseph B. Kirsner 93 Milk Sickness (Tremetol Poisoning) I 218 76 Influenza I 178 Thomas E. Cone, Jr. Alfred W. Crosby 94 Multiple Sclerosis I 220 77 Japanese B Encephalitis I 181 W. I. McDonald Edward H. Kass 95 Mumps I 222 78 Lactose Intolerance and Malabsorption I Robert J. Kim-Farley 182 96 Muscular Dystrophy I 224 Norman Kretchmer Thomas G. Benedek I 79 Lassa Fever 184 97 Myasthenia Gravis I 225 Wilbur G. Downs Bernard M. Patten 80 Lead Poisoning I 185 98 Nematode Infection I 227 Arthur C. Aufderheide K. David Patterson 81 Legionnaires’ Disease (Legionellosis, 99 Onchocerciasis I 228 Pontiac Fever, Legionella Pneumonia) I 189 K. David Patterson David W. Fraser 100 Ophthalmia (Trachoma, Conjunctivitis) 82 Leishmaniasis I 191 I 230 Marvin J. Allison Mary C. Karasch ix P1: FCH/SPH P2: FCH/SPH QC: FCH/SPH T1: FCH CB518-FM CB518-Kiple-V1.cls March 4, 2003 15:23 Contents 101 Osteoarthritis I 234 118 Rabies I 270 Charles W. Denko K. David Patterson 102 Osteoporosis I 236 119 Relapsing Fever I 275 R. Ted Steinbock Anne Hardy 103 Paget’s Disease of Bone I 238 120 Rheumatic Fever and Rheumatic Heart Roy D. Altman Disease I 277 Thomas G. Benedek 104 Paragonimiasis I 240 K. David Patterson 121 Rickets and Osteomalacia I 280 R. Ted Steinbock 105 Parkinson’s Disease (Parkinsonism) I 240 122 Rickettsial Diseases I 282 Bernard M. Patten Victoria A. Harden 106 Pellagra I 242 123 Rocky Mountain Spotted Fever and Elizabeth W. Etheridge Related Diseases I 283 Victoria A. Harden 107 Periodontal Disease (Pyorrhea) I 244 Jeffrey Levin 124 Rubella I 285 Robert J. Kim-Farley 108 Pica I 247 Brian T. Higgins 125 Saint Anthony’s Fire I 287 Ann G. Carmichael 109 Pinta I 250 Don R. Brothwell 126 Scarlet Fever I 288 Anne Hardy 110 Plague of Athens I 251 Ann G. Carmichael 127 Schistosomiasis I 290 John Farley 111 Pneumocystis Pneumonia (Interstitial Plasma Cell Pneumonia, Pneumocystosis) I 128 Scrofula I 292 254 Roger K. French K. David Patterson 129 Scurvy I 295 112 Pneumonia I 255 Roger K. French Jacalyn Duffin 130 Sickle-Cell Anemia I 298 113 Poliomyelitis I 258 Georges C. Benjamin H. V. Wyatt 131 Smallpox I 300 114 Protein-Energy Malnutrition I 261 Alfred W. Crosby James L. Newman 132 Streptococcal Diseases I 304 115 Protozoan Infection I 265 Peter C. English K. David Patterson 133 Strongyloidiasis I 306 116 Puerperal Fever I 265 K. David Patterson K. Codell Carter 134 Sudden Infant Death Syndrome (SIDS) 117 Q Fever I 267 I 306 S. R. Palmer Todd L. Savitt x P1: FCH/SPH P2: FCH/SPH QC: FCH/SPH T1: FCH CB518-FM CB518-Kiple-V1.cls March 4, 2003 15:23 Contents 135 Sudden Unexplained Death Syndrome 149 Trichuriasis I 336 (Asian SUDS) I 309 K. David Patterson Neal R. Holtan 150 Tuberculosis I 336 136 Sweating Sickness I 311 William D. Johnston Ann G. Carmichael 151 Tularemia I 342 137 Syphilis I 312 Patrick D. Horne Jon Arrizabalaga 152 Typhoid Fever I 345 138 Syphilis, Nonvenereal I 317 Charles W. LeBaron and David N. Taylor Kenneth F. Kiple 153 Typhomalarial Fever I 349 I 139 Tapeworm Infection 319 Dale Smith K. David Patterson 154 Typhus, Epidemic I 352 I 140 Tay-Sachs Disease 320 Victoria A. Harden Bradford Towne 155 Typhus, Murine I 355 141 Tetanus I 323 Victoria A.
Recommended publications
  • Unsolved Mysteries
    © 1993 Nature Publishing Group http://www.nature.com/naturebiotechnology /COMMENTARY• Unsolved Mysteries BERNARD DIXON he hand of the Lord was against fever nine years earlier, and show that most of those the city with a very great destruc­ patients had also developed rising levels of antibodies tion: and he smote the men of the against Legionella pneumophila. city, both small and great, and But what are the prospects of applying PCR to the ' they had emerods in their secret study of pathogens from previous centuries? As with parts" (1 Samuel 5:9). animal cells, the question hinges largely on finding What on earth was going on microbial DNA that has not been denatured and which here? An outbreak of hemor­ therefore still contains meaningful coding sequences. rhoids? Venereal disease? Bibli­ It's a fanciful idea at first, but on reflection the cal citations are not exactly thick on the ground at possibility of retrieving such material seems more scientific meetings, but these were the quotes with plausible than might be imagined. which veteran microbiologist Chris Collins opened Viruses can be maintained indefinitely, like chemi­ part of the Society for Applied Bacteriology's Sum­ cals, on a laboratory shelf. There seems every reason mer Conference in Nottingham last month. Theses­ to suppose, therefore, that some viruses of past times sion ranged over several "old plagues," including may have persisted in, for example, permafrost or several of disputed identity. It also prompted sugges­ desiccated burial vaults. The prospect of viable organ­ tions that the polymerase chain reaction (PCR), now isms being released from such sites was taken very widely famed for the fact and fantasy of amplifying seriously by the World Health Organization during human and other DNA from ancient bones, might also the smallpox eradication campaign of the 1970s, be used to retrieve microbial DNA and thus learn when Peter Razzell of Bedford College, London, something about infections in the mists of history.
    [Show full text]
  • Were the English Sweating Sickness and the Picardy Sweat Caused by Hantaviruses?
    Viruses 2014, 6, 151-171; doi:10.3390/v6010151 OPEN ACCESS viruses ISSN 1999-4915 www.mdpi.com/journal/viruses Review Were the English Sweating Sickness and the Picardy Sweat Caused by Hantaviruses? Paul Heyman 1,2,*, Leopold Simons 1,2 and Christel Cochez 1,2 1 Research Laboratory for Vector-Borne Diseases, Queen Astrid Military Hospital, Brussels B-1120, Belgium; E-Mails: [email protected] (L.S.); [email protected] (C.C.) 2 Reference Laboratory for Hantavirus infections, Queen Astrid Military Hospital, Brussels B-1120, Belgium * Author to whom correspondence should be addressed; E-Mail: [email protected]; Tel.: +32-2-264-4044. Received: 12 October 2013; in revised form: 4 December 2013 / Accepted: 9 December 2013 / Published: 7 January 2014 Abstract: The English sweating sickness caused five devastating epidemics between 1485 and 1551, England was hit hardest, but on one occasion also mainland Europe, with mortality rates between 30% and 50%. The Picardy sweat emerged about 150 years after the English sweat disappeared, in 1718, in France. It caused 196 localized outbreaks and apparently in its turn disappeared in 1861. Both diseases have been the subject of numerous attempts to define their origin, but so far all efforts were in vain. Although both diseases occurred in different time frames and were geographically not overlapping, a common denominator could be what we know today as hantavirus infections. This review aims to shed light on the characteristics of both diseases from contemporary as well as current knowledge and suggests hantavirus infection as the most likely cause for the English sweating sickness as well as for the Picardy sweat.
    [Show full text]
  • Infection Control Through the Ages
    American Journal of Infection Control 40 (2012) 35-42 Contents lists available at ScienceDirect American Journal of Infection Control American Journal of Infection Control journal homepage: www.ajicjournal.org Major article Infection control through the ages Philip W. Smith MD a,*, Kristin Watkins MBA b, Angela Hewlett MD a a Division of Infectious Diseases, Department of Internal Medicine, University of Nebraska Medical Center, Omaha, NE b Center for Preparedness Education, College of Public Health, University of Nebraska Medical Center, Omaha, NE Key Words: To appreciate the current advances in the field of health care epidemiology, it is important to understand History the history of hospital infection control. Available historical sources were reviewed for 4 different Hospitals historical time periods: medieval, early modern, progressive, and posteWorld War II. Hospital settings Nosocomial for the time periods are described, with particular emphasis on the conditions related to hospital infections. Copyright Ó 2012 by the Association for Professionals in Infection Control and Epidemiology, Inc. Published by Elsevier Inc. All rights reserved. Approximately 1.7 million health careeassociated infections One of the few public health measures was the collection of (HAIs) occur in the United States each year.1 Hospital infection bodies of plague victims. The bodies were left in the street to be control programs are nearly universal in developed nations and have picked up by carts and placed in mass graves outside of town.3,4 significantly lowered the risk of acquiring a HAI since their inception Other infection control measures included hanging people who in the mid 20th century. As we debate the preventability of HAIs, as wandered in from an epidemic region into an uninfected area, well as the ethical and logistic aspects of patient safety, it is impor- shutting up plague victims in their homes, and burning clothing tant to recall the historical context of hospital infection control.
    [Show full text]
  • THESIS for DOCTORAL DEGREE (Ph.D.)
    From the Center for Infectious Medicine, Department of Medicine, Huddinge Karolinska Institutet, Stockholm, Sweden HANTAVIRUSES, ESCAPEES FROM THE DEATH ROW – VIRAL MECHANISMS TOWARDS APOPTOSIS RESISTANCE Carles Solà Riera Stockholm 2019 Front cover: “The anti-apoptotic engine of hantaviruses” A graphical representation of the strategies by which hantaviruses hinder the cellular signalling towards apoptosis: downregulation of death receptor 5 from the cell surface, interference with mitochondrial membrane permeabilization, and direct inhibition of caspase-3 activity. All previously published papers were reproduced with permission from the publisher. Published by Karolinska Institutet. Printed by E-print AB 2019 © Carles Solà-Riera, 2019 ISBN 978-91-7831-525-3 Hantaviruses, escapees from the death row – Viral mechanisms towards apoptosis resistance THESIS FOR DOCTORAL DEGREE (Ph.D.) By Carles Solà Riera Public defence: Friday 15th of November, 2019 at 09:30 am Lecture Hall 9Q Månen, Alfred Nobels allé 8, Huddinge Principal Supervisor: Opponent: Associate Professor Jonas Klingström PhD Christina Spiropoulou Karolinska Institutet Centers for Disease Control and Prevention, Department of Medicine, Huddinge Atlanta, Georgia, USA Center for Infectious Medicine Viral Special Pathogens Branch, NCEZID, DHCPP Co-supervisor(s): Examination Board: Professor Hans-Gustaf Ljunggren Associate Professor Lisa Westerberg Karolinska Institutet Karolinska Institutet Department of Medicine, Huddinge Department of Microbiology, Tumor and Cell Center for Infectious
    [Show full text]
  • Trends in Microbiology
    Trends in Microbiology Microbial Genomics of Ancient Plagues and Outbreaks --Manuscript Draft-- Manuscript Number: TIMI-D-16-00114R1 Article Type: Review Corresponding Author: Cheryl P Andam, Ph.D Harvard T. H. Chan School of Public Health Boston, MA UNITED STATES First Author: Cheryl P Andam, Ph.D Order of Authors: Cheryl P Andam, Ph.D Colin J Worby, Ph.D. Qiuzhi Chang Michael G Campana, Ph.D. Abstract: The recent use of next generation sequencing methods to investigate historical disease outbreaks has provided us with an unprecedented ability to address important and long-standing questions in epidemiology, pathogen evolution and human history. In this review, we present major findings that illustrate how microbial genomics has provided new insights into the nature and etiology of infectious diseases of historical importance, such as plague, tuberculosis, and leprosy. Sequenced isolates collected from archaeological remains also provide evidence for the timing of historical evolutionary events as well as geographic spread of these pathogens. Elucidating the genomic basis of virulence in historical diseases can provide relevant information on how we can effectively understand the emergence and re-emergence of infectious diseases today and in the future. © 2016. This manuscript version is made available under the CC-BY-NC-ND 4.0 license http://creativecommons.org/licenses/by-nc-nd/4.0/ Powered by Editorial Manager® and ProduXion Manager® from Aries Systems Corporation Trends Box 1 Trends 2 3 ñ Important challenges to ancient genomic analyses include limited DNA sampling and 4 methodological issues (DNA authentication, recovery, isolation, enrichment, 5 sequencing, false positives). 6 ñ Genome sequencing of pathogens from historically notable disease outbreaks 7 provides insight into the nature of long-term co-evolution of humans and pathogens.
    [Show full text]
  • The English Sweating Sickness, 1485-1551: a Viral Pulmonary Disease?
    Medical History, 1998, 42: 96-98 Comment The English Sweating Sickness, 1485-1551: A Viral Pulmonary Disease? MARK TAVINER, GUY THWAITES, VANYA GANT* A recent article in this journal describes an analysis of the 1551 outbreak of the sweating sickness.1 Dr Dyer's research, based on 680 extant parish registers, represents to date the most comprehensive and detailed analysis of the demographic impact of any of the five outbreaks of the sweating sickness of 1485, 1508, 1517, 1528 and 1551. Furthermore, his article supersedes previous analyses of the demographic impact of the sweating sickness based on either parish registers2 or testamentary evidence.3 Contemporary impressions of strong age, class, and sex predispositions of the victims of the sweating sickness to young, rich males are modified to give a more dispassionate and informed picture. He also shows how the sweating sickness was predominantly a rural rather than an urban disease, with a limited overall demographic impact, and that there may have been occurrences outside the five "classic" epidemic years.4 This extensive demographic material is then used to provide a fuller epidemiological explanation for the aetiology of the sweating sickness. The underlying hypothesis is that the causative agent of the sweating sickness was spread by human-to-human contact as well as initially through a zoonosis or an environmental vector. This suggestion of human- to-human transmission stems from two aspects of the register data: first, the observable sequences of gender biases and intra-familial trends of mortalities at a parish level, and secondly from the spread of the epidemic at a national level.
    [Show full text]
  • English Sweating Sickness and the 1529 Continental Outbreak”
    Phi Alpha Theta Pacific Northwest Conference, 8–10 April 2021 Anika Esther Martin, Eastern Washington University, undergraduate student, “The ‘English Bath’: English Sweating Sickness and the 1529 Continental Outbreak” Abstract: Sudor Anglicus, or "English Sweating Sickness," was a peculiar disease which afflicted England during the Tudor period. First appearing in the late summer of 1485, Sweating Sickness quickly proved itself to be a terrifying killer. Those who contracted the Sweat were struck ill suddenly, often died within the first twenty-four hours, and suffered from a host of symptoms, the most visible of which being a raging fever and oppressive sweat. Between 1485 and 1551, five major outbreaks of the disease wracked the country, attracting the worried attention of those beyond England. In 1529, those anxieties were realized when a German ship unknowingly carried twelve sick passengers into the city of Hamburg, Germany, introducing the Sweat to Europe. This paper centers on the 1529 continental outbreak of English Sweating Sickness, tracking its path across Europe and exploring the different ways in which mainland Europeans reacted to the disease. Did they follow English precedents in handling the Sweat; or did they develop new policies and remedies? In approaching these questions, I will pay special attention to the German public health system, including a discussion of their city quarantines and medical pamphlets. The “English Bath:” English Sweating Sickness and the 1529 Continental Outbreak Anika Esther Martin Eastern Washington University Undergraduate [email protected] Abstract: 1 Sudor Anglicus, or "English Sweating Sickness," was a peculiar disease which afflicted England during the Tudor period.
    [Show full text]
  • (SARS-Cov-2, 2019-Coronavirus). J Mycol Mycological Sci 2020, 3(1): 000123
    Open Access Journal of Mycology & Mycological Sciences ISSN: 2689-7822 MEDWIN PUBLISHERS Committed to Create Value for Researchers The Historical/Evolutionary Cause and Possible Treatment of Pandemic COVID-19 (SARS-CoV-2, 2019-Coronavirus) Niknamian S* Associate Professor of Medicine, John D Dingell VA Medical Center and Wayne State University, Review Article USA Volume 3 Issue 1 Received Date: May 02, 2020 *Corresponding author: Sorush Niknamian, Chief, Division of Infectious Diseases, John D Published Date: June 15, 2020 Dingell VA Medical Center, Associate Professor of Medicine, Wayne State University Detroit, MI DOI: 10.23880/oajmms-16000123 USA, Tel: 313-576-3057; Fax: 313-576-1242; Email: [email protected] Abstract Background: A virus is a small infectious agent that replicates only inside the living cells of an organism. Viruses can infect all types of life forms, from animals and plants to microorganisms, including bacteria and archaea. In evolution, viruses are an important means of horizontal gene transfer, which increases genetic diversity in a way virus. Some viruses especially smallpox, throughout history, has killed between 300-500 million people in its 12,000- analogous to sexual reproduction. Influenza (Including (COVID-19), is an infectious disease caused by an influenza year existence. As modern humans increased in numbers, new infectious diseases emerged, including SARS-CoV-2. We have two groups of virus, RNA and DNA viruses. The most brutal viruses are RNA ones like COVID-19 Sars-CoV-2. Introduction: Coronaviruses are a group of viruses that cause diseases in mammals and birds. In humans, coronaviruses cause respiratory tract infections that are typically mild, such as some cases of the common cold and COVID-19.
    [Show full text]
  • Bislol'y 01 Medicine the SWEATING SICKNESS in ENGLAND* ARCHIBALD W
    24 April 1971 S.A. MEDICAL JOURNAL 473 Bislol'Y 01 Medicine THE SWEATING SICKNESS IN ENGLAND* ARCHIBALD W. SLOAN, Professor of Physiology, University of Cape Town SUMMARY blood with a most ardent heat ... so that, of all of them An acute infect;ous fever, called the sweating sickness, that sickened, there was not one among a hundred that broke out in England in five major epidemics in the years escaped." 1485, 1508, 1517, 1528 and 1551. Only one epidemic, thal HoIinshed, who closely follows Hall's description of the of 1528, spread also on the continent of Europe. The outbreak, states that the epidemic began on about 21 disease I-vas characterized by headache, pain in the chest, September and lasted until the end of October," but Webb and profuse sweating, and frequently proved fatal within maintains that it reached Oxford at the end of August, 24 hours. it can be distinguished from plague, malaria, and killing many students and causing many others to flee, and typhus, all of which were prevalent in the 161h century, reached London only a few days later: and was probably not influenza but anoTher virus infection It is widely assumed that the disease was brought into which has not reappeared in England since 1551. the country by the mercenary troops which the Earl of Richmond (later Henry VII) brought with him from France One of the unsolved mysteries of medical history is the to the battle of Bosworth Field (22 August 1485), but nature of the sweating sickness, an acute and often fatal there is no account of any such epidemic at the time on illness, which swept across England in five great epidemics the continent of Europe.
    [Show full text]
  • An Assessment of the “Sweating Sickness” Affecting England During the Tudor Dynasty
    AN ABSTRACT OF THE DISSERTATION OF Edwin Del Wollert for the degree of Doctor of Philosophy in History of Science presented on March 15, 2017. Title: An Assessment of the “Sweating Sickness” Affecting England During the Tudor Dynasty Abstract approved: Paul E. Kopperman Abstract. While historiography and interest in Tudor England at both the popular and specialist levels presents few signs of diminishing, there may nonetheless exist a sense that we have little left to learn about this period and its culture. A notable gap in our knowledge, however, remains regarding the mysterious disease known only as “sweating sickness” or sudor anglicus. This dissertation addresses and evaluates this disease from the perspective of the history of science, and in doing so, it makes three key arguments. First, this project examines how the early modern science and medicine known and practiced by Tudor subjects influenced their perceptions of this new disease, leaving them in a mostly helpless position from which to combat it and indeed often wondering if the unknown illness might represent a divine judgment, especially in the form of questioning a dubious claim to monarchy made by the first Tudor ruler, Henry VII. Second, the dissertation offers a detailed and layered thesis concluding that the disease was ultimately caused by an earlier version of the louping-ill virus, or LIV, a virus and accompanying illness which continued to affect parts of Western Europe, with its own unique strain still extant within Britain. The third argument will return to the opening statement of this abstract, and reveal how this more thorough and unique treatment of Tudor historiography does much to further our understanding of the Tudors and their citizens, all the more relevant since the “Sweat” even now is typically either mentioned in passing, or not at all, but those who write about this period of history.
    [Show full text]
  • On the Origin of Influenza a Hemagglutinin
    Indian352 J Microbiol (December 2009) 49:352–357 Indian J Microbiol (December 2009) 49:352–357 ORIGINAL ARTICLE On the origin of infl uenza A hemagglutinin Derek Gatherer Received: 22 September 2009 / Accepted: 30 September 2009 © Association of Microbiologists of India 2009 Abstract Recent advances in phylogenetic methods have produced some reassessments of the ages of the most recent Introduction common ancestor of hemagglutinin proteins in known strains of infl uenza A. This paper applies Bayesian Infl uenza A was the cause of three pandemics in the phylogenetic analysis implemented in BEAST to date the 20th century, in 1918, 1957 and 1968, associated with nodes on the infl uenza A hemagglutinin tree. The most recent hemagglutinin serotypes H1, H2 and H3 respectively. common ancestor (MRCA) of infl uenza A hemagglutinin The arrival of novel hemagglutinin serotypes in infl uenza proteins is located with 95% confi dence between 517 and A viruses infecting human populations is referred to as 1497 of the Common Era (AD), with the center of the antigenic shift. Sixteen different serotypes of infl uenza probability distribution at 1056 AD. The implications of this A hemagglutinin are found in avian wildfowl, the major revised dating for both historical and current epidemiology reservoir of the disease, and it is believed that zoonotic are discussed. Infl uenza A can be seen as an emerging transfer from avian species, is the major mechanism for disease of mediaeval and early modern times. antigenic shift in mammals. In addition to the three serotypes introduced to humans in the 20th century pandemics, H5, H7 and H9 have also been found in human infl uenza A cases, Keywords Infl uenza A · Hemagglutinin · H1N1 although without extensive human-to-human transmission.
    [Show full text]
  • DECEASED DISEASES* by DAVID RIESMAN, M.D., SC.D
    DECEASED DISEASES* By DAVID RIESMAN, M.D., SC.D. PHILADELPHIA ISEASES, at least many of conditions, but war may revive them. them, are like human be- Of the diseases that have died be- ings. They are born, they cause their causal agent has become flourish and they die. Some spontaneously extinct, the sweating may be eternal or at least coevalsickness with is one of the most interesting Dthe race, but seeing how many have examples. As far as we know this disease disappeared or are in the process of dis- has disappeared utterly. Also known as appearing, it would hardly be a wise the English sweat, it was a devastating prophecy to predict eternity for any of pestilence, of whose symptomatology, them. however, we have little knowledge. One Diseases may die from a variety of of the best descriptive accounts is by causes—thus the agent causing them Dr. John Caius or Kaye. Nor have we may disappear, as in the case of the any ideas as to its etiology. Some have sweating sickness of the Middle Ages. identified it with influenza, but I be- Some have become rare or nearly ex- lieve on insufficient grounds. On the tinct through efficient sanitary meas- Continent, especially in France, the dis- ures of various kinds, as is true of lep- ease was accompanied by an eruption, rosy. In some cases the disappearance is hence the names suer miliaire, suette only apparent, having been brought migliare. about through a change in name. Oth- The sweating sickness appeared first ers have disappeared because they were in England after the battle of Bosworth, really not diseases at all but symptoms August 22, 1485.
    [Show full text]