Dissociative Disorders Types of Dissociative Disorders Dissociative
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Paranoid – Suspicious; Argumentative; Paranoid; Continually on The
Disorder Gathering 34, 36, 49 Answer Keys A N S W E R K E Y, Disorder Gathering 34 1. Avital Agoraphobia – 2. Ewelina Alcoholism – 3. Martyna Anorexia – 4. Clarissa Bipolar Personality Disorder –. 5. Lysette Bulimia – 6. Kev, Annabelle Co-Dependant Relationship – 7. Archer Cognitive Distortions / all-of-nothing thinking (Splitting) – 8. Josephine Cognitive Distortions / Mental Filter – 9. Mendel Cognitive Distortions / Disqualifying the Positive – 10. Melvira Cognitive Disorder / Labeling and Mislabeling – 11. Liat Cognitive Disorder / Personalization – 12. Noa Cognitive Disorder / Narcissistic Rage – 13. Regev Delusional Disorder – 14. Connor Dependant Relationship – 15. Moira Dissociative Amnesia / Psychogenic Amnesia – (*Jason Bourne character) 16. Eylam Dissociative Fugue / Psychogenic Fugue – 17. Amit Dissociative Identity Disorder / Multiple Personality Disorder – 18. Liam Echolalia – 19. Dax Factitous Disorder – 20. Lorna Neurotic Fear of the Future – 21. Ciaran Ganser Syndrome – 22. Jean-Pierre Korsakoff’s Syndrome – 23. Ivor Neurotic Paranoia – 24. Tucker Persecutory Delusions / Querulant Delusions – 25. Lewis Post-Traumatic Stress Disorder – 26. Abdul Proprioception – 27. Alisa Repressed Memories – 28. Kirk Schizophrenia – 29. Trevor Self-Victimization – 30. Jerome Shame-based Personality – 31. Aimee Stockholm Syndrome – 32. Delphine Taijin kyofusho (Japanese culture-specific syndrome) – 33. Lyndon Tourette’s Syndrome – 34. Adar Social phobias – A N S W E R K E Y, Disorder Gathering 36 Adjustment Disorder – BERKELEY Apotemnophilia -
Psychogenic and Organic Amnesia. a Multidimensional Assessment of Clinical, Neuroradiological, Neuropsychological and Psychopathological Features
Behavioural Neurology 18 (2007) 53–64 53 IOS Press Psychogenic and organic amnesia. A multidimensional assessment of clinical, neuroradiological, neuropsychological and psychopathological features Laura Serraa,∗, Lucia Faddaa,b, Ivana Buccionea, Carlo Caltagironea,b and Giovanni A. Carlesimoa,b aFondazione IRCCS Santa Lucia, Roma, Italy bClinica Neurologica, Universita` Tor Vergata, Roma, Italy Abstract. Psychogenic amnesia is a complex disorder characterised by a wide variety of symptoms. Consequently, in a number of cases it is difficult distinguish it from organic memory impairment. The present study reports a new case of global psychogenic amnesia compared with two patients with amnesia underlain by organic brain damage. Our aim was to identify features useful for distinguishing between psychogenic and organic forms of memory impairment. The findings show the usefulness of a multidimensional evaluation of clinical, neuroradiological, neuropsychological and psychopathological aspects, to provide convergent findings useful for differentiating the two forms of memory disorder. Keywords: Amnesia, psychogenic origin, organic origin 1. Introduction ness of the self – and a period of wandering. According to Kopelman [33], there are three main predisposing Psychogenic or dissociative amnesia (DSM-IV- factors for global psychogenic amnesia: i) a history of TR) [1] is a clinical syndrome characterised by a mem- transient, organic amnesia due to epilepsy [52], head ory disorder of nonorganic origin. Following Kopel- injury [4] or alcoholic blackouts [20]; ii) a history of man [31,33], psychogenic amnesia can either be sit- psychiatric disorders such as depressed mood, and iii) uation specific or global. Situation specific amnesia a severe precipitating stress, such as marital or emo- refers to memory loss for a particular incident or part tional discord [23], bereavement [49], financial prob- of an incident and can arise in a variety of circum- lems [23] or war [21,48]. -
Cotard's Syndrome: Two Case Reports and a Brief Review of Literature
Published online: 2019-09-26 Case Report Cotard’s syndrome: Two case reports and a brief review of literature Sandeep Grover, Jitender Aneja, Sonali Mahajan, Sannidhya Varma Department of Psychiatry, Post Graduate Institute of Medical Education and Research, Chandigarh, India ABSTRACT Cotard’s syndrome is a rare neuropsychiatric condition in which the patient denies existence of one’s own body to the extent of delusions of immortality. One of the consequences of Cotard’s syndrome is self‑starvation because of negation of existence of self. Although Cotard’s syndrome has been reported to be associated with various organic conditions and other forms of psychopathology, it is less often reported to be seen in patients with catatonia. In this report we present two cases of Cotard’s syndrome, both of whom had associated self‑starvation and nutritional deficiencies and one of whom had associated catatonia. Key words: Catatonia, Cotard’s syndrome, depression Introduction Case Report Cotard’s syndrome is a rare neuropsychiatric condition Case 1 characterized by anxious melancholia, delusions Mr. B, 65‑year‑old retired teacher who was pre‑morbidly of non‑existence concerning one’s own body to the well adjusted with no family history of mental illness, extent of delusions of immortality.[1] It has been most with personal history of smoking cigarettes in dependent commonly seen in patients with severe depression. pattern for last 30 years presented with an insidious However, now it is thought to be less common possibly onset mental illness of one and half years duration due to early institution of treatment in patients precipitated by psychosocial stressors. -
Does Amnesia Specifically Predict Alzheimer's Pathology?
Does amnesia specifically predict Alzheimer’s pathology? A neuropathological study Maxime Bertoux, Pascaline Cassagnaud, Thibaud Lebouvier, Florence Lebert, Marie Sarazin, Isabelle Le Ber, Bruno Dubois, Brain Bank, Sophie Auriacombe, Didier Hannequin, et al. To cite this version: Maxime Bertoux, Pascaline Cassagnaud, Thibaud Lebouvier, Florence Lebert, Marie Sarazin, et al.. Does amnesia specifically predict Alzheimer’s pathology? A neuropathological study. Neurobiology of Aging, Elsevier, In press. hal-02898941 HAL Id: hal-02898941 https://hal.archives-ouvertes.fr/hal-02898941 Submitted on 14 Jul 2020 HAL is a multi-disciplinary open access L’archive ouverte pluridisciplinaire HAL, est archive for the deposit and dissemination of sci- destinée au dépôt et à la diffusion de documents entific research documents, whether they are pub- scientifiques de niveau recherche, publiés ou non, lished or not. The documents may come from émanant des établissements d’enseignement et de teaching and research institutions in France or recherche français ou étrangers, des laboratoires abroad, or from public or private research centers. publics ou privés. Amnesia/AD pathology 1 Does amnesia specifically predict Alzheimer’s pathology? A neuropathological study. Maxime Bertoux*1a, Pascaline Cassagnaud*b, Thibaud Lebouvier*c, Florence Leberta, Marie Sarazinde, Isabelle Le Berfg, Bruno Duboisfg, NeuroCEB Brain Bank, Sophie Auriacombeh, Didier Hannequini, David Walloni, Mathieu Ceccaldij, Claude-Alain Mauragek, Vincent Deramecourtc, Florence Pasquiera a Univ Lille, Lille Neuroscience & Cognition (Inserm UMRS1172) Degenerative and vascular cognitive disorders, CHU Lille, Laboratory of Excellence Distalz (Development of Innovative Strategies for a Transdisciplinary approach to ALZheimer’s disease). F-59000, Lille, France.F-59000, Lille, France. b Univ Lille, CHU Lille, Laboratory of Excellence Distalz (Development of Innovative Strategies for a Transdisciplinary approach to ALZheimer’s disease). -
When the Mind Falters: Cognitive Losses in Dementia
T L C When the Mind Falters: Cognitive Losses in Dementia by L Joel Streim, MD T Associate Professor of Psychiatry C Director, Geriatric Psychiatry Fellowship Program University of Pennsylvania VISN 4 Mental Illness Research Education and Clinical Center Philadelphia VA Medical Center Delaware Valley Geriatric Education Center The goal of this module is to teach direct staff about the syndrome of dementia and its clinical effects on residents. It focuses on the ways that the symptoms of dementia affect persons’ functional ability and behavior. We begin with an overview of the symptoms of cognitive impairment. We continue with a description of the causes, epidemiology, and clinical course (stages) of dementia. We then turn to a closer look at the specific areas of cognitive impairment, and examine how deficits in different areas of cognitive function can interfere with the person’s daily functioning, causing disability. The accompanying videotape illustrates these principles, using the example of a nursing home resident whose cognitive impairment interferes in various ways with her eating behavior and ability to feed herself. 1 T L Objectives C At the end of this module you should be able to: Describe the stages of dementia Distinguish among specific cognitive impairments from dementia L Link specific cognitive impairments with the T disabilities they cause C Give examples of cognitive impairments and disabilities Describe what to do when there is an acute change in cognitive or functional status Delaware Valley Geriatric Education Center At the end of this module you should be able to • Describe the stages of dementia. These are early, middle and late, and we discuss them in more detail. -
PAVOL JOZEF ŠAFARIK UNIVERSITY in KOŠICE Dissociative Amnesia: a Clinical and Theoretical Reconsideration DEGREE THESIS
PAVOL JOZEF ŠAFARIK UNIVERSITY IN KOŠICE FACULTY OF MEDICINE Dissociative amnesia: a clinical and theoretical reconsideration Paulo Alexandre Rocha Simão DEGREE THESIS Košice 2017 PAVOL JOZEF ŠAFARIK UNIVERSITY IN KOŠICE FACULTY OF MEDICINE FIRST DEPARTMENT OF PSYCHIATRY Dissociative amnesia: a clinical and theoretical reconsideration Paulo Alexandre Rocha Simão DEGREE THESIS Thesis supervisor: Mgr. MUDr. Jozef Dragašek, PhD., MHA Košice 2017 Analytical sheet Author Paulo Alexandre Rocha Simão Thesis title Dissociative amnesia: a clinical and theoretical reconsideration Language of the thesis English Type of thesis Degree thesis Number of pages 89 Academic degree M.D. University Pavol Jozef Šafárik University in Košice Faculty Faculty of Medicine Department/Institute Department of Psychiatry Study branch General Medicine Study programme General Medicine City Košice Thesis supervisor Mgr. MUDr. Jozef Dragašek, PhD., MHA Date of submission 06/2017 Date of defence 09/2017 Key words Dissociative amnesia, dissociative fugue, dissociative identity disorder Thesis title in the Disociatívna amnézia: klinické a teoretické prehodnotenie Slovak language Key words in the Disociatívna amnézia, disociatívna fuga, disociatívna porucha identity Slovak language Abstract in the English language Dissociative amnesia is a one of the most intriguing, misdiagnosed conditions in the psychiatric world. Dissociative amnesia is related to other dissociative disorders, such as dissociative identity disorder and dissociative fugue. Its clinical features are known -
Department of Veterans Affairs § 4.130
Department of Veterans Affairs § 4.130 agency shall evaluate it using a diag- termine whether a change in evalua- nostic code which represents the domi- tion is warranted. nant (more disabling) aspect of the (Authority: 38 U.S.C. 1155) condition (see § 4.14). [61 FR 52700, Oct. 8, 1996] (Authority: 38 U.S.C. 1155) § 4.130 Schedule of ratings—Mental [61 FR 52700, Oct. 8, 1996, as amended at 79 FR disorders. 45099, Aug. 4, 2014] The nomenclature employed in this § 4.127 Intellectual disability (intellec- portion of the rating schedule is based tual developmental disorder) and upon the American Psychiatric Asso- personality disorders. ciation’s Diagnostic and Statistical Manual of Mental Disorders, Fifth Edi- Intellectual disability (intellectual tion (DSM–5) (see § 4.125 for availability developmental disorder) and person- information). Rating agencies must be ality disorders are not diseases or inju- thoroughly familiar with this manual ries for compensation purposes, and, to properly implement the directives in except as provided in § 3.310(a) of this § 4.125 through § 4.129 and to apply the chapter, disability resulting from them general rating formula for mental dis- may not be service-connected. How- orders in § 4.130. The schedule for rating ever, disability resulting from a men- for mental disorders is set forth as fol- tal disorder that is superimposed upon lows: intellectual disability (intellectual de- 9201 Schizophrenia velopmental disorder) or a personality 9202 [Removed] disorder may be service-connected. 9203 [Removed] 9204 [Removed] (Authority: 38 U.S.C. 1155) 9205 [Removed] [79 FR 45100, Aug. 4, 2014] 9208 Delusional disorder 9210 Other specified and unspecified schizo- § 4.128 Convalescence ratings fol- phrenia spectrum and other psychotic lowing extended hospitalization. -
The Effect of Depersonalization and Derealization Symptoms
THE EFFECT OF DEPERSONALIZATION AND DEREALIZATION SYMPTOMS ON OLFACTION AND OLFACTORY HEDONICS Thesis Submitted to The College of Arts and Sciences of the UNIVERSITY OF DAYTON In Partial Fulfillment of the Requirements for The Degree of Master of Arts in Psychology By Rhiannon A. Gibbs UNIVERSITY OF DAYTON Dayton, Ohio May 2018 THE EFFECT OF DEPERSONALIZATION AND DEREALIZATION SYMPTOMS ON OLFACTION AND OLFACTORY HEDONICS Name: Gibbs, Rhiannon A. APPROVED BY: _______________________________________ Julie Walsh-Messinger, Ph.D. Faculty Advisor ______________________________________ Roger R. Reeb, Ph.D. Committee Member ______________________________________ Jackson A. Goodnight, Ph.D. Committee Member Concurrence: _______________________________________ Lee Dixon, Ph.D. Chair, Department of Psychology ii © Copyright by Rhiannon A. Gibbs All rights reserved 2018 ABSTRACT THE EFFECT OF DEPERSONALIZATION AND DEREALIZATION SYMPTOMS ON OLFACTION AND OLFACTORY HEDONICS Name: Gibbs, Rhiannon A. University of Dayton Advisor: Dr. Julie Walsh-Messinger. Depersonalization and derealization symptoms affect sensation, perception, and emotion, producing subjective experiences of unreality and affective numbing (Simeon, 2004). Abnormalities in the amygdala, which is associated with emotional reactions such as anxiety and fear (LeDoux, 1993), have been observed in depersonalization and derealization and other psychiatric disorders, such as anxiety and depression (Sierra & Berrios, 1998). Olfactory deficits have been posited as a potential marker for psychiatric -
Dissociative Experiences in Bipolar Disorder II: Are They Related to Childhood Trauma and Obsessive-Compulsive Symptoms?
Original article Dissociative experiences in bipolar disorder II: Are they related to childhood trauma and obsessive-compulsive symptoms? GUL ERyılmaz1, SERMıN KESEBıR1, IşIl GöğceGöz Gül1, eylem özten1, KayIhan Oğuz KaramustafalIOğlu1 1 Uskudar University Neuropsychiatry Hospital, Psychiatry, Istanbul, Turkey. Received: 12/29/2014 – Accepted: 3/2/2015 DOI: 10.1590/0101-60830000000045 Abstract Objective: The aim of this study is to investigate the presence of dissociative symptoms and whether they are related to childhood trauma and obsessive- -compulsive symptoms in bipolar disorder type II (BD-II). Methods: Thirty-three euthymic patients (HDRS<8, YMRS<5) and 50 healthy subjects were eva- luated by SCID-I and SCID-NP. We excluded all first and second-axis comorbidities. All patients and healthy subjects were examined with the Dissociative Experiences Scale (DES), Childhood Trauma Questionnaire (CTQ-53), and Yale-Brown Obsessive-Compulsive Disorder scale (Y-BOCS). Results: In pairwise comparisons between the BD-II and control groups, the total CTQ, emotional abuse, emotional neglect, DES, and total Y-BOCS scores in the BD-II group were significantly higher than those in the control group (p < 0.05). There were five cases with DES scores over 30 (15.2%) and one case (2%) in the control group. DES was weakly correlated with total CTQ and Y-BOCS in patients diagnosed with BD-II (r = 0.278, p < 0.05 and r = 0.217, p < 0.05, respectively). While there was no correlation between total CTQ and Y-BOCS, the CTQ sexual abuse subscale was found to be related to Y-BOCS (r = 0.330, p < 0.05). -
The Three Amnesias
The Three Amnesias Russell M. Bauer, Ph.D. Department of Clinical and Health Psychology College of Public Health and Health Professions Evelyn F. and William L. McKnight Brain Institute University of Florida PO Box 100165 HSC Gainesville, FL 32610-0165 USA Bauer, R.M. (in press). The Three Amnesias. In J. Morgan and J.E. Ricker (Eds.), Textbook of Clinical Neuropsychology. Philadelphia: Taylor & Francis/Psychology Press. The Three Amnesias - 2 During the past five decades, our understanding of memory and its disorders has increased dramatically. In 1950, very little was known about the localization of brain lesions causing amnesia. Despite a few clues in earlier literature, it came as a complete surprise in the early 1950’s that bilateral medial temporal resection caused amnesia. The importance of the thalamus in memory was hardly suspected until the 1970’s and the basal forebrain was an area virtually unknown to clinicians before the 1980’s. An animal model of the amnesic syndrome was not developed until the 1970’s. The famous case of Henry M. (H.M.), published by Scoville and Milner (1957), marked the beginning of what has been called the “golden age of memory”. Since that time, experimental analyses of amnesic patients, coupled with meticulous clinical description, pathological analysis, and, more recently, structural and functional imaging, has led to a clearer understanding of the nature and characteristics of the human amnesic syndrome. The amnesic syndrome does not affect all kinds of memory, and, conversely, memory disordered patients without full-blown amnesia (e.g., patients with frontal lesions) may have impairment in those cognitive processes that normally support remembering. -
FF #145 Panic. 3Rd Ed
! FAST FACTS AND CONCEPTS #145 PANIC DISORDER AT THE END-OF-LIFE VJ Periyakoil MD Background Anxiety and fear occur commonly in the dying patient. However, disabling anxiety and/or panic is not a normal aspect of the dying process. Separating “normal” death-related anxiety from pathological panic is an important palliative care skill. Definitions • A panic attack is defined in the DSM-IV as “a discrete period of intense fear or discomfort, in which four (or more) of the following symptoms develop abruptly and reach a peak within 10 minutes: palpitations, pounding heart or accelerated heart rate, sweating, trembling or shaking, sensations of shortness of breath or smothering, feeling of choking, chest pain or discomfort, nausea or abdominal distress, feeling dizzy, unsteady, lightheaded or faint, derealization or depersonalization, fear of losing control or going crazy, fear of dying. • Derealization describes a sensation of feeling estranged or detached from one’s environment. • Depersonalization is an altered and unreal perception of self, one’s feelings and/or situation. Described by one patient as “feeling like you are on the outside looking in”. Diagnosis • A combination of physical symptoms (feeling dizzy, weak, nauseous, unsteady, lightheaded, breathless) and affective symptoms (fear of loss of control) are used to diagnose a panic disorder. • Terminally ill patients may often have many of the physical symptoms listed above as a part of their illness process. Thus the presence of derealization, depersonalization and fear of loss of control are more useful in making the diagnosis of panic disorder in the terminally ill. • A contributing feature to the diagnosis of panic disorder is if a patient develops recurrent symptoms, worries about future ‘attacks’ and alters her/his behavior in anticipation of such attacks. -
A Neurostructural Biomarker of Dissociative Amnesia: a Hippocampal Study in Dissociative Cambridge.Org/Psm Identity Disorder
Psychological Medicine A neurostructural biomarker of dissociative amnesia: a hippocampal study in dissociative cambridge.org/psm identity disorder 1,2, 3, 4,5,† Original Article Lora I. Dimitrova * , Sophie L. Dean *, Yolanda R. Schlumpf , Eline M. Vissia6,† , Ellert R. S. Nijenhuis5, Vasiliki Chatzi7, Lutz Jäncke4,8, *These authors contributed equally to this 2 9 1 work Dick J. Veltman , Sima Chalavi and Antje A. T. S. Reinders † These authors contributed equally to this 1Department of Psychological Medicine, Institute of Psychiatry, Psychology & Neuroscience, King’s College work London, London, UK; 2Department of Psychiatry, Amsterdam UMC, Location VUmc, VU University Amsterdam, Amsterdam, The Netherlands; 3Department of Psychosis Studies, Institute of Psychiatry, King’s College London, Cite this article: Dimitrova LI et al (2021). A 4 neurostructural biomarker of dissociative London, UK; Division of Neuropsychology, Department of Psychology, University of Zurich, Zurich, Switzerland; 5 6 amnesia: a hippocampal study in dissociative Clienia Littenheid AG, Private Clinic for Psychiatry and Psychotherapy, Littenheid, Switzerland; Heelzorg, Centre 7 identity disorder. Psychological Medicine 1–9. for Psychotrauma, Zwolle, The Netherlands; Department of Biomedical Engineering, King’s College London, https://doi.org/10.1017/S0033291721002154 London, UK; 8Research Unit for Plasticity and Learning of the Healthy Aging Brain, University of Zurich, Zurich, Switzerland and 9Movement Control and Neuroplasticity Research Group, Department of Movement Sciences, KU Received: 14 September 2020 Leuven, Leuven, Belgium Revised: 12 February 2021 Accepted: 11 May 2021 Abstract Key words: Background. Little is known about the neural correlates of dissociative amnesia, a transdiag- Dissociative experience scale; DES; childhood trauma; CA1; Freesurfer; dissociation nostic symptom mostly present in the dissociative disorders and core characteristic of dis- sociative identity disorder (DID).