A Novel Technique for Modification of Images for Deuteranopic Viewers
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Theoretical Part Eye Examinations 1
Name and Surrname number Study group Theoretical part Eye examinations 1. Astigmatism Astigmatism is an optical defect in which vision is blurred due to the inability of the optics of the eye to focus a point object into a sharp focused image on the retina. Astigmatism can sometimes be asymptomatic, while higher degrees of astigmatism may cause symptoms such as blurry vision, squinting, eye strain, fatigue, or headaches. Types Regular astigmatism: Principal meridians are perpendicular. Simple astigmatism – the first focal line is on the retina, while the second is located behind the retina, or, the first focal line is in front of the retina, while the second is on the retina. Compound astigmatism – both focal lines are located behind or before the retina. Mixed astigmatism – focal lines are on both sides of the retina (straddling the retina). Irregular astigmatism: Principal meridians are not perpendicular. This type cannot be corrected by a lens. Tests Objective Refractometer, autorefractometer Placido keratoscope – A placido keratoscope consists of a handle and a circular part with a hole in the middle. The hole with a magnifying glass is viewed from a distance of 10–15 cm to the patients cornea. In the 200 mm wide circular portion there are concentric alternating black and white circles. They reflect the patient’s cornea. In the event of astigmatism, a deformation appears at the corresponding location. Sciascope Ophthalmometry Subjective Fuchs figure – This is a tool for evaluating astigmatism where examinee stands up against a pattern of circular shape (circular or striped rectangles) and fixes his/her gaze on the center of the pattern with one open eye. -
Comprehensive Pediatric Eye and Vision Examination
American Optometric Association – Peer/Public Review Document 1 2 3 EVIDENCE-BASED CLINICAL PRACTICE GUIDELINE 4 5 6 7 8 9 10 11 12 13 14 15 16 17 Comprehensive 18 Pediatric Eye 19 and Vision 20 Examination 21 22 For Peer/Public Review May 16, 2016 23 American Optometric Association – Peer/Public Review Document 24 OPTOMETRY: THE PRIMARY EYE CARE PROFESSION 25 26 The American Optometric Association represents the thousands of doctors of optometry 27 throughout the United States who in a majority of communities are the only eye doctors. 28 Doctors of optometry provide primary eye care to tens of millions of Americans annually. 29 30 Doctors of optometry (O.D.s/optometrists) are the independent primary health care professionals for 31 the eye. Optometrists examine, diagnose, treat, and manage diseases, injuries, and disorders of the 32 visual system, the eye, and associated structures, as well as identify related systemic conditions 33 affecting the eye. Doctors of optometry prescribe medications, low vision rehabilitation, vision 34 therapy, spectacle lenses, contact lenses, and perform certain surgical procedures. 35 36 The mission of the profession of optometry is to fulfill the vision and eye care needs of the 37 public through clinical care, research, and education, all of which enhance quality of life. 38 39 40 Disclosure Statement 41 42 This Clinical Practice Guideline was funded by the American Optometric Association (AOA), 43 without financial support from any commercial sources. The Evidence-Based Optometry 44 Guideline Development Group and other guideline participants provided full written disclosure 45 of conflicts of interest prior to each meeting and prior to voting on the strength of evidence or 46 clinical recommendations contained within this guideline. -
Acquired Colour Vision Defects in Glaucoma—Their Detection and Clinical Significance
1396 Br J Ophthalmol 1999;83:1396–1402 Br J Ophthalmol: first published as 10.1136/bjo.83.12.1396 on 1 December 1999. Downloaded from PERSPECTIVE Acquired colour vision defects in glaucoma—their detection and clinical significance Mireia Pacheco-Cutillas, Arash Sahraie, David F Edgar Colour vision defects associated with ocular disease have The aims of this paper are: been reported since the 17th century. Köllner1 in 1912 + to provide a review of the modern literature on acquired wrote an acute description of the progressive nature of col- colour vision in POAG our vision loss secondary to ocular disease, dividing defects + to diVerentiate the characteristics of congenital and into “blue-yellow” and “progressive red-green blindness”.2 acquired defects, in order to understand the type of This classification has become known as Köllner’s rule, colour vision defect associated with glaucomatous although it is often imprecisely stated as “patients with damage retinal disease develop blue-yellow discrimination loss, + to compare classic clinical and modern methodologies whereas optic nerve disease causes red-green discrimina- (including modern computerised techniques) for tion loss”. Exceptions to Köllner’s rule34 include some assessing visual function mediated through chromatic optic nerve diseases, notably glaucoma, which are prima- mechanisms rily associated with blue-yellow defects, and also some reti- + to assess the eVects of acquired colour vision defects on nal disorders such as central cone degeneration which may quality of life in patients with POAG. result in red-green defects. Indeed, in some cases, there might be a non-specific chromatic loss. Comparing congenital and acquired colour vision Colour vision defects in glaucoma have been described defects since 18835 and although many early investigations Congenital colour vision deficiencies result from inherited indicated that red-green defects accompanied glaucoma- cone photopigment abnormalities. -
Method for Compensating for Color Differences Between Different Images of a Same Scene
(19) TZZ¥ZZ__T (11) EP 3 001 668 A1 (12) EUROPEAN PATENT APPLICATION (43) Date of publication: (51) Int Cl.: 30.03.2016 Bulletin 2016/13 H04N 1/60 (2006.01) (21) Application number: 14306471.5 (22) Date of filing: 24.09.2014 (84) Designated Contracting States: (72) Inventors: AL AT BE BG CH CY CZ DE DK EE ES FI FR GB • Sheikh Faridul, Hasan GR HR HU IE IS IT LI LT LU LV MC MK MT NL NO 35576 CESSON-SÉVIGNÉ (FR) PL PT RO RS SE SI SK SM TR • Stauder, Jurgen Designated Extension States: 35576 CESSON-SÉVIGNÉ (FR) BA ME • Serré, Catherine 35576 CESSON-SÉVIGNÉ (FR) (71) Applicants: • Tremeau, Alain • Thomson Licensing 42000 SAINT-ETIENNE (FR) 92130 Issy-les-Moulineaux (FR) • CENTRE NATIONAL DE (74) Representative: Browaeys, Jean-Philippe LA RECHERCHE SCIENTIFIQUE -CNRS- Technicolor 75794 Paris Cedex 16 (FR) 1, rue Jeanne d’Arc • Université Jean Monnet de Saint-Etienne 92443 Issy-les-Moulineaux (FR) 42023 Saint-Etienne Cedex 2 (FR) (54) Method for compensating for color differences between different images of a same scene (57) The method comprises the steps of: - for each combination of a first and second illuminants, applying its corresponding chromatic adaptation matrix to the colors of a first image to compensate such as to obtain chromatic adapted colors forming a chromatic adapted image and calculating the difference between the colors of a second image and the chromatic adapted colors of this chromatic adapted image, - retaining the combination of first and second illuminants for which the corresponding calculated difference is the smallest, - compensating said color differences by applying the chromatic adaptation matrix corresponding to said re- tained combination to the colors of said first image. -
Colour Vision Deficiency
Eye (2010) 24, 747–755 & 2010 Macmillan Publishers Limited All rights reserved 0950-222X/10 $32.00 www.nature.com/eye Colour vision MP Simunovic REVIEW deficiency Abstract effective "treatment" of colour vision deficiency: whilst it has been suggested that tinted lenses Colour vision deficiency is one of the could offer a means of enabling those with commonest disorders of vision and can be colour vision deficiency to make spectral divided into congenital and acquired forms. discriminations that would normally elude Congenital colour vision deficiency affects as them, clinical trials of such lenses have been many as 8% of males and 0.5% of femalesFthe largely disappointing. Recent developments in difference in prevalence reflects the fact that molecular genetics have enabled us to not only the commonest forms of congenital colour understand more completely the genetic basis of vision deficiency are inherited in an X-linked colour vision deficiency, they have opened the recessive manner. Until relatively recently, our possibility of gene therapy. The application of understanding of the pathophysiological basis gene therapy to animal models of colour vision of colour vision deficiency largely rested on deficiency has shown dramatic results; behavioural data; however, modern molecular furthermore, it has provided interesting insights genetic techniques have helped to elucidate its into the plasticity of the visual system with mechanisms. respect to extracting information about the The current management of congenital spectral composition of the visual scene. colour vision deficiency lies chiefly in appropriate counselling (including career counselling). Although visual aids may Materials and methods be of benefit to those with colour vision deficiency when performing certain tasks, the This article was prepared by performing a evidence suggests that they do not enable primary search of Pubmed for articles on wearers to obtain normal colour ‘colo(u)r vision deficiency’ and ‘colo(u)r discrimination. -
Congenital Or Acquired Color Vision Defect – If Acquired What Caused It?
Congenital or Acquired Color Vision Defect – If Acquired What Caused it? Terrace L. Waggoner O.D. 3730 Tiger Point Blvd. Gulf Breeze, FL 32563 [email protected] Course Handout AOA 2017 Conference: I. Different types of congenital colorblindness. A. Approximately 8% of the population has a congenital color vision deficiency. B. Dichromate 1. Protanopia is a severe type of color vision deficiency caused by the complete absence of red retinal photoreceptors (L cone absent). 2. Deuteranopia is a type of color vision deficiency where the green photoreceptors are absent (M cone absent). 3. Tritanopia is a very rare color vision disturbance in which there are only two cone pigments present and a total absence of blue retinal receptors (S cone absent). It is related to chromosome 7. C. Anomalous Trichromate 1. Protanomaly is a mild color vision defect in which an altered spectral sensitivity of red retinal receptors (closer to green receptor response) results in poor red–green hue discrimination. 2. Deuteranomaly, caused by a similar shift in the green retinal receptors, is by far the most common type of color vision deficiency, mildly affecting red–green hue discrimination in 5% males. 3. Tritanomaly is a rare, hereditary color vision deficiency affecting blue–green and yellow–red/pink hue discrimination. D. Rates of color blindness 1. Dichromacy Males 2.4% Females .03% a. Protanopia (red deficient: L cone absent) Males 1.3% Females 0.02% b. Deuteranopia (green deficient: M cone absent) Males 1.2% Females 0.01 c. Tritanopia (blue deficient: S cone absent) Males 0.001% Females 0.03% 2. -
PEDIATRIC EYE and VISION EXAMINATION Reference Guide for Clinicians
OPTOMETRIC CLINICAL PRACTICE GUIDELINE OPTOMETRY: THE PRIMARY EYE CARE PROFESSION Doctors of optometry are independent primary health care providers who examine, diagnose, treat, and manage diseases and disorders of the visual system, the eye, and associated structures as well as diagnose related systemic conditions. Optometrists provide more than two-thirds of the primary eye care services in the United States. They are more widely distributed geographically than other eye care providers and are readily accessible for the delivery of eye and vision care services. There are approximately 32,000 full-time equivalent doctors of optometry currently in practice in Pediatric Eye the United States. Optometrists practice in more than 7,000 communities across the United States, serving as the sole primary eye care provider in more than 4,300 communities. And Vision The mission of the profession of optometry is to fulfill the vision and eye care needs of the public through clinical care, research, and education, all Examination of which enhance the quality of life. OPTOMETRIC CLINICAL PRACTICE GUIDELINE PEDIATRIC EYE AND VISION EXAMINATION Reference Guide for Clinicians First Edition Originally Prepared by (and Second Edition Reviewed by) the American Optometric Association Consensus Panel on Pediatric Eye and Vision Examination: Mitchell M. Scheiman, O.D., M.S., Principal Author Catherine S. Amos, O.D. Elise B. Ciner, O.D. Wendy Marsh-Tootle, O.D. Bruce D. Moore, O.D. Michael W. Rouse, O.D., M.S. Reviewed by the AOA Clinical Guidelines Coordinating Committee: John C. Townsend, O.D., Chair (2nd Edition) John F. Amos, O.D., M.S. -
Book of Abstracts of the International Colour Association (AIC) Conference 2020
NATURAL COLOURS - DIGITAL COLOURS Book of Abstracts of the International Colour Association (AIC) Conference 2020 Avignon, France 20, 26-28th november 2020 Sponsored by le Centre Français de la Couleur (CFC) Published by International Colour Association (AIC) This publication includes abstracts of the keynote, oral and poster papers presented in the International Colour Association (AIC) Conference 2020. The theme of the conference was Natural Colours - Digital Colours. The conference, organised by the Centre Français de la Couleur (CFC), was held in Avignon, France on 20, 26-28th November 2020. That conference, for the first time, was managed online and onsite due to the sanitary conditions provided by the COVID-19 pandemic. More information in: www.aic2020.org. © 2020 International Colour Association (AIC) International Colour Association Incorporated PO Box 764 Newtown NSW 2042 Australia www.aic-colour.org All rights reserved. DISCLAIMER Matters of copyright for all images and text associated with the papers within the Proceedings of the International Colour Association (AIC) 2020 and Book of Abstracts are the responsibility of the authors. The AIC does not accept responsibility for any liabilities arising from the publication of any of the submissions. COPYRIGHT Reproduction of this document or parts thereof by any means whatsoever is prohibited without the written permission of the International Colour Association (AIC). All copies of the individual articles remain the intellectual property of the individual authors and/or their -
Intermixing the OPN1LW and OPN1MW Genes Disrupts the Exonic Splicing Code Causing an Array of Vision Disorders
G C A T T A C G G C A T genes Review Intermixing the OPN1LW and OPN1MW Genes Disrupts the Exonic Splicing Code Causing an Array of Vision Disorders Maureen Neitz * and Jay Neitz Department of Ophthalmology and Vision Science Center, University of Washington, Seattle, WA 98109, USA; [email protected] * Correspondence: [email protected]; Tel.: +1-206-543-7998 Abstract: Light absorption by photopigment molecules expressed in the photoreceptors in the retina is the first step in seeing. Two types of photoreceptors in the human retina are responsible for image formation: rods, and cones. Except at very low light levels when rods are active, all vision is based on cones. Cones mediate high acuity vision and color vision. Furthermore, they are critically important in the visual feedback mechanism that regulates refractive development of the eye during childhood. The human retina contains a mosaic of three cone types, short-wavelength (S), long-wavelength (L), and middle-wavelength (M) sensitive; however, the vast majority (~94%) are L and M cones. The OPN1LW and OPN1MW genes, located on the X-chromosome at Xq28, encode the protein component of the light-sensitive photopigments expressed in the L and M cones. Diverse haplotypes of exon 3 of the OPN1LW and OPN1MW genes arose thru unequal recombination mechanisms that have intermixed the genes. A subset of the haplotypes causes exon 3- skipping during pre-messenger RNA splicing and are associated with vision disorders. Here, we review the mechanism by which splicing defects in these genes cause vision disorders. Citation: Neitz, M.; Neitz, J. -
International Journal for Scientific Research & Development
IJSRD - International Journal for Scientific Research & Development| Vol. 1, Issue 12, 2014 | ISSN (online): 2321-0613 Modifying Image Appearance for Improvement in Information Gaining For Colour Blinds Prof.D.S.Khurge1 Bhagyashree Peswani2 1Professor, ME2, 1, 2 Department of Electronics and Communication, 1,2L.J. Institute of Technology, Ahmedabad Abstract--- Color blindness is a color perception problem of Light transmitted by media i.e., Television uses additive human eye to distinguish colors. Persons who are suffering color mixing with primary colors of red, green, and blue, from color blindness face many problem in day to day life each of which is stimulating one of the three types of the because many information are contained in color color receptors of eyes with as little stimulation as possible representations like traffic light, road signs etc. of the other two. Its called "RGB" color space. Mixtures of Daltonization is a procedure for adapting colors in an image light are actually a mixture of these primary colors and it or a sequence of images for improving the color perception covers a huge part of the human color space and thus by a color-deficient viewer. In this paper, we propose a re- produces a large part of human color experiences. That‟s coloring algorithm to improve the accessibility for the color because color television sets or color computer monitors deficient viewers. In Particular, we select protanopia, a type needs to produce mixtures of primary colors. of dichromacy where the patient does not naturally develop Other primary colors could in principle be used, but the “Red”, or Long wavelength, cones in his or her eyes. -
Application of Contrast Sensitivity Functions in Standard and High Dynamic Range Color Spaces
https://doi.org/10.2352/ISSN.2470-1173.2021.11.HVEI-153 This work is licensed under the Creative Commons Attribution 4.0 International License. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. Color Threshold Functions: Application of Contrast Sensitivity Functions in Standard and High Dynamic Range Color Spaces Minjung Kim, Maryam Azimi, and Rafał K. Mantiuk Department of Computer Science and Technology, University of Cambridge Abstract vision science. However, it is not obvious as to how contrast Contrast sensitivity functions (CSFs) describe the smallest thresholds in DKL would translate to thresholds in other color visible contrast across a range of stimulus and viewing param- spaces across their color components due to non-linearities such eters. CSFs are useful for imaging and video applications, as as PQ encoding. contrast thresholds describe the maximum of color reproduction In this work, we adapt our spatio-chromatic CSF1 [12] to error that is invisible to the human observer. However, existing predict color threshold functions (CTFs). CTFs describe detec- CSFs are limited. First, they are typically only defined for achro- tion thresholds in color spaces that are more commonly used in matic contrast. Second, even when they are defined for chromatic imaging, video, and color science applications, such as sRGB contrast, the thresholds are described along the cardinal dimen- and YCbCr. The spatio-chromatic CSF model from [12] can sions of linear opponent color spaces, and therefore are difficult predict detection thresholds for any point in the color space and to relate to the dimensions of more commonly used color spaces, for any chromatic and achromatic modulation. -
Visual Impairment Age-Related Macular
VISUAL IMPAIRMENT AGE-RELATED MACULAR DEGENERATION Macular degeneration is a medical condition predominantly found in young children in which the center of the inner lining of the eye, known as the macula area of the retina, suffers thickening, atrophy, and in some cases, watering. This can result in loss of side vision, which entails inability to see coarse details, to read, or to recognize faces. According to the American Academy of Ophthalmology, it is the leading cause of central vision loss (blindness) in the United States today for those under the age of twenty years. Although some macular dystrophies that affect younger individuals are sometimes referred to as macular degeneration, the term generally refers to age-related macular degeneration (AMD or ARMD). Age-related macular degeneration begins with characteristic yellow deposits in the macula (central area of the retina which provides detailed central vision, called fovea) called drusen between the retinal pigment epithelium and the underlying choroid. Most people with these early changes (referred to as age-related maculopathy) have good vision. People with drusen can go on to develop advanced AMD. The risk is considerably higher when the drusen are large and numerous and associated with disturbance in the pigmented cell layer under the macula. Recent research suggests that large and soft drusen are related to elevated cholesterol deposits and may respond to cholesterol lowering agents or the Rheo Procedure. Advanced AMD, which is responsible for profound vision loss, has two forms: dry and wet. Central geographic atrophy, the dry form of advanced AMD, results from atrophy to the retinal pigment epithelial layer below the retina, which causes vision loss through loss of photoreceptors (rods and cones) in the central part of the eye.