Central Venous Catheters: a Closer Look at the Subclavian Vein Approach

Total Page:16

File Type:pdf, Size:1020Kb

Central Venous Catheters: a Closer Look at the Subclavian Vein Approach CASE REPORT Central Venous Catheters: A Closer Look at the Subclavian Vein Approach KEVIN SUN, MD; GREGORY M. SOARES, MD KEYWORDS: Central venous catheter, Subclavian Figure 1. AP chest under fluoroscopy showing a chemotherapy port placed in the vein, internal jugular vein subclavian vein illustrating the “pinch off” sign. Fracture occurred at the location of the clavicle and first rib. INTRODUCTION Central venous catheters (CVCs) are commonly used and have a range of outpatient and inpatient indications. A subclavian vein approach has tradi- tionally been used for placement of these cathe- ters; however, this method exposes the patient to the high risk of subclavian stenosis as well as an increased risk for catheter fracture. In this report, we describe a patient with a chemotherapy port placed in the subclavian vein that underwent spon- taneous fracture. We therefore advocate for the use of an internal jugular approach for CVCs. CASE REPORT A 62-year-old man with a history of Kaposi’s sar- coma was referred to interventional radiology for a percutaneous chemotherapy port study. The per- cutaneous port was originally placed through the Figure 2. Digital subtraction angiography showing extravasation of left subclavian vein for adjuvant chemotherapy. contrast revealing the catheter fracture. Port malfunction was first noticed during a routine follow-up appointment with the patient’s hematol- ogy oncologist. Blood return was sluggish and there was a noticeable soft lump at the upper sternum after flushing. A Port study was performed under fluoroscopic guidance. The initial AP view of the chest (Figure 1) revealed luminal narrowing and “pinch off” sign at the intersection of the clavicle and first rib. Digital subtraction acquisition with contrast confirmed the location of the fracture Fig( - ure 2). Contrast extravasation was documented at the location of the soft swelling. (Figure 3). The device was removed in the interventional radiology suite. Gentle traction was used to remove the cath- eter, given the known damage and possible risk for embolization of the catheter tip. Upon removal, parallel 1cm long longitudinal fractures were iden- tified at the fluoroscopically identified point of extravasation (Figure 4). RIMJ ARCHIVES | MAY ISSUE WEBPAGE | RIMS MAY 2018 RHODE ISLAND MEDICAL JOURNAL 31 CASE REPORT Figure 3. Extravasation of contrast into the subcutaneous tissue. Figure 4. Extent of the catheter fracture after removal. DISCUSSION object in a narrow vessel lumen at the restricted anatomic Various factors leading to catheter fracture have been rec- space between the first rib and clavicle. Utilization of larger ognized. It has been well established that catheters placed caliber vessels such as the internal jugular vein for catheter in the subclavian vein are exposed to high mechanical fric- placement has been shown to minimize this complication, tion from the clavicle and first rib. 1 Compressive forces can with reported stenosis rates as low as 3%.8 cause transient obstruction. Over time, repetitive stress on Though the subclavian vein has been the preferred site the catheter causes structural degradation leading to frac- for many proceduralists, given the evidence of complica- ture. Previously reported incidences for catheter fracture tions with long-term use, many have advocated for the have ranged from .1–1.3%.2,3 internal jugular vein as the first-line approach. 1,9,10 It is well Occult fracture may first be noticed with difficulty admin- documented that an internal jugular approach with image istering or aspirating fluid through the line. More serious guidance provides a safe and reliable method for long-term symptoms may present as extravascular administration of central venous catheters. The course of the internal jugular medications through the fractured line or embolization of vein is free of anatomic features that may cause compression the catheter tip. or catheter damage. It has a large caliber and high flow to Early diagnosis of catheter fracture is key to manage- reduce the risk for thrombosis. Other risks such as infection ment. Chest x-ray can provide the earliest radiographic evi- are comparable to the subclavian approach, while pneumo- dence for possible catheter fracture with a positive “pinch thorax risk is diminished. 11 Finally, complications such as off” sign. Patients with a positive “pinch off” sign have an brachial plexus injuries and thoracic duct injuries are unique estimated 40% risk for catheter fracture and such catheters to a subclavian catheter and are also avoided. should be removed and replaced using another vessel.4 If fracture is suspected and complete transection has occurred, the patient should undergo emergent percutaneous retrieval CONCLUSION by interventional radiology, which has been shown to be a Central venous catheter placement through the subclavian highly successful and safe procedure.5 vein has a high rate of vein stenosis and increased risk for Catheter fractures are a rare event. Stenosis is a more com- catheter fracture. Catheter fracture is less common, but may mon and insidious complication of subclavian venous cath- lead to dangerous complications such as extravascular extrav- eter placement. Venous stenosis in the setting of subclavian asation of medication or embolization. Subclavian stenosis catheters has a reported incidence of 32–50%, typically seen can severely limit venous access which becomes problem- with catheters used for greater than 2 weeks of duration.6,7 atic for patients requiring long-term parenteral therapy. The The mechanism for stenosis is catheter-induced throm- Internal Jugular approach with imaging guidance minimizes bosis and intimal fibrosis due to the presence of a foreign risk and provides a proven, safe and reliable alternative. RIMJ ARCHIVES | MAY ISSUE WEBPAGE | RIMS MAY 2018 RHODE ISLAND MEDICAL JOURNAL 32 CASE REPORT References Authors 1. Mirza B, Vanek VW, Kupensky DT. Pinch-off syndrome: case Kevin Sun, MD, Department of Internal Medicine, Roger Williams report and collective review of the literature. Am Surg. 2004 Medical Center, Providence, RI. Jul;70(7):635-44. Gregory M. Soares, MD, Associate Professor of Diagnostic & 2. Wu CY, Fu JY, Feng PH, Kao TC, Yu SY, Li HJ, Ko PJ, Hsieh Interventional Radiology, Warren Alpert Medical School of HC. Catheter fracture of intravenous ports and its management. World J Surg. 2011 Nov;35(11):2403-10. Brown University; Rhode Island Medical Imaging. 3. Amr Mahmoud Abdel Samad, Yosra Abdelzaher Ibrahim. Com- Correspondence plications of Port A Cath implantation: A single institution ex- perience, In The Egyptian Journal of Radiology and Nuclear Med- [email protected] icine, Volume 46, Issue 4, 2015, Pages 907-911, ISSN 0378-603X 4. Fazeny-Dörner B, Wenzel C, Berzlanovich A, Sunder-Plassmann G, Greinix H, Marosi C, Muhm M. Central venous catheter pinch-off and fracture: recognition, prevention and management. Bone Marrow Transplant. 2003 May;31(10):927-30. Review. 5. Dinkel HP, Muhm M, Exadaktylos AK, Hoppe H, Triller J Emer- gency percutaneous retrieval of a silicone port catheter fragment in pinch-off syndrome by means of an Amplatz gooseneck snare. Emerg Radiol. 2002 Sep;9(3):165-8. 6. Beenen L, van Leusen R, Deenik B, Bosch FH. The incidence of subclavian vein stenosis using silicone catheters for hemodialy- sis. Artif Organs. 1994 Apr;18(4):289-92. 7. Vanherweghem JL. Thrombosis and stenosis of central venous access in hemodialysis]. Nephrologie. 1994;15(2):117-21. 8. Surratt RS, Picus D, Hicks ME, Darcy MD, Kleinhoffer M, Jen- drisak M. The importance of preoperative evaluation of the sub- clavian vein in dialysis access planning. AJR Am J Roentgenol. 1991 Mar;156(3):623-5. 9. Andris DA, Krzywda EA, Schulte W, Ausman R, Quebbeman EJ. Pinch-off syndrome: a rare etiology for central venous catheter occlusion. JPEN J Parenter Enteral Nutr. 1994 Nov- Dec;18(6):531-3. 10. Cho, Jin-Beom et al. “Pinch-off Syndrome.” Journal of the Kore- an Surgical Society 85.3 (2013): 139–144. PMC. Web. 2 Dec. 2017. 11. Arvaniti K, Lathyris D, Blot S, Apostolidou-Kiouti F, Koulen- ti D, Haidich AB. Cumulative Evidence of Randomized Con- trolled and Observational Studies on Catheter-Related Infection Risk of Central Venous Catheter Insertion Site in ICU Patients: A Pairwise and Network Meta-Analysis. Crit Care Med. 2017 Apr;45(4):e437-e448. RIMJ ARCHIVES | MAY ISSUE WEBPAGE | RIMS MAY 2018 RHODE ISLAND MEDICAL JOURNAL 33.
Recommended publications
  • Prep for Practical II
    Images for Practical II BSC 2086L "Endocrine" A A B C A. Hypothalamus B. Pineal Gland (Body) C. Pituitary Gland "Endocrine" 1.Thyroid 2.Adrenal Gland 3.Pancreas "The Pancreas" "The Adrenal Glands" "The Ovary" "The Testes" Erythrocyte Neutrophil Eosinophil Basophil Lymphocyte Monocyte Platelet Figure 29-3 Photomicrograph of a human blood smear stained with Wright’s stain (765). Eosinophil Lymphocyte Monocyte Platelets Neutrophils Erythrocytes "Blood Typing" "Heart Coronal" 1.Right Atrium 3 4 2.Superior Vena Cava 5 2 3.Aortic Arch 6 4.Pulmonary Trunk 1 5.Left Atrium 12 9 6.Bicuspid Valve 10 7.Interventricular Septum 11 8.Apex of The Heart 9. Chordae tendineae 10.Papillary Muscle 7 11.Tricuspid Valve 12. Fossa Ovalis "Heart Coronal Section" Coronal Section of the Heart to show valves 1. Bicuspid 2. Pulmonary Semilunar 3. Tricuspid 4. Aortic Semilunar 5. Left Ventricle 6. Right Ventricle "Heart Coronal" 1.Pulmonary trunk 2.Right Atrium 3.Tricuspid Valve 4.Pulmonary Semilunar Valve 5.Myocardium 6.Interventricular Septum 7.Trabeculae Carneae 8.Papillary Muscle 9.Chordae Tendineae 10.Bicuspid Valve "Heart Anterior" 1. Brachiocephalic Artery 2. Left Common Carotid Artery 3. Ligamentum Arteriosum 4. Left Coronary Artery 5. Circumflex Artery 6. Great Cardiac Vein 7. Myocardium 8. Apex of The Heart 9. Pericardium (Visceral) 10. Right Coronary Artery 11. Auricle of Right Atrium 12. Pulmonary Trunk 13. Superior Vena Cava 14. Aortic Arch 15. Brachiocephalic vein "Heart Posterolateral" 1. Left Brachiocephalic vein 2. Right Brachiocephalic vein 3. Brachiocephalic Artery 4. Left Common Carotid Artery 5. Left Subclavian Artery 6. Aortic Arch 7.
    [Show full text]
  • Pictures of Central Venous Catheters
    Pictures of Central Venous Catheters Below are examples of central venous catheters. This is not an all inclusive list of either type of catheter or type of access device. Tunneled Central Venous Catheters. Tunneled catheters are passed under the skin to a separate exit point. This helps stabilize them making them useful for long term therapy. They can have one or more lumens. Power Hickman® Multi-lumen Hickman® or Groshong® Tunneled Central Broviac® Long-Term Tunneled Central Venous Catheter Dialysis Catheters Venous Catheter © 2013 C. R. Bard, Inc. Used with permission. Bard, are trademarks and/or registered trademarks of C. R. Bard, Inc. Implanted Ports. Inplanted ports are also tunneled under the skin. The port itself is placed under the skin and accessed as needed. When not accessed, they only need an occasional flush but otherwise do not require care. They can be multilumen as well. They are also useful for long term therapy. ` Single lumen PowerPort® Vue Implantable Port Titanium Dome Port Dual lumen SlimPort® Dual-lumen RosenblattTM Implantable Port © 2013 C. R. Bard, Inc. Used with permission. Bard, are trademarks and/or registered trademarks of C. R. Bard, Inc. Non-tunneled Central Venous Catheters. Non-tunneled catheters are used for short term therapy and in emergent situations. MAHURKARTM Elite Dialysis Catheter Image provided courtesy of Covidien. MAHURKAR is a trademark of Sakharam D. Mahurkar, MD. © Covidien. All rights reserved. Peripherally Inserted Central Catheters. A “PICC” is inserted in a large peripheral vein, such as the cephalic or basilic vein, and then advanced until the tip rests in the distal superior vena cava or cavoatrial junction.
    [Show full text]
  • Piccs, Ports and Lines: Clarifying the Options
    Current Concepts in Vascular Therapies 2011 Mid-Atlantic Conference PICCs, Ports and Lines: Clarifying the Options Babatunde Almaroof, MD April 2, 2011 Objectives • State the indications for central venous access • Discuss types of central venous catheters • “Clarifying the options”/indications for each kind of catheter. Need for central vascular access • There is an increasing need for vascular access as medical care has become more complex. • Most inpatients are able to get their needs served by a peripheral i.v access • Sometimes however, a central access will be needed due to limitations of a peripheral access – Infiltration, extravasation, thrombosis – Infection and sclerosis • This makes central venous access, the preferred choice for long term use as they allow a higher flow and tolerate hyperosmolar solutions not tolerated by peripheral veins Indications for central venous access • TPN • Chemotherapy • Long term antibiotics – Osteomyelitis, endocarditis, fungal infections • Patients with difficult peripheral vein access • Hemodynamic monitoring • Temporary hemodialysis access • Plasmapheresis Historical Background • The first i.v infusion was performed using a cannula made from quill in 1657 • First successful human blood transfusion was performed in 1667 • Seldinger described his technique for catheter insertion in 1953 • Percutaneous placement of a subclavian vein catheter was reported in 1956 Sites of central venous access • Internal Jugular vein • Subclavian vein – Higher risk of pneumothorax • Femoral vein – Higher risk of
    [Show full text]
  • Vessels and Circulation
    CARDIOVASCULAR SYSTEM OUTLINE 23.1 Anatomy of Blood Vessels 684 23.1a Blood Vessel Tunics 684 23.1b Arteries 685 23.1c Capillaries 688 23 23.1d Veins 689 23.2 Blood Pressure 691 23.3 Systemic Circulation 692 Vessels and 23.3a General Arterial Flow Out of the Heart 693 23.3b General Venous Return to the Heart 693 23.3c Blood Flow Through the Head and Neck 693 23.3d Blood Flow Through the Thoracic and Abdominal Walls 697 23.3e Blood Flow Through the Thoracic Organs 700 Circulation 23.3f Blood Flow Through the Gastrointestinal Tract 701 23.3g Blood Flow Through the Posterior Abdominal Organs, Pelvis, and Perineum 705 23.3h Blood Flow Through the Upper Limb 705 23.3i Blood Flow Through the Lower Limb 709 23.4 Pulmonary Circulation 712 23.5 Review of Heart, Systemic, and Pulmonary Circulation 714 23.6 Aging and the Cardiovascular System 715 23.7 Blood Vessel Development 716 23.7a Artery Development 716 23.7b Vein Development 717 23.7c Comparison of Fetal and Postnatal Circulation 718 MODULE 9: CARDIOVASCULAR SYSTEM mck78097_ch23_683-723.indd 683 2/14/11 4:31 PM 684 Chapter Twenty-Three Vessels and Circulation lood vessels are analogous to highways—they are an efficient larger as they merge and come closer to the heart. The site where B mode of transport for oxygen, carbon dioxide, nutrients, hor- two or more arteries (or two or more veins) converge to supply the mones, and waste products to and from body tissues. The heart is same body region is called an anastomosis (ă-nas ′tō -mō′ sis; pl., the mechanical pump that propels the blood through the vessels.
    [Show full text]
  • Infusaport Insertion in Patients with Haemophilia
    Infusaport insertion in patients with haemophilia PURPOSE This guideline is designed to assist medical and nursing staff in the management of children with haemophilia having an infusaport inserted at the Royal Children’s Hospital. DEFINITIONS Infusaport or portacath is an implantable Central Venous Access Device. BACKGROUND Most children with severe haemophilia (<1% Factor VIII or IX) will require prophylactic intravenous clotting factor administration 2-3 times per week to prevent spontaneous bleeding. Accessing peripheral veins can be difficult and traumatic for children and in particular infants/toddlers where veins are often difficult to identify. A number of boys develop significant behavioural issues around treatment after traumatic experiences in their early years. Approximately 80% of children with severe haemophilia treated at the Royal Children’s Hospital will require an infusaport for venous access. Most families report that insertion of a “port” dramatically improves their quality of life in that venous access is no longer fearful and difficult and parents are able to administer clotting factor to their child at home for both prevention and treatment of bleeds. Ports are removed as soon as parents are able to administer clotting factor peripherally. In general ports are removed prior to commencement of primary school. PROCEDURE Once the need for a port has been identified and discussed with the family a referral is made. Mr Joe Crameri performs the majority of infusaport surgery in haemophilia patients at the Royal Children’s Hospital. Many families appreciate the opportunity to see a port (there is one in the haemophilia centre) and to speak with a family whose child is established on home prophylaxis via a port.
    [Show full text]
  • Venous Access and Ports
    Venous Access and Ports Helen Starosta Venous access and ports Peripheral IV access Arterio-Venous Fistula Central venous access Peripherally Inserted Central Catheter (PICC) Non Tunnelled Central Venous Catheter (CVC) Tunnelled (e.g. Hickman) Central Venous Access Device Implanted Central Venous Access Device e.g. Infusaport Jesse’s Story Charles’s Story Vein Training Why do we need venous access Treatment for bleeding disorders involves intravenous therapy Therefore reliable venous access is essential to make effective treatment possible The choices for IV access Peripheral IV access Arterio-Venous Fistula Central venous access Peripherally Inserted Central Catheter (PICC) Non Tunnelled Central Venous Catheter (CVC) Tunnelled (e.g. Hickman) Central Venous Access Device Implanted Central Venous Access Device e.g. Infusaport Peripheral Venous Access Butterfly & IV Short term (days) or intermittent therapy Short catheters generally placed in forearm, hand or scalp veins Arterio-Venous Fistula Can last many years Connects an artery directly to a vein → results in more blood flow to the vein → the vein grows larger and stronger Fistula takes a while after surgery to develop (as long as 24 months) Properly formed fistula is less likely than other kinds of vascular access to form clots or become infected Peripherally Inserted Central Catheters (PICC) Short term use (days to several weeks) Peripheral central venous catheter inserted at or above the antecubital space and the distal tip of the catheter is positioned
    [Show full text]
  • Anatomy and Physiology in Relation to Compression of the Upper Limb and Thorax
    Clinical REVIEW anatomy and physiology in relation to compression of the upper limb and thorax Colin Carati, Bren Gannon, Neil Piller An understanding of arterial, venous and lymphatic flow in the upper body in normal limbs and those at risk of, or with lymphoedema will greatly improve patient outcomes. However, there is much we do not know in this area, including the effects of compression upon lymphatic flow and drainage. Imaging and measuring capabilities are improving in this respect, but are often expensive and time-consuming. This, coupled with the unknown effects of individual, diurnal and seasonal variances on compression efficacy, means that future research should focus upon ways to monitor the pressure delivered by a garment, and its effects upon the fluids we are trying to control. More is known about the possible This paper will describe the vascular Key words effects of compression on the anatomy of the upper limb and axilla, pathophysiology of lymphoedema when and will outline current understanding of Anatomy used on the lower limbs (Partsch and normal and abnormal lymph drainage. It Physiology Junger, 2006). While some of these will also explain the mechanism of action Lymphatics principles can be applied to guide the use of compression garments and will detail Compression of compression on the upper body, it is the effects of compression on fluid important that the practitioner is movement. knowledgeable about the anatomy and physiology of the upper limb, axilla and Vascular drainage of the upper limb thorax, and of the anatomical and vascular It is helpful to have an understanding of Little evidence exists to support the differences that exist between the upper the vascular drainage of the upper limb, use of compression garments in the and lower limb, so that the effects of these since the lymphatic drainage follows a treatment of lymphoedema, particularly differences can be considered when using similar course (Figure 1).
    [Show full text]
  • What Is Venous Thoracic Outlet Syndrome?
    Venous Thoracic Outlet Syndrome An In-Depth Guide for Patients and Healthcare Providers By: Holly Grunebach, MSPH, PA-C, Ying-Wei Lum, MD, MPH Division of Vascular Surgery and Endovascular Therapy Johns Hopkins Hospital 600 N Wolfe St, Halsted 668 Baltimore, MD 21287 1. What is venous thoracic outlet syndrome? Venous thoracic outlet syndrome is a condition that occurs when the subclavian vein is compressed by the first rib and the subclavius/anterior scalene muscle resulting in a blood clot. The anterior scalene is a muscle located in the neck that attaches to the first rib in the area known as the thoracic outlet. Typically, the structures that control the function of the arm including the subclavian artery, subclavian vein and the nerves of the brachial plexus are able to pass through this area without incident. (Figure) In the case of venous thoracic outlet syndrome the veins are compressed which results in a blood clot. 2. What causes venous thoracic outlet syndrome? Venous thoracic outlet syndrome occurs over time as the subclavian vein is compressed by the subclavius/anterior scalene muscle and first rib with arm use. With the arms in an extended or overhead position, the space around the structures within the thoracic outlet space narrows. In thoracic outlet syndrome this space becomes so narrow that compression of the subclavian vein occurs triggering blood clot formation. 3. What are signs and symptoms of venous thoracic outlet syndrome? Persistent swelling with pain and color change in the arm is typical in patients with venous thoracic outlet syndrome. These symptoms are usually persistent and unable to be relieved with other conservative treatments.
    [Show full text]
  • The Cardiovascular System
    11 The Cardiovascular System WHAT The cardiovascular system delivers oxygen and HOW nutrients to the body tissues The heart pumps and carries away wastes blood throughout the body such as carbon dioxide in blood vessels. Blood flow via blood. requires both the pumping action of the heart and changes in blood pressure. WHY If the cardiovascular system cannot perform its functions, wastes build up in tissues. INSTRUCTORS Body organs fail to function properly, New Building Vocabulary and then, once oxygen becomes Coaching Activities for this depleted, they will die. chapter are assignable in hen most people hear the term cardio- only with the interstitial fluid in their immediate Wvascular system, they immediately think vicinity. Thus, some means of changing and of the heart. We have all felt our own “refreshing” these fluids is necessary to renew the heart “pound” from time to time when we are ner- nutrients and prevent pollution caused by vous. The crucial importance of the heart has been the buildup of wastes. Like a bustling factory, the recognized for ages. However, the cardiovascular body must have a transportation system to carry system is much more than just the heart, and its various “cargoes” back and forth. Instead of from a scientific and medical standpoint, it is roads, railway tracks, and subways, the body’s important to understand why this system is so vital delivery routes are its hollow blood vessels. to life. Most simply stated, the major function of the Night and day, minute after minute, our tril- cardiovascular system is transportation. Using lions of cells take up nutrients and excrete wastes.
    [Show full text]
  • Central Venous Lines in Haemophilia. Ljung, Rolf
    Central venous lines in haemophilia. Ljung, Rolf Published in: Haemophilia DOI: 10.1046/j.1365-2516.9.s1.7.x 2003 Link to publication Citation for published version (APA): Ljung, R. (2003). Central venous lines in haemophilia. Haemophilia, 9(Suppl 1), 88-92. https://doi.org/10.1046/j.1365-2516.9.s1.7.x Total number of authors: 1 General rights Unless other specific re-use rights are stated the following general rights apply: Copyright and moral rights for the publications made accessible in the public portal are retained by the authors and/or other copyright owners and it is a condition of accessing publications that users recognise and abide by the legal requirements associated with these rights. • Users may download and print one copy of any publication from the public portal for the purpose of private study or research. • You may not further distribute the material or use it for any profit-making activity or commercial gain • You may freely distribute the URL identifying the publication in the public portal Read more about Creative commons licenses: https://creativecommons.org/licenses/ Take down policy If you believe that this document breaches copyright please contact us providing details, and we will remove access to the work immediately and investigate your claim. LUND UNIVERSITY PO Box 117 221 00 Lund +46 46-222 00 00 Haemophilia (2003), 9, (Suppl. 1), 88–93 Central venous lines in haemophilia R. LJUNG Departments of Paediatrics and Coagulation Disorders, Lund University, University Hospital, Malmoo,€ Sweden Summary. Infections and technical problems are the been seen in some but not in others.
    [Show full text]
  • Vascular Access Device (VAD) Management
    Vascular Access Device (VAD) Management Page 1 of 21 Disclaimer: This algorithm has been developed for MD Anderson using a multidisciplinary approach considering circumstances particular to MD Anderson’s specific patient population, services and structure, and clinical information. This is not intended to replace the independent medical or professional judgment of physicians or other health care providers in the context of individual clinical circumstances to determine a patient's care. TABLE OF CONTENTS Definitions………………………………....…………………………....………………………………………….. Page 3 CVAD Post-Insertion Dressing Care………….………………………………………………………………….. Page 4 VAD Maintenance Care………….………………………………………………………………………………... Pages 5-8 Dressing Care …………………………………………………………………………………………………. Page 5 Flush Management …………………………………………………………………………………………. Page 6 Needleless Connector Management………………………………………………………………………….. Page 7 Tubing Management………………....…………….……………………………...………………………….. Page 8 Implanted Venous Ports: Access and Management……….…………………………………………………….. Page 9 VAD Complications………………………………………………………………………….…………………….. Pages 10-13 Skin Impairment…….………………………………………………………………………………………… Page 10 Site Complication/Infection…………………………………………………………………………………… Page 11 Phlebitis …………………………....…………….……………………………...……………………………... Page 12 CVAD Device-Related …………………………....…………….……………………………...……………… Page 13 CVAD = central venous access device PICC = peripherally inserted central catheter CICC = centrally inserted central catheter Department of Clinical Effectiveness V4 Approved
    [Show full text]
  • Double Left Brachiocephalic Veins with Persistent Left Superior Vena Cava: a Case Report 지속 좌측 상대정맥과 동반된 중복 좌측 팔머리 정맥: 증례 보고
    Case Report pISSN 1738-2637 / eISSN 2288-2928 J Korean Soc Radiol 2014;71(2):55-57 http://dx.doi.org/10.3348/jksr.2014.71.2.55 Double Left Brachiocephalic Veins with Persistent Left Superior Vena Cava: A Case Report 지속 좌측 상대정맥과 동반된 중복 좌측 팔머리 정맥: 증례 보고 O Hyun Kwon, MD, Ji Kyoung Lim, MD, Jae Kyo Lee, MD, Mi Soo Hwang, MD Department of Radiology, Yeungnam University College of Medicine, Daegu, Korea Anomalous left brachiocephalic vein (ALBCV) is a rare condition of the major tho- racic veins. It is usually associated with a congenital cardiac anomaly. Most reports Received January 10, 2014; Accepted May 29, 2014 on ALBCV are on aberrant left BCV, and there are few reports on double left BCV. Corresponding author: Jae Kyo Lee, MD Persistent left superior vena cava (PLSVC) is also a rare vascular anomaly that is Department of Radiology, Yeungnam University College of Medicine, 170 Hyeonchung-ro, Nam-gu, caused by the failure of the left anterior cardinal vein to regress. To our knowledge, Daegu 705-717, Korea. double left BCV with PLSVC has not been reported. Here, we report a case of double Tel. 82-53-620-3047 Fax. 82-53-653-5484 left BCV with PLSVC in a 72-year-old male patient with no previous cardiac abnor- E-mail: [email protected] mality. This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial Index terms License (http://creativecommons.org/licenses/by-nc/3.0) which permits unrestricted non-commercial use, distri- Anomalous Left Brachiocephalic Vein bution, and reproduction in any medium, provided the Double Left Brachiocephalic Veins original work is properly cited.
    [Show full text]