Master Day 2

Total Page:16

File Type:pdf, Size:1020Kb

Master Day 2 Master Day 2 1. Identify “AOT” On radiographs, the adenomatoid odontogenic tumor presents as a radiolucency (dark area) around an unerupted tooth extending past the cementoenamel junction. It should be differentially diagnosed from a dentigerous cyst and the main difference is that the radiolucency in case of AOT extends apically beyond the cementoenamel junction. Radiographs will exhibit faint flecks of radiopacities surrounded by a radiolucent zone. It is sometimes misdiagnosed as a cyst. And frequently in anterior maxilla! 2. Identify Dentigerous Cyst 3. Identify Eruption Cyst May God be with us and help us nail NBDE PART 2 J 4. Identify Doubtful Answer Molar appears to have DD and Premolar – DI 5. Identify ? Amelogenesis Imperfecta: How to identify? See the outer boundary n shape. Compare it with adjacent teeth moreover i see it like a prepared tooth for crown to diagnose. May God be with us and help us nail NBDE PART 2 J 6. Identify Dentinogenesis imperfecta 7. Identify External Resorption May God be with us and help us nail NBDE PART 2 J 8. Identify Genial tubercle 9. Identify Bilateral Tori May God be with us and help us nail NBDE PART 2 J 10. Identify Mandibular Canal 11. Identify Ameloblastoma May God be with us and help us nail NBDE PART 2 J 12. Identify Fusion 13. Identify Lateral Periodontal Cyst May God be with us and help us nail NBDE PART 2 J 14. Identify Complex Odentoma It's a complex odontome. Compound would show enamel dentin and cementum separately. 15. Identify Cementoblastoma May God be with us and help us nail NBDE PART 2 J 16. Identify Underexposed / OverFixed 17. Identify Intermaxillary Suture May God be with us and help us nail NBDE PART 2 J 18. Identify Zygomatic Process 19. Identify Coronoid Process May God be with us and help us nail NBDE PART 2 J 20. 21. May God be with us and help us nail NBDE PART 2 J 22. Identify Leukoplakia 23. Identify Herpes Labialis May God be with us and help us nail NBDE PART 2 J 24. Identify Stafne’s bone Cyst May God be with us and help us nail NBDE PART 2 J 25. Identify Recurrent Caries : 26. Identify Age and Diagnosis ? Over 7 Years Diagnosis would be retained deciduous lower incisors and coming to age is would be 7-8 here we have to consider the development of roots of 6 and roots of upper central incisors and lower lateral incisors. Coming to loss of D in 3rd arch, ref; Mc Donald pediatric book, if any primary teeth lost before 7 yr there May God be with us and help us nail NBDE PART 2 J would be delayed eruption of permanent teeth and if loss of primary teeth after 7 yrs would lead to premature eruption of permanent teeth. Here premolar is in eruption stage so it should be considered too. So age would be 7-8 yrs 27. Learn the fact Complete craniofacial dysjunction by the Le Fort III osteotomy allows the surgeon to alter the orbital position and volume, zygomatic projection, position of the nasal root, frontonasal angle, and position of the maxilla and to lengthen the nose. The Le Fort II osteotomy allows the surgeon to alter the nasomaxillary projection without altering the orbital volume and zygomatic projection. The Le Fort I osteotomy allows for correction primarily at the occlusal level affecting the upper lip position, nasal tip and alar base region, and the columella labial angle without altering the orbitozygomatic region 28. The appearance of the curve of spee on the panoramic suggests that the patients chin was positioned too: A. Far forward B. Far bacK C. High D. Low Answer is HIGH May God be with us and help us nail NBDE PART 2 J 29. No answer available May be lingual tori 30. Identify Underexposed/Overfixed +Lower border of mx.sinus May God be with us and help us nail NBDE PART 2 J 31. Identify gemination, fusion has just two roots 32. Identify Answer is below the picture May God be with us and help us nail NBDE PART 2 J 33. Identify flap Semilunar Flap 34. Identify Answer is below the Picture May God be with us and help us nail NBDE PART 2 J 35. Identify Answer is below the picture May God be with us and help us nail NBDE PART 2 J 36. Identify Answer is below the picture May God be with us and help us nail NBDE PART 2 J 37. Identify we have retained primary 1ts molar in lower right side Missing lower 3rd molar bilateral or extracted Chin is up means � frown Chin is down means � smile In this Pano ... Chin down smiling Pt is 23 years’ old 38. Pt adult male. Present asymptomatic, necrotic, ulcerated area involving palatal mucosa. Histologically presents lobular necrosis of glandular parenchyma with sQuamous metaplasia and hyperplasia of ductal epithelium. Dx & Tx? Necrotizing sialometaplasia. it is caused by ischemic necrosis of minor salivary glands. it will heal in 6-10 wks. May God be with us and help us nail NBDE PART 2 J 39. Identify 8 is angle of Mandible and 9 is Hyoid Bone 40. Identify Peutz-Jeghers Syndrome. Intraorally pigmentations may be located anywhere in the mucosa. Most frequently hard palate, Buccal mucosa and gingiva May God be with us and help us nail NBDE PART 2 J 41. Identify 1- middle cranial fossa 5- styloid process 17- ear 42. Identify Dens in Dente May God be with us and help us nail NBDE PART 2 J 43. Identify 10- glenoid fossa 11- articular eminence 12-condyle 13-vertebra May God be with us and help us nail NBDE PART 2 J 44. Identify Pulp Stone: Compare with Dens in dente : 42nd Question 45. Identify 2-orbit 3- zygomatic arch 4- hard palate 6- max tuberosity May God be with us and help us nail NBDE PART 2 J 46. Identify Eagle’s Syndrome Elongated Styloid and calcification of stylohyoid ligaments. 47. Identify odontogenic myxoma: non-encapsulated, no resorption of teeth, cortical expansion, honey-comb appearance May God be with us and help us nail NBDE PART 2 J 48. Identify Punched out lesions without sclerotic border...multiple myeloma Punched out lesions with or without sclerotic border.... Langerhans cell histiocytosis 49. Identify The arrow is pointing to alternating RLs at the level of border of mandible. This is also known as "onion skin" and it's seen in GARRE's Osteomyelitis. May God be with us and help us nail NBDE PART 2 J 50. Identify Y is formed between nasal floor and maxillary sinus Y line of Ennis The straight line resembles the basal floor and the curved one for the maxillary sinus. May God be with us and help us nail NBDE PART 2 J 51. Identify Answer: Antral pseudocyst (mucous retention pseudocyst) 52. Identify May God be with us and help us nail NBDE PART 2 J 53. Identify Internal Resorption 54. Turner's Hypoplasia***-Frequent pattern of enamel defects seen in permanent teeth secondary to periapical inflammatory disease of the overlying deciduous tooth. -The altered tooth is called Turner's tooth. May God be with us and help us nail NBDE PART 2 J 55. Characteristics of enamel defects in Turner's Hypoplasia -Vary from focal areas of white, yellow or brown discoloration to extensive hypoplasia which can involve the entire crown. Most frequently noted in permanent bicuspids because of their relationship to the overlying deciduous molars. 56. In regards to traumatic injury, which teeth are most affected by Turner's Hypoplasia Maxillary central incisors 57. Identify problem, cause, treatment Dental Fluorosis clinical features and treatment - Ingestion of fluoride results in enamel defects called dental fluorosis. -Permanent hypomutation of the enamel in which there is an increased surface and subsurface porosity of the enamel. -Altered tooth structure presents as areas of lusterless, white opaque enamel with zones of yellow to dark-brown discoloration. treatment: composites and full crown coverage May God be with us and help us nail NBDE PART 2 J 58. Critical years for dental fluorosis During the 2nd and third years of life when anterior teeth are forming and fluoride levels are greater than 1 pt per million ingested. 59. Attrition definiton*** Loss of tooth structure caused by tooth to tooth contact during occlusion and mastication Destruction acclerated by: 1) poor quality or absent enamel 2) Premature contacts, edge to edge occlusion 3) intraoral abrasives, erosion or grinding habit May God be with us and help us nail NBDE PART 2 J 60. Abrasion definition Loss of tooth structure secondary to the action of an external agent. Most common source is toothbrushing. 61. Erosion definition Loss of tooth structure caused by chemical reaction (other than bacteria) -usually to an acid external: lemon, vinegar, soft drinks internal: gastric secretions such as perimylolysis (people suffering from anorexia or buliema present this) 62. Attrition most often seen in these teeth and specific surfaces -Deciduous and permanent teeth -Occlusal and Incisor region of maxillary and mandibular teeth -Lingual of Maxillary anteriors -Labial of mandibular anteriors May God be with us and help us nail NBDE PART 2 J 63. Abrasion 64. Internal Resorption Destruction of teeth accomplished by cells located in the dental pulp.-Rare occurence, when injury to pupal tissue occurs such as physical trauma or pulpitis. -Process continues as long as vital pulp tissue is present. -Uniform well circumscribed symmetric radiolucent enlargement of the pulp chamber or canal. Pinktooth of Mummery. -When it affects the coronal pulp and expands it, the crown can display a pink discoloration: May God be with us and help us nail NBDE PART 2 J 65.
Recommended publications
  • The Role of Alpha V Beta 6 Integrin in Enamel Biomineralization
    The Role of Alpha v Beta 6 Integrin in Enamel Biomineralization by Leila Mohazab A THESIS SUBMITTED IN PARTIAL FULFILLMENT OF THE REQUIREMENTS FOR THE DEGREE OF MASTERS OF SCIENCE in The Faculty of Graduate and Postdoctoral Studies (Craniofacial Science) THE UNIVERSITY OF BRITISH COLUMBIA (Vancouver) October 2013 ⃝c Leila Mohazab 2013 Abstract Tooth enamel has the highest degree of biomineralization of all vertebrate hard tissues. During the secretory stage of enamel formation, ameloblasts deposit an extracellular matrix that is in direct contact with ameloblast plasma membrane. Although it is known that integrins mediate cell-matrix adhesion and regulate cell signaling in most cell types, the receptors that reg- ulate ameloblast adhesion and matrix production are not well characterized. Thus, we hypothesized that αvβ6 integrin is expressed in ameloblasts where it regulates biomineralization of enamel. Human and mouse ameloblasts were found to express both β6 integrin mRNA and protein. The maxil- lary incisors of Itgb6-/- mice lacked yellow pigment and their mandibular incisors appeared chalky and rounded. Molars of Itgb6-/- mice showed signs of reduced mineralization and severe attrition. The mineral-to-protein ra- tio in the incisors was significantly reduced in Itgb6-/- enamel, mimicking hypomineralized amelogenesis imperfecta. Interestingly, amelogenin-rich ex- tracellular matrix abnormally accumulated between the ameloblast layer of Itgb6-/- mouse incisors and the forming enamel surface, and also between ameloblasts. This accumulation was related to increased synthesis of amel- ogenin, rather than to reduced removal of the matrix proteins. This was confirmed in cultured ameloblast-like cells, which did not use αvβ6 integrin as an endocytosis receptor for amelogenins, although it participated in cell adhesion on this matrix indirectly via endogenously produced matrix pro- teins.
    [Show full text]
  • Free PDF Download
    Eur opean Rev iew for Med ical and Pharmacol ogical Sci ences 2014; 18: 440-444 Radiographic evaluation of the prevalence of enamel pearls in a sample adult dental population H. ÇOLAK, M.M. HAMIDI, R. UZGUR 1, E. ERCAN, M. TURKAL 1 Department of Restorative Dentistry, Kirikkale University School of Dentistry, Kirikkale, Turkey 1Department of Prosthodontics, Kirikkale University School of Dentistry, Kirikkale, Turkey Abstract. – AIM: Enamel pearls are a tooth One theory of the enamel pearl etiology is that anomaly that can act as contributing factors in the enamel pearls develop as a result of a localized development of periodontal disease. Studies that developmental activity of a remnant of Hertwig’s have addressed the prevalence of enamel pearls in epithelial root sheath which has remained adher - populations were scarce. The purpose of this study 5 was to evaluate the prevalence of enamel pearls in ent to the root surface during root development . the permanent dentition of Turkish dental patients It is believed that cells differentiate into function - by means of panoramic radiographs. ing ameloblasts and produce enamel deposits on PATIENTS AND METHODS: Panoramic radi - the root. The conditions needed for local differ - ographs of 6912 patients were examined for the entiation and functioning of ameloblasts in this presence of enamel pearls. All data (age, sex and ectopic position are not fully understood 6,7 . systemic disease or syndrome) were obtained from the patient files and analyzed for enamel The most common site for enamel pearls is at pearls. Descriptive characteristics of sexes, the cementoenamel junction of multirooted jaws, and dental localization were recorded.
    [Show full text]
  • Supernumerary Teeth)
    Lecture 7 Paediatric Dentistry Dr. Israa Ali “Dental Anomalies” Dental anomalies are malformations or defects affecting teeth. They usually result from either disturbances to teeth development or they could be as result of environmental influences on teeth. Dental anomalies Developmental Environmental Defects in Effects on teeth number tooth structure Defects in Effects on teeth size tooth color Defects in Effects on teeth shape eruption Defects in teeth structure 11/04/2019 1 Developmental anomalies of dentition: Anomalies Anomalies Anomalies Anomalies of of number of size of shape structure Gemination Ameloenesis Hypodontia Fusion Imperfecta (AI) Microdontia Concrescence Accessory cusps Dentinoenesis Oliodontia Imperfecta (DI) Dens invaginatus Ectopic enamel Anodontia Dentin dysplasia Taurodontism Macrodontia Hypercementosis Accessory roots Regional Hyperdontia Odontodysplasia Dilaceration Developmental anomalies in the number of teeth: 1- Hypodontia: It is agenesis of some teeth (fewer than 6 teeth, not including the third molars). It can occur alone (isolated) or it could be associated with syndromes such as Down’s syndrome. It is uncommon in primary teeth. However, the permanent teeth are more likely to be affected with the most commonly affected teeth to be the third molars followed by mandibular second premolars, maxillary lateral incisors, maxillary second premolars, and mandibular central incisors. 2- Oligodontia: It is a term used to describe the developmental absence of more than six permanent teeth. It is also uncommon in primary teeth; and when the permanent 11/04/2019 2 teeth are affected, collapse of the dental arch and drifting of the few present teeth will result due to presence of excess space. Treatment of both hypodontia and oligodontia involve prosthodontic and orthodontic rehabilitation.
    [Show full text]
  • Day 2 Unicorn 1
    MasterDay2 A word from the author: MasterRQs and MasterDay2 are helpful only once you have basic knowledge of your subjects. Both of these files have the best and compile almost all radiographic images and cases available online. Extensive image coverage of each of topic has been done. Day 2 tests your basic knowledge of the subjects. I sincerely advise you to go through the mentioned topics properly- as the cases are mostly focussed on these. Know these diseases and medicines used, contraindications of them and MOA. What to note/write on the paper provided in the exam ? You will be given 2 sheets to write on/if anything. Here is what you need to write. Draw a line in the centre of the first page, and write Generic name on one side and Trade name on another. During the exam in the first 4-5 cases they mention you both the class/Drug/generic name and the trade name eg : Generic Trade Alendronate (Bisphosphonates) Fosamax Ethambutol (Anti TB) Abitol Zoledronic acid(Bisphosphonates) Reclast By the time you are done with 4-5 cases they will skip the Generic names in few questions because they presume you should know them as they have been mentioned in the exam only. So If you are unaware of this, you will have to go back and look for the important information again question by question, but if u have made this small chart, it saves your time! Must read topics 1. Hypertension and management 2. Myocardial infarction 3. Stroke 4. Diabetes 5. Syncope 6.
    [Show full text]
  • Enamel Pearl Associated with Localized Periodontitis in Hellenistic Age Woman
    T o m o v e t a l . C A S E R E P O R T Enamel pearl associated with localized periodontitis in Hellenistic age woman • Georgi Tomov (1), Elka Popova (2), Rumen Ivanov (3), Nadezhda Atanassova (4) • 1 - Оral Pathology Department, Faculty of Dental Medicine, Medical University, Plovdiv, Bulgaria 2 - Periodontology Department, Faculty of Dental Medicine, Medical University, Plovdiv, Bulgaria 3 - Archeologist 4 – National Anthropological Museum at Institute of Experimental Morphology, Pathology and Anthropology with Museum Address for correspondence: Assoc.Prof. Georgi Tomov, PhD Medical University Plovdiv, Faculty of Dental Medicine, Oral Pathology Department, Plovdiv, Bulgaria Phone: +359896742065 E- mail: [email protected] Bull Int Assoc Paleodont. 2017;11(2):62-66. Abstract Tooth anatomic factors like ectopic enamel pearls are often associated with localized periodontal inflammation and bone loss. There are no existing paleopathological data for such structural anomalies in ancient populations associated with periodontal pathology in the literature. A rare case of enamel pearl on the maxillary right first molar of women associated with localized periodontitis is presented and discussed. Keywords: enamel pearl; localized periodontitis; paleopathology; Hellenistic age Bull Int Assoc Paleodont. Volume 11, Number 2, 2017 www.paleodontology.com 62 Bulletin of the International Association for Paleodontology NO-FEE OPEN ACCESS JOURNAL T o m o v e t a l . C A S E R E P O R T Introduction are found in Plovdiv, Bulgaria (archeological site Bacterial plaque has been implicated as the “Kirkor Azarian” №4) and are provided for primary etiologic factor in the initiation and anthropological study in the Medical University progression of gingivitis and periodontitis (1).
    [Show full text]
  • Defects of Permanent Teeth
    ORAL PATHOLOGY Dr. Muna Lec.10 DEVELOPMENTAL DEFECTS OF THE ORAL AND MAXILLOFACIAL REGION 1- DEVELOPMENTAL DISORDERS OF TEETH. 2- DEVELOPMENTAL DEFECT OF THE ORAL MUCOSA. 3- DEVELOPMENTAL DEFECT OF THE TONGUE. 4- DEVELOPMENTAL DEFECT OF THE LIPS AND PALATE. 5- DEVELOPMENTAL DEFECT OF THE JAW BONES. 6- DEVELOPMENTAL CYST. 1- Developmental Disorders of Teeth Development of an ideal dentition depends on many factors:- • Formation of a full complement of teeth • Normal structural development of the dental tissues • Eruption of each group of teeth at the appropriate time into an adequate space • Eruption of teeth into correct relationship to occlude with their opposite numbers Developmental Alterations of Teeth 1- Developmental alteration in the size of teeth. 2- Developmental alteration in the number of teeth. 3- Developmental alteration in the shape of teeth. 4- Developmental alteration in the eruption of teeth. 5- Developmental alteration in the structure of teeth. 1- Alteration in size of teeth Microdontia Generalized microdontia: all teeth in the dentition appear smaller than normal, as in pituitary dwarfism Focal or localized microdontia: a single tooth is smaller than normal. The shape of these microdonts is also often altered with the reduced size. This phenomenon is most commonly seen with maxillary lateral incisors, in which the tooth crown appears cone or peg shaped, (peg lateral). Macrodontia Generalized macrodontia is characterized by the appearance of enlarged teeth throughout the dentition. This may be absolute, as seen in pituitary gigantism, or it may be relative owing to a disproportionately small maxilla and mandible. Focal, or localized, macrodontia: is characterized by an abnormally large tooth or group of teeth.
    [Show full text]
  • Bibliograficzne Z´Ro´Dła Ilustracji
    BIBLIOGRAFICZNE Z´RO´ DŁA ILUSTRACJI Dzięki uprzejmości: Acuson Computed Sonography Corporation, Mountain View, CA (Doppler ultrasonography). Zaczerpnięto z: Agur AMR, Lee M. Grant’s Atlas of Anatomy. 10th ed. Baltimore, MD: Lippincott Williams & Wilkins; 1999 (spinal column). Dzięki uprzejmości: Alzheimer’s Disease Education and Referral Center, a service of the National Institute on Aging (PET scan). Przedrukowano za zgodą: American Cancer Society, Inc. (malignant melanoma). Jennifer Anderson @ USDA-NRCS PLANTS Database (Podophyllum peltatum, Dicentra cucullaria). Zaczerpnięto z: Anderson SC, Poulsen K. Anderson’s Atlas of Hematology. Baltimore, MD: Lippincott Williams & Wilkins; 2003 (pronormoblast, basophilic normoblast, polychromatophilic normoblast, orthochromatophilic normoblast, reticulocyte, erythrocyte, plasmablast, proplasmacyte, plasma cells: plasmacytes, lymphoblast, lymphocyte, monoblast, premonocyte, monocyte, eosinophilic myelocyte, eosinophilic metamyelocyte, eosinophilic band, eosinophil, myeloblast, promyelocyte, neutrophilic myelocyte, neutrophilic metamyelocyte, neutrophilic band, neutrophil, basophilic myelocyte, basophilic metamyelocyte, basophilic band, basophil, megakaryoblast, promega- karyocyte, megakaryocyte, platelets, hypersegmentation, Pelger-Huët, Döhle body, Faggot cell, LE cell, cleaved cell, hairy cell, Dutcher body, Mott cell, Chédiak-Higashi granules, bilobed plasma cell, bacteria: spirochetes, stomatocytosis). Zaczerpnięto z: Baker CL. The Hughston Clinic Sports Medicine Book. 1st ed. Baltimore, MD:
    [Show full text]
  • Developmental Anomalies Affecting the Morphology of Teeth – a Review
    View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Portal de Periódicos da UNIVILLE (Universidade da Região de Joinville) ISSN: Electronic version: 1984-5685 RSBO. 2015 Jan-Mar;12(1):68-78 Literature Review Article Developmental anomalies affecting the morphology of teeth – a review Ashish Shrestha1 Vinay Marla1 Sushmita Shrestha2 Iccha K Maharjan3 Corresponding author: Ashish Shrestha Department of Oral Histology and Pathology, College of Dental Surgery B. P. Koirala Institute of Health Sciences Dharan – Sunsari – Nepal E-mail: [email protected] 1 Department of Oral Histology and Pathology, College of Dental Surgery, B. P. Koirala Institute of Health Sciences – Dharan – Nepal. 2 Department of Conservative Dentistry and Endodontics, College of Dental Surgery, B. P. Koirala Institute of Health Sciences – Dharan – Nepal. 3 Department of Oral Medicine and Radiology, College of Dental Surgery, B. P. Koirala Institute of Health Sciences – Dharan – Nepal. Received for publication: May 5, 2014. Accepted for publication: August 12, 2014. Abstract Keywords: developmental Introduction: The development of tooth is a complex process anomaly, diagnostic wherein there is series of interactions between the ectoderm and criteria, tooth ectomesenchyme. The role of genes in determining the shape and morphology. form of a specific tooth has already been defined, the alterations in which can lead to a variety of anomalies in regards to number, size, form, shape, structure, etc. Objective: To review the literature on the developmental anomalies of teeth. Literature review: The developmental anomalies affecting the morphology exists in both deciduous & permanent dentition and shows various forms such as gemination, fusion, concrescence, dilacerations, dens evaginatus, dens invaginatus, enamel pearls, taurodontism or peg laterals.
    [Show full text]
  • Role of Bmp2 in Dental Hard Tissue Formation
    Zurich Open Repository and Archive University of Zurich Main Library Strickhofstrasse 39 CH-8057 Zurich www.zora.uzh.ch Year: 2016 Role of Bmp2 in dental hard tissue formation Malik, Zeba Posted at the Zurich Open Repository and Archive, University of Zurich ZORA URL: https://doi.org/10.5167/uzh-204719 Dissertation Published Version Originally published at: Malik, Zeba. Role of Bmp2 in dental hard tissue formation. 2016, University of Zurich, Faculty of Science. Role of Bmp2 in Dental Hard Tissue Formation Dissertation (Juni 2016) zur Erlangung der naturwissenschaftlichen Doktorwürde (Dr.sc.nat) vorgelegt der Mathematisch-naturwissenschaftlichen Fakultät der Universität Zürich von Zeba Malik aus Indien Promotionkomitee Prof. Dr. Michael Hengartner Prof. Dr. Daniel Graf Prof. Dr. Lukas Sommer Dr. Aimee Zuniga Zurich 2016 1 Table of Contents 1. Summary .......................................................................................................................................................................... 4 2. Summary in German ................................................................................................................................................. 6 3. Introduction ..................................................................................................................................................................... 8 3.1 Bone morphogenetic proteins (Bmps) ............................................................................ 8 3.2 Tooth: Structure and Function .....................................................................................
    [Show full text]
  • The National Oral Health Research Initiative (Nohri)
    THE NATIONAL ORAL HEALTH RESEARCH INITIATIVE (NOHRI) The National Oral Health Research Conference 2008 organised by the Oral Health Division, Ministry of Health Malaysia, identified the need to establish a multiagency initiative to identify oral health research priorities for the country and to better manage the oral health research agenda in the country. The establishment of the National Health Research Initiative (NOHRI) in the 10th Malaysia Plan (10 MP) was mooted by the Oral Health Division in 2010 towards enhancing evidence-based policy decisions in the oral healthcare delivery system in the country. Stakeholders with oral health research interests from various organisations in the country which are currently represented in NOHRI are as follows: Oral Health Division, Ministry of Health Malaysia Faculty of Dentistry, University of Malaya (UM) Faculty of Dentistry, National University of Malaysia (UKM) Faculty of Dentistry, Science University of Malaysia (USM) Faculty of Dentistry, International Islamic University of Malaysia (UIAM) Faculty of Dentistry, Islamic Science University of Malaysia (USIM) Faculty of Dentistry, Mara University of Technology (UiTM) Faculty of Dentistry, International University of Malaysia (IMU) Faculty of Dentistry, Asian Institute of Medicine, Science & Technology (AIMST) Faculty of Dentistry, Malaysia Allied Health Science Academy (MAHSA) Dental Corp, Ministry of Defence Oral Cancer Research and Coordinating Centre, University Malaya (OCRCC) Malaysian Dental Association (MDA) The establishment of NOHRI is timely to better manage the oral health research agenda in the country in the following ways: Oral health research priorities of relevant stakeholders in oral health in the country are identified and collated. These priorities will be taken into consideration in the national health research priority setting.
    [Show full text]
  • Oral Pathology Disorders of Development of Teeth
    Oral Pathology DEVELOPMENTAL DEFECTS OF THE ORAL AND MAXILLOFACIAL REGION Developmental disturbances of the oral region are of these broad categories: (1) Developmental disturbances affecting teeth. (2) Developmental disturbances limited to soft tissue. (3) Developmental disturbances affecting bone. Disorders of development of teeth The development of teeth is regulated by genes, but the genetic program is very sensitive to disturbances in the environment such as infection, or toxic chemicals. The causes of developmental disorders of teeth are multifactorial, involving the interaction of genetic and environmental factors. These disorders may be prenatal or postnatal in origin and may inherit or acquired. Disorders of development of teeth may be due to abnormalities in the differentiation of the dental lamina and the tooth germs, causing anomalies number, size, form of teeth and abnormalities of morph differentiation or abnormalities in the formation of the dental hard tissue resulting in disturbances in tooth structure ((abnormalities of histo differentiation)). Developmental Alterations in the Number of Teeth Anodontia Absence of teeth is known as anodontia. This condition is further qualified as complete anodontia, when all teeth are missing; as partial anodontia or hypodontia, when one or several teeth are missing; as pseudoanodontia, when teeth are absent clinically because of impaction or delayed eruption; or as false anodontia, when teeth have been exfoliated or extracted. Partial anodontia is relatively common. Congenitally missing teeth are usually third molars, followed by second premolars and maxillary lateral incisors. 1-several regulatory genes are involved in tooth formation and development which may be mutated in hypodontia e.g. (MSX1 and Pax9). These genes are involved in the control of development of the face and of many other tissues and organs in the embryo.
    [Show full text]
  • PROGRAMMA DELL'insegnamento in Inglese
    PROGRAMMA DELL’INSEGNAMENTO in inglese I - ANATOMICAL-PATHOLOGY PROPEDEUTICS (Prof. G. Palmieri) - Definition and purpose of the discipline - Methodologies of histopathological investigation - Diagnostic limits of histopathology - Method of request for histopathological examination - Method of collection, storage and forwarding of the material to be examined - Evaluation and interpretation of the histopathological and cytopathological examination report and communication with the histopathologist II - ORAL PATHOLOGY (Prof. G. Palmieri) ANOMALIES OF OROFACIAL DEVELOPMENT Orofacial fissures, labial fasades (commissural and paramedian), double lip, Fordyce granules, language abnormalities (aglossia, microglossia, macroglossia, anchilogloxia, fixed language, language hairy, lingual thyroid), varicose veins, exostoses, bulls, Stafne defect, rare syndromes (hemi- hyperplasia, progressive facial atrophy, Crouzon syndrome, Apert syndrome, jaw-facial dysostosis). TOOTH ANOMALIES Developmental abnormalities due to environmental influences (enamel abnormalities, Turner's tooth, fluorosis, Luetic hypoplasia - Hutchinson's teeth and strawberry molar) Regressive alterations (friction, abrasion, erosion, abfractione), internal resorption and external, anomalies of coloring of the teeth (extrinsic and intrinsic). Localized disorders of the rash (primary impact, ankylosis) Congenital anomalies of the teeth: number (anodontia, hypodontia, oligodontia, hyperdontia), dimension (microdontia, macrodontia), of shape (gemination, fusion, with crescenza, cuspidi
    [Show full text]