1) Cervical Pain, Radiculopathy, and Neck Surgery
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Pierre Robin and the Syndrome That Bears His Name PETER RANDALL
Pierre Robin and the Syndrome That Bears His Name PETER RANDALL, M.D. WILTON M. KROGMAN, Ph.D. SOONA JAHINA, B.D.S., M.Sc. Philadelphia, Pennsylvania The Pierre Robin Syndrome refers to a combination of micrognathia (a small jaw) and glossoptosis (literally, a falling downward or back- ward of the tongue) in the newborn infant (Figure 1). These conditions are likely to cause obstruction of the upper airway, and they are fre- quently associated with an incomplete cleft of the palate. Patients with the Pierre Robin Syndrome may present a real emer- gency in the delivery room because of the obstructed upper airway, or the airway problem may not become manifest for several days or weeks (10, 11, 38). There is frequently a feeding problem, as well as problems associated with the cleft of the palate (if one is present) and also an unusual malocclusion (2, 5, 12, 16). In addition, it presents a fascinating anthropological puzzle (22, 23). This paper will review the work of Dr. Robin, consider some possible etiologies of this syndrome, and report on some work on mandibular bone growth in a group of such patients. History Pierre Robin was far from the first person to recognize this syndrome. One account is recorded in 1822 by St. Hilaire. In 1891 Taruffi men- tioned two subclassifications-hypomicrognatus (small jaw) and hypo- agnathus (absent jaw). In 1891, four cases, two of them having cleft palates, were reported by Lanneloague and Monard (12, 14). Shukow- sky in 1902 described a tongue to lip surgical adhesion to overcome the respiratory obstruction (34). -
Podo Pediatrics Podo Pediatrics
Podo Pediatrics Identifying Biomechanical Pathologies David Lee, D.P.M., D. A.B.P.S. Purpose • Identification of mechanical foot and ankle conditions • Base treatments • Knowing when to refer to a podiatrist Topics • Flatfoot (Pes Plano Valgus) • Equinus • Intoed feet (Cavo-adductor Varus) • Heel pain (Calcaneodynia) • Shin Splints • Various Pedal deformities 1 WHAT IS NORMAL? At birth to ~9 months • Ankle flexible to over 20 deg DF • No “C” shaped foot • No clicking or popping sounds • Babinski sign • Pull up 7-8mo. 9-16 months… • Begin walking • Feet are fat, flat and floppy • Knees are always center or externally rotated, never internal. • Stance is wide and less stable • Stomping gait pattern 2 16-18 months • Able to walk upstairs • Knee never internal • Still wide base and flat and floppy feet • Stomping still 3-7 years • Able toe walk downstairs • Heel-to-toe walk • Watch for – Intoeing – Tripping – Tight ankle joint (equinus) 7 years and up • Arch should be developed • Heel-to-toe walk • Heel is perpendicular to ground • Knees straight ahead 3 Neutral Internal Rotation Early detection is important • Prevent long term adaptation • Joint damage • Adult pathology – Heel pain, bunions, hammertoes, ankle instability, knee pain, shin splints, etc. • Ability to thrive physically and socially 4 THE FLAT FOOT Visual Complaints by the Parent • Tripping or falling • Poor balance- Clumsy • Feet look funny, walks funny • Shoes wearing out quickly Social Complaints by the Parent • Lazy, inactive, “doesn’t like going outside to play or play sports -
Knee Osteoarthritis
BRIGHAM AND WOMEN’S HOSPITAL Department of Rehabilitation Services Standard of Care: _Osteoarthritis of the Knee Case Type / Diagnosis: Knee Osteoarthritis. ICD-9: 715.16, 719.46 Osteoarthritis/Osteoarthrosis (OA) is the most common joint disease causing disability, affecting more than 7 million people in the United States 1. OA is a disease process of axial and peripheral joints. It is characterized by progressive deterioration and loss of articular cartilage and by reactive bone changes at the margins of the joints and in the subchondral bone. Clinical manifestations are characterized by slowly developing joint pain, stiffness, and joint enlargement with limitations of motion. Knee osteoarthritis (OA) results from mechanical and idiopathic factors that alter the balance between degradation and synthesis of articular cartilage and subchondral bone. The etiology of knee OA is not entirely clear, yet its incidence increases with age and in women. 1 The etiology may have genetic factors affecting collagen, or traumatic factors, such as fracture or previous meniscal damage. Obesity is a risk factor for the development and progression of OA. Early degenerative changes predict progression of the disease. Underlying biomechanical factors, such as varum or valgum of the tibial femoral joint may predispose people to OA. However Hunter et al 2reported knee alignment did not predict OA, but rather was a marker of the disease severity. Loss of quadriceps muscle strength is associated with knee pain and disability in OA. Clinical criteria for the diagnosis of OA of the knee has been established by Altman3 Subjects with examination finding consistent with any of the three categories were considered to have Knee OA. -
Knee Evaluation
Evaluation of Knee Injuries Dr. Alan A. Zakaria, D.O., M.S. 1080 Kirts Blvd., Suite 400 Troy, Mi., 48084 Team Physician United States Soccer Federation University of Michigan Men’s and Women’s Soccer Objective Identify main anatomic components of the knee Perform basic knee exam along with special tests Identify common knee injury patterns and their physical exam findings. Anatomy ➢ Bony Anatomy ➢ Ligaments ➢ Cartilage ➢ Musculature ➢ Other Soft Tissue Knee Anatomy Two functional joints – Femorotibial – Femoropatellar Femoral condyles – Flex/extend Knee Anatomy Patella – Sesamoid with two concave surfaces and vertical ridge – Increases efficiency of extension Knee Anatomy: Anterior Cruciate Ligament (ACL) Run inferior, anterior, and medially Arises from medial aspect lateral femoral condyle Insert lateral to medial tibial eminence Restrains anterior subluxation of tibia on femur Knee Anatomy: Posterior Cruciate Ligament (PCL) Arises from the posterior intercondylar area of the tibia Inserts at the medial condyle of the femur Restrains posterior subluxation of the tibia on the femur Knee Anatomy: Medial Collateral Ligament (MCL) Postero-superior medial femoral condyle to proximal end of tibia Maximum tension at full extension Restraint to valgus stress Knee Anatomy: Lateral Collateral Ligament (LCL) Posterosuperior lateral femoral condyle to lateral head of fibula Restraint to varus stress Knee Anatomy: Meniscus Load bearing, joint stability, shock absorption Peripheral third vascularized Knee Anatomy: Articular Cartilage Hyaline cartilage -
Cervical Fusion
Medical Coverage Policy Effective Date ............................................. 6/15/2021 Next Review Date ....................................... 6/15/2022 Coverage Policy Number .................................. 0527 Cervical Fusion Table of Contents Related Coverage Resources Overview .............................................................. 1 Acupuncture Coverage Policy ................................................... 1 Bone, Cartilage and Ligament Graft Substitutes General Background ............................................ 4 Bone Growth Stimulators: Electrical (Invasive, Medicare Coverage Determinations .................... 9 Noninvasive), Ultrasound Coding/Billing Information .................................... 9 Discography Intervertebral Disc (IVD) Prostheses References ........................................................ 11 Intraoperative Monitoring Lumbar Fusion for Spinal Instability and Degenerative Disc Conditions, Including Sacroiliac Fusion Minimally Invasive Intradiscal/Annular Procedures and Trigger Point Injections INSTRUCTIONS FOR USE The following Coverage Policy applies to health benefit plans administered by Cigna Companies. Certain Cigna Companies and/or lines of business only provide utilization review services to clients and do not make coverage determinations. References to standard benefit plan language and coverage determinations do not apply to those clients. Coverage Policies are intended to provide guidance in interpreting certain standard benefit plans administered by Cigna Companies. -
Examination of the Spine
Page 1 of 7 Examination of the Spine Neck and back pain are common presentations in primary care. Many cases of neck and back pain are due to benign functional or postural causes but a thorough history and examination is essential to assess the cause (see articles Low Back Pain and Sciatica, Thoracic Back Pain and Neck Pain), any associated psychological difficulties (eg depression, anxiety or somatisation disorder), and any functional impairment, including restrictions with work, leisure and domestic activities. General examination of the spine The examination should begin as soon as you first see the patient and continues with careful observation during the whole consultation. It is essential to observe the patient's gait and posture. Inconsistency between observed function and performance during specific tests may help to differentiate between physical and functional causes for the patient's symptoms. Inspection Examination of any localised spinal disorder requires inspection of the entire spine. The patient should therefore undress to their underwear. Look for any obvious swellings or surgical scars. Assess for deformity: scoliosis, kyphosis, loss of lumbar lordosis or hyperlordosis of the lumbar spine. Look for shoulder asymmetry and pelvic tilt. Observe the patient walking to assess for any abnormalities of gait. Palpation Palpate for tenderness over bone and soft tissues. Perform an abdominal examination to identify any masses, and consider a rectal examination (cauda equina syndrome may present with low back pain, pain in the legs and unilateral or bilateral lower limb motor and/or sensory abnormality, bowel and/or bladder dysfunction with saddle and perineal anaesthesia, urinary dysfunction and bowel disturbances, and rectal examination may reveal loss of anal tone and sensation). -
Delayed Diagnosis of Tandem Spinal Stenosis: a Retrospective Institutional Review
International Journal of Spine Surgery, Vol. 13, No. 3, 2019, pp. 283–288 https://doi.org/10.14444/6038 ÓInternational Society for the Advancement of Spine Surgery Delayed Diagnosis of Tandem Spinal Stenosis: A Retrospective Institutional Review AMIT BHANDUTIA, MD, LUKE BROWN, MD, MBA, ALYSA NASH, MD, IAN BUSSEY, MD, MARK SHASTI, MD, EUGENE KOH, MD, PHD, KELLEY BANAGAN, MD, STEVEN LUDWIG, MD, DANIEL GELB, MD Department of Orthopaedics, University of Maryland Medical Center, Baltimore, Maryland ABSTRACT Background: Tandem spinal stenosis (TSS) is defined as simultaneous spinal stenosis in the cervical, thoracic, and/or lumbar regions and may present with both upper and lower motor neuron symptoms, neurogenic claudication, and gait disturbance. Current literature has focused mainly on the prevalence of TSS and treatment methods, while the incidence of delayed TSS diagnosis is not well defined. The purpose of this study was to determine the incidence of delayed TSS diagnosis at our institution and describe the clinical characteristics commonly observed in their particular presentation. Methods: Following institutional review board approval, an institutional billing database review was performed for patients who underwent a spinal decompression procedure between 2006 and 2016. Thirty-three patients who underwent decompression on 2 separate spinal regions within 1 year were included for review. Patients with delayed diagnosis of TSS following the first surgery were differentiated from those with preoperative diagnosis of TSS. Results: TSS requiring surgical decompression occurred in 33 patients, with the incidence being 2.06% in this cohort. Fifteen patients received a delayed diagnosis after the first surgical decompression (45%) and were found to have a longer interval between decompressions (7.6 6 2.1 months versus 4.01 6 3 months, P ¼ .0004). -
Escharotomy Incisions 17
Emergency War Surgery Second United States Revision of The Emergency War Surgery NATO Handbook Thomas E. Bowen, M.D. Editor BG, MC, U.S. Army Ronald Bellamy, M.D. Co-Editor COL, MC, U.S. Army United States Department of Defense United States Government Printing Office Washington, D.C. 1988 Peer Review Status: Internally Peer Reviewed Creation Date: Unknown Last Revision Date: 1988 Contents Foreword Preface Prologue Acknowledgments Chapter I. General Considerations of Forward Surgery Part I. Types of Wounds and Injuries II. Missile-Caused Wounds III. Burn Injury IV. Cold Injury V. Blast Injuries VI. Chemical Injury VII. Mass Casualties in Thermonuclear Warfare VIII. Multiple Injuries Part II. Response of the Body to Wounding IX. Shock and Resuscitation X. Compensatory and Pathophysiological Responses to Trauma XI. Infection Part III. General Considerations of Wound Management XII. Sorting of Casualties XIII. Aeromedical Evacuation XIV. War Surgery Within the Division XV. Anesthesia and Analgesia XVI. Wounds and Injuries of the Soft Tissues XVII. Crush Injury XVIII. Vascular Injuries XIX. Wounds and Injuries of Bones and Joints XX. Wounds and Injuries of Peripheral Nerves XXI. Amputations Part IV. Regional Wounds and Injuries XXII. Craniocerebral Injury XXIII. Maxillofacial Wounds and Injuries XXIV. Wounds and Injuries of the Eye XXV. Laser Injury of the Eye XXVI. Wounds and Injuries of the Ear XXVII. Wounds and Injuries of the Neck XXVIII. Wounds and Injuries of the Chest XXIX. Wounds of the Abdomen XXX. Reoperative Abdominal Surgery XXXI. Wounds and Injuries of the Genitourinary Tract XXXII. Wounds and Injuries of the Hand XXXIII. Wounds and Injuries of the Spinal Column and Cord Appendixes A. -
Protocols Full Document
STATE OF RHODE ISLAND WORKERS’ COMPENSATION COURT MEDICAL ADVISORY BOARD PROTOCOLS Approved by the Medical Advisory Board Rhode Island Workers’ Compensation Court John Parziale, MD Robert M. Ferrieri Chair Chief Judge Workers’ Compensation Court Sheila G. Mitchell, RN, BSN Medical Advisory Board Administrator Effective: 1/28/2020 1 TABLE OF CONTENTS YEAR PROTCOL WAS LAST REVIEWED . 3 PREFACE. 4 CARPAL TUNNEL SYNDROME . 5 CERVICAL MUSCULOLIGAMENTOUS INJURY (Sprain/Strain) . 8 HERNIATED CERVICAL DISC . 11 PROTOCOLS FOR INJURIES TO THE EYE . 15 PROTOCOL FOR THE EVALUATION AND MANAGEMENT OF ACUTE SHOULDER INJURIES . 26 PROTOCOL FOR THE MANAGEMENT OF ACUTE INJURIES TO THE KNEE . 29 LOW BACK MUSCULOLIGAMENTOUS INJURY (SPRAIN/STRAIN). 33 HERNIATED LUMBAR DISC. 36 LUMBAR FUSION.. 40 TRAUMATIC BRAIN INJURY.(formerly Post Traumatic Headache) . 42 CHRONIC REGIONAL PAIN SYNDROME (formerly Symp. Dyst.). 47 THORACIC OUTLET SYNDROME . 51 PROTOCOLS FOR INJURIES TO THE FOOT AND ANKLE. 54 WORKERS COMP. PROTOCOLS WHEN PRIMARY INJURY IS PSYCHIATRIC/PSYCHOLOGICAL. 69 OUTPATIENT PHYSICAL AND OCCUPATIONAL THERAPY PROTOCOL GUIDELINES. 84 ACOUSTIC TRAUMA. 91 INTERVENTIONAL PAIN MANAGEMENT TREATMENT PROTOCOL . 95 WORK HARDENING PROTOCOLS . 113 PROTOCOL FOR THE MANAGEMENT OF HERNIAS . .. 118 ACUPUNCTURE.. 123 TEMPOROMANDIBULAR JOINT DISORDERS.. 126 ACUTE HAND INJURY PROTOCOLS.. 131 PHARMACEUTICAL PROTOCOLS . 144 CONTACT DERMATITIS PROTOCOL. 145 CUBITAL TUNNEL SYNDROME.. 147 RADIAL TUNNEL SYNDROME . 150 SPINAL COLUMN STIMULATORS . 153 ANTERIOR CRUCIATE RUPTURES . 155 HEARING LOSS PROTOCOL . 157 INITIAL VOCATIONAL ASSESSMENT PROTOCOL GUIDELINES. 159 HIERARCHY OF VOCATIONAL REHABILITATION. 161 OCCUPATIONAL HEARING IMPAIRMENT TREATMENT PROTOCOL . 162 DIAGNOSIS AND INITIAL TREATMENT OF OCCUPATIONAL ASTHMA . 166 CHRONIC NONINTERVENTIONAL, NONCANCER PAIN PROTOCOL . 174 2 YEAR PROTOCOL WAS LAST REVIEWED PREFACE CARPAL TUNNEL SYNDROME . -
Nonoperative and Operative Management of Snapping Scapula
Clinical Sports Medicine Update Nonoperative and Operative Management of Snapping Scapula Robert C. Manske,*†‡ MEd, MPT, SCS, ATC, CSCS, Michael P. Reiman,‡ MEd, PT, ATC, CSCS, and Mark L. Stovak,‡ MD From †Wichita State University, Wichita, Kansas, and ‡Via Christi Regional Medical Center, Wichita, Kansas Snapping scapula is a painful crepitus of the scapulothoracic articulation. This crepitus is a grinding or snapping noise with scapulothoracic motion that may or may not accompany pain. This condition is commonly seen in overhead-throwing athletes. Treatment of patients with this syndrome begins with nonoperative methods; when nonoperative treatment fails, several surgi- cal options exist. This article will discuss both nonoperative and operative management of this common shoulder condition. Keywords: scapulothoracic crepitus; scapulothoracic bursitis; scapular disorders; shoulder rehabilitation Scapular function is crucial to not only the shoulder but ranges from simple annoyance to a truly disabling condi- also the entire upper extremity.As knowledge of the shoul- tion for the symptomatic patient. This crepitus is usually der and its surrounding structures has increased over the described as production of a snapping, grinding, thumping, past decade, so has interest in the scapula. The scapula’s or popping sound with scapulothoracic motion. This sound role is 2-fold: it is required to maintain a stable base of is amplified by the thoracic cavity, which acts as a reso- support for the humerus; it is also required to be mobile, nance chamber as in the body of a stringed instrument.61 allowing dynamic positioning of the glenoid fossa during Historically identified initially by Boinet,5 scapular crepi- glenohumeral elevation. -
Flexible Flatfoot
REVIEW ORTHOPEDICS & TRAUMATOLOGY North Clin Istanbul 2014;1(1):57-64 doi: 10.14744/nci.2014.29292 Flexible flatfoot Aziz Atik1, Selahattin Ozyurek2 1Department of Orthopedics and Tarumatology, Balikesir University Faculty of Medicine, Balikesir, Turkey; 2Department of Orthopedics and Traumatology, Aksaz Military Hospital, Marmaris, Mugla, Turkey ABSTRACT While being one of the most frequent parental complained deformities, flatfoot does not have a universally ac- cepted description. The reasons of flexible flatfoot are still on debate, but they must be differentiated from rigid flatfoot which occurs secondary to other pathologies. These children are commonly brought up to a physician without any complaint. It should be kept in mind that the etiology may vary from general soft tissue laxities to intrinsic foot pathologies. Every flexible flatfoot does not require radiological examination or treatment if there is no complaint. Otherwise further investigation and conservative or surgical treatment may necessitate. Key words: Children; flatfoot; flexible; foot problem; pes planus. hough the term flatfoot (pes planus) is gener- forms again (Figure 2). When weight-bearing forces Tally defined as a condition which the longitu- on feet are relieved this arch can be observed. If the dinal arch of the foot collapses, it has not a clinically foot is not bearing any weight, still medial longitu- or radiologically accepted universal definition. Flat- dinal arch is not seen, then it is called rigid (fixed) foot which we frequently encounter in routine out- flatfoot. To differentiate between these two condi- patient practice will be more accurately seen as a re- tions easily, Jack’s test (great toe is dorisflexed as the sult of laxity of ligaments of the foot. -
Osseous and Soft Tissue Pathology of the Thoracolumbar Spine and Pelvic Region
AAEP 360° Back Pain and Pelvic Dysfunction / 2018 Osseous and Soft Tissue Pathology of the Thoracolumbar Spine and Pelvic Region Kevin K. Haussler, DVM, DC, PhD, DACVSMR Author’s address—Gail Holmes Equine Orthopaedic Research bar articular processes, lumbar intertransverse joints, and the Center, Department of Clinical Sciences, College of Veterinary sacroiliac region are commonly used in performance horses Medicine and Biomedical Sciences, Colorado State University, with back or pelvic pain.13,14 Nuclear scintigraphy can provide Fort Collins, CO 80523; e-mail: [email protected] useful insights into areas of inflammation or horses with poorly 15,16 Take Home Message—Spinal disorders and sacroiliac joint localized back or pelvic pain. Advanced diagnostic imaging injuries have been identified as significant causes of reduced (CT, MRI) can be applied to the trunk and pelvic region of foals 17 performance in horses. and some small ponies or horses. Unfortunately, small gantry size has prevented full spine imaging in adult horses. I. INTRODUCTION II. CONGENITAL SPINAL MALFORMATIONS Clinical conditions affecting the axial skeleton can be Developmental variations in the morphology of thoracolumbar categorized as: (1) osseous disorders of the vertebral body, vertebral bodies, processes, and joints in horses are known to vertebral arch, or vertebral processes; (2) soft tissue disorders occur.18-23 Knowledge of normal spinal morphology and involving musculotendinous or ligamentous structures; and (3) vertebral anomalies is important for distinction of pathologic neurologic disorders that compromise the spinal cord or spinal change from normal anatomic variations. Congenital alterations nerves. Osseous lesions known to occur in the equine axial in the normal spinal curvature include variable degrees of 1,2 skeleton include spinous process impingement, degenerative scoliosis, lordosis and kyphosis.