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Sgs1
Helicase Mechanisms During Homologous Recombination in Saccharomyces Cerevisiae
A Mutation in the Putative MLH3 Endonuclease Domain Confers a Defect in Both Mismatch Repair and Meiosis in Saccharomyces Cerevisiae
A Genome-Wide Screen for Genes Affecting Spontaneous Direct-Repeat Recombination In
Fanconi Anemia, Bloom Syndrome and Breast Cancer
Pdf
Linking the Multiple Functions of Xpf-Ercc1 Endonuclease in Dna Repair to Health Outcomes: Cancer and Aging
Suppression of Spontaneous Genome Rearrangements in Yeast DNA Helicase Mutants
Crossing and Zipping: Molecular Duties of the ZMM Proteins in Meiosis Alexandra Pyatnitskaya, Valérie Borde, Arnaud De Muyt
Implications of Metastable Nicks and Nicked Holliday Junctions in Processing Joint Molecules in Mitosis and Meiosis
BLM Ortholog, Sgs1, Prevents Aberrant Crossing-Over by Suppressing Formation of Multichromatid Joint Molecules
Exo1 Recruits Cdc5 Polo Kinase to Mutlγ to Ensure Efficient Meiotic Crossover Formation
Examination of the Roles of Sgs1 and Srs2 Helicases in the Enforcement of Recombination Fidelity in Saccharomyces Cerevisiae
Molecular Mechanism of Double Holliday Junction Dissolution Paolo Swuec and Alessandro Costa*
SHOC1 Is a ERCC4-(Hhh)2-Like Protein, Integral to the Formation of Crossover Recombination Intermediates During Mammalian Meiosis
Mutation in SGS1 Does Not Increase the Prevalence of Mmbir but Causes a Reduction in the Proportion of Gene Conversions
Downloaded from the Saccharomyces Genome Database Segregation
Resection Activity of the Sgs1 Helicase Alters the Affinity of DNA
Insights Into the Conserved Regulatory Mechanisms of Human and Yeast Aging
Top View
Holliday Junction-Containing DNA Structures Persist in Cells Lacking Sgs1 Or Top3 Following Exposure to DNA Damage
Structure-Specific Endonucleases and the Resolution of Chromosome Underreplication
WRN Helicase Defective in the Premature Aging Disorder Werner Syndrome Genetically Interacts with Topoisomerase 3 and Restores the Top3 Slow Growth Phenotype of Sgs1 Top3
Maintenance of Genome Integrity and Active Homologous Recombination
DNA Helicases Sgs1 and BLM Promote DNA Double-Strand Break Resection
Heteroduplex Rejection During Single-Strand Annealing Requires Sgs1 Helicase and Mismatch Repair Proteins Msh2 and Msh6 but Not Pms1
Functional Overlap Between Sgs1–Top3 and the Mms4–Mus81 Endonuclease
1 the Absence of Top3 Reveals an Interaction Between the Sgs1 And
The Human Bloom Syndrome Gene Suppresses the DNA Replication and Repair Defects of Yeast Dna2 Mutants
Sgs1 Binding to Rad51 Stimulates Homology-Directed DNA Repair in Saccharomyces Cerevisiae
Al-Sweel Et Al. 1 Mlh3 Separation of Function and Endonuclease
The Werner Syndrome Gene Reviews the Werner Syndrome Gene the Molecular Basis of Recq Helicase- Deficiency Diseases
The Regulation of Homologous Recombination by Helicases
Regulatory Control of Sgs1 and Dna2 During Eukaryotic DNA End Resection
The Dissolution of Double Holliday Junctions
The Role of Sgs1 and Mph1 in DNA Repair and Mismatch Correction A
Structure-Specific Endonucleases Xpf and Mus81 Play Overlapping but Essential Roles in DNA Repair by Homologous Recombination
SGS1/BLM Suppresses R-Loop Formation 1 Conserved
Regulation of DNA Pairing in Homologous Recombination
Mechanisms of Ageing and Development Bloom's Syndrome
Functional Conservation of Recq Helicase BLM Between Humans and Drosophila Melanogaster Rebecca L
LOSS of BLOOM SYNDROME PROTEIN CAUSES DESTABILIZATION of GENOMIC ARCHITECTURE and IS COMPLEMENTED by ECTOPIC EXPRESSION of Escherichia Coli Recg in HUMAN CELLS
Regulation of Structure-Specific Endonucleases in Replication Stress
Distinct DNA Repair Pathways Cause Genomic Instability at Alternative DNA Structures
The Bloom's Syndrome Helicase Interacts Directly with the Human
Functions of Recq Family Helicases: Possible Involvement of Bloom's and Werner's Syndrome Gene Products in Guarding Genome Integrity During DNA Replication
Mechanisms and Functions of DNA Mismatch Repair Guo-Min Li1
A Meiotic XPF–ERCC1-Like Complex Recognizes Joint Molecule Recombination Intermediates to Promote Crossover Formation
Maintenance of Yeast Genome Integrity by Recq Family DNA Helicases
Rad51-Dependent DNA Structures Accumulate at Damaged Replication Forks in Sgs1 Mutants Defective in the Yeast Ortholog of BLM Recq Helicase
Sgs1’S Roles in DNA End Resection, HJ Dissolution, and Crossover Suppression Require a Two-Step SUMO Regulation Dependent on Smc5/6
A Senataxin-Associated Exonuclease SAN1 Is Required for Resistance to DNA Interstrand Cross-Links