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SEC63
A Clearer Picture of the ER Translocon Complex Max Gemmer and Friedrich Förster*
Aneuploidy: Using Genetic Instability to Preserve a Haploid Genome?
Genetic Editing of SEC61, SEC62, and SEC63 Abrogates Human
Membrane Phospholipid Alteration Causes Chronic ER Stress Through
Sec63 and Xbp1 Regulate Ire1α Activity and Polycystic Disease Severity
Protein Folding and Quality Control in the ER
Renoprotective Effect of Combined Inhibition of Angiotensin-Converting Enzyme and Histone Deacetylase
Protein Translocation Across the Rough Endoplasmic Reticulum
Stepwise Gating of the Sec61 Protein-Conducting Channel by Sec63 and Sec62
Different Effects of Sec61a, Sec62 and Sec63 Depletion on Transport of Polypeptides Into the Endoplasmic Reticulum of Mammalian Cells
ER Membrane Protein Complex Required for Nuclear Fusion Davis T.W
Signal Sequences Specify the Targeting Route to the Endoplasmic Reticulum Membrane Davis T.W
Determining the Causes of Recessive Retinal Dystrophy
Chromosomal Abnormalities in Hepatic Cysts Point to Novel Polycystic Liver Disease Genes
Mutation of Sec63 in Zebrafish Causes Defects in Myelinated Axons and Liver Pathology Kelly R
The Sec62–Sec63 Translocon Facilitates Translocation of the C
The Protein Translocation Channel Mediates Glycopeptide Export Across the Endoplasmic Reticulum Membrane
Data-Driven and Knowledge-Driven Computational Models of Angiogenesis in Application to Peripheral Arterial Disease
Top View
Identification of Signal Peptide Features for Substrate Specificity in Human
And Posttranslational Protein Translocation Into Theyeast
TMEM258 Is a Component of the Oligosaccharyltransferase Complex Controlling ER Stress and Intestinal Inflammation
Whole-Exome Sequencing Reveals LRP5 Mutations and Canonical Wnt Signaling Associated with Hepatic Cystogenesis
Human Diseases Affecting Gating of the Sec61 Complex
Investigating the Influence of Perinatal Nicotine and Alcohol Exposure On
The Fate of Endoplasmic Reticulum-Targeted Proteins in the Face of Proteasome Failure
The Sec63/Bip Complex Suppresses Higher-Order Oligomerization and Rnase Activity of Ire1α During ER Stress Xia Li1, Sha Sun1, S
Endoplasmic Reticulum Associated Protein Degradation (ERAD): the Function of Dfm1 and Other Novel Components of the Pathway
Sec63 and Xbp1 Regulate Ire1α Activity and Polycystic Disease Severity
Pulling Force Studies of Secretory Protein Translocation Into The
The Sec63/Bip Complex Suppresses Higher-Order Oligomerization And