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- Retinoblastoma Protein and the Leukemia-Associated PLZF Transcription Factor Interact to Repress Target Gene Promoters
- The Regulation of E2F by Prb-Family Proteins
- AP-1-MEDIATED REGULATION of HPV CHROMATIN TRANSCRIPTION by WEI-MING WANG Submitted in Partial Fulfillment of the Requirements Fo
- The Retinoblastoma Gene Product Protects E2F-1 from Degradation by the Ubiquitin-Proteasome Pathway
- Lh14e's Effects Are Mediated Through P21 and Related
- Differential Action on Transcriptional Programs Related to Cell Cycle Control and Immune Function
- Cell Stemness Is Maintained Upon Concurrent Expression of RB and the Mitochondrial Ribosomal Protein S18-2
- Retinoblastoma Protein and CCAAT/Enhancer-Binding Protein  Are
- The Retinoblastoma Gene Product Regulates Spl-Mediated Transcription SEONG-JIN KIM,1 UCHE S
- Hyperphosphorylation of Retinoblastoma Protein and P53 by Okadaic Acid, a Tumor Promoter
- Retinoblastoma Tumor Suppressor Protein Roles in Epigenetic Regulation
- Multiple Mechanisms for E2F Binding Inhibition by Phosphorylation of the Retinoblastoma Protein C-Terminal Domain
- Role of C-Fos and E2F in the Induction of Cyclin a Transcription and Vascular Smooth Muscle Cell Proliferation
- Defective Human Retinoblastoma Protein Identified by Lack of Interaction with the E1A Oncoprotein I
- Reversal of an Antiestrogen-Mediated Cell Cycle Arrest of MCF-7 Cells by Viral Tumor Antigens Requires the Retinoblastoma Protein-Binding Domain
- E2F Transcription Factor in Mouse Cancer Models (3, 15), and Mutations Are More Com- Family
- Adenovirus E1A Downregulates Cjun- and Junb-Mediated Transcription by Targeting Their Coactivator P300
- The Disease, Gene and Protein Provide Critical Leads to Understand Cancer
- Histone Chaperone Jun Dimerization Protein 2 (Jdp2): Role in Cellular Senescence and Aging
- WT1 Promotes Cell Proliferation in Non-Small Cell Lung Cancer Cell Lines Through Up-Regulating Cyclin D1 and P-Prb in Vitro and in Vivo
- Involvement of Retinoblastoma Protein and HBP1 in Histone H1(0) Gene Expression
- The Retinoblastoma Protein-Associated Transcription Repressor Rbak Interacts with the Androgen Receptor and Enhances Its Transcriptional Activity
- Retinoblastoma Protein Associates with SP1 and Activates the Hamster Dihydrofolate Reductase Promoter
- Role of the Retinoblastoma Protein Family, Prb, P107 and P130 in the Negative Control of Cell Growth
- Regulation of Kru¨Ppel-Like Factor 6 Tumor Suppressor Activity by Acetylation
- REVIEW the Emerging Role of Kru¨Ppel-Like Factors in Endocrine-Responsive Cancers of Female Reproductive Tissues
- Targeting Cancer Cell Metabolism with Mitochondria- Immobilized Phosphorescent Cyclometalated Iridium(III) Complexes
- Wilmsâ•Ž Tumor-1 (Wt1) Protein Expression in Glioma Cells
- The Retinoblastoma Protein Is an Essential Mediator That Links the Interferon-Inducible 204 Gene to Cell-Cycle Regulation
- Retinoblastoma Protein Positively Regulates Terminal Adipocyte Differentiation Through Direct Interaction with C/Ebps
- Subnuclear Localization of WT1 in Splicing Or Transcription Factor Domains Is Regulated by Alternative Splicing
- Expression of Krüppel-Like Factor 6, KLF6, in Rat Pituitary Stem
- Induction of Androgen Receptor-Dependent Apoptosis in Prostate Cancer Cells by the Retinoblastoma Protein
- A Gatekeeper to Hormone Independence in Prostate Cancer?
- Tumor-Suppressor Genes: Cardinal Factors in Inherited Predisposition to Human Cancers by H
- Direct Regulation of DNA Repair by E2F and RB in Mammals and Plants: Core Function Or Convergent Evolution?
- Paradoxical Increase in Retinoblastoma Protein in Colorectal Carcinomas May Protect Cells from Apoptosis1
- Retinoblastoma Gene Mutations Detected by Whole Exome Sequencing of Merkel Cell Carcinoma
- Myoblasts Rely on Tap63 to Control Basal Mitochondria Respiration
- Cyclin-Dependent Kinase Inhibition by the KLF6 Tumor Suppressor Protein Through Interaction with Cyclin D1
- Functional Interactions Between the Retinoblastoma (Rb) Protein and Sp
- Stable Binding to E2F Is Not Required for the Retinoblastoma Protein to Activate Transcription, Promote Differentiation, and Suppress Tumor Cell Growth