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PRDX4
Peroxiredoxins in Neurodegenerative Diseases
PRDX4 (Human) ELISA Kit 1
The Prognostic Values of the Peroxiredoxins Family in Ovarian Cancer
The Role of Vitamin C in the Gene Expression of Oxidative Stress Markers in Fibroblasts from Burn Patients1
Anti-Oxidant Pathogenesis of High-Grade Glioma DISSERTATION
Integrative Analysis the Characterization of Peroxiredoxins in Pan-Cancer
Functional Genomics Study of Acute Heat Stress Response in the Small
Hudson Udel 0060D 13
World Journal of Biological Chemistry
Suppression of MG132-Mediated Cell Death by Peroxiredoxin 1 Through Influence on ASK1 Activation in Human Thyroid Cancer Cells
Murine Perinatal Beta Cell Proliferation and the Differentiation of Human Stem Cell Derived Insulin Expressing Cells Require NEUROD1
Phospholipase A2 of Peroxiredoxin 6 Has a Critical Role in Tumor Necrosis Factor-Induced Apoptosis
Peroxiredoxin 4 (PRDX4): Its Critical in Vivo Roles in Animal Models of Metabolic Syndrome Ranging from Atherosclerosis to Nonalcoholic Fatty Liver Disease
Peroxiredoxins and Immune Infiltrations in Colon Adenocarcinoma
Intracellular Sources of ROS/H2O2 in Health and Neurodegeneration: Spotlight on Endoplasmic Reticulum
Comprehensive Analysis of Peroxiredoxins Expression Profiles
1 Electronic Supporting Information 2 Effect of Secondary Organic
The Role of Gstπ Isoform in the Cells Signalling and Anticancer Therapy
Top View
Clinical Relevance of Thyroid Cell Models in Redox Research
Cancer Cell International
The Influence of Selol on the Expression of Oxidative Stress Genes in Normal and Malignant Prostate Cells
Prdx Overexpression in Tumor Tissue of Breast Cancer Patients Jillian Muhlbauer1, Shelley a Phelan2*
Destroy and Exploit: Catalyzed Removal of Hydroperoxides from the Endoplasmic Reticulum
Peroxiredoxins—The Underrated Actors During Virus-Induced Oxidative Stress
The Enzymatic Antioxidant System of Human Spermatozoa
Testis-Specific Peroxiredoxin 4 Variant Is Not Absolutely Required
Catalyzed Removal of Hydroperoxides from the Endoplasmic Reticulum
Supporting Information
(Thioredoxin Peroxidase-2) Protects Cancer Cells Against Hypoxia and Drug-Induced Hydrogen Peroxide-Dependent Apoptosis
Pulmonary Endoplasmic Reticulum Stress: Scars, Smoke and Suffocation
Antioxidants
Peroxiredoxin 2 Specifically Regulates the Oxidative and Metabolic Stress Response of Human Metastatic Breast Cancer Cells in Lu
Prdx6 Deficiency Ameliorates DSS Colitis: Relevance of Compensatory Antioxidant Mechanisms
Two Phases of Disulfide Bond Formation Have Differing Requirements for Oxygen
Supplementary Informations
Thioredoxin and Glutaredoxin Systems
Roles of Antioxidative Enzymes in Wound Healing
PRDX2 and PRDX4 Are Negative Regulators of Hypoxia-Inducible Factors Under Conditions of Prolonged Hypoxia
The Ketogenic Diet Reverses Gene Expression Patterns and Reduces
Redox Regulation Via Glutaredoxin-1 and Protein S-Glutathionylation
PRDX5 Is Differentially Expressed in the Brains of Patients with Psychotic
ERO1-Independent Production of H Within the Endoplasmic Reticulum
Detection and Identification of Peroxiredoxin 3 As a Biomarker in Hepatocellular Carcinoma by a Proteomic Approach