DOCSLIB.ORG
Explore
Sign Up
Log In
Upload
Search
Home
» Tags
» NDUFA9
NDUFA9
A Computational Approach for Defining a Signature of Β-Cell Golgi Stress in Diabetes Mellitus
Low Abundance of the Matrix Arm of Complex I in Mitochondria Predicts Longevity in Mice
Defective NDUFA9 As a Novel Cause of Neonatally Fatal
Dehydroepiandrosterone Induces Growth Arrest of Hepatoma Cells Via Alteration of Mitochondrial Gene Expression and Function
Coupling Mitochondrial Respiratory Chain to Cell Death: an Essential Role of Mitochondrial Complex I in the Interferon-B and Retinoic Acid-Induced Cancer Cell Death
Electron Transport Chain Activity Is a Predictor and Target for Venetoclax Sensitivity in Multiple Myeloma
Transcriptomic and Proteomic Landscape of Mitochondrial
Pioglitazone Leads to an Inactivation and Disassembly of Complex I of The
Respiratory Chain Activity and Protein Expression in Skeletal Muscles from High and Low Feed Efficient Swine Within the Same Genetic Line
Autocrine IFN Signaling Inducing Profibrotic Fibroblast Responses By
Accessory Subunits Are Integral for Assembly and Function of Human Mitochondrial Complex I
Table S1. 103 Ferroptosis-Related Genes Retrieved from the Genecards
Structures of the Human LONP1 Protease Reveal Regulatory Steps Involved in Protease Activation
Mitochondrial Sirt3 Contributes to the Bone Loss Caused by Aging Or Estrogen Deficiency
NDUFA9 (20C11): Sc-58392
Many Mitochondrial Proteins Require the Assistance of Chaperones to Fold Into Their Native Conformation After Their Import Into Mitochondria
Analysis of Human Mutations in the Supernumerary Subunits of Complex I
Table 2. List of 113 Potential Blastocystis Mitochondrial Proteins and Proteins Associated with Mitochondria with Their EST Accession Numbers
Top View
LONP1 and Mthsp70 Cooperate to Promote Mitochondrial Protein Folding
A Role for the Mitochondrial Deacetylase Sirt3 in Regulating Energy Homeostasis
Sirtuin 3 Expression and Acetylation of Three Downstream Targets in Painted Turtle Liver and Brain During Anoxia
Cardiomyocyte-Specific Deletion of GCN5L1 in Mice Restricts
Mammalian Mitochondrial Complex I Structure and Disease-Causing Mutations
Mitochondrial Metabolism, Sirtuins, and Aging
Subcellular Compartmentalization of NAD+ and Its Role in Cancer: a Serenade of Metabolic Melodies
Modeling Gene Regulation from Paired Expression and Chromatin Accessibility Data
Defective NDUFA9 As a Novel Cause Of
P53-Inducible DPYSL4 Associates with Mitochondrial Supercomplexes and Regulates Energy Metabolism in Adipocytes and Cancer Cells
Supplementary Table 1. the Clinical Characteristics of SCC Patients Analyzed by RNA 2 Sequencing
Expression of LONP1 Is High in Visceral Adipose Tissue in Obesity, and Is Associated with Glucose and Lipid Metabolism
SIRT3 Deficiency Exacerbates P53/Parkin‑Mediated Mitophagy Inhibition and Promotes Mitochondrial Dysfunction: Implication for Aged Hearts
NDUFA9 Rabbit Polyclonal Antibody – TA342228 | Origene
NDUFA10 Mutations Cause Complex I Deficiency in a Patient with Leigh Disease
Table of Contents List of Investigators
Inbred Mouse Strains Expression in Primary Immunocytes Across
Interplay Between TRAP1 and Sirtuin-3 Modulates Mitochondrial