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Complement Factor I
Clinical Study High Complement Factor I Activity in the Plasma of Children with Autism Spectrum Disorders
Advances in Hematology
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Development and Validation of a Protein-Based Risk Score for Cardiovascular Outcomes Among Patients with Stable Coronary Heart Disease
Structural and Functional Analysis of Complement Factor H: a Crucial Protein in Several Disorders
Complement in Tumourigenesis and the Response to Cancer Therapy
Complement System and Potential Therapeutics in Age-Related Macular Degeneration
Implications in C3 Glomerulopathies This Information Is Current As of September 26, 2021
Functional Expression of Complement Factor I Following AAV-Mediated Gene Delivery in the Retina of Mice
Probe Set Name Symbol 1598 G at Growth Arres
Electronic Supplementary Material (ESI) for Journal of Materials Chemistry B
Complement Factor I Deficiency
Complement As a Therapeutic Target in Systemic Autoimmune Diseases
SUPPLEMENTARY DATA Retinoic Acid Mediates Visceral-Specific
Complement Factor I in Health and Disease. Nilsson, Sara
Comparative Proteomic Analysis in Scar-Free Skin Regeneration in Acomys Cahirinus and Scarring Mus Musculus Jung Hae Yoon1, Kun Cho 2, Timothy J
Comprehensive Genetic Analysis of Complement and Coagulation Genes in Atypical Hemolytic Uremic Syndrome
Top View
Structural Basis for Complement Factor I Control and Its Disease-Associated Sequence Polymorphisms
CFI Gene Complement Factor I
A New Player in the Alternative Complement Pathway: MASP-1 Is Essential for LPS-Induced but Not for Zymosan-Induced Alternative Pathway Activation
Glomerular Endothelial Cells Impairs Activation of Protein C in Human
Glycerol-3-Phosphate Is a FGF23 Regulator Derived from the Injured Kidney
Complement Factor I Deficiency Associated with Recurrent Meningitis Coinciding with Menstruation
Complement Factor H: Solution Structures and Interactions with Ligands
Article Factor I Autoantibodies in Patients with Atypical
Proteomic Landscape of the Human Choroid–Retinal Pigment Epithelial Complex
Cutaneous Vasculitis and Recurrent Infection Caused by Deficiency in Complement Factor I
Tryptase from C3, C4, and C5 Β Generation of Anaphylatoxins By
PQ500™ Reference Peptides Kit Manual Available At
Complement System in Cutaneous Squamous Cell Carcinoma
Diagnostic and Prognostic Significance of Complement in Patients with Alcohol-Associated Hepatitis
Article Concentration (A), Mode Sizes (B) and Size Distribution Curves (C) Were Assessed by Nanoparticle Tracking Analysis
Complement Activation in Peritoneal Dialysis–Induced Arteriolopathy
Pearls of Laboratory Medicine
Complement in Age-Related Macular Degeneration: a Focus on Function
Targeted Proteomic Analysis of Human Plasma on a Discovery Scale Pq500 & Tsq Altis Mass Spectrometer
Common Polymorphisms in C3, Factor B, and Factor H Collaborate to Determine Systemic Complement Activity and Disease Risk
The Story of Complement Factor I T Peter J Lachmann
Variation of Complement Factor H and Mannan Binding Lectin in Human
Molecular Immunology Dense Deposit Disease
Enrichment and Identification of Differentially Expressed Genes in Hepatocellular Carcinoma Stem‑Like Cells
European Society for Immunodeficiencies (ESID) And
Factor I-Dpl Catalog Number
The Genetic and Molecular Bases of Monogenic Disorders Affecting Proteolytic Systems
Modified Proteases That Inhibit Complement Activation
C3 Mutation Reveals a Mechanism of Dense Deposit Disease Pathogenesis and Provides Insights Into Complement Activation and Regulation
The Role of the Immune Response in Age-Related Macular Degeneration." International Journal of Inflammation
Text S1: Supporting Information
Mutations in Complement Factor I Predispose to Development of Atypical Hemolytic Uremic Syndrome
Complement C2
Complement Proteins As Soluble Pattern Recognition Receptors for Pathogenic Viruses
Mouse Genetics and Proteomic Analyses Demonstrate a Critical Role for Complement in a Model of DHRD/ML, an Inherited Macular Degeneration
The Footprint of the Ageing Stroma in Older Patients with Breast Cancer
Cutaneous Vasculitis and Digital Ischaemia Caused by Heterozygous Gain-Of-Function Mutation in C3