Cdc25
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- Chk1-Mediated Cdc25a Degradation As a Critical Mechanism for Normal Cell Cycle Progression Hidemasa Goto1,*, Toyoaki Natsume2,3, Masato T
- UNIVERSITY of CALIFORNIA, SAN DIEGO The
- Oncogenic Tyrosine Phosphatases: Novel Therapeutic Targets for Melanoma Treatment
- Multiple Isoforms of CDC25 Oppose ATM Activity to Maintain Cell Proliferation During Vertebrate Development
- 4. WNK Kinases in Invasion and Metastasis
- Roles of Cyclin Dependent Kinase and Cdk
- Molecular Characterisation of Crypα, an Axonal Receptor Protein Tyrosine
- UNIVERSITY of CALIFORNIA, SAN DIEGO Identification of Specific
- 1 Pharmacophore-Guided Discovery of CDC25 Inhibitors Causing Cell
- Protein Tyrosine Phosphatase Structure–Function Relationships In
- Cdc25 Inhibition and Cell Cycle Arrest by a Synthetic Thioalkyl Vitamin K Analogue1
- Role of Receptor Protein Tyrosine Phosphatases (Rptps) in Insulin Signaling and Secretion
- A Tumor Cell-Selective Inhibitor of Mitogen-Activated Protein Kinase Phosphatases Sensitizes Breast Cancer Cells to Lymphokine-Activated Killer Cell Activity S
- The Phosphoinositol Phosphatase Activity of PTEN Mediates a Serum-Sensitive Gl Growth Arrest in Glioma Cells
- Cycling at the Interface Between Neurodevelopment and Neurodegeneration
- The Vulnerability of RB Loss in Breast Cancer: Targeting a Void in Cell Cycle Control
- Customized Array Comparative Genomic Hybridization Analysis Of
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