Neurologic System

Total Page:16

File Type:pdf, Size:1020Kb

Neurologic System Neurologic System ✽ The neurological or nervous system is composed of c. Occipital: center for visual perception and two primary areas: the central nervous system, which integration. includes the brain and the spinal cord, and the peripheral d. Temporal: controls auditory, verbal, visual, nervous system, which includes the entire network of memory, and personality functions. nerves extending from the central nervous system. e. Some functions of the brain occur in more than one lobe. PHYSIOLOGY OF THE 2. Cerebellum: contains more neurons than the rest of the brain combined. It serves to interpret motor and NEUROLOGICAL SYSTEM mental dexterity, as well as sense of balance. Central Nervous System 3. Brain stem: conduit for all information transmission between upper and lower nervous system. Consists The brain and the spinal cord within the vertebral column of pons, midbrain, and medulla. make up the central nervous system (CNS). a. Pons: responsible for alertness; relays sensory A. The brain and the spinal cord are protected by the rigid information between cerebellum and cerebrum. bony structure of the skull and by the vertebral column, b. Midbrain: interprets auditory and visual refl exes. respectively (Figure 15-1). c. Medulla: lower portion of the brain stem. Con- B. Meninges: the protective membranes that cover the trols autonomic functions. brain and are continuous with those of the spinal cord. (1) Respiratory center for changes in rate and 1. Pia mater: covers the surfaces of the brain and the depth of breathing. spinal column. (2) Controls heart rate. 2. Arachnoid: waterproof membrane that encases the (3) Vomiting refl ex center. entire CNS; the subarachnoid space contains the (4) Swallowing refl ex center. cerebrospinal fl uid. 4. Diencephalon: contains thalamus and hypo- 3. Dura mater: a tough membrane that provides pro- thalamus. tection to the brain and spinal cord. C. Cerebrospinal fl uid (CSF) (Figure 15-2). 1. Serves to cushion and protect the brain and spinal cord; the brain literally fl oats in CSF. 2. CSF is clear, colorless, watery fl uid; approximately 100- to 200-ml total volume, with a normal fl uid pressure of 70 to 150 cm of water (average: 125 cm of water pressure). 3. Formation and circulation of CSF: CSF is formed continuously by the choroid plexus located in the ventricles of the brain. It is reabsorbed by the arachnoid villi in the ventricles at the same rate it is formed. D. Brain: The basic brain anatomy consists of the cere- brum, the cerebellum, the brain stem, and the interior structures. 1. Cerebrum: the largest portion of the brain; sepa- rated into hemispheres. Each hemisphere is divided into four lobes: frontal, parietal, occipital, and temporal. a. Frontal: memory, language, personality, and emotions are primarily controlled here; highly vulnerable to traumatic brain injury. FIGURE 15-1 Major divisions of the central nervous system. (From Lewis SL et al: Medical-surgical nursing: assessment and b. Parietal: integrates sensory information and management of clinical problems, ed 7, St. Louis, 2007, Mosby.) interprets spatial relationships. 307 308 CHAPTER 15 Neurological System FIGURE 15-2 Cerebrospinal fluid circulation. Cerebrospinal fluid is produced by choroid plexus in lateral ventricles and flows around brain and spinal cord until it reaches arachnoid villi, from which it is absorbed into venous circulation. Arrows indicate major pathway of cerebrospinal fluid flow.From From Monahan FD et al: Medical-surgical nursing: health and illness perspectives, ed 8, St. Louis, 2007, Mosby. a. Thalamus: receives and relays auditory, sensory, a. Cranial nerves: responsible for a variety of func- and visual signals. tions, from hearing and balance to regulation of b. Hypothalamus: controls body temperature regu- chemoreceptors in the aortic body. lation, sleep/wake cycles, and appetite; regulates b. Spinal nerves: each pair of nerves is numbered the pituitary gland. according to the level of the spinal cord from 5. Limbic system: regulates emotions, drives, and which it originates. appetite. 2. Autonomic nervous system: regulates involuntary E. Spinal cord: a nerve bundle transmitting messages to activity (e.g., cardiovascular, respiratory, metabolic, and from the body. body temperature) (Table 15-1). 1. Extends from medulla to first or second lumbar a. Parasympathetic division: maintains normal vertebra. body functions. 2. Nerves exit and enter the spinal cord at each b. Sympathetic division: prepares the body to meet vertebral body and communicate with specific areas a challenge or an emergency; “fight or flight.” of the body. c. Most of the organs of the body receive innerva- 3. Rings of bony vertebrae surround and protect cord tion from both the parasympathetic and the sym- and nerve roots. pathetic divisions. 4. Intervertebral disks cushion and provide flexibility to the spinal column. System Data Collection Peripheral Nervous System (PNS) A. History. The PNS consists of sensory and motor neurons. The PNS is 1. Neurological history. subdivided into the sensory/somatic nervous system and the 2. Medical history. autonomic nervous system. a. Chronic, concurrent medical problems. A. Types of nerves. b. Medications (especially tranquilizers, sedatives, 1. Sensory (afferent or ascending) nerves relay sensa- and narcotics). tions to the brain for response. c. If client is an infant or young child, a pregnancy 2. Motor components of cranial nerves (efferent or de- and delivery history of the mother is obtained. scending) send messages from the brain d. Sequence of growth and development. to muscles, glands, and specialized tissues (e.g., 3. Family history: presence of hereditary or congenital heart and lungs). problems. B. Functions of PNS: both sensory/somatic and autonomic 4. Personal history: activities of daily living; any responsibilities. change in routine. 1. Sensory/somatic nervous system consists of cranial 5. History and symptoms of current problem. nerves and spinal nerves. a. Paralysis or paresthesia. CHAPTER 15 Neurological System 309 TABLE 15-1 AUTONOMIC NERVOUS SYSTEM AREA AFFECTED SYMPATHETIC (FIGHT OR FLIGHT) PARASYMPATHETIC Pupil Dilates Constricts Bronchi Dilates Constricts Heart Increases rate Decreases rate Gastrointestinal Inhibits peristalsis Stimulates peristalsis Stimulates sphincter Inhibits sphincters Bladder Relaxes bladder Contracts bladder muscle muscle Constricts sphincter Relaxes sphincter TABLE b. 15- Syncope, dizziness.2 GLASGOW COMA SCALE 2. Mental status (must take into consideration the c. Headache. client’s age, culture, and educational background) CATE GORY d. Speech problems.APPROPRIATE RESPONSE (Box 15-1, Box 15-2). SCORE e. Visual problems. a. General appearance and behavior. OF RESPONSE STIMULUS f. Changes in personality. b. Level of consciousness. Eyes open g. Memory loss.Approach bedside Spontaneous response (1) Oriented to person, place, and time. 4 h. Nausea, vomiting. (2) Appropriate response to verbal and tactile Verbal command Opening of eyes to name or command 3 B. Physical assessment. stimuli. 1. General observation of client.Pain Lack of opening of eyes (3) to previous Memory, problem-solving abilities.stimuli but opening 2 a. Posture, gait. to pain c. Mood. b. Position of rest for the infant or young child. d. Thought content and intellectual capacity. c. Personal hygiene, grooming. Lack of opening of eyes e. Judgment and abstract thinking.to any stimulus 1 d. Evaluate speech and ability to communicate. f. Perceptual distortion. (1) Pace of speech: rapid, slow, halting. Untestable 3. Assess pupillary status and eye movements. U (2) Clarity: slurred or distinct. a. Size of pupils should be equal. Best verbal response Verbal questioning with maximum Appropriate orientation, conversant, correct identifica- 5 (3) Tone: high pitched, rough.arousal tion of self, place, year b. , and Reaction of pupils. month (4) Vocabulary: appropriate choice of words. (1) Direct light refl ex: constriction of pupil e. Facial features may suggest specifi c syndromes Confusion; conversant, but disorientated when light is shown directly into eye.in one or more 4 in children. spheres (2) Accommodation: pupillary constriction to accommodate near vision. BOX 15-1 OLDER ADULT CARE FOCUS Inappropriate or disorganized c. BOX Evaluate eye movements. 15-2use of wordsOLDER ADULT 3CARE FOCUS (e.g., curs ing), lack of sustained (1) Note nystagmus: fi ne, jerking eye move- conversation Assessing Neurologic Function Causes of Confusion in the Older Adult Client ment. in Older Adults Incomprehensible words, Decreased (2) sounds Ability of eyes to move together.(e.g., Cardiacmoaning) Output 2 • (3) Myocardial Resting position should be at mid-position infarction Signs of Cognitive Impairment Lack of sound, even with painful stimuli 1 • Dysrhythmias of the eye socket. • Significant memory loss (person, place, and time). • Congestive heart failure • Person: Does client know who he or she is, and can client Untestable d. PERRLA: Indicates that Pupils are UEqual, give you his or her full name? Hypoxia/RespiratoryRound, and Reactive to AcidosisLight, and that Accom- Best• Place:motor Canresponse client identifyVerbal hiscommand or her home(e.g., address“raise your and Obedience in response • to command modation is present. Pneumonia 6 where he or she is now?arm, hold up two fingers.” 4. Evaluate motor function.• Infection • Time: What was the most recent holiday; what month, a. • Assess face and upper extremities for equality of Hypoventilation time of day, day of thePain week(pressure
Recommended publications
  • Postural Determinants in the Blind. Final Report. INSTITUTION Illinois Visually Handicapped Inst., Chicago, Ill
    DOCUMENT RESUME ED 048 714 EC 031 990 AUTHOR Siegal, Irwin M.; Murphy, Thomas J. TITLE Postural Determinants in the Blind. Final Report. INSTITUTION Illinois Visually Handicapped Inst., Chicago, Ill. SPONS AGENCY Social and Rehabilitation Service (DHEW) , Washington, D.C. Div. of Research and Demonstration Grants. PUB DATE Aug 70 NOTE 113p. EDRS PRICE EDRS Price MF-$0.65 HC-$6.58 DESCRIPTORs Body Image, *Exceptional Child Research, Exercise (Physiology), *Human Posture, Physical Therapy, *Visually Handicapped, *Visually Handicapped Mobility, *Visually Handicapped Orientation ABSTRACT The problem of malposture in the blind and its attect on orientation and travel skills was explored. A group of 45 students were enrolled in a standard 3-month mobility training program.Ea,_:h student suite red a postural problem, some compounded by severe orthopedic and/or neurological deficit. All subjects were given complete orthopedic and neurological examinations as well as a battery of special psychometric tests. Postural problems were diagnosed and treated by a variety of therapeutic techniques, some newly describd, including specialized exercise, splintage, and postural physical education programs. Improvement evaluation (by motion picture photography) was made before, during and after the 3-month program. The hypothesis tested was that improvement in posture contributed to improvement in mobility. The final results indicated such a correlation to exist. One implication is that postural training plays an important role in the development of mobility skills and thus in the total rehabilitation of the blind. (Author) ,'~ EC031990 FINAL REPORT POSTURAL DETERMINANTS IN THE BLIND (The Influence of Posture on Mobility and Orientation) IRWIN M. SIEGEL, M.D., Chief Investigator THOMAS J.
    [Show full text]
  • A Dictionary of Neurological Signs
    FM.qxd 9/28/05 11:10 PM Page i A DICTIONARY OF NEUROLOGICAL SIGNS SECOND EDITION FM.qxd 9/28/05 11:10 PM Page iii A DICTIONARY OF NEUROLOGICAL SIGNS SECOND EDITION A.J. LARNER MA, MD, MRCP(UK), DHMSA Consultant Neurologist Walton Centre for Neurology and Neurosurgery, Liverpool Honorary Lecturer in Neuroscience, University of Liverpool Society of Apothecaries’ Honorary Lecturer in the History of Medicine, University of Liverpool Liverpool, U.K. FM.qxd 9/28/05 11:10 PM Page iv A.J. Larner, MA, MD, MRCP(UK), DHMSA Walton Centre for Neurology and Neurosurgery Liverpool, UK Library of Congress Control Number: 2005927413 ISBN-10: 0-387-26214-8 ISBN-13: 978-0387-26214-7 Printed on acid-free paper. © 2006, 2001 Springer Science+Business Media, Inc. All rights reserved. This work may not be translated or copied in whole or in part without the written permission of the publisher (Springer Science+Business Media, Inc., 233 Spring Street, New York, NY 10013, USA), except for brief excerpts in connection with reviews or scholarly analysis. Use in connection with any form of information storage and retrieval, electronic adaptation, computer software, or by similar or dis- similar methodology now known or hereafter developed is forbidden. The use in this publication of trade names, trademarks, service marks, and similar terms, even if they are not identified as such, is not to be taken as an expression of opinion as to whether or not they are subject to propri- etary rights. While the advice and information in this book are believed to be true and accurate at the date of going to press, neither the authors nor the editors nor the publisher can accept any legal responsibility for any errors or omis- sions that may be made.
    [Show full text]
  • 001 2009 4 E.Pdf
    # 2003 University of South Africa All rights reserved Printed and published by the University of South Africa Muckleneuk, Pretoria ETH306-W/1/2004±2006 97441767 Karinnew Style CONTENTS Study unit Page FOREWORD (viii) SECTION A LEARNERS WHO EXPERIENCE BARRIERS TO LEARNING STUDY UNIT 1 WHO ARE THESE LEARNERS WHO EXPERIENCE BARRIERS TO LEARNING? 2 1.1 The terms ``learners with special educational needs'' and ``learners who experience barriers to learning'' 3 1.2 Manifestations of barriers to learning 5 1.3 The interrelationship between barriers to learning 11 1.4 Phases in which barriers to learning and development come to the fore 11 1.5 Classification of learners who experience barriers to learning 12 1.6 Degrees of barriers to learning 15 1.7 Extent of barriers to learning 15 1.8 Consequences of barriers to learning 16 1.9 Summary 16 Bibliography 17 STUDY UNIT 2 CAUSES OF BARRIERS TO LEARNING 18 2.1 Why a knowledge of what causes barriers to learning is important 18 2.2 Disability 20 2.3 Extrinsic causes of barriers to learning 23 2.4 Summary 33 Bibliography 34 STUDY UNIT 3 PARENTS AND FAMILIES OF LEARNERS WHO EXPERIENCE BARRIERS TO LEARNING 36 3.1 Introduction 36 3.2 Some factors that may determine parental attitudes towards a learners with a physical and/or physiological impairment 38 ETH306-W/1/2004±2006 (iii) Study unit Page 3.3 Different patterns of parental attitudes 41 3.4 Life-cycle events and parental attitudes 45 3.5 The effect of the birth of a child with a physical and/or physiological impairment on the different members of
    [Show full text]
  • Assessment of Neurologic Function
    Chapter 60 ● Assessment of Neurologic Function LEARNING OBJECTIVES ● On completion of this chapter, the learner will be able to: 1. Describe the structures and functions of the central and peripheral nervous systems. 2. Differentiate between pathologic changes that affect motor control and those that affect sensory pathways. 3. Compare the functioning of the sympathetic and parasympathetic nervous systems. 4. Describe the significance of physical assessment to the diagnosis of neurologic dysfunction. 5. Describe changes in neurologic function associated with aging and their impact on neurologic assessment findings. 6. Describe diagnostic tests used for assessment of suspected neuro- logic disorders and the related nursing implications. 1820 Chapter 60 Assessment of Neurologic Function 1821 Nurses in many types of practice settings encounter patients impulses away from the cell body. Nerve cell bodies occurring in with altered neurologic function. Disorders of the nervous system clusters are called ganglia or nuclei. A cluster of cell bodies with can occur at any time during the life span and can vary from mild, the same function is called a center (eg, the respiratory center). self-limiting symptoms to devastating, life-threatening disorders. Neuroglial cells, another type of nerve cell, support, protect, and The nurse must be skilled in the assessment of the neurologic sys- nourish neurons. tem whether the assessment is generalized or focused on specific areas of function. Assessment in either case requires knowledge Neurotransmitters of the anatomy and physiology of the nervous system and an understanding of the array of tests and procedures used to diag- Neurotransmitters communicate messages from one neuron to nose neurologic disorders.
    [Show full text]
  • Management of Chronic Conditions in the Foot and Lower
    Management of Chronic Conditions in the Foot and Lower Leg Content Strategist: Rita Demetriou-Swanwick Content Development Specialists: Catherine Jackson/Nicola Lally Project Manager: Umarani Natarajan Designer/Design Direction: Christian Bilbow Illustration Manager: Jennifer Rose Illustrator: Antbits Management of Chronic Conditions in the Foot and Lower Leg Edited by Keith Rome BSc(Hons), MSc, PhD, FCPodMed, SRCh Professor in Podiatry and Co-Director, Health and Research Rehabilitation Institute, Department of Podiatry, School of Rehabilitation and Occupation Studies, Faculty of Health and Environmental Sciences, Auckland University of Technology, Auckland, New Zealand Peter McNair DipPhysEd, DipPT, MPhEd(Distn), PhD Professor of Physiotherapy and Director, Health and Rehabilitation Research Institute, Auckland University of Technology, Auckland, New Zealand Foreword by Christopher Nester BSc(Hons), PhD Professor, Research Lead: Foot and Ankle Research Programme, School of Health Sciences, University of Salford, Salford, UK Edinburgh London New York Oxford Philadelphia St Louis Sydney Toronto 2015 © 2015 Elsevier Ltd All rights reserved. No part of this publication may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, recording, or any information storage and retrieval system, without permission in writing from the publisher. Details on how to seek permission, further information about the Publisher’s permissions policies and our arrangements with organizations such as the Copyright
    [Show full text]
  • A Dictionary of Neurological Signs.Pdf
    A DICTIONARY OF NEUROLOGICAL SIGNS THIRD EDITION A DICTIONARY OF NEUROLOGICAL SIGNS THIRD EDITION A.J. LARNER MA, MD, MRCP (UK), DHMSA Consultant Neurologist Walton Centre for Neurology and Neurosurgery, Liverpool Honorary Lecturer in Neuroscience, University of Liverpool Society of Apothecaries’ Honorary Lecturer in the History of Medicine, University of Liverpool Liverpool, U.K. 123 Andrew J. Larner MA MD MRCP (UK) DHMSA Walton Centre for Neurology & Neurosurgery Lower Lane L9 7LJ Liverpool, UK ISBN 978-1-4419-7094-7 e-ISBN 978-1-4419-7095-4 DOI 10.1007/978-1-4419-7095-4 Springer New York Dordrecht Heidelberg London Library of Congress Control Number: 2010937226 © Springer Science+Business Media, LLC 2001, 2006, 2011 All rights reserved. This work may not be translated or copied in whole or in part without the written permission of the publisher (Springer Science+Business Media, LLC, 233 Spring Street, New York, NY 10013, USA), except for brief excerpts in connection with reviews or scholarly analysis. Use in connection with any form of information storage and retrieval, electronic adaptation, computer software, or by similar or dissimilar methodology now known or hereafter developed is forbidden. The use in this publication of trade names, trademarks, service marks, and similar terms, even if they are not identified as such, is not to be taken as an expression of opinion as to whether or not they are subject to proprietary rights. While the advice and information in this book are believed to be true and accurate at the date of going to press, neither the authors nor the editors nor the publisher can accept any legal responsibility for any errors or omissions that may be made.
    [Show full text]
  • Comparison Between Forward and Backward Gait Retraining for Mobility in Individuals with Mild to Moderate Parkinson’S Disease
    COMPARISON BETWEEN FORWARD AND BACKWARD GAIT RETRAINING FOR MOBILITY IN INDIVIDUALS WITH MILD TO MODERATE PARKINSON’S DISEASE by Roné Grobbelaar Article-format MSc Thesis presented in partial fulfilment of the requirements for the degree of Master of Science in the Faculty of Education at Stellenbosch University Supervisor: Dr Karen Welman Co-supervisor: Prof Ranel Venter March 2017 Stellenbosch University https://scholar.sun.ac.za Declaration By submitting this thesis electronically, I declare that the entirety of the work contained therein is my own, original work, that I am the sole author thereof (save to the extent explicitly otherwise stated), that reproduction and publication thereof by Stellenbosch University will not infringe any third party rights and that I have not previously in its entirety or in part submitted it for obtaining any qualification. March 2017 Copyright © 2017 Stellenbosch University All rights reserved i Stellenbosch University https://scholar.sun.ac.za ABSTRACT Background Dysfunctional gait and transitional movements are the most disabling features of Parkinson‟s disease (PD) and often relates to falls. Due to executive dysfunction in PD, dual tasking (DT) is detrimental to already impaired mobility parameters. Backwards walking (BW) might be a useful training alternative to improve aberrant PD gait and transitional movements to consequently improve the quality of complex, multi-directional daily activities, which most often involve DT. Over ground BW gait retraining has shown to be beneficial for neurological gait rehabilitation; however, has not yet been investigated in PD. Training in complex, novel tasks may induce enhanced cortical activity for movement preparation that is beyond training in automatic tasks.
    [Show full text]
  • 腦血管疾病學 the Tingling Sensation and Limbs Weakness Recurred and She Was Referred 1 to Our Hospital for Further Survey
    Poster Session performed one hour later was negative. 壁報論文 Her symptoms recovered gradually after 8 hours of staying in hospital. Nevertheless, five hours after discharge, 腦血管疾病學 the tingling sensation and limbs weakness recurred and she was referred 1 to our hospital for further survey. 個案研究:以急性全身刺痛與無力表現 Neurological examination revealed the 的雙內側延髓中風 muscle power was 3/5 in all four limbs 朱海瑞 湯頌君 鄭建興 with bilateral extensor type of plantar 國立臺灣大學醫學院附設醫院 神經部 response. The gag reflex was trance Bilateral medial medullary infarction and tongue could not be protruded. Ten presenting as acute fluctuating tingling hours after arriving in our emergency sensation and weakness over all limbs room, the muscle strength deteriorated and trunk: a case report Hai-Jui Chu, Sung-Chun Tang, Jiann-Shing Jeng to 2/5 in upper limbs and 1/5 in lower Department of Neurology, National Taiwan University Hospital ones. Magnetic resonance imaging revealed bilateral MMI infarction with Background Bilateral medial heart shape sign. Digital subtraction medullary infarction (MMI) is extremely angiography showed normal rare among patients with acute ischemic appearance of bilateral vertebral stroke. Here we reported a case of arteries but suspected an azygos bilateral MMI presenting as an unusual anterior spinal artery mainly derived course of acute fluctuating tingling from the left vertebral artery. For the sensation and weakness in all limbs and consequently respiratory distress, she trunk for 2 days. was intubated. Heparinization was given for 2 days and was shifted to Case Report A 41-year-old female had Clopidogrel 1 tablet per day. Three newly diagnosed hypertension but did not months later, her muscle strength take regular medication for several improved significantly and she could months.
    [Show full text]
  • NEUROLOGY in Lectures (Selected Lectures)
    MINISTRY OF UKRAINE PUBLIC HEALTH I.Y. HORBACHEVSKYY TERNOPIL STATE MEDICAL UNIVERSITY Department of Neurology, Psychiatry, Science of Narcotics and Medical Psychology NEUROLOGY in lectures (Selected lectures) Ternopil TSMU Ukrmedknyha 2006 1 The redaction of Head of Department of Neurology, Psychia- try, Science of Narcotics and Medical Psychology Honoured Science and Technic Worker, d.m.s. prof. Shkrobot S.I. The authors of lectures: d.m.s. prof. Shkrobot S.I. , k.m.s. assoc. prof. Hara I.I. Literature redaction: Milevska L.S. It was approved by CMC (the report ¹ 4 28.05.2003) Reviewers: The Head of Department of Neurology, Lviv National Medical University d.m.s. prof. Pshyk S.S. The Head of Department of Neurology, Psychiatry and Medical Psychology Bukovinskyy State Medical University d.m.n. prof. Pashkovskyy V.M. Responsibility for this edition is on the Rector of I.Y. Horbachevskyy Ternopil State Medical University the Member of Ukrainian Academy of Medical Science prof. Kovalchuk L.Y. 2 CONTENS Introduction ..................................................................................... 4 Functions of nervous system. Conditioned and unconditioned reflexes. Motor system (symptomatic and topical diagnostics of movement disturbances) ................................................................................... 5 Sensation. Signs of sensation disturbances ...................................... 25 Extrapyramidal system and Cerebellum. The main anatomic and physiological data. Physiological functions and pathologic syndromes
    [Show full text]
  • A Textbook of Chiropractic Symptomatology
    A TEXT-BOOK ON Chiropractic Symptomatology OR THE Manifestations of Incoordmation Considered From a Chiropractic Standpoint JAMES N. FIRTH, D. C.. Ph. C. Professor of Symptomatology in the Palmer School of Chiropractic, (Chiropractic'i Fountain Head) Davenport, Iowa COPYRIGHT. 1914, BY JAMES N. FIRTH. D. C., Ph. C. 1914 DRIFFILL PRINTING CO. ROCK ISLAND. ILL PREFACE Upon accepting the chair of Symptomatology in Tin Palmer School of Chiropractic, Davenport, Iowa, I found it difficult for students and others seeking Chiropractic in- formation on diseased conditions to obtain a comprehensive idea of such when they sought their information in medical text-books. That is, they were able to find an accurate de- scription of the pathology and its accompanying symptoms, but were unable to apply the Chiropractic idea or philosophy to such conditions. The aim of this volume, as indicated by its title, is that the student, practitioner and layman may obtain a concise, yet comprehensive, explanation of incoordinations from a Chiropractic standpoint. This volume is exclusively de- voted to the subject of Symptomatology and should not be considered as an authority on philosophy. Many of the theories advanced here have been derived or suggested from Dr. Palmer's works on philosophy, which are the only works published on that subject. It is very essential that the introductory chapter and the chapter on preliminary considerations be read and studied first, as therein are contained the fundamental principles upon which subsequent deductions are based. It is hoped that this volume may be useful to the student, practitioner and layman. JAMES N. FIRTH, D.
    [Show full text]
  • Pediatric Orthopedics in Practice, DOI 10.1007/978-3-662-46810-4, © Springer-Verlag Berlin Heidelberg 2015 880 Backmatter
    879 Backmatter Subject Index – 880 F. Hefti, Pediatric Orthopedics in Practice, DOI 10.1007/978-3-662-46810-4, © Springer-Verlag Berlin Heidelberg 2015 880 Backmatter Subject index Bold letters: Principal article Italics: Illustrations A Acetylsalicylic acid 303, 335 Adolescent scoliosis Amyloidosis 663 Acheiropodia 804 7 Scoliosis Amyoplasia 813–814 Abducent nerve paresis 752, Achievement by proxy 10, 11 AFO 7 Ankle Foot Orthosis Anaerobes 649, 652, 657 816 Achilles tendon Aggrecan 336, 367, 762 ANA 7 antinuclear antibodies Abducted pes planovalgus – lengthening 371, 426, 431, aggressive osteomyelitis Analysis, gait 488, 490–497 433, 434, 436, 439, 443, 464, 7 osteomyelitis, aggressive 7 Gait analysis Abduction contracture 468, 475, 485, 487–490, 493, Agonist 281, 487, 492, 493, Anchor 169, 312, 550, 734 7 contracture 496, 816, 838, 840 495, 498, 664, 832, 835, 840, Andersen classification abduction pants 219–221 – shortening 358, 418, 431, 868 7 classification, Andersen Abduction splint 212, 218–221, 433, 464, 465, 467, 468, 475, Ahn classification 366 Andry, Nicolas 21, 22 248, 850 489, 496, 838 Aitken classification (congenital Anesthesia 26, 38, 135, 154, Abduction Achondrogenesis 750, 751, femoral deficiency ) 7 classi- 162, 174, 221, 243, 247, 248, – hip 195, 198, 199, 212, 213, 756, 758–760, 769 fication, femoral deficiency 255, 281, 303, 385, 386, 400, 214, 218, 219, 220, 221, Achondroplasia 56, 163, 166, Akin osteotomy 477, 479 500, 506, 559, 568, 582–585, 241–245, 247, 248, 251, 255, 242, 270, 271, 353, 409, 628, Albers-Schönberg
    [Show full text]
  • Igait: Vision-Based Low-Cost, Reliable Machine Learning Framework for Gait
    iGait: Vision-based Low-Cost, Reliable Machine Learning Framework for Gait Abnormality Detection by SAIF IFTEKAR SAYED Presented to the Faculty of the Graduate School of The University of Texas at Arlington in Partial Fulfillment of the Requirements for the Degree of MASTER OF SCIENCE THE UNIVERSITY OF TEXAS AT ARLINGTON December 2017 Copyright c by Saif Iftekar Sayed 2017 All Rights Reserved TABLE OF CONTENTS LIST OF ILLUSTRATIONS . iv LIST OF TABLES . vi Chapter Page 1. Abstract . .1 2. Literature Review . .2 2.1 Impact of health on gait . .2 2.2 Data Acquisition Types . .3 2.3 Chronic Low back pain . .8 3. Methodology . 10 3.1 The iGait System . 10 3.1.1 Body Features Extraction . 15 3.1.2 Fall Prediction . 22 3.1.3 The iGait System Protocol . 24 4. Experimental Results . 28 4.1 Experimental Results . 28 4.1.1 Classification metrics . 28 4.1.2 Feature Significance . 31 4.1.3 ROC Curves . 35 5. Conclusion and Future Work . 39 5.1 Conclusion and Future Work . 39 REFERENCES . 40 iii LIST OF ILLUSTRATIONS Figure Page 2.1 setup of 3 force resistive resistors(left) and a gyroscope(right) . .4 2.2 setup of a BSN sensor behind the ear and a RGB-D Camera . .6 2.3 Computation of features at µ = 0 with non-overlapping(a), overlapping(b) and at a viewpoint at µ = 20(c) . .7 2.4 Individual ellipse model attached to the respective segments of the body . .8 3.1 Block Diagram of iGait System . 11 3.2 Kinect V1 Sensor .
    [Show full text]