Practice Standards and Guidelines for Acquired Cognitive Communication Disorders
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Assessing the Severity of Traumatic Brain Injury—Time for a Change?
Journal of Clinical Medicine Review Assessing the Severity of Traumatic Brain Injury—Time for a Change? Olli Tenovuo 1,2,*, Ramon Diaz-Arrastia 3, Lee E. Goldstein 4, David J. Sharp 5,6, Joukje van der Naalt 7 and Nathan D. Zasler 8,9 1 Division of Clinical Neurosciences, Turku Brain Injury Centre, Turku University Hospital, 20521 Turku, Finland 2 Department of Neurology, Institute of Clinical Medicine, University of Turku, 20500 Turku, Finland 3 Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA 19104, USA; [email protected] 4 Alzheimer’s Disease Research Center, College of Engineering, Boston University School of Medicine, Boston, MA 02118, USA; [email protected] 5 Clinical, cognitive and computational neuroimaging laboratory (C3NL), Department of Brain Sciences, Faculty of Medicine, Imperial College London, London, W12 0NN, UK; [email protected] 6 UK Dementia Research Institute Care Research and Technology Centre, Imperial College London and the University of Surrey, London, W12 0NN UK 7 Department of Neurology, University of Groningen, University Medical Center Groningen, 9713 GZ Groning-en, The Netherlands; [email protected] 8 Concussion Care Centre of Virginia and Tree of Life, Richmond, VA 23233, USA; [email protected] 9 Department of Physical Medicine and Rehabilitation, Virginia Commonwealth University, Richmond, VA 23284, USA * Correspondence: olli.tenovuo@tyks.fi; Tel.: +358-50-438-3802 Abstract: Traumatic brain injury (TBI) has been described to be man’s most complex disease, in man’s most complex organ. Despite this vast complexity, variability, and individuality, we still classify the severity of TBI based on non-specific, often unreliable, and pathophysiologically poorly understood measures. -
Common Disorders of Speech in Children by T
Arch Dis Child: first published as 10.1136/adc.34.177.444 on 1 October 1959. Downloaded from A DESCRIPTION AND CLASSIFICATION OF THE COMMON DISORDERS OF SPEECH IN CHILDREN BY T. T. S. INGRAM From the Department of Child Life and Health, University of Edinburgh and the Royal Hospitalfor Sick Children, Edinburgh (RECEIVED FOR PUBLICATION MARCH 9, 1959) The full assessment of children suffering from speech defects, speech therapy is rarely provided. speech defects requires a team consisting of speech Children with speech defects attending these schools therapist, paediatrician, psychologist and otologist. were frequently referred to the Speech Clinic in the The additional services of a phonetician, neurologist, Hospital. orthodontist, plastic surgeon, radiologist, social Detailed family, birth and developmental histories worker or psychiatric social worker and child were taken, with particular emphasis on the rate, and psychiatrist are often helpful. any abnormalities, of speech development. The Unfortunately the majority of descriptions and children were subjected to a detailed paediatric and classifications of speech disorders in childhood are neurological examination, and behaviour in play by speech therapists working without much medical was observed for as long as possible in each case. co-operation and advice. With honourable excep- The child's speech was studied jointly by the by copyright. tions, they tend to regard speech disorders as rather paediatrician and the speech therapist, and detailed isolated phenomena, dissociated from the other notes were made of its intelligibility and of the behavioural and psychological characteristics of the particular defects which were observed. In many child. In the present paper an attempt is made to cases tape recordings were made, though it is always describe and classify the disorders of speech most easier, in fact, to examine speech for defects of frequently encountered in a hospital speech clinic articulation in the presence of the patient. -
Chronic Anxiety and Stammering Ashley Craig & Yvonne Tran
Advances in PsychiatricChronic Treatment anxiety (2006), and vol. stammering 12, 63–68 Fear of speaking: chronic anxiety and stammering Ashley Craig & Yvonne Tran Abstract Stammering results in involuntary disruption of a person’s capacity to speak. It begins at an early age and can persist for life for at least 20% of those stammering at 2 years old. Although the aetiological role of anxiety in stammering has not been determined, evidence is emerging that suggests people who stammer are more chronically and socially anxious than those who do not. This is not surprising, given that the symptoms of stammering can be socially embarrassing and personally frustrating, and have the potential to impede vocational and social growth. Implications for DSM–IV diagnostic criteria for stammering and current treatments of stammering are discussed. We hope that this article will encourage a better understanding of the consequences of living with a speech or fluency disorder as well as motivate the development of treatment protocols that directly target the social fears associated with stammering. Stammering (also called stuttering) is a fluency childhood, many recover naturally in early disorder that results in involuntary disruptions of adulthood (Bloodstein, 1995). We found a higher a person’s verbal utterances when, for example, prevalence rate (of up to 1.4%) in children and they are speaking or reading aloud (American adolescents (2–19 years of age), with males in this Psychiatric Association, 1994). The primary symptoms of the disorder are shown in Box 1. If the symptoms are untreated in early childhood, Box 1 Symptoms of stammering there is a risk that the concomitant behaviours will become more pronounced (Bloodstein, 1995; Craig Behavioural symptoms • et al, 2003a). -
Processing of Degraded Speech in Brain Disorders
brain sciences Review Processing of Degraded Speech in Brain Disorders Jessica Jiang 1, Elia Benhamou 1, Sheena Waters 2 , Jeremy C. S. Johnson 1, Anna Volkmer 3, Rimona S. Weil 1 , Charles R. Marshall 1,2, Jason D. Warren 1,† and Chris J. D. Hardy 1,*,† 1 Dementia Research Centre, Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, London WC1N 3BG, UK; [email protected] (J.J.); [email protected] (E.B.); [email protected] (J.C.S.J.); [email protected] (R.S.W.); [email protected] (C.R.M.); [email protected] (J.D.W.) 2 Preventive Neurology Unit, Wolfson Institute of Preventive Medicine, Queen Mary University of London, London EC1M 6BQ, UK; [email protected] 3 Division of Psychology and Language Sciences, University College London, London WC1H 0AP, UK; [email protected] * Correspondence: [email protected]; Tel.: +44-203-448-3676 † These authors contributed equally to this work. Abstract: The speech we hear every day is typically “degraded” by competing sounds and the idiosyncratic vocal characteristics of individual speakers. While the comprehension of “degraded” speech is normally automatic, it depends on dynamic and adaptive processing across distributed neural networks. This presents the brain with an immense computational challenge, making de- graded speech processing vulnerable to a range of brain disorders. Therefore, it is likely to be a sensitive marker of neural circuit dysfunction and an index of retained neural plasticity. Consid- ering experimental methods for studying degraded speech and factors that affect its processing in healthy individuals, we review the evidence for altered degraded speech processing in major Citation: Jiang, J.; Benhamou, E.; neurodegenerative diseases, traumatic brain injury and stroke. -
Social Anxiety Disorder and Stuttering: Current Status And
G Model JFD-5535; No. of Pages 14 ARTICLE IN PRESS Journal of Fluency Disorders xxx (2013) xxx–xxx Contents lists available at ScienceDirect Journal of Fluency Disorders Social anxiety disorder and stuttering: Current status and future directions ∗ Lisa Iverach , Ronald M. Rapee Centre for Emotional Health, Department of Psychology, Macquarie University, Australia a r t i c l e i n f o a b s t r a c t Article history: Anxiety is one of the most widely observed and extensively studied psychological concomi- Received 17 March 2013 tants of stuttering. Research conducted prior to the turn of the century produced evidence Received in revised form 11 August 2013 of heightened anxiety in people who stutter, yet findings were inconsistent and ambiguous. Accepted 20 August 2013 Failure to detect a clear and systematic relationship between anxiety and stuttering was Available online xxx attributed to methodological flaws, including use of small sample sizes and unidimensional measures of anxiety. More recent research, however, has generated far less equivocal find- Keywords: ings when using social anxiety questionnaires and psychiatric diagnostic assessments in Stuttering larger samples of people who stutter. In particular, a growing body of research has demon- Anxiety strated an alarmingly high rate of social anxiety disorder among adults who stutter. Social Social anxiety disorder anxiety disorder is a prevalent and chronic anxiety disorder characterised by significant fear Social phobia of humiliation, embarrassment, and negative evaluation in social or performance-based Cognitive Behaviour Therapy situations. In light of the debilitating nature of social anxiety disorder, and the impact of stuttering on quality of life and personal functioning, collaboration between speech pathol- ogists and psychologists is required to develop and implement comprehensive assessment and treatment programmes for social anxiety among people who stutter. -
II./2.9. Language and Higher Cerebral Functions II./2.9.1. Disorders Of
II./2.9. Language and higher cerebral functions II./2.9.1. Disorders of language Disorders of language include disturbance of speech, writing and reading. II./2.9.1.1. Disorders of speech (aphasias) Aphasia is a speech disorder caused by lesion of cortical speech centers and their connections, characterized by the disturbance in the use and production of verbal symbols. Dysarthria is a disturbance of phonation (voice production), articulation, pronunciation and prosody (intonation, rhythm). Dysarthria may be caused by the lesion of cortical centers of innervating the muscles of speech, bilateral lesion of corticobulbar fibers, bulbar motoneuron disorders, myasthenia gravis, myopathies, and cerebellar What is the difference disorders. between aphasia and dysarthria? Anatomy Brain regions related to speech are in most people located in the dominant (left) hemisphere: the Wernicke’s speech center (Br22 and a small part of Br39 and Br40), responsible for the comprehension of speech, lies adjacent to the primary auditory cortex in the posterior two-thirds of the superior temporal gyrus. Its connection with the angular gyrus ensures the coupling of perceptual and learned memories with speech. The angular and the supramarginal gyri are associative areas at the border of the auditory, sensory and visual cortices. The Broca’s motor speech center is in the middle and inferior frontal gyrus, and the lower part of the precentral gyrus (Br44). The motor speech area is connected by the arcuate bundle with the angular and the supramarginal gyri, and the Wernicke area. The occipitofrontal bundle connects the angular and the supramarginal gyri with the frontal opercular areas. -
Diagnosing and Managing Concussion Tool
Sections: A B C D References Diagnosing and Managing Concussion Tool This tool has been developed to support family physicians and nurse practitioners in consistently diagnosing and managing concussion in adult patients (≥ 18 years). Section A: Concussion overview Section C: Patient encounter forms pg.1 Clinical definition pg.5 Form: Diagnostic assessment pg.1 Common misconceptions about concussion pg.7 Form: Return to activities Section B: General treatment approach pg.9 Form: Symptom monitoring log (patient resource) pg.2 Assessment and diagnosis Section D: Additional resources pg.3 Management and recovery planning pg.10 Patient resources pg.4 Monitoring and follow-up pg.11 Provider resources Appendices Appendix A: Pharmacotherapy guidance Appendix B: Clinical symptom management guidance Section A: Concussion overview A concussion is an acute neurophysiological event related to blunt impact or mechanical injury applied to the head, neck, or body, with transmitting forces to the brain.1,2,3,4,5 • Although complex, concussion is manageable in a primary care setting. • There is no perfect diagnostic test or marker for concussion.3 • Although the terms concussion and mild traumatic brain injury (mTBI) are often used interchangeably, they are different conditions along a continuum.3 Evidence of intracranial injury or a persistent neurologic deficit are indicative of a mTBI.1,4 Clarifying common misconceptions about concussions • A concussion does not require a direct blow to the head. Concussion can be caused by impulsive forces acting elsewhere on the body transmitting to the head – such as sudden acceleration, rotation, deceleration (whiplash) or by multiple sub-concussive hits.1,3,4 • A concussion may or may not involve a loss of consciousness. -
An Overview on Primary Progressive Aphasia and Its Variants
1 TITLE An Overview on Primary Progressive Aphasia and its variants Serena Amici a,d *M.D., Maria Luisa Gorno -Tempini a, M.D., PhD, Jennifer M. Ogar a,b , M.S., Nina F. Dronkers a,b,c , PhD, Bruce L. Miller a, M.D. aMemory and Aging Center, Department of Ne urology , University of California, San Francisco, USA bVA Northern California Health Care System, Martinez, CA, USA cUniversity of California, Davis, Davis, CA, USA dDepartment of Neurosciences, University of Perugia, Perugia, ITALY *Corresponding author: Serena Amici 350 Parnassus Avenue, San Francisco, CA 94143 -1207 (415) 476 3572 Keywords : primary progressive aphasia, nonfluent progressive aphasia, semantic dementia, logopenic progressive aphasia, neuroimaging studies, language symptoms Words: 415 6 Tables : 2 Figures: 1 Reference s: 64 2 Abstract We present a review of the literature on Primary Progressive Aphasia (PPA) together with the analysis of neuropschychological and neuroradiologic profile s of 42 PPA patients. Mesulam originally defined PPA as a progressive degenerative disorder characterized by isolated language impairment for at least two years. The most common variants of PPA are: 1) Progressive nonfluent aphasia (PNFA), 2) semantic dementia (SD), 3) logopenic progressive aphasia (LPA). PNFA is characterized by labored speech, agrammatism in production, and/or comprehension . In some cases the syndrome begins with isolated deficits in speech. SD patients typically present with loss of word and object meaning and surface dyslexia. LPA patients have word -finding difficulties, syntactically simple but accurate language output and impaired sentence comprehension. The neuropsychological data demonstrated that SD patients show the most characteristic pattern of impairment, while PNFA and LPA overlap with in many cognitive domains. -
Childhood-Onset Fluency Disorder (Stuttering)
0 Running head: STUTTERING Childhood-Onset Fluency Disorder (Stuttering) An Informational Handout Anna Birstein 4.20.2015 Stuttering is a Speech Disorder Definition Childhood-Onset Fluency Disorder (stuttering) reflects a marked impairment in speech fluency that is not attributable to stroke or another medical condition, and developmental or mental disorder (Weis, 2013). The Diagnostic and Statistical Manual of Mental Disorders (Fifth Edition), edited by American Psychiatric Association, defines Childhood-Onset Fluency Disorder (Stuttering) as a condition characterized by disturbances in the normal fluency and time patterning of speech that are inappropriate for the individual’s age and language skills, and persist over time. The onset of symptoms is usually in the early developmental period (APA, 2013, p. 45). Diagnosis Criteria According to the DSM-V, the disturbances in the normal speech fluency are characterized by one or more of the following: Sound and syllable repetitions (part-word repetitions) (ba – baby) Sound prolongations (S>>>>sometimes) Broken words (pauses within a word) (Ta – table) Audible or silent blocking (filled or unfilled pauses in speech) (I like to – go home) Circumlocutions (word substitutions to avoid a problematic word) Words pronounced with an excess of physical tension Monosyllabic whole-word repetitions (“I-I-I see him”) The other criterion is that “the disturbance [in speech fluency] causes anxiety about speaking or limitations in effective communication, social participation, and academic or occupational performance. .” (DSM V, 2013) According to Weis (2013), many children who stutter show co-occurring behaviors when they exhibit their disfluencies. These behaviors usually involve repetitive movement of the head, face, and neck (p. -
Apraxia of Speech and Nonverbal School-Aged Children with Autism I
Topics Apraxia of Speech and Nonverbal School-Aged Children with Autism I. Introduction A. Apraxia of Speech Lawrence D. Shriberg B. The CAS/ASD Hypothesis II. Overviews Waisman Center A. Speech Sound Disorders (SSD) University of Wisconsin-Madison B. Childhood Apraxia of Speech (CAS) III. Speech Research in ASD Nonverbal School-aged Children with Autism A. Verbal ASD: Findings NIH Workshop, Bethesda, MD, April 13-14, 2010 B. Nonverbal ASD: Perspectives Locus of Apraxia of Speech in a Four-Phase Speech Processing Frameworka Apraxia of Speech in an Adolescent with Classic Galactosemia Encoding (auditory-perceptual) Memorial (storage, retrieval) Transcoding (planning-programming) Execution (neuromotor) aAfter Van der Merwe (2009) Multisyllabic Words Task 2 (MWT2) What is Apraxia of Speech? 1. emphasis 11. consciousness 2. probably 12. suspicious Say the following as quickly as you can: 3. sympathize 13. municipal 4. terminal 14. orchestra 5. synthesis 15. specific Six thick thistle sticks 6. especially 16. statistics Six thick thistle sticks 7. peculiar 17. fire extinguisher Six thick thistle sticks 8. skeptical 18. Episcopal church 9. fudgesicle 19. statistician 10. vulnerable 20. Nicaragua 1 Topics The CAS/ASD Hypothesisa I. Introduction CAS is a sufficient cause for: A. Apraxia of Speech B. The CAS/ASD Hypothesis Weak version: speech and prosody- II. Overviews voice deficits in persons with verbal ASD A. Speech Sound Disorders (SSD) B. Childhood Apraxia of Speech (CAS) Strong version: failure to acquire speech in III. Speech Research in ASD persons with nonverbal ASD A. Verbal ASD: Findings aChildhood Apraxia of Speech (CAS) is the term adopted in the ASHA (2007) report; Developmental Verbal Dyspraxia (DVD) continues to be B. -
Brain Injury in Children and Youth: a Manual
Brain Injury in Children and Youth A Manual for Educators Revised 2018 ACKNOWLEDGMENTS In 2001, the Traumatic Brain injury (TBI) Manual was written as a joint effort between the Colorado Depart- ment of Education, the New Start Project within the Center for Community Participation at Colorado State University in Fort Collins, Colorado, and the Children’s Hospital Colorado. Since 2001, brain injury research and practice has changed significantly. In 2012, the Colorado Brain Injury Steering Committee, led by Karen McAvoy and Judy Dettmer, was instrumental in updating the TBI Manual and creating the stand alone spe- cial education eligibility category and criteria for TBI, which was formally adopted into the Colorado Rules for the Exceptional Children’s Educational Act (ECEA). The newest version of this manual was revised in 2018 by: Judy Dettmer, B.S.W., Director, MINDSOURCE Brain Injury Network, Colorado Department of Human Services Jeanne E. Dise-Lewis (†), Ph.D., Psychologist, Rehabilitative Medicine, Children’s Hospital Colorado, Died September 18, 2014 † Deceased. Patricia W. Colella, M.A., C.A.G.S., School Psychologist Nicole Crawford, Ph.D., Psychologist, Brain Injury Specialist, Colorado Department of Education Heather Hotchkiss, M.S.W., Principal Brain Injury Specialist, Exceptional Student Services Unit, Colorado Department of Education Karen McAvoy, Psy.D., Psychologist, Brain Injury Specialist, Colorado Department of Education Peter Thompson, Ph.D., School Psychologist, Douglas County School District Janet Tyler, Ph.D., Senior -
Coping with Memory Problems After Brain Injury Practical Strategies
Coping with Memory Problems after Brain Injury Practical Strategies Introduction Memory problems are one of the most common effects of acquired brain injury. Sadly there are no cures available, but there are a number of ways of coping and making life easier. This factsheet is designed to provide basic, practical suggestions on coping with memory problems and making the most of memory. The first main section outlines ways of adapting the environment and lifestyle in order to minimise the impact of memory problems. The second main section deals with techniques and strategies which can help people to make the most of their existing memory and to store and recall information more efficiently. Adapting and Coping with Memory Problems There are a number of ways to make life easier. These include: Adapting the environment Using external memory aids Following a set routine Combining several strategies to make a substitute ‘memory system’ Improving general well-being The rest of this section will provide suggestions for each of these categories. Adapting the environment One of the simplest ways to help people with memory problems is to adapt their environment so they rely on memory less. Some ideas for doing so which have helped others are: Keeping a notepad by the phone to make a note of phone calls and messages Putting essential information on a noticeboard Deciding on a special place to keep important objects like keys, wallets or spectacles and always putting them back in the same place Attaching important items to your person so they can’t be mislaid, for example using a neck cord for reading glasses Labelling cupboards and storage vessels as a reminder of where things are kept Labelling perishable food with the date it was opened Painting the toilet door a distinctive colour so it is easier to find Labelling doors as a reminder of which room is which Using external memory aids Many people use external memory aids, regardless of whether they have a brain injury or not.