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Product List Product Name Product Name Albendazole Clomiphene Citrate Allopurinol Clomipramine Amiloride Closantel Base / Sodium Amisulpride Croscarmellose Sodium Aripiprazole Clotrimazole Avanafil Dapoxetine Hcl Beclomethasone Dipropionate Deflazacort Benfotiamine Dexamethasone Sodium Phosphate Benzocaine Dexchlorpheniramine Maleate Betahistine Di Hcl Dicyclomine Hcl Betamethasone Dipropionate Diethylcarbamazine Citrate Betamethasone Sodium Phosphate Dimenhydrinate Betamethasone Valerate Diphenhydramine Hcl Brexpiprazole Divalproex Sodium Calcium Chloride Duloxetine Hcl Carbamazepine Econazole Nitrate / Base Carbidopa Escitalopram Oxalate Carvedilol Fenbendazole BP Vet Cetrimide Fexofenadine Hcl Cetirizine Hcl Fluocinolone Acetonide Chlorhexidine Gluconate Fluorometholone Chlorpheniramine Maleate Fluticasone Propionate Chlorpromazine Glimepiride (Micronized) Ciclopirox Glipizide Ciclopirox Olamine Halobetasol Propionate Citalopram HBR Hydrocortisone Acetate / Butyrate Clobetasol Propionate / Butyrate Hydroquinone USP Product List Product Name Product Name Imatinib Mesylate Piroctone Olamine Indapamide Piroxicam Isosorbide Dinitrate / Mononitrate Potassium Chloride Ketorolac Tromethamine Prednisolone Acetate Levosulpiride Prednisolone Sodium Phosphate Lidocaine Base / Hcl Prilocaine Base / Hcl Lithium Carbonate IP/BP/USP Prochlorperazine Magnesium Chloride Quetiapine Fumarate Magnesium Valproate Rafoxanide BP VET Malt Extract Ricobendazole Mebendazole Risperidone Meloxicam Silver Sulfadiazine Methylcobalamin Sodium Bicarbonate Methylprednisolone Base / Acetate Sodium Chloride Metoclopramide Hcl Sodium Valproate Metoprolol Succinate Tadalafil Metoprolol Tartrate Tolperisone Hcl Miconazole Nitrate / Base Triamcinolone Acetonide Minoxidil Triclabendazole Misoprostol 1 % with HPMC Triclosan Mometasone Furoate Trihexyphenidyl Nitroglycerine 10 % Trifluoperazine Hcl Olanzapine Trimetazidine Ondansetron Base / Hcl Triprolidine Hcl Oxfendazole Valproic Acid Oxyclozanide BP VET Vardenafil Oxymetazoline Hcl Xylometazolline HCL Paroxetine Hcl Zinc Gluconate Permethrin Zinc Pyrithione Pheniramine Maleate Ziprasidone Hcl Mono .

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The Management of Seborrheic Dermatitis 2020: an Update
Review Article The management of seborrheic dermatitis 2020: An update Sandra Widaty1, Kusmarinah Bramono1, Muhammad Yulianto Listiawan2, Ariyati Yosi3, Eliza Miranda1, Githa Rahmayunita1, Herwinda Brahmanti4, Henry W Lim5 1Department of Dermatology and Venereology, Faculty of Medicine Universitas Indonesia, Dr. Cipto Mangunkusumo National General Hospital, Jakarta, Indonesia 2Department of Dermatology and Venereology, Faculty of Medicine Universitas Airlangga, Dr. Soetomo General Hospital, Surabaya, Indonesia 3Department of Dermatology and Venereology, Faculty of Medicine Universitas Sumatera Utara, H. Adam Malik General Hospital, Medan, Indonesia 4Department of Dermatology and Venereology, Faculty of Medicine Universitas Brawijaya, Dr. Saiful Anwar General Hospital, Malang, Indonesia 5Department of Dermatology, Henry Ford Hospital, Detroit, Michigan, USA Email: [email protected] Abstract Background: Seborrheic dermatitis (SD) is a chronic relapsing dermatitis manifesting in the seborrheic area, affecting infants or adults. In Indonesia, the prevalence of SD is 0.99–5.8% of all dermatology cases from 2013 to 2015. SD has been known to be a prominent manifestation among HIV patients, but there is an increasing trend in the general population. Therefore, in 2017, the Indonesian Society for Dermatology and Venereology proposed a consensus for the management of SD in Indonesia based on the discussion from 12 dermatological centers. Concurrent with the development of new drugs, this study aims to evaluate and develop a guideline for the treatment of seborrheic dermatitis in Indonesia to update the previous guidelines in 2017. Methods: Systematic review was based on evidence-based methods, and scientific evidences were acquired through systematic search. Evidence analysis was in accordance with the level of evidence. The available evidences were evaluated, and conclusion was based on the grade of recommendation.
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Seborrheic Dermatitis and Its Relationship with Malassezia Spp
REVIEW Seborrheic dermatitis and its relationship with Malassezia spp Manuel Alejandro Salamanca-Córdoba†,1,2, Carolina Alexandra Zambrano-Pérez2,3, Carlos Mejía-Arbeláez2,4, Adriana Motta5, Pedro Jiménez6, Silvia Restrepo-Restrepo7, Adriana Marcela Celis-Ramírez1,8* Abstract Seborrheic dermatitis (SD) is a chronic inflammatory disease that that is difficult to manage and with a high impact on the individual’s quality of life. Besides, it is a multifactorial entity that typically occurs as an inflammatory response toMalassezia species, along with specific triggers that contribute to its pathophysiology. Sin- ce the primary underlying pathogenic mechanisms include Malassezia proliferation and skin inflammation, the most common treatment includes topical antifungal keratolytics and anti-inflammatory agents. However, the consequences of eliminating the yeast population from the skin, the resistance profiles ofMalassezia spp. and the effectivity among different groups of medications are unknown. Thus, in this review, we summarize the current knowledge on the disease´s pathophysio- logy and the role of Malassezia sp. on it, as well as, the different antifungal treatment alternatives, including topical and oral treatment in the management of SD. Key words: seborrheic dermatitis, Malassezia, pathogenic role, treatment. Dermatitis seborreica y su relación con Malassezia spp Resumen La dermatitis seborreica (DS) es una enfermedad inflamatoria crónica, con un elevado impacto en la calidad de vida del individuo. Además, DS es una entidad multifactorial que ocurre como respuesta inflamatoria a las levaduras del género Malassezia spp., junto con factores desencadenantes que contribuyen a la fisio- patología de la enfermedad. Dado que el mecanismo patogénico principal involucra la proliferación e inflamación generada por Malassezia spp., el tratamiento más usado son los agentes tópicos antifúngicos y antiinflamatorios.
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Seborrheic Dermatitis and Its Relationship with Malassezia Spp
REVIEW Seborrheic dermatitis and its relationship with Malassezia spp Manuel Alejandro Salamanca-Córdoba†,1,2, Carolina Alexandra Zambrano-Pérez2,3, Carlos Mejía-Arbeláez2,4, Adriana Motta5, Pedro Jiménez6, Silvia Restrepo-Restrepo7, Adriana Marcela Celis-Ramírez1,8* Abstract Seborrheic dermatitis (SD) is a chronic inflammatory disease that that is difficult to manage and with a high impact on the individual’s quality of life. Besides, it is a multifactorial entity that typically occurs as an inflammatory response toMalassezia species, along with specific triggers that contribute to its pathophysiology. Sin- ce the primary underlying pathogenic mechanisms include Malassezia proliferation and skin inflammation, the most common treatment includes topical antifungal keratolytics and anti-inflammatory agents. However, the consequences of eliminating the yeast population from the skin, the resistance profiles ofMalassezia spp. and the effectivity among different groups of medications are unknown. Thus, in this review, we summarize the current knowledge on the disease´s pathophysio- logy and the role of Malassezia sp. on it, as well as, the different antifungal treatment alternatives, including topical and oral treatment in the management of SD. Key words: seborrheic dermatitis, Malassezia, pathogenic role, treatment. Dermatitis seborreica y su relación con Malassezia spp Resumen La dermatitis seborreica (DS) es una enfermedad inflamatoria crónica, con un elevado impacto en la calidad de vida del individuo. Además, DS es una entidad multifactorial que ocurre como respuesta inflamatoria a las levaduras del género Malassezia spp., junto con factores desencadenantes que contribuyen a la fisio- patología de la enfermedad. Dado que el mecanismo patogénico principal involucra la proliferación e inflamación generada por Malassezia spp., el tratamiento más usado son los agentes tópicos antifúngicos y antiinflamatorios.
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349 © Springer Nature Switzerland AG 2020 A. Tosti Et Al. (Eds.), Hair
Index A diagnosis, 237 Abatacept, 118, 344 management, 237, 238 Acitretin, 183, 184 phototherapy, 240 Acne keloidalis nuchae (AKN), 18–20, 155, poor prognostic factors, 237 156 systemic treatment, 239 Acute telogen effluvium, 125, 129 topical and intralesional treatment Adalimumab, 185, 186 options, 238 Adams-Oliver syndrome, 242 impregnate a cotton swab, 28 Adjuvant therapy, 121 intralesional corticosteroid therapy, African hair, 262 111, 112 African sensitive hair, 263 intralesional steroid dosing, 112 5 alfa reductase inhibitors, 49 intramuscular steroid therapy, 114 Aloe vera, 169 intravenous steroid therapy, 114 Alopecia areata (AA), 21, 81, 85, 237, 247, JAK inhibitors, 119 300, 301, 324, 328, 329 low/no efficacy, 122 abatacept, 344 microneedling in, 40 adjuvant therapy, 119–121 mild dermatitis, 28 affecting beard/eyelashes/eyebrows, 111 modified DPCP protocol, 27 allergic reaction, 28, 31 oral JAK inhibitors, 342, 343 anthralin, 116 oral steroid therapy, 114 apremilast, 344 patch test, 27 BMD1141, 343, 344 patient’s reaction, 29 BNZ-1, 343 phototherapy and excimer laser, 116–118 in children and adults treatment, 110 phototherapy and laser therapy, 117 children and pregnant women, 33, 34 prostaglandin F2a analogs, 116 corticosteroid-sparing agents, 115 SADBE, application of, 25, 30 different corticosteroid-sparing agents, 114 scalp micropigmentation, 110 DMARD, 114, 115 schedule visits, 29 DPCP, 25, 343 sensitize patients, 27 dupilumab, 345 side effects, 31, 33 efficacy assessment, 31 suitable therapeutic regimen, 109–111 evidence-based treatment options, 120, 121 systemic steroids + corticosteroid-sparing/ feedback about allergic reaction, 29 DMARD agents, 115 fumaric acid, 344 systemic therapy, 113 hair loss targeted therapeutics, 118 course of, 237 topical immunotherapy, 25, 30, 115, 116 © Springer Nature Switzerland AG 2020 349 A.
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Efficacy and Safety of Cream Containing Climbazole/Piroctone Olamine for Facial Seborrheic Dermatitis: a Single-Center, Open-Label Split-Face Clinical Study
Efficacy and Safety of C/P Cream for Facial SD pISSN 1013-9087ㆍeISSN 2005-3894 Ann Dermatol Vol. 28, No. 6, 2016 https://doi.org/10.5021/ad.2016.28.6.733 ORIGINAL ARTICLE Efficacy and Safety of Cream Containing Climbazole/Piroctone Olamine for Facial Seborrheic Dermatitis: A Single-Center, Open-Label Split-Face Clinical Study Hae Jeong Youn1, Soo Young Kim1, Minji Park2, Won Hee Jung2, Yang Won Lee1,3, Yong Beom Choe1,3, Kyu Joong Ahn1,3 1Department of Dermatology, Konkuk University School of Medicine, Seoul, 2Department of Systems Biotechnology, Chung-Ang University, Anseong, 3Research Institute of Medical Science, Konkuk University, Seoul, Korea Background: Seborrheic dermatitis (SD) is a multifactorial lient cream-treated left side. For the C/P cream, the MICs disease; Malassezia species play an important role in its were 0.625, 5, 0.625, and 2.5 mg/ml for Malassezia restricta, pathogenesis. Objective: We aimed to determine whether a M. globosa, M. sympodialis, and M. slooffiae, respectively. cream containing climbazole/piroctone olamine (C/P Conclusion: Based on the reduced casual sebum level and cream), antifungal agents with expected efficacy against extent of erythema, the antifungal activity of C/P cream Malassezia species, could improve SD symptoms. Methods: against Malassezia species seems useful for the treatment of We instructed 24 patients with mild-to-moderate SD to apply mild to moderate SD. (Ann Dermatol 28(6) 733∼739, 2016) the C/P cream and emollient cream on the right and left sides of the face, respectively, every morning and evening for 4 -Keywords- weeks. The casual sebum level (measured with SebumeterⓇ; Climbazole, Malassezia species, Piroctone olamine, Courage & Khazaka Electronic GmbH, Germany) and the ex- Seborrheic dermatitis tent of erythema (measured with MexameterⓇ; Courage & Khazaka Electronic GmbH) on the face were measured at baseline and after 4 weeks.
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