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January 2007 January 2007 Published electronically by The Grand Lodge of Free & Accepted Masons of New York Volume 1 Issue 8 Message from the Grand East In this Issue To My Brothers: Message from the Grand East Welcome back from your holiday season. I hope each of you will have a MMRL Press Release Happy and Healthy New Year. Masonic Compact Bro. Gerald R. Ford Today, our Craft is moving forward in many areas. The strides made in From the Editor Communications, Community Involvement, Education and in the Masonic Breakfast presentation of our Ritual are becoming apparent. One obvious measure of Mark Russell success is the number of new raisings in our Craft. We are beginning to see the results of your efforts in presenting our Craft to the public and to your The Knights Templars, the Holy Grail, and the Tarot friends. Remember, Encourage Your Friends to Become Your Brother, along with Brother Bring A Friend Night programs, will Lodge Services help your Lodges grow. Use these programs and see the results. From the Grand Lecturer Grand Lodge Calendar As we start our New Year, we have some exciting news from our Masonic Committee Chairmen Medical Research Laboratory. Their ongoing work in the area of cardiac Brotherhood Fund arrhythmia research continues to result in new breakthroughs that not From the Chairman of Grand Lodge only help our members to live longer and healthier lives but their research Communication Umbrella Public Relations has impact on the entire world. For almost 50 years, the scientists at your Community Involvement Umbrella Committee Masonic Medical Research Laboratory have worked to aid humanity. Be Child ID proud of their efforts and stand proud that we as members of this great Masonic Student Assistance Training Program Fraternal Order are making a difference and contribute every day to the (MSAT) welfare of the world around us. Masonic Care Community Fellowshiip committee Best regards to all, Masonic University of New York (MUNY) Neal Bidnick Grand Master Camp Turk / Youth Committee Chancellor Livingston Library Lodge Renewal 225th Anniversary Masonic Medical Research Laboratory From Whose Bourne Around the State Bro. Ron Kamp [email protected] MMRL Scientists Discover New Genes Responsible for Sudden Cardiac Arrest Scientists at the Masonic Medical Research various ions to move in and out of the cardiac Laboratory (MMRL) in Utica, NY have cell leads to an electrical imbalance that can uncovered a new genetic basis for abnormal disrupt the normal distribution of the electrical rhythms of the heart responsible for sudden charge throughout the heart, resulting in cardiac arrest. abnormal rhythms. Some arrhythmias are benign, such as premature beats, and others are The landmark discovery is reported in the deadly, including ventricular tachycardia and current issue of Circulation, the leading fibrillation. The ECG, which records the Cardiology journal published by the American electrical activity of the heart from electrodes Heart Association placed on the body surface, can be used to (AHA). Dr. Charles detect these rhythm disturbances. The ECG is Antzelevitch and a normally comprised of a P wave, reflecting the team of activation of the upper chambers of the heart investigators and (atria), a QRS wave denoting the activation of collaborators from the lower chambers of the heart (ventricles) and Canada, Germany, a T wave, which is inscribed as a result of the France and Italy repolarization associated with relaxation of the describe a new heart. The interval between the QRS and T clinical entity waves (QT interval) normally ranges between characterized by 360 and 460 milliseconds and the ST segment distinctive changes of the normal ECG is isoelectric (neither in the electrocardiogram (ECG) in three families elevated or depressed). with a history of sudden cardiac death. Affected family members were all found to have The new clinical entity is a combination of two mutations in the genes that encode the cardiac distinct sudden death syndromes known as the calcium channels. These channels permit the Brugada and Short QT syndromes. The flow of calcium ion in and out of cells in the Brugada syndrome is characterized by an heart. The defective genes called CACNA1C and elevation of the ST segment in the ECG and the CACNB2b were found to generate a smaller short QT syndrome distinguished by a briefer than normal electrical current and thus to be than normal QT interval (less than or equal to responsible for creating an electrical imbalance 360 milliseconds). These syndromes have that results in potentially fatal abnormal heart previously been shown to be due to defects in rhythms, known as cardiac arrhythmias. genes that control the flow of sodium and potassium ions across the membrane of the Although the heart is a mechanical pump, each cardiac cell. Antzelevitch, who serves as and every beat is initiated by electrical activity Executive Director and Director of Research of that originates in the upper part of the heart the MMRL, and his colleagues at the MMRL called the sinus node and is transmitted in a were the first to identify the KCNH2 gene very orderly fashion through the remainder of responsible for the Short QT syndrome and the heart. The electrical current is due to the together with colleagues at Baylor College of movement of ions such as sodium, potassium Medicine were the first to describe the SCN5A and calcium across the cell membrane. Defects gene responsible for the Brugada syndrome. in the function of the channels that permit these Each syndrome is capable of independently Page 2 January 2007 producing sudden cardiac arrest. The new found to affect ion channels within the heart clinical entity, by combining the two leading to sudden cardiac arrest.” Antzelevitch malfunctioning electrical features, presents a agreed, indicating that “we are at the tip of the situation of double jeopardy. iceberg and have a great deal to learn before we can routinely use genetic screening to identify Antzelevitch, the leading investigator in the new children and adults at risk for sudden cardiac study, recently presented preliminary results of arrest.” the research at the late-breaking abstract session at the annual scientific sessions of the “The impressive progress that we have made in AHA. Dr. Guido Pollevick, acting head of the this field of medicine in recent years is Molecular Genetics program at the MMRL, encouraging and with appropriate commitment presented a poster at the AHA meeting of resources, we can look forward to dramatic regarding additional aspects of this new clinical advances in the months and years ahead”, he entity. added According to Founded in 1958 by the Grand Lodge of Free & Antzelevitch “our Accepted Masons of the State of New York, the ability to link calcium MMRL is internationally renowned medical channel loss of research and educational institute dedicated to function mutations to studies of the electrical activity of the heart and sudden cardiac death the mechanisms responsible for abnormal opens exciting new rhythms of the heart. In recent years, the avenues for better MMRL has also become a central hub for diagnosis and genetic screening of inherited arrhythmic treatment of inherited diseases in the United States. MMRL scientists sudden death have uncovered the mechanisms responsible for syndromes that affect many forms of life-threatening cardiac young adults, children and infants.” Scientists arrhythmias as well as the mechanisms by at the MMRL, in collaboration with colleagues which some drugs act to precipitate in Italy, were the first to identify the genetic arrhythmias. In recent years, they have defect responsible for Sudden Infant Death delineated the genetic basis for several inherited Syndrome, linking SIDS to a malignant cardiac sudden cardiac death syndromes. Prominent arrhythmia. among their most recent achievements is the identification of a novel strategy for the Dr. Jonathan Cordeiro, a research scientist pharmacologic treatment of atrial fibrillation, involved with assessing the effects of the one of the greatest unmet medical needs facing mutated genes on electrical function, thought our society. that “in time many more mutations will be Page 3 January 2007 Page 4 January 2007 In Honor of Gerald Rudolph Ford, The 38th President of the United States, Born 1913 - Called Home by the Grand Architect December 2006. GERALD RUDOLPH FORD(1913-2006 ) Thirty-eighth President (1974-1977) MASONIC RECORD Initiated: September 30, 1949, Malta Lodge No. 465, Grand Rapids, Michigan, along with his half-brothers Thomas Gardner Ford (1918-1995), Richard Addison Ford (1924-) and James Francis Ford (1927- ). The Fellowcraft and Master Mason Degrees were Conferred by Columbia Lodge No. 3, Washington, D.C., on April 20 and May 18, 1951, as a courtesy to Malta Lodge. Brother Ford was made a Sovereign Grand Inspector General, 33°, and Honorary Member, Supreme Council A.A.S.R. Northern Jurisdiction at the Academy of Music in Philadelphia, on September 26, 1962, for which he served as Exemplar (Representative) for his Class. Brother and President Ford was unanimously elected an Active Member of the International Supreme Council, Order of DeMolay and its Honorary Grand Master, at its Annual Session held at Orlando, Florida, April 6-9, 1975; Brother Ford held this post until January 1977, at which time he became a Past Honorary Grand Master, receiving his Collar and Jewel on October 24, 1978 in Topeka, Kansas, from the Hon. Thomas C. Raum, Jr., Grand Master, Order of DeMolay. Brother Ford was the last President who is a Freemason. The following is the text of a message of condolence on the passing of our Brother Mason, Gerald R. Ford, 38th President of the United States of America and a member of Malta Lodge No 465, Grand Rapids, Michigan. This statement was issued December 27, 2006 by the Grand Masters of New York and Michigan and the Executive Secretary of the Masonic Service Association of North America.
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