HEENT EXAMINIATION ______HEENT Exam Exam Overview
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Advanced Interpretation of Adult Vital Signs in Trauma William D
Advanced Interpretation of Adult Vital Signs in Trauma William D. Hampton, DO Emergency Physician 26 March 2015 Learning Objectives 1. Better understand vital signs for what they can tell you (and what they can’t) in the assessment of a trauma patient. 2. Appreciate best practices in obtaining accurate vital signs in trauma patients. 3. Learn what teaching about vital signs is evidence-based and what is not. 4. Explain the importance of vital signs to more accurately triage, diagnose, and confidently disposition our trauma patients. 5. Apply the monitoring (and manipulation of) vital signs to better resuscitate trauma patients. Disclosure Statement • Faculty/Presenters/Authors/Content Reviewers/Planners disclose no conflict of interest relative to this educational activity. Successful Completion • To successfully complete this course, participants must attend the entire event and complete/submit the evaluation at the end of the session. • Society of Trauma Nurses is accredited as a provider of continuing nursing education by the American Nurses Credentialing Center's Commission on Accreditation. Vital Signs Vital Signs Philosophy: “View vital signs as compensatory to the illness/complaint as opposed to primary.” Crowe, Donald MD. “Vital Sign Rant.” EMRAP: Emergency Medicine Reviews and Perspectives. February, 2010. Vital Signs Truth over Accuracy: • Document the true status of the patient: sick or not? • Complete vital signs on every patient, every time, regardless of the chief complaint. • If vital signs seem misleading or inaccurate, repeat them! • Beware sending a patient home with abnormal vitals (especially tachycardia)! •Treat vital signs the same as any other diagnostics— review them carefully prior to disposition. The Mother’s Vital Sign: Temperature Case #1 - 76-y/o homeless ♂ CC: 76-y/o homeless ♂ brought to the ED by police for eval. -
CASE REPORT 48-Year-Old Man
THE PATIENT CASE REPORT 48-year-old man SIGNS & SYMPTOMS – Acute hearing loss, tinnitus, and fullness in the left ear Dennerd Ovando, MD; J. Walter Kutz, MD; Weber test lateralized to the – Sergio Huerta, MD right ear Department of Surgery (Drs. Ovando and Huerta) – Positive Rinne test and and Department of normal tympanometry Otolaryngology (Dr. Kutz), UT Southwestern Medical Center, Dallas; VA North Texas Health Care System, Dallas (Dr. Huerta) Sergio.Huerta@ THE CASE UTSouthwestern.edu The authors reported no A healthy 48-year-old man presented to our otolaryngology clinic with a 2-hour history of potential conflict of interest hearing loss, tinnitus, and fullness in the left ear. He denied any vertigo, nausea, vomiting, relevant to this article. otalgia, or otorrhea. He had noticed signs of a possible upper respiratory infection, including a sore throat and headache, the day before his symptoms started. His medical history was unremarkable. He denied any history of otologic surgery, trauma, or vision problems, and he was not taking any medications. The patient was afebrile on physical examination with a heart rate of 48 beats/min and blood pressure of 117/68 mm Hg. A Weber test performed using a 512-Hz tuning fork lateral- ized to the right ear. A Rinne test showed air conduction was louder than bone conduction in the affected left ear—a normal finding. Tympanometry and otoscopic examination showed the bilateral tympanic membranes were normal. THE DIAGNOSIS Pure tone audiometry showed severe sensorineural hearing loss in the left ear and a poor speech discrimination score. The Weber test confirmed the hearing loss was sensorineu- ral and not conductive, ruling out a middle ear effusion. -
Bates' Pocket Guide to Physical Examination and History Taking
Lynn S. Bickley, MD, FACP Clinical Professor of Internal Medicine School of Medicine University of New Mexico Albuquerque, New Mexico Peter G. Szilagyi, MD, MPH Professor of Pediatrics Chief, Division of General Pediatrics University of Rochester School of Medicine and Dentistry Rochester, New York Acquisitions Editor: Elizabeth Nieginski/Susan Rhyner Product Manager: Annette Ferran Editorial Assistant: Ashley Fischer Design Coordinator: Joan Wendt Art Director, Illustration: Brett MacNaughton Manufacturing Coordinator: Karin Duffield Indexer: Angie Allen Prepress Vendor: Aptara, Inc. 7th Edition Copyright © 2013 Wolters Kluwer Health | Lippincott Williams & Wilkins. Copyright © 2009 by Wolters Kluwer Health | Lippincott Williams & Wilkins. Copyright © 2007, 2004, 2000 by Lippincott Williams & Wilkins. Copyright © 1995, 1991 by J. B. Lippincott Company. All rights reserved. This book is protected by copyright. No part of this book may be reproduced or transmitted in any form or by any means, including as photocopies or scanned-in or other electronic copies, or utilized by any information storage and retrieval system without written permission from the copyright owner, except for brief quotations embodied in critical articles and reviews. Materials appear- ing in this book prepared by individuals as part of their official duties as U.S. government employees are not covered by the above-mentioned copyright. To request permission, please contact Lippincott Williams & Wilkins at Two Commerce Square, 2001 Market Street, Philadelphia PA 19103, via email at [email protected] or via website at lww.com (products and services). 9 8 7 6 5 4 3 2 1 Printed in China Library of Congress Cataloging-in-Publication Data Bickley, Lynn S. Bates’ pocket guide to physical examination and history taking / Lynn S. -
Current Audiometric Tests for the Detection of Functional Hearing Loss
CURRENT AUDIOMETRIC TESTS FOR THE DETECTION OF FUNCTIONAL HEARING LOSS by JOAN PHYLLIS COOPER X^^ Be A., Union College, 1965 A MASTER'S REPORT submitted in partial fulfillment of the J requirements for the degree MASTER OF ARTS Department of Speech KANSAS STATE UNIVERSITY Manhattan, Kansas 1963 Approved by: Ma j or ? ro re s s or . bo 11 C6£ TABLE OF CONTENTS Chapter Page I , Introduction .••••••••••••••••••«••• • • • 1 Definitions ....?................«« •••••••• 2 II. Tests for Functional Hearing Loss 4 General Behavior in the Clinical Evaluation 4 The Ear, Nose, and Throat Examination 9 Pure-tone Audiometry 12 Saucer-shaped Audiograms 14 False-alarm Responses During Pure-tone Audiometry ...................... c 17 Inappropriate Lateralization ,« 17 Bone Conduction Audiometry 20 Speech Audiometry ••••••••••••••••• • 22 Speech Discrimination ••••••••••• 22 Speech Reception Threshold ........ ............... 23 Errors During Measurement of Spondee Threshold ... 2 3 Inappropriate Lateralization 26 Pure-tone Stenger Test 26 Modified (Speech) Stenger Test 29 Doerf ler-Stewart Test 30 Lombard Test ............ o 36 De layed Auditory Feedback ., 39 Psychogalvanic Skin Resistance Test 45 Modification of EDR Test , . .. 50 Bekesy Type V Tracing ......... o ..... ....... ... s ...... 51 c ^ . iii Chapter Page ( II. Rainville Test 5S Sensorineural Acuity Level Test ..... 59 Lipreading Test .....«•..•.<.«•......,....•............ 61 The Variable Intensity Pulse Count Method 62 Rapid Random Loudness Judgments ..................... c 63 Middle Ear -
Copyrighted Material
Index Page numbers in italics denote fi gures, anion gap 263 Baker’s cyst 100, 104 those in bold denote tables. ankle swelling 195–8 bamboo spine 189 ankle-brachial pressure index 70–1 basal cell carcinoma 77 abdominal aortic aneurysm 145 ankylosing spondylitis 179, 189 basic life support 282–5 abdominal distension 143–7, 145 antalgic gait 25, 42, 94 Behçet’s disease 181 abdominal examination 10–19 anti-tuberculosis drugs 223 benign paroxysmal positional vertigo abdominal masses 17–18, 17 antiepileptics 223 173 abdominal pain 137–42 antihypertensives 223 benign prostatic hypertrophy 57 acute 138 anxiety berry aneurysm 33 chronic 139 faintness 175 biceps tendonitis 81 investigations 139–40 palpitations 120 bleeding diathesis 149 origin of 140–1, 141 tremor 169 blood gas analysis see arterial blood gas abducens nerve 34 aortic dissection 116 analysis abscess 193 aortic regurgitation 3 Boerhaave’s syndrome 149 breast 54 aortic stenosis 3 Bouchard’s nodes 90 perianal 57 aorto-enteric fi stula 149 bowel cancer see colorectal cancer acidosis apex beat 4 bowel obstruction 144, 146 metabolic 263, 265 Apley’s test 102 brachial plexus 85 respiratory 264 apologising 237 brain natriuretic peptide 6 acoustic neuroma 34, 175 appendicectomy 218 breaking bad news 208–10 acquired immunodefi ciency syndrome apraxic gait 25 SPIKES framework 209 see HIV/AIDS arterial blood gas analysis 262–6, 264–5 breast abscess 54 acromioclavicular joint arterial examination 68–72 breast examination 53–5 arthritis 81 arterial ulcer 70 description of lumps 55, 55 Scarf test -
Blood Pressure Year 1 Year 2 Core Clinical/Year 3+
Blood Pressure Year 1 Year 2 Core Clinical/Year 3+ Do Do • Patient at rest for 5 minutes • Measure postural BP and pulse in patients with a • Arm at heart level history suggestive of volume depletion or • Correct size cuff- bladder encircles 80% of arm syncope • Center of cuff aligns with brachial artery -Measure BP and pulse in supine position • Cuff wrapped snugly on bare arm with lower -Slowly have patient rise and stand (lie them down edge 2-3 cm above antecubital fossa promptly if symptoms of lightheadedness occur) • Palpate radial artery, inflate cuff to 70 mmHg, -Measure BP and pulse after 1 minute of standing then increase in 10 mmHg increments to 30 mmHg above point where radial pulse disappears. Know Deflate slowly until pulse returns; this is the • Normally when a person stands fluid shifts to approximate systolic pressure. lower extremities causing a compensatory rise in • Auscultate the Korotkoff sounds pulse by up to 10 bpm with BP dropping slightly -place bell lightly in antecubital fossa • Positive postural vital signs are defined as -inflate BP to 20-30mmHg above SBP as determined symptoms of lightheadedness and/or a drop in by palpation SBP of 20 mmHg with standing -deflate cuff at rate 2mmHg/second while auscultating • Know variations in BP cuff sizes -first faint tapping (Phase I Korotkoff) = SBP; • A lack of rise in pulse in a patient with an Disappearance of sound (Phase V Korotkoff)=DBP orthostatic drop in pressure is a clue that the Know cause is neurologic or related or related to -Korotkoff sounds are lower pitch, better heard by bell medications (eg. -
Patient Assessment: 3 Techniques of Physical Examination: 2
Patient Assessment: 3 Techniques of Physical Examination: 2 W4444444444444444444444444444444444444444444444444444444444444444444444444444444444444444444444444444444444444 UNIT TERMINAL OBJECTIVE 3-2 At the completion of this unit, the EMT-Critical Care Technician student will be able to explain the significance of physical exam findings commonly found in emergency situations. COGNITIVE OBJECTIVES At the completion of this unit, the EMT-Critical Care Technician student will be able to: 3-2.1 Define the terms inspection, palpation, percussion, auscultation. (C-1) 3-2.2 Describe the techniques of inspection, palpation, percussion, and auscultation. (C-1) 3-2-3 Review the procedure for taking and significance of vital signs (pulse, respiration, and blood pressure.) (C-2) 3-2.4 Describe the evaluation of mental status. (C-1) 3-2.5 Evaluate the importance of a general survey. (C-3) 3-2.6 Describe the examination of skin and nails. (C-1) 3-2.7 Differentiate normal and abnormal findings of the assessment of the skin. (C-3) 3-2.8 Distinguish the importance of abnormal findings of the assessment of the skin. (C-3) 3-2.9 Describe the normal and abnormal assessment findings of the head (including the scalp, skull, face and skin). (C-1) 3-2.10 Describe the examination of the head (including the scalp, skull, face, and skin). (C-1) 3-2.11 Describe the examination of the eyes. (C-1) 3-2.12 Distinguish between normal and abnormal assessment findings of the eyes. (C-3) 3-2.13 Describe the examination of the ears. (C-1) 3-2.14 Differentiate normal and abnormal assessment findings of the ears. -
Examination of the Knee
Examination of the Knee The Examination For every joint of the lower extremity always begin with the patient in standing IN STANDING INSPECTION 1. Cutaneous Structures: Look for Erythema, scarring, bruising, and swelling in the following areas: a. Peripatellar grooves b. Suprapatellar bursa c. Prepatellar bursa d. Infrapatellar tendon e. Anserine bursa f. Popliteal fossa 2. Muscle & Soft Tissue: a. Quadriceps atrophy b. Hamstring atrophy c. Calf atrophy 3. Bones & Alignment: a. Patella position (Alta, Baha, Winking, Frog eyed), b. Varus or Valgus alignment c. Flexion contracture or Genu recurvatum RANGE OF MOTION - ACTIVE Standing is the best opportunity to assess active range of motion of the knee. 1. Ask the patient to squat into a deep knee bend. Both knees should bend symmetrically. 2. Ask the patient to then stand and extend the knee fully – lock the knee. The knee should straighten to 0 degrees of extension. Some people have increased extension referred to as genu recurvatum. GAIT 1. Look for a short stance phase on the affected limb and an awkward gait if a concomitant leg length discrepancy 2. Look for turning on block 3. Screening 1. Walk on the toes 2. Walk on the heels 3. Squat down – Active Range of Motion testing SPECIAL TESTS 1. Leg Length Discrepancy a. Look at patients back for evidence of a functional scoliosis b. Place your hands on the patients Iliac crests looking for inequality which may mean a leg length discrepancy IN SITTING NEUROLOGIC EXAMINATION 1. Test the reflexes a. L4 – Quadriceps reflex VASCULAR EXAMINATION 1. Feel for the posterior tibial artery SUPINE POSITION INSPECTION 1. -
Primary Care Assessment of Suspected Stroke-TIA Pathway
Primary Care Assessment of Rapid onset of new neurological deficit or symptom Suspected Stroke/TIA Rapid means over seconds or minutes, or rarely, hours Exclude/ treat Click for hypoglycaemia more info Perform face, arm, speech test (FAST) • Has the person’s face fallen on one side? Can they smile? • Can they raise both arms and keep them there? • Is their speech slurred? FAST positive FAST negative See pathway Negative, but history Primary Care Risk of FAST positive now Stratification of Suspected resolved TIA Upper or Lower Motor Neurone Sparing of the forehead, and possibly maintenance of spontaneous smiling = UMN Quick assessment Facial nerve weakness lesion. Lower Motor Neurone Leg weakness YES for other neurological only? Whole of one side affected, including the facial weakness forehead = LMN lesion. This could be caused by a deficit brainstem stroke in which case it would be Diplopia squint 3rd, 4th, accompanied by other signs such as tremor, 6th ataxia, vertigo and horizontal gaze palsy Weakness 11th of Rapid onset of other shoulder elevation, NO Upper Motor Neurone motor symptoms or Other cranial nerves ninging of scapula, (sparing of forehead) signs weakness of head turning See pathway Deviation of tongue or Management of uvula 12th nerve Suspected Stroke Numbness or altered Exclude peripheral nerve sensation or dermatomal pattern Rapid onset sensory Click for more symptoms or signs info Sensory inattention Comprehension difficulties may be for the spoken or written word or for both or for one language only Speech difficulties -
Examples of Questions for the Exam
MedIS exam 2013 Course title: Nervesystemet og bevægeapparatet II - MedIS Programme: Bachelor in education Semester: 5. semester Exam date: 21-1-12 Time: 9:00 - 12:00 Evaluation form 7-point scale – External censur Important information: Remember to bring your student identification card Remember to put your student number – not your name and cpr no – on all sheets that you hand in for evaluation Remember to hand in the assignment if you leave the exam before it has ended No written aid is allowed Quicktionary pen is allowed Your paper must be handed in on paper in hand written form The study board/the university cannot be held liable if any problems should occur regarding the electronic aid during the examination It will be considered cheating or attempting to cheat if the technical equipment of the student is communicating or trying to communicate with other equipment not relevant to the exam, without an explicit permission. Before the beginning of the exam, the student should make sure that all communication devices in the equipment at the exam are turned off. Communication is not allowed The exam consists of a mixture of multiple choice questions and essay questions. The total amount of questions is 36. The total amount of points is 60. This lists the grades corresponding to the points: 12 (passed): 56-60 points 10 (passed): 51-55.5 points 07 (passed): 46-50.5 points 04 (passed): 41-45.5 points 02 (passed): 36-40.5 points 00 (failed): 18-35.5 points -2 (failed): 0-17.5 points 1 1. -
Bedside Neuro-Otological Examination and Interpretation of Commonly
J Neurol Neurosurg Psychiatry: first published as 10.1136/jnnp.2004.054478 on 24 November 2004. Downloaded from BEDSIDE NEURO-OTOLOGICAL EXAMINATION AND INTERPRETATION iv32 OF COMMONLY USED INVESTIGATIONS RDavies J Neurol Neurosurg Psychiatry 2004;75(Suppl IV):iv32–iv44. doi: 10.1136/jnnp.2004.054478 he assessment of the patient with a neuro-otological problem is not a complex task if approached in a logical manner. It is best addressed by taking a comprehensive history, by a Tphysical examination that is directed towards detecting abnormalities of eye movements and abnormalities of gait, and also towards identifying any associated otological or neurological problems. This examination needs to be mindful of the factors that can compromise the value of the signs elicited, and the range of investigative techniques available. The majority of patients that present with neuro-otological symptoms do not have a space occupying lesion and the over reliance on imaging techniques is likely to miss more common conditions, such as benign paroxysmal positional vertigo (BPPV), or the failure to compensate following an acute unilateral labyrinthine event. The role of the neuro-otologist is to identify the site of the lesion, gather information that may lead to an aetiological diagnosis, and from there, to formulate a management plan. c BACKGROUND Balance is maintained through the integration at the brainstem level of information from the vestibular end organs, and the visual and proprioceptive sensory modalities. This processing takes place in the vestibular nuclei, with modulating influences from higher centres including the cerebellum, the extrapyramidal system, the cerebral cortex, and the contiguous reticular formation (fig 1). -
Sudden Sensoryneural Hearing Loss As a Rare Attack Type in Multiple Sclerosis
380 Arch Neuropsychiatry 2018;55:380−382 LETTER TO EDITOR https://doi.org/10.5152/npa.2017.19270 Sudden Sensoryneural Hearing Loss As a Rare Attack Type in Multiple Sclerosis Amber EKER1 , Bahar KAYMAKAMZADE1 , Aslı TUNCER KURNE2 , Çağrı Mesut TEMUÇİN2 , Rana KARABUDAK2 1Department of Neurology, Near East University Faculty of Medicine, Nicasia, Cyprus 2Department of Neurology, Hacettepe University Faculty of Medicine, Ankara, Turkey ultiple sclerosis (MS) is a chronic, multifocal, inflammatory, A 35-year-old male patient who had been following with MS was Mdemyelinating disorder of the central nervous system. In addition admitted to hospital with the complaint of acute hearing loss. He had to common symptoms like optic neuritis (ON), sensorial, pyramidal, diagnosed with definite MS two years ago. The presenting symptom was and cerebellar dysfunction, a wide range of rare symptoms could be a left sided weakness, and hypoesthesia on the left upper extremity. At feature of MS. Rare symptoms could be the presenting symptom of MS. that time, the magnetic resonance imaging (MRI) of the brain revealed Herein, we report a case with MS with a very rare symptom; unilateral demyelinating lesions in periventricular white matter, corpus callosum vestibulocochlear nerve involvement and aimed to emphasize the and left capsula interna. Cervical MRI showed C3-C4 intramedullary importance of electrophysiological studies in the diagnosis. demyelinating plaque. None of the lesions had contrast enhancement. (a) Figure 1. a, b. T2 (a) and contrast enhanced T1 (b) sequences of the brain MRI does not (b) reveal any new lesion. Cite this article as: Eker A, Kaymakamzade B, Tuncer Kurne A, Temuçin ÇM, Karabudak R.