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Clinical Study Minimally Invasive Treatment of Infrabony Periodontal Defects Using Dual-Wavelength Laser Therapy
Hindawi Publishing Corporation International Scholarly Research Notices Volume 2016, Article ID 7175919, 9 pages http://dx.doi.org/10.1155/2016/7175919 Clinical Study Minimally Invasive Treatment of Infrabony Periodontal Defects Using Dual-Wavelength Laser Therapy Rana Al-Falaki,1 Francis J. Hughes,2 and Reena Wadia2 1 Al-FaPerio Clinic, 48AQueensRoad,BuckhurstHill,EssexIG95BY, UK 2Department of Periodontology, King’s College London Dental Institute, Floor 21,TowerWingGuysHospital,LondonSE19RT, UK Correspondence should be addressed to Rana Al-Falaki; [email protected] Received 23 January 2016;Revised15 April 2016;Accepted26 April 2016 Academic Editor: Jiiang H. Jeng Copyright © 2016 Rana Al-Falaki et al. Tis is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Introduction. Surgical management of infrabony defects is an invasive procedure, frequently requiring the use of adjunctive material such as grafs or biologics, which is time-consuming and associated with expense and morbidity to the patient. Lasers in periodontal regeneration have been reported in the literature, with each wavelength having potential benefts through diferent laser-tissue interactions. Te purpose of this case series was to assess the efcacy of a new dual-wavelength protocol in the management of infrabony defects. Materials and Methods. 32 defects (one in each patient) were treated using ultrasonic debridement, followed by fapless application of Erbium, Chromium:Yttrium, Scandium, Gallium, Garnet (Er,Cr:YSGG) laser (wavelength 2780 nm), and fnal application of diode laser (wavelength 940 nm). Pocket depths (PD) were measured afer 6 months and repeat radiographs taken afer one year. -
ABC of Oral Health Periodontal Disease John Coventry, Gareth Griffiths, Crispian Scully, Maurizio Tonetti
Clinical review ABC of oral health Periodontal disease John Coventry, Gareth Griffiths, Crispian Scully, Maurizio Tonetti Most periodontal disease arises from, or is aggravated by, accumulation of plaque, and periodontitis is associated particularly with anaerobes such as Porphyromonas gingivalis, Bacteroides forsythus, and Actinobacillus actinomycetemcomitans. Calculus (tartar) may form from calcification of plaque above or below the gum line, and the plaque that collects on calculus exacerbates the inflammation. The inflammatory reaction is associated with progressive loss of periodontal ligament and alveolar bone and, eventually, with mobility and loss of teeth. Periodontal diseases are ecogenetic in the sense that, in subjects rendered susceptible by genetic or environmental factors (such as polymorphisms in the gene for interleukin 1, cigarette smoking, immune depression, and diabetes), the infection leads to more rapidly progressive disease. Osteoporosis also seems to have some effect on periodontal bone loss. The possible effects of periodontal disease on systemic Chronic marginal gingivitis showing erythematous oedematous appearance health, via pro-inflammatory cytokines, have been the focus of much attention. Studies to test the strength of associations with atherosclerosis, hypertension, coronary heart disease, cerebrovascular disease, and low birth weight, and any effects on diabetic control, are ongoing. Gingivitis Chronic gingivitis to some degree affects over 90% of the population. If treated, the prognosis is good, but otherwise it may progress to periodontitis and tooth mobility and loss. Marginal gingivitis is painless but may manifest with bleeding from the gingival crevice, particularly when brushing the teeth. The gingival margins are slightly red and swollen, eventually with mild gingival hyperplasia. Management—Unless plaque is assiduously removed and Gingivitis with hyperplasia kept under control by tooth brushing and flossing and, where necessary, by removal of calculus by scaling and polishing by dental staff, the condition will recur. -
Generalized Aggressive Periodontitis Associated with Plasma Cell Gingivitis Lesion: a Case Report and Non-Surgical Treatment
Clinical Advances in Periodontics; Copyright 2013 DOI: 10.1902/cap.2013.130050 Generalized Aggressive Periodontitis Associated With Plasma Cell Gingivitis Lesion: A Case Report and Non-Surgical Treatment * Andreas O. Parashis, Emmanouil Vardas, † Konstantinos Tosios, ‡ * Private practice limited to Periodontics, Athens, Greece; and, Department of Periodontology, School of Dental Medicine, Tufts University, Boston, MA, United States of America. †Clinic of Hospital Dentistry, Dental Oncology Unit, University of Athens, Greece. ‡ Private practice limited to Oral Pathology, Athens, Greece. Introduction: Plasma cell gingivitis (PCG) is an unusual inflammatory condition characterized by dense, band-like polyclonal plasmacytic infiltration of the lamina propria. Clinically, appears as gingival enlargement with erythema and swelling of the attached and free gingiva, and is not associated with any loss of attachment. The aim of this report is to present a rare case of severe generalized aggressive periodontitis (GAP) associated with a PCG lesion that was successfully treated and maintained non-surgically. Case presentation: A 32-year-old white male with a non-contributory medical history presented with gingival enlargement with diffuse erythema and edematous swelling, predominantly around teeth #5-8. Clinical and radiographic examination revealed generalized severe periodontal destruction. A complete blood count and biochemical tests were within normal limits. Histological and immunohistochemical examination were consistent with PCG. A diagnosis of severe GAP associated with a PCG lesion was assigned. Treatment included elimination of possible allergens and non- surgical periodontal treatment in combination with azithromycin. Clinical examination at re-evaluation revealed complete resolution of gingival enlargement, erythema and edema and localized residual probing depths 5 mm. One year post-treatment the clinical condition was stable. -
A Concise Guide to Treating Painful Oral Lesions
Drugs Used to Treat Osteoporosis and Bone Cancer Perio & Implant Centers The Team for of the Monterey Bay (831) 648-8800 Jochen P. Pechak, DDS, MSD in Silicon Valley (408) 738-3423 Which May Cause Osteonecrosis of the Jaws mobile: www.DrPechakapp.com he many bisphosphonates and monoclonal antibodies which are used to treat osteoporosis and bone cancer often web: GumsRus.com causeDrugsDrugs osteonecrosis Used Used of the to jaws.to Treat AsTreat dental clinicians,Osteoporosis Osteoporosis it is important that and andwe are Bone awareBone of this Cancers Cancers side effect before Ttreating our patients who are taking these drugs. The tables below summarize these drugs, the route these drugs are administered, andWhich Whichtheir likelihood May May of causing Cause Cause osteonecrosis Osteonecrosis Osteonecrosis of the jaws as reported byof of Dr. the theRobert Jaws JawsMarx at the University of Miami Division of Oral and Maxillofacial Surgery. PDL tm Osteoporosis Drugs Drugs Osteoporosis Used to Treat Drugs Osteoporosis PerioDontaLetter Jochen P. Pechak, DDS, MSD, Periodontics and Implant Dentistry Spring DrugDrug ClassificationClassification ActionAction DoseDose RouteRoute %% of of ReportedReported CasesCases of of OsteonecrosisOsteonecrosis AlendronateAlendronate BisphosphonateBisphosphonate OsteoclastOsteoclast 7070 mg/wk mg/wk OralOral 8282%% From Our Office A Concise Guide to Treating (Fosamax(Fosamax ToxicityToxicity to Yours... Generic)Generic) Painful Oral Lesions ResidronateResidronate BisphosphonateBisphosphonate OsteoclastOsteoclast 3535 mg/wk mg/wk OralOral 1%1% As dentists specializing in treat- (Actonel Toxicity (Actonel Toxicity ment of diseases of the oral cavity atients present frequently with Treating Cold Sores Atelvia)Atelvia) and associated structures, we are painful oral lesions. They are often also called upon to treat pain- IbandronateIbandronate BisphosphonateBisphosphonate OsteoclastOsteoclast 150150 mg/mos mg/mos OralOral 1%1% usually not serious, but patients And Canker Sores (Boniva) Toxicity IV ful oral lesions in the mouth. -
Desquamative Gingivitis Desquamative Gingivitis
DOI: 10.5772/intechopen.69268 Provisional chapter Chapter 1 Desquamative Gingivitis Desquamative Gingivitis Hiroyasu Endo, Terry D. Rees, Hideo Niwa, HiroyasuKayo Kuyama, Endo, Morio Terry D.Iijima, Rees, Ryuuichi Hideo Niwa, KayoImamura, Kuyama, Takao Morio Kato, Iijima, Kenji Doi,Ryuuichi Hirotsugu Imamura, TakaoYamamoto Kato, and Kenji Takanori Doi, Hirotsugu Ito Yamamoto and TakanoriAdditional information Ito is available at the end of the chapter Additional information is available at the end of the chapter http://dx.doi.org/10.5772/intechopen.69268 Abstract Desquamative gingivitis (DG) is characterized by erythematous, epithelial desquama‐ tion, erosion of the gingival epithelium, and blister formation on the gingiva. DG is a clinical feature of a variety of diseases or disorders. Most cases of DG are associated with mucocutaneous diseases, the most common ones being lichen planus, mucous membrane pemphigoid, and pemphigus vulgaris. Proper diagnosis of the underlying cause is important because the prognosis varies, depending on the disease. This chapter presents the underlying etiology that is most commonly associated with DG. The current literature on the diagnostic and management modalities of patients with DG is reviewed. Keywords: gingival diseases/pemphigus/pemphigoid, benign mucous membrane/lichen planus, oral/hypersensitivity/autoimmune diseases 1. Introduction Manifestations of desquamative gingivitis (DG) include erythematous gingiva, epithelial des‐ quamation, and erosion of the gingival epithelium, as well as blister formation on the gingiva [1, 2] (Figure 1). The DG lesions may be localized or generalized and may extend into the alveolar mucosa. Similar lesions are often found on the buccal mucosa, tongue, and palate in the oral cavity. The signs of DG are clearly different from those of dental plaque‐induced gingivitis. -
A Mini Review on Non-Augmentative Surgical Therapy of Peri-Implantitis—What Is Known and What Are the Future Challenges?
MINI REVIEW published: 20 April 2021 doi: 10.3389/fdmed.2021.659361 A Mini Review on Non-augmentative Surgical Therapy of Peri-Implantitis—What Is Known and What Are the Future Challenges? Kristina Bertl 1,2* and Andreas Stavropoulos 1,3,4 1 Department of Periodontology, Faculty of Odontology, University of Malmö, Malmö, Sweden, 2 Division of Oral Surgery, University Clinic of Dentistry, Medical University of Vienna, Vienna, Austria, 3 Division of Regenerative Dental Medicine and Periodontology, University Clinics of Dental Medicine, University of Geneva, Geneva, Switzerland, 4 Division of Conservative Dentistry and Periodontology, University Clinic of Dentistry, Medical University of Vienna, Vienna, Austria Non-augmentative surgical therapy of peri-implantitis is indicated for cases with primarily horizontal bone loss or wide defects with limited potential for bone regeneration and/or re-osseointegration. This treatment approach includes a variety of different techniques (e.g., open flap debridement, resection of peri-implant mucosa, apically positioned flaps, bone re-contouring, implantoplasty, etc.) and various relevant aspects should be considered during treatment planning. The present mini review provides an overview on what is known for the following components of non-augmentative surgical treatment of Edited by: Priscila Casado, peri-implantitis and on potential future research challenges: (1) decontamination of the Fluminense Federal University, Brazil implant surface, (2) need of implantoplasty, (3) prescription of antibiotics, -
Denture Technology Curriculum Objectives
Health Licensing Agency 700 Summer St. NE, Suite 320 Salem, Oregon 97301-1287 Telephone (503) 378-8667 FAX (503) 585-9114 E-Mail: [email protected] Web Site: www.Oregon.gov/OHLA As of July 1, 2013 the Board of Denture Technology in collaboration with Oregon Students Assistance Commission and Department of Education has determined that 103 quarter hours or the equivalent semester or trimester hours is equivalent to an Associate’s Degree. A minimum number of credits must be obtained in the following course of study or educational areas: • Orofacial Anatomy a minimum of 2 credits; • Dental Histology and Embryology a minimum of 2 credits; • Pharmacology a minimum of 3 credits; • Emergency Care or Medical Emergencies a minimum of 1 credit; • Oral Pathology a minimum of 3 credits; • Pathology emphasizing in Periodontology a minimum of 2 credits; • Dental Materials a minimum of 5 credits; • Professional Ethics and Jurisprudence a minimum of 1 credit; • Geriatrics a minimum of 2 credits; • Microbiology and Infection Control a minimum of 4 credits; • Clinical Denture Technology a minimum of 16 credits which may be counted towards 1,000 hours supervised clinical practice in denture technology defined under OAR 331-405-0020(9); • Laboratory Denture Technology a minimum of 37 credits which may be counted towards 1,000 hours supervised clinical practice in denture technology defined under OAR 331-405-0020(9); • Nutrition a minimum of 4 credits; • General Anatomy and Physiology minimum of 8 credits; and • General education and electives a minimum of 13 credits. Curriculum objectives which correspond with the required course of study are listed below. -
Periodontal Surgery in Furcation-Involved Maxillary Molars Revisited—An Introduction of Guidelines for Comprehensive Treatment
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by RERO DOC Digital Library Clin Oral Invest (2011) 15:9–20 DOI 10.1007/s00784-010-0431-9 REVIEW Periodontal surgery in furcation-involved maxillary molars revisited—an introduction of guidelines for comprehensive treatment Clemens Walter & Roland Weiger & Nicola Ursula Zitzmann Received: 1 November 2009 /Accepted: 1 June 2010 /Published online: 23 June 2010 # Springer-Verlag 2010 Abstract Maxillary molars with interradicular loss of overview, including what constitutes accurate diagnosis and periodontal tissue have an increased risk of additional what indications there are for the different surgical attachment loss with an impaired long-term prognosis. periodontal treatment options. Since accurate clinical analysis of furcation involvement is not feasible due to limited access, morphological variations Keywords Furcation involvement . Furcation surgery. and measurement errors, additional diagnostics, e.g., with Diagnosis . Decision making . 3D imaging cone-beam computed tomography, may be required. Surgi- cal treatment options have graduated from a less invasive Abbreviations and acronyms approach, i.e., keeping as much periodontal attachment as FI Furcation involvement possible, to a more invasive approach: (1) open flap PPD Probing pocket depth debridement with/without gingivectomy or apically reposi- PAL Probing attachment level tioned flap and/or tunnelling; (2) root separation; (3) Sc&Rp Scaling and root planning amputation/trisection of a root (with/without root separation RCT Root canal treatment or tunnel preparation); (4) amputation/trisection of two SPT Supportive periodontal treatment roots; and (5) extraction of the entire tooth. Tunnelling is FDP Fixed dental prosthesis indicated when the degree of root separation allows for RDP Removable dental prosthesis opening of the interradicular region. -
The International Journal of Periodontics & Restorative Dentistry
The International Journal of Periodontics & Restorative Dentistry © 2013 BY QUINTESSENCE PUBLISHING CO, INC. PRINTING OF THIS DOCUMENT IS RESTRICTED TO PERSONAL USE ONLY. NO PART MAY BE REPRODUCED OR TRANSMITTED IN ANY FORM WITHOUT WRITTEN PERMISSION FROM THE PUBLISHER. 217 Open Flap Debridement in Combination with Acellular Dermal Matrix Allograft for the Prevention of Postsurgical Gingival Recession: A Case Series Ramesh Sundersing Chavan, BDS, MDS1 The therapeutic objective of peri- Manohar Laxman Bhongade, MSc, BDS, MDS2 odontal flap surgery is to provide Ishan Ramakant Tiwari, BDS, MDS1 accessibility to the underlying Priyanka Jaiswal, BDS, MDS1 root surface to reduce the pocket depth,1 arrest further breakdown, and prevent additional attach- Open flap debridement with flap repositioning may result in significant gingival ment loss. Open flap debridement recession. Patients with chronic periodontitis were treated with open flap 2 debridement followed by placement of an acellular dermal matrix allograft (OFD) is a common procedure for (ADMA) underneath the flap to minimize the occurrence of postsurgical gingival the treatment of deep periodontal recession. Ten patients (total, 60 teeth) with periodontal pockets in the anterior pockets associated with horizontal dentition underwent open flap debridement combined with ADMA. Probing bone loss. This procedure is indi- pocket depth, relative attachment level, and relative gingival margin level were cated when pocket elimination is recorded at baseline and 6 months postsurgery. The mean probing pocket undesirable because of esthetic depth at baseline and 6 months was 4.4 and 1.7 mm, respectively (P < .05); the mean relative attachment level at baseline and 6 months was 12.9 and 10.7 mm, considerations, particularly in the respectively (P < .05); and the mean relative gingival margin level at baseline and anterior dentition. -
Severe Gingival Swelling and Erythema
PHOTO CHALLENGE Severe Gingival Swelling and Erythema Mohammed Bindakhil, DDS; Thomas P. Sollecito, DMD; Eric T. Stoopler, DMD A 62-year-old man presented to an oral medi- cine specialist with gingival inflammation of at least 1 year’s duration. He reported mild discom- fort when consuming spicy foods and denied associated extraoral lesions. His medical history revealed hypertension, hypothyroidism,copy and pso- riasis. Medications included lisinopril 10 mg and levothyroxine 100 µg daily. No known drug aller- gies were reported. His family and social history were noncontributory, and a detailed review of systems was unremarkable. Extraoral examina- tion revealednot no lymphadenopathy, salivary gland enlargement, or thyromegaly. Intraoral examina- tion revealed diffuse enlargement of the maxillary and mandibular gingiva accompanied by severe erythema and bleeding on provocation. A 3-mm punch biopsy of the gingiva was performed for Doroutine analysis and direct immunofluorescence. WHAT’S YOUR DIAGNOSIS? a. extramedullary plasmacytoma b. mucous membrane pemphigoid c. oral lichen planus d. pemphigus vulgaris e. plasma cell gingivitis CUTIS PLEASE TURN TO PAGE E20 FOR THE DIAGNOSIS From the Department of Oral Medicine, University of Pennsylvania School of Dental Medicine, Philadelphia. The authors report no conflict of interest. Correspondence: Eric T. Stoopler, DMD, University of Pennsylvania School of Dental Medicine, 240 S 40th St, Philadelphia, PA 19104 ([email protected]). WWW.MDEDGE.COM/DERMATOLOGY VOL. 105 NO. 6 I JUNE 2020 E19 Copyright Cutis 2020. No part of this publication may be reproduced, stored, or transmitted without the prior written permission of the Publisher. PHOTO CHALLENGE DISCUSSION THE DIAGNOSIS: Plasma Cell Gingivitis icroscopic analysis demonstrated an acanthotic stratified squamous epithelium with an edema- Mtous fibrous stroma containing dense perivascular infiltrates of plasma cells and lymphocytes (Figure 1). -
Article Download
wjpls, 2018, Vol. 4, Issue 8, 182-184 Case Report ISSN 2454-2229 Gavali et al. World Journal of Pharmaceutical World Journaland Life of Pharmaceutical Sciences and Life Sciences WJPLS www.wjpls.org SJIF Impact Factor: 5.088 THE MODIFIED WIDMAN FLAP TECHNIQUE: A CASE REPORT Dr. Neelam Gavali*1, Dr. Pramod Waghmare2, Dr. Vishakha Patil3, Dr. Nilima Landge4 1Post Graduate Student, Department of Periodontology, Bharati Vidyapeeth (Deemed to be University) Dental College and Hospital- Pune. 2Professor, Department of Periodontology, Bharati Vidyapeeth (Deemed to be University) Dental College and Hospital- Pune. 3Professor, Department of Periodontology, Bharati Vidyapeeth (Deemed to be University) Dental College and Hospital- Pune. 4Associate Professor, Department of Periodontology, Bharati Vidyapeeth (Deemed to be University) Dental College and Hospital- Pune. *Corresponding Author: Dr. Neelam Gavali Post Graduate Student, Department of Periodontology, Bharati Vidyapeeth (Deemed to be University) Dental College and Hospital- Pune. Article Received on 24/05/2018 Article Revised on 14/06/2018 Article Accepted on 05/07/2018 ABSTRACT The Modified Widman flap (MWF), one of the most common and conservative surgical approaches that aims to eliminate the inflamed gingival tissue and also provide access for root debridement. It is classified with the “access flap operations” because the goal of the flap reflection is primarily to provide improved visual access to the periodontally involved tissues. The modified widman flap surgery is not aimed at surgical eradication of pocket walls, including bony walls. It is aimed at maximum healing in areas of previous periodontal pockets with minimum loss of periodontal tissues during and after the surgery.[6] KEYWORDS: Modified widman flap, Gingival enlargement. -
Surgical Management of Peri-Implantitis
Current Oral Health Reports (2020) 7:283–303 https://doi.org/10.1007/s40496-020-00278-y PERI-IMPLANTITIS (I DARBY, SECTION EDITOR) Surgical Management of Peri-implantitis Ausra Ramanauskaite1 & Karina Obreja1 & Frank Schwarz1 Published online: 1 August 2020 # The Author(s) 2020 Abstract Purpose of Review To provide an overview of current surgical peri-implantitis treatment options. Recent Findings Surgical procedures for peri-implantitis treatment include two main approaches: non-augmentative and aug- mentative therapy. Open flap debridement (OFD) and resective treatment are non-augmentative techniques that are indicated in the presence of horizontal bone loss in aesthetically nondemanding areas. Implantoplasty performed adjunctively at supracrestally and buccally exposed rough implant surfaces has been shown to efficiently attenuate soft tissue inflammation compared to control sites. However, this was followed by more pronounced soft tissue recession. Adjunctive augmentative measures are recommended at peri-implantitis sites exhibiting intrabony defects with a minimum depth of 3 mm and in the presence of keratinized mucosa. In more advanced cases with combined defect configurations, a combination of augmentative therapy and implantoplasty at exposed rough implant surfaces beyond the bony envelope is feasible. Summary For the time being, no particular surgical protocol or material can be considered as superior in terms of long-term peri- implant tissue stability. Keywords Peri-implantitis . Treatment . Surgical therapy Introduction of further bone loss [5]. To achieve these treatment endpoints, it is currently accepted that surgical approaches that allow ade- Peri-implantitis is a plaque-associated pathological condition quate access to the contaminated implant surface are required occurring around dental implants that results in a breakdown [6–8].