How Hippocampal Memory Shapes, and Is Shaped By, Attention
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Semantic Memory - Psychology - Oxford Bibliographies
1/16/2019 Semantic Memory - Psychology - Oxford Bibliographies Semantic Memory Michael N. Jones, Johnathan Avery LAST MODIFIED: 15 JANUARY 2019 DOI: 10.1093/OBO/97801998283400231 Introduction Semantic memory refers to our general world knowledge that encompasses memory for concepts, facts, and the meanings of words and other symbolic units that constitute formal communication systems such as language or math. In the classic hierarchical view of memory, declarative memory was subdivided into two independent modules: episodic memory, which is our autobiographical store of individual events, and semantic memory, which is our general store of abstracted knowledge. However, more recent theoretical accounts have greatly reduced the independence of these two memory systems, and episodic memory is typically viewed as a gateway to semantic memory accessed through the process of abstraction. Modern accounts view semantic memory as deeply rooted in sensorimotor experience, abstracted across many episodic memories to highlight the stable characteristics and mute the idiosyncratic ones. A great deal of research in neuroscience has focused on both how the brain creates semantic memories and what brain regions share the responsibility for storage and retrieval of semantic knowledge. These include many classic experiments that studied the behavior of individuals with brain damage and various types of semantic disorders but also more modern studies that employ neuroimaging techniques to study how the brain creates and stores semantic memories. Classically, semantic memory had been treated as a miscellaneous area of study for anything in declarative memory that was not clearly within the realm of episodic memory, and formal models of meaning in memory did not advance at the pace of models of episodic memory. -
Compare and Contrast Two Models Or Theories of One Cognitive Process with Reference to Research Studies
! The following sample is for the learning objective: Compare and contrast two models or theories of one cognitive process with reference to research studies. What is the question asking for? * A clear outline of two models of one cognitive process. The cognitive process may be memory, perception, decision-making, language or thinking. * Research is used to support the models as described. The research does not need to be outlined in a lot of detail, but underatanding of the role of research in supporting the models should be apparent.. * Both similarities and differences of the two models should be clearly outlined. Sample response The theory of memory is studied scientifically and several models have been developed to help The cognitive process describe and potentially explain how memory works. Two models that attempt to describe how (memory) and two models are memory works are the Multi-Store Model of Memory, developed by Atkinson & Shiffrin (1968), clearly identified. and the Working Memory Model of Memory, developed by Baddeley & Hitch (1974). The Multi-store model model explains that all memory is taken in through our senses; this is called sensory input. This information is enters our sensory memory, where if it is attended to, it will pass to short-term memory. If not attention is paid to it, it is displaced. Short-term memory Research. is limited in duration and capacity. According to Miller, STM can hold only 7 plus or minus 2 pieces of information. Short-term memory memory lasts for six to twelve seconds. When information in the short-term memory is rehearsed, it enters the long-term memory store in a process called “encoding.” When we recall information, it is retrieved from LTM and moved A satisfactory description of back into STM. -
The Baseline Structure of the Enteric Nervous System and Its Role in Parkinson’S Disease
life Review The Baseline Structure of the Enteric Nervous System and Its Role in Parkinson’s Disease Gianfranco Natale 1,2,* , Larisa Ryskalin 1 , Gabriele Morucci 1 , Gloria Lazzeri 1, Alessandro Frati 3,4 and Francesco Fornai 1,4 1 Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, 56126 Pisa, Italy; [email protected] (L.R.); [email protected] (G.M.); [email protected] (G.L.); [email protected] (F.F.) 2 Museum of Human Anatomy “Filippo Civinini”, University of Pisa, 56126 Pisa, Italy 3 Neurosurgery Division, Human Neurosciences Department, Sapienza University of Rome, 00135 Rome, Italy; [email protected] 4 Istituto di Ricovero e Cura a Carattere Scientifico (I.R.C.C.S.) Neuromed, 86077 Pozzilli, Italy * Correspondence: [email protected] Abstract: The gastrointestinal (GI) tract is provided with a peculiar nervous network, known as the enteric nervous system (ENS), which is dedicated to the fine control of digestive functions. This forms a complex network, which includes several types of neurons, as well as glial cells. Despite extensive studies, a comprehensive classification of these neurons is still lacking. The complexity of ENS is magnified by a multiple control of the central nervous system, and bidirectional communication between various central nervous areas and the gut occurs. This lends substance to the complexity of the microbiota–gut–brain axis, which represents the network governing homeostasis through nervous, endocrine, immune, and metabolic pathways. The present manuscript is dedicated to Citation: Natale, G.; Ryskalin, L.; identifying various neuronal cytotypes belonging to ENS in baseline conditions. -
Distance Learning Program Anatomy of the Human Brain/Sheep Brain Dissection
Distance Learning Program Anatomy of the Human Brain/Sheep Brain Dissection This guide is for middle and high school students participating in AIMS Anatomy of the Human Brain and Sheep Brain Dissections. Programs will be presented by an AIMS Anatomy Specialist. In this activity students will become more familiar with the anatomical structures of the human brain by observing, studying, and examining human specimens. The primary focus is on the anatomy, function, and pathology. Those students participating in Sheep Brain Dissections will have the opportunity to dissect and compare anatomical structures. At the end of this document, you will find anatomical diagrams, vocabulary review, and pre/post tests for your students. The following topics will be covered: 1. The neurons and supporting cells of the nervous system 2. Organization of the nervous system (the central and peripheral nervous systems) 4. Protective coverings of the brain 5. Brain Anatomy, including cerebral hemispheres, cerebellum and brain stem 6. Spinal Cord Anatomy 7. Cranial and spinal nerves Objectives: The student will be able to: 1. Define the selected terms associated with the human brain and spinal cord; 2. Identify the protective structures of the brain; 3. Identify the four lobes of the brain; 4. Explain the correlation between brain surface area, structure and brain function. 5. Discuss common neurological disorders and treatments. 6. Describe the effects of drug and alcohol on the brain. 7. Correctly label a diagram of the human brain National Science Education -
Mnemonics in a Mnutshell: 32 Aids to Psychiatric Diagnosis
Mnemonics in a mnutshell: 32 aids to psychiatric diagnosis Clever, irreverent, or amusing, a mnemonic you remember is a lifelong learning tool ® Dowden Health Media rom SIG: E CAPS to CAGE and WWHHHHIMPS, mnemonics help practitioners and trainees recall Fimportant lists (suchCopyright as criteriaFor for depression,personal use only screening questions for alcoholism, or life-threatening causes of delirium, respectively). Mnemonics’ effi cacy rests on the principle that grouped information is easi- er to remember than individual points of data. Not everyone loves mnemonics, but recollecting diagnostic criteria is useful in clinical practice and research, on board examinations, and for insurance reimbursement. Thus, tools that assist in recalling di- agnostic criteria have a role in psychiatric practice and IMAGES teaching. JUPITER In this article, we present 32 mnemonics to help cli- © nicians diagnose: • affective disorders (Box 1, page 28)1,2 Jason P. Caplan, MD Assistant clinical professor of psychiatry • anxiety disorders (Box 2, page 29)3-6 Creighton University School of Medicine 7,8 • medication adverse effects (Box 3, page 29) Omaha, NE • personality disorders (Box 4, page 30)9-11 Chief of psychiatry • addiction disorders (Box 5, page 32)12,13 St. Joseph’s Hospital and Medical Center Phoenix, AZ • causes of delirium (Box 6, page 32).14 We also discuss how mnemonics improve one’s Theodore A. Stern, MD Professor of psychiatry memory, based on the principles of learning theory. Harvard Medical School Chief, psychiatric consultation service Massachusetts General Hospital How mnemonics work Boston, MA A mnemonic—from the Greek word “mnemonikos” (“of memory”)—links new data with previously learned information. -
Post-Encoding Stress Does Not Enhance Memory Consolidation: the Role of Cortisol and Testosterone Reactivity
brain sciences Article Post-Encoding Stress Does Not Enhance Memory Consolidation: The Role of Cortisol and Testosterone Reactivity Vanesa Hidalgo 1,* , Carolina Villada 2 and Alicia Salvador 3 1 Department of Psychology and Sociology, Area of Psychobiology, University of Zaragoza, 44003 Teruel, Spain 2 Department of Psychology, Division of Health Sciences, University of Guanajuato, Leon 37670, Mexico; [email protected] 3 Department of Psychobiology and IDOCAL, University of Valencia, 46010 Valencia, Spain; [email protected] * Correspondence: [email protected]; Tel.: +34-978-645-346 Received: 11 October 2020; Accepted: 15 December 2020; Published: 16 December 2020 Abstract: In contrast to the large body of research on the effects of stress-induced cortisol on memory consolidation in young people, far less attention has been devoted to understanding the effects of stress-induced testosterone on this memory phase. This study examined the psychobiological (i.e., anxiety, cortisol, and testosterone) response to the Maastricht Acute Stress Test and its impact on free recall and recognition for emotional and neutral material. Thirty-seven healthy young men and women were exposed to a stress (MAST) or control task post-encoding, and 24 h later, they had to recall the material previously learned. Results indicated that the MAST increased anxiety and cortisol levels, but it did not significantly change the testosterone levels. Post-encoding MAST did not affect memory consolidation for emotional and neutral pictures. Interestingly, however, cortisol reactivity was negatively related to free recall for negative low-arousal pictures, whereas testosterone reactivity was positively related to free recall for negative-high arousal and total pictures. -
Types of Memory and Models of Memory
Inf1: Intro to Cognive Science Types of memory and models of memory Alyssa Alcorn, Helen Pain and Henry Thompson March 21, 2012 Intro to Cognitive Science 1 1. In the lecture today A review of short-term memory, and how much stuff fits in there anyway 1. Whether or not the number 7 is magic 2. Working memory 3. The Baddeley-Hitch model of memory hEp://www.cartoonstock.com/directory/s/ short_term_memory.asp March 21, 2012 Intro to Cognitive Science 2 2. Review of Short-Term Memory (STM) Short-term memory (STM) is responsible for storing small amounts of material over short periods of Nme A short Nme really means a SHORT Nme-- up to several seconds. Anything remembered for longer than this Nme is classified as long-term memory and involves different systems and processes. !!! Note that this is different that what we mean mean by short- term memory in everyday speech. If someone cannot remember what you told them five minutes ago, this is actually a problem with long-term memory. While much STM research discusses verbal or visuo-spaal informaon, the disNncNon of short vs. long-term applies to other types of sNmuli as well. 3/21/12 Intro to Cognitive Science 3 3. Memory span and magic numbers Amount of informaon varies with individual’s memory span = longest number of items (e.g. digits) that can be immediately repeated back in correct order. Classic research by George Miller (1956) described the apparent limits of short-term memory span in one of the most-cited papers in all of psychology. -
Glutamate Binding in Primate Brain
Journal of Neuroscience Research 27512-521 (1990) Quisqualate- and NMDA-Sensitive [3H]Glutamate Binding in Primate Brain A.B. Young, G.W. Dauth, Z. Hollingsworth, J.B. Penney, K. Kaatz, and S. Gilman Department of Neurology. University of Michigan, Ann Arbor Excitatory amino acids (EAA) such as glutamate and Young and Fagg, 1990). Because EAA are involved in aspartate are probably the neurotransmitters of a general cellular metabolic functions, they were not orig- majority of mammalian neurons. Only a few previous inally considered to be likcl y neurotransmitter candi- studies have been concerned with the distribution of dates. Electrophysiological studies demonstrated con- the subtypes of EAA receptor binding in the primate vincingly the potency of these substances as depolarizing brain. We examined NMDA- and quisqualate-sensi- agents and eventually discerned specific subtypes of tive [3H]glutamate binding using quantitative autora- EAA receptors that responded preferentially to EAA an- diography in monkey brain (Macaca fascicularis). alogs (Dingledine et al., 1988). Coincident with the elec- The two types of binding were differentially distrib- trophysiological studies, biochemical studies indicated uted. NMDA-sensitive binding was most dense in that EAA were released from slice and synaptosome dentate gyrus of hippocampus, stratum pyramidale preparations in a calcium-dependent fashion and were of hippocampus, and outer layers of cerebral cortex. accumulated in synaptosomc preparations by a high-af- Quisqualate-sensitive binding was most dense in den- finity transport system. With these methodologies, EAA tate gyrus of hippocampus, inner and outer layers of release and uptake were found to be selectively de- cerebral cortex, and molecular layer of cerebellum. -
The Absent Minded Consumer
The Absentminded Consumer John Ameriks, Andrew Caplin and John Leahy∗ March 2003 (Preliminary) Abstract We present a model of an absentminded consumer who does not keep constant track of his spending. The model generates a form of precautionary consumption, in which absentminded agents tend to consume more than attentive agents. We show that wealthy agents are more likely to be absentminded, whereas young and retired agents are more likely to pay attenition. The model presents new explanations for a relationship between spending and credit card use and for the decline in consumption at retirement. Key Words: JEL Classification: 1 Introduction Doyouknowhowmuchyouspentlastmonth,andwhatyouspentiton?Itis only if you answer this question in the affirmative that the classical life-cycle model of consumption applies to you. Otherwise, you are to some extent an absent-minded consumer. In this paper we develop a theory that applies to thoseofuswhofallintothiscategory. The concept of absentmindedness that we employ in our analysis was introduced by Rubinstein and Piccione [1997]. They define absentmindedness as the inability to distinguish between decision nodes that lie along the same branch of a decision tree. By identifying these distinct nodes with different ∗We would like to thank Mark Gertler, Per Krusell, and Ricardo Lagos for helpful comments. 1 levels of spending, we are able to model consumers who are uncertain as to how much they spend in any given period. The effect of this change in model structure is to make it difficult for consumers to equate the marginal utility of consumption with the marginal utility of wealth. We show that this innocent twist in a standard consumption model may not only provide insight into some existing puzzles in the consumption literature, but also shed light on otherwise puzzling results concerning linkages between financial planning and wealth accumulation (Ameriks, Caplin, and Leahy [2003]). -
Contributions of the Hippocampus and the Striatum to Simple Association and Frequency-Based Learning
www.elsevier.com/locate/ynimg NeuroImage 27 (2005) 291 – 298 Contributions of the hippocampus and the striatum to simple association and frequency-based learning Dima Amso,a,b,* Matthew C. Davidson,a Scott P. Johnson,b Gary Glover,c and B.J. Caseya aSackler Institute for Developmental Psychobiology, Weill Medical College of Cornell University, 1300 York Avenue, Box 140, New York, NY 10021, USA bDepartment of Psychology, New York University, NY 10003, USA cDepartment of Radiology, Stanford University, CA 94305, USA Received 8 September 2004; revised 30 January 2005; accepted 8 February 2005 Available online 8 April 2005 Using fMRI and a learning paradigm, this study examined the result from how often an event is encountered or how often it is independent contributions of the hippocampus and striatum to simple presented in a particular context or paired with other events. association and frequency-based learning. We scanned 10 right-handed Understanding the neural bases of these types of learning is the young adult subjects using a spiral in/out sequence on a GE 3.0 T objective of this study. scanner during performance of the learning paradigm. The paradigm A common measure of learning, used in both human infant and consisted of 2 cues that predicted each of 3 targets with varying nonhuman animal research, is response to novelty. The theoretical probabilities. Simultaneously, we varied the frequency with which each target was presented throughout the task, independent of cue framework (Sokolov, 1963) underlying these novelty-preference associations. Subjects had shorter response latencies to frequently paradigms is that attention is oriented more toward novel, relative occurring and highly associated target stimuli and longer response to familiar, stimuli. -
Handbook of Metamemory and Memory Evolution of Metacognition
This article was downloaded by: 10.3.98.104 On: 29 Sep 2021 Access details: subscription number Publisher: Routledge Informa Ltd Registered in England and Wales Registered Number: 1072954 Registered office: 5 Howick Place, London SW1P 1WG, UK Handbook of Metamemory and Memory John Dunlosky, Robert A. Bjork Evolution of Metacognition Publication details https://www.routledgehandbooks.com/doi/10.4324/9780203805503.ch3 Janet Metcalfe Published online on: 28 May 2008 How to cite :- Janet Metcalfe. 28 May 2008, Evolution of Metacognition from: Handbook of Metamemory and Memory Routledge Accessed on: 29 Sep 2021 https://www.routledgehandbooks.com/doi/10.4324/9780203805503.ch3 PLEASE SCROLL DOWN FOR DOCUMENT Full terms and conditions of use: https://www.routledgehandbooks.com/legal-notices/terms This Document PDF may be used for research, teaching and private study purposes. Any substantial or systematic reproductions, re-distribution, re-selling, loan or sub-licensing, systematic supply or distribution in any form to anyone is expressly forbidden. The publisher does not give any warranty express or implied or make any representation that the contents will be complete or accurate or up to date. The publisher shall not be liable for an loss, actions, claims, proceedings, demand or costs or damages whatsoever or howsoever caused arising directly or indirectly in connection with or arising out of the use of this material. Evolution of Metacognition Janet Metcalfe Introduction The importance of metacognition, in the evolution of human consciousness, has been emphasized by thinkers going back hundreds of years. While it is clear that people have metacognition, even when it is strictly defined as it is here, whether any other animals share this capability is the topic of this chapter. -
How Trauma Impacts Four Different Types of Memory
How Trauma Impacts Four Different Types of Memory EXPLICIT MEMORY IMPLICIT MEMORY SEMANTIC MEMORY EPISODIC MEMORY EMOTIONAL MEMORY PROCEDURAL MEMORY What It Is What It Is What It Is What It Is The memory of general knowledge and The autobiographical memory of an event The memory of the emotions you felt The memory of how to perform a facts. or experience – including the who, what, during an experience. common task without actively thinking and where. Example Example Example Example You remember what a bicycle is. You remember who was there and what When a wave of shame or anxiety grabs You can ride a bicycle automatically, with- street you were on when you fell off your you the next time you see your bicycle out having to stop and recall how it’s bicycle in front of a crowd. after the big fall. done. How Trauma Can Affect It How Trauma Can Affect It How Trauma Can Affect It How Trauma Can Affect It Trauma can prevent information (like Trauma can shutdown episodic memory After trauma, a person may get triggered Trauma can change patterns of words, images, sounds, etc.) from differ- and fragment the sequence of events. and experience painful emotions, often procedural memory. For example, a ent parts of the brain from combining to without context. person might tense up and unconsciously make a semantic memory. alter their posture, which could lead to pain or even numbness. Related Brain Area Related Brain Area Related Brain Area Related Brain Area The temporal lobe and inferior parietal The hippocampus is responsible for The amygdala plays a key role in The striatum is associated with producing cortex collect information from different creating and recalling episodic memory.