(IQI 25) Bilateral Cardiac Catheterization Rate July 2016
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Pub 100-04 Medicare Claims Processing Centers for Medicare & Medicaid Services (CMS) Transmittal 3054 Date: August 29, 2014 Change Request 8803
Department of Health & CMS Manual System Human Services (DHHS) Pub 100-04 Medicare Claims Processing Centers for Medicare & Medicaid Services (CMS) Transmittal 3054 Date: August 29, 2014 Change Request 8803 SUBJECT: Ventricular Assist Devices for Bridge-to-Transplant and Destination Therapy I. SUMMARY OF CHANGES: This Change Request (CR) is effective for claims with dates of service on and after October 30, 2013; contractors shall pay claims for Ventricular Assist Devices as destination therapy using the criteria in Pub. 100-03, part 1, section 20.9.1, and Pub. 100-04, Chapter 32, sec. 320. EFFECTIVE DATE: October 30, 2013 *Unless otherwise specified, the effective date is the date of service. IMPLEMENTATION DATE: September 30, 2014 Disclaimer for manual changes only: The revision date and transmittal number apply only to red italicized material. Any other material was previously published and remains unchanged. However, if this revision contains a table of contents, you will receive the new/revised information only, and not the entire table of contents. II. CHANGES IN MANUAL INSTRUCTIONS: (N/A if manual is not updated) R=REVISED, N=NEW, D=DELETED-Only One Per Row. R/N/D CHAPTER / SECTION / SUBSECTION / TITLE D 3/90.2.1/Artifiical Hearts and Related Devices R 32/Table of Contents N 32/320/Artificial Hearts and Related Devices N 32/320.1/Coding Requirements for Furnished Before May 1, 2008 N 32/320.2/Coding Requirements for Furnished After May 1, 2008 N 32/320.3/ Ventricular Assist Devices N 32/320.3.1/Postcardiotomy N 32/320.3.2/Bridge-To -Transplantation (BTT) N 32/320.3.3/Destination Therapy (DT) N 32/320.3.4/ Other N 32/320.4/ Replacement Accessories and Supplies for External Ventricular Assist Devices or Any Ventricular Assist Device (VAD) III. -
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Phenotype definition for the Vanderbilt Genome-Electronic Records project Identifying genetics determinants of normal QRS duration (QRSd) Patient population: • Patients with DNA whose first electrocardiogram (ECG) is designated as “normal” and lacking an exclusion criteria. • For this study, case and control are drawn from the same population and analyzed via continuous trait analysis. The only difference will be the QRSd. Hypothetical timeline for a single patient: Notes: • The study ECG is the first normal ECG. • The “Mildly abnormal” ECG cannot be abnormal by presence of heart disease. It can have abnormal rate, be recorded in the presence of Na-channel blocking meds, etc. For instance, a HR >100 is OK but not a bundle branch block. • Y duration = from first entry in the electronic medical record (EMR) until one month following normal ECG • Z duration = most recent clinic visit or problem list (if present) to one week following the normal ECG. Labs values, though, must be +/- 48h from the ECG time Criteria to be included in the analysis: Criteria Source/Method “Normal” ECG must be: • QRSd between 65-120ms ECG calculations • ECG designed as “NORMAL” ECG classification • Heart Rate between 50-100 ECG calculations • ECG Impression must not contain Natural Language Processing (NLP) on evidence of heart disease concepts (see ECG impression. Will exclude all but list below) negated terms (e.g., exclude those with possible, probable, or asserted bundle branch blocks). Should also exclude normalization negations like “LBBB no longer present.” -
A Case of Stenosis of Mitral and Tricuspid Valves in Pregnancy, Treated by Percutaneous Sequential Balloon Valvotomy
Case Report Annals of Clinical Medicine and Research Published: 30 Jun, 2020 A Case of Stenosis of Mitral and Tricuspid Valves in Pregnancy, Treated by Percutaneous Sequential Balloon Valvotomy Vipul Malpani, Mohan Nair*, Pritam Kitey, Amitabh Yaduvanshi, Vikas Kataria and Gautam Singal Department of Cardiology, Holy Family Hospital, New Delhi, India Abstract Rheumatic mitral stenosis is associated with other lesions, but combination of mitral stenosis and tricuspid stenosis is unusual. We are reporting a case of mitral and tricuspid stenosis in a pregnant lady that was successfully treated by sequential balloon valvuloplasty in a single sitting. Keywords: Mitral stenosis; Tricuspid stenosis; Balloon valvotomy Abbreviations MS: Mitral Stenosis; TS: Tricuspid Stenosis; BMV: Balloon Mitral Valvotomy; CMV: Closed Mitral Valvotomy; BTV: Balloon Tricuspid Valvotomy; PHT: Pressure Half Time; MVA: Mitral Valve Area; TVA: Tricuspid Valve Area; LA: Left Atrium; RA: Right Atrium; TR: Tricuspid Regurgitation Introduction Rheumatic Tricuspid valve Stenosis (TS) is rare, and it generally accompanies mitral valve disease [1]. TS is found in 15% cases of rheumatic heart disease but it is of clinical significance in only 5% cases [2]. Isolated TS accounts for about 2.4% of all cases of organic tricuspid valve disease and is mostly seen in young women [3,4]. Combined stenosis of mitral and tricuspid valves is extremely uncommon. Combined stenosis of both the valves has never been reported in pregnancy. Balloon Mitral Valvotomy (BMV) and surgical Closed Mitral Valvotomy (CMV) are two important OPEN ACCESS therapeutic options in the management of rheumatic mitral stenosis. Significant stenosis of the *Correspondence: tricuspid valve can also be treated by Balloon Tricuspid Valvotomy (BTV) [5,6]. -
Severe Tricuspid Valve Stenosis
Severe Tricuspid Valve Stenosis A Cause of Silent Mitral Stenosis Abdolhamid SHEIKHZADEH, M.D., Homayoon MOGHBELI, M.D., Parviz GHABUSSI, M.D., and Siavosh TARBIAT, M.D. SUMMARY The diastolic rumbling murmur of mitral stenosis (MS) may be attenuated in the presence of low cardiac output, right ventri- cular enlargement, Lutembacher's syndrome, pulmonary emphy- sema, and obesity. In this report we would like to stress that the presence of tricuspid stenosis (TS) is an additional significant cause of silent MS. The clinical material consisted of 73 patients with rheumatic TS who had undergone cardiac surgery. Five of these cases had clinical findings of TS without auscultatory findings of MS. They were found to have severe MS at the time of operation and to re- quire mitral valve surgery. At cardiac catheterization the mean diastolic gradient (MDG) across the mitral valve (MV) was less than 3mmHg and pulmonary arterial systolic pressure was 29- 42mmHg. The MDG across the tricuspid valve was 6-17mmHg. In conclusion, TS can mask clinical and hemodynamic find- ings of MS. The reason for this is the mechanical barrier imposed by TS proximal to the MV. Additional Indexing Words: Rheumatic valvular disease Atrial imprint Tricuspid valve surgery HE most common silent valvular lesion is that of mitral stenosis (MS).1) T The auscultatory findings of MS particularly the diastolic rumble, can be masked in patients with low cardiac output,2) severe pulmonary hyperten- sion right ventricular hypertrophy,3) Lutembacher's syndrome,4) pulmonary emphysema, and obesity.2) The purpose of this communication is to report another cause of true silent MS. -
Early Outcomes of Percutaneous Pulmonary Valve Implantation with Pulsta and Melody Valves: the First Report from Korea
Journal of Clinical Medicine Article Early Outcomes of Percutaneous Pulmonary Valve Implantation with Pulsta and Melody Valves: The First Report from Korea Ah Young Kim 1,2 , Jo Won Jung 1,2, Se Yong Jung 1,2 , Jae Il Shin 1,2 , Lucy Youngmin Eun 1,2 , Nam Kyun Kim 3 and Jae Young Choi 1,2,* 1 Division of Pediatric Cardiology, Center for Congenital Heart Disease, Severance Cardiovascular Hospital, Yonsei University College of Medicine, Seoul 03722, Korea; [email protected] (A.Y.K.); [email protected] (J.W.J.); [email protected] (S.Y.J.); [email protected] (J.I.S.); [email protected] (L.Y.E.) 2 Department of Pediatrics, Yonsei University College of Medicine, Seoul 03722, Korea 3 Department of Pediatrics, Emory University, Atlanta, GA 30322, USA; [email protected] * Correspondence: [email protected] Received: 25 July 2020; Accepted: 24 August 2020; Published: 26 August 2020 Abstract: Percutaneous pulmonary valve implantation (PPVI) is used to treat pulmonary stenosis (PS) or pulmonary regurgitation (PR). We described our experience with PPVI, specifically valve-in-valve transcatheter pulmonary valve replacement using the Melody valve and novel self-expandable systems using the Pulsta valve. We reviewed data from 42 patients undergoing PPVI. Twenty-nine patients had Melody valves in mostly bioprosthetic valves, valved conduits, and homografts in the pulmonary position. Following Melody valve implantation, the peak right ventricle-to-pulmonary artery gradient decreased from 51.3 11.5 to 16.7 3.3 mmHg and right ventricular systolic pressure ± ± fell from 70.0 16.8 to 41.3 17.8 mmHg. -
Building Blocks of Clinical Practice Helping Athletic Trainers Build a Strong Foundation Issue #7: Cardiac Assessment: Basic Cardiac Auscultation Part 2 of 2
Building Blocks of Clinical Practice Helping Athletic Trainers Build a Strong Foundation Issue #7: Cardiac Assessment: Basic Cardiac Auscultation Part 2 of 2 AUSCULTATION Indications for Cardiac Auscultation • History of syncope, dizziness • Chest pain, pressure or dyspnea during or after activity / exercise • Possible indication of hypertrophic cardiomyopathy • Sensations of heart palpitations • Tachycardia or bradycardia • Sustained hypertension and/or hypercholesterolemia • History of heart murmur or heart infection • Noted cyanosis • Trauma to the chest • Signs of Marfan’s syndrome * Enlarged or bulging aorta * Ectomorphic, scoliosis or kyphosis, pectus excavatum or pectus carinatum * Severe myopia Stethoscope • Diaphragm – best for hearing high pitched sounds • Bell – best for hearing low pitched sounds • Ideally, auscultate directly on skin, not over clothes Adult Rate • > 100 bpm = tachycardia • 60-100 bpm = normal (60-95 for children 6-12 years old) • < 60 bpm = bradycardia Rhythm • Regular • Irregular – regularly irregular or “irregularly” irregular Auscultation Sites / Valvular Positions (see part 1 of 2 for more information) • Aortic: 2nd right intercostal space • Pulmonic: 2nd left intercostal space • Tricuspid: 4th left intercostal space • Mitral: Apex, 5th intercostal space (mid-clavicular line) Auscultate at each valvular area with bell and diaphragm and assess the following: • Cardiac rhythm – regular or irregular • Heart sounds – note the quality • Murmurs – valvular locations • Extra-Cardiac Sounds – clicks, snaps and -
Cardiovascular Magnetic Resonance (CMR) Page 1 of 11
Cardiovascular Magnetic Resonance (CMR) Page 1 of 11 No review or update is scheduled on this Medical Policy as it is unlikely that further published literature would change the policy position. If there are questions about coverage of this service, please contact Blue Cross and Blue Shield of Kansas customer service, your professional or institutional relations representative, or submit a predetermination request. Medical Policy An independent licensee of the Blue Cross Blue Shield Association Title: Cardiovascular Magnetic Resonance (CMR) Professional Institutional Original Effective Date: August 4, 2005 Original Effective Date: July 1, 2006 Revision Date(s): February 27, 2006, Revision Date(s): May 2, 2007; May 2, 2007; November 1, 2007; November 1, 2007; January 1, 2010; January 1, 2010; February 15, 2013; February 15, 2013; December 11, 2013; December 11, 2013; April 15, 2014; April 15, 2014; July 15, 2014; June 10, 2015; July 15, 2014; June 10, 2015; June 8, 2016; June 8, 2016; October 1, 2016; October 1, 2016; May 10, 2017; May 10, 2017; April 25, 2018; April 25, 2018; October 1, 2018 October 1, 2018 Current Effective Date: July 15, 2014 Current Effective Date: July 15, 2014 Archived Date: July 3, 2019 Archived Date: July 3, 2019 State and Federal mandates and health plan member contract language, including specific provisions/exclusions, take precedence over Medical Policy and must be considered first in determining eligibility for coverage. To verify a member's benefits, contact Blue Cross and Blue Shield of Kansas Customer Service. The BCBSKS Medical Policies contained herein are for informational purposes and apply only to members who have health insurance through BCBSKS or who are covered by a self-insured group plan administered by BCBSKS. -
Surgical Management of Tricuspid Stenosis
Art of Operative Techniques Surgical management of tricuspid stenosis Marisa Cevasco, Prem S. Shekar Division of Cardiac Surgery, Brigham and Women’s Hospital, Boston, MA, USA Correspondence to: Prem S. Shekar, MD. Chief of Cardiac Surgery, 75 Francis Street, Boston, MA 02115, USA. Email: [email protected]. Tricuspid valve stenosis (TS) is rare, affecting less than 1% of patients in developed nations and approximately 3% of patients worldwide. Detection requires careful evaluation, as it is almost always associated with left-sided valve lesions that may obscure its significance. Primary TS is most frequently caused by rheumatic valvulitis. Other causes include carcinoid, radiation therapy, infective endocarditis, trauma from endomyocardial biopsy or pacemaker placement, or congenital abnormalities. Surgical management of TS is not commonly addressed in standard cardiac texts but is an important topic for the practicing surgeon. This paper will elucidate the anatomy, pathophysiology, and surgical management of TS. Keywords: Tricuspid stenosis (TS); tricuspid valve replacement (TVR) Submitted Feb 26, 2017. Accepted for publication Apr 21, 2017. doi: 10.21037/acs.2017.05.14 View this article at: http://dx.doi.org/10.21037/acs.2017.05.14 Introduction has also been accommodated in the design of annuloplasty rings, which have a gap in the ring in this area. Anatomy The anterior papillary muscle provides chordae to the The tricuspid valve consists of three leaflets (anterior, anterior and posterior leaflets, the posterior papillary muscle posterior, and septal), their associated chordae tendineae provides chordae to the posterior and septal leaflets, and the and papillary muscles, the fibrous tricuspid annulus, and the septal wall gives chordae to the anterior and septal leaflets. -
2010 Common Diagnosis Codes: Cardiology
22010010 CommonCommon DiagnoDiagnossisis CCodeodess:: CardiologyCardiology Code Description Code Description Code Description 038.9 Unspec septicemia 405.91 Unspec renovascular hypertension 425.8 Cardiomyopathy diseases classifi ed elsewhere 244.9 Unspec acquired hypothyroidism 410 AMI anterolateral wall episode care unspec 425.9 Secondary cardiomyopathy unspec 250 Diabetes mellitus w/o comp Type II not uncontrolled 410.01 initial episode care 426.0 Atrioventricular block complete 250.01 Diabetes mellitus w/o complication Type I not 410.02 AMI anterolateral wall subsequent episode care 426.10 Atrioventricular block unspec uncontrolled 410.1 AMI other anterial wall episode care unspec 426.11 First degree atrioventricular block 250.02 Diabetes mellitus w/o comp Type II uncontrolled 410.11 initial episode care 426.12 Mobitz (type) ii atrioventricular block 250.8 Type II diabetes mellitus w/ other spec 410.12 subsequent episode care 426.13 Other second degree atrioventricular block manifestations, not uncontrolled 410.21 AMI inferolarteral wall initial episode care 426.2 Left bundle branch hemiblock 250.9 Type II diabetes mellitus w/unspec complication, not 410.31 AMI inferoposterior wall initial episode of care 426.3 Other left bundle branch block uncontrolled 410.4 AMI other inferior wall episode care unspec 426.4 Right bundle branch block 272 Pure hypercholesterolemia 410.41 initial episode care 426.50 Bunble branch block unspec 272.1 Pure hyperglyceridemia 410.42 AMI other inferior wall subsequent episode care 426.52 Right bundle branch -
Vena Cava Backflow and Right Ventricular Stiffness in Pulmonary Arterial Hypertension
Early View Original article Vena cava backflow and right ventricular stiffness in pulmonary arterial hypertension J. Tim Marcus, Berend E. Westerhof, Joanne A. Groeneveldt, Harm Jan Bogaard, Frances S. de Man, Anton Vonk Noordegraaf Please cite this article as: Marcus JT, Westerhof BE, Groeneveldt JA, et al. Vena cava backflow and right ventricular stiffness in pulmonary arterial hypertension. Eur Respir J 2019; in press (https://doi.org/10.1183/13993003.00625-2019). This manuscript has recently been accepted for publication in the European Respiratory Journal. It is published here in its accepted form prior to copyediting and typesetting by our production team. After these production processes are complete and the authors have approved the resulting proofs, the article will move to the latest issue of the ERJ online. Copyright ©ERS 2019 Vena cava backflow and right ventricular stiffness in pulmonary arterial hypertension J. Tim Marcus1, Berend E. Westerhof2,3, Joanne A. Groeneveldt2, Harm Jan Bogaard2, Frances S. de Man2 and Anton Vonk Noordegraaf2 Affiliations: 1Amsterdam UMC, Vrije Universiteit Amsterdam, Radiology and Nuclear Medicine, Amsterdam Cardiovascular Sciences, Amsterdam, the Netherlands; 2Amsterdam UMC, Vrije Universiteit Amsterdam, Pulmonary Medicine, Amsterdam Cardiovascular Sciences, Amsterdam, the Netherlands; 3Amsterdam UMC, University of Amsterdam, Medical Biology, Section of Systems Physiology, Amsterdam Cardiovascular Sciences, Amsterdam, the Netherlands. Correspondence: Dr. J.T. Marcus Amsterdam UMC, Vrije Universiteit Amsterdam, Radiology and Nuclear Medicine, Amsterdam Cardiovascular Sciences, De Boelelaan 1117, Amsterdam Netherlands phone: +31 20 4440179 e-mail: [email protected] Abstract Vena cava (VC) backflow is a well-recognized clinical hallmark of right ventricular (RV) failure in pulmonary arterial hypertension (PAH). -
Primary Prevention of Pulmonary Heart Disease
Heart disease resources Primary prevention of pulmonary vented or diagnosed, and adequately treated, heart disease it must be recognized as a specific form of both acute and chronic heart disease. Such PULMONARY HEART DISEASE STUDY GROUP: recognition can be facilitated by the accep- Chairman: ROY H. BEHNKE, M.D. tance of a single definition of the disease Members: S. GILBERT BLOUNT, M.D.; entity. Pulmonary heart disease, cor pulmo- J. DAVID BRISTOW, M.D.; VIRGINIA CARRIERI, R.N.; JOHN A. PIERCE, M.D.; nale, is here defined as: Alteration in structure ARTHUR SASAHARA, M.D.; or function of the right ventricle resulting ALFRED SOFFER, M.D. from disease affecting the structure or func- Consultant: RICHARD GREENSPAN, M.D. tion of the lung or its vasculature, except when Introduction this alteration results from disease of the left Pulmonary heart disease, cor pulmonale, is side of the heart or congenital heart disease. a major form of heart disease yet there is reasonable evidence that it can be prevented. Incidence and prevalence Support for this contention rests in the fact Effective programs of prevention are facili- that the causative or precedent pulmonary tated by an accurate assessment of the scope diseases are in large part preventable. Primary and magnitude of the health problem. Chronic prevention of the related pulmonary diseases obstructive lung disease (bronchitis and/or is thus the initial and basic step in the pre- emphysema) is by far the most important of vention of cor pulmonale. Such an under- the respiratory diseases associated with pul- taking is a major task. It can only be achieved monary heart disease. -
2014 AHA/ACC Guideline for the Management of Patients With
Journal of the American College of Cardiology Vol. 63, No. 22, 2014 Ó 2014 by the American Heart Association, Inc., and the American College of Cardiology Foundation ISSN 0735-1097/$36.00 Published by Elsevier Inc. http://dx.doi.org/10.1016/j.jacc.2014.02.536 PRACTICE GUIDELINE 2014 AHA/ACC Guideline for the Management of Patients With Valvular Heart Disease A Report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines Developed in Collaboration With the American Association for Thoracic Surgery, American Society of Echocardiography, Society for Cardiovascular Angiography and Interventions, Society of Cardiovascular Anesthesiologists, and Society of Thoracic Surgeons Writing Rick A. Nishimura, MD, MACC, FAHA, Paul Sorajja, MD, FACC, FAHA# Committee Co-Chairy Thoralf M. Sundt III, MD***yy Members* Catherine M. Otto, MD, FACC, FAHA, James D. Thomas, MD, FASE, FACC, FAHAzz Co-Chairy *Writing committee members are required to recuse themselves from voting on sections to which their specific relationships with industry and Robert O. Bonow, MD, MACC, FAHAy other entities may apply; see Appendix 1 for recusal information. yACC/ y AHA representative. zACC/AHA Task Force on Performance Measures Blase A. Carabello, MD, FACC* x { z liaison. ACC/AHA Task Force on Practice Guidelines liaison. Society John P. Erwin III, MD, FACC, FAHA of Cardiovascular Anesthesiologists representative. #Society for Cardio- Robert A. Guyton, MD, FACC*x vascular Angiography and Interventions representative. **American ’ y Association for Thoracic Surgery representative. yySociety of Thoracic Patrick T. O Gara, MD, FACC, FAHA Surgeons representative. zzAmerican Society of Echocardiography Carlos E. Ruiz, MD, PHD, FACCy representative.