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The Primary Care Pediatrician and the Care of Children with Cleft Lip And/Or Cleft Palate Charlotte W
CLINICAL REPORT Guidance for the Clinician in Rendering Pediatric Care The Primary Care Pediatrician Charlotte W. Lewis, MD, MPH, FAAP, a Lisa S. Jacob, DDS, MS, b Christoph U. andLehmann, MD, the FAAP, FACMI, Care c SECTION ON ORALof HEALTH Children With Cleft Lip and/or Cleft Palate Orofacial clefts, specifically cleft lip and/or cleft palate (CL/P), are among the abstract most common congenital anomalies. CL/P vary in their location and severity and comprise 3 overarching groups: cleft lip (CL), cleft lip with cleft palate (CLP), and cleft palate alone (CP). CL/P may be associated with one of many syndromes that could further complicate a child’s needs. Care of patients aDivision of General Pediatrics and Hospital Medicine, Department of with CL/P spans prenatal diagnosis into adulthood. The appropriate timing Pediatrics, University of Washington School of Medicine and Seattle Children’s Hospital, Seattle, Washington; bGeorgetown Pediatric and order of specific cleft-related care are important factors for optimizing Dentistry and Orthodontics, Georgetown, Texas; and Departments of cBiomedical Informatics and Pediatrics, Vanderbilt University Medical outcomes; however, care should be individualized to meet the specific needs Center, Nashville, Tennessee of each patient and family. Children with CL/P should receive their specialty All three authors participated extensively in developing, researching, cleft-related care from a multidisciplinary cleft or craniofacial team with and writing the manuscript and revising it based on reviewers’ comments; Dr Lehmann made additional revisions after review by the sufficient patient and surgical volume to promote successful outcomes. board of directors. The primary care pediatrician at the child’s medical home has an essential This document is copyrighted and is property of the American role in making a timely diagnosis and referral; providing ongoing health Academy of Pediatrics and its Board of Directors. -
Glossary for Narrative Writing
Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper -
Soonerstart Automatic Qualifying Syndromes and Conditions 001
SoonerStart Automatic Qualifying Syndromes and Conditions 001 Abetalipoproteinemia 272.5 002 Acanthocytosis (see Abetalipoproteinemia) 272.5 003 Accutane, Fetal Effects of (see Fetal Retinoid Syndrome) 760.79 004 Acidemia, 2-Oxoglutaric 276.2 005 Acidemia, Glutaric I 277.8 006 Acidemia, Isovaleric 277.8 007 Acidemia, Methylmalonic 277.8 008 Acidemia, Propionic 277.8 009 Aciduria, 3-Methylglutaconic Type II 277.8 010 Aciduria, Argininosuccinic 270.6 011 Acoustic-Cervico-Oculo Syndrome (see Cervico-Oculo-Acoustic Syndrome) 759.89 012 Acrocephalopolysyndactyly Type II 759.89 013 Acrocephalosyndactyly Type I 755.55 014 Acrodysostosis 759.89 015 Acrofacial Dysostosis, Nager Type 756.0 016 Adams-Oliver Syndrome (see Limb and Scalp Defects, Adams-Oliver Type) 759.89 017 Adrenoleukodystrophy, Neonatal (see Cerebro-Hepato-Renal Syndrome) 759.89 018 Aglossia Congenita (see Hypoglossia-Hypodactylia) 759.89 019 Albinism, Ocular (includes Autosomal Recessive Type) 759.89 020 Albinism, Oculocutaneous, Brown Type (Type IV) 759.89 021 Albinism, Oculocutaneous, Tyrosinase Negative (Type IA) 759.89 022 Albinism, Oculocutaneous, Tyrosinase Positive (Type II) 759.89 023 Albinism, Oculocutaneous, Yellow Mutant (Type IB) 759.89 024 Albinism-Black Locks-Deafness 759.89 025 Albright Hereditary Osteodystrophy (see Parathyroid Hormone Resistance) 759.89 026 Alexander Disease 759.89 027 Alopecia - Mental Retardation 759.89 028 Alpers Disease 759.89 029 Alpha 1,4 - Glucosidase Deficiency (see Glycogenosis, Type IIA) 271.0 030 Alpha-L-Fucosidase Deficiency (see Fucosidosis) -
Non-Syndromic Occurrence of True Generalized Microdontia with Mandibular Mesiodens - a Rare Case Seema D Bargale* and Shital DP Kiran
Bargale and Kiran Head & Face Medicine 2011, 7:19 http://www.head-face-med.com/content/7/1/19 HEAD & FACE MEDICINE CASEREPORT Open Access Non-syndromic occurrence of true generalized microdontia with mandibular mesiodens - a rare case Seema D Bargale* and Shital DP Kiran Abstract Abnormalities in size of teeth and number of teeth are occasionally recorded in clinical cases. True generalized microdontia is rare case in which all the teeth are smaller than normal. Mesiodens is commonly located in maxilary central incisor region and uncommon in the mandible. In the present case a 12 year-old boy was healthy; normal in appearance and the medical history was noncontributory. The patient was examined and found to have permanent teeth that were smaller than those of the average adult teeth. The true generalized microdontia was accompanied by mandibular mesiodens. This is a unique case report of non-syndromic association of mandibular hyperdontia with true generalized microdontia. Keywords: Generalised microdontia, Hyperdontia, Permanent dentition, Mandibular supernumerary tooth Introduction [Ullrich-Turner syndrome], Chromosome 13[trisomy 13], Microdontia is a rare phenomenon. The term microdontia Rothmund-Thomson syndrome, Hallermann-Streiff, Oro- (microdentism, microdontism) is defined as the condition faciodigital syndrome (type 3), Oculo-mandibulo-facial of having abnormally small teeth [1]. According to Boyle, syndrome, Tricho-Rhino-Phalangeal, type1 Branchio- “in general microdontia, the teeth are small, the crowns oculo-facial syndrome. short, and normal contact areas between the teeth are fre- Supernumerary teeth are defined as any supplementary quently missing” [2] Shafer, Hine, and Levy [3] divided tooth or tooth substance in addition to usual configuration microdontia into three types: (1) Microdontia involving of twenty deciduous and thirty two permanent teeth [7]. -
Skeletal Malocclusion: a Developmental Disorder with a Life-Long Morbidity
Elmer ress Review J Clin Med Res. 2014;6(6):399-408 Skeletal Malocclusion: A Developmental Disorder With a Life-Long Morbidity Nishitha Joshia, Ahmad M. Hamdanb, Walid D. Fakhouric, d Abstract Keyword: Skeletal malocclusion; Micrognathia; Retrognathia; Prognathia; Late-onset diseases The likelihood of birth defects in orofacial tissues is high due to the structural and developmental complexity of the face and the sus- ceptibility to intrinsic and extrinsic perturbations. Skeletal maloc- clusion is caused by the distortion of the proper mandibular and/or Introduction maxillary growth during fetal development. Patients with skeletal malocclusion may suffer from dental deformities, bruxism, teeth Disorders of the head and face are very common birth de- crowding, trismus, mastication difficulties, breathing obstruction fects in all racial populations, and can appear as isolated phe- and digestion disturbance if the problem is left untreated. In this notype or as part of a syndrome. The prevalence of cranio- review, we focused on skeletal malocclusion that affects 27.9% of facial anomalies varies among different ethnicities based on the US population with different severity levels. We summarized genetic background, geography, socio-economical status and the prevalence of class I, II and III of malocclusion in different ethnic groups and discussed the most frequent medical disorders environmental factors. Because of the structural complexity associated with skeletal malocclusion. Dental anomalies that lead of the craniofacial region, variations in genetic and environ- to malocclusion such as tooth agenesis, crowding, missing teeth mental factors may have a profound effect on development, and abnormal tooth size are not addressed in this review. We pro- and could lead to congenital birth defects. -
Research Article
z Available online at http://www.journalcra.com INTERNATIONAL JOURNAL OF CURRENT RESEARCH International Journal of Current Research Vol. 10, Issue, 07, pp.71222-71228, July, 2018 ISSN: 0975-833X RESEARCH ARTICLE THE TONGUE SPEAKS A LOT OF HEALTH. 1,*Dr. Firdous Shaikh, 2Dr. Sonia Sodhi, 3Dr Zeenat Fatema Farooqui and 4Dr. Lata Kale 1PG Student, Department of Oral Medicine and Radiology, CSMSS Dental College and Hospital, Aurangabad 2Professor, Department of Oral Medicine and Radiology, CSMSS Dental College and Hospital, Aurangabad 3Fatema Farooqui, Chief Medical Officer, Sri Ram Homeopathic Clinic and Research Center, Solapur 4Professor and Head, Department of Oral Medicine and Radiology, CSMSS Dental College and Hospital, Aurangabad ARTICLE INFO ABSTRACT Article History: Multifunctional organ of the human body without a bone yet strong is the tongue. It mainly consists Received 26th April, 2018 of the functional portion of muscle mass, mucosa, fat and the specialized tissue of taste i.e. the Received in revised form papillae. Diseases may either result from internal/ systemic causes of extrinsic causes like trauma, 14th May, 2018 infection, etc. A new method for classification has been proposed in this review for diseases of Accepted 09th June, 2018 tongue. This review mainly focuses on encompassing almost each aspect that the body reflects via its th Published online 30 July, 2018 mirror in mouth, the tongue. Key Words: Tongue, Diseases of Tongue, Discoloration of Tongue, Oral health, Hairy Tongue. Copyright © 2018, Firdous Shaikh et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. -
Psykisk Utviklingshemming Og Forsinket Utvikling
Psykisk utviklingshemming og forsinket utvikling Genpanel, versjon v03 Tabellen er sortert på gennavn (HGNC gensymbol) Navn på gen er iht. HGNC >x10 Andel av genet som har blitt lest med tilfredstillende kvalitet flere enn 10 ganger under sekvensering x10 er forventet dekning; faktisk dekning vil variere. Gen Gen (HGNC Transkript >10x Fenotype (symbol) ID) AAAS 13666 NM_015665.5 100% Achalasia-addisonianism-alacrimia syndrome OMIM AARS 20 NM_001605.2 100% Charcot-Marie-Tooth disease, axonal, type 2N OMIM Epileptic encephalopathy, early infantile, 29 OMIM AASS 17366 NM_005763.3 100% Hyperlysinemia OMIM Saccharopinuria OMIM ABCB11 42 NM_003742.2 100% Cholestasis, benign recurrent intrahepatic, 2 OMIM Cholestasis, progressive familial intrahepatic 2 OMIM ABCB7 48 NM_004299.5 100% Anemia, sideroblastic, with ataxia OMIM ABCC6 57 NM_001171.5 93% Arterial calcification, generalized, of infancy, 2 OMIM Pseudoxanthoma elasticum OMIM Pseudoxanthoma elasticum, forme fruste OMIM ABCC9 60 NM_005691.3 100% Hypertrichotic osteochondrodysplasia OMIM ABCD1 61 NM_000033.3 77% Adrenoleukodystrophy OMIM Adrenomyeloneuropathy, adult OMIM ABCD4 68 NM_005050.3 100% Methylmalonic aciduria and homocystinuria, cblJ type OMIM ABHD5 21396 NM_016006.4 100% Chanarin-Dorfman syndrome OMIM ACAD9 21497 NM_014049.4 99% Mitochondrial complex I deficiency due to ACAD9 deficiency OMIM ACADM 89 NM_000016.5 100% Acyl-CoA dehydrogenase, medium chain, deficiency of OMIM ACADS 90 NM_000017.3 100% Acyl-CoA dehydrogenase, short-chain, deficiency of OMIM ACADVL 92 NM_000018.3 100% VLCAD -
Oral Health Abnormalities in Children Born with Macrosomia Established During Mixed Dentition Period
© Wydawnictwo Aluna Wiadomości Lekarskie 2019, tom LXXII, nr 5 cz I PRACA ORYGINALNA ORIGINAL ARTICLE ORAL HEALTH ABNORMALITIES IN CHILDREN BORN WITH MACROSOMIA ESTABLISHED DURING MIXED DENTITION PERIOD Olga V. Garmash KHARKIV NATIONAL MEDICAL UNIVERSITY, KHARKIV, UKRAINE ABSTRACT Introduction: The prevalence of soft tissue and hard tooth tissuediseases in the oral cavity and the morphofunctional disorders of craniofacial complex, require attention ofspecialistsin various branches of medicine. Scientists began to pay attention to metabolic and other violations that have occurred in the fetal development and led to the occurrence of certain changes in the dental status of the child. The aim of thisresearch is to study the features of the dental health condition in the children of Northeast of Ukraine, who were born with macrosomia during the period of mixed dentition. The study takes into account intrauterine body length growth acceleration, intrauterine obesity or well-balanced acceleration of both the body weight and length gain. Materials and methods: Thirty 6.5–11-year-old children with fetal macrosomia were examined (MainGroup). A Comparison Group was comprised of sixteen children, whose weight-height parameters at birth were normal (fetal normosomia). All children in the Main group were split into four subgroups in accordance with weight-height parameters at birth using the V. I. Grischenko and his co-authors’ harmonious coefficient. The evaluation of the hygiene status of the oral cavity, the dental caries intensity evaluation, and the quantitative analysis of minor salivary gland secretion have been performed. The prevalence of dentoalveolar abnormalities was evaluated. Results: The highest values of caries intensity were recorded in macrosomic-at-birth children born with harmonious (well-balanced) intrauterine development, with intrauterine obesity and increased body length, or with intrauterine obesity and an average body length. -
Lieshout Van Lieshout, M.J.S
EXPLORING ROBIN SEQUENCE Manouk van Lieshout Van Lieshout, M.J.S. ‘Exploring Robin Sequence’ Cover design: Iliana Boshoven-Gkini - www.agilecolor.com Thesis layout and printing by: Ridderprint BV - www.ridderprint.nl ISBN: 978-94-6299-693-9 Printing of this thesis has been financially supported by the Erasmus University Rotterdam. Copyright © M.J.S. van Lieshout, 2017, Rotterdam, the Netherlands All rights reserved. No parts of this thesis may be reproduced, stored in a retrieval system, or transmitted in any form or by any means without permission of the author or when appropriate, the corresponding journals Exploring Robin Sequence Verkenning van Robin Sequentie Proefschrift ter verkrijging van de graad van doctor aan de Erasmus Universiteit Rotterdam op gezag van de rector magnificus Prof.dr. H.A.P. Pols en volgens besluit van het College voor Promoties. De openbare verdediging zal plaatsvinden op woensdag 20 september 2017 om 09.30 uur door Manouk Ji Sook van Lieshout geboren te Seoul, Korea PROMOTIECOMMISSIE Promotoren: Prof.dr. E.B. Wolvius Prof.dr. I.M.J. Mathijssen Overige leden: Prof.dr. J.de Lange Prof.dr. M. De Hoog Prof.dr. R.J. Baatenburg de Jong Copromotoren: Dr. K.F.M. Joosten Dr. M.J. Koudstaal TABLE OF CONTENTS INTRODUCTION Chapter I: General introduction 9 Chapter II: Robin Sequence, A European survey on current 37 practice patterns Chapter III: Non-surgical and surgical interventions for airway 55 obstruction in children with Robin Sequence AIRWAY OBSTRUCTION Chapter IV: Unravelling Robin Sequence: Considerations 79 of diagnosis and treatment Chapter V: Management and outcomes of obstructive sleep 95 apnea in children with Robin Sequence, a cross-sectional study Chapter VI: Respiratory distress following palatal closure 111 in children with Robin Sequence QUALITY OF LIFE Chapter VII: Quality of life in children with Robin Sequence 129 GENERAL DISCUSSION AND SUMMARY Chapter VIII: General discussion 149 Chapter IX: Summary / Nederlandse samenvatting 169 APPENDICES About the author 181 List of publications 183 Ph.D. -
Treatments for Ankyloglossia and Ankyloglossia with Concomitant Lip-Tie Comparative Effectiveness Review Number 149
Comparative Effectiveness Review Number 149 Treatments for Ankyloglossia and Ankyloglossia With Concomitant Lip-Tie Comparative Effectiveness Review Number 149 Treatments for Ankyloglossia and Ankyloglossia With Concomitant Lip-Tie Prepared for: Agency for Healthcare Research and Quality U.S. Department of Health and Human Services 540 Gaither Road Rockville, MD 20850 www.ahrq.gov Contract No. 290-2012-00009-I Prepared by: Vanderbilt Evidence-based Practice Center Nashville, TN Investigators: David O. Francis, M.D., M.S. Sivakumar Chinnadurai, M.D., M.P.H. Anna Morad, M.D. Richard A. Epstein, Ph.D., M.P.H. Sahar Kohanim, M.D. Shanthi Krishnaswami, M.B.B.S., M.P.H. Nila A. Sathe, M.A., M.L.I.S. Melissa L. McPheeters, Ph.D., M.P.H. AHRQ Publication No. 15-EHC011-EF May 2015 This report is based on research conducted by the Vanderbilt Evidence-based Practice Center (EPC) under contract to the Agency for Healthcare Research and Quality (AHRQ), Rockville, MD (Contract No. 290-2012-00009-I). The findings and conclusions in this document are those of the authors, who are responsible for its contents; the findings and conclusions do not necessarily represent the views of AHRQ. Therefore, no statement in this report should be construed as an official position of AHRQ or of the U.S. Department of Health and Human Services. The information in this report is intended to help health care decisionmakers—patients and clinicians, health system leaders, and policymakers, among others—make well-informed decisions and thereby improve the quality of health care services. This report is not intended to be a substitute for the application of clinical judgment. -
Plastic Surgery Essentials for Students Handbook to All Third Year Medical Students Concerned with the Effect of the Outcome on the Entire Patient
AMERICAN SOCIETY OF PLASTIC SURGEONS YOUNG PLASTIC SURGEONS STEERING COMMITTEE Lynn Jeffers, MD, Chair C. Bob Basu, MD, Vice Chair Eighth Edition 2012 Essentials for Students Workgroup Lynn Jeffers, MD Adam Ravin, MD Sami Khan, MD Chad Tattini, MD Patrick Garvey, MD Hatem Abou-Sayed, MD Raman Mahabir, MD Alexander Spiess, MD Howard Wang, MD Robert Whitfield, MD Andrew Chen, MD Anureet Bajaj, MD Chris Zochowski, MD UNDERGRADUATE EDUCATION COMMITTEE OF THE PLASTIC SURGERY EDUCATIONAL FOUNDATION First Edition 1979 Ruedi P. Gingrass, MD, Chairman Martin C. Robson, MD Lewis W.Thompson, MD John E.Woods, MD Elvin G. Zook, MD Copyright © 2012 by the American Society of Plastic Surgeons 444 East Algonquin Road Arlington Heights, IL 60005 All rights reserved. Printed in the United States of America ISBN 978-0-9859672-0-8 i INTRODUCTION PREFACE This book has been written primarily for medical students, with constant attention to the thought, A CAREER IN PLASTIC SURGERY “Is this something a student should know when he or she finishes medical school?” It is not designed to be a comprehensive text, but rather an outline that can be read in the limited time Originally derived from the Greek “plastikos” meaning to mold and reshape, plastic surgery is a available in a burgeoning curriculum. It is designed to be read from beginning to end. Plastic specialty which adapts surgical principles and thought processes to the unique needs of each surgery had its beginning thousands of years ago, when clever surgeons in India reconstructed individual patient by remolding, reshaping and manipulating bone, cartilage and all soft tissues. -
2016-Chapter-143-Oropharyngeal-Growth-And-Malformations-PPSM-6E-1.Pdf
To protect the rights of the author(s) and publisher we inform you that this PDF is an uncorrected proof for internal business use only by the author(s), editor(s), reviewer(s), Elsevier and typesetter Toppan Best-set. It is not allowed to publish this proof online or in print. This proof copy is the copyright property of the publisher and is confidential until formal publication. Chapter c00143 Oropharyngeal Growth and Skeletal Malformations 143 Stacey Dagmar Quo; Benjamin T. Pliska; Nelly Huynh Chapter Highlights p0010 • Sleep-disordered breathing (SDB) is marked by manifestations has not been determined. The varying degrees of collapsibility of the length and volume of the airway increase until pharyngeal airway. The hard tissue boundaries the age of 20 years, at which time there is a of the airway dictate the size and therefore the variable period of stability, followed by a slow responsiveness of the muscles that form this decrease in airway size after the fifth decade of part of the upper airway. Thus, the airway is life. The possibility of addressing the early forms shaped not only by the performance of the of this disease with the notions of intervention pharyngeal muscles to stimulation but also by and prevention can change the landscape the surrounding skeletal framework. of care. u0015 • The upper and lower jaws are key components • Correction of specific skeletal anatomic u0025 of the craniofacial skeleton and the deficiencies can improve or eliminate SDB determinants of the anterior wall of the upper symptoms in both children and adults. It is airway. The morphology of the jaws can be possible that the clinician may adapt or modify negatively altered by dysfunction of the upper the growth expression, although the extent of airway during growth and development.