The Refractory VF Arrest Patient: a Review of the Current Treatment
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Cardiogenetics 2017; volume 7:6304 Sudden death in a young patient with atrial fibrillation Case Report Correspondence: María Angeles Espinosa Castro, Inherited Cardiovascular Disease A 22-year-old man suffered a sudden Program, Cardiology Department, Gregorio María Tamargo, cardiac arrest without previous symptoms Marañón Hospital, Dr. Esquerdo, 46, 28007, María Ángeles Espinosa, while he was at rest, waiting for a subway Madrid, Spain. Víctor Gómez-Carrillo, Miriam Juárez, train. Cardiopulmonary resuscitation was Tel.: +34.91.586.82.90. immediately started using an Automated E-mail: [email protected] Francisco Fernández-Avilés, External Defibrillation that identified the Raquel Yotti Key words: KCNQ1; mutation; channelopa- presence of ventricular fibrillation and thy; sudden cardiac death; atrial fibrillation. Inherited Cardiovascular Disease delivered a shock. Return of spontaneous Program, Cardiology Department, circulation was achieved after three Contributions: MT, acquisition and interpreta- Gregorio Marañón Hospital, Madrid, attempts, being atrial fibrillation (AF) the tion of data for the work, ensuring that ques- Spain patient’s rhythm at this point (Figure 1). tions related to the accuracy or integrity of any He was admitted to our Cardiovascular part of the work is appropriately investigated Intensive Care Unit and therapeutic and resolved; MAE, conception of the work, hypothermia was performed over a period critical revision of the intellectual content, final approval of the version to be published, Abstract of 24 h. After completing hypothermia, ensuring that questions related to the accuracy rewarming, and another 24 h of controlled of any part of the work is appropriately inves- Sudden cardiac death (SCD) in young normothermia the patient awakened with no tigated and resolved; VG-C, acquisition and patients without structural heart disease is residual neurologic damage. -
Public Access Defibrillation/AED Program Implementation Guide
Public Access Defibrillation/AED Program Implementation Guide There are 4 key elements to establishing a Public Access Defibrillation program. These include: Having designated rescuers trained in CPR and in how to use an automated external defibrillator (AED) Having a physician to provide medical oversight and direction Integrating your program with the local emergency medical services (EMS) system Using and maintaining the AED(s) according to the manufacturers specifications Steps: 1. Gain consensus Within your company or organization begin to identify the key decision makers and arrange a meeting to gain support. The American Heart Association or your local EMS Agency can assist you with presentation materials. 2. Review the law and regulations Review Federal, State and local laws and regulations regarding Public Access Defibrillation program requirements. Consult your local Emergency Medical Services (EMS) Agency The California laws ang regulations that pertain to AEDs are Title 22, and AB 658. 3. Consult your local Emergency Medical Services (EMS) Agency The local EMS Agency can provide you with information regarding training, purchasing AEDs, medical direction and laws and regulations. 4. Identify your response team Identify who would be most likely to respond in an emergency – this will help determine how and where AEDs are mounted or stored. 5. Select equipment and vendor Some considerations in selecting the AED may include: Reputation of the AED manufacturer for the product’s quality and customer service, compatibility with the equipment used in the local EMS system, and ease of operation of the AED. 6. Design Policies and Procedures These may include: Who manages the AED program When the AED should be used, when it should not be used Training required to use the AED Locations of AEDs and other equipment (such as gloves and pocket mask for CPR) Notification process for internal AED responders and external emergency medical services responders Maintenance schedule for equipment Training and refresher training policies 7. -
Atrial Arrhythmia Triggering Electromechanical Dissociation And
EP CASE REPORT ....................................................................................................................................................... Atrial arrhythmia triggering electromechanical dissociation and ventricular fibrillation in a patient with atrial switch operation Nicolas Combes1,2,3*, Stefano Bartoletti1,2,Se´bastien Hascoet€ 3, Olivier Vahdat2, Franc¸ois Heitz2, and Victor Waldmann 4,5 1Electrophysiology Unit, Clinique Pasteur, Toulouse, France; 2Pediatric and Adult Congenital Heart Disease Department, Clinique Pasteur, 45, Avenue de Lombez, 31076 Toulouse, France; 3Pediatric and Adult Congenital Heart Disease Department, Hoˆpital Marie Lannelongue, Le Plessis-Robinson, France; 4Cardiology Department, Electrophysiology Unit, European Georges Pompidou Hospital, Paris, France; and 5Cardiology Department, Adult Congenital Heart Disease Unit, European Georges Pompidou Hospital, Paris, France * Corresponding author. Tel: 133 562213131; fax: 133 562211641. E-mail address: [email protected] A 26-year-old man with D- transposition of the great arteries (D-TGA) and pre- vious Mustard atrial switch surgery was referred for catheter ablation of a recur- rent symptomatic paroxys- mal atrial flutter. The arrhythmia was easily induci- ble (Figure 1A, cycle length 310 ms, rate 194 b.p.m.), with rapid conduction to the ventricles. While mapping flutter, simultaneous record- ing of endocardial signals and invasive blood pressure monitoring showed haemo- dynamic deterioration with intermittent electromechan- ical dissociation (Figure 1B) and then pulseless electrical activity (Figure 1C). After ini- tiation of cardiopulmonary resuscitation, several electric shocks failed to restore sinus rhythm and atrial flutter transitioned a few minutes later into ventricular tachy- cardia and then ventricular fibrillation (Figure 1D); this was ultimately terminated by defibrillation after an Figure 1 (A) Atrial flutter on 12-lead ECG induced by atrial bursts (240 ms), average ventricular response adrenaline bolus. -
Mitral Valve Prolapse, Arrhythmias, and Sudden Cardiac Death: the Role of Multimodality Imaging to Detect High-Risk Features
diagnostics Review Mitral Valve Prolapse, Arrhythmias, and Sudden Cardiac Death: The Role of Multimodality Imaging to Detect High-Risk Features Anna Giulia Pavon 1,2,*, Pierre Monney 1,2,3 and Juerg Schwitter 1,2,3 1 Cardiac MR Center (CRMC), Lausanne University Hospital (CHUV), 1100 Lausanne, Switzerland; [email protected] (P.M.); [email protected] (J.S.) 2 Cardiovascular Department, Division of Cardiology, Lausanne University Hospital (CHUV), 1100 Lausanne, Switzerland 3 Faculty of Biology and Medicine, University of Lausanne (UniL), 1100 Lausanne, Switzerland * Correspondence: [email protected]; Tel.: +41-775-566-983 Abstract: Mitral valve prolapse (MVP) was first described in the 1960s, and it is usually a benign condition. However, a subtype of patients are known to have a higher incidence of ventricular arrhythmias and sudden cardiac death, the so called “arrhythmic MVP.” In recent years, several studies have been published to identify the most important clinical features to distinguish the benign form from the potentially lethal one in order to personalize patient’s treatment and follow-up. In this review, we specifically focused on red flags for increased arrhythmic risk to whom the cardiologist must be aware of while performing a cardiovascular imaging evaluation in patients with MVP. Keywords: mitral valve prolapse; arrhythmias; cardiovascular magnetic resonance Citation: Pavon, A.G.; Monney, P.; Schwitter, J. Mitral Valve Prolapse, Arrhythmias, and Sudden Cardiac Death: The Role of Multimodality 1. Mitral Valve and Arrhythmias: A Long Story Short Imaging to Detect High-Risk Features. In the recent years, the scientific community has begun to pay increasing attention Diagnostics 2021, 11, 683. -
Cardiac Arrest and Ventricular Fibrillation * a Method of Treatment by Electrical Shock
Thorax: first published as 10.1136/thx.7.3.205 on 1 September 1952. Downloaded from Thorax (1952), 7, 205. CARDIAC ARREST AND VENTRICULAR FIBRILLATION * A METHOD OF TREATMENT BY ELECTRICAL SHOCK BY I. K. R. MCMILLANt F. B. COCKETT, AND P. STYLES Froml the Departnment of Cardiology, tile Professorial Surgical Unit, and tile Departalzent of Phlysical Medicine, St. Thomlas's Hospital, London (RECEIVED FOR PUBLICATION APRIL 30, 1952) Cardiac arrest during operation is a subject of VENTRICULAR FIBRILLATION importance to all surgeons. During intra-thoracic The treatment of ventricular fibrillation bv operations cardiac arrest and ventricular fibrilla- electrical shock therapy is not new and was first tion can be differentiated by direct observation of tried experimentally in 1899 (Prevost and Battelli). the heart. In recent years this method has been extensively Ventricular fibrillation is an incoordinated type investigated in the United States and France of contraction which produces no useful beats. (Kouwenhoven and Kay, 1951; Johnson and The typical rippling movements of the ventricular Kirby, 1951 ; Santy and Marion, 1950). muscle are unmistakable when seen or felt with The treatment of ventricular fibrillation by the hand directly on the heart. The treatment of zlectrical shock gives encouraging results in the copyright. a heart that has stopped is cardiac massage and experimental animal, and may be a useful adjunct adequate oxygenation. The treatment of ventri- in the operating theatre during cardiac and general cular fibrillation which we recommend is, first, surgery. Under these conditions shock therapy massage to maintain the oxygen supply to the can be rapidly applied. Provided that adequate heart through the coronary arteries, followed oxygenation is maintained and direct cardiac mas- http://thorax.bmj.com/ rapidly by electrical shocking to restore normal sage promptly initiated and maintained, shock rhythm therapy can be applied without undue haste. -
Delaying Defibrillation to Give Basic Cardiopulmonary Resuscitation to Patients with Out-Of-Hospital Ventricular Fibrillation a Randomized Trial
ORIGINAL CONTRIBUTION Delaying Defibrillation to Give Basic Cardiopulmonary Resuscitation to Patients With Out-of-Hospital Ventricular Fibrillation A Randomized Trial Lars Wik, MD, PhD Context Defibrillation as soon as possible is standard treatment for patients with ven- Trond Boye Hansen tricular fibrillation. A nonrandomized study indicates that after a few minutes of ven- Frode Fylling tricular fibrillation, delaying defibrillation to give cardiopulmonary resuscitation (CPR) first might improve the outcome. Thorbjørn Steen, MD Objective To determine the effects of CPR before defibrillation on outcome in pa- Per Vaagenes, MD, PhD tients with ventricular fibrillation and with response times either up to or longer than Bjørn H. Auestad, PhD 5 minutes. Design, Setting, and Patients Randomized trial of 200 patients with out-of- Petter Andreas Steen, MD, PhD hospital ventricular fibrillation in Oslo, Norway, between June 1998 and May 2001. ARLY DEFIBRILLATION IS CRITICAL Patients received either standard care with immediate defibrillation (n=96) or CPR first for survival from ventricular fi- with 3 minutes of basic CPR by ambulance personnel prior to defibrillation (n=104). If initial defibrillation was unsuccessful, the standard group received 1 minute of CPR brillation. The survival rate de- before additional defibrillation attempts compared with 3 minutes in the CPR first group. creases by 3% to 4% or 6% to E10% per minute depending on whether Main Outcome Measure Primary end point was survival to hospital discharge. Sec- ondary end points were hospital admission with return of spontaneous circulation (ROSC), basic cardiopulmonary resuscitation 1,2 1-year survival, and neurological outcome. A prespecified analysis examined sub- (CPR) is performed. -
Basic Rhythm Recognition
Electrocardiographic Interpretation Basic Rhythm Recognition William Brady, MD Department of Emergency Medicine Cardiac Rhythms Anatomy of a Rhythm Strip A Review of the Electrical System Intrinsic Pacemakers Cells These cells have property known as “Automaticity”— means they can spontaneously depolarize. Sinus Node Primary pacemaker Fires at a rate of 60-100 bpm AV Junction Fires at a rate of 40-60 bpm Ventricular (Purkinje Fibers) Less than 40 bpm What’s Normal P Wave Atrial Depolarization PR Interval (Normal 0.12-0.20) Beginning of the P to onset of QRS QRS Ventricular Depolarization QRS Interval (Normal <0.10) Period (or length of time) it takes for the ventricles to depolarize The Key to Success… …A systematic approach! Rate Rhythm P Waves PR Interval P and QRS Correlation QRS Rate Pacemaker A rather ill patient……… Very apparent inferolateral STEMI……with less apparent complete heart block RATE . Fast vs Slow . QRS Width Narrow QRS Wide QRS Narrow QRS Wide QRS Tachycardia Tachycardia Bradycardia Bradycardia Regular Irregular Regular Irregular Sinus Brady Idioventricular A-Fib / Flutter Bradycardia w/ BBB Sinus Tach A-Fib VT PVT Junctional 2 AVB / II PSVT A-Flutter SVT aberrant A-Fib 1 AVB 3 AVB A-Flutter MAT 2 AVB / I or II PAT PAT 3 AVB ST PAC / PVC Stability Hypotension / hypoperfusion Altered mental status Chest pain – Coronary ischemic Dyspnea – Pulmonary edema Sinus Rhythm Sinus Rhythm P Wave PR Interval QRS Rate Rhythm Pacemaker Comment . Before . Constant, . Rate 60-100 . Regular . SA Node Upright in each QRS regular . Interval =/< leads I, II, . Look . Interval .12- .10 & III alike .20 Conduction Image reference: Cardionetics/ http://www.cardionetics.com/docs/healthcr/ecg/arrhy/0100_bd.htm Sinus Pause A delay of activation within the atria for a period between 1.7 and 3 seconds A palpitation is likely to be felt by the patient as the sinus beat following the pause may be a heavy beat. -
Pub 100-04 Medicare Claims Processing Centers for Medicare & Medicaid Services (CMS) Transmittal 3054 Date: August 29, 2014 Change Request 8803
Department of Health & CMS Manual System Human Services (DHHS) Pub 100-04 Medicare Claims Processing Centers for Medicare & Medicaid Services (CMS) Transmittal 3054 Date: August 29, 2014 Change Request 8803 SUBJECT: Ventricular Assist Devices for Bridge-to-Transplant and Destination Therapy I. SUMMARY OF CHANGES: This Change Request (CR) is effective for claims with dates of service on and after October 30, 2013; contractors shall pay claims for Ventricular Assist Devices as destination therapy using the criteria in Pub. 100-03, part 1, section 20.9.1, and Pub. 100-04, Chapter 32, sec. 320. EFFECTIVE DATE: October 30, 2013 *Unless otherwise specified, the effective date is the date of service. IMPLEMENTATION DATE: September 30, 2014 Disclaimer for manual changes only: The revision date and transmittal number apply only to red italicized material. Any other material was previously published and remains unchanged. However, if this revision contains a table of contents, you will receive the new/revised information only, and not the entire table of contents. II. CHANGES IN MANUAL INSTRUCTIONS: (N/A if manual is not updated) R=REVISED, N=NEW, D=DELETED-Only One Per Row. R/N/D CHAPTER / SECTION / SUBSECTION / TITLE D 3/90.2.1/Artifiical Hearts and Related Devices R 32/Table of Contents N 32/320/Artificial Hearts and Related Devices N 32/320.1/Coding Requirements for Furnished Before May 1, 2008 N 32/320.2/Coding Requirements for Furnished After May 1, 2008 N 32/320.3/ Ventricular Assist Devices N 32/320.3.1/Postcardiotomy N 32/320.3.2/Bridge-To -Transplantation (BTT) N 32/320.3.3/Destination Therapy (DT) N 32/320.3.4/ Other N 32/320.4/ Replacement Accessories and Supplies for External Ventricular Assist Devices or Any Ventricular Assist Device (VAD) III. -
Resuscitation and Defibrillation
AARC GUIDELINE: RESUSCITATION AND DEFIBRILLATION AARC Clinical Practice Guideline Resuscitation and Defibrillation in the Health Care Setting— 2004 Revision & Update RAD 1.0 PROCEDURE: signs, level of consciousness, and blood gas val- Recognition of signs suggesting the possibility ues—included in those conditions are or the presence of cardiopulmonary arrest, initia- 4.1 Airway obstruction—partial or complete tion of resuscitation, and therapeutic use of de- 4.2 Acute myocardial infarction with cardio- fibrillation in adults. dynamic instability 4.3 Life-threatening dysrhythmias RAD 2.0 DESCRIPTION/DEFINITION: 4.4 Hypovolemic shock Resuscitation in the health care setting for the 4.5 Severe infections purpose of this guideline encompasses all care 4.6 Spinal cord or head injury necessary to deal with sudden and often life- 4.7 Drug overdose threatening events affecting the cardiopul- 4.8 Pulmonary edema monary system, and involves the identification, 4.9 Anaphylaxis assessment, and treatment of patients in danger 4.10 Pulmonary embolus of or in frank arrest, including the high-risk de- 4.11 Smoke inhalation livery patient. This includes (1) alerting the re- 4.12 Defibrillation is indicated when cardiac suscitation team and the managing physician; (2) arrest results in or is due to ventricular fibril- using adjunctive equipment and special tech- lation.1-5 niques for establishing, maintaining, and moni- 4.13 Pulseless ventricular tachycardia toring effective ventilation and circulation; (3) monitoring the electrocardiograph and recogniz- -
The Transthoracic Impedance Cardiogram Is a Potential Haemodynamic Sensor for an Automated External Defibrillator
European Heart Journal (1998) 19, 1879–1888 Article No. hj981199 The transthoracic impedance cardiogram is a potential haemodynamic sensor for an automated external defibrillator P. W. Johnston*, Z. Imam†, G. Dempsey†, J. Anderson† and A. A. J. Adgey *Regional Medical Cardiology Centre, Royal Victoria Hospital, Belfast; †Northern Ireland Bioengineering Centre, Belfast, Northern Ireland Aims The American Heart Association has endorsed the agonal rhythm (20), electromechanical dissociation (22), concept of Public Access Defibrillation. However, there ventricular tachycardia (27) and sinus rhythm (5). These have been reports of inappropriate direct current shocks rhythms were divided into those associated with haemo- from automatic external defibrillators. The specificity of dynamic collapse i.e. no pulse — asystole, ventricular fibril- automatic external defibrillators for shockable rhythms lation, agonal rhythm, electromechanical dissociation and may be improved by the incorporation of a haemodynamic shockable ventricular tachycardia (associated with loss of sensor. consciousness, pulselessness or a systolic blood pressure of <80 mmHg) (Group 1) and those associated with a satis- Methods and Results This study examined the use of four factory cardiac output i.e. non-shockable ventricular tachy- parameters extracted from the impedance cardiogram i.e. cardia (conscious with a pulse) and sinus rhythm (Group 2). Peak dz/dt (the peak of the impedance cardiogram On univariate analysis each of the four impedance cardio- measured from the line dz/dt=0 Ùs"1), Peak-trough (the gram parameters were significantly greater in Group 2 than peak-to-trough measurement of the impedance cardiogram Group 1 (P<0·001). On multivariate analysis the par- Ùs"1), Area 1 (the area under the C wave of the impedance ameters which best differentiated the two groups were Area cardiogram above the line dz/dt=0 mÙ) and Area 2 (the 1 and Peak-trough. -
PUBLIC ACCESS to AUTOMATED EXTERNAL DEFIBRILLATORS (Aeds) FACTS
POSITION STATEMENT PUBLIC ACCESS TO AUTOMATED EXTERNAL DEFIBRILLATORS (AEDs) FACTS • Cardiac refers to the heart. Arrest means stop. Sudden cardiac arrest is the sudden and unexpected loss of heart function. • Signs of cardiac arrest include: no breathing or only gasping, no movement, and no pulse. • Up to 40,000 cardiac arrests occur each year in Canada. That’s one cardiac arrest every 12 minutes. Without rapid and appropriate treatment, most of these cardiac arrests will result in death. Thousands of lives could be saved through public access to automated external defibrillators. • An automated external defibrillator (AED) is a small, portable device used to identify cardiac rhythms and deliver a shock to correct abnormal electrical activity in the heart. As a result of the sophisticated electronics in an AED the operator will only be advised to deliver a shock if the • A landmark study of the City of Chicago airports public heart is in a rhythm which can be corrected by defibrillation. access to AED program showed survival rates as high as If a shockable rhythm is not detected, no shock can be 75%. This success is directly related to highly visible, readily given and the provider will be instructed to perform accessible automated external defibrillators for public use cardiopulmonary resuscitation (CPR) until emergency and an integrated structured emergency response system.7 medical services arrive. • Research shows that AEDs are most effectively used by • When an AED and CPR are immediately available, the trained individuals. However, AEDs are safe and easy to use chance of survival from sudden cardiac arrest is substantially by almost anyone. -
Persistent Ventricular Fibrillation During Therapeutic Hypothermia and Prolonged High-Dose Vasopressor Therapy: Case Report
ARTICLE IN PRESS The Journal of Emergency Medicine, Vol. xx, No. x, pp. xxx, 2009 Copyright © 2009 Elsevier Inc. Printed in the USA. All rights reserved 0736-4679/09 $–see front matter doi:10.1016/j.jemermed.2009.05.027 Clinical Communications: Adults PERSISTENT VENTRICULAR FIBRILLATION DURING THERAPEUTIC HYPOTHERMIA AND PROLONGED HIGH-DOSE VASOPRESSOR THERAPY: CASE REPORT Joshua C. Poles, BA* Tyler F. Vadeboncoeur, MD† and Bentley J. Bobrow, MD‡ *Kansas City University of Medicine and Biosciences, Kansas City, Missouri, †Department of Emergency Medicine, Mayo Clinic Florida, Jacksonville, Florida, and ‡Department of Emergency Medicine, Maricopa Medical Center, Phoenix, Arizona Reprint Address: [email protected] e Abstract—Background: Recent emphasis on high qual- survival rates remain low, many communities have real- ity prehospital cardiopulmonary resuscitation has resulted ized substantial improvements in meaningful survival in more out-of-hospital cardiac arrest victims surviving to the after OHCA. This has occurred primarily through emergency department. As such, standardized in-hospital emergency medical services (EMS) systems focusing post-cardiac arrest care is necessary to assure optimal neuro- logical recovery. Although therapeutic hypothermia has on delivering high quality cardiopulmonary resuscita- arisen as a key component in the post-cardiac arrest care tion (CPR) (1,2). paradigm, its interaction with other therapies remains Therapeutic hypothermia (TH) is currently an American poorly defined. Objective: The purpose of this communica- Heart Association (AHA) 2005 Guideline IIa therapy for tion is to demonstrate a potential interaction between thera- adult victims who remain unconscious after a ventricular peutic hypothermia and routinely administered resuscitation fibrillation (VF) arrest in the out-of-hospital setting (3).