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Effects of Direct Dental Restorations on Periodontium - Clinical and Radiological Study
Effects of direct dental restorations on periodontium - clinical and radiological study Luiza Ungureanu, Albertine Leon, Cristina Nuca, Corneliu Amariei, Doru Petrovici Constanta, Romania Summary The authors have performed a clinical study - 175 crown obturations of class II, II, V and cavities have been analyzed in 125 patients, following their impact on the marginal periodontium and a radi- ological study - consisting of the analysis of 108 proximal amalgam obturations and of their nega- tive effects on the profound periodontium. The results showed alarming percentages (over 80% in the clinical examination and 87% in the radiological examination of improper restorations, which generated periodontal alterations, from gingivitis to chronic marginal progressive periodontitis. The percentage of 59.26% obturations that triggered different degrees of osseous lysis imposed the need of knowing the negative effects of direct restorations on the periodontium and also the impor- tance of applying the specific preventive measures. Key words: dental anatomy, gingival embrasure contact area, under- and over sizing, cervical exten- sion, osseous lysis. Introduction the antagonist tooth can trigger enlargement of the contact point during functional movements. Dental restorations and periodontal health This allows interdental impact of foodstuff, with are closely related: periodontal health is needed devastating consequent effects on interproximal for the correct functioning of all restorations periodontal tissues. while the functional stimulation due to dental Marginal occlusal ridges must be placed restorations is essential for periodontal protection. above the proximal contact surface, and must be Coronal obturations with improper occlusal rounded and smooth so as to allow the access of modeling, oversized proximally or on the dental floss. -
DENTIN HYPERSENSITIVITY: Consensus-Based Recommendations for the Diagnosis & Management of Dentin Hypersensitivity
October 2008 | Volume 4, Number 9 (Special Issue) DENTIN HYPERSENSITIVITY: Consensus-Based Recommendations for the Diagnosis & Management of Dentin Hypersensitivity A Supplement to InsideDentistry® Published by AEGISPublications,LLC © 2008 PUBLISHER Inside Dentistry® and De ntin Hypersensitivity: Consensus-Based Recommendations AEGIS Publications, LLC for the Diagnosis & Management of Dentin Hypersensitivity are published by AEGIS Publications, LLC. EDITORS Lisa Neuman Copyright © 2008 by AEGIS Publications, LLC. Justin Romano All rights reserved under United States, International and Pan-American Copyright Conventions. No part of this publication may be reproduced, stored in a PRODUCTION/DESIGN Claire Novo retrieval system or transmitted in any form or by any means without prior written permission from the publisher. The views and opinions expressed in the articles appearing in this publication are those of the author(s) and do not necessarily reflect the views or opinions of the editors, the editorial board, or the publisher. As a matter of policy, the editors, the editorial board, the publisher, and the university affiliate do not endorse any prod- ucts, medical techniques, or diagnoses, and publication of any material in this jour- nal should not be construed as such an endorsement. PHOTOCOPY PERMISSIONS POLICY: This publication is registered with Copyright Clearance Center (CCC), Inc., 222 Rosewood Drive, Danvers, MA 01923. Permission is granted for photocopying of specified articles provided the base fee is paid directly to CCC. WARNING: Reading this supplement, Dentin Hypersensitivity: Consensus-Based Recommendations for the Diagnosis & Management of Dentin Hypersensitivity PRESIDENT / CEO does not necessarily qualify you to integrate new techniques or procedures into your practice. AEGIS Publications expects its readers to rely on their judgment Daniel W. -
Long-Term Uncontrolled Hereditary Gingival Fibromatosis: a Case Report
Long-term Uncontrolled Hereditary Gingival Fibromatosis: A Case Report Abstract Hereditary gingival fibromatosis (HGF) is a rare condition characterized by varying degrees of gingival hyperplasia. Gingival fibromatosis usually occurs as an isolated disorder or can be associated with a variety of other syndromes. A 33-year-old male patient who had a generalized severe gingival overgrowth covering two thirds of almost all maxillary and mandibular teeth is reported. A mucoperiosteal flap was performed using interdental and crevicular incisions to remove excess gingival tissues and an internal bevel incision to reflect flaps. The patient was treated 15 years ago in the same clinical facility using the same treatment strategy. There was no recurrence one year following the most recent surgery. Keywords: Gingival hyperplasia, hereditary gingival hyperplasia, HGF, hereditary disease, therapy, mucoperiostal flap Citation: S¸engün D, Hatipog˘lu H, Hatipog˘lu MG. Long-term Uncontrolled Hereditary Gingival Fibromatosis: A Case Report. J Contemp Dent Pract 2007 January;(8)1:090-096. © Seer Publishing 1 The Journal of Contemporary Dental Practice, Volume 8, No. 1, January 1, 2007 Introduction Hereditary gingival fibromatosis (HGF), also Ankara, Turkey with a complaint of recurrent known as elephantiasis gingiva, hereditary generalized gingival overgrowth. The patient gingival hyperplasia, idiopathic fibromatosis, had presented himself for examination at the and hypertrophied gingival, is a rare condition same clinic with the same complaint 15 years (1:750000)1 which can present as an isolated ago. At that time, he was treated with full-mouth disorder or more rarely as a syndrome periodontal surgery after the diagnosis of HGF component.2,3 This condition is characterized by had been made following clinical and histological a slow and progressive enlargement of both the examination (Figures 1 A-B). -
Epidemiology and Indices of Gingival and Periodontal Disease Dr
PEDIATRIC DENTISTRY/Copyright ° 1981 by The American Academy of Pedodontics Vol. 3, Special Issue Epidemiology and indices of gingival and periodontal disease Dr. Poulsen Sven Poulsen, Dr Odont Abstract Validity of an index indicates to what extent the This paper reviews some of the commonly used indices index measures what it is intended to measure. Deter- for measurement of gingivitis and periodontal disease. mination of validity is dependent on the availability Periodontal disease should be measured using loss of of a so-called validating criterion. attachment, not pocket depth. The reliability of several of Pocket depth may not reflect loss of periodontal the indices has been tested. Calibration and training of attachment as a sign of periodontal disease. This is be- examiners seems to be an absolute requirement for a cause gingival swelling will increase the distance from satisfactory inter-examiner reliability. Gingival and periodontal disease is much more severe in several the gingival margin to the bottom of the clinical populations in the Far East than in Europe and North pocket (pseudo-pockets). Thus, depth of the periodon- America, and gingivitis seems to increase with age resulting tal pocket may not be a valid measurement for perio- in loss of periodontal attachment in approximately 40% of dontal disease. 15-year-old children. Apart from the validity and reliability of an index, important factors such as the purpose of the study, Introduction the level of disease in the population, the conditions under which the examinations are going to be per- Epidemiological data form the basis for planning formed etc., will have to enter into choice of an index. -
Intrusion of Incisors to Facilitate Restoration: the Impact on the Periodontium
Note: This is a sample Eoster. Your EPoster does not need to use the same format style. For example your title slide does not need to have the title of your EPoster in a box surrounded with a pink border. Intrusion of Incisors to Facilitate Restoration: The Impact on the Periodontium Names of Investigators Date Background and Purpose This 60 year old male had severe attrition of his maxillary and mandibular incisors due to a protrusive bruxing habit. The patient’s restorative dentist could not restore the mandibular incisors without significant crown lengthening. However, with orthodontic intrusion of the incisors, the restorative dentist was able to restore these teeth without further incisal edge reduction, crown lengthening, or endodontic treatment. When teeth are intruded in adults, what is the impact on the periodontium? The purpose of this study was to determine the effect of adult incisor intrusion on the alveolar bone level and on root length. Materials and Methods We collected the orthodontic records of 43 consecutively treated adult patients (aged > 19 years) from four orthodontic practices. This project was approved by the IRB at our university. Records were selected based upon the following criteria: • incisor intrusion attempted to create interocclusal space for restorative treatment or correction of excessive anterior overbite • pre- and posttreatment periapical and cephalometric radiographs were available • no incisor extraction or restorative procedures affecting the cementoenamel junction during the treatment period pretreatment pretreatment Materials and Methods We used cephalometric and periapical radiographs to measure incisor intrusion. The radiographs were imported and the digital images were analyzed with Image J, a public-domain Java image processing program developed at the US National Institutes of Health. -
Dental Cementum Reviewed: Development, Structure, Composition, Regeneration and Potential Functions
Braz J Oral Sci. January/March 2005 - Vol.4 - Number 12 Dental cementum reviewed: development, structure, composition, regeneration and potential functions Patricia Furtado Gonçalves 1 Enilson Antonio Sallum 1 Abstract Antonio Wilson Sallum 1 This article reviews developmental and structural characteristics of Márcio Zaffalon Casati 1 cementum, a unique avascular mineralized tissue covering the root Sérgio de Toledo 1 surface that forms the interface between root dentin and periodontal Francisco Humberto Nociti Junior 1 ligament. Besides describing the types of cementum and 1 Dept. of Prosthodontics and Periodontics, cementogenesis, attention is given to recent advances in scientific Division of Periodontics, School of Dentistry understanding of the molecular and cellular aspects of the formation at Piracicaba - UNICAMP, Piracicaba, São and regeneration of cementum. The understanding of the mechanisms Paulo, Brazil. involved in the dynamic of this tissue should allow for the development of new treatment strategies concerning the approach of the root surface affected by periodontal disease and periodontal regeneration techniques. Received for publication: October 01, 2004 Key Words: Accepted: December 17, 2004 dental cementum, review Correspondence to: Francisco H. Nociti Jr. Av. Limeira 901 - Caixa Postal: 052 - CEP: 13414-903 - Piracicaba - S.P. - Brazil Tel: ++ 55 19 34125298 Fax: ++ 55 19 3412 5218 E-mail: [email protected] 651 Braz J Oral Sci. 4(12): 651-658 Dental cementum reviewed: development, structure, composition, regeneration and potential functions Introduction junction (Figure 1). The areas and location of acellular Cementum is an avascular mineralized tissue covering the afibrillar cementum vary from tooth to tooth and along the entire root surface. Due to its intermediary position, forming cementoenamel junction of the same tooth6-9. -
The Cementum: Its Role in Periodontal Health and Disease*
THE JOURNAL OF PERIODONTOLOGY JULY, NINETEEN HUNDRED SIXTY ONE The Cementum: Its Role In Periodontal Health and Disease* by DONALD A. KERR, D.D.S., M.S.,** Ann Arbor, Michigan HE cementum is a specialized calcified tissue of mesenchymal origin which provides for the attachment of the periodontal fibers to the surface of the Troot. It consists of 45 to 50 per cent inorganic material and 50 to 55 per cent organic material with the inorganic material in a hydroxyl apatite structure. The primary cementum is formed initially by appositional growth from the dental sac and later from the periodontal membrane under the influence of cementoblasts. It is formed in laminated layers with the incorporation of Sharpey's fibers into a fibrillar matrix which undergoes calcification. Cementum deposition is a Continuous process throughout life with new cementum being deposited over the old cemental surface. Cementum is formed by the organiza• tion of collagen fibrils which are cemented together by a matrix produced by the polymerization of mucopolysaccharides. This material is designated as cementoid and becomes mature cementum upon calcification. The significance of the continuous deposition of cementum has received various interpretations. 1. Continuous deposition of cementum is necessary for the reattachment of periodontal fibers which have been destroyed or which require reorientation due to change in position of teeth. It is logical that there should be a continuous deposition of cementum because it is doubtful that the initial fibers are retained throughout the life of the tooth, and therefore new fibers must be continually formed and attached by new cementum. -
Acute Dental Pain I: Pulpal and Dentinal Pain Pulpal And
VIDENSKAB & KLINIK | Oversigtsartikel ABSTRACT Acute dental pain I: Acute dental pain I: pulpal and dentinal pain pulpal and The specialized anatomy of the pulp-dentin dentinal pain complex and the dense, predominantly noci- ceptive pulpal innervation from the trigeminal nerve explains the variety of pain sensations from this organ. Matti Närhi, professor, ph.d., Department of Dentistry/Physiology, Institute of Medicine, University of Eastern Finland, Finland Brief, sharp pain is typical of A-fibre-mediated pain, while long-lasting, dull/aching pain indi- Lars Bjørndal, associate professor, dr.odont., ph.d., Department of Cariology and Endodontics, Faculty of Health and Medical Sciences, cates C-fibre involvement. A-fibres react to University of Copenhagen cold or mechanical stimuli, such as cold drinks Maria Pigg, senior lecturer, dr.odont., Department of Endodontics or toothbrushing, whereas C-fibres are mainly and Department of Orofacial Pain and Jaw Function, Malmö activated by inflammatory mediators. Thus, lin- University, Sweden gering pain suggests presence of irreversible Inge Fristad, professor, ph.d., Department of Clinical Dentistry, pulpal inflammation. Faculty of Medicine and Dentistry,University of Bergen, Norway During pulpitis, structural changes of the pul- Sivakami Rethnam Haug, associate professor and head, dr.odont., pal nerves (sprouting) occur and neuropeptide Section for Endodontics, Department of Clinical Dentistry, Faculty of release triggers an immune response; neuro- Medicine and Dentistry, University of Bergen, Norway, Årstadveien 19 N5009, Bergen, Norway genic inflammation. Pain sensations during pul- pitis can range from hypersensitivity to thermal stimuli to severe throbbing. There might also be aching pain, possibly referred and often difficult to localize. Thus, diagnosis is challenging for ain localized to teeth is among the most frequently ex- the clinician. -
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International Journal of Stomatological Research 2012, 1(2): 6-16 DOI: 10.5923/j.ijsr.20120102.01 Periodontium and Orthodontic Implications: Biological Basics Marinho Del Santo Orthodontist in Private Practice, Neo Face Dental Clinic Abstract As more and better orthodontic and periodontal scientific evidences are contextualized, potentially richer will be their achieved clinical results. The primary goal of this article is to study the main anatomic, histologic, physiologic and pathologic features of the periodontal tissues, and such knowledge to be applied in the Orthodontics and Periodontics fields. Keywords Periodontics, Orthodontics, Periodontal Development, Periodontal Anatomy, Periodontal Physiology the tissues and host resistance. When the equilibrium is 1. Introduction broken the periodontal disease is installed or re-installed, and diverse degenerative levels can possibly happen, and are In general, the periodontal tissues of patients who seek revertible or not. periodontal treatment suffer constant and dynamic oscillation between the physiologic and pathologic states, and the natural ongoing process to achieve periodontal 2. Goal homeostasis depends upon tissue reposition, remodeling, The main goal of this article is to study the periodontal degradation and repair. In practice, as more and better tissues, illustrating their innate features, and highlighting orthodontic and periodontal treatments are contextualized, pertinent clinical parameters. The target-specialists of these more potentially richer will be the achieved clinical results. publications practice in the Periodontics and Orthodontics In Orthodontics, dental movements target the correction of fields, and must know the mandatory periodontal requisites malocclusions. Such goal includes the leveling and to recommend orthodontic treatment, and the benefits of the alignment of the teeth in the arches, and such movements orthodontic treatment for the periodontal homeostasis. -
Pro-Argin, a Breakthrough Technology Based Upon Arginine
American Journal of Dentistry, Vol. 22, Special Issue A, March, 2009 A, March, 22, Special Issue Vol. American Journal of Dentistry, Vol. 22, Special Issue A, March, 2009 - p. 1A - 24A Introducing Pro-Argin™ A Breakthrough Technology Based upon Arginine and Calcium for In-Office Treatment of Dentin Hypersensitivity _______________________________________________________________________________________________________________________________________________________________ Editorial _______________________________________________________________________________________________________________________________________________________________ Dentin hypersensitivity: Beneficial effects of an arginine-calcium carbonate desensitizing paste Dentin hypersensitivity is a common occurrence diately after dental scaling procedures and its and is often a chief concern among patients. The sustained relief over 4 weeks. Another paper pre- pain associated with dentin hypersensitivity is sents the results of a double-blind, stratified, caused by some type of external stimulus and the randomized clinical study showing the successful sensitivity can range in its intensity from patient to desensitizing effect of the 8% arginine-calcium patient. The successful management of dentin carbonate paste tested, when applied as a pre- hypersensitivity is often very challenging for the procedure to professional dental cleaning. dental professional. The cause of the pain and the This Special Issue also includes a study con- description of the discomfort reported by -
Cervical Restorations Useful When Assessing Gingival and Periodontal Health
http://dx.doi.org/10.17159/2519-0105/2018/v73no10a9 INDUSTRY NEWS < 633 Cervical restorations useful when assessing gingival and periodontal health SADJ November 2018, Vol. 73 No. 10 p633 - p634 A Volchansky “Once a Periodontal Patient, always a Periodontal Patient”. ACRONYM This comment could be describing the affliction of Chronic CEJ: Cemento-enamel-junction Periodontitis or Refractory Periodontitis which refer to the periodontal status of patients who require monitoring over extended periods and who demonstrate severe attach- ment loss (derived from Parameters of Care: Journal of Periodontology, 20001). The progression of periodontal diseases is assessed by the extent of gingival recession, the severity of clinical attachment loss, and the probing depths of pockets. Periodontal disease is generally described as a slow and continually progressive condition.2 It may well be important that a fixed reference point is available to ensure repeatability when these measurements are recorded. The landmark habitually used is the cemento-enamel -junction (CEJ). The normal gingival margin position is 0.5 – 2.0 mm coronal to the CEJ. Gingival recession is defined as the increase in the location of the gingival margin apical to the CEJ.4 Figure 1. Cervical restorations on 13; 14, obscuring the CEJ. A cervical restoration is one that is placed adjacent There are three options, supragingival, equigingival and to the CEJ or the gingival margin (G V Black (19023). subgingival. Much has been written about the importance The margins of such a restoration are clearly visible of the restorative margin, its location, the materials adjacent to the gingival attachment and the location may and the contours of any restoration in relation to peri- approximate the cervical line / cemento-enamel junction odontal health.5,6 of the tooth. -
Diagnosis Questions and Answers
1.0 DIAGNOSIS – 6 QUESTIONS 1. Where is the narrowest band of attached gingiva found? 1. Lingual surfaces of maxillary incisors and facial surfaces of maxillary first molars 2. Facial surfaces of mandibular second premolars and lingual of canines 3. Facial surfaces of mandibular canines and first premolars and lingual of mandibular incisors* 4. None of the above 2. All these types of tissue have keratinized epithelium EXCEPT 1. Hard palate 2. Gingival col* 3. Attached gingiva 4. Free gingiva 16. Which group of principal fibers of the periodontal ligament run perpendicular from the alveolar bone to the cementum and resist lateral forces? 1. Alveolar crest 2. Horizontal crest* 3. Oblique 4. Apical 5. Interradicular 33. The width of attached gingiva varies considerably with the greatest amount being present in the maxillary incisor region; the least amount is in the mandibular premolar region. 1. Both statements are TRUE* 39. The alveolar process forms and supports the sockets of the teeth and consists of two parts, the alveolar bone proper and the supporting alveolar bone; ostectomy is defined as removal of the alveolar bone proper. 1. Both statements are TRUE* 40. Which structure is the inner layer of cells of the junctional epithelium and attaches the gingiva to the tooth? 1. Mucogingival junction 2. Free gingival groove 3. Epithelial attachment * 4. Tonofilaments 1 49. All of the following are part of the marginal (free) gingiva EXCEPT: 1. Gingival margin 2. Free gingival groove 3. Mucogingival junction* 4. Interproximal gingiva 53. The collar-like band of stratified squamous epithelium 10-20 cells thick coronally and 2-3 cells thick apically, and .25 to 1.35 mm long is the: 1.