Hip Dysplasia
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Turnedhead Adductedhip Truncal Curvature Syndrome
Archives ofDisease in Childhood 1994; 70: 515-519 515 Turned head adducted hip truncal curvature syndrome Arch Dis Child: first published as 10.1136/adc.70.6.515 on 1 June 1994. Downloaded from Chiaki Hamanishi, Seisuke Tanaka Abstract curvature. An epidemiological analysis was One hundred and eight neonates and carried out to determine whether the intra- infants who showed the clinical triad of a uterine environment of these asymmetrically head turned to one side, adduction con- deformed babies had been restricted. The tracture of the hip joint on the occipital clinical course of each feature of the clinical side of the turned head, and truncal cur- triad was also analysed to determine whether vature, which we named TAC syndrome, TAC syndrome is aetiologically related to any were studied. These cases included seven subsequent paediatric disorders. with congenital and five with late infantile dislocations of the hip joint and 14 who developed muscular torticollis. Forty one Patients and methods were among 7103 neonates examined by We studied a total of 108 cases with TAC one of the authors. An epidemiological syndrome. Of them, 41 were among a total analysis confirmed the aetiology of the number of 7103 neonates personally examined syndrome to be environmental. The side by one of the authors (CH) at newborn exami- to which the head was turned and that of nations conducted at hospitals in four cities the adducted hip contracture showed a since 1981. Thirteen were referred neonates. high correlation with the side of the The remaining 54 were among infants aged maternal spine on which the fetus had from 10 days to 3 months who were referred to been lying. -
Canine Hip Dysplasia - by Patricia Long October, 2001
Canine Hip Dysplasia - by Patricia Long October, 2001 Edited by Melissa Zebley, DVM Contributions by: Sue Bacig, Brenda Briggs, Sue Brightman, Cathy Burlile, Janice Cagwin, Steve Dudley, Lisa Ebnet, Sue German, Kathy Maher, Ruth Reynolds, Rose Tierney, Sue Sanvido, Doug Smith Much has been written on hip dysplasia (HD), and this article is simply an attempt to summarize much of that information. For a more in-depth examination of HD, I urge you to read the articles by Susan Thorpe-Vargas and John Cargill which can be found on http://workingdogs.com/ Prevention - the ounce worth more than a pound Breeders have several methods of trying to minimize the risk of HD: OFA, GDC, PennHIP. But these registries are only able to identify some dogs that may have HD. They are not able to identify the dogs that carry the genes for HD. It is important to understand that no matter how many generations there are in the pedigree with hips rated clear, all this can do is to minimize the risk of HD - it can't eliminate it. HD is a polygenetic multifactorial condition, which means that we still don't know exactly what causes it. But without the genes for it, a dog won't get it without some other event such as an injury. What is HD? The hip joint is a clever device, the classic ball and socket joint. Properly constructed, the top of the thigh bone, or femoral head, is the ball that fits into the socket, or acetabulum, in the pelvis. But in dogs, as in humans, this joint does not always develop properly. -
British Veterinary Association / Kennel Club Hip Dysplasia Scheme
British Veterinary Association / Kennel Club Hip Dysplasia Scheme Breed Specific Statistics – 1 January 2001 to 31 December 2016 Hip scores should be considered along with other criteria as part of a responsible breeding programme, and it is recommended that breeders choose breeding stock with hip scores around and ideally below the breed median score, depending on the level of HD in the breed. HD status of parents, siblings and progeny for Kennel Club registered dogs should also be considered, and these together with a three generation Health Test Pedigree may be downloaded via the Health Test Results Finder, available on the Kennel Club’s online health tool Mate Select (www.mateselect.org.uk). In addition, estimated breeding values (EBVs) are available for breeds in which a significant number of dogs have been graded, via the same link. For further advice on the interpretation and use of hip scores see www.bva.co.uk/chs The breed median score is the score of the ‘average’ dog in that breed (i.e. an equal number of dogs in that breed have better and worse scores). No. 15 year No. 15 year 5 year 5 year Breed score in Breed score in Range Median Median Range Median Median 15 years 15 years Affenpinscher 40 8 – 90 13 14 Beagle 62 8 - 71 16 17 Afghan Hound 18 0 – 73 8.5 27 Bearded Collie 1511 0 – 70 9 9 Airedale Terrier 933 4 – 72 11 10 Beauceron 42 2 – 23 10 10 Akita 1029 0 – 91 7 7 Belgian Shepherd 249 0 – 37 8 8 Dog (Groenendael) Alaskan Malamute 1248 0 – 78 10 10 Belgian Shepherd 16 5 - 16 10 14 Dog (Laekenois) Anatolian 63 3 – 67 9 -
Sciatica and Chronic Pain
Sciatica and Chronic Pain Past, Present and Future Robert W. Baloh 123 Sciatica and Chronic Pain Robert W. Baloh Sciatica and Chronic Pain Past, Present and Future Robert W. Baloh, MD Department of Neurology University of California, Los Angeles Los Angeles, CA, USA ISBN 978-3-319-93903-2 ISBN 978-3-319-93904-9 (eBook) https://doi.org/10.1007/978-3-319-93904-9 Library of Congress Control Number: 2018952076 © Springer International Publishing AG, part of Springer Nature 2019 This work is subject to copyright. All rights are reserved by the Publisher, whether the whole or part of the material is concerned, specifically the rights of translation, reprinting, reuse of illustrations, recitation, broadcasting, reproduction on microfilms or in any other physical way, and transmission or information storage and retrieval, electronic adaptation, computer software, or by similar or dissimilar methodology now known or hereafter developed. The use of general descriptive names, registered names, trademarks, service marks, etc. in this publication does not imply, even in the absence of a specific statement, that such names are exempt from the relevant protective laws and regulations and therefore free for general use. The publisher, the authors, and the editors are safe to assume that the advice and information in this book are believed to be true and accurate at the date of publication. Neither the publisher nor the authors or the editors give a warranty, express or implied, with respect to the material contained herein or for any errors or omissions that may have been made. The publisher remains neutral with regard to jurisdictional claims in published maps and institutional affiliations. -
Orthopedic-Conditions-Treated.Pdf
Orthopedic and Orthopedic Surgery Conditions Treated Accessory navicular bone Achondroplasia ACL injury Acromioclavicular (AC) joint Acromioclavicular (AC) joint Adamantinoma arthritis sprain Aneurysmal bone cyst Angiosarcoma Ankle arthritis Apophysitis Arthrogryposis Aseptic necrosis Askin tumor Avascular necrosis Benign bone tumor Biceps tear Biceps tendinitis Blount’s disease Bone cancer Bone metastasis Bowlegged deformity Brachial plexus injury Brittle bone disease Broken ankle/broken foot Broken arm Broken collarbone Broken leg Broken wrist/broken hand Bunions Carpal tunnel syndrome Cavovarus foot deformity Cavus foot Cerebral palsy Cervical myelopathy Cervical radiculopathy Charcot-Marie-Tooth disease Chondrosarcoma Chordoma Chronic regional multifocal osteomyelitis Clubfoot Congenital hand deformities Congenital myasthenic syndromes Congenital pseudoarthrosis Contractures Desmoid tumors Discoid meniscus Dislocated elbow Dislocated shoulder Dislocation Dislocation – hip Dislocation – knee Dupuytren's contracture Early-onset scoliosis Ehlers-Danlos syndrome Elbow fracture Elbow impingement Elbow instability Elbow loose body Eosinophilic granuloma Epiphyseal dysplasia Ewing sarcoma Extra finger/toes Failed total hip replacement Failed total knee replacement Femoral nonunion Fibrosarcoma Fibrous dysplasia Fibular hemimelia Flatfeet Foot deformities Foot injuries Ganglion cyst Genu valgum Genu varum Giant cell tumor Golfer's elbow Gorham’s disease Growth plate arrest Growth plate fractures Hammertoe and mallet toe Heel cord contracture -
The Hip's Influence on Low Back Pain
Journal of Sport Rehabilitation, 2009, 18, 24-32 © 2009 Human Kinetics, Inc. The Hip’s Influence on Low Back Pain: A Distal Link to a Proximal Problem Michael P. Reiman, P. Cody Weisbach, and Paul E. Glynn Low back pain (LBP) is a multifactorial dysfunction, with one of the potential con- tributing factors being the hip joint. Currently, research investigating the examination and conservative treatment of LBP has focused primarily on the lumbar spine. The objective of this clinical commentary is to discuss the potential link between hip impairments and LBP using current best evidence and the concept of regional inter- dependence as tools to guide decision making and offer ideas for future research. Keywords: strength, rehabilitation In day-to-day clinical practice it is often difficult to identify the source of symptoms in patients with low back pain (LBP).1 Abenhaim et al2 noted that a small percentage of individuals with LBP have an identifiable pathoanatomical source. Further clouding the picture are multiple studies indicating the potential inability of diagnostic imaging to identify the pain source, influence prognosis, or affect outcomes.3–6 Research has demonstrated the effectiveness of subgrouping patients into a classification system based on signs and symptoms indicating their likelihood to respond to specific treatments. This classification approach has pro- duced improved outcomes and high levels of reliability as compared with clinical- practice guidelines.7–9 For treating clinicians, these findings help guide decision making and improve results; however, not all patients will fit into a treatment- based subgroup. The treating therapist must then rely on an impairment-based approach, identifying potential local or remote contributors to the patient’s area of primary concern. -
Hip Dysplasia: Understanding a Very Misunderstood Puppy Abnormality
PET OWNER SERIES Congenital Hip Dysplasia: Understanding a very misunderstood puppy abnormality It is worth noting at the outset that all orthopedic conditions and post-operative recoveries are made worse by an obese or over-weight body condition. Since dogs come in all shapes and sizes, even within one breed, body weight is a difficult variable to guide. Body "condition" is easier to evaluate and recognize when "ideal". Also worth noting is that carrying excess fat tissue is not “bad” just because the animal is “heavy”, but probably more importantly because fat tissue is pro-inflammatory. It is an active, dynamic group of cells throughout the body that, when in excess, accelerates many degenerative processes leading to disease and injury. A lifetime-study of a large group of dogs demonstrated that lean dogs lived almost two years longer than their genetically similar overweight counterparts! The "ideal" body condition is leaner than you think. Very few dogs these days are ideal (65% are overweight or obese), so our frame of reference is skewed. To evaluate body condition, you use your eyes and hands. You should be able to feel the ribs, pelvic bones and shoulder bones easily, but not see them. You should be able to see (or feel in the fluffy dogs) a "waist" behind the ribs when viewed from the top. You should be able to see the belly tuck up behind the ribs when viewed from the side. Hip dysplasia is a common developmental problem in large breed dogs that is both hereditary and affected by nutrition, body weight and activity level. -
Cartilage Borderline
Prevalence of High-Grade Cartilage Defects in Patients With Borderline Dysplasia With Femoroacetabular Impingement: A Comparative Cohort Study Ioanna K. Bolia, M.D., M.S., Ph.D., Karen K. Briggs, M.P.H., Renato Locks, M.D., Jorge Chahla, M.D., Hajime Utsunomiya, M.D., Ph.D., and Marc J. Philippon, M.D. Purpose: To compare the prevalence, size, and location of Outerbridge grade III and IV cartilage defects on the femoral head and acetabulum between patients with borderline acetabular dysplasia and patients with non-borderline dysplasia who underwent hip arthroscopy for femoroacetabular impingement (FAI). Methods: Patients aged 18 years or older who underwent primary hip arthroscopy for correction of FAI and labral repair from November 2005 to April 2016 were included. We excluded patients with previous hip surgery, a radiographic hip joint space of 2 mm or less, and/or a lateral center-edge angle (LCEA) of less than 20 or greater than 40. The study patients were divided into 2 groups based on the LCEA on the anteroposterior pelvic radiograph: Patients with an LCEA between 20 and 25 were included in the borderline group, and patients with an LCEA between 25 and 40 were included in the non-borderline group. The prevalence, size, and location of Outerbridge grade III and IV chondral lesions on the femoral head and acetabulum were recorded intraoperatively. Comparisons between groups were performed with the Mann-Whitney U test for nonpara- metric testing and the t test for data that were normally distributed. Data were analyzed to calculate odds ratios associated with the various factors. -
PERIACETABULAR OSTEOTOMY (PAO) All Information Here Is General in Nature and Needs to Be Tailored to Your Circumstances
PERIACETABULAR OSTEOTOMY (PAO) All information here is general in nature and needs to be tailored to your circumstances Overview PAO surgery is a hip preservation surgery performed to correct a deformity in the acetabulum (hip socket) such as acetabular dysplasia. If this condition remains untreated, secondary arthritis commonly develops. Therefore, in order to relieve symptoms and improve the prognosis of the hip, this surgery is done to correct the bony anatomy and help normalize the load across the joint. “Periacetabular” means around the acetabulum (hip socket). “Osteotomy” means to cut bone. Therefore, periacetabular osteotomy means to cut the bone around the acetabulum and reposition the hip socket. The PAO is a very effective procedure for the treatment of symptomatic acetabular dysplasia. Mr Slattery is trained in a minimally invasive form of periacetabular osteotomy using a groin crease incision. An incision is made across the front of the hip joint to allow exposure of the hip and surrounding pelvis. Then specialised instruments are used with X-ray vision to perform controlled cuts of the pelvis and free the acetabulum from the pelvis. The acetabulum is repositioned and fixed in the new position with three or four screws after further checks with X-ray. This is a highly specialised procedure which is done by only a few surgeons worldwide. It was pioneered in Switzerland with Prof. Ganz, which is where Dr Slattery undertook sub-specialist fellowship training to learn the art of this procedure. A pelvic model is shown and demonstrates the bony cuts and repositioning of the acetabulum. The gaps between the repositioned bone fill in with new bone, just like the healing of a fracture. -
Hip Dysplasia Scheme Breed Specific Statistics 2019
Hip Dysplasia Scheme Breed Specific Statistics 2019 The below table outlines the median hip score for each breed screened under the CHS Hip Dysplasia Scheme. The breed median score is the ‘middle’ score for all dogs’ in that breed (i.e. an equal number of dogs in that breed have scored higher or lower than the median score). Hip scores should be considered along with other criteria as part of responsible breeding programme. It is recommended that breeders choose breeding stock with hips scores around, and ideally below, the 5-year breed median score. By representing dogs scored in the last 15 years, a more accurate reflection of each breed’s current state of health and improvement is given. The 5-year median here refers to dogs scored between 1st January 2015 and 31st December 2019. Hip dysplasia status of the parents, siblings and progeny for Kennel Club registered dogs should also be considered, and these together with a three generation Health Test Pedigree may be downloaded via the Health Test Results Finder, available on the Kennel Club's online health tool Mate Select. In addition, estimated breeding values (EBVs) are available for breeds in which a significant number of breeds have been scored, via the same link. Tested 15 15 years 5 years Breed Tested 2019 years Mean Min Max Median Mean Median Affenpinscher 40 0 17.9 8.0 90.0 13.0 23.8 23.0 Afghan Hound 85 33 12.3 4.0 73.0 10.0 12.6 10.0 Airedale Terrier 910 58 13.9 4.0 77.0 11.0 13.8 11.0 Akita 883 27 7.7 0.0 58.0 6.0 8.0 7.0 Alaskan Malamute 1242 25 11.7 0.0 78.0 10.0 10.1 9.0 -
Lieshout Van Lieshout, M.J.S
EXPLORING ROBIN SEQUENCE Manouk van Lieshout Van Lieshout, M.J.S. ‘Exploring Robin Sequence’ Cover design: Iliana Boshoven-Gkini - www.agilecolor.com Thesis layout and printing by: Ridderprint BV - www.ridderprint.nl ISBN: 978-94-6299-693-9 Printing of this thesis has been financially supported by the Erasmus University Rotterdam. Copyright © M.J.S. van Lieshout, 2017, Rotterdam, the Netherlands All rights reserved. No parts of this thesis may be reproduced, stored in a retrieval system, or transmitted in any form or by any means without permission of the author or when appropriate, the corresponding journals Exploring Robin Sequence Verkenning van Robin Sequentie Proefschrift ter verkrijging van de graad van doctor aan de Erasmus Universiteit Rotterdam op gezag van de rector magnificus Prof.dr. H.A.P. Pols en volgens besluit van het College voor Promoties. De openbare verdediging zal plaatsvinden op woensdag 20 september 2017 om 09.30 uur door Manouk Ji Sook van Lieshout geboren te Seoul, Korea PROMOTIECOMMISSIE Promotoren: Prof.dr. E.B. Wolvius Prof.dr. I.M.J. Mathijssen Overige leden: Prof.dr. J.de Lange Prof.dr. M. De Hoog Prof.dr. R.J. Baatenburg de Jong Copromotoren: Dr. K.F.M. Joosten Dr. M.J. Koudstaal TABLE OF CONTENTS INTRODUCTION Chapter I: General introduction 9 Chapter II: Robin Sequence, A European survey on current 37 practice patterns Chapter III: Non-surgical and surgical interventions for airway 55 obstruction in children with Robin Sequence AIRWAY OBSTRUCTION Chapter IV: Unravelling Robin Sequence: Considerations 79 of diagnosis and treatment Chapter V: Management and outcomes of obstructive sleep 95 apnea in children with Robin Sequence, a cross-sectional study Chapter VI: Respiratory distress following palatal closure 111 in children with Robin Sequence QUALITY OF LIFE Chapter VII: Quality of life in children with Robin Sequence 129 GENERAL DISCUSSION AND SUMMARY Chapter VIII: General discussion 149 Chapter IX: Summary / Nederlandse samenvatting 169 APPENDICES About the author 181 List of publications 183 Ph.D. -
Just How Bad Is Hip Dysplasia
Facts about Hip Dysplasia For years this word has been passed around as something that Common breeds affected with is seen occasionally in large breed dogs, but what is hip dysplasia and hip dysplasia: what can be done to prevent it? Hip dysplasia is literally arthritis of 1) English Bulldog 74% bad the hip joints. Hip dysplasia is considered to be an inherited disease 2) Pug 63% bad since hip conformation is passed down from the dam and sire to the 3) Dogue de Bordeux 56% bad puppies. The hip joint is a ball and socket joint, so the more of the 4) Neopolitan Mastiff 49% bad ball that is covered by the socket, the less likely the dog is to develop 5) St. Bernard 47% bad hip dysplasia. Evaluating hip joint conformation is very difficult to 6) Cane Corso 40% bad do without doing x-rays. Manual palpation of the hip joint can 7) Basset 36% bad be done but many poorly conformed hip joints may feel “tight” at 8) French Bulldog 34% bad 8-12 weeks of age. There have been many discussions about the 9) American Bulldog 33% bad hereditary aspect of hip dysplasia and if any other factors contribute to 10) Newfoundland 26% bad hip dysplasia. Hip dysplasia can be affected by the body condition of the dog. Purina foods did a study with a litter of Labradors in which they split up a litter of puppies and kept ½ the litter in an obese condition (>5 body condition score) and the other ½ of the litter in good lean body condition (<5 body condition score).