Portal Hypertensionand Its Radiological Investigation
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Byoung Chan Kang, MD, Da Jeong Nam, MD*, Eun Kyoung Ahn, MD*, Duck Mi Yoon, MD, and Joung Goo Cho, MD*
Korean J Pain 2013 July; Vol. 26, No. 3: 299-302 pISSN 2005-9159 eISSN 2093-0569 http://dx.doi.org/10.3344/kjp.2013.26.3.299 |Case Report| Secondary Erythromelalgia - A Case Report - Department of Anesthesiology and Pain Medicine, Yonsei University College of Medicine, Seoul, *National Health Insurance Service Ilsan Hospital, Goyang, Korea Byoung Chan Kang, MD, Da Jeong Nam, MD*, Eun Kyoung Ahn, MD*, Duck Mi Yoon, MD, and Joung Goo Cho, MD* Erythromelalgia is a rare neurovascular pain syndrome characterized by a triad of redness, increased temperature, and burning pain primarily in the extremities. Erythromelalgia can present as a primary or secondary form, and secondary erythromelalgia associated with a myeloproliferative disease such as essential thrombocythemia often responds dramatically to aspirin therapy, as in the present case. Herein, we describe a typical case of a 48-year-old woman with secondary erythromelalgia linked to essential thrombocythemia in the unilateral hand. As this case demonstrates, detecting and visualizing the hyperthermal area through infrared thermography of an erythromelalgic patient can assist in diagnosing the patient, assessing the therapeutic results, and understanding the disease course of erythromelalgia. (Korean J Pain 2013; 26: 299-302) Key Words: aspirin, erythromelalgia, infrared thermography, neuropathic pain. Erythromelalgia is a rare clinical syndrome charac- Adults are more commonly involved than children and are terized by a triad of redness, increased temperature, and more likely to have the secondary form. Secondary eryth- burning pain primarily in the extremities. The term eryth- romelalgia is usually associated with myeloproliferative romelalgia, derived from the Greek words for redness and disorders such as essential thrombocythemia (ET) and pol- pain in the extremities, was coined in 1878 by Mitchell [1]. -
Splenic Artery Embolization for the Treatment of Gastric Variceal Bleeding Secondary to Splenic Vein Thrombosis Complicated by Necrotizing Pancreatitis: Report of a Case
Hindawi Publishing Corporation Case Reports in Medicine Volume 2016, Article ID 1585926, 6 pages http://dx.doi.org/10.1155/2016/1585926 Case Report Splenic Artery Embolization for the Treatment of Gastric Variceal Bleeding Secondary to Splenic Vein Thrombosis Complicated by Necrotizing Pancreatitis: Report of a Case Hee Joon Kim, Eun Kyu Park, Young Hoe Hur, Yang Seok Koh, and Chol Kyoon Cho Department of Surgery, Chonnam National University Medical School, Gwangju, Republic of Korea Correspondence should be addressed to Chol Kyoon Cho; [email protected] Received 11 August 2016; Accepted 1 November 2016 Academic Editor: Omer Faruk Dogan Copyright © 2016 Hee Joon Kim et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Splenic vein thrombosis is a relatively common finding in pancreatitis. Gastric variceal bleeding is a life-threatening complication of splenic vein thrombosis, resulting from increased blood flow to short gastric vein. Traditionally, splenectomy is considered the treatment of choice. However, surgery in necrotizing pancreatitis is dangerous, because of severe inflammation, adhesion, and bleeding tendency. In the Warshaw operation, gastric variceal bleeding is rare, even though splenic vein is resected. Because the splenic artery is also resected, blood flow to short gastric vein is not increased problematically. Herein, we report a case of gastric variceal bleeding secondary to splenic vein thrombosis complicated by necrotizing pancreatitis successfully treated with splenic artery embolization. Splenic artery embolization could be the best treatment option for gastric variceal bleeding when splenectomy is difficult such as in case associated with severe acute pancreatitis or associated with severe adhesion or in patients withhigh operation risk. -
Venous Symposium: Overview
5/30/2017 1 5/30/2017 VENOUS SYMPOSIUM: OVERVIEW Robert W. Vorhies, M.D., F.A.C.S. Vascular and Endovascular Surgery Endovenous Therapy and Vein Aesthetics Ferrell-Duncan Clinic, Cox Health Systems WHY DO WE CARE? • Epidemiology • Disability • Historical experience • Opportunity 2 5/30/2017 EPIDEMIOLOGY QUESTION #1 Which state has nearly the same population as all the people in the US with venous disease? • A. New York • B. Florida • C. California • D. Missouri EPIDEMIOLOGY QUESTION #1 Which state has nearly the population as all the people in the US with venous disease? • A. New York • B. Florida • C. California • More than 11 million men and 22 million women between the ages of 40 and 80 years in the United States have varicose veins. • Prevalence of 20% (range, 21.8%-29.4%) • D. Missouri Peter Gloviczki, Anthony J. Comerota, Michael C. Dalsing, Bo G. Eklof, David L. Gillespie, Monika L. Gloviczki, Joann M. Lohr, Robert B. McLafferty, Mark H. Meissner, M. Hassan Murad, Frank T. Padberg, Peter J. Pappas, Marc A. Passman Joseph D. Raffetto, Michael A. Vasquez, and Thomas W. Wakefield. “The care of patients with varicose veins and associated chronic venous diseases: Clinical practice guidelines of the Society for Vascular Surgery and the American Venous Forum.” J Vasc Surg . 2011;53:2S-48S. 3 5/30/2017 EPIDEMIOLOGY QUESTION #2 • Which mid western metropolitan area has about the same number of people as those with severe chronic venous insufficiency including ulcers and skin changes? • A. Minneapolis • B. Chicago • C. St. Louis • D. Kansas City. EPIDEMIOLOGY QUESTION #2 • Which mid western city has about the same number of people as those with severe chronic venous insufficiency including ulcers and skin changes? • A. -
Hemorrhoids Information, Pictures, Treatments, and Cures by Rick Shacket DO, 1989 ©
Hemorrhoids Information, Pictures, Treatments, and Cures By Rick Shacket DO, 1989 © Hemorrhoids are cushions of tissue and varicose veins located in and around the rectal area. When they become inflamed, hemorrhoids can itch, bleed, and cause pain. Unfortunately a hemorrhoidal condition only tends to get worse over the years. That is why safe, gentle, and effective treatment for hemorrhoids is recommended as soon as they occur. Hemorrhoids bother about 89% of all Americans at some time in their lives. Hemorrhoids caused Napoleon to sit side-saddle, sent President Jimmy Carter to the operating room, and benched baseball star George Brett during the 1980 World Series. Over two thirds of all healthy people reporting for physical examinations have hemorrhoids. For more information about Hemorrhoids visit the links below: • Pictures: Hemorrhoids and Anal Fissure • Stapled Hemorrhoidopexy (PPH Procedure) • What Are Hemorrhoids? • Harmonic Scalpel Hemorrhoid surgery • What Are the Symptoms of Hemorrhoids? • Laser Surgery for Hemorrhoids • How Common Are Hemorrhoids? • Atomizing Hemorrhoids • How Are Hemorrhoids Diagnosed? • Complications of Hemorrhoid Surgery • What Is the Treatment? • Knowing What to Ask Your Surgeon • How Are Hemorrhoids Prevented? • Allopathic Hemorrhoid Medications • Painless Treatment of Hemorrhoids • Herbal Hemorrhoid Medications • HAL-RAR Method Hemorrhoidectomy • Homeopathic Hemorrhoid Medications • Hemorrhoids Grades 1 to 4 • References • Surgical Classification of Hemorrhoids • Video References • Traditional Surgery for Hemorrhoids Pictures: Hemorrhoids and Anal Fissure Internal hemorrhoids occur higher up in the anal canal, out of sight. Bleeding is the most common symptom of internal hemorrhoids, and often the only one in mild cases. View hemorrhoid gallery for detailed photos. Hemorrhoids Information, Pictures, Treatments, and Cures Page 1 of 21 External hemorrhoids are visible-occurring out side the anus. -
Arteries and Veins) of the Gastrointestinal System (Oesophagus to Anus)
2021 First Sitting Paper 1 Question 07 2021-1-07 Outline the anatomy of the blood supply (arteries and veins) of the gastrointestinal system (oesophagus to anus) Portal circulatory system + arterial blood flow into liver 1100ml of portal blood + 400ml from hepatic artery = 1500ml (30% CO) Oxygen consumption – 20-35% of total body needs Arterial Supply Abdominal Aorta • It begins at the aortic hiatus of the diaphragm, anterior to the lower border of vertebra T7. • It descends to the level of vertebra L4 it is slightly to the left of midline. • The terminal branches of the abdominal aorta are the two common iliac arteries. Branches of Abdominal Aorta Visceral Branches Parietal Branches Celiac. Inferior Phrenics. Superior Mesenteric. Lumbars Inferior Mesenteric. Middle Sacral. Middle Suprarenals. Renals. Internal Spermatics. Gonadal Anterior Branches of The Abdominal Aorta • Celiac Artery. Superior Mesenteric Artery. Inferior Mesenteric Artery. • The three anterior branches supply the gastrointestinal viscera. Basic Concept • Fore Gut - Coeliac Trunk • Mid Gut - Superior Mesenteric Artery • Hind Gut - Inferior Mesenteric Artery Celiac Trunk • It arises from the abdominal aorta immediately below the aortic hiatus of the diaphragm anterior to the upper part of vertebra LI. • It divides into the: left gastric artery, splenic artery, common hepatic artery. o Left gastric artery o Splenic artery ▪ Short gastric vessels ▪ Lt. gastroepiploic artery o Common hepatic artery ▪ Hepatic artery proper JC 2019 2021 First Sitting Paper 1 Question 07 • Left hepatic artery • Right hepatic artery ▪ Gastroduodenal artery • Rt. Gastroepiploic (gastro-omental) artery • Sup pancreatoduodenal artery • Supraduodenal artery Oesophagus • Cervical oesophagus - branches from inferior thyroid artery • Thoracic oesophagus - branches from bronchial arteries and aorta • Abd. -
Dr. ALSHIKH YOUSSEF Haiyan
Dr. ALSHIKH YOUSSEF Haiyan General features The peritoneum is a thin serous membrane Consisting of: 1- Parietal peritoneum -lines the ant. Abdominal wall and the pelvis 2- Visceral peritoneum - covers the viscera 3- Peritoneal cavity - the potential space between the parietal and visceral layer of peritoneum - in male, is a closed sac - but in the female, there is a communication with the exterior through the uterine tubes, the uterus, and the vagina ▪ Peritoneum cavity divided into Greater sac Lesser sac Communication between them by the epiploic foramen The peritoneum The peritoneal cavity is the largest one in the body. Divided into tow sac : .Greater sac; extends from diaphragm down to the pelvis. Lesser Sac .Lesser sac or omental bursa; lies behind the stomach. .Both cavities are interconnected through the epiploic foramen(winslow ). .In male : the peritoneum is a closed sac . .In female : the sac is not completely closed because it Greater Sac communicates with the exterior through the uterine tubes, uterus and vagina. Peritoneum in transverse section The relationship between viscera and peritoneum Intraperitoneal viscera viscera is almost totally covered with visceral peritoneum example, stomach, 1st & last inch of duodenum, jejunum, ileum, cecum, vermiform appendix, transverse and sigmoid colons, spleen and ovary Intraperitoneal viscera Interperitoneal viscera Retroperitoneal viscera Interperitoneal viscera Such organs are not completely wrapped by peritoneum one surface attached to the abdominal walls or other organs. Example liver, gallbladder, urinary bladder and uterus Upper part of the rectum, Ascending and Descending colon Retroperitoneal viscera some organs lie on the posterior abdominal wall Behind the peritoneum they are partially covered by peritoneum on their anterior surfaces only Example kidney, suprarenal gland, pancreas, upper 3rd of rectum duodenum, and ureter, aorta and I.V.C The Peritoneal Reflection The peritoneal reflection include: omentum, mesenteries, ligaments, folds, recesses, pouches and fossae. -
Vessels and Circulation
CARDIOVASCULAR SYSTEM OUTLINE 23.1 Anatomy of Blood Vessels 684 23.1a Blood Vessel Tunics 684 23.1b Arteries 685 23.1c Capillaries 688 23 23.1d Veins 689 23.2 Blood Pressure 691 23.3 Systemic Circulation 692 Vessels and 23.3a General Arterial Flow Out of the Heart 693 23.3b General Venous Return to the Heart 693 23.3c Blood Flow Through the Head and Neck 693 23.3d Blood Flow Through the Thoracic and Abdominal Walls 697 23.3e Blood Flow Through the Thoracic Organs 700 Circulation 23.3f Blood Flow Through the Gastrointestinal Tract 701 23.3g Blood Flow Through the Posterior Abdominal Organs, Pelvis, and Perineum 705 23.3h Blood Flow Through the Upper Limb 705 23.3i Blood Flow Through the Lower Limb 709 23.4 Pulmonary Circulation 712 23.5 Review of Heart, Systemic, and Pulmonary Circulation 714 23.6 Aging and the Cardiovascular System 715 23.7 Blood Vessel Development 716 23.7a Artery Development 716 23.7b Vein Development 717 23.7c Comparison of Fetal and Postnatal Circulation 718 MODULE 9: CARDIOVASCULAR SYSTEM mck78097_ch23_683-723.indd 683 2/14/11 4:31 PM 684 Chapter Twenty-Three Vessels and Circulation lood vessels are analogous to highways—they are an efficient larger as they merge and come closer to the heart. The site where B mode of transport for oxygen, carbon dioxide, nutrients, hor- two or more arteries (or two or more veins) converge to supply the mones, and waste products to and from body tissues. The heart is same body region is called an anastomosis (ă-nas ′tō -mō′ sis; pl., the mechanical pump that propels the blood through the vessels. -
The Diameter of the Originating Vein Determines Esophageal and Gastric Fundic Varices in Portal Hypertension Secondary to Posthepatitic Cirrhosis
CLINICS 2012;67(6):609-614 DOI:10.6061/clinics/2012(06)11 CLINICAL SCIENCE The diameter of the originating vein determines esophageal and gastric fundic varices in portal hypertension secondary to posthepatitic cirrhosis Hai-ying Zhou, Tian-wu Chen, Xiao-ming Zhang, Li-ying Wang, Li Zhou, Guo-li Dong, Nan-lin Zeng, Hang Li, Xiao-li Chen, Rui Li Sichuan Key Laboratory of Medical Imaging, and Department of Radiology, Affiliated Hospital of North Sichuan Medical College, Sichuan/China. OBJECTIVE: The aim of this study was to determine whether and how the diameter of the vein that gives rise to the inflowing vein of the esophageal and gastric fundic varices secondary to posthepatitic cirrhosis, as measured with multidetector-row computed tomography, could predict the varices and their patterns. METHODS: A total of 106 patients with posthepatitic cirrhosis underwent multidetector-row computed tomography. Patients with and without esophageal and gastric fundic varices were enrolled in Group 1 and Group 2, respectively. Group 1 was composed of Subgroup A, consisting of patients with varices, and Subgroup B consisted of patients with varices in combination with portal vein-inferior vena cava shunts. The diameters of the originating veins of veins entering the varices were reviewed and statistically analyzed. RESULTS: The originating veins were the portal vein in 8% (6/75) of patients, the splenic vein in 65.3% (49/75) of patients, and both the portal and splenic veins in 26.7% (20/75) of patients. The splenic vein diameter in Group 1 was larger than that in Group 2, whereas no differences in portal vein diameters were found between groups. -
Reprint Of: Why Are Hemorrhoids Symptomatic? the Pathophysiology and Etiology of Hemorrhoids
Seminars in Colon and Rectal Surgery 29 (2018) 160 166 À Contents lists available at ScienceDirect Seminars in Colon and Rectal Surgery journal homepage: www.elsevier.com/locate/yscrs Reprint of: Why are hemorrhoids symptomatic? the pathophysiology and etiology of hemorrhoids WilliamD1X X C. Cirocco, MD,D2X X FACS, FASCRS* Department of Surgery, University of Missouri Kansas City, Kansas City, Missouri. À ABSTRACT Hemorrhoids are a normal component of the anorectum and contribute to the mechanism of anal closure, thus providing fine adjustment of anal continence. There are numerous myths and legends associated with the disordered and diseased state of hemorrhoids. Fortunately, information obtained from modern technolo- gies including microscopic histopathology defined first the actual substance and makeup of hemorrhoids and was later combined with anorectal physiology to provide evidence establishing the underlying pathophysiol- ogy of this universal finding of the human anorectum. The sliding anal canal theory of Gass and Adams has held up and is further supported by other anatomic studies including the work of WHF Thomson, who popu- larized the term “cushions” to describe the complex intertwining of muscle, connective tissue, veins, arteries, and arteriovenous communications which constitute hemorrhoids. A loss of muscle mass in favor of connec- tive tissue over time helps explain the role of aging as a predisposing factor for symptomatic hemorrhoids. Other factors include the modern “rich” or low-residue diet leading to constipation and straining which con- tributes to prolapsing cushions. Pathologic studies also demonstrated arteriovenous communications explain- ing why hemorrhoid bleeding is typically bright red or arterial in nature as opposed to dark or venous bleeding. -
Treatment Strategies for Patients with Lower Extremity Chronic Venous Disease (LECVD)
Evidence-based Practice Center Systematic Review Protocol Project Title: Treatment Strategies for Patients with Lower Extremity Chronic Venous Disease (LECVD) Project ID: DVTT0515 Initial publication date if applicable: March 7, 2016 Amendment Date(s) if applicable: May 6th, 2016 (Amendments Details–see Section VII) I. Background for the Systematic Review Lower extremity chronic venous disease (LECVD) is a heterogeneous term that encompasses a variety of conditions that are typically classified based on the CEAP classification, which defines LECVD based on Clinical, Etiologic, Anatomic, and Pathophysiologic parameters. This review will focus on treatment strategies for patients with LECVD, which will be defined as patients who have had signs or symptoms of LE venous disease for at least 3 months. Patients with LECVD can be asymptomatic or symptomatic, and they can exhibit a myriad of signs including varicose veins, telangiectasias, LE edema, skin changes, and/or ulceration. The etiology of chronic venous disease includes venous dilation, venous reflux, (venous) valvular incompetence, mechanical compression (e.g., May-Thurner syndrome), and post-thrombotic syndrome. Because severity of disease and treatment are influenced by anatomic segment, LECVD is also categorized by anatomy (iliofemoral vs. infrainguinal veins) and type of veins (superficial veins, perforating veins, and deep veins). Finally, the pathophysiology of LECVD is designated typically as due to the presence of venous reflux, thrombosis, and/or obstruction. LECVD is common -
Technical Aspects of Orthotopic Liver Transplantation for Hepatocellular Carcinoma
Technical Aspects of Orthotopic Liver Transplantation for Hepatocellular Carcinoma a a,b, Lung-Yi Lee, MD , David P. Foley, MD * KEYWORDS Liver transplantation Surgery Hepatocellular carcinoma Piggyback technique Portal vein thrombosis KEY POINTS In the majority of cases, patients with cirrhosis and hepatocellular carcinoma (HCC) who undergo liver transplantation are transplanted based on their higher Model for End-Stage Liver Disease (MELD) exception score and not their physiologic MELD score; this usually results in fewer physiologic derangements during liver transplantation. Patients who have previously undergone locoregional therapy or liver resection for HCC can develop significant perihepatic adhesions that increase the complexity of the hepa- tectomy during transplant. Implantation strategy of the inferior vena cava (IVC) during liver transplant may need to be modified based on location of previously treated HCC. Patients who undergo transarterial chemoembolization for pretransplant HCC therapy may have higher rates of hepatic artery thrombosis after liver transplant; therefore, aorto- hepatic bypass grafting with donor iliac artery may be required for arterial in flow to the liver allograft. Patients with portal vein (PV) thrombosis with a bland thrombus and a patent superior mesenteric vein (SMV) can undergo successful liver transplant through either PV throm- bectomy and standard end-to-end PV-PV anastomosis, or the use of SMV-PV bypass graft with donor iliac vein. a Department of Surgery, University of Wisconsin School of Medicine and Public Health, Clinical Sciences Center, H4/766, 600 Highland Avenue, Madison, WI 53792-3284, USA; b Veterans Administration Surgical Services, William S. Middleton Memorial Veterans Hospital, 2500 Overlook Terrace, Madison, WI 53705, USA * Corresponding author. -
Venous Drainage from the Tail of the Pancreas to the Lienal Vein and Its Relationship with the Distal Splenorenal Shunt Selectivity1
20 – ORIGINAL ARTICLE Surgical Anatomy Venous drainage from the tail of the pancreas to the lienal vein and its relationship with the distal splenorenal shunt selectivity1 Drenagem venosa da cauda do pâncreas para a veia lienal e sua relação com a seletividade da anastomose esplenorrenal Cláudio PirasI, Danilo Nagib Salomão PauloII, Isabel Cristina Andreatta Lemos PauloIII, Hildegardo RodriguesIV, Alcino Lázaro da SilvaV I Associate Professor, Department of Surgery, School of Sciences, EMESCAM, Espirito Santo, Brazil. II Full Professor of Surgery, Department of Surgery, School of Sciences, EMESCAM, Espirito Santo, Brazil. III Associate Professor, Department of Surgery, School of Sciences, EMESCAM, Espirito Santo, Brazil. IV Professor of Anatomy, Department of Morphology, School of Sciences, EMESCAM, Espirito Santo, Brazil. V Emeritus Professor of Surgery, School of Medicine, Federal University of Minas Gerais, Brazil. ABSTRACT Purpose: To identify the veins draining from the pancreatic tail to the lienal vein and its possible relationship with the loss of the distal splenorenal shunt selectivity. Methods: Thirty eight human blocks including stomach, duodenum, spleen, colon and pancreas, removed from fresh corpses, were studied with the replenish and corrosion technique, using vinilic resin and posterior corrosion of the organic tissue with commercial hydrochloric acid, in order to study the lienal vein and its tributaries. Results: The number of veins flowing directly to the splenic vein varied from seven to twenty two (14.52 ± 3.53). Pancreatic branches of the pancreatic tail flowing to the segmentary veins of the spleen were found in 25 of the anatomical pieces studied (65.79%). These branches varied from one to four, predominating one branch (60%) and two branches (24%).