DOCSLIB.ORG

Bbm:978-3-319-12105-5/1.Pdf

Total Page：16

File Type：pdf, Size：1020Kb

Download full-text PDF Read full-text
Bbm:978-3-319-12105-5/1.Pdf Index A Autosomal recessive disorder. See Cystine stones; Absorptive hypercalcicuria , 35, 51 Cystinuria; 2,8-Dihydroxyadenine (DHA) ACA. See Affordable Health Care Act (ACA) stones Acetaminophen , 163, 165 Acetazolamide (Diamox) , 61, 81 Acetohydroxamic acid (AHA) , 38, 126 B Acute kidney injury (AKI) , 154 Bacteriuria , 76 Adenine phosphoribosyl transferase (APRT) , 39 Benign prostatic hypertrophy (BPH) , 59, 71, 76, 94, 99, Adult stone disease , 79 140, 141, 150, 216 Afferent arterioles , 20 Beta agonists , 164–165 Affordable Health Care Act (ACA) Billing and coding process advantages , 236–237 coinsurance , 232 catastrophic coverage plan , 236 copay , 232–233 disadvantages , 237–238 CPT ® , 229–230 four metal tiers plans , 236 E & M services , 232 health care law , 235 global period fee , 230 health services , 235 ICD-9 codes , 229 impact of , 235 ICD-10 coding system , 229 AHA. See Acetohydroxamic acid (AHA) modifi er , 230–232 ALARA (“As Low As Reasonably Achievable”) , 81 questionnaires , 233 Allopurinol (Zyloprim) , 52, 58, 64, 124 Bladder , 20 Aloe Vera , 184 exstrophy , 23, 24 Alpha-1 blockers , 76–77 function , 21 Alpha-mercaptopropionylglycine (Alpha-MPG) , 58 overactive , 165, 178 American Society of Nephrology and the Endourological stones Society , 123 augmentation cystoplasty , 148–149 American Urological Association (AUA) guidelines in children , 80 diet therapies , 219 gender , 71 evaluation , 219 between prostate , 151 follow up , 219–220 symptoms , 27 pharmacologic therapies , 219 treatment , 27, 150 American Urological Association Meeting AUA (2014) , Urat-1, clinical device , 29 123 structure and location , 21–22 Ammonium acid urate calculi , 61, 70 surgical procedure Angiotensin converting enzyme (ACE) , 20 Greenlight™ laser ablation of prostate , 140–141 Antegrade progression , 92 open surgical cystotomy with stone Antibiotics , 38, 62, 120, 211 removal , 141 Anticholinergics , 156, 163–164 transurethral stone removal (cystolitholapaxy) , Anticoagulants , 120 139–140 Anti-epileptic drugs (AEDs) , 64 treatment , 139 Antimicrobial therapy , 38 Blood urea nitrogen (BUN) , 170, 172 Appendicitis , 3, 93, 207 Body mass index (BMI) , 64, 86 Aspirin , 63, 94, 119, 120, 130, 160, 162, 172 BPH. See Benign prostatic hypertrophy (BPH) Asymptomatic stone. See Incidental stones Bucillamine (Rimatil) , 58 Atkins diet , 195, 203–204 Bumetanide , 61 D.A. Schulsinger (ed.), Kidney Stone Disease: Say NO to Stones!, 241 DOI 10.1007/978-3-319-12105-5, © Springer International Publishing Switzerland 2015 242 Index C IV dye (contrast) , 105 Calcium , 35, 184 multiple non-contrast renal protocol , 104 Calcium oxalate (CaOx) stones , 188 non contrast Calcium oxalate dihydrate (COD) advantage and disadvantage , 105 composition and fragility , 46 patient indications , 104–105 fragility of stones , 144 right lower pole renal stone , 148 radiopaque , 46 Congenital anomalies , 81 shape, size and color , 37, 43–44 Cranberry , 184 urinary , 44 Creatinine (Cr) , 172 Calcium oxalate monohydrate (COM) , 36 Crixivan ® stones , 40, 62 composition and fragility , 46 Crohn’s disease , 71 radiopaque , 46 Crystallization , 178 shape, size and color , 44 Current Procedural Terminology (CPT® ) , 229–230 Calcium phosphate stones , 37 Cyclooxygenase (COX) , 162 radiopaque , 46 Cystine dimethyl ester (CDME) , 218 shape, size and color , 44, 45 Cystine, Ornithine, Lysine and Arginine (COLA) , 38 Calcium stones. See also Calcium oxalate Cystine stones , 35 dihydrate (COD); Calcium oxalate autosomal recessive inborn error , 38 monohydrate (COM) composition and fragility , 46 associated with obesity , 86 poorly radiopaque , 46 hypercalciuria , 35 prevalence of , 38 metabolic factors , 35 risk factor , 38 oxalate dihydrate , 35, 37 shape, size and color , 45–46 phosphate , 35, 37 treatment , 38–39 surgical therapy , 35 Cystinuria treatment , 36 metabolic and hereditary factors , 53–54 Calcium + Vitamin D 3 (OsCal) , 125 stone formation , 58 Cancer , 52 Cystoscope , 157 leukemia , 57 lymphoma , 57 metastatic , 172 D obesity-related conditions , 85 Darifenacin (Enablex) , 164 patient, ureteral stent in , 153 DASH (Dietary Approaches to Stop Hypertension) prostate , 115 diet , 201–202, 209 Captopril , 58, 126 Dehydration , 51, 83, 175–178 Carbonic anhydrase inhibitors , 61 Diabetes , 57 Cardiovascular disease , 71 Diabetic nephropathy , 100 Celebrex (celecoxib) , 58 Diarrhea , 6, 56, 59, 61, 64, 70, 124, 125, 175, 182, 197 Chelating agent , 54, 58 Diet. See also Dietary patterns Chemolysis , 38 kidney stones, infl uence of , 187 Chemotherapeutic agents , 63 Dietary factors Children with stones calcium oxalate stones , 188 composition , 80 condition in digestive tract , 191–192 inherited abnormality , 83 into digestive tract , 190–191 prevention , 82 high urinary calcium excretion , 188–190 risk factors , 80–81 high urinary oxalate excretion , 190 symptomatology , 81 low urinary citrate excretion , 192 treatment , 82 low urinary magnesium excretion , 192–193 types of , 79–80 calcium phosphate stones work-up , 81 high urinary calcium excretion , 195 Chlorthaladone , 124 low urinary citrate excretion , 194 Cholecystitis , 3 case study , 199 Cinnamon , 184 cystine stones , 196 Ciprofl oxacin , 62 diabetes , 196–197 Citric acid (Citrate) , 182–183 macular degeneration , 198 Colchicine , 58 malabsorption , 197 Complete ureteral duplication , 23 nutrition therapy, goals for , 187–188 Computed tompography (CT scan) osteoporosis , 197–198 Index 243 special medical situations , 196 kidney stone , 135 uric acid stones ureter stones , 138–139 high urinary uric acid excretion , 195 urinary stone treatment , 29–30 low (acid) urine pH , 195–196 urine volume ( see Hydration) Dietary patterns F lithogenic diets ( see Lithogenic diets) Flank pain/renal colic , 92 with lower risk of stones Flomax ® , 130 DASH diet , 201–202 Fluids Mediterranean diet , 202 consumption raw food diets , 203 kidney stone formation, prevention of , 179 vegetarian diet , 203 requirement for , 177–178 Weight Watchers diet , 202–203 timings , 178 Dietary supplements types of cautious about certain , 184 citrus based , 176 citric acid , 182–183 coffee , 177 magnesium , 181–182 food , 176 NSF® International seal , 184 soda , 177 Omega-3 fats , 183 tea , 177 potassium , 182 water , 176 pumpkin seeds , 183 Food and Drug Administration (FDA) , 184 rose hips , 183 Furosemide , 61 safety , 183–184 USP® seal , 184 vitamin B6 (pyridoxine) , 183 G 2,8-Dihydroxyadenine (DHA) stones , 39 Gastric/duodenal ulcer , 3 radiolucent calculi , 46, 143 Gastro-Intestinal Stromal Tumor (GIST) , 5–6 Diuretics , 63 Gender , 73 Dornier Compact Delta II lithotripter , 135 age , 69 Dornier HM1 , 29 anatomical barrier , 71 D-Penicillamine , 125 bladder stones , 71 Drosphila melanogaster , 217 case study , 73 Dual-energy CT (DECT) , 215–216 diseases Dukan diet , 205 cardiovascular disease , 71 Crohn’s disease , 71 eating disorders , 70–71 E laxative abuse , 70 Ectopic kidney , 145, 146 osteoporosis , 71 Elderly with stones factors diagnosis , 83 post-menopause , 72 management , 83–84 during pregnancy , 72 prevalence , 83 vasectomy , 72 risk factors , 83 hormones , 72 Electrohydraulic lithotripter , 29–30 risk factors , 72 Electrohydrolic force , 77 statistics , 69 Endoscopy equipment , 141–142 stone types , 70 Endourology fellowship , 113, 114 symptoms , 70 Ephedrine , 62–63 Gerota’s Fascia , 20 Epididymitis , 208 Good Manufacturing Practices (GMPs) , 184 Epispadias , 25 Gout , 57–58 Estrogen , 58, 72 Gross hematuria , 94 Ethylene glycol , 217 Guaifenesin stone , 63 Evaluation and management (E & M) services , 232 Extracorporeal shock wave lithotripsy (ESWL) technique , 77 complication , 148 H lower pole stone , 147 Health care plan , 238–239 stone(s) fragment , 82 Hematuria , 173 surgical procedure Heterologous nucleation , 52 244 Index Horeshoe kidney , 147 Intractable pain , 62, 77, 92, 98, 116, 120, 156 Hounsfi eld units (HU) , 46 Intramural ureter , 92 24 hours urine test Intravenous Urography (IVU) , 106, 107 appropriate time for collecting , 170 determination of urine volume , 179 method , 171 J multiple factors on , 171–172 Jackson Pratt drains , 12 poor man’s , 171 preparation , 170–171 results , 171 K urine container , 170 Ketorolac , 163 “World Series” of urine tests , 170 Kidney Hydration , 129, 179 congenital anomalies fl uids intake ( see Fluids) horseshoe kidney , 22 goals , 178 renal ectopia , 23 management function , 20 fl uid by numbers , 179 retroperitoneum , 20 urine by colors , 178–179 structure and location , 20–21 Hydrochlorothiazide (HCTZ) , 51, 124 Kidney stones. See also Stones Hydromorphone (Dilaudid) , 161–162 in animal models , 216–217 Hydronephrosis , 92, 104, 105, 207 in children , 79–80 Hydroureteronephrosis , 75, 76, 81 management of Hydroxyapatite. See Calcium phosphate stones AUA guidelines for , 219–220 Hyperoxaluria , 35, 52–53, 125 imaging technology , 215–216 Hyperparathyroidism , 58, 172 new medications and probiotics , 217–218 Hypertension , 57, 61, 63, 83, 85, 126, 135, 165, 201, SDM , 216 202, 209 preventing formation of Hyperuricosuria , 35, 36 complimentary medicine ( see Dietary metabolic and hereditary factors , 52 supplements) stone formation , 59 nutrition recommendations ( see Dietary Hypocitraturia , 35, 51 factors) medical therapy , 124 robotic approaches , 218–219 metabolic and hereditary factors , 52 treatment options , 215 stone formation , 59 Klebsiella , 93 Hypospadias , 23–24 K. pneumonia , 38 Hypoxanthine-guanine phosphoribosyl transferase KUB (Kidneys, Ureters and Bladder)
Load more

Recommended publications
  • Contemporary Concepts and Imaging Findings in Paediatric Cystic Kidney Disease, P
    Contemporary concepts and imaging findings in paediatric cystic kidney disease, p. 65-79 HR VOLUME 3 | ISSUE 2 J Urogenital Imaging Review Contemporary concepts and imaging findings in paediatric cystic kidney disease Vasiliki Dermentzoglou, Virginia Grigoraki, Maria Zarifi Department of Radiology, Agia Sophia Children's Hospital, Athens, Greece Submission: 31/1/2018 | Acceptance: 27/5/2018 Abstract The purpose of this article is to review the renal cyst- cystic tumours. Imaging plays an important role, as ic diseases in children with regard to classification, it helps to detect and characterise many of the cyst- genetic background, antenatal and postnatal ultra- ic diseases based primarily on detailed sonographic sonographic appearances and evolution of findings analysis. Diagnosis can be achieved in many condi- in childhood. Numerous classifications exist, even tions during foetal life with ultrasound (US) and in though the prevailing one divides cystic diseases in selected cases with foetal magnetic resonance imag- hereditary and non-hereditary. Contemporary data ing (MRI). After birth, combined use of conventional are continuously published for most of the sub-cat- and high-resolution US allows detailed definition of egories. Genetic mutations at the level of primary the extent and evolution of kidney manifestations. cilia are considered a causative factor for many re- Appropriate monitoring with US seems crucial for nal cystic diseases which are now included in the patients’ management. In selected cases (e.g. hepa- spectrum of ciliopathies. Genetic mapping has doc- tobiliary disease, cystic tumours) primarily MRI and umented gene mutations in cystic diseases that are occasionally computed tomography (CT) are valua- generally considered non-hereditary, as well as in ble diagnostic tools.
    [Show full text]
  • Renal Relevant Radiology: Use of Ultrasonography in Patients with AKI
    Renal Relevant Radiology: Use of Ultrasonography in Patients with AKI Sarah Faubel,* Nayana U. Patel,† Mark E. Lockhart,‡ and Melissa A. Cadnapaphornchai§ Summary As judged by the American College of Radiology Appropriateness Criteria, renal Doppler ultrasonography is the most appropriate imaging test in the evaluation of AKI and has the highest level of recommendation. Unfortunately, nephrologists are rarely specifically trained in ultrasonography technique and interpretation, and *Division of Internal Medicine, Nephrology, important clinical information obtained from renal ultrasonography may not be appreciated. In this review, the University of Colorado strengths and limitations of grayscale ultrasonography in the evaluation of patients with AKI will be discussed and Denver Veterans with attention to its use for (1) assessment of intrinsic causes of AKI, (2) distinguishing acute from chronic kidney Affairs Medical Center, 3 Denver, Colorado; diseases, and ( ) detection of obstruction. The use of Doppler imaging and the resistive index in patients with † AKI will be reviewed with attention to its use for (1) predicting the development of AKI, (2) predicting the Department of 3 Radiology and prognosis of AKI, and ( ) distinguishing prerenal azotemia from intrinsic AKI. Finally, pediatric considerations in §Department of Internal the use of ultrasonography in AKI will be reviewed. Medicine and Clin J Am Soc Nephrol 9: 382–394, 2014. doi: 10.2215/CJN.04840513 Pediatrics, Nephrology, University of Colorado Denver, Denver, Colorado; and Introduction structures on ultrasonography images. The renal cap- ‡Department of Renal ultrasonography is typically the most appro- sule consists of thin fibrous tissue, which is next to Radiology, University of priate and useful radiologic test in the evaluation of fat, and thus the kidney often appears to be surroun- Alabama at Birmingham, patients with AKI (1).
    [Show full text]
  • 10 Renal Infarction 13 Renal
    1 Kidneys and Adrenals P. Hein, U. Lemke, P. Asbach Renal Anomalies 1 Angiomyolipoma 47 Medullary Sponge Kidney 5 Hypovascular Renal Cell Accessory Renal Arteries 8 Carcinoma 50 Renal Artery Stenosis (RAS) 10 Oncocytoma 52 Renal Infarction 13 Renal Cell Carcinoma 54 Renal VeinThrombosis 16 Cystadenoma and Cystic Renal Renal Trauma/Injuries 19 Cell Carcinoma 59 Acute Pyelonephritis 23 Renal Lymphoma 62 Chronic Pyelonephritis 26 Renal Involvement in Xanthogranulomatous Phakomatoses 65 Pyelonephritis 29 Kidney Transplantation I 67 Pyonephrosis 31 Kidney Transplantation II 70 Renal Abscess 33 Adrenocortical Hyperplasia 73 Renal Tuberculosis 36 Adrenal Adenoma 76 Renal Cysts I Adrenocortical Carcinoma 81 (Simple, Parapelvic, Cortical) 38 Pheochromocytoma 85 Renal Cysts II Adrenal Metastasis 88 (Complicated, Atypical) 41 Adrenal Calcification 91 Polycystic Kidney Disease 44 AdrenalCysts 93 2 The Urinary Tract P. Asbach, D. Beyersdorff Ureteral Duplication Anomalies.. 96 BladderDiverticula 127 Megaureter 99 Urothelial Carcinoma Ureterocele 101 oftheBIadder 129 Anomalies of the Male Urethral Strictures 133 Ureteropelvicjunction 103 Female Urethral Pathology 135 Vesicoureteral Reflux (VUR) 106 Vesicovaginal and Vesicorectal Acute Urinary Obstruction 109 Fistulas 138 Chronic Urinary Obstruction 112 The Postoperative Lower Retroperitoneal Fibrosis 115 Urinary Tract 140 Urolithiasis 118 BladderRupture 142 Ureteral Injuries 122 Urethral and Penile Trauma 145 Urothelial Carcinoma of the Renal Peivis and Ureter 124 Bibliografische Informationen digitalisiert durch http://d-nb.info/987804278 gescannt durch Contents 3 The Male Cenitals U. Lemke. D. Beyersdorff, P. Asbach Scrotal Anatomy 148 Varicocele 169 Hydrocele 151 Benign Prostatic Hyperplasia Testicular and Epididymal Cysts 153 (BPH) 171 Testicular Microlithiasis 155 Prostatitis 174 Epididymoorchitis 157 Prostate Cancer 176 Testicular Tumors 160 Penile Cavernosal Fibrosis 179 Testicular Torsion 164 Peyronie Disease 181 Testicular Trauma 166 Penile Malignancies 184 4 The Female Cenitals U.
    [Show full text]
  • Autosomal Dominant Medullary Cystic Kidney Disease (ADMCKD)
    Autosomal Dominant Medullary Cystic Kidney Disease (ADMCKD) Author: Doctor Antonio Amoroso1 Creation Date: June 2001 Scientific Editor: Professor Francesco Scolari 1Servizio Genetica e Cattedra di Genetica, Istituto per l'infanzia burlo garofolo, Via dell'Istria 65/1, 34137 Trieste, Italy. [email protected] Abstract Keywords Disease name Synonyms Diagnostic criteria Differential diagnosis Prevalence Clinical description Management Etiology Genetic counseling References Abstract Autosomal dominant medullary cystic kidney disease (ADMCKD) belongs, together with nephronophthisis (NPH), to a heterogeneous group of inherited tubulo-interstitial nephritis, termed NPH-MCKD complex. The disorder, usually first seen clinically at an average age of 28 years, is characterized by structural defects in the renal tubules, leading to a reduction of the urine–concentrating ability and decreased sodium conservation. Clinical onset and course of ADMCKD are insidious. The first sign is reduced urine– concentrating ability. Clinical symptoms appear when the urinary concentrating ability is markedly reduced, producing polyuria. Later in the course, the clinical findings reflect the progressive renal insufficiency (anemia, metabolic acidosis and uremic symptoms). End-stage renal disease typically occurs in the third-fifth decade of life or even later. The pathogenesis of ADMCKD is still obscure and how the underlying genetic abnormality leads to renal disease is unknown. ADMCKD is considered to be a rare disease. Until 2000, 55 affected families had been described. There is no specific therapy for ADMCKD other than correction of water and electrolyte imbalances that may occur. Dialysis followed by renal transplantation is the preferred approach for end-stage renal failure. Keywords Autosomal dominant medullary cystic disease, medullary cysts, nephronophthisis, tubulo-interstitial nephritis Disease name Diagnostic criteria Autosomal dominant medullary cystic kidney The renal presentation of MCKD is relatively disease (ADMCKD) non-specific.
    [Show full text]
  • Irish Rare Kidney Disease Network (IRKDN)
    Irish Rare kidney Disease Network (IRKDN) Others Cork University Mater, Waterford University Dr Liam Plant Hospital Galway Dr Abernathy University Hospital Renal imaging Dr M Morrin Prof Griffin Temple St and Crumlin Beaumont Hospital CHILDRENS Hospital Tallaght St Vincents Dr Atiff Awann Rare Kidney Disease Clinic Hospital University Hospital Prof Peter Conlon Dr Lavin Prof Dr Holian Little Renal pathology Lab Limerick University Dr Dorman and Hospital Dr Doyle Dr Casserly Patient Renal Council Genetics St James Laboratory Hospital RCSI Dr Griffin Prof Cavaller MISION Provision of care to patients with Rare Kidney Disease based on best available medical evidence through collaboration within Ireland and Europe Making available clinical trials for rare kidney disease to Irish patients where available Collaboration with other centres in Europe treating rare kidney disease Education of Irish nephrologists on rare Kidney Disease. Ensuring a seamless transition of children from children’s hospital with rare kidney disease to adult centres with sharing of knowledge of rare paediatric kidney disease with adult centres The provision of precise molecular diagnosis of patients with rare kidney disease The provision of therapeutic plan based on understanding of molecular diagnosis where available Development of rare disease specific registries within national renal It platform ( Emed) Structure Beaumont Hospital will act as National rare Kidney Disease Coordinating centre working in conjunction with a network of Renal unit across the country
    [Show full text]
  • Urology 2002
    UROLOGY Dr. S. Herschorn Ryan Groll, Alexandra Nevin and David Rebuck, chapter editors Gilbert Tang, associate editor HISTORY AND PHYSICAL . 2 PENIS AND URETHRA . .26 Peyronie's Disease KIDNEY AND URETER . 2 Priapism Renal Stone Disease Phimosis Stone Types Paraphimosis Benign Renal Tumours Penile Tumours Malignant Renal Tumours Erectile Dysfunction Carcinoma of the Renal Pelvis and Ureter Urethritis Renal Trauma Urethral Syndrome Urethral Stricture BLADDER . 9 Urethral Trauma Bladder Carcinoma Neurogenic Bladder HEMATURIA . .30 Incontinence Urinary Tract Infections (UTI) INFERTILITY . .31 Recurrent/Chronic Cystitis Interstitial Cystitis PEDIATRIC UROLOGY . .32 Bladder Stones Congenital Abnormalities Urinary Retention Hypospadias Bladder Trauma Epispadias-Exstrophy Bladder Catheterization Antenatal Hydronephrosis Posterior Urethral Valves PROSTATE . 16 UPJ Obstruction Benign Prostatic Hyperplasia (BPH) Vesicoureteral Reflux (VUR) Prostate Specific Antigen (PSA) Urinary Tract Infection (UTI) Prostatic Carcinoma Enuresis Prostatitis/Prostatodynia Nephroblastoma Cryptorchidism / Ectopic Testes SCROTUM AND CONTENTS . .21 Ectopic Testes Epididymitis Ambiguous Genitalia Orchitis Torsion SURGICAL PROCEDURES . .35 Testicular Tumours Hematocele REFERENCES . .36 Hydrocele Spermatocele/Epididymal Cyst Varicocele Hernia MCCQE 2002 Review Notes Urology – U1 HISTORY AND PHYSICAL HISTORY ❏ pain: location (CVA, genitals, suprapubic), onset, quality (colicky, burning), severity, radiation ❏ associated symptoms: fever, chills, weight loss, nausea, vomiting
    [Show full text]
  • Kidney Cysts Fact Sheet
    Last Reviewed August 2016 Page 1 Prevent, Detect, Support. Fact sheet Kidney Cysts Cystic kidney disease causes pockets Benign cysts are non-cancerous This fact sheet covers the three of clear, watery fluid (cysts) to form in sacs filled with clear, watery fluid. most common types of cystic the kidneys. The cysts slowly replace They range from small blisters to kidney disease: healthy kidney tissue causing the large sacs filled with several litres • Polycystic kidney disease kidneys to become larger. This makes of fluid. Having a few cysts is not it harder for your kidneys to work unusual, particularly as you become • Medullary cystic disease properly. older. These cysts don’t usually need • Medullary sponge kidney treatment and do not mean that you have cystic kidney disease. Cysts can develop if you have had long-term kidney problems, such as kidney failure and have been on dialysis for a long time. This is called Acquired Cystic Kidney Disease (ACKD). These cysts are not inherited Kidney cysts Normal kidney (passed on from your parents) and do not usually need treatment. What is polycystic kidney disease (PKD)? Polycystic Kidney Disease (PKD) is Autosomal Dominant PKD - This is Autosomal Recessive PKD - This is the most common inherited cystic the most common inherited form of a less common form of inherited PKD kidney disease. It causes the growth PKD. If you have autosomal dominant where both parents have to carry of thousands of cysts (fluid filled sacs) PKD you have a one in two chance this gene. In this case there is a one in your kidneys.
    [Show full text]
  • PQI 12 Urinary Tract Infection Admission Rate
    AHRQ QITM Version 4.5, Prevention Quality Indicators #12, Technical Specifications, Urinary Tract Infection Admission Rate www.qualityindicators.ahrq.gov Urinary Tract Infection Admission Rate Technical Specifications Prevention Quality Indicators #12 (PQI #12) AHRQ Quality IndicatorsTM, Version 4.5, May 2013 Area-Level Indicator Type of Score: Rate Description Admissions with a principal diagnosis of urinary tract infection per 100,000 population, ages 18 years and older. Excludes kidney or urinary tract disorder admissions, other indications of immunocompromised state admissions, obstetric admissions, and transfers from other institutions. [NOTE: The software provides the rate per population. However, common practice reports the measure as per 100,000 population. The user must multiply the rate obtained from the software by 100,000 to report admissions per 100,000 population.] Numerator Discharges, for patients ages 18 years and older, with a principal ICD-9-CM diagnosis code for urinary tract infection. [NOTE: By definition, discharges with a principal diagnosis of urinary tract infection are precluded from an assignment of MDC 14 by grouper software. Thus, obstetric discharges should not be considered in the PQI rate, though the AHRQ QITM software does not explicitly exclude obstetric cases.] ICD-9-CM Urinary tract infection diagnosis codes: 59010 AC PYELONEPHRITIS NOS 59081 PYELONEPHRIT IN OTH DIS 59011 AC PYELONEPHR W MED NECR 5909 INFECTION OF KIDNEY NOS 5902 RENAL/PERIRENAL ABSCESS 5950 ACUTE CYSTITIS 5903 PYELOURETERITIS CYSTICA
    [Show full text]
  • UNILATERAL MEDULLARY SPONGE KIDNEY FIG. 1 Multiple Right
    Case Report doi: 10.22374/jeleu.v1i1.12 UNILATERAL MEDULLARY SPONGE KIDNEY Rodrigo Neira S, Diego Barrera C, Gastón Astroza E Facultad de Medicina, Pontifi cia Universidad Católica de Chile, Santiago, Chile. Corresponding Author Gastón Astroza E, MD: gaeulufi @gmail.com Submitted: May 15, 2018. Accepted: July 5, 2018. Published: August 21, 2018. ABSTRACT A 40-year-old female with an allergy to medium contrast, presented with bilateral loin pain and renal colic type radiation. Image study with non-contrast computerized tomography and magnetic resonance imaging revealed multiple nephrolithiasis and a complex cyst at lower pole of the right kidney and no findings in the left one. The patient was treated with laser lithotripsy by flexible ureterorenoscopy revealing multiple lithiasis within cystic dilatations of the urothelium. The complex cyst was submitted to partial nephrec- tomy. Finally, biopsy revealed renal parenchyma with polycystic changes in the external corticomedullary area. All findings were compatible with unilateral medullary sponge kidney diagnosis. CLINICAL CASE A 40-year-old female patient presented with a his- tory of insulin resistance (treated with metformin), allergy to contrast medium, and maternal urolithia- sis. The patient consulted for bilateral loin pain and right renal colic pain. Non-contrast computerized tomography (NCCT) (Figures 1 and 2) demonstrated multiple right nephrolithiasis and a 3-cm hypodense right lower pole lesion with some calcifications. This was diagnosed as a complex cyst. Related to this cyst, a magnetic resonance imaging (MRI) of the abdo- men was requested which showed a 3.5 cm Bosniak 3 right renal cyst. Resolution of the lithiasis was decided by means of flexible ureterorenoscopy (fURS) with laser lithotripsy.
    [Show full text]
  • Medullary Sponge Kidney with Distal Renal Tubular Acidosis
    Open Access SAJ Case Reports CASE REPORT ISSN: 2375-7043 Medullary Sponge Kidney with Distal Renal Tubular Acidosis: A Case Report and Review of the Literature Jamshidian M1*, Coombs RJ2, Ratnam S1 and Malhotra D1 1Department of Nephrology, University of Toledo Medical Center, 3000 Arlington Ave, Toledo, United States 2Department of Radiology, University of Toledo Medical Center, 3000 Arlington Ave, Toledo, United States *Corresponding author: Jamshidian M, Department of Nephrology University of Toledo Medical Center, 3000 Arlington Ave, Toledo, OH 43614, United States, E-mail: [email protected] Citation: Jamshidian M, Coombs RJ, Ratnam S, Malhotra D (2018) Medullary Sponge Kidney with Distal Renal Tubular Acidosis: A Case Report and Review of the Literature. SAJ Case Rep 5: 204 Article history: Received: 22 December 2017, Accepted: 28 May 2018, Published: 30 May 2018 Abstract Medullary sponge kidney (MSK) is a rare disease, with a prevalence of 0.0002 – 0.0005%, characterized by cystic dilations of the collecting ducts. Patients with MSK have the propensity for recurrent formation of kidney stones, nephrocalcinosis and urinary tract infections (UTIs). Unfamiliar to many practitioners and commonly asymptomatic, MSK patients often remain undiagnosed. In addition to a comprehensive review of the literature published to date, we report a case of how we diagnosed MSK coexisting with distal renal tubular acidosis (dRTA) in an adult patient who presented with severe hypokalemia and hyperchloremic metabolic acidosis. Keywords: Renal Tubular Acidosis; Nephrocalcinosis; Medullary Sponge Kidney; Hypokalemia; Metabolic Acidosis Introduction Medullary sponge kidney (MSK) is a rare disorder, usually congenital, defined by cystic dilations of the precalyceal collecting ducts within the medullary pyramids of the kidney [1-3].
    [Show full text]
  • Medullary Cystic Disease of the Kidney: Its Occurrence in Two Siblings
    Postgrad. med. J. (October 1968) 44, 792-798. Postgrad Med J: first published as 10.1136/pgmj.44.516.792 on 1 October 1968. Downloaded from Medullary cystic disease of the kidney: its occurrence in two siblings SATYA PAUL HANDA* ROBERT TENNANT M.B. M.D. The Hartford Hospital, Hartford, Connecticut Summary Necropsy findings reveal gross or microscopic Two cases of medullary cystic disease of the cysts in the medullary region of the kidney. At kidney in two siblings are presented. In both present there is an ample evidence that medullary siblings there was an insidious onset of azotaemia cystic disease of the kidney and familial juvenile and anaemia at an early age. The urinalyses were nephronophthisis represent one and the same dis- normal except for a trace of proteinuria and order (Mongeau & Worthen, 1967; Strauss & persistent low specific gravity. The kidneys were Sommers, 1967). small by radiological studies and this was proved The occurrence of medullary cystic disease in at necropsy. The gross microscopic appearances two siblings prompted the present report. of the kidneys were consistent with medullary cystic disease. The literature on this subject and Case reports current views on the similarities between familial Protected by copyright. juvenile nephronophthisis and this condition are Case 1 discussed. R.B., a 20-year-old white male, was admitted to Hartford Hospital for the first time because of Introduction progressive weakness, headache and epigastric Medullary cystic disease of the kidney, a pro- pain associated with vomiting. gressive renal disorder occuring in children and The patient had had enuresis until the age of young adults, first described by Smith & Graham 7 and nocturia during his entire life.
    [Show full text]
  • Diseases and Disorders of the Urinary System
    Chapter 10 Diseases and Disorders of the Urinary System Learning Objectives After studying this chapter, you should be able to L Describe the anatomy and the functions of kidneys, nephrons, ureters, urinary bladder, and urethra L Identify the etiology, signs and symptoms, diagnostic tests, and treatment for acute kidney injury and other acute and inflammatory diseases of the urinary system L Know the etiology, and describe the signs and symptoms, diagnostic tests, and treatment of urinary tract infections L Identify the etiology, signs and symptoms, diagnostic tests, and treatment for chronic kidney disease, hypertensive kidney disease, diabetic nephropathy, nephrotic syndrome, end-stage renal disease, and other chronic diseases of the urinary system L Describe kidney dialysis L Recognize the etiology, signs and symptoms, and modes of treat- Histopathology of kidney showing ment for renal cell carcinoma, Wilm’s tumor, and bladder cancer nodular glomerulosclerosis characteristic of diabetes mellitus. (Courtesy of L Describe common congenital disorders of the urinary system the Centers for Disease Control and Prevention/Dr. Edwin P. Wing, Jr., 1974) L Describe common age-related diseases of the urinary system Fact or Fiction? Kidney stones occur only in the kidneys. Fiction: Kidney stones may form anywhere within the urinary system, but they usually form in the renal pelvis or calyces of the kidney and they can lodge in the ureters. 198 M10_ZELM4744_08_SE_C10.indd 198 04/04/14 3:20 PM Disease Chronicle What’s in a Name? Many anatomical structures once bore the names of the scientists who first discovered them. Recently anatomists have revised anatomical nomenclature and we no longer formally name organs after scientists.
    [Show full text]