Childhood Psychotic Experiences and Objective and Subjective Sleep Problems
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2020 Question Book
2020 QUESTION BOOK 13TH EDITION WHO WE ARE Welcome to the thirteenth edition of the Ninja’s Guide to PRITE! Loma Linda University Medical Center is located in sunny Southern California. about 60 miles east of Los Angeles. A part of the Adventist Health System, we provide patient care in one of the largest non-profit health systems in the nation. Loma Linda's mission is to excel in medical education, global healthcare, and community outreach, all under a central tenant: "To Make Man Whole." At the Loma Linda Department of Psychiatry, our residents are trained in many diverse patient care settings. As an official World Health Organization Collaboration Center, our department funds resident electives in Global Mental Health at locations around the world. Additionally, our residents can participate in national and international disaster relief on the LLU Behavioral Health Trauma Team. We were proud to welcome our first group of Child and Adolescent Psychiatry fellows in the Summer of 2019 and work collaboratively with 3 other residency programs within the region. Our residency didactic education is constantly evolving based upon resident feedback, and our residents have the opportunity to aid in course development. More than anything, our residency fosters an environment where residents and faculty treat each other like family. Our faculty are dedicated to resident education and professional development. We believe in "taking 'No' off the table", encouraging innovative change, and passionately supporting our residents to achieve anything they set their minds to. For over a decade our residents have volunteered their time to create The Ninja's Guide to PRITE at our Annual Ninja PRITE Workshop. -
Sleep Environment and Non-Rapid Eye Movement-Related Parasomnia Among Children: 42 Case Series
pISSN 2093-9175 / eISSN 2233-8853 BRIEF COMMUNICATION https://doi.org/10.17241/smr.2020.00535 Sleep Environment and Non-Rapid Eye Movement-Related Parasomnia Among Children: 42 Case Series Joohee Lee, MD, Sungook Yeo, MD, Kyumin Kim, MD, Seockhoon Chung, MD, PhD Department of Psychiatry, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Korea The purpose of this study was to identify the clinical features related to sleep environment of non- rapid eye movement (NREM)-related parasomnia. It was a retrospective medical record review of 42 children. We investigated demographic information, sleep pattern, sleep environment, and the mother’s dysfunctional beliefs about the child’s sleep. The mean age of subjects was 6.3± 3.1. The diagnosis was night terror (n = 21), sleepwalking (n = 8), confusional arousal (n = 2), and unspeci- fied (n = 11). The average time of sleep pattern was as follow; bedtime 21:39± 0:54 pm, sleep onset time 22:13 ± 0:54 pm, wake-up time 7:37 ± 0:42 am and NREM-related parasomnia occurrence time 1:09 ± 2:04 am. The average number of co-sleeping members was 2.8. 48.5% (n = 16) mothers experienced coldness while sleeping, and 64.7% (n = 22) parents had dysfunctional beliefs about their children’s sleep. The large number of co-sleeping members, coldness mothers experienced while sleeping, and dysfunctional beliefs about their children’s sleep may influence the NREM-pa- rasomnia in children. Sleep Med Res 2020;11(1):49-52 Key WordsaaParasomnia, Sleep environment, Co-sleep, Children. INTRODUCTION Received: April 3, 2020 A significant number of children are impacted by sleep disorders, reported in 25–62% of Revised: April 27, 2020 Accepted: May 4, 2020 such children [1,2]. -
PTSD and Sleep Corporal Michael J
VOLUME 27/NO. 4 • ISSN: 1050-1835 • 2016 Research Quarterly advancing science and promoting understanding of traumatic stress Published by: Philip Gehrman, PhD National Center for PTSD University of Pennsylvania, Department of Psychiatry VA Medical Center (116D) 215 North Main Street Gerlinde Harb, PhD White River Junction Estadt Psychological Services and Vermont 05009-0001 USA PTSD and Sleep Corporal Michael J. Crescenz VA Medical Center (802) 296-5132 Richard Ross, MD, PhD FAX (802) 296-5135 Department of Veterans Affairs Medical Center and Email: [email protected] University of Pennsylvania, Department of Psychiatry All issues of the PTSD Research Quarterly are available online at: www.ptsd.va.gov Introduction Study, 52% of combat Veterans with PTSD reported a significant nightmare problem (Neylan et al., 1998). Editorial Members: PTSD is unique among mental health disorders in In a general community sample, nightmares were Editorial Director that sleep problems represent two of the diagnostic endorsed by 71% of individuals with PTSD (Leskin, Matthew J. Friedman, MD, PhD criteria of the fifth edition of the American Psychiatric Woodward, Young, & Sheikh, 2002). Posttraumatic Bibliographic Editor Association’s (APA) Diagnostic and Statistical Manual nightmares are independently associated with Misty Carrillo, MLIS of Mental Disorders (DSM-5); recurrent nightmares daytime distress and impaired functioning over and Managing Editor are part of the intrusion cluster of symptoms, and above the impact of overall PTSD severity (Levin & Heather Smith, BA Ed insomnia is a component of the arousal cluster. Nielsen, 2007; Littlewood, Gooding, Panagioti, & While these sleep problems are symptoms of PTSD, Kyle, 2016). National Center Divisions: the evidence suggests that they tend to become Executive independent problems over time, warranting sleep- Insomnia and recurrent nightmares are traditionally White River Jct VT focused assessment and treatment. -
The ICD-10 Classification of Mental and Behavioural Disorders : Clinical Descriptions and Diagnostic Guidelines
ICD-10 ThelCD-10 Classification of Mental and Behavioural Disorders Clinical descriptions and diagnostic guidelines | World Health Organization I Geneva I 1992 Reprinted 1993, 1994, 1995, 1998, 2000, 2002, 2004 WHO Library Cataloguing in Publication Data The ICD-10 classification of mental and behavioural disorders : clinical descriptions and diagnostic guidelines. 1.Mental disorders — classification 2.Mental disorders — diagnosis ISBN 92 4 154422 8 (NLM Classification: WM 15) © World Health Organization 1992 All rights reserved. Publications of the World Health Organization can be obtained from Marketing and Dissemination, World Health Organization, 20 Avenue Appia, 1211 Geneva 27, Switzerland (tel: +41 22 791 2476; fax: +41 22 791 4857; email: [email protected]). Requests for permission to reproduce or translate WHO publications — whether for sale or for noncommercial distribution — should be addressed to Publications, at the above address (fax: +41 22 791 4806; email: [email protected]). The designations employed and the presentation of the material in this publication do not imply the expression of any opinion whatsoever on the part of the World Health Organization concerning the legal status of any country, territory, city or area or of its authorities, or concerning the delimitation of its frontiers or boundaries. Dotted lines on maps represent approximate border lines for which there may not yet be full agreement. The mention of specific companies or of certain manufacturers' products does not imply that they are endorsed or recommended by the World Health Organization in preference to others of a similar nature that are not mentioned. Errors and omissions excepted, the names of proprietary products are distinguished by initial capital letters. -
Cognitive Behavioral Therapy for Insomnia Enhances Depression Outcome in Patients with Comorbid Major Depressive Disorder and Insomnia
INSOMNIA AND DEPRESSION Cognitive Behavioral Therapy for Insomnia Enhances Depression Outcome in Patients with Comorbid Major Depressive Disorder and Insomnia Rachel Manber, PhD1; Jack D. Edinger, PhD2; Jenna L. Gress, BA1; Melanie G. San Pedro-Salcedo, MA1; Tracy F. Kuo, PhD1; Tasha Kalista, MA1 1Stanford University, Stanford, CA; 2VA Medical Center and Duke University Medical Center, Durham, NC Study Objective: Insomnia impacts the course of major depressive severity index (ISI), daily sleep diaries, and actigraphy. disorder (MDD), hinders response to treatment, and increases risk for EsCIT + CBTI resulted in a higher rate of remission of depression depressive relapse. This study is an initial evaluation of adding cogni- (61.5%) than EsCIT + CTRL (33.3%). EsCIT + CBTI was also associat- tive behavioral therapy for insomnia (CBTI) to the antidepressant medi- ed with a greater remission from insomnia (50.0%) than EsCIT + CTRL cation escitalopram (EsCIT) in individuals with both disorders. (7.7%) and larger improvement in all diary and actigraphy measures of Design and setting: A randomized, controlled, pilot study in a single sleep, except for total sleep time. academic medical center. Conclusion: This pilot study provides evidence that augmenting an Participants: 30 individuals (61% female, mean age 35±18) with MDD antidepressant medication with a brief, symptom focused, cognitive- and insomnia. behavioral therapy for insomnia is promising for individuals with MDD Interventions: EsCIT and 7 individual therapy sessions of CBTI or and comorbid insomnia in terms of alleviating both depression and in- CTRL (quasi-desensitization). somnia. Measurements and results: Depression was assessed with the Keywords: Major depressive disorder, Insomnia, Cognitive behavioral HRSD17 and the depression portion of the SCID, administered by raters therapy, Remission masked to treatment assignment, at baseline and after 2, 4, 6, 8, and Citation: Manber R; Edinger JD; Gress JL; San Pedro-Salcedo MG; 12 weeks of treatment. -
Parasomnias and Antidepressant Therapy: a Review of the Literature
REVIEW ARTICLE published: 12 December 2011 PSYCHIATRY doi: 10.3389/fpsyt.2011.00071 Parasomnias and antidepressant therapy: a review of the literature Lara Kierlin1,2 and Michael R. Littner 1,2* 1 David Geffen School of Medicine at University of California Los Angeles, Los Angeles, CA, USA 2 Pulmonary, Critical Care and Sleep Medicine, VA Greater Los Angeles Healthcare System, Los Angeles, CA, USA Edited by: There exists a varying level of evidence linking the use of antidepressant medication to Ruth Benca, University of the parasomnias, ranging from larger, more comprehensive studies in the area of REM Wisconsin – Madison School of Medicine, USA sleep behavior disorder to primarily case reports in the NREM parasomnias. As such, prac- Reviewed by: tice guidelines are lacking regarding specific direction to the clinician who may be faced Ruth Benca, University of with a patient who has developed a parasomnia that appears to be temporally related to Wisconsin – Madison School of use of an antidepressant. In general, knowledge of the mechanisms of action of the med- Medicine, USA ications, particularly with regard to the impact on sleep architecture, can provide some David Plante, University of Wisconsin, USA guidance. There is a potential for selective serotonin reuptake inhibitors, tricyclic antide- *Correspondence: pressants, and serotonin–norepinephrine reuptake inhibitors to suppress REM, as well Michael R. Littner, 10736 Des Moines as the anticholinergic properties of the individual drugs to further disturb normal sleep Avenue, Porter Ranch, Los Angeles, architecture. CA 91326, USA. e-mail: [email protected] Keywords: parasomnias, REM sleep behavior disorder, non-REM parasomnias, selective serotonin reuptake inhibitors, depression INTRODUCTION and night terrors (Ohayon et al., 1999; Yeh et al., 2009). -
Early-Onset Parkinson's Disease Caused by PLA2G6 Compound
CASE REPORT published: 21 August 2019 doi: 10.3389/fneur.2019.00915 Early-Onset Parkinson’s Disease Caused by PLA2G6 Compound Heterozygous Mutation, a Case Report and Literature Review Ting Shen 1,2,3, Jing Hu 1,2, Yasi Jiang 2,3, Shuai Zhao 1,2, Caixiu Lin 1,2, Xinzhen Yin 1,2, Yaping Yan 1,2, Jiali Pu 1,2, Hsin-Yi Lai 2,3* and Baorong Zhang 1,2* 1 Department of Neurology of the Second Affiliated Hospital, Zhejiang University School of Medicine, Zhejiang University, Hangzhou, China, 2 Department of Neurology of the Second Affiliated Hospital, Interdisciplinary Institute of Neuroscience and Technology, Zhejiang University School of Medicine, Key Laboratory of Medical Neurobiology of Zhejiang Province, Zhejiang University, Hangzhou, China, 3 Key Laboratory of Biomedical Engineering of Ministry of Education, Qiushi Academy for Edited by: Advanced Studies, College of Biomedical Engineering and Instrument Science, Zhejiang University, Hangzhou, China Ruey-Meei Wu, National Taiwan University, Taiwan Reviewed by: PLA2G6 has been certified as a causative gene in patients with autosomal recessive Yih-Ru Wu, early-onset Parkinson’s disease (EOPD). We reported an EOPD case caused by Chang Gung Memorial Hospital, Taiwan PLA2G6 gene mutation, and performed neurological examination, genetic analysis, Chin-Hsien Lin, and multimodal neuroimaging to describe this phenotype. A compound heterozygous National Taiwan University, Taiwan mutation c.991G>T/c.1472+1G>A was detected in this patient. Heterozygous for the *Correspondence: c.991G>T and c.1472+1G>A were separately detected in his parents. Pathogenicity Hsin-Yi Lai [email protected] of these two mutations were predicted according to the American college of medical Baorong Zhang genetics and genomics (ACMG) guideline. -
Management Recommendations on Sleep Disturbance of Patients with Parkinson’S Disease
Consensus Management Recommendations on Sleep Disturbance of Patients with Parkinson’s Disease Chun‑Feng Liu1,2, Tao Wang3, Shu‑Qin Zhan4, De‑Qin Geng5, Jian Wang6, Jun Liu7, Hui‑Fang Shang8, Li‑Juan Wang9, Piu Chan4, Hai‑Bo Chen10, Sheng‑Di Chen7, Yu‑Ping Wang4, Zhong‑Xin Zhao11, K Ray Chaudhuri12 1Department of Neurology, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu 215004, China 2Institute of Neuroscience, Soochow University, Suzhou, Jiangsu 215004, China 3Department of Neurology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430022, China 4Department of Neurology, Xuan Wu Hospital, Capital Medical University, Beijing 100053, China 5Department of Neurology, Affiliated Hospital of Xuzhou Medical University, Xuzhou, Jiangsu 221006, China 6Department of Neurology and National Clinical Research Center for Aging and Medicine, Huashan Hospital, Fudan University, Shanghai 200040, China 7Department of Neurology and Institute of Neurology, Ruijin Hospital Affiliated to Shanghai JiaoTong University School of Medicine, Shanghai 200025, China 8Department of Neurology, West China Hospital, Sichuan University, Chengdu, Sichuan 610041, China 9Department of Neurology, Guangdong Neuroscience Institute, Guangdong General Hospital, Guangdong Academy of Medical Sciences, Guangzhou, Guangdong 510080, China 10Department of Neurology, Beijing Hospital, National Center of Gerontology, Beijing 100730, China 11Department of Neurology, Changzheng Hospital, Second Military Medical -
Dyssomnia Sleep Disorders.Pdf
Dyssomnia Sleep Disorders Dyssomnia Sleep Disorders By Yolanda Smith, BPharm Dyssomnia is a broad type of sleep disorders involving difficulty falling or remaining asleep, which can lead to excessive sleepiness during the day due to the reduced quantity, quality or timing of sleep. This is distinct from parasomnias, which involves abnormal behavior of the nervous system during sleep. Symptoms indicative a dyssomnia sleep disorder may include difficulty falling asleep, intermittent awakenings during the night or waking up earlier than usual. As a result of the reduced or disrupted sleep, most patients do not feel well rested and have less ability to perform during the day. In many cases, lifestyle habits have an impact on the disorder, including stress, physical pain or discomfort, napping during the day, bedtime or use of stimulants. Dyssomnia Sleep Disorders There are two main types of dyssomnia sleep disorders according to the origin or cause or the disorder: extrinsic and intrinsic. Both of these are covered in more detail below, in addition to general principles in the diagnosis and treatment of the disorders. Extrinsic dyssomnias are sleep disorders that originate from external causes and may include: Insomnia Sleep apnea Narcolepsy Restless legs syndrome Periodic Limb movement disorder Hypersomnia Toxin-induced sleep disorder Kleine-Levin syndrome Intrinsic dyssomnias are sleep disorders that originate from internal causes and may include: Altitude insomnia Substance use insomnia Sleep-onset association disorder Nocturnal paroxysmal dystonia Limit-setting sleep disorder Diagnosis The differential diagnosis between the types of dyssomnias usually begins with a consultation about the sleep history of the individual, including the onset, frequency and duration of sleep. -
The Co-Occurrence of Sexsomnia, Sleep Bruxism and Other Sleep Disorders
Journal of Clinical Medicine Review The Co-Occurrence of Sexsomnia, Sleep Bruxism and Other Sleep Disorders Helena Martynowicz 1, Joanna Smardz 2, Tomasz Wieczorek 3, Grzegorz Mazur 1, Rafal Poreba 1, Robert Skomro 4, Marek Zietek 5, Anna Wojakowska 1, Monika Michalek 1 ID and Mieszko Wieckiewicz 2,* 1 Department of Internal Medicine, Occupational Diseases and Hypertension, Wroclaw Medical University, 50-367 Wroclaw, Poland; [email protected] (H.M.); [email protected] (G.M.); [email protected] (R.P.); [email protected] (A.W.); [email protected] (M.M.) 2 Department of Experimental Dentistry, Wroclaw Medical University, 50-367 Wroclaw, Poland; [email protected] 3 Department and Clinic of Psychiatry, Wroclaw Medical University, 50-367 Wroclaw, Poland; [email protected] 4 Division of Respiratory Critical Care and Sleep Medicine, Department of Medicine, University of Saskatchewan, Saskatoon, SK S7N 5A2, Canada; [email protected] 5 Department of Periodontology, Wroclaw Medical University, 50-367 Wroclaw, Poland; [email protected] * Correspondence: [email protected]; Tel.: +48-660-47-87-59 Received: 3 August 2018; Accepted: 19 August 2018; Published: 23 August 2018 Abstract: Background: Sleep sex also known as sexsomnia or somnambulistic sexual behavior is proposed to be classified as NREM (non-rapid eye movement) parasomnia (as a clinical subtype of disorders of arousal from NREM sleep—primarily confusional arousals or less commonly sleepwalking), but it has also been described in relation to REM (rapid eye movement) parasomnias. Methods: The authors searched the PubMed database to identify relevant publications and present the co-occurrence of sexsomnia and other sleep disorders as a non-systematic review with case series. -
Hypersomnia (Hypersomnolence) Symptoms and Diagnosis
Hypersomnia (Hypersomnolence) Symptoms and Diagnosis Hypersomnia (Hypersomnolence) Symptoms and Diagnosis By Yolanda Smith, BPharm Hypersomnia, also known as hypersomnolence, is a condition involving excessive daytime sleepiness or prolonged nighttime sleep on a recurring basis. Adolescents and young adults are most likely to be affected by the condition. It often causes affected individuals to take repeated naps throughout the day, which may disrupt other activities, such as work, study or social activities. These naps typically only provide temporary relief of symptoms and the desire to nap returns shortly afterwards. Common Symptoms It is common for people with hypersomnia to have difficulty waking up, particularly after a long sleep. They may feel disorientated and confused, which can continue for several hours in some patients. Excessive daytime sleepiness is the defining symptom of hypersomnia, despite getting a full night’s sleep. This may inhibit affected individuals from participating in daily routines or events. Additionally, it can be more difficult for them to maintain normal function in family, social and work environments. It can cause affected individuals to perform poorly and may lead to distress about other areas of their life. In particular, patients affected by hypersomnia are more likely to suffer from depression and anxiety than the general population. Although not all patients experience other signs and symptoms, hypersomnia may also be associated with: Anxiety Agitation Clouded thought processes and decision-making Depression Hallucinations Low energy levels Reduced appetite Reduced memory Restlessness Slow speech Diagnostic Techniques The primary diagnostic criterion for primary hypersomnia is excessive daytime sleepiness for at least one month in acute conditions or three months in persistent conditions. -
Pharmacological Sleep Interventions for Person with Dementia February 2011
Sleep and Dementia: A report on the evidence-base for non- pharmacological sleep interventions for person with dementia February 2011 Cary A Brown, Robyn Berry, Maria Tan, Anjalee Khoshla Department of Occupational Therapy, Faculty of Rehabilitation Medicine University of Alberta, Canada Correspondence: Dr Cary Brown, Associate Professor, University of Alberta [email protected] 1.0 Abstract Funding support provided by the Canadian Dementia Knowledge Translation Network (CDKTN). Any information included in this publication and/or opinions expressed therein do not necessarily reflect vies Network but remain solely those of the ABSTRACTauthors. 1 Summary Expanded Background: Disordered sleep1, prevalent in older persons (1), has been show to have significant cognitive, physical and psychological consequences (2). For example, the rate of falls in older persons with disordered sleep is demonstrated to be higher (3). The literature shows that falls, and fear of falling, have a negative impact on older persons’ self-confidence, and continued engagement in social activities. These activities are important because they provide the physical and social stimulation necessary to maintain cognitive and emotional health (4, 5). British researchers found that disordered sleep was a strong predictor of future depression in older community dwelling adults (6). In turn, depression is one of the risk factors for dementia (7). Disordered sleep patterns in persons with dementia have been found to accelerate functional decline and contribute to increased caregiver stress (8). Caregiver support and wellbeing is critical for successful management of dementia in the home setting. However, living with someone who has dementia and disordered sleep results in sleep deprivation in the caregiver as well, further decreasing caregiver resilience and coping.