AFTD 2017 Annual Education Conference Sheraton Inner Harbor Hotel | Baltimore, MD May 5, 2017, 9:00 A.M
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2020 Question Book
2020 QUESTION BOOK 13TH EDITION WHO WE ARE Welcome to the thirteenth edition of the Ninja’s Guide to PRITE! Loma Linda University Medical Center is located in sunny Southern California. about 60 miles east of Los Angeles. A part of the Adventist Health System, we provide patient care in one of the largest non-profit health systems in the nation. Loma Linda's mission is to excel in medical education, global healthcare, and community outreach, all under a central tenant: "To Make Man Whole." At the Loma Linda Department of Psychiatry, our residents are trained in many diverse patient care settings. As an official World Health Organization Collaboration Center, our department funds resident electives in Global Mental Health at locations around the world. Additionally, our residents can participate in national and international disaster relief on the LLU Behavioral Health Trauma Team. We were proud to welcome our first group of Child and Adolescent Psychiatry fellows in the Summer of 2019 and work collaboratively with 3 other residency programs within the region. Our residency didactic education is constantly evolving based upon resident feedback, and our residents have the opportunity to aid in course development. More than anything, our residency fosters an environment where residents and faculty treat each other like family. Our faculty are dedicated to resident education and professional development. We believe in "taking 'No' off the table", encouraging innovative change, and passionately supporting our residents to achieve anything they set their minds to. For over a decade our residents have volunteered their time to create The Ninja's Guide to PRITE at our Annual Ninja PRITE Workshop. -
Theory of Mind Deficit in the Behavioral Variant of Frontotemporal Dementia and in Amyotrophic Lateral Sclerosis: Why Does It Matter?
Open Access Journal of Neurology & Neurosurgery Editorial Open Access J Neurol Neurosurg Volume 1 Issue 1 - December 2015 Copyright © All rights are reserved by Marco Cavallo Theory of Mind deficit in the behavioral variant of frontotemporal dementia and in amyotrophic lateral sclerosis: Why does it matter? Marco Cavallo1,2* 1eCampus University, Novedrate (Como), Italy 2Mental Health Department - Azienda Sanitaria Locale Torino 3, Italy Submission: November 15, 2015; Published: December 02, 2015 *Corresponding author: Marco Cavallo, Neuropsychologist, Researcher at the faculty of Psychology of the eCampus University of Novedrate, Italy, Tel no: +39.3478306430; Email: Editorial stories that explicitly referred to social situations. Our results underlined the needs for further research to gain a deeper Theory of Mind (hereinafter referred to as ToM) is the ability understanding on the possible link between patient’s behavioral to explain and predict other people’s behavior by attributing problems and their limited understanding of social situations. independent mental states to them. It allows us to recognize that mental states such as beliefs, intentions, and desires play a key role in driving and monitoring human behavior. Impairment in associated with ALS, a neurodegenerative disease that from We also aimed at clarifying the nature of the ToM deficits ToM ability can be highly disabling. ToM had been extensively cognitive and neuroimaging points of view appears to share studied in neuropsychiatric conditions such as autism and some relevant -
Social Behaviour Vs. Psychiatric Features Of
Psychiatria Danubina, 2010; Vol. 22, No. 2, pp 179–182 Case report © Medicinska naklada - Zagreb, Croatia SOCIAL BEHAVIOUR VS. PSYCHIATRIC FEATURES OF FRONTOTEMPORAL DEMENTIA Clinical report of two cases Rajka Marija Liščić1 & Aleš Kogoj2 1Institute for Medical Research and Occupational Health, Zagreb, Croatia 2Department of Geriatric Psychiatry, University Psychiatric Hospital, Ljubljana, Slovenia received: 6.10.2009; revised: 12.1.2010; accepted: 9.2.2010 SUMMARY Behavioural disturbances are prominent in frontotemporal dementia (FTD), a focal, non-Alzheimer type of dementia. Although most patients with FTD present with socially inappropriate behaviour, compulsive-like acts, poor insight and disinhibition, the presence of psychiatric features including delusions, hallucinations, and paranoia can lead to a misclassification of FTD as psychiatric disorder. In the absence of cognitive deficits non-experts fail to recognize these social changes as dementia symptoms. We report two individuals who met current clinical criteria for behavioural or frontal variant FTD (bv-FTD), with the aim of distinguishing between psychotic symptoms and the often bizarre personality and behaviour change found in FTD. Also we review the literature on the noncognitive neuropsyhiatric manifestation of this disorder. Clinical findings presented, and a literature review, indicate that psychotic symptoms are rare in FTD. Better awareness of behavioural symptoms in clinical practice is necessary in order to avoid misdiagnosis of FTD as psychiatric disorder. Key words: bv-frontotemporal dementia - behavioural disturbances - psychotic symptoms * * * * * INTRODUCTION and paranoia) must be distinguished from the often bizarre personality and behaviour changes of FTD. The With baby boomers now reaching late middle age, overlap between FTD and psychotic symptoms exists degenerative diseases are becoming an increasingly and therefore may lead to misleading diagnoses. -
Psychotic Symptoms in Frontotemporal Dementia: a Diagnostic Dilemma?
International Psychogeriatrics (2015), 27:4, 531–539 C International Psychogeriatric Association 2014. This is an Open Access article, distributed under the terms of the Creative Commons Attribution licence (http://creativecommons.org/licenses/by/3.0/), which permits unrestricted re-use, distribution, and reproduction in any medium, provided the original work is properly cited. doi:10.1017/S1041610214002580 Psychotic symptoms in frontotemporal dementia: a diagnostic dilemma? ........................................................................................................................................................................................................................................................................................................................................................................................................................................................................................................................................................................................................................................... Maria Landqvist Waldö,1 Lars Gustafson,1 Ulla Passant1 and Elisabet Englund2 1Section of Geriatric Psychiatry, Department of Clinical Sciences, Lund University, Klinikgatan 22, Lund SE-221 85, Sweden 2Section of Oncology and Pathology, Department of Clinical Sciences, Lund University, Lund, SE-221 85, Sweden ABSTRACT Background: Frontotemporal dementia (FTD) constitutes a spectrum of neurodegenerative disorders associated with degeneration of, predominantly, -
The ICD-10 Classification of Mental and Behavioural Disorders : Clinical Descriptions and Diagnostic Guidelines
ICD-10 ThelCD-10 Classification of Mental and Behavioural Disorders Clinical descriptions and diagnostic guidelines | World Health Organization I Geneva I 1992 Reprinted 1993, 1994, 1995, 1998, 2000, 2002, 2004 WHO Library Cataloguing in Publication Data The ICD-10 classification of mental and behavioural disorders : clinical descriptions and diagnostic guidelines. 1.Mental disorders — classification 2.Mental disorders — diagnosis ISBN 92 4 154422 8 (NLM Classification: WM 15) © World Health Organization 1992 All rights reserved. Publications of the World Health Organization can be obtained from Marketing and Dissemination, World Health Organization, 20 Avenue Appia, 1211 Geneva 27, Switzerland (tel: +41 22 791 2476; fax: +41 22 791 4857; email: [email protected]). Requests for permission to reproduce or translate WHO publications — whether for sale or for noncommercial distribution — should be addressed to Publications, at the above address (fax: +41 22 791 4806; email: [email protected]). The designations employed and the presentation of the material in this publication do not imply the expression of any opinion whatsoever on the part of the World Health Organization concerning the legal status of any country, territory, city or area or of its authorities, or concerning the delimitation of its frontiers or boundaries. Dotted lines on maps represent approximate border lines for which there may not yet be full agreement. The mention of specific companies or of certain manufacturers' products does not imply that they are endorsed or recommended by the World Health Organization in preference to others of a similar nature that are not mentioned. Errors and omissions excepted, the names of proprietary products are distinguished by initial capital letters. -
Early-Onset Parkinson's Disease Caused by PLA2G6 Compound
CASE REPORT published: 21 August 2019 doi: 10.3389/fneur.2019.00915 Early-Onset Parkinson’s Disease Caused by PLA2G6 Compound Heterozygous Mutation, a Case Report and Literature Review Ting Shen 1,2,3, Jing Hu 1,2, Yasi Jiang 2,3, Shuai Zhao 1,2, Caixiu Lin 1,2, Xinzhen Yin 1,2, Yaping Yan 1,2, Jiali Pu 1,2, Hsin-Yi Lai 2,3* and Baorong Zhang 1,2* 1 Department of Neurology of the Second Affiliated Hospital, Zhejiang University School of Medicine, Zhejiang University, Hangzhou, China, 2 Department of Neurology of the Second Affiliated Hospital, Interdisciplinary Institute of Neuroscience and Technology, Zhejiang University School of Medicine, Key Laboratory of Medical Neurobiology of Zhejiang Province, Zhejiang University, Hangzhou, China, 3 Key Laboratory of Biomedical Engineering of Ministry of Education, Qiushi Academy for Edited by: Advanced Studies, College of Biomedical Engineering and Instrument Science, Zhejiang University, Hangzhou, China Ruey-Meei Wu, National Taiwan University, Taiwan Reviewed by: PLA2G6 has been certified as a causative gene in patients with autosomal recessive Yih-Ru Wu, early-onset Parkinson’s disease (EOPD). We reported an EOPD case caused by Chang Gung Memorial Hospital, Taiwan PLA2G6 gene mutation, and performed neurological examination, genetic analysis, Chin-Hsien Lin, and multimodal neuroimaging to describe this phenotype. A compound heterozygous National Taiwan University, Taiwan mutation c.991G>T/c.1472+1G>A was detected in this patient. Heterozygous for the *Correspondence: c.991G>T and c.1472+1G>A were separately detected in his parents. Pathogenicity Hsin-Yi Lai [email protected] of these two mutations were predicted according to the American college of medical Baorong Zhang genetics and genomics (ACMG) guideline. -
Schizophrenia and Frontotemporal Dementia: Shared Causation?
International Review of Psychiatry, April 2013; 25(2): 168–177 Schizophrenia and frontotemporal dementia: Shared causation? MICHA Ł HARCIAREK 1 , DOLORES MALASPINA2 , TAO SUN3 & ELKHONON GOLDBERG4 1 Division of Clinical Psychology and Neuropsychology, Institute of Psychology, University of Gdansk, Poland, 2 Department of Psychiatry, New York University School of Medicine, New York, USA, 3 Department of Cell and Developmental Biology, Cornell University Weill Medical College, New York, USA, 4 Department of Neurology, New York University School of Medicine, New York, USA Abstract The relationship between specifi c genes and particular diseases in neuropsychiatry is unclear, and newer studies focus on shared domains of neurobiological and cognitive pathology across different disorders. This paper reviews the evidence for an association between schizophrenia and frontotemporal dementia, including symptom similarity, familial co-morbidity, and neuroanatomical changes. Genetic as well as epigenetic fi ndings from both schizophrenia and frontotemporal demen- tia are also discussed. As a result, we introduce the hypothesis of a shared susceptibility for certain subgroups of schizo- phrenia and frontotemporal dementia. This common causation may involve the same gene(s) at different stages of life: early in schizophrenia and late in frontotemporal dementia. Additionally, we provide a rationale for future research that should emphasize both genetic and cognitive parallels between certain forms of schizophrenia and frontotemporal dementia in a synergistic, coordinated way, placing both in the context of aberrant lateralization patterns. Introduction of what constitutes a ‘ disorder ’ in neuropsychiatry, The relationship between specifi c genes and particu- and invited the revision of, and departure from, tra- lar diseases in neuropsychiatry remains unclear, and ditional diagnostic taxonomies. -
Management Recommendations on Sleep Disturbance of Patients with Parkinson’S Disease
Consensus Management Recommendations on Sleep Disturbance of Patients with Parkinson’s Disease Chun‑Feng Liu1,2, Tao Wang3, Shu‑Qin Zhan4, De‑Qin Geng5, Jian Wang6, Jun Liu7, Hui‑Fang Shang8, Li‑Juan Wang9, Piu Chan4, Hai‑Bo Chen10, Sheng‑Di Chen7, Yu‑Ping Wang4, Zhong‑Xin Zhao11, K Ray Chaudhuri12 1Department of Neurology, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu 215004, China 2Institute of Neuroscience, Soochow University, Suzhou, Jiangsu 215004, China 3Department of Neurology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430022, China 4Department of Neurology, Xuan Wu Hospital, Capital Medical University, Beijing 100053, China 5Department of Neurology, Affiliated Hospital of Xuzhou Medical University, Xuzhou, Jiangsu 221006, China 6Department of Neurology and National Clinical Research Center for Aging and Medicine, Huashan Hospital, Fudan University, Shanghai 200040, China 7Department of Neurology and Institute of Neurology, Ruijin Hospital Affiliated to Shanghai JiaoTong University School of Medicine, Shanghai 200025, China 8Department of Neurology, West China Hospital, Sichuan University, Chengdu, Sichuan 610041, China 9Department of Neurology, Guangdong Neuroscience Institute, Guangdong General Hospital, Guangdong Academy of Medical Sciences, Guangzhou, Guangdong 510080, China 10Department of Neurology, Beijing Hospital, National Center of Gerontology, Beijing 100730, China 11Department of Neurology, Changzheng Hospital, Second Military Medical -
Differentiating Delirium, Dementia, and Depression Elderly Patients Are at High Risk of Mood and Cognitive Impairments Such As Depression, Delirium and Dementia
February 2021 www.nursingcenter.com Differentiating Delirium, Dementia, and Depression Elderly patients are at high risk of mood and cognitive impairments such as depression, delirium and dementia. Delirium is an acute, transient and reversible cause of brain dysfunction, usually triggered by one or more precipitating factors, including infection, medications, pain and dehydration. Dementia is usually subtle in its onset and may not be recognized until it has affected one or more cognitive domains. Depression is characterized by low mood, loss of interest or pleasure in most activities, sleep disturbance, anxiety, and social withdrawal. Delirium, dementia, and depression have overlapping characteristics, and patients may experience more than one of these conditions at the same time. It is essential to differentiate between these conditions, particularly if delirium is present, because this is an acute medical emergency that requires rapid assessment and management. Nurses in both outpatient and hospital settings can have a significant role in the identification, assessment and management of patients with dementia, delirium and depression. Dementia Signs and Symptoms Dementia is the most common disorder of cognition, and is characterized by a decline in one or more of these cognitive domains (Larson, 2019): • Memory (remote memories versus recent memories) • Language (word retrieval, comprehension) • Learning new skills (following linear instructions, with ability to repeat skills) • Executive function (ability to shop, do laundry, write a check) • Complex attention (completing multi-step tasks) • Social cognition (remembering family connections, names) • Perceptual-motor skills (dressing, bathing) The decline in function must not be attributable to other organic disease, must not be due to an episode of delirium, and must be severe enough to interfere with independence or daily functioning. -
Dyssomnia Sleep Disorders.Pdf
Dyssomnia Sleep Disorders Dyssomnia Sleep Disorders By Yolanda Smith, BPharm Dyssomnia is a broad type of sleep disorders involving difficulty falling or remaining asleep, which can lead to excessive sleepiness during the day due to the reduced quantity, quality or timing of sleep. This is distinct from parasomnias, which involves abnormal behavior of the nervous system during sleep. Symptoms indicative a dyssomnia sleep disorder may include difficulty falling asleep, intermittent awakenings during the night or waking up earlier than usual. As a result of the reduced or disrupted sleep, most patients do not feel well rested and have less ability to perform during the day. In many cases, lifestyle habits have an impact on the disorder, including stress, physical pain or discomfort, napping during the day, bedtime or use of stimulants. Dyssomnia Sleep Disorders There are two main types of dyssomnia sleep disorders according to the origin or cause or the disorder: extrinsic and intrinsic. Both of these are covered in more detail below, in addition to general principles in the diagnosis and treatment of the disorders. Extrinsic dyssomnias are sleep disorders that originate from external causes and may include: Insomnia Sleep apnea Narcolepsy Restless legs syndrome Periodic Limb movement disorder Hypersomnia Toxin-induced sleep disorder Kleine-Levin syndrome Intrinsic dyssomnias are sleep disorders that originate from internal causes and may include: Altitude insomnia Substance use insomnia Sleep-onset association disorder Nocturnal paroxysmal dystonia Limit-setting sleep disorder Diagnosis The differential diagnosis between the types of dyssomnias usually begins with a consultation about the sleep history of the individual, including the onset, frequency and duration of sleep. -
Qt8nt665v8.Pdf
UC Berkeley Berkeley Scientific Journal Title Acquired Savantism: The Genesis of Accidental Genius Permalink https://escholarship.org/uc/item/8nt665v8 Journal Berkeley Scientific Journal, 14(2) ISSN 1097-0967 Author Gururangan, Kapil Publication Date 2011 DOI 10.5070/BS3142011708 Undergraduate eScholarship.org Powered by the California Digital Library University of California ACQUIRED SAVANTISM: THE GENESIS OF ACCIDENTAL GENIUS Kapil Gururangan The autistic model for savantism stems from At age ten, Orlando Serrell was struck on the the high incidence of autism in the savant population. left side of his head with a baseball; he is able to clearly Nearly half of all savants display some condition that remember the weather and details about his personal can be classified as an autism spectrum disorder, leading activities for every day since that accident (Hughes 2010, scientists to presume that some facet of autism may 149). Some elderly patients battling the debilitation of also predispose the human mind to the formation of dementia have discovered newfound abilities in music, these amazing abilities (Treffert 2009, 2). This model is art, and language (Miller 2008, 8). A former molecular based on the principle of weak central coherence theory, biologist loses her ability to speak but becomes amazingly talented in painting that fuses music, vision, and feeling onto a canvas titled Unraveling Bolero (8). These examples all have one thing in common: the individuals acquire BSJ impressive abilities following damage to the central nervous system, predominantly in the left hemisphere of the brain. Savant syndrome is the presence of extreme talent in the wake of profound disabilities and while this normally presents in early development, scientists have observed a fascinating mechanism for the occurrence of savant syndrome after an accident (Hughes 2010, 149). -
Management Recommendations on Sleep Disturbance of Patients with Parkinson’S Disease
King’s Research Portal Link to publication record in King's Research Portal Citation for published version (APA): Liu, CF., Tao, W., ShuQin, Z., DeQin, G., Jian, W., Liu, J., HuiFang, S., LiJuan, W., Piu, C., HaiBo, C., Chen, SD., YuPing, W., ZhongXin, Z., & Ray Chaudhuri, K. (2018). Management Recommendations on Sleep Disturbance of Patients with Parkinson’s Disease. Chinese Medical Journal, 131(24), 2976-2985. Citing this paper Please note that where the full-text provided on King's Research Portal is the Author Accepted Manuscript or Post-Print version this may differ from the final Published version. If citing, it is advised that you check and use the publisher's definitive version for pagination, volume/issue, and date of publication details. And where the final published version is provided on the Research Portal, if citing you are again advised to check the publisher's website for any subsequent corrections. General rights Copyright and moral rights for the publications made accessible in the Research Portal are retained by the authors and/or other copyright owners and it is a condition of accessing publications that users recognize and abide by the legal requirements associated with these rights. •Users may download and print one copy of any publication from the Research Portal for the purpose of private study or research. •You may not further distribute the material or use it for any profit-making activity or commercial gain •You may freely distribute the URL identifying the publication in the Research Portal Take down policy If you believe that this document breaches copyright please contact [email protected] providing details, and we will remove access to the work immediately and investigate your claim.