Acute Care Interventions of Brain Injuries
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A Rare Case of Penetrating Trauma of Frontal Sinus with Anterior Table Fracture Himanshu Raval1*, Mona Bhatt2 and Nihar Gaur3
ISSN: 2643-4474 Raval et al. Neurosurg Cases Rev 2020, 3:046 DOI: 10.23937/2643-4474/1710046 Volume 3 | Issue 2 Neurosurgery - Cases and Reviews Open Access CASE REPORT Case Report: A Rare Case of Penetrating Trauma of Frontal Sinus with Anterior Table Fracture Himanshu Raval1*, Mona Bhatt2 and Nihar Gaur3 1 Department of Neurosurgery, NHL Municipal Medical College, SVP Hospital Campus, Gujarat, India Check for updates 2Medical Officer, CHC Dolasa, Gujarat, India 3GAIMS-GK General Hospital, Gujarat, India *Corresponding author: Dr. Himanshu Raval, Resident, Department of Neurosurgery, NHL Municipal Medical College, SVP Hospital Campus, Elisbridge, Ahmedabad, Gujarat, 380006, India, Tel: 942-955-3329 Abstract Introduction Background: Head injury is common component of any Road traffic accident (RTA) is the most common road traffic accident injury. Injury involving only frontal sinus cause of cranio-facial injury and involvement of frontal is uncommon and unique as its management algorithm is bone fractures are rare and constitute 5-9% of only fa- changing over time with development of radiological modal- ities as well as endoscopic intervention. Frontal sinus inju- cial trauma. The degree of association has been report- ries may range from isolated anterior table fractures causing ed to be 95% with fractures of the anterior table or wall a simple aesthetic deformity to complex fractures involving of the frontal sinuses, 60% with the orbital rims, and the frontal recess, orbits, skull base, and intracranial con- 60% with complex injuries of the naso-orbital-ethmoid tents. Only anterior table injury of frontal sinus is rare in pen- region, 33% with other orbital wall fractures and 27% etrating head injury without underlying brain injury with his- tory of unconsciousness and questionable convulsion which with Le Fort level fractures. -
Traumatic Brain Injury
REPORT TO CONGRESS Traumatic Brain Injury In the United States: Epidemiology and Rehabilitation Submitted by the Centers for Disease Control and Prevention National Center for Injury Prevention and Control Division of Unintentional Injury Prevention The Report to Congress on Traumatic Brain Injury in the United States: Epidemiology and Rehabilitation is a publication of the Centers for Disease Control and Prevention (CDC), in collaboration with the National Institutes of Health (NIH). Centers for Disease Control and Prevention National Center for Injury Prevention and Control Thomas R. Frieden, MD, MPH Director, Centers for Disease Control and Prevention Debra Houry, MD, MPH Director, National Center for Injury Prevention and Control Grant Baldwin, PhD, MPH Director, Division of Unintentional Injury Prevention The inclusion of individuals, programs, or organizations in this report does not constitute endorsement by the Federal government of the United States or the Department of Health and Human Services (DHHS). Suggested Citation: Centers for Disease Control and Prevention. (2015). Report to Congress on Traumatic Brain Injury in the United States: Epidemiology and Rehabilitation. National Center for Injury Prevention and Control; Division of Unintentional Injury Prevention. Atlanta, GA. Executive Summary . 1 Introduction. 2 Classification . 2 Public Health Impact . 2 TBI Health Effects . 3 Effectiveness of TBI Outcome Measures . 3 Contents Factors Influencing Outcomes . 4 Effectiveness of TBI Rehabilitation . 4 Cognitive Rehabilitation . 5 Physical Rehabilitation . 5 Recommendations . 6 Conclusion . 9 Background . 11 Introduction . 12 Purpose . 12 Method . 13 Section I: Epidemiology and Consequences of TBI in the United States . 15 Definition of TBI . 15 Characteristics of TBI . 16 Injury Severity Classification of TBI . 17 Health and Other Effects of TBI . -
Trauma Treating Traumatic Facial Nerve Paralysis
LETTERS TO EDITOR 205 206 LETTERS TO EDITOR In conclusion, the most frequent type of HBV R. Hepatitis B virus genotype A is more often hearing loss, and an impedance audiogram Strands of facial nerve interconnect with genotype in our study was D type, which associated with severe liver disease in northern showed absent stapedial reflex. CT scan cranial nerves V, VIII, IX, X, XI, and XII and usually causes a mild liver disease. Hence India than is genotype D. Indian J Gastroenterol showed no fracture of the temporal bone and with the cervical cutaneous nerves. This free proper vaccination, e ducational programs, 2005;24:19-22. intact ossicles. The patient was prescribed intermingling of Þ bers of the facial nerve with and treatment with lamivudine are efficient 6. Thakur V, Sarin SK, Rehman S, Guptan RC, high-dose steroids, along with vigorous facial Þ bers of other neural structures (particularly Kazim SN, Kumar S. Role of HBV genotype strategies in controlling HBV infection in our nerve stimulation and massages. the cranial nerve V) has been proposed as in predicting response to lamivudine therapy area. the mechanism of spontaneous return of facial in patients with chronic hepatitis B. Indian J After 3 weeks of treatment, the facial nerve nerve function after peripheral injury to the Gastroenterol 2005;24:12-5. stimulation tests were repeated and the ABDOLVAHAB MORADI, nerve.[4] VAHIDEH KAZEMINEJHAD1, readings were compared to those taken prior to GHOLAMREZA ROSHANDEL, treatment. There was negligible improvement. KHODABERDI KALAVI, Early onset/complete palsy indicates disruption EZZAT-OLLAH GHAEMI2, SHAHRYAR SEMNANI The patient was offered an exploratory of continuity of the nerve. -
5 Things to Know About Traumatic Brain Injuries
5 Things to Know About Traumatic Brain Injuries What is a Traumatic Brain Injury? A traumatic brain injury (TBI) is defined as a blow or jolt to the head or a penetrating head injury that disrupts the function of the brain. The severity of such an injury may range from: “mild” – i.e., a brief change in mental status or consciousness, to “severe” – i.e., an extended period of unconsciousness or amnesia after the injury. What Causes a Traumatic Brain Injury? A TBI occurs when an outside force impacts the head hard enough to cause the brain to move within the skull, or if the force causes the skull to break and directly hurts the brain. Rapid acceleration/deceleration of the head can also force the brain the move back and forth inside the skull, which pulls apart nerve fibers and causes damage to brain tissue. The most common causes of TBI are: Falls Motor vehicle-traffic crashes Physical violence Sports accidents What are the symptoms of a TBI? A person with a brain injury can experience a variety of symptoms, but not necessarily all of the following symptoms: Lethargy (sluggish, sleepy, gets tired easily) Continuous headache Confusion Ringing in the ears, or changes in ability to hear Vision changes (blurred vision, seeing double, light-sensitive) Dilated pupils Difficulty thinking (memory problems, poor judgment, poor attention span, slow thought process) Dizziness or balance problems Inappropriate emotional responses (irritability, easily frustrated, inappropriate crying or laughing) Difficulty speaking (slurred speech) Respiratory problems (slow or uneven breathing) Vomiting Body numbness or tingling Paralysis (difficulty moving body parts, weakness, poor coordination) Semi-comatose (not alert and unable to respond to others) Loss of consciousness Who is at Highest Risk for TBI? The two age groups at the highest first for TBI are 0-4 year olds and 15-19 year olds. -
Middle Ear Disorders
3/2/2014 ENT CARRLENE DONALD, MMS PA-C ANTHONY MENDEZ, MMS PA-C MAYO CLINIC ARIZONA DEPARTMENT OF OTOLARYNGOLOGY AND HEAD & NECK SURGERY No Disclosures OBJECTIVES • 1. Identify important anatomic structures of the ears, nose, and throat • 2. Assess and treat disorders of the external, middle and inner ear • 3. Assess and treat disorders of the nose and paranasal sinuses • 4. Assess and treat disorders of the oropharynx and larynx • 5. Educate patients on the risk factors for head and neck cancers 1 3/2/2014 Otology External Ear Disorders External Ear Anatomy 2 3/2/2014 Trauma • Variety of presentations. • Rule out temporal bone trauma (battle’s sign & hemotympanum). CT head w/out contrast. • Tx lacerations/avulsions with copious irrigation, closure with dissolvable sutures (monocryl), tetanus update, and antibiotic coverage (anti- Pseudomonal). May need to bolster if concerned for a hematoma . Auricular Hematoma • Due to blunt force trauma. • Drain/aspirate, cover with anti- biotics (anti- Pseudomonals), and apply bolster or passive drain if needed. • Infection and/or cauliflower ear may result if not treated. Chondritis • Inflammation and infection of the auricular cartilage. usually due to Pseudomonas aeruginosa. • Cultures • Treat with empiric antibiotics (anti-Pseudomonal) and I&D if needed. • Differentiate from relapsing polychondritis, which is an autoimmune disorder. 3 3/2/2014 External Auditory Canal Foreign Body • Children –Foreign bodies • Adults – Cerumen plugs • May present with hearing loss, ear pain and drainage • Exam under microscopic otoscopy. Check for otitis externa. • Remove under direct visualization. Can try to neutralize bugs with mineral oil. Do not attempt to irrigate organic material with water as this may cause an infection. -
Penetrating Injury to the Head: Case Reviews K Regunath, S Awang*, S B Siti, M R Premananda, W M Tan, R H Haron**
CASE REPORT Penetrating Injury to the Head: Case Reviews K Regunath, S Awang*, S B Siti, M R Premananda, W M Tan, R H Haron** *Department of Neurosciences, Universiti Sains Malaysia, 16150 Kubang Kerian, Kelantan, **Department of Neurosurgery, Hospital Kuala Lumpur the right frontal lobe to a depth of approximately 2.5cm. SUMMARY (Figure 1: A & B) There was no obvious intracranial Penetrating injury to the head is considered a form of severe haemorrhage along the track of injury. The patient was taken traumatic brain injury. Although uncommon, most to the operating theatre and was put under general neurosurgical centres would have experienced treating anaesthesia. The nail was cut proximal to the entry wound patients with such an injury. Despite the presence of well and the piece of wood removed. The entry wound was found written guidelines for managing these cases, surgical to be contaminated with hair and debris. The nail was also treatment requires an individualized approach tailored to rusty. A bicoronal skin incision was fashioned centred on the the situation at hand. We describe a collection of three cases entry wound. A bifrontal craniotomy was fashioned and the of non-missile penetrating head injury which were managed bone flap removed sparing a small island of bone around the in two main Neurosurgical centres within Malaysia and the nail (Figure 1: C&D). Bilateral “U” shaped dural incisions unique management approaches for each of these cases. were made with the base to the midline. The nail was found to have penetrated with dura about 0.5cm from the edge of KEY WORDS: Penetrating head injury, nail related injury, atypical penetrating the sagittal sinus. -
Traumatic Brain Injury(Tbi)
TRAUMATIC BRAIN INJURY(TBI) B.K NANDA, LECTURER(PHYSIOTHERAPY) S. K. HALDAR, SR. OCCUPATIONAL THERAPIST CUM JR. LECTURER What is Traumatic Brain injury? Traumatic brain injury is defined as damage to the brain resulting from external mechanical force, such as rapid acceleration or deceleration impact, blast waves, or penetration by a projectile, leading to temporary or permanent impairment of brain function. Traumatic brain injury (TBI) has a dramatic impact on the health of the nation: it accounts for 15–20% of deaths in people aged 5–35 yr old, and is responsible for 1% of all adult deaths. TBI is a major cause of death and disability worldwide, especially in children and young adults. Males sustain traumatic brain injuries more frequently than do females. Approximately 1.4 million people in the UK suffer a head injury every year, resulting in nearly 150 000 hospital admissions per year. Of these, approximately 3500 patients require admission to ICU. The overall mortality in severe TBI, defined as a post-resuscitation Glasgow Coma Score (GCS) ≤8, is 23%. In addition to the high mortality, approximately 60% of survivors have significant ongoing deficits including cognitive competency, major activity, and leisure and recreation. This has a severe financial, emotional, and social impact on survivors left with lifelong disability and on their families. It is well established that the major determinant of outcome from TBI is the severity of the primary injury, which is irreversible. However, secondary injury, primarily cerebral ischaemia, occurring in the post-injury phase, may be due to intracranial hypertension, systemic hypotension, hypoxia, hyperpyrexia, hypocapnia and hypoglycaemia, all of which have been shown to independently worsen survival after TBI. -
Final Program N
XXII Congress The International Society on Thrombosis and Haemostasis B July 11-16 2009 O 55th Annual Meeting S of the Scientific and Standardization Committee of the ISTH T O Final Program N Boston - July 11-16 2009 XXII Congress of the International Society on Thrombosis and Haemostasis 2009 Table ISTH of Contents Venue and Contacts 2 Wednesday 209 Welcome Messages 3 – Plenary Lectures 210 Committees 7 – State of the Art Lectures 210 Congress Awards and Grants 15 – Abstract Symposia Lectures 212 Other Meetings 19 – Oral Communications 219 – Posters 239 ISTH Information 20 Program Overview 21 Thursday 305 SSC Meetings and – Plenary Lectures 306 Educational Sessions 43 – State of the Art Lectures 306 – Abstract Symposia Lectures 309 Scientific Program 89 – Oral Communications 316 Monday 90 – Posters 331 – Plenary Lectures 90 Nursing Program 383 – State of the Art Lectures 90 Special Symposia 389 – Abstract Symposia Lectures 92 Satellite Symposia 401 – Oral Communications 100 – Posters 118 Technical Symposia Sessions 411 Exhibition and Sponsors 415 Tuesday 185 – Plenary Lectures 186 Exhibitor and Sponsor Profiles 423 – State of the Art Lectures 186 Congress Information 445 – Abstract Symposia Lectures 188 Map of BCEC 446 – Oral Communications 196 Hotel and Transportation Information 447 ISTH 2009 Congress Information 452 Boston Information 458 Social Events 463 Excursions 465 Authors’ Index 477 1 Venue & Contacts Venue Boston Convention & Exhibition Center 415 Summer Street - Boston, Massachusetts 02210 - USA Phone: +1 617 954 2800 - Fax: +1 617 954 3326 The BCEC is only about 10 minutes by taxi from Boston Logan International Airport. The 2009 Exhibition is located in Hall A and B of the Exhibit Level of the BCEC, along with posters and catering. -
Approach to the Trauma Patient Will Help Reduce Errors
The Approach To Trauma Author Credentials Written by: Nicholas E. Kman, MD, The Ohio State University Updated by: Creagh Boulger, MD, and Benjamin M. Ostro, MD, The Ohio State University Last Update: March 2019 Case Study “We have a motor vehicle accident 5 minutes out per EMS report.” 47-year-old male unrestrained driver ejected 15 feet from car arrives via EMS. Vital Signs: BP: 100/40, RR: 28, HR: 110. He was initially combative at the scene but now difficult to arouse. He does not open his eyes, withdrawals only to pain, and makes gurgling sounds. EMS placed a c-collar and backboard, but could not start an IV. What do you do? Objectives Upon completion of this self-study module, you should be able to: ● Describe a focused rapid assessment of the trauma patient using an organized primary and secondary survey. ● Discuss the components of the primary survey. ● Discuss possible pathology that can occur in each domain of the primary survey and recommend treatment/stabilization measures. ● Describe how to stabilize a trauma patient and prioritize resuscitative measures. ● Discuss the secondary survey with particular attention to head/central nervous system (CNS), cervical spine, chest, abdominal, and musculoskeletal trauma. ● Discuss appropriate labs and diagnostic testing in caring for a trauma patient. ● Describe appropriate disposition of a trauma patient. Introduction Nearly 10% of all deaths in the world are caused by injury. Trauma is the number one cause of death in persons 1-50 years of age and results in significant life years lost. According to the National Trauma Data Bank, falls were the leading cause of trauma followed by motor vehicle collisions (MVCs) and firearm related injuries with an overall mortality rate of 4.39% in 2016. -
Joint Faculty of Intensive Care Medicine
Joint Faculty of Intensive Care Medicine Australian and New Zealand The Royal Australasian College of Anaesthetists College of Physicians ABN 82 055 042 852 Exam Report Oct 2009 This report is prepared to provide candidates, tutors and their supervisors of training with information about the way in which the Examiners assessed the performance of candidates in the Examination. Answers provided are not model answers but guides to what was expected. Candidates should discuss the report with their tutors so that they may prepare appropriately for the future examinations The exam included two 2.5 hour written papers comprising of 15 ten-minute short answer questions each. Candidates were required to score at least 50% in the written paper before being eligible to sit the oral part of the exam. The oral exam comprised 8 interactive vivas and two separate hot cases. This is the fourth examination with the new regulations which came into force in 2008. The tables below provide an overall statistical analysis as well as information regarding performance in the individual sections. A comparison with April 2008, October 2008 and April 2009 data is also provided. Examiner comments Written paper 1) Lack of specificity and precision in the answers. 2) Poor pass rate on clinical methods questions – suggests that candidates are not taking clinical examination seriously and they will need to read books like Talley and O’Connor thoroughly. 3) Candidates seem to score well largely on Data interpretation / OSCE type questions. The inability to score well on other questions reflects a general lack of preparation and knowledge even of common topics in intensive care. -
Guidelines for the Management of Severe Traumatic Brain Injury 4Th Edition
Guidelines for the Management of Severe Traumatic Brain Injury 4th Edition Nancy Carney, PhD Oregon Health & Science University, Portland, OR Annette M. Totten, PhD Oregon Health & Science University, Portland, OR Cindy O'Reilly, BS Oregon Health & Science University, Portland, OR Jamie S. Ullman, MD Hofstra North Shore-LIJ School of Medicine, Hempstead, NY Gregory W. J. Hawryluk, MD, PhD University of Utah, Salt Lake City, UT Michael J. Bell, MD University of Pittsburgh, Pittsburgh, PA Susan L. Bratton, MD University of Utah, Salt Lake City, UT Randall Chesnut, MD University of Washington, Seattle, WA Odette A. Harris, MD, MPH Stanford University, Stanford, CA Niranjan Kissoon, MD University of British Columbia, Vancouver, BC Andres M. Rubiano, MD El Bosque University, Bogota, Colombia; MEDITECH Foundation, Neiva, Colombia Lori Shutter, MD University of Pittsburgh, Pittsburgh, PA Robert C. Tasker, MBBS, MD Harvard Medical School & Boston Children’s Hospital, Boston, MA Monica S. Vavilala, MD University of Washington, Seattle, WA Jack Wilberger, MD Drexel University, Pittsburgh, PA David W. Wright, MD Emory University, Atlanta, GA Jamshid Ghajar, MD, PhD Stanford University, Stanford, CA Reviewed for evidence-based integrity and endorsed by the American Association of Neurological Surgeons and the Congress of Neurological Surgeons. September 2016 TABLE OF CONTENTS PREFACE ...................................................................................................................................... 5 ACKNOWLEDGEMENTS ............................................................................................................................................. -
Head Injuries/Concussions
HEAD INJURY/CONCUSSIONS Overview p. 1 Definitions p. 2 Incidence/Causes p. 2-4 Signs/Symptoms (Early) p. 4 First Aid p. 4-5 Health Office Steps p. 5-6 Follow-up/Educational Implications p. 6-7 CA Sports Rules (Ed. Code; CIF) p. 8 Second Impact Syndrome p. 9 Prevention/Guidelines for p. 9-10 the Management of Sport-Related Concussion Resources/References p. 11-12 OVERVIEW Although most head injuries and concussions are mild, they are potentially serious and may result in death or have long term serious negative consequences. These include: Changes in the ability to learn, communicate and think Increased mental health problems- depression, anxiety, personality changes, aggression, acting out, and social inappropriateness Traumatic brain injury can also cause epilepsy and increase the risk for conditions such as Alzheimer’s disease, Parkinson’s disease, and other brain disorders that become more prevalent with age Relative to sports, data shows that many catastrophic head injuries are a direct result of injured athletes returning to play too soon, not having fully recovered from the first head injury. (From California Interscholastic Federation (CIF) - “All concussions are potentially serious and may 1 San Diego County Office of Education- School Nursing (2013) result in complications including prolonged brain damage and death if not recognized and managed properly. In other words, even a “ding” or a bump on the head can be serious. …. most sports concussions occur without loss of consciousness.” Definitions Head injury Medline Plus - http://www.nlm.nih.gov/medlineplus/ency/article/000028.htm ) A head injury is any trauma that injures the scalp, skull, or brain.