X-Ray Interpretation
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Chest and Abdominal Radiograph 101
Chest and Abdominal Radiograph 101 Ketsia Pierre MD, MSCI July 16, 2010 Objectives • Chest radiograph – Approach to interpreting chest films – Lines/tubes – Pneumothorax/pneumomediastinum/pneumopericar dium – Pleural effusion – Pulmonary edema • Abdominal radiograph – Tubes – Bowel gas pattern • Ileus • Bowel obstruction – Pneumoperitoneum First things first • Turn off stray lights, optimize room lighting • Patient Data – Correct patient – Patient history – Look at old films • Routine Technique: AP/PA, exposure, rotation, supine or erect Approach to Reading a Chest Film • Identify tubes and lines • Airway: trachea midline or deviated, caliber change, bronchial cut off • Cardiac silhouette: Normal/enlarged • Mediastinum • Lungs: volumes, abnormal opacity or lucency • Pulmonary vessels • Hila: masses, lymphadenopathy • Pleura: effusion, thickening, calcification • Bones/soft tissues (four corners) Anatomy of a PA Chest Film TUBES Endotracheal Tubes Ideal location for ETT Is 5 +/‐ 2 cm from carina ‐Normal ETT excursion with flexion and extension of neck 2 cm. ETT at carina Right mainstem Intubation ‐Right mainstem intubation with left basilar atelectasis. ETT too high Other tubes to consider DHT down right mainstem DHT down left mainstem NGT with tip at GE junction CENTRAL LINES Central Venous Line Ideal location for tip of central venous line is within superior vena cava. ‐ Risk of thrombosis decreased in central veins. ‐ Catheter position within atrium increases risk of perforation Acceptable central line positions • Zone A –distal SVC/superior atriocaval junction. • Zone B – proximal SVC • Zone C –left brachiocephalic vein. Right subclavian central venous catheter directed cephalad into IJ Where is this tip? Hemiazygous Or this one? Right vertebral artery Pulmonary Arterial Catheter Ideal location for tip of PA catheter within mediastinal shadow. -
Cardiothoracic Fellowship Program
Cardiothoracic Fellowship Program Table of Contents Program Contact ............................................................................................ 3 Other contact numbers .................................................................................. 4 Introduction ........................................................................................................... 5 Goals and Objectives of Fellowship: ..................................................................... 6 Rotation Schedule: ........................................................................................ 7 Core Curriculum .................................................................................................... 8 Fellow’s Responsibilities ..................................................................................... 22 Resources ........................................................................................................... 23 Facilities ....................................................................................................... 23 Educational Program .......................................................................................... 26 Duty Hours .......................................................................................................... 29 Evaluation ........................................................................................................... 30 Table of Appendices .................................................................................... 31 Appendix A -
TB: Recognizing It on a Chest X-Ray
TB: Recognizing it on a Chest X‐Ray Disclosures • Grant support from Michigan Department of Community Health – Despite conflict of interest I still want to: – There’s enough TB for job security. Objectives • You will – Be able to identify major structures on a normal chest x‐ray – Identify and correctly name CXR abnormalities seen commonly in TB – Recognize chest x‐ray patterns that suggest TB & when you find them you will Basics of Diagnostic X‐ray Physics • X‐rays are directed at the . patient and variably absorbed – When not absorbed • Pass through patient & strike the x‐ray film or – When completely absorbed • Don’t strike x‐ray film or – When scattered • Some strike the x‐ray film Absorption Shade / Density • Absorption depends • Whitest = Most Dense on the – Metal – Energy of the x‐ray beam – Contrast material (dye) – Density of the tissue – Calcium – Bone – Water – Soft Tissue – Fat – Air / Gas • Blackest = Least Dense Normal Frontal Chest X‐ray: Posterior Anterior Note silhouette formed by • lung adjacent to heart • lung adjacent to diaphragm Silhouette Sign Lifeinthefastlane.com Normal Lateral Chest X‐ray Normal PA & Lateral X‐ray: Hilum Hilum –Major bronchi, Pulmonary veins & arteries, Lymph nodes at the root of the lung. Normal PA & Lateral X‐ray: Mediastinum Mediastinum –Central chest organs (not lungs) – Heart, Aorta, Trachea, Thymus, Esophagus, Lymph nodes, Nerves (Between 2 pleuras or linings of the lungs) Normal PA & Lateral X‐ray: Apex • Apex of lung – Area of lung above the level of the anterior end of the 1st rib Wink -
Chest X-Ray Made Ridiculously Plain, Basic, Or Uncomplicated in Form, Nature, Or Design; Without Much Decoration Or Ornamentation
Chest X-Ray Made ridiculously plain, basic, or uncomplicated in form, nature, or design; without much decoration or ornamentation Steve Cohen, MD, PA-C Associate Professor, Dept. of Surgery & Director of PA Clinical Education Florida International University Really Simple Step 3 The END, Bye-Bye Thanks for Coming! Posterior Anterior (PA) Workhorse PA Films Properties • X-ray is a “NEGATIVE” – Light is dark (ex. Air) – Less x-ray get through = shows white (eg. bone) • Beam is divergent – Posterior elements magnified – Dense posterior elements may hide anterior structures What can you see on CXR? 4 3 2 5 5 1 3 6 What are we looking at? Quickie Anatomy What are we looking at? Quickie Anatomy Airway • Film “Position” – Patients left on your right • Trachea – Midline (mostly) – Open • Mostly equal from wall to wall Ribs • Rib count – Extent quality • Bony integrity – Fracture, dislocation • Left first rib – Posterior at top • Beam hits first – Anterior at bottom Ribs • Use 1st rib to count • Posterior ribs usually most visible – beam hits first • Seen here = 8 ribs • Clavicles – Integrity (fx, displacement) – Assessing midline • Same distance from trachea Diaphragm – Stomach Bubble • Right higher than left diaphragm – Liver • Costophrenic angles – Fluid “haziness” • Air under diaphragm • Stomach bubble – variable Left Vessels and Heart • Subclavian Artery • Coming off arch • Aortic Arch • Pulmonary Artery • Returning to heart • Left Atrium • Left Ventricle Right Vessels and Heart • Ascending Aorta • Leaving heart • Superior Vena Cava • Entering -
Upper Airway Obstruction in Children: Imaging Essentials
Acute upper airway obstruction in children: Imaging essentials Carlos J. Sivit MD Rainbow Babies and Children’s Hospital Case Western Reserve School of Medicine ►Clinical perspective ►Infections ►Foreign body ►Masses 1 Clinical Clinical ► Common cause of respiratory failure in children ► Potentially life-threatening in younger children because of smaller airway diameter ► Narrowing of upper airway has exponential effect on airflow 2 Clinical ► Majority of children are otherwise healthy ► Appropriate management results in good outcomes ► Improper management has dire consequences ► Imaging plays critical role in diagnosis Clinical ► Signs and symptoms § Respiratory distress § Dysphagia § Odynophagia § Stridor § Absence of air entry § Tachycardia 3 Stridor ► Harsh respiratory noise caused by turbulent air flow through narrowed airway ► Specific for severe upper airway obstruction ► Intensifies in inspiration ► Does not help specify nature or location Infections 4 Infections ► Acute laryngotracheobronchitis ► Acute epiglottitis ► Acute bacterial tracheitis ► Retropharyngeal abscess ► Infectious mononucleosis Croup ► Heterogenous group of acute infections characterized by brassy “croupy” cough ► May or may not be accompanied by stridor, hoarseness and respiratory distress ► Typically seen in younger children § 6 months – 5 years 5 Croup ► Parainfluenza viruses account for 75% ► Adenoviruses, RSV, influenza and measles cause most remaining cases ► Secondary bacterial infection is rare Imaging ►Imaging § Performed to exclude other conditions -
12. Shortness of Breath
12. Shortness of breath Shortness of breath (dyspnea) is described as an intense tightening in the chest, air hunger, difficulty breathing, breathlessness or a feeling of suffocation. It is a subjective symptom of many diseases. The causes of shortness of breath can be various, but most often pulmonary or cardiac. The main causes of pulmonary or cardiac dyspnea include heart congestion, pulmonary embolism, emphysema, atelectasis, pneumothorax and interstitial lung processes. The basic imaging method in the diagnosis of dyspnea that follows clinical examination is a chest X- ray. It is used to evaluate heart congestion, fluid effusions, atelectasis, pneumothorax, and a rough assessment of interstitial lung processes. Only if there is a clinical suspicion of pulmonary embolization, the CT angiography (with intravenous contrast), perfusion lung scintigraphy or echocardiography are imaging methods of choice. If interstitial lung disease or pulmonary emphysema are suspected or there is uncertain finding on chest X-ray, high-resolution CT (HRCT) of the chest is recommended. HRCT allows detailed assessment of pulmonary parenchyma. Non-contrast CT is usually sufficient to assess interstitial changes. Heart congestion Heart congestion is the result of insufficient cardiac output due to heart failure or fluid overload. The most common cause of heart congestion is left heart failure with increased pressure in pulmonary veins and capillaries. On chest X- ray can be distinguished 3 degrees of heart congestion. Note: Right-sided heart failure most often occurs in long-term left-sided heart failure or lung disease, which leads to high pulmonary artery resistance (various etiologies of pulmonary hypertension, e.g. advanced COPD). -
Interstitial Lung Disease
Interstitial Lung Disease Camille Washowich, MSN, ACNP, CCRN Center for Advanced Lung Disease Stanford University Medical Center Lung Physiology ILD Classification Interstitial Lung Disease Connective Tissue Diseases Primary (unclassified) Idiopathic Fibrotic Disorders Drug and Treatment Induced Connective Tissue Diseases Scleroderma Systemic Lupus Erythematous (SLE) Rheumatoid Arthritis Mixed Connective Tissue Disease Primary (unclassified) Sarcoidosis Stage I-IV Neurofibromatosis Tuberous Sclerosis AIDS ARDS Bone Marrow Transplantation Post infectious Occupational & Environmental Exposures: Inorganic & Organic Agriculture Workers and Animal Handlers Construction: wood/metal Auto repair Military Chemicals (plastic, paint, polyurethane) Organisms: fungus/molds/bacterium Idiopathic Fibrotic Disorders Pulmonary fibrosis Familial pulmonary fibrosis Autoimmune pulmonary fibrosis Respiratory bronchiolitis Nonspecific interstitial pneumonitis (NSIP) Drug Induced Antibiotics Anti-arrhythmics Anti-inflammatory Anti-convulsant Radiation/Chemotherapy Oxygen toxicity Narcotics ILD Epidemiology in the US 100K admissions/year Occupation DILD Sarcoidosis 11% 15% pulmonologist patients 5% DAH 8% 4% CTD Incidence: 5/100K 9% Men (31%) versus Women (26%) Other 11% IPF 45% of all ILD patients Pulmonary Fibrosis 52% Age/Gender/Race Specifications to Assist in Diagnosis 20-40yrs: Inherited Interstitial Lung diseases Familial idiopathic pulmonary fibrosis Collagen vascular disease- associated ILD LAM Pulmonary Langerhans’ cell granulomatosis Sarcoidosis 50yrs: -
Since January 2020 Elsevier Has Created a COVID-19 Resource Centre with Free Information in English and Mandarin on the Novel Coronavirus COVID- 19
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by IUPUIScholarWorks Since January 2020 Elsevier has created a COVID-19 resource centre with free information in English and Mandarin on the novel coronavirus COVID- 19. The COVID-19 resource centre is hosted on Elsevier Connect, the company's public news and information website. Elsevier hereby grants permission to make all its COVID-19-related research that is available on the COVID-19 resource centre - including this research content - immediately available in PubMed Central and other publicly funded repositories, such as the WHO COVID database with rights for unrestricted research re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for free by Elsevier for as long as the COVID-19 resource centre remains active. A02842_052 4/11/06 3:59 PM Page 813 Chapter 52 Otolaryngologic Disorders William P. Potsic and Ralph F. Wetmore EAR vibrating tympanic membrane to the stapes footplate. Anatomy Stapes movement creates a fluid wave in the inner ear that travels to the round window membrane and is dissi- The ear is divided into three anatomic and functional pated by reciprocal motion to the stapes. areas: the external ear, the middle ear, and the inner ear. There are two striated muscles in the middle ear. The The external ear consists of the auricle, external auditory tensor tympani muscle lies along the side of the eustachian canal, and the lateral surface of the tympanic membrane. tube, and its tendon attaches to the medial surface of the The auricle is a complex fibroelastic skeleton that is cov- malleus. -
CHEST RADIOLOGY: Goals and Objectives
Harlem Hospital Center Department of Radiology Residency Training Program CHEST RADIOLOGY: Goals and Objectives ROTATION 1 (Radiology Years 1): Resident responsibilities: • ED chest CTs • Inpatient and outpatient plain films including the portable intensive care unit radiographs • Consultations with referring clinicians MEDICAL KNOWLEDGE: • Residents must demonstrate knowledge about established and evolving biomedical, clinical, and cognitive sciences and the application of this knowledge to patient care. At the end of the rotation, the resident should be able to: • Identify normal radiographic and CT anatomy of the chest • Identify and describe common variants of normal, including aging changes. • Demonstrate a basic knowledge of radiographic interpretation of atelectasis, pulmonary infection, congestive heart failure, pleural effusion and common neoplastic diseases of the chest • Identify the common radiologic manifestation of thoracic trauma, including widened mediastinum, signs of aortic laceration, pulmonary contusion/laceration, esophageal and diaphragmatic rupture. • Know the expected postoperative appearance in patients s/p thoracic surgery and the expected location of the life support and monitoring devices on chest radiographs of critically ill patients (intensive care radiology); be able to recognize malpositioned devices. • Identify cardiac enlargement and know the radiographic appearance of the dilated right vs. left atria and right vs. left ventricles, and pulmonary vascular congestion • Recognize common life-threatening -
Chest Radiology: a Resident's Manual
Chest Radiology: A Resident's Manual Bearbeitet von Johannes Kirchner 1. Auflage 2011. Buch. 300 S. Hardcover ISBN 978 3 13 153871 0 Format (B x L): 23 x 31 cm Weitere Fachgebiete > Medizin > Sonstige Medizinische Fachgebiete > Radiologie, Bildgebende Verfahren Zu Inhaltsverzeichnis schnell und portofrei erhältlich bei Die Online-Fachbuchhandlung beck-shop.de ist spezialisiert auf Fachbücher, insbesondere Recht, Steuern und Wirtschaft. Im Sortiment finden Sie alle Medien (Bücher, Zeitschriften, CDs, eBooks, etc.) aller Verlage. Ergänzt wird das Programm durch Services wie Neuerscheinungsdienst oder Zusammenstellungen von Büchern zu Sonderpreisen. Der Shop führt mehr als 8 Millionen Produkte. 1 Heart Failure Acute left heart failure is most commonly caused by a hyperten- " Compare pulmonary vessels that are equidistant to a central sive crisis. Radiographic signs on the plain chest radiograph ob- point in the respective hilum. tained with the patient standing include: " Compare the diameter of a random easily identifiable superior " Redistribution of pulmonary perfusion lobe artery (often the anterior segmental artery is most easily " Presence of interstitial patterns (Kerley lines, peribronchial identifiable) with the diameter of the corresponding ipsilateral cuffing) bronchus (Fig. 1.62). " Alveolar densities with indistinct vascular structures (ad- vanced stage) As the pulmonary artery and corresponding ipsilateral bronchus " Pleural effusions are normally of precisely equal diameter, a larger arterial diameter is indicative of redistribution of perfusion (Fig. 1.63). The diagnos- All of these signs are essentially attributable to increased fluid tic criteria of caudal-to-cranial redistribution cannot be evaluated content in the abnormally heavy “wet” lung. The fluid accumula- on radiographs obtained in the supine patient. -
Pleural Effusion, Hypovascularity in Lung Zone (Westermark’S Sign) & Pyramid Shape Infiltrate with Peak Directed to Hilus (Hampton’S Hump)
Author(s): Michele M. Nypaver, MD, 2011 License: Unless otherwise noted, this material is made available under the terms of the Creative Commons Attribution Share Alike 3.0 License: http://creativecommons.org/licenses/by-sa/3.0/ We have reviewed this material in accordance with U.S. Copyright Law and have tried to maximize your ability to use, share, and adapt it. Copyright holders of content included in this material should contact [email protected] with any questions, corrections, or clarification regarding the use of content. For more information about how to cite these materials visit http://open.umich.edu/privacy-and-terms-use. Any medical information in this material is intended to inform and educate and is not a tool for self-diagnosis or a replacement for medical evaluation, advice, diagnosis or treatment by a healthcare professional. Please speak to your physician if you have questions about your medical condition. Viewer discretion is advised: Some medical content is graphic and may not be suitable for all viewers. Citation Key for more information see: http://open.umich.edu/wiki/CitationPolicy Use + Share + Adapt { Content the copyright holder, author, or law permits you to use, share and adapt. } Public Domain – Government: Works that are produced by the U.S. Government. (17 USC § 105) Public Domain – Expired: Works that are no longer protected due to an expired copyright term. Public Domain – Self Dedicated: Works that a copyright holder has dedicated to the public domain. Creative Commons – Zero Waiver Creative Commons – Attribution License Creative Commons – Attribution Share Alike License Creative Commons – Attribution Noncommercial License Creative Commons – Attribution Noncommercial Share Alike License GNU – Free Documentation License Make Your Own Assessment { Content Open.Michigan believes can be used, shared, and adapted because it is ineligible for copyright. -
Redalyc.POSTERS EXPOSTOS
Revista Portuguesa de Pneumología ISSN: 0873-2159 [email protected] Sociedade Portuguesa de Pneumologia Portugal POSTERS EXPOSTOS Revista Portuguesa de Pneumología, vol. 23, núm. 3, noviembre, 2017 Sociedade Portuguesa de Pneumologia Lisboa, Portugal Disponível em: http://www.redalyc.org/articulo.oa?id=169753668003 Como citar este artigo Número completo Sistema de Informação Científica Mais artigos Rede de Revistas Científicas da América Latina, Caribe , Espanha e Portugal Home da revista no Redalyc Projeto acadêmico sem fins lucrativos desenvolvido no âmbito da iniciativa Acesso Aberto Document downloaded from http://www.elsevier.es, day 06/12/2017. This copy is for personal use. Any transmission of this document by any media or format is strictly prohibited. POSTERS EXPOSTOS PE 001 PE 002 A PLEASANT FINDING MALIGNANT CHEST PAIN A Pais, AI Coutinho, M Cardoso, A Pignatelli, C Bárbara A Pais, C Pereira, C Antunes, V Pereira, AI Coutinho, A Feliciano, Centro Hospitalar de Lisboa Norte C Quadros, A Ribeiro, L Carvalho, C Bárbara Centro Hospitalar de Lisboa Norte Key-words: mass, debridement, hamartoma Key-words: pain, S100, sarcoma 37-year-old male patient, salesman. Sporadic smoker. With a past history of allergic rhinitis and chronic gastritis. With no usual 26-year-old male patient, supermarket employee. Smoker of 10 medication. In January 2017, he was diagnosed with a respiratory pack-year. Past history of bronchial asthma in childhood. No rel - infection, having completed ten days of empirical antibiotic ther - evant family history. Without usual ambulatory medication. With apy with amoxicillin / clavulanic acid, with clinical improvement. a history of dry cough and chest pain in the posterior region of In May 2017, he underwent thoracic xray, which revealed homoge - the left hemithorax, for about two years, having had at that time, neous opacity of triangular morphology in the middle lobe of the chest X-ray without pathological findings, and the clinical picture right lung.