Capnography- Measuring Life and Breath

Total Page:16

File Type:pdf, Size:1020Kb

Capnography- Measuring Life and Breath DECEMBER 2010 Measuring LIFE & BREATH The benefits of capnography in EMS An exclusive supplement to JEMS sponsored by Oridion. A LOOK INSIDE TABLE OF CONTENTS 4 CAPNOGRAPHY BASICS An invaluable tool for providers & their patients Pat Brandt, RN 10 POSITIVE JUSTIFICATION A cost-benefit analysis of capnography use can prove value to patients & your budget Pat Brandt, RN 14 A FORM OF TRIAGE Vice President/Publisher Advertising Director Capnography use for the conscious & Jeff Berend Judi Leidiger non-intubated patient Bob Page, AAS, NREMT-P, CCEMT-P, NCEE Editorial Director Art Director 18 GOTHCA! A.J. Heightman, MPA, EMT-P Leah Bergman Challenging waveforms that could fool an expert Pat Brandt, RN Managing Editor Cover Photo Lauren Hardcastle Oridion INTRODUCTION THE ULTIMATE TREND SETTER By A.J. Heightman, MPA, EMT-P he 2010 AHA Guidelines for CPR and ECC have the earliest indication of hypoventilation, respiratory depression T a major new Class I recommendation for and respiratory failure. use of quantitative waveform capnography for Most importantly, changes in the capnography waveform confirmation and monitoring of endotracheal provide the earliest indication of apnea, upper airway obstruction (ET) tube placement. Real-time monitoring and optimization and laryngospasm. A glance at the waveform by a trained of CPR quality using physiologic parameters, such as partial provider allows them to instantly see a patient’s response to pressure of end-tidal carbon dioxide (PEtCO2), are encouraged. airway alignment maneuvers and further distinguish upper The guidelines also recommend the use of capnography PEtCO2 airway obstruction from laryngospasm. Providers can also easily values to monitor CPR quality, detect return of spontaneous recognize the curved waveform of obstructive lung disease, which circulation and guide vasopressor therapy during cardiac arrest indicates bronchospasm. (Class IIb). Capnography can also be used effectively to detect, assess In EMS circles, capnometry used to just be a tool for and triage victims of chemical terrorism. It’s helpful as an determining whether an ET tube was properly placed. It soon assessment and triage tool because chemical agents are primarily became the gold standard for this function. However, medical absorbed through the skin and respiratory tract and have their directors, educators and field crews in most EMS systems soon greatest effect on the central nervous and respiratory systems. realized that capnography was a multi-purpose assessment tool. Because EtCO2 waveforms and trends alert crews to Capnography is the only single-monitoring modality worsening conditions and allow them to intervene much earlier available to EMS crews that provides a visual reference to a to correct or reverse a critical condition, capnography is one of patient’s ABCs in less than 15 seconds, with a normal waveform the most important EMS assessment tools that can be carried in instantly telling us our patient’s airway is patent, they’re a critical care toolkit for conscious and unconscious patients. breathing, and they’re adequately perfusing. Pass this special supplement to December JEMS along to Capnography also provides the only direct, non-invasive your personnel and ensure your crews are fully utilizing all the measure of ventilatory status available to EMS crews and offers im­­portant capabilities offered by capnography. Measuring Life & Breath is an editorial supplement sponsored by Oridion and published by Elsevier Public Safety, 525 B Street, Ste. 1800, San Diego, CA 92101-4495; 800/266-5367 (Fed ID # 13-1958712). Copyright 2010 Elsevier Inc. No material may be reproduced or uploaded on computer network services without the expressed permission of the publisher. Subscription information: To subscribe to an Elsevier publication, visit www.jems.com. Advertising information: Rates are available on request. Contact Elsevier Public Safety, Advertising Department, 525 B Street, Ste. 1800, San Diego, CA 92101-4495; 800/266-536. MEASURING LIFE & BREATH 3 THE BASICS CAPNOGRAPHY BASICS An invaluable tool for providers & their patients Photo Fred Wurster & Al Kalbach Wurster Photo Fred By Pat Brandt, RN Capnography is now required in most EMS systems to ensure successful patient endotracheal intubation. he goal of this supplement is to review key are perfusing well. In addition, it gives you key infor- T aspects of capnography, its powerful assess- mation about the patient’s metabolic status, such as ment capabilities on intubated and conscious whether they have normal metabolic activity or if it patients, and its importance as a prehospital triage increased or decreased. and treatment guiding tool. This information, when combined with the Capnography provides valuable and rapid patient’s history and your physical assessment, can assessment information that greatly assists EMS provide you with an accurate working diagnosis of providers and enables them to develop, monitor the emergency condition. Once that clinical obser- and modify patient care plans. This valuable assess- vation is made, you can initiate the appropriate ment tool supplies immediate breath-to-breath treatment for that condition. Then you can con- information about the patient’s respiratory sta- tinue to observe the capnography trend and use it tus: Are they being adequately ventilated? Are they to assist you in determining the effectiveness of the breathing too quickly or slowly? Are they experi- treatment and guide you in continuing or adjusting encing bronchospasm? your treatment as required. Capnography also provides you with key infor- These outstanding assessment capabilities mation about the patient’s circulatory status, such exhibit why capnography is required in multiple as whether they have adequate cardiac output and states and on every ALS unit in Europe. 4 JEMS DEC. 2010 Definition Capnography represents Capnography offers a quantitative numerical read- ing and graphic waveform that measures, illustrates real-time information and documents your patient’s exhaled carbon diox- regarding CO exhalation ide (CO2). All living human beings produce CO2 as a 2 byproduct of metabolism. The carbon dioxide, once and respiratory rates. produced, is diffused into the blood and transported to the lungs via the circulatory system. It’s then released by the alveoli and eliminated from the body measured with arterial blood gases in patients with during exhalation. normal lung function and also in patients with Therefore, capnography enables you to eval- abnormal lung function due to conditions other uate the current status of the patient’s ventilatory, than a ventilation-perfusion mismatch. circulatory and metabolic systems by measuring In patients with a ventilation-perfusion mis- the exhaled CO2 and graphically depicting its path match, the gradient between the ventilation and of exhalation. the perfusion will widen based on the severity of the mismatch. In those cases, the EtCO2 should be used Capnography Use to trend the ventilatory status. An elevated EtCO2 Capnography not only provides you with a rapid level is typically an indication of hypoventilation or and reliable assessment of the patient’s ventilatory, increased metabolic activity. A low exhaled CO2 level circulatory and metabolic function, it also—and more importantly—represents real-time information FIGURE 1 regarding CO2 exhalation and respiratory rates. In the articles that follow, several actual cases will illustrate the effectiveness of capnography as an assessment and monitoring tool in the field. The measurement of CO2 content in each exha- lation reflects the CO2 produced by metabolism, transported by the circulatory system and exhaled by the respiratory system at the time of that particular breath. This allows you to make rapid adjustments in your treatment based on current information. may be an indication of hyperventilation, decreased cardiac output or poor pulmonary perfusion, which Application can occur in shock. It’s important you understand capnography’s The capnography waveform can be compared numerical readings and graphic waveforms to use to an ECG because the “normal” waveform has cer- it effectively. The numeric readings are derived tain rules. Each waveform represents the various from a point in the respiratory cycle known as the phases of inhalation and exhalation and is divided end-tidal CO2 (EtCO2). This is the point at the end into four phases, (see Figure 1 above). of exhalation when the CO2 reaches its highest con- Phase I (A–B) occurs at the beginning of exha- centration. This concentration is generally in the lation when no CO2 is present in the upper airway, range of 35–45 mmHg. trachea, posterior pharynx, mouth and nose. No gas The reading closely correlates to the CO2 levels exchange occurs in these areas, so this “dead space” Hyperventilation Example This is a hyperventilation waveform. Note the high RR and low EtCO2. Figure Pat Brandt Figure MEASURING LIFE & BREATH 5 THE BASICS CONTINUED to the presence of more CO2 per breath, (see Fig- FIGURE 2 ure 3, p. 8). There are, however, other reasons for an increased EtCO2 and increased waveform height. These include a decreased tidal volume with or with- out a decreased respiratory rate, an increased meta- bolic rate and an increased body temperature. The hyperventilation waveform, related to an increased respiratory rate, will have a higher number of waveforms with a decreased height of the waveforms due to the presence of less CO2 per breath, (see Figure 2). As mentioned earlier, other reasons for a decreased EtCO2 and decreased wave- form height include increased tidal volume, a decreased metabolic rate, a decrease in circulation and hypothermia. The bronchospastic capnography waveform is is represented as the flat baseline of the waveform. recognized by a shark-fin shape instead of the nor- Phase II (B–C) is the ascending phase. During mal box-like waveform, (see Figure 4, p. 9). This is this phase, CO2 from the alveoli begins to reach the because bronchospasm causes a slower and more upper airway and mix with the dead space air, caus- erratic emptying of CO2 from the alveoli, which ing a rapid rise in the amount of CO2 detected. results in a slower rise in the expiratory upstroke.
Recommended publications
  • Den170044 Summary
    DE NOVO CLASSIFICATION REQUEST FOR CLEARMATE REGULATORY INFORMATION FDA identifies this generic type of device as: Isocapnic ventilation device. An isocapnic ventilation device is a prescription device used to administer a blend of carbon dioxide and oxygen gases to a patient to induce hyperventilation. This device may be labeled for use with breathing circuits made of reservoir bags (21 CFR 868.5320), oxygen cannulas (21 CFR 868.5340), masks (21 CFR 868.5550), valves (21 CFR 868.5870), resuscitation bags (21 CFR 868.5915), and/or tubing (21 CFR 868.5925). NEW REGULATION NUMBER: 21 CFR 868.5480 CLASSIFICATION: Class II PRODUCT CODE: QFB BACKGROUND DEVICE NAME: ClearMateTM SUBMISSION NUMBER: DEN170044 DATE OF DE NOVO: August 23, 2017 CONTACT: Thornhill Research, Inc. 5369 W. Wallace Ave Scottsdale, AZ 85254 INDICATIONS FOR USE ClearMateTM is intended to be used by emergency department medical professionals as an adjunctive treatment for patients suffering from carbon monoxide poisoning. The use of ClearMateTM enables accelerated elimination of carbon monoxide from the body by allowing isocapnic hyperventilation through simulated partial rebreathing. LIMITATIONS Intended Patient Population is adults aged greater than 16 years old and a minimum of 40 kg (80.8 lbs) ClearMateTM is intended to be used by emergency department medical professionals. This device should always be used as adjunctive therapy; not intended to replace existing protocol for treating carbon monoxide poisoning. When providing treatment to a non-spontaneously breathing patient using the ClearMate™ non-spontaneous breathing patient circuit, CO2 monitoring equipment for the measurement of expiratory carbon dioxide concentration must be used. PLEASE REFER TO THE LABELING FOR A MORE COMPLETE LIST OF WARNINGS AND CAUTIONS.
    [Show full text]
  • Relationship Between PCO 2 and Unfavorable Outcome in Infants With
    nature publishing group Articles Clinical Investigation Relationship between PCO2 and unfavorable outcome in infants with moderate-to-severe hypoxic ischemic encephalopathy Krithika Lingappan1, Jeffrey R. Kaiser2, Chandra Srinivasan3, Alistair J. Gunn4; on behalf of the CoolCap Study Group BACKGROUND: Abnormal PCO2 is common in infants with term infants with HIE, both minimum and cumulative expo- hypoxic ischemic encephalopathy (HIE). The objective was to sure to hypocapnia in the first 12 h of the trial were associated determine whether hypocapnia was independently associated with death or adverse neurodevelopmental outcome (13). The with unfavorable outcome (death or severe neurodevelopmen- dose–response relationship between PCO2 and outcome for tal disability at 18 mo) in infants with moderate-to-severe HIE. infants with moderate-to-severe HIE is unknown. METHODS: This was a post hoc analysis of the CoolCap Study The risk of developing hypocapnia or hypercapnia may be in which infants were randomized to head cooling or standard affected by severity of brain injury, intensity and duration of care. Blood gases were measured at prespecified times after newborn resuscitation, timing after the primary injury, and/or randomization. PCO2 and follow-up data were available for 196 response to metabolic acidosis. Thus, in this secondary analy- of 234 infants. Analyses were performed to investigate the rela- sis of the CoolCap Study (14), we sought to confirm the obser- tionship between hypocapnia in the first 72 h after randomiza- vation that
    [Show full text]
  • Laryngeal Spasm Mimicking Asthma and Vitamin D Deficiency
    Case Report Allergy Asthma Immunol Res. 2014 May;6(3):267-269. http://dx.doi.org/10.4168/aair.2014.6.3.267 pISSN 2092-7355 • eISSN 2092-7363 Laryngeal Spasm Mimicking Asthma and Vitamin D Deficiency Monica Masoero,1 Michela Bellocchia,1 Antonio Ciuffreda,1 Fabio LM Ricciardolo,2 Giovanni Rolla,1 Caterina Bucca1* 1Department of Medical Sciences, University of Turin, Italy 2Department of Clinical and Biological Sciences, University of Turin, Italy This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. We present a woman with heterozygous carnitine palmitoyl transferase 2 (CPT-2) deficiency who in the last 6 months suffered from episodic dyspnea and choking. Symptoms could not be attributed to her muscular energy defect, since heterozygous CPT-2 deficiency is usually asymptomatic or causes only mild muscle fatigability. Myopathy is usually triggered by concurrent factors, either genetic (additional muscle enzymes defects) or acquired (metabolic stress). The patient was referred to our respiratory clinic for suspect bronchial asthma. Spirometry showed mild decrease in inspiratory flows. Methacholine challenge was negative. Dyspnea was triggered by hyperventilation-induced hypocapnia, which produced marked decrease in airflow rates, particularly in inspiratory flows, consistent with laryngospasm. Nutritional assessment of the patient showed low serum level of calci- um and vitamin D, attributable to avoidance of milk and dairy products for lactose intolerance and to insufficient sunlight exposure. After calcium and vitamin D supplementation episodic laryngospasm disappeared and hypocapnic hyperventilation test induced very mild change in airflow rates.
    [Show full text]
  • Hyperventilation and the Body
    Hyperventilation and the body C. Gilbert Hyperventilation has rapid and far-ranging physiological effects via its alteration of pH and depletion of CO 2 in the body, resulting in respiratory alkalosis, acute or chronic. The general effects on skeletal and smooth muscles, as well as neural tissue, are summarized. A wide variety of symptoms such as pain, tension, disturbances of consciousness, circulatory problems and cardiovascular effects can confound treatment efforts but may respond to alterations of breathing patterns. Suggestions are given for detecting this condition. Introduction Background Imagine the following set of symptoms reported The 'Fat folder' syndrome is one vivid description to you: episodes of light-headedness attached by Claude Lum to the so-called accompanied by cool, moist hands, pale skin, hyperventilation syndrome, indicating the perhaps some tunnel vision, chest pain, tortuous, and tortured, doctor-to-doctor path headaches, feelings of unreality, dread and followed by many individuals pursuing relief agitation; heart palpitations, uncomfortable from their problems. Each specialist performs his breathing and tender shoulder muscles. Suppose tests, finds little that is specifically diagnostic, and that medical consultation has found little of note, dutifully relays the individual on to the next but at least there is no evidence of major disease. specialist because typically, with this disorder, Where would you start? A break for lunch, symptoms come and go in many different perhaps? systems. Hyperventilation simply means breathing in Christopher Gilbert In working with such a diffuse set of PhD Social Sciences complaints spread out over many systems, a excess of the body's needs for oxygen at a and Human practitioner will naturally tend to focus on the particular moment.
    [Show full text]
  • Essential Expectations-Avoiding Hypocapnia and Hyperoxemia in Neonatal Encephalopathy
    Journal of Pediatrics and Neonatal Care Essential Expectations-Avoiding Hypocapnia and Hyperoxemia in Neonatal Encephalopathy Mini Review Mini Review Therapeutic hypothermia (TH) is the accepted treatment modality for newborns with moderate to severe neonatal Volume 6 Issue 5 - 2017 encephalopathy likely originating from a hypoxic-ischemic event. Numerous randomized, controlled trials and meta-analyses have shown that TH reduces that the risk of death or major Baystate Children’s Hospital, USA neurodevelopmental disability by 25-35% among treated infants *Corresponding author: Jeffrey Shenberger, Baystate [1,2]. Lung injury, with or without effective respiratory control, Children’s Hospital, Division of Newborn Medicine 759 is present in as many as 80% of infants with moderate-to-severe neonatal encephalopathy who meet the inclusion criteria for TH [3]. 009-3; Email: Although these encephalopathic newborns often require oxygen Chestnut Street Springfield, MA 01199, USA, Tel: 413-794- supplementation or ventilatory support to provide adequate Received: March 08, 2017 | Published: April 04, 2017 gas exchange, their rapidly changing pulmonary mechanics and respiratory drive makes achieving and maintaining eucapnia and normoxemia clinically challenging. Accumulating evidence from clinical trials illustrates the importance of controlling the Although hypocapnia in neonatal encephalopathy may be partial pressures of carbon dioxide (PCO2) and oxygen (PO2) in a surrogate marker for brain injury severity, PCO2 represents order to maximize neurodevelopmental outcomes and ameliorate secondary brain injury resulting from over ventilation and recovery. As many infants undergoing TH are intubated for apnea, oxygenation. frequenta readily bloodmodifiable gas monitoring factor capable and/or of transcutaneousmaximizing neurologic or end- tidal CO monitoring are essential.
    [Show full text]
  • Diving Physiology 3
    Diving Physiology 3 SECTION PAGE SECTION PAGE 3.0 GENERAL ...................................................3- 1 3.3.3.3 Oxygen Toxicity ........................3-21 3.1 SYSTEMS OF THE BODY ...............................3- 1 3.3.3.3.1 CNS: Central 3.1.1 Musculoskeletal System ............................3- 1 Nervous System .........................3-21 3.1.2 Nervous System ......................................3- 1 3.3.3.3.2 Lung and 3.1.3 Digestive System.....................................3- 2 “Whole Body” ..........................3-21 3.2 RESPIRATION AND CIRCULATION ...............3- 2 3.2.1 Process of Respiration ..............................3- 2 3.3.3.3.3 Variations In 3.2.2 Mechanics of Respiration ..........................3- 3 Tolerance .................................3-22 3.2.3 Control of Respiration..............................3- 4 3.3.3.3.4 Benefits of 3.2.4 Circulation ............................................3- 4 Intermittent Exposure..................3-22 3.2.4.1 Blood Transport of Oxygen 3.3.3.3.5 Concepts of and Carbon Dioxide ......................3- 5 Oxygen Exposure 3.2.4.2 Tissue Gas Exchange.....................3- 6 Management .............................3-22 3.2.4.3 Tissue Use of Oxygen ....................3- 6 3.3.3.3.6 Prevention of 3.2.5 Summary of Respiration CNS Poisoning ..........................3-22 and Circulation Processes .........................3- 8 3.2.6 Respiratory Problems ...............................3- 8 3.3.3.3.7 The “Oxygen Clock” 3.2.6.1 Hypoxia .....................................3-
    [Show full text]
  • Gas Exchange and Ventilation–Perfusion Relationships in the Lung
    ERJ Express. Published on July 25, 2014 as doi: 10.1183/09031936.00037014 REVIEW IN PRESS | CORRECTED PROOF Gas exchange and ventilation–perfusion relationships in the lung Johan Petersson1,2 and Robb W. Glenny3,4 Affiliations: 1Section of Anaesthesiology and Intensive Care Medicine, Dept of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden. 2Dept of Anaesthesiology, Surgical Services and Intensive Care, Karolinska University Hospital, Stockholm, Sweden. 3Dept of Medicine, Division of Pulmonary and Critical Care Medicine, University of Washington, Seattle, WA, USA. 4Dept of Physiology and Biophysics, University of Washington, Seattle, WA, USA. Correspondence: Johan Petersson, Dept of Anaesthesiology, Surgical Services and Intensive Care, Karolinska University Hospital, 17176 Stockholm, Sweden. E-mail: [email protected] ABSTRACT This review provides an overview of the relationship between ventilation/perfusion ratios and gas exchange in the lung, emphasising basic concepts and relating them to clinical scenarios. For each gas exchanging unit, the alveolar and effluent blood partial pressures of oxygen and carbon dioxide (PO2 and PCO2) are determined by the ratio of alveolar ventilation to blood flow (V9A/Q9) for each unit. Shunt and low V9A/Q9 regions are two examples of V9A/Q9 mismatch and are the most frequent causes of hypoxaemia. Diffusion limitation, hypoventilation and low inspired PO2 cause hypoxaemia, even in the absence of V9A/Q9 mismatch. In contrast to other causes, hypoxaemia due to shunt responds poorly to supplemental oxygen. Gas exchanging units with little or no blood flow (high V9A/Q9 regions) result in alveolar dead space and increased wasted ventilation, i.e. less efficient carbon dioxide removal.
    [Show full text]
  • AIRWAY MANAGEMENT: HYPE ABOUT VENTILATION and the ANETHETIZED PATIENT Donna M
    AIRWAY MANAGEMENT: HYPE ABOUT VENTILATION AND THE ANETHETIZED PATIENT Donna M. Sisak, CVT, LVT, VTS (Anesthesia/Analgesia) Seattle Veterinary Specialists Kirkland, WA [email protected] “Life is not measured by the number of breaths we take but by the moments that take our breath away.” Author unknown An anesthetic event can be divided into the following components: premedication, induction, maintenance, and recovery. Each component is equally important and requires extreme vigilance by the anesthetist. Most of the drugs used (anesthetics/analgesics) throughout an anesthetic event are respiratory depressants. Until we obtain the patient’s airway (intubation) we really have no control over our patient’s ventilatory status and well-being. The patient is at risk of becoming hypoxic (inadequate O2 at the tissue level); eventually leading to other possible complications – aspiration pneumonia, decreased cardiac output (CO), decreased perfusion, and potentially cardiac arrest. Induction of an anesthetic event is defined as: drug administration to change the state of the patient from AWAKE to ANESTHETIZED and eventually securing the patient’s airway by placing a flexible rubber (endotracheal) down the trachea. Endotracheal intubation allows us to isolate the patient’s airway and support respiratory function during an anesthetic event. Safe anesthetic management requires adequate support and function to the respiratory system. It is the responsibility of the anesthetist to assure appropriate airway management during an anesthetic event. The anesthetist should be familiar with respiratory terminology as well as the equipment available for the patient. Endotracheal Intubation Endotracheal intubation is the placement of an endotracheal (ET) tube positioned between the vocal cords and down the trachea.
    [Show full text]
  • Near-Drowning: Prevention Is the Key!
    Near-drowning: Prevention is the key! Lisa Reichter BSN, RN, CCRN Trauma Nurse Coordinator AAP Latest Statistics: (Pediatrics (2012; 129:1-7) • Hospitalizations fell from an estimated 3623 in 1993 to 1781 in 2008 – Pediatric (age 0-19 years) hospitalizations associated with near drowning declined 51%! – From 3623 in 1993 to 1781 in 2008 – Annual hospital days declined • From 14,570 days in 1993; 6295 days in 2008 Relative Contribution of Various Submersion Media to Drowning Accidents Salt Water 1 - 2% Fresh Water 98% swimming pools: public 50% swimming pools: private 3% lakes, rivers, streams, storm drains 20% bathtubs 15% buckets of water 4% fish tanks or pools 4% toilets 1% washing machines 1% Drowning Characteristics • Differ by age and sex • Young children (<4 yrs) have the greatest mortality rate – More likely to drown from bathing or falling into water – Reduction in these drownings from targeted injury prevention efforts • Older children – More likely to drown while swimming in open water • Males are 4-6x more likely than females – Overestimation of swimming ability – Greater use of alcohol by adolescent males Near Drowning Risk Factors: Age 600 500 400 300 Male Female 200 100 0 0-4 yr 5-9 yr 10-14 yr 15-19 Other Causes Spas, Hot Tubs • Entrapment – drains • hair, body parts, clothing – winter pool/spa covers Decrease in Death Rates • In-hospital mortality decreased 42% – From estimated 359 deaths in 1993 to 207 in 2008 ―Although improvements in treatment might be having an impact on survival, it is not clear from these data what level of neurologic functioning survivors may have.
    [Show full text]
  • Breath Hold Training Position Statement.Pdf
    BREATH HOLD TRAINING: POSITION STATEMENT Mark Wilson, Nirmala Perera, Richard Saw, David Hughes (Australian Institute of Sport), Ana Holt (Victorian Institute of Sport) Breath Hold Training: Position Statement 1 INTRODUCTION Breath hold training (BHT) is a technique utilised in sport and exercise science to increase athletes’ tolerance for oxygen deprivation. Another desired outcome for non-breath-hold sports is improved oxygen delivery. BHT is applied across many different sports, but most work has focused on swimming and water-based activities. There have been significant safety concerns raised about the practice of BHT in Australia and internationally. Much research has focused on the proven risks to athletes and the modest benefits in terms of tolerance to oxygen deprivation.1-3 The risks of BHT are potentially higher when training limits are pushed in the quest of sporting success. Given the health risks posed by BHT, medical oversight should be built into all relevant sporting environments. Education and support should be provided to stakeholders to mitigate the risks. Swimming Australia recognised the importance of this issue and created a position statement in 2003.4 There have been no recent cross-sport recommendations regarding the practice of BHT. This Australian Institute of Sport (AIS) position statement will guide athletes, coaches and support personnel in their considerations of BHT, prioritising athlete safety and wellbeing in the high-performance environment. WHAT ARE THE DIFFERENT TYPES OF BREATH HOLD TRAINING? There are a range of activities which constitute BHT; many of these are sport specific. The practice of BHT can involve underwater only, shallow water, deep dives, on-land or above water, passive or dynamic, maximal or sub-maximal durations, repeated or single breath holds.
    [Show full text]
  • Simple New Method to Accelerate Clearance of Carbon Monoxide
    PublisherInfo PublisherName : BioMed Central PublisherLocation : London PublisherImprintName : BioMed Central Simple new method to accelerate clearance of carbon monoxide ArticleInfo ArticleID : 4212 ArticleDOI : 10.1186/ccf-2000-5638 ArticleCitationID : 5638 ArticleSequenceNumber : 71 ArticleCategory : Paper Report ArticleFirstPage : 1 ArticleLastPage : 3 RegistrationDate : 2000–6–30 ArticleHistory : OnlineDate : 2000–6–30 ArticleCopyright : Current Science Ltd2000 ArticleGrants : ArticleContext : 1305422 Jonathan Ball,Aff1 Aff1 St George's Hospital, London, UK Keywords Carbon monoxide poisoning, respiratory therapy Comments This comparatively simple and potentially portable technique appears to be safe and efficacious in the treatment of carbon monoxide (CO) poisoning. The authors argue that the potential deleterious effects of hypocapnia in CO poisoning (being a rightward shift in the oxygen dissociation curve as well as cerebral vasoconstriction) could be prevented by this technique. In addition, the enhanced clearance of CO achieved should reduce the duration of hypoxia and the direct intracellular toxic effects of CO. This well conducted study justifies a clinical trial of this technique, especially in the pre-hospital environment. Introduction CO inhalation is the leading cause of fatal poisoning in the industrialised world. Current therapy consists of either the administration of 100% oxygen, or, where available, hyperbaric oxygen. Inhaled carbon dioxide (CO2) (5%) therapy as an adjunct to oxygen was advocated in the 1920s but fell from routine use due to its poor tolerability. Similarly, hyperpnea, despite enhancing CO elimination, provokes a respiratory alkalosis with consequent aggravation of already impaired cerebral oxygen delivery. Fisher and colleagues have previously demonstrated the efficacy of a novel breathing circuit in dogs which provokes normocapnic hyperoxic hyperpnea producing a > 50% reduction in CO half life.
    [Show full text]
  • Treatment of Carbon Monoxide Poisoning by Mechanical Ventilation: Case Report*
    TREATMENT OF CARBON MONOXIDE POISONING BY MECHANICAL VENTILATION: CASE REPORT* GARY L. TOWNSEND, M.D., AND JOHN B. STETSON, M.D.~ StacmE is among the ten leading causes of death in industrialized countries. During 1965 there were 22,000 reported suicides in the United States (statistics compiled by the Center for Studies of Suicide Prevention). There is no way of estimating the incidence of unreported suicides, or of the number of survivors after attempted suicide. One of the more frequent modes of suicide is carbon monoxide inhalation. As an accidental cause of death, carbon monoxide poisoning ranks second only to alcohol,x This, combined with the increasing incidence of non-accidental carbon monoxide poisoning, has made the acute management of these cases a pertinent problem in modern medicine. As an example of the extent of the "disease," in the period 1950 to 1955, carbon monoxide poisoning represented 2.1 per cent of eases investigated by the Los Angeles county coroner's oflqce (875 of 41,615). 2 Mann, in 1958, found 1,156 cases of carbon monoxide poisoning out of a total of 18,513 suicides) In the state of Indiana, in 1965, there were 80 suicides by carbon monoxide inhalation and 47 accidental poisonings.4 Carbon monoxide has been termed the perfect asphyxiant. Haemoglobin com- bines with carbon monoxide 200-300 times as readily as it does with oxygen and dissociates from it 250 times as slowlyP The poisonous effects of carbon monoxide are derived from its double action in the production of asphyxia. First, any excess in inspired air reduces the percentage of oxygen inhaled.
    [Show full text]