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Occupational Airborne Particulates
Environmental Burden of Disease Series, No. 7 Occupational airborne particulates Assessing the environmental burden of disease at national and local levels Tim Driscoll Kyle Steenland Deborah Imel Nelson James Leigh Series Editors Annette Prüss-Üstün, Diarmid Campbell-Lendrum, Carlos Corvalán, Alistair Woodward World Health Organization Protection of the Human Environment Geneva 2004 WHO Library Cataloguing-in-Publication Data Occupational airborne particulates : assessing the environmental burden of disease at national and local levels / Tim Driscoll … [et al.]. (Environmental burden of disease series / series editors: Annette Prüss-Ustun ... [et al.] ; no. 7) 1.Dust - adverse effects 2.Occupational exposure 3.Asthma - chemically induced 4.Pulmonary disease, Chronic obstructive - chemically induced 5.Pneumoconiosis - etiology 6.Cost of illness 7.Epidemiologic studies 8.Risk assessment - methods 9.Manuals I.Driscoll, Tim. II.Prüss-Üstün, Annette. III.Series. ISBN 92 4 159186 2 (NLM classification: WA 450) ISSN 1728-1652 Suggested Citation Tim Driscoll, et al. Occupational airborne particulates: assessing the environmental burden of disease at national and local levels. Geneva, World Health Organization, 2004. (Environmental Burden of Disease Series, No. 7). © World Health Organization 2004 All rights reserved. Publications of the World Health Organization can be obtained from Marketing and Dissemination, World Health Organization, 20 Avenue Appia, 1211 Geneva 27, Switzerland (tel: +41 22 791 2476; fax: +41 22 791 4857; email: [email protected]). -
Interstitial Lung Diseases in Developing Countries
Rivera-Ortega P and Molina-Molina M. Interstitial Lung Diseases in Developing Countries. Annals of Global Health. 2019; 85(1): 4, 1–14. DOI: https://doi.org/10.5334/aogh.2414 REVIEW Interstitial Lung Diseases in Developing Countries Pilar Rivera-Ortega*,† and Maria Molina-Molina*,† More than 100 different conditions are grouped under the term interstitial lung disease (ILD). A diag- nosis of an ILD primarily relies on a combination of clinical, radiological, and pathological criteria, which should be evaluated by a multidisciplinary team of specialists. Multiple factors, such as environmental and occupational exposures, infections, drugs, radiation, and genetic predisposition have been implicated in the pathogenesis of these conditions. Asbestosis and other pneumoconiosis, hypersensitivity pneumonitis (HP), chronic beryllium disease, and smoking-related ILD are specifically linked to inhalational exposure of environmental agents. The recent Global Burden of Disease Study reported that ILD rank 40th in relation to global years of life lost in 2013, which represents an increase of 86% compared to 1990. Idiopathic pulmonary fibrosis (IPF) is the prototype of fibrotic ILD. A recent study from the United States reported that the incidence and prevalence of IPF are 14.6 per 100,000 person-years and 58.7 per 100,000 persons, respectively. These data suggests that, in large populated areas such as Brazil, Russia, India, and China (the BRIC region), there may be approximately 2 million people living with IPF. However, studies from South America found much lower rates (0.4–1.2 cases per 100,000 per year). Limited access to high- resolution computed tomography and spirometry or to multidisciplinary teams for accurate diagnosis and optimal treatment are common challenges to the management of ILD in developing countries. -
Guidelines for Human Exposure Assessment Risk Assessment Forum
EPA/100/B-19/001 October 2019 www.epa.gov/risk Guidelines for Human Exposure Assessment Risk Assessment Forum EPA/100/B-19/001 October 2019 Guidelines for Human Exposure Assessment Risk Assessment Forum U.S. Environmental Protection Agency DISCLAIMER This document has been reviewed in accordance with U.S. Environmental Protection Agency (EPA) policy. Mention of trade names or commercial products does not constitute endorsement or recommendation for use. Preferred citation: U.S. EPA (U.S. Environmental Protection Agency). (2019). Guidelines for Human Exposure Assessment. (EPA/100/B-19/001). Washington, D.C.: Risk Assessment Forum, U.S. EPA. Page | ii TABLE OF CONTENTS DISCLAIMER ..................................................................................................................................... ii LIST OF TABLES.............................................................................................................................. vii LIST OF FIGURES ........................................................................................................................... viii LIST OF BOXES ................................................................................................................................ ix ABBREVIATIONS AND ACRONYMS .............................................................................................. x PREFACE ........................................................................................................................................... xi AUTHORS, CONTRIBUTORS AND -
Personal Protective Equipment (PPE) Guide
Personal Protective Equipment (PPE) Guide Volume 1: General PPE February 2003 F417-207-000 This guide is designed to be used by supervisors, lead workers, managers, employers, and anyone responsible for the safety and health of employees. Employees are also encouraged to use information in this guide to analyze their own jobs, be aware of work place hazards, and take active responsibility for their own safety. Photos and graphic illustrations contained within this document were provided courtesy of the Occupational Safety and Health Administration (OSHA), Oregon OSHA, United States Coast Guard, EnviroWin Safety, Microsoft Clip Gallery (Online), and the Washington State Department of Labor and Industries. TABLE OF CONTENTS (If viewing this pdf document on the computer, you can place the cursor over the section headings below until a hand appears and then click. You can also use the Adobe Acrobat Navigation Pane to jump directly to the sections.) How To Use This Guide.......................................................................................... 4 A. Introduction.........................................................................................6 B. What you are required to do ..............................................................8 1. Do a Hazard Assessment for PPE and document it ........................................... 8 2. Select and provide appropriate PPE to your employees................................... 10 3. Provide training to your employees and document it ........................................ 11 -
7. Tasks/Procedures 8. Hazards 9. Abatement
FS-6700-7 (11/99) 1. WORK PROJECT/ 2. LOCATION 3. UNIT(S) U.S. Department of Agriculture ACTIVITY Coconino National Forest All Districts Forest Service Trail Maintenance JOB HAZARD ANALYSIS (JHA) 4. NAME OF ANALYST 5. JOB TITLE 6. DATE PREPARED References-FSH 6709.11 and 12 Amy Racki Partnership Coordinator 10/28/2013 9. ABATEMENT ACTIONS 7. TASKS/PROCEDURES 8. HAZARDS Engineering Controls * Substitution * Administrative Controls * PPE Personal Protective Equipment Wear helmet, work gloves, boots with slip-resistant heels and soles with firm, flexible support, eye protection, long sleeve shirts, long pants, hearing protection where appropriate Carry first aid kit Vehicle Operation Fatigue Drive defensively and slow. Watch for animals Narrow, rough roads Always wear seatbelts and turn lights on Poor visibility Mechanical failure Ensure that you have reliable communication Vehicle Accients Obey speed limits Weather Keep vehicles maintained. Keep windows and windshield clean Animals on Road Anticipate careless actions by other drivers Use spotter when backing up Stay clear of gullies and trenches, drive slowly over rocks. Carry and use chock blocks, use parking brake, and do not leave vehicle while it is running Inform someone of your destination and estimated time of return, call in if plans change Carry extra food, water, and clothing Stop and rest if fatigued Hiking on the Trail Dehydration Drink 12-15 quarts of water per day, increase fluid on hotter days or during extremely strenuous activity Contaminated water Drink -
Safety Manual
Eastern Elevator Safety and Health Manual January 2017 Page 1 of 125 SAFETY AND HEALTH MANUAL Index by Section Number Safety Policy ................................................................................................................. …4 Section 1: General Health and Safety Policies ............................................................. …5 Section 2: Hazard Communication ................................................................................ ..13 Section 3: Personal Protective Equipment (PPE) ......................................................... ..18 Section 4: Emergency Action Plan ................................................................................ ..31 Section 5: Fall Protection .............................................................................................. ..34 Section 6: Ladder Safety ............................................................................................... ..38 Section 7: Bloodborne Pathogens ................................................................................ ..42 Section 8: Motor Vehicle / Fleet Safety ......................................................................... ..47 Section 9: Hearing Conservation .................................................................................. ..50 Section 10: Lockout / Tagout ........................................................................................ ..53 Section 11: Fire Prevention .......................................................................................... -
Pneumoconiosis
Prim Care Respir J 2013; 22(2): 249-252 PERSPECTIVE Pneumoconiosis *Paul Cullinan1, Peter Reid2 1 Consultant Physician, Royal Brompton and Harefield NHS Foundation Trust, London, UK 2 Consultant Physician, Western General Hospital, Edinburgh, UK Introduction Figure 1. Asbestosis; the HRCT scan shows the typical The pneumoconioses are parenchymal lung diseases that arise from picture of subpleural fibrosis (solid arrow); in addition inhalation of (usually) inorganic dusts at work. Some such dusts are there is diffuse, left-sided pleural thickening (broken biologically inert but visible on a chest X-ray or CT scan; thus, while arrow), characteristic too of heavy asbestos exposure they are radiologically alarming they do not give rise to either clinical disease or deficits in pulmonary function. Others – notably asbestos and crystalline silica – are fibrogenic so that the damage they cause is through the fibrosis induced by the inhaled dust rather than the dust itself. Classically these give rise to characteristic radiological patterns and restrictive deficits in lung function with reductions in diffusion capacity; importantly, they may progress long after exposure to the causative mineral has finished. In the UK and similar countries asbestosis is the commonest form of pneumoconiosis but in less developed parts of the world asbestosis is less frequent than silicosis; these two types are discussed in detail below. Other, rarer types of pneumoconiosis include stannosis (from tin fume), siderosis (iron), berylliosis (beryllium), hard metal disease (cobalt) and coal worker’s pneumoconiosis. Asbestosis Clinical scenario How is the diagnosis made? Asbestosis is the ‘pneumoconiosis’ that arises from exposure to A man of 78 reports gradually worsening breathlessness; he has asbestos in the workplace.1 The diagnosis is made when, on the no relevant medical history of note and has never been a regular background of heavy occupational exposure to any type of asbestos, smoker. -
A PRACTICAL GUIDE for USE of REAL TIME DETECTION SYSTEMS for WORKER PROTECTION and COMPLIANCE with OCCUPATIONAL EXPOSURE LIMITS May 2019
A PRACTICAL GUIDE FOR USE OF REAL TIME DETECTION SYSTEMS FOR WORKER PROTECTION AND COMPLIANCE WITH OCCUPATIONAL EXPOSURE LIMITS May 2019 WHITE PAPER May 2019 A PRACTICAL GUIDE FOR USE OF REAL TIME DETECTION SYSTEMS FOR WORKER PROTECTION AND COMPLIANCE WITH OCCUPATIONAL EXPOSURE LIMITS Prepared by: Energy Facility Contractor’s Group (EFCOG) Industrial Hygiene and Safety Task Group and Members of the American Industrial Hygiene Association (AIHA) Exposure Assessment Strategies Group Dina Siegel1, David Abrams 2, John Hill3, Steven Jahn2, Phil Smith2, Kayla Thomas4 1 Los Alamos National Laboratory 2 AIHA Exposure Assessment Strategies Committee 3 Savannah River Site 4 Kansas City National Security Campus A PRACTICAL GUIDE FOR USE OF REAL TIME DETECTION SYSTEMS FOR WORKER PROTECTION AND COMPLIANCE WITH OCCUPATIONAL EXPOSURE LIMITS May 2019 Table of Contents 1.0 Executive Summary 2.0 Introduction 3.0 Discussion 3.1 Occupational Exposure Assessment 3.2 Regulatory Compliance 3.3 Occupational Exposure Limits 3.4 Traditional Use of Real Time Detection Systems 3.5 Use and Limitations of Real Time Detection Systems 3.6 Use of Real Time Detection Systems for Compliance 3.7 Documentation/Reporting of Real Time Detection Systems Results 3.8 Peak Exposures Data Interpretations 3.9 Conclusions 4.0 Matrices 5.0 References 6.0 Attachments Page | 2 A PRACTICAL GUIDE FOR USE OF REAL TIME DETECTION SYSTEMS FOR WORKER PROTECTION AND COMPLIANCE WITH OCCUPATIONAL EXPOSURE LIMITS May 2019 1.0 Executive Summary This white paper presents practical guidance for field industrial hygiene personnel in the use and application of real time detection systems (RTDS) for exposure monitoring. -
Respiratory Function Changes After Asbestos Pleurisy
Thorax: first published as 10.1136/thx.35.1.31 on 1 January 1980. Downloaded from Thorax, 1980, 35, 31-36 Respiratory function changes after asbestos pleurisy P H WRIGHT, A HANSON, L KREEL, AND L H CAPEL From the Cardiothoracic Institute, London Chest Hospital, East Ham Chest Clinic, London, and the Division of Radiology, Clinical Research Centre, Northwick Park Hospital, Harrow ABSTRACT Six patients with radiographic evidence of diffuse pleural thickening after industrial asbestos exposure are described. Five had computed tomography of the thorax. All the scans showed marked circumferential pleural thickening often with calcification, and four showed no significant evidence of intrapulmonary fibrosis (asbestosis). Lung function testing showed reduc- tion of the inspiratory capacity and the single-breath carbon monoxide transfer factor (TLco). The transfer coefficient, calculated as the TLCO divided by the alveolar volume determined by helium dilution during the measurement of TLco, was increased. Pseudo-static compliance curves showed markedly more negative intrapleural pressures at all lung volumes than found in normal people. These results suggest that the circumferential pleural thickening was preventing normal lung expansion despite abnormally great distending pressures. The pattern of lung function tests is sufficiently distinctive for it to be recognised in clinical practice, and suggests that the lungs are held rigidly within an abnormal pleura. The pleural thickening in our patients may have been related to the condition described as "benign asbestos pleurisy" rather than the copyright. interstitial fibrosis of asbestosis. Malignant pleural effusion associated with meso- necessary in many of these early cases, and opera- thelioma or bronchial carcinoma is a recognised tion notes recorded the presence of marked http://thorax.bmj.com/ complication of asbestos exposure. -
Supplementary File 2: Assessment of Quality and Bias
Supplementary File 2: Assessment of quality and bias In preparing the tool, it was not possible to identify a dominant factor for which studies should be controlled for, instead recognising several key variables. Comparability was therefore scored against a single criterion of ‘effectively controlling for relevant factors’, meaning a maximum of eight points, rather than nine, were available. Where a nested cohort or matched case group was identified within an interventional study, it was agreed that the quality of the study as applied to impact of ETS would be appraised, and not the main interventional study outcomes. This acknowledged that high quality trials not relevant to the impact of ETS exposure could contain poor quality cohorts that were appropriate for inclusion, or vice versa. A point was awarded for satisfactory response on each question, indicated by a tick in the relevant table. Cohort Outcome Measures Domain and outcomes Point Representativeness of the exposed cohort A Truly representative of the paediatric population undergoing surgery B Somewhat representative of the paediatric population undergoing surgery C Selected subgroup of the population undergoing surgery D No description of the cohort selection process Selection of the non-exposed cohort A Drawn from the same community as the exposed cohort B Drawn from a different source C No description of the derivation of the non-exposed cohort Ascertainment of exposure A Secure record (e.g. surgical record, biological test) B Structured interview C Written self-report of self-completed questionnaire D No description Demonstration that outcome of interest was not present at start of the study A Yes B No Comparability of cohorts on the basis of the design or analysis A Study controls for reasonable factors (e.g. -
Hazard Control Selection and Management Requirements
ENVIRONMENT, SAFETY & HEALTH DIVISION Chapter 1: General Policy and Responsibilities Hazard Control Selection and Management Requirements Product ID: 671 | Revision ID: 1744 | Date published: 27 May 2015 | Date effective: 27 May 2015 URL: http://www-group.slac.stanford.edu/esh/eshmanual/references/eshReqControls.pdf 1 Purpose This document defines how a risk-based approach is used to determine the need for controls on facilities, systems, or components to protect the public, workers, and the environment. For controls necessary to prevent or mitigate serious events, specific devices and procedures will be formally credited as part of the approved safety envelope. How these controls are selected, evaluated, and approved, and the process for maintaining and modifying controls, are described in these requirements1. As used here, controls and hazard controls mean those engineered, administrative, or personal protective elements that are used to protect against a hazard. Normal process or operational controls are not included in these requirements except to the extent that their use is directly tied to safety. The concept of credited control is well established in the accelerator safety community. The concept of credited control is borrowed from DOE Order 420.2C, “Safety of Accelerator Facilities” (DOE O 420.2C), but this document neither extends the requirements of DOE O 420.2C to non-accelerator hazards nor modifies those requirements for accelerator hazards. The intent is to extend those robust principles to management of controls for non-accelerator hazards of similar risk. 2 Roles and Responsibilities 2.1 Associate Laboratory Director . Ensures that technical systems under his or her directorate’s management are properly analyzed to determine the type and level of controls necessary to control risk to an acceptable level . -
Code of Practice
HOW TO MANAGE WORK HEALTH AND SAFETY RISKS Code of Practice DECEMBER 2011 Safe Work Australia is an Australian Government statutory agency established in 2009. Safe Work Australia consists of representatives of the Commonwealth, state and territory governments, the Australian Council of Trade Unions, the Australian Chamber of Commerce and Industry and the Australian Industry Group. Safe Work Australia works with the Commonwealth, state and territory governments to improve work health and safety and workers’ compensation arrangements. Safe Work Australia is a national policy body, not a regulator of work health and safety. The Commonwealth, states and territories have responsibility for regulating and enforcing work health and safety laws in their jurisdiction. NSW note: This code of practice has been approved under section 274 of the Work Health and Safety Act 2011. Notice of that approval was published in the NSW Government Gazette referring to this code of practice as How to manage work health and safety risks (page 7194) on Friday 16 December 2011. This code of practice commenced on 1 January 2012. ISBN 978-0-642-33301-8 [PDF] ISBN 978-0-642-33302-5 [RTF] Creative Commons Except for the logos of Safe Work Australia, SafeWork SA, Workplace Standards Tasmania, WorkSafe WA, Workplace Health and Safety QLD, NT WorkSafe, WorkCover NSW, Comcare and WorkSafe ACT, this copyright work is licensed under a Creative Commons Attribution-Noncommercial 3.0 Australia licence. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc/3.0/au/ In essence, you are free to copy, communicate and adapt the work for non commercial purposes, as long as you attribute the work to Safe Work Australia and abide by the other licence terms.