NBRC Therapist Written RRT Examination
Total Page：16
File Type：pdf, Size：1020Kb
Load more
Recommended publications
-
Respiratory Therapy Program
UNIVERSITY OF THE DISTRICT OF COLUMBIA RESPIRATORY THERAPY PROGRAM The University offers the A.A.S. Degree in Respiratory ASSOCIATE IN APPLIED SCIENCE DEGREE IN Therapy. The curriculum reflects high standards of RESPIRATORY THERAPY professional practice and incorporates guidelines from practice trends, professional organizations and accrediting Total Credit Hours of College-Level Courses Required agencies. Students develop the knowledge base and For Graduation: 70 clinical competencies required to meet the health care needs of patients with cardiopulmonary disorders. The program offers both a day and an evening option. Respiratory Therapists treat patients along the age and health-care continuums – from premature infants to the PROGRAM OF STUDY aged in critical care, acute care, rehabilitation, and home care settings. PREREQUISITES 1535-101 General College Math I 3 ACCREDITATION & CREDENTIALING 1133-111 English Composition I 3 1401-112 Anatomy and Physiology II – Lecture 3 The UDC Respiratory Therapy Program is accredited by 1401-114 Anatomy and Physiology II – Lab 1 the Commission on Accreditation of Allied Health Total 10 Credits Education Programs (CAAHEP), in collaboration with the FIRST YEAR – FALL SEMESTER Committee on Accreditation for Respiratory Care 1431-170 Introduction to Health Sciences 2 1431-171 Principles and Practice of Resp Therapy I 4 (CoARC). Graduates are eligible for both the entry-level licensure/ CRT examination (required by the District of 1401-112 Anatomy and Physiology II – Lecture 3 Columbia, Maryland and Virginia) and the advanced 1401-114 Anatomy and Physiology II – Lab 1 1133-112 or 1535-102 English Composition II or practice RRT examinations, both offered by the National Board for Respiratory Care (NBRC). -
Lung Function Studies in Diagnostics and Follow-Up of Pulmonary Sarcoidosis
Lung Function Studies in Diagnostics and Follow-up of Pulmonary Sarcoidosis By INGELA BRÅDVIK Lund 1994 From the Department of Lung Medicine and the Department of Clinical Physiology University of Lund, Sweden Lung Function Studies in Diagnostics and Follow-up of Pulmonary Sarcoidosis Ingela Brådvik Lund 1994 Organization Document name LUND UNIVERSITY DOCTORAL DISSERTATION Department of Lung Medicine Date of issue University Hospital 94 06 09 S-22I 85 Lund CODEN: ISRN LUMEDW/MELL- - 1007- - SE Authorfj) Sponsoring organization Ingela Brådvik The Swedish Heart Lung Foundation Title and subtitle Lung function studies in diagnostics and follow-up of pulmonary sarcoidosis Abstract In 66 patients the relationship between lung volumes and lung mechanics in pulmonary sarcoidosis was investigated Lung volumes, static lung mechanics, lung resistance, dynamic lung mechanics and arterial blood gases at rest and during exercise were obtained. Fifteen functionally compromised patients received steroids during one year. They were re-investigated during the treatment and at a follow-up after an average of 7 years. In another 41 patients with newly diagnosed sarcoidosis, the kinetics of the lung clearance of ^9mTc-DTPA measured over 180 minutes was explored, and compared to kinetics in healthy smokers. The relationship between lung clearance and lung volumes, lung mechanics, arterial blood gases and disease activity assessed with serum angiotensin-converting enzyme and "'Ga scintigraphy was studied. Reduced lung volumes and compliance, increased resistance and decreased arterial oxygen tension were common. Vital i capacity (VC), and changes of VC at follow-up, corresponded to the slope of the static elastic pressure/volume curve, . and to the variation of it. -
Bronchodilators Accelerate the Dynamics of Muscle O2 Delivery
Chronic obstructive pulmonary disease Bronchodilators accelerate the dynamics of muscle Thorax: first published as 10.1136/thx.2009.120857 on 13 July 2010. Downloaded from O2 delivery and utilisation during exercise in COPD Danilo C Berton,1 Priscila B Barbosa,1 Luciana S Takara,1 Gaspar R Chiappa,1 Ana Cristina B Siqueira,1 Daniela M Bravo,1 Leonardo F Ferreira,2 J Alberto Neder1 See Editorial, p 573 ABSTRACT (QT), suggesting an important role for the central Background Expiratory flow limitation and lung cardiovascular adjustments in setting the limits of < Supplementary methods are hyperinflation promote cardiocirculatory perturbations increase in peripheral QO at the onset of exercise. published online only. To view 2 these files please visit the that might impair O2 delivery to locomotor muscles in Further experimental evidence for this contention journal online (http://thorax.bmj. patients with chronic obstructive pulmonary disease was obtained in a subsequent study in which com). (COPD). The hypothesis that decreases in lung hyperinflation a strategy aimed to reduce the resistive work of 1Pulmonary Function and Clinical after the inhalation of bronchodilators would improve breathing and dynamic hyperinflation (heliox) Exercise Physiology Unit skeletal muscle oxygenation during exercise was tested. simultaneously accelerated the dynamics of QTand (SEFICE), Division of Respiratory 5 Methods Twelve non- or mildly hypoxaemic males QO2 in patients with moderate to severe COPD. Diseases, Department of (forced expiratory volume in 1 s (FEV1)¼38.5612.9% -
The Pulmonary Manifestations of Left Heart Failure*
The Pulmonary Manifestations of Left Heart Failure* Brian K. Gehlbach, MD; and Eugene Geppert, MD Determining whether a patient’s symptoms are the result of heart or lung disease requires an understanding of the influence of pulmonary venous hypertension on lung function. Herein, we describe the effects of acute and chronic elevations of pulmonary venous pressure on the mechanical and gas-exchanging properties of the lung. The mechanisms responsible for various symptoms of congestive heart failure are described, and the significance of sleep-disordered breathing in patients with heart disease is considered. While the initial clinical evaluation of patients with dyspnea is imprecise, measurement of B-type natriuretic peptide levels may prove useful in this setting. (CHEST 2004; 125:669–682) Key words: Cheyne-Stokes respiration; congestive heart failure; differential diagnosis; dyspnea; pulmonary edema; respiratory function tests; sleep apnea syndromes Abbreviations: CHF ϭ congestive heart failure; CSR-CSA ϭ Cheyne-Stokes respiration with central sleep apnea; CPAP ϭ continuous positive airway pressure; Dlco ϭ diffusing capacity of the lung for carbon monoxide; DM ϭ membrane conductance; FRC ϭ functional residual capacity; OSA ϭ obstructive sleep apnea; TLC ϭ total lung ϭ ˙ ˙ ϭ capacity; VC capillary volume; Ve/Vco2 ventilatory equivalent for carbon dioxide early 5 million Americans have congestive heart For a detailed review of the pathophysiology of N failure (CHF), with 400,000 new cases diag- high-pressure pulmonary edema, the reader is re- nosed each year.1 Unfortunately, despite the consid- ferred to several excellent recent reviews.2–4 erable progress that has been made in understanding the pathophysiology of pulmonary edema, the pul- monary complications of this condition continue to The Pathophysiology of Pulmonary challenge the bedside clinician. -
Effect of Heliox Breathing on Flow Limitation in Chronic Heart Failure Patients
Eur Respir J 2009; 33: 1367–1373 DOI: 10.1183/09031936.00117508 CopyrightßERS Journals Ltd 2009 Effect of heliox breathing on flow limitation in chronic heart failure patients M. Pecchiari*, T. Anagnostakos#, E. D’Angelo*, C. Roussos#, S. Nanas# and A. Koutsoukou# ABSTRACT: Patients with chronic heart failure (CHF) exhibit orthopnoea and tidal expiratory flow AFFILIATIONS limitation in the supine position. It is not known whether the flow-limiting segment occurs in the *Istituto di Fisiologia Umana I, Universita` degli Studi di Milano, peripheral or central part of the tracheobronchial tree. The location of the flow-limiting segment Milan, Italy, and can be inferred from the effects of heliox (80% helium/20% oxygen) administration. If maximal #Dept of Critical Care and Pulmonary expiratory flow increases with this low-density mixture, the choke point should be located in the Services, Evangelismos General central airways, where the wave-speed mechanism dominates. If the choke point were located in Hospital, Medical School, University of Athens, Athens, Greece. the peripheral airways, where maximal flow is limited by a viscous mechanism, heliox should have no effect on flow limitation and dynamic hyperinflation. CORRESPONDENCE Tidal expiratory flow limitation, dynamic hyperinflation and breathing pattern were assessed in M. Pecchiari 14 stable CHF patients during air and heliox breathing at rest in the sitting and supine position. Istituto di Fisiologia Umana I via L. Mangiagalli 32 No patient was flow-limited in the sitting position. In the supine posture, eight patients exhibited 20133 Milan tidal expiratory flow limitation on air. Heliox had no effect on flow limitation and dynamic Italy hyperinflation and only minor effects on the breathing pattern. -
The Measurement of Pulmonary Diffusing Capacity for Carbon Monoxide by a Rebreathing Method
THE MEASUREMENT OF PULMONARY DIFFUSING CAPACITY FOR CARBON MONOXIDE BY A REBREATHING METHOD Benjamin M. Lewis, … , Ernest J. Hayford-Welsing, Erma Flaherty J Clin Invest. 1959;38(11):2073-2086. https://doi.org/10.1172/JCI103985. Research Article Find the latest version: https://jci.me/103985/pdf THE MEASUREMENT OF PULMONARY DIFFUSING CAPACITY FOR CARBON MONOXIDE BY A REBREATHING METHOD*t By BENJAMIN M. LEWIS, TAI-HON LIN, FRANCES E. NOET AND ERNEST J. HAYFORD-WELSING§ WITH THE TECHNICAL ASSISTANCE OF ERMA FLAHERTY (From the Pulmonary Function Laboratories, Departments of Medicine, Wayne State University College of Medicine, and City of Detroit Receiving Hospital, Detroit, Mich.) (Submitted for publication February 18, 1959; accepted June 19, 1959) The pulmonary diffusing capacity for oxygen capacity and one second vital capacity are first determined. is of great physiological and clinical sig- The analyzer circuit of the apparatus is then flushed with (DLo,) tank oxygen. A sealed bag containing a volume of 0.3 nificance (1). Its measurement, however, is rela- per cent CO and 10 per cent He in air (or in oxygen)2 tively complex (2). Pulmonary diffusing capac- equal to the subject's one second vital capacity is attached ity for carbon monoxide (DLco) which, it is to the three-way tap, the clamps on the bag are removed usually assumed,' can be converted to DL02 from and the bag and analyzer circuit mixed by the pump.3 the known solubilities and molecular weights of A bag-in-box device attached to a spirometer has been found convenient for filling the bag. -
Making the Diagnosis of Asthma
Making the Diagnosis of Asthma Meredith C McCormack MD MHS and Paul L Enright MD Introduction Signs and Symptoms Differential Diagnosis Diagnostic Testing Spirometry Bronchodilator Response Testing Inhalation Challenge Test Radioallergosorbent Test and Allergen Skin Test Exhaled Nitric Oxide Radiographic Imaging Asthma Versus Chronic Obstructive Pulmonary Disease Asthma Severity Summary: Take-Home Messages for the Respiratory Therapist Diagnostic tests can only increase or decrease the probability of the asthma diagnosis, so a thorough history is very important. In patients with asthma-like symptoms, spirometric evidence of airway obstruction plus a large bronchodilator response makes asthma much more likely. However, nor- mal spirometry is common in patients with mild asthma who are not symptomatic at the time of testing, and patients with poorly controlled asthma may lack substantial bronchodilator response. Inhalation challenge test often helps confirm asthma in patients with normal spirometry. Adult smokers with intermittent respiratory symptoms may have either asthma or chronic obstructive pulmonary disease (COPD). Normal post-bronchodilator spirometry rules out COPD. In patients with airway obstruction, a low diffusing capacity of the lung for carbon monoxide increases the probability of COPD and makes asthma much less likely. A high exhaled nitric oxide level makes allergic asthma more likely. Response to inhaled corticosteroids makes asthma more likely and COPD less likely. Key words: asthma, spirometry, methacholine, bronchodilator, pulmonary function, diagnosis. [Respir Care 2008;53(5):583–590. © 2008 Daedalus Enterprises] Meredith C McCormack MD MHS is affiliated with the Division of consulting on spirometry quality-assurance programs for a phase-3 clin- Pulmonary and Critical Care Medicine, Johns Hopkins School of Med- ical trial of varenicline for smoking cessation. -
Table of Contents 1
GENERAL THORACIC SURGERY DATABASE v.2.3 TRAINING MANUAL August 2017 Table of Contents 1. Demographics ................................................................................................................................................................. 2 2. Follow Up ........................................................................................................................................................................ 9 3. Admission ..................................................................................................................................................................... 10 4. Pre-Operative Evaluation ............................................................................................................................................. 14 5. Diagnosis (Category of Disease) ................................................................................................................................... 48 6. Procedure ..................................................................................................................................................................... 70 7. Post-Operative Events ................................................................................................................................................ 111 8. Discharge .................................................................................................................................................................... 135 9. Quality Measures ...................................................................................................................................................... -
Performance Characteristics in Idiopathic Pulmonary Fibrosis
ORIGINAL ARTICLE INTERSTITIAL LUNG DISEASE AND RADIOLOGY Quantitative high-resolution computed tomography fibrosis score: performance characteristics in idiopathic pulmonary fibrosis Stephen M. Humphries1, Jeffrey J. Swigris2, Kevin K. Brown2, Matthew Strand3, Qi Gong4, John S. Sundy4, Ganesh Raghu5, Marvin I. Schwarz6, Kevin R. Flaherty7, Rohit Sood8, Thomas G. O’Riordan4 and David A. Lynch1 Affiliations: 1Dept of Radiology, National Jewish Health, Denver, CO, USA. 2Division of Pulmonary and Critical Care Medicine, National Jewish Health, Denver, CO, USA. 3Division of Biostatistics and Bioinformatics, National Jewish Health, Denver, CO, USA. 4Gilead Sciences Inc., Foster City, CA, USA. 5Center for Interstitial Lung Diseases, Dept of Medicine, University of Washington, Seattle, WA, USA. 6Division of Pulmonary Sciences and Critical Care Medicine, University of Colorado, Aurora, CO, USA. 7Division of Pulmonary and Critical Care Medicine, University of Michigan, Ann Arbor, MI, USA. 8PAREXEL International, Billerica, MA, USA. Correspondence: Stephen M. Humphries, Quantitative Imaging Laboratory, Dept of Radiology, National Jewish Health, 1400 Jackson Street, Denver, CO 80206-2761, USA. E-mail: [email protected] @ERSpublications In subjects with IPF, quantification of lung fibrosis extent on HRCT using data-driven texture analysis shows acceptable performance characteristics and minimal clinically important difference in the range of 3.4–6.4% http://ow.ly/fFNc30lfAGh Cite this article as: Humphries SM, Swigris JJ, Brown KK, et al. Quantitative high-resolution computed tomography fibrosis score: performance characteristics in idiopathic pulmonary fibrosis. Eur Respir J 2018; 52: 1801384 [https://doi.org/10.1183/13993003.01384-2018]. ABSTRACT We evaluated performance characteristics and estimated the minimal clinically important difference (MCID) of data-driven texture analysis (DTA), a high-resolution computed tomography (HRCT)-derived measurement of lung fibrosis, in subjects with idiopathic pulmonary fibrosis (IPF). -
Respiratory Therapy Handbook
WASHINGTON STATE COMMUNITY COLLEGE RESPIRATORY THERAPY STUDENT HANDBOOK 2020-2021 Written: July, 1996 Revised: December, 2020 TABLE OF CONTENTS Statement of Non-Discrimination…………………………………………………………………………….. 2 Introduction……………………………………………………………………………………………………………..3 Goal………………………………………………………………………………………………………………………5-6 Accreditation……………………………………………………………………………………………………………6 Program Organization………………………………………………………………………………………………7 Plan for Consistency of Clinical Instruction & Evaluation of Clinical courses, Preceptors & Clinical Sites…………………………………………………………………………………………………………9 STUDENT POLICIES Course of Study………………………………………………………………………………………………………11 Student Schedule/Class sessions……………………………………………………………………………..11 Clinical Experiences…………………………………………………………………………………………..11-12 Tardiness……………………………………………………………………………………………………………….12 Absenteeism, Clinical…………………………………………………………………………………………12-13 Absenteeism, Classroom………………………………………………………………………………………….13 Absenteeism, Lab……………………………………………………………………………………………….13-14 Clinical Evaluations……………………………………………………………………………………………15-16 Clinical Competency…………………………………………………………………………………………..17-20 ACADEMIC POLICIES Clinical Evaluation Forms…………………………………………………………………………………..15-16 Clinical Competency Evaluation………………………………………………………………………….17-20 Promotion………………………………………………………………………………………………………………21 Evaluation………………………………………………………………………………………………………………21 Remediation …………………………………………………………………………………………………………..22 Probation/Dismissal……………………………………………………………………………………………….22 Leave of Absence…………………………………………………………………………………………………….22 -
Respiratory Therapy (RTH) 1
Respiratory Therapy (RTH) 1 RTH-205. Cardiopulmonary Pathophysiology. 2 Credits. Respiratory Therapy LECT 2 hrs An overview of the pathophysiology of diseases of the (RTH) cardiopulmonary system with an emphasis on pathophysiologic processes such as hypoxemia, hypoventilation, diffusion defects and ventilation perfusion mismatch; a survey of diseases encountered Courses by the respiratory therapist, including pathophysiology, diagnostic RTH-199. Respiratory Therapeutics. 5 Credits. methods and findings, clinical manifestations, treatment and LECT 4 hrs, LAB 3 hrs prognosis. An introduction to respiratory care, including history of the Prerequisites: RTH-203 and permission of program director. profession, ethical and legal responsibilities of the respiratory RTH-206. Mechanical Ventilation. 4 Credits. therapist; medical terminology, basic respiratory care procedures LECT 3 hrs, LAB 3 hrs including the physics, physiology and administration of medical Techniques of airway management and the provision of mechanical gas therapy, basic patient communication and assessment skills. ventilation; includes types of airways and appropriate uses; the Basic respiratory care procedures, humidity and aerosol therapy, physics and physiology of mechanical ventilation; classification hyperinflation therapy, chest physiotherapy and bronchial hygiene; of mechanical ventilators; indications for clinical application an overview of microbiology as applied to respiratory care; infection and complications of mechanical ventilation; management control; and equipment sterilization procedures. Course requires and monitoring of the patient requiring ventilatory support; and that students have completed the pre-professional phase of the appropriate methods of withdrawing ventilatory support. Respiratory Therapy program and have permission of the program Prerequisites: RTH-199, RTH-202, RTH-203, RTH-210 and director to enroll. permission of program director Prerequisites: Permission of Program Director Corequisites: RTH-204, RTH-205 and RTH-211 Additional Fees: Course fee applies. -
ICD-9-CM Procedures (FY10)
2 PREFACE This sixth edition of the International Classification of Diseases, 9th Revision, Clinical Modification (ICD-9-CM) is being published by the United States Government in recognition of its responsibility to promulgate this classification throughout the United States for morbidity coding. The International Classification of Diseases, 9th Revision, published by the World Health Organization (WHO) is the foundation of the ICD-9-CM and continues to be the classification employed in cause-of-death coding in the United States. The ICD-9-CM is completely comparable with the ICD-9. The WHO Collaborating Center for Classification of Diseases in North America serves as liaison between the international obligations for comparable classifications and the national health data needs of the United States. The ICD-9-CM is recommended for use in all clinical settings but is required for reporting diagnoses and diseases to all U.S. Public Health Service and the Centers for Medicare & Medicaid Services (formerly the Health Care Financing Administration) programs. Guidance in the use of this classification can be found in the section "Guidance in the Use of ICD-9-CM." ICD-9-CM extensions, interpretations, modifications, addenda, or errata other than those approved by the U.S. Public Health Service and the Centers for Medicare & Medicaid Services are not to be considered official and should not be utilized. Continuous maintenance of the ICD-9- CM is the responsibility of the Federal Government. However, because the ICD-9-CM represents the best in contemporary thinking of clinicians, nosologists, epidemiologists, and statisticians from both public and private sectors, no future modifications will be considered without extensive advice from the appropriate representatives of all major users.