Contraindications for Massage Therapy
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
Byoung Chan Kang, MD, Da Jeong Nam, MD*, Eun Kyoung Ahn, MD*, Duck Mi Yoon, MD, and Joung Goo Cho, MD*
Korean J Pain 2013 July; Vol. 26, No. 3: 299-302 pISSN 2005-9159 eISSN 2093-0569 http://dx.doi.org/10.3344/kjp.2013.26.3.299 |Case Report| Secondary Erythromelalgia - A Case Report - Department of Anesthesiology and Pain Medicine, Yonsei University College of Medicine, Seoul, *National Health Insurance Service Ilsan Hospital, Goyang, Korea Byoung Chan Kang, MD, Da Jeong Nam, MD*, Eun Kyoung Ahn, MD*, Duck Mi Yoon, MD, and Joung Goo Cho, MD* Erythromelalgia is a rare neurovascular pain syndrome characterized by a triad of redness, increased temperature, and burning pain primarily in the extremities. Erythromelalgia can present as a primary or secondary form, and secondary erythromelalgia associated with a myeloproliferative disease such as essential thrombocythemia often responds dramatically to aspirin therapy, as in the present case. Herein, we describe a typical case of a 48-year-old woman with secondary erythromelalgia linked to essential thrombocythemia in the unilateral hand. As this case demonstrates, detecting and visualizing the hyperthermal area through infrared thermography of an erythromelalgic patient can assist in diagnosing the patient, assessing the therapeutic results, and understanding the disease course of erythromelalgia. (Korean J Pain 2013; 26: 299-302) Key Words: aspirin, erythromelalgia, infrared thermography, neuropathic pain. Erythromelalgia is a rare clinical syndrome charac- Adults are more commonly involved than children and are terized by a triad of redness, increased temperature, and more likely to have the secondary form. Secondary eryth- burning pain primarily in the extremities. The term eryth- romelalgia is usually associated with myeloproliferative romelalgia, derived from the Greek words for redness and disorders such as essential thrombocythemia (ET) and pol- pain in the extremities, was coined in 1878 by Mitchell [1]. -
Venous Symposium: Overview
5/30/2017 1 5/30/2017 VENOUS SYMPOSIUM: OVERVIEW Robert W. Vorhies, M.D., F.A.C.S. Vascular and Endovascular Surgery Endovenous Therapy and Vein Aesthetics Ferrell-Duncan Clinic, Cox Health Systems WHY DO WE CARE? • Epidemiology • Disability • Historical experience • Opportunity 2 5/30/2017 EPIDEMIOLOGY QUESTION #1 Which state has nearly the same population as all the people in the US with venous disease? • A. New York • B. Florida • C. California • D. Missouri EPIDEMIOLOGY QUESTION #1 Which state has nearly the population as all the people in the US with venous disease? • A. New York • B. Florida • C. California • More than 11 million men and 22 million women between the ages of 40 and 80 years in the United States have varicose veins. • Prevalence of 20% (range, 21.8%-29.4%) • D. Missouri Peter Gloviczki, Anthony J. Comerota, Michael C. Dalsing, Bo G. Eklof, David L. Gillespie, Monika L. Gloviczki, Joann M. Lohr, Robert B. McLafferty, Mark H. Meissner, M. Hassan Murad, Frank T. Padberg, Peter J. Pappas, Marc A. Passman Joseph D. Raffetto, Michael A. Vasquez, and Thomas W. Wakefield. “The care of patients with varicose veins and associated chronic venous diseases: Clinical practice guidelines of the Society for Vascular Surgery and the American Venous Forum.” J Vasc Surg . 2011;53:2S-48S. 3 5/30/2017 EPIDEMIOLOGY QUESTION #2 • Which mid western metropolitan area has about the same number of people as those with severe chronic venous insufficiency including ulcers and skin changes? • A. Minneapolis • B. Chicago • C. St. Louis • D. Kansas City. EPIDEMIOLOGY QUESTION #2 • Which mid western city has about the same number of people as those with severe chronic venous insufficiency including ulcers and skin changes? • A. -
Skin Injuries – Can We Determine Timing and Mechanism?
Skin injuries – can we determine timing and mechanism? Jo Tully VFPMS Seminar 2016 What skin injuries do we need to consider? • Bruising • Commonest accidental and inflicted skin injury • Basic principles that can be applied when formulating opinion • Abrasions • Lacerations }we need to be able to tell the difference • Incisions • Stabs/chops • Bite marks – animal v human / inflicted v ‘accidental’ v self-inflicted Our role…. We are often/usually/always asked…………….. • “What type of injury is it?” • “When did this injury occur?” • “How did this injury occur?” • “Was this injury inflicted or accidental?” • IS THIS CHILD ABUSE? • To be able to answer these questions (if we can) we need knowledge of • Anatomy/physiology/healing - injury interpretation • Forces • Mechanisms in relation to development, plausibility • Current evidence Bruising – can we really tell which bruises are caused by abuse? Definitions – bruising • BLUNT FORCE TRAUMA • Bruise =bleeding beneath intact skin due to BFT • Contusion = bruise in deeper tissues • Haematoma - extravasated blood filling a cavity (or potential space). Usually associated with swelling • Petechiae =Pinpoint sized (0.1-2mm) hemorrhages into the skin due to acute rise in venous pressure • medical causes • direct forces • indirect forces Medical Direct Indirect causes mechanical mechanical forces forces Factors affecting development and appearance of a bruise • Properties of impacting object or surface • Force of impact • Duration of impact • Site - properties of body region impacted (blood supply, -
Immune Thrombocytopenia Purpura (ITP)
Immune Thrombocytopenia Purpura (ITP) Information for patients and carers from the Haematology Department What is ITP? Immune thrombocytopenic purpura (ITP) is a condition which causes the number of platelets in Spleen your blood to be reduced. Platelets are cells that help blood to clot and they help to prevent bleeding and bruising after an injury. If you do not have enough platelets in your blood, you are likely to bruise easily or may be unable to stop bleeding if you cut yourself. In ITP, your body’s immune system destroys your own platelets. White blood cells in your blood and your spleen (an organ in your abdomen) are part of your immune system. One of their actions is to produce antibodies which help your body to fight Diagram showing the position of infections. If you develop ITP, your immune system the spleen becomes overactive and produces antibodies that cause your platelets to be destroyed in the spleen; this results in a low platelet count. ITP is a type of autoimmune condition (which means your immune system is acting against your body rather than for it). ITP in adults is more common in women than men. It is very different from ITP in children, who usually get ITP after a viral infection but who recover without any treatment. ITP in adults normally needs treatment. Some people with ITP have other autoimmune conditions, such as rheumatoid arthritis, or infections such as hepatitis or HIV. If you have any of these medical issues, your ITP may be treated slightly differently. 1 of 8 ITP (August 2021) A normal platelet count is between 150 and 400 thousand million platelets per litre of blood. -
Hemorrhoids Information, Pictures, Treatments, and Cures by Rick Shacket DO, 1989 ©
Hemorrhoids Information, Pictures, Treatments, and Cures By Rick Shacket DO, 1989 © Hemorrhoids are cushions of tissue and varicose veins located in and around the rectal area. When they become inflamed, hemorrhoids can itch, bleed, and cause pain. Unfortunately a hemorrhoidal condition only tends to get worse over the years. That is why safe, gentle, and effective treatment for hemorrhoids is recommended as soon as they occur. Hemorrhoids bother about 89% of all Americans at some time in their lives. Hemorrhoids caused Napoleon to sit side-saddle, sent President Jimmy Carter to the operating room, and benched baseball star George Brett during the 1980 World Series. Over two thirds of all healthy people reporting for physical examinations have hemorrhoids. For more information about Hemorrhoids visit the links below: • Pictures: Hemorrhoids and Anal Fissure • Stapled Hemorrhoidopexy (PPH Procedure) • What Are Hemorrhoids? • Harmonic Scalpel Hemorrhoid surgery • What Are the Symptoms of Hemorrhoids? • Laser Surgery for Hemorrhoids • How Common Are Hemorrhoids? • Atomizing Hemorrhoids • How Are Hemorrhoids Diagnosed? • Complications of Hemorrhoid Surgery • What Is the Treatment? • Knowing What to Ask Your Surgeon • How Are Hemorrhoids Prevented? • Allopathic Hemorrhoid Medications • Painless Treatment of Hemorrhoids • Herbal Hemorrhoid Medications • HAL-RAR Method Hemorrhoidectomy • Homeopathic Hemorrhoid Medications • Hemorrhoids Grades 1 to 4 • References • Surgical Classification of Hemorrhoids • Video References • Traditional Surgery for Hemorrhoids Pictures: Hemorrhoids and Anal Fissure Internal hemorrhoids occur higher up in the anal canal, out of sight. Bleeding is the most common symptom of internal hemorrhoids, and often the only one in mild cases. View hemorrhoid gallery for detailed photos. Hemorrhoids Information, Pictures, Treatments, and Cures Page 1 of 21 External hemorrhoids are visible-occurring out side the anus. -
Bleeds and Bruises in Children with Haemophilia
Bleeds and Bruises in CHildren WiTH HaeMOPHilia MusCle ANd/or JoiNt Bleeds Call the parent/guardian P.r.i.C.e. siGNs oF A serious HeAd Bleed P : Protection * Headache. Lower Limb: Take weight off the joint or muscle * drowsiness. Upper Limb: No carrying using affected arm * Nausea. r : rest * Vomiting. • Rest means rest! * unsteady Balance. • Try not to allow use of the joint or muscle where * irritability. possible. * Confusion. * seizures. i : ice * loss of consciousness. • Regular ice packs can help with pain & reduce swelling. • Put an ice pack over the affected area for 20 minutes. Repeat every two hours. DO NOT leave the ice pack on for more than 20 minutes siGNs oF A soFt tissue DO NOT place ice pack directly on skin (Use a tea Bleed towel/cold pack cover) * Bruising, discolouring of skin. C : Compression * Mild swelling. • Use an elasticated bandage to compress the affected area to reduce swelling. e : elevation • Elevate the affected limb to help reduce swelling. siGNs oF AN ABdoMiNAl • Keep the affected joint or muscle above the level of the Bleed heart. * Bloody, black or tar-like First Aid bowel motions. * red or brown urine. Mouth & Gum Bleeds * Pain. These can be hard to control because clots that form are * Vomiting of blood (blood washed away by saliva or knocked off by the tongue or food. Try giving the child an ice cube or ice pop to suck. may be red or black). These bleeds may need treatment by parents or the treatment centre. Nosebleeds siGNs oF BleediNG iNto tHe Tilt head forward and pinch the bridge of the nose below the bone for 10 - 20 minutes and / or put an ice-pack on JoiNts or MusCles the bridge of the nose for not more than 5 minutes. -
Isolated Plantar Vein Thrombosis Resembling a Corn with a Bruise
JE Hahm, et al pISSN 1013-9087ㆍeISSN 2005-3894 Ann Dermatol Vol. 31, No. 1, 2019 https://doi.org/10.5021/ad.2019.31.1.66 CASE REPORT Isolated Plantar Vein Thrombosis Resembling a Corn with a Bruise Ji Eun Hahm, Kang Su Kim, Jae Won Ha, Chul Woo Kim, Sang Seok Kim Department of Dermatology, Kangdong Sacred Heart Hospital, College of Medicine, Hallym University, Seoul, Korea Plantar vein thrombosis, rarely-reported disease, is usually or callus, plantar fibromatosis, or plantar verruca1. Among accompanied by pain and tenderness in the plantar region laborers, they may develop from excess pressure on the and should be differentiated from other dermatological con- bony prominences of the feet, repetitive uneven friction ditions causing plantar pain, such as hemorrhagic corn/cal- from footwear, or gait abnormalities. Plantar vein throm- lus, plantar epidermal cyst, verruca, or plantar fibromatosis. bosis is a rare condition causing plantar pain. The exact A 52-year-old man presented with a violaceous tender sub- cause of plantar vein thrombosis is yet unclear, but predis- cutaneous nodule overlying a hyperkeratotic plaque on his posing conditions, such as prior trauma, surgery, paraneo- sole. Initially, he thought it was a corn and applied keratolytic plastic syndromes, or coagulation disorders have been agents, which failed to work. Sonography revealed a well-de- described. To date, there is no established treatment ex- marcated mass with increased peripheral vascularity. His cept surgical excision, but reportedly, nonsteroidal anti-in- pain was relieved after a complete wide excision, which con- flammatory drug or heparin with elastic bandage is known firmed the mass to be plantar vein thrombosis after histo- to be effective for symptomatic control2-5. -
Portal Hypertensionand Its Radiological Investigation
Postgrad Med J: first published as 10.1136/pgmj.39.451.299 on 1 May 1963. Downloaded from POSTGRAD. MED. J. (I963), 39, 299 PORTAL HYPERTENSION AND ITS RADIOLOGICAL INVESTIGATION J. H. MIDDLEMISS, M.D., F.F.R., D.M.R.D. F. G. M. Ross, M.B., B.Ch., B.A.O., F.F.R., D.M.R.D. From the Department of Radiodiagnosis, United Bristol Hospitals PORTAL hypertension is a condition in which there branch of the portal vein but may drain into the right is an blood in the branch. abnormally high pressure Small veins which are present on the serosal surface portal system of veins which eventually leads to of the liver and in the surrounding peritoneal folds splenomegaly and in chronic cases, to haematem- draining the diaphragm and stomach are known as esis and melaena. accessory portal veins. They may unite with the portal The circulation is in that it vein or enter the liver independently. portal unique The hepatic artery arises normally from the coeliac exists between two sets of capillaries, i.e. the axis but it may arise as a separate trunk from the aorta. capillaries of the spleen, pancreas, gall-bladder It runs upwards and to the right and divides into a and most of the gastro-intestinal tract on the left and right branch before entering the liver at the one hand and the sinusoids of the liver on the porta hepatis. The venous return starts as small thin-walled branches other hand. The liver parallels the lungs in that in the centre of the lobules in the liver. -
Understanding Haemophilia
Understanding haemophilia Understanding haemophilia Contents Introduction 3 Haemophilia and your child 4 What is haemophilia? 5 What causes haemophilia? 5 Can females have haemophilia? 6 Carriers 8 Who is affected by haemophilia? 9 How severe is haemophilia? 9 Signs and symptoms of haemophilia 11 How is haemophilia diagnosed? 14 Diagnosis 14 Treatment 16 Port-a-cath 19 Managing joint bleeds with PRICE 19 Gene therapy 21 Possible complications of haemophilia 22 Inhibitors 22 Joint damage 22 Medical and dental treatment 23 Surgery Circumcision Dental care Medicines Vaccinations Bleeding disorder card Living with haemophilia 26 Sport and exercise 27 School, college and work 28 Travel 29 Pregnancy and haemophilia 30 Glossary of terms 32 About The Haemophilia Society 33 2 Understanding haemophilia Introduction This booklet is about haemophilia A and B. It gives a general overview of haemophilia and information on diagnosing, treating and living with the condition that we hope will answer your main questions. It has been written for people directly affected by haemophilia and for anyone interested in learning about haemophilia. If you are a parent and your child has recently been diagnosed with haemophilia you may be feeling quite overwhelmed. Remember, you’re not alone and many families are facing the same concerns and issues. Please do get in touch – we have lots of support and information available as well as services for parents and children. You can find out more via our website or Facebook pages, by emailing [email protected] or calling us on 020 7939 0780. The outlook is now the best it has ever been for people with haemophilia in the UK. -
Reprint Of: Why Are Hemorrhoids Symptomatic? the Pathophysiology and Etiology of Hemorrhoids
Seminars in Colon and Rectal Surgery 29 (2018) 160 166 À Contents lists available at ScienceDirect Seminars in Colon and Rectal Surgery journal homepage: www.elsevier.com/locate/yscrs Reprint of: Why are hemorrhoids symptomatic? the pathophysiology and etiology of hemorrhoids WilliamD1X X C. Cirocco, MD,D2X X FACS, FASCRS* Department of Surgery, University of Missouri Kansas City, Kansas City, Missouri. À ABSTRACT Hemorrhoids are a normal component of the anorectum and contribute to the mechanism of anal closure, thus providing fine adjustment of anal continence. There are numerous myths and legends associated with the disordered and diseased state of hemorrhoids. Fortunately, information obtained from modern technolo- gies including microscopic histopathology defined first the actual substance and makeup of hemorrhoids and was later combined with anorectal physiology to provide evidence establishing the underlying pathophysiol- ogy of this universal finding of the human anorectum. The sliding anal canal theory of Gass and Adams has held up and is further supported by other anatomic studies including the work of WHF Thomson, who popu- larized the term “cushions” to describe the complex intertwining of muscle, connective tissue, veins, arteries, and arteriovenous communications which constitute hemorrhoids. A loss of muscle mass in favor of connec- tive tissue over time helps explain the role of aging as a predisposing factor for symptomatic hemorrhoids. Other factors include the modern “rich” or low-residue diet leading to constipation and straining which con- tributes to prolapsing cushions. Pathologic studies also demonstrated arteriovenous communications explain- ing why hemorrhoid bleeding is typically bright red or arterial in nature as opposed to dark or venous bleeding. -
Treatment Strategies for Patients with Lower Extremity Chronic Venous Disease (LECVD)
Evidence-based Practice Center Systematic Review Protocol Project Title: Treatment Strategies for Patients with Lower Extremity Chronic Venous Disease (LECVD) Project ID: DVTT0515 Initial publication date if applicable: March 7, 2016 Amendment Date(s) if applicable: May 6th, 2016 (Amendments Details–see Section VII) I. Background for the Systematic Review Lower extremity chronic venous disease (LECVD) is a heterogeneous term that encompasses a variety of conditions that are typically classified based on the CEAP classification, which defines LECVD based on Clinical, Etiologic, Anatomic, and Pathophysiologic parameters. This review will focus on treatment strategies for patients with LECVD, which will be defined as patients who have had signs or symptoms of LE venous disease for at least 3 months. Patients with LECVD can be asymptomatic or symptomatic, and they can exhibit a myriad of signs including varicose veins, telangiectasias, LE edema, skin changes, and/or ulceration. The etiology of chronic venous disease includes venous dilation, venous reflux, (venous) valvular incompetence, mechanical compression (e.g., May-Thurner syndrome), and post-thrombotic syndrome. Because severity of disease and treatment are influenced by anatomic segment, LECVD is also categorized by anatomy (iliofemoral vs. infrainguinal veins) and type of veins (superficial veins, perforating veins, and deep veins). Finally, the pathophysiology of LECVD is designated typically as due to the presence of venous reflux, thrombosis, and/or obstruction. LECVD is common -
EFFIENT Safely and Effectively
1 HIGHLIGHTS OF PRESCRIBING INFORMATION • Patients with unstable angina or, non-ST-elevation myocardial These highlights do not include all the information needed to use infarction (NSTEMI) (1.1). EFFIENT safely and effectively. See full prescribing information • Patients with ST-elevation myocardial infarction (STEMI) when for EFFIENT. managed with either primary or delayed PCI (1.1). EFFIENT (prasugrel) tablets, for oral use ------------------------ DOSAGE AND ADMINISTRATION ----------------------- Initial U.S. Approval: 2009 • Initiate treatment with a single 60-mg oral loading dose (2). • Continue at 10-mg once daily with or without food. Consider 5-mg WARNING: BLEEDING RISK once daily for patients <60 kg (2). See full prescribing information for complete boxed warning. • Patients should also take aspirin (75-mg to 325-mg) daily (2). • Effient can cause significant, sometimes fatal, bleeding (5.1, ---------------------- DOSAGE FORMS AND STRENGTHS --------------------- 5.2, 6.1). • Do not use Effient in patients with active pathological bleeding 5-mg and 10-mg tablets (3) or a history of transient ischemic attack or stroke (4.1, 4.2). ------------------------------- CONTRAINDICATIONS ------------------------------ • In patients ≥75 years of age, Effient is generally not • Active pathological bleeding (4.1) recommended, except in high-risk patients (diabetes or prior • Prior transient ischemic attack or stroke (4.2) MI), where its use may be considered (8.5). • Hypersensitivity to prasugrel or any component of the product • Do not start Effient in patients likely to undergo urgent (4.3) coronary artery bypass graft surgery (CABG). When possible, discontinue Effient at least 7 days prior to any surgery (5.2). ------------------------ WARNINGS AND PRECAUTIONS ----------------------- • Additional risk factors for bleeding include: body weight • CABG-related bleeding: Risk increases in patients receiving <60 kg; propensity to bleed; concomitant use of medications Effient who undergo CABG (5.2).