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Management of Dental Trauma in a Primary Care Setting Abstract
Guidance for the Clinician in Rendering Pediatric Care CLINICAL REPORT Management of Dental Trauma in a Primary Care Setting Martha Ann Keels, DDS, PhD, and THE SECTION ON ORAL abstract HEALTH The American Academy of Pediatrics and its Section on Oral Health have KEY WORDS developed this clinical report for pediatricians and primary care physi- dental trauma, dental injury, tooth, teeth, dentist, pediatrician cians regarding the diagnosis, evaluation, and management of dental ABBREVIATION trauma in children aged 1 to 21 years. This report was developed CT—computed tomography through a comprehensive search and analysis of the medical and den- This document is copyrighted and is property of the American tal literature and expert consensus. Guidelines published and updated Academy of Pediatrics and its Board of Directors. All authors have filed conflict of interest statements with the American by the International Association of Dental Traumatology (www.dental- Academy of Pediatrics. Any conflicts have been resolved through traumaguide.com) are an excellent resource for both dental and non- a process approved by the Board of Directors. The American dental health care providers. Pediatrics 2014;133:e466–e476 Academy of Pediatrics has neither solicited nor accepted any commercial involvement in the development of the content of this publication. The guidance in this report does not indicate an exclusive INTRODUCTION course of treatment or serve as a standard of medical care. Variations, taking into account individual circumstances, may be By 14 years of age, 30% of children have experienced a dental injury.1 appropriate. Many of these children are taken directly to their medical home, an urgent care center, or an emergency department for evaluation and treatment. -
Journal 2017
Journal of ENT masterclass ISSN 2047-959X Journal of ENT MASTERCLASS® Year Book 2017 Volume 10 Number 1 YEAR BOOK 2017 VOLUME 10 NUMBER 1 JOURNAL OF ENT MASTERCLASS® Volume 10 Issue 1 December 2017 Contents Free Courses for Trainees, Consultants, SAS grades, GPs & Nurses Welcome Message 3 CALENDER OF FREE RESOURCES 2018-19 Hesham Saleh Increased seats for specialist registrars & exam candidates ENT aspects of cystic fibrosis management 4 Gary J Connett ® 15th Annual International ENT Masterclass Paediatric swallowing disorders 8 Venue: Doncaster Royal Infirmary, 25-27th January 2019 Hayley Herbert and Shyan Vijayasekaran Special viva sessions for exam candidates Paediatric tongue-tie 14 Steven Frampton, Ciba Paul, Andrea Burgess and Hasnaa Ismail-Koch rd ® 3 ENT Masterclass China Paediatric oesophageal foreign bodies 20 Beijing, China, 12-13th May 2018 Emily Lowe, Jessica Chapman, Ori Ron and Michael Stanton Biofilms in paediatric otorhinolaryngology 26 3rd ENT Masterclass® Europe S Goldie, H Ismail-Koch, P.G. Harries and R J Salib Berlin, Germany, 14-15th Sept 2018 Intracranial complications of ear, nose and throat infections in childhood 34 Alice Lording, Sanjay Patel and Andrea Whitney ® ENT Masterclass Switzerland The superior canal dehiscence syndrome 41 Lausanne, 5-6th Oct 2018 Simon Richard Mackenzie Freeman Tympanosclerosis 46 ® ENT Masterclass Sri Lanka Priya Achar and Harry Powell Colombo, 16-17th Nov 2018 Endoscopic ear surgery 49 Carolina Wuesthoff, Nicholas Jufas and Nirmal Patel o Limited places, on first come basis. Early applications advised. o Masterclass lectures, Panel discussions, Clinical Grand Rounds Vestibular function testing 57 o Oncology, Plastics, Pathology, Radiology, Audiology, Medico-legal Karen Lindley and Charlie Huins Auditory brainstem implantation 63 Website: www.entmasterclass.com Harry R F Powell and Shakeel S Saeed CYBER TEXTBOOK on operative surgery, Journal of ENT Masterclass®, Surgical management of temporal bone meningo-encephalocoele and CSF leaks 69 Application forms Mr. -
Pathophysiological Mechanisms of Root Resorption After Dental Trauma: a Systematic Scoping Review Kerstin M
Galler et al. BMC Oral Health (2021) 21:163 https://doi.org/10.1186/s12903-021-01510-6 RESEARCH ARTICLE Open Access Pathophysiological mechanisms of root resorption after dental trauma: a systematic scoping review Kerstin M. Galler1*, Eva‑Maria Grätz1, Matthias Widbiller1 , Wolfgang Buchalla1 and Helge Knüttel2 Abstract Background: The objective of this scoping review was to systematically explore the current knowledge of cellular and molecular processes that drive and control trauma‑associated root resorption, to identify research gaps and to provide a basis for improved prevention and therapy. Methods: Four major bibliographic databases were searched according to the research question up to Febru‑ ary 2021 and supplemented manually. Reports on physiologic, histologic, anatomic and clinical aspects of root resorption following dental trauma were included. Duplicates were removed, the collected material was screened by title/abstract and assessed for eligibility based on the full text. Relevant aspects were extracted, organized and summarized. Results: 846 papers were identifed as relevant for a qualitative summary. Consideration of pathophysiological mechanisms concerning trauma‑related root resorption in the literature is sparse. Whereas some forms of resorption have been explored thoroughly, the etiology of others, particularly invasive cervical resorption, is still under debate, resulting in inadequate diagnostics and heterogeneous clinical recommendations. Efective therapies for progres‑ sive replacement resorptions have not been established. Whereas the discovery of the RANKL/RANK/OPG system is essential to our understanding of resorptive processes, many questions regarding the functional regulation of osteo‑/ odontoclasts remain unanswered. Conclusions: This scoping review provides an overview of existing evidence, but also identifes knowledge gaps that need to be addressed by continued laboratory and clinical research. -
Pulp Canal Obliteration After Traumatic Injuries in Permanent Teeth – Scientific Fact Or Fiction?
CRITICAL REVIEW Endodontic Therapy Pulp canal obliteration after traumatic injuries in permanent teeth – scientific fact or fiction? Juliana Vilela BASTOS(a) Abstract: Pulp canal obliteration (PCO) is a frequent finding associated (b) Maria Ilma de Souza CÔRTES with pulpal revascularization after luxation injuries of young permanent teeth. The underlying mechanisms of PCO are still unclear, (a) Universidade Federal de Minas Gerais - and no experimental scientific evidence is available, except the results UFMG, School of Dentistry, Department of Restorative Dentistry, Belo Horizonte, MG, of a single histopathological study. The lack of sound knowledge Brazil. concerning this process gives rise to controversies, including the (b) Pontifícia Universidade Católica de Minas most suitable denomination. More than a mere semantic question, Gerais – PUC-MG, Department of Dentistry, the denomination is an important issue, because it reflects the nature Belo Horizonte, MG, Brazil. of this process, and directly impacts the treatment plan decision. The hypothesis that accelerated dentin deposition is related to the loss of neural control over odontoblastic secretory activity is well accepted, but demands further supportive studies. PCO is seen radiographically as a rapid narrowing of pulp canal space, whereas common clinical features are yellow crown discoloration and a lower or non-response to sensibility tests. Late development of pulp necrosis and periapical disease are rare complications after PCO, rendering prophylactic endodontic intervention -
Pulp Canal Obliteration- a Daunting Clinical Challenge
IOSR Journal of Dental and Medical Sciences (IOSR-JDMS) e-ISSN: 2279-0853, p-ISSN: 2279-0861.Volume 19, Issue 3 Ser.16 (March. 2020), PP 56-60 www.iosrjournals.org Pulp Canal Obliteration- A Daunting Clinical Challenge Ashish Jain1, P Shanti Priya2, Ronald Tejpaul3, Basa Srinivas Karteek4 1(Reader, Department of Conservative dentistry and Endodontics, PanineeyaInstitute of Dental Sciences, India) 2(Senior lecturer, Department of Conservative dentistry and Endodontics, Panineeya Institute of Dental Sciences, India) 3 (Senior lecturer, Department of Conservative dentistry and Endodontics, Oxford Dental College, India) 4(Senior lecturer, Department of Conservative dentistry and Endodontics, Panineeya Institute of Dental Sciences, India) Abstract: Traumatic injuries of primary or permanent dentition can lead to certain clinical complications and its management presents a considerable challenge for apractitioner. Pulp canal obliteration (PCO) also called as calcific metamorphosis (CM) following dental trauma has been stated to be approximately 37 - 40% and commonly noticed after luxation injuries. The diagnostic status and treatment planning decision regarding PCO always remains controversial. The decision on when to start treatment of such cases, whether on early detection of PCO or to wait until detection of signs and symptoms of pulp necrosis, always remains a clinical dilemma. Following calcific metamorphosis, scouting for the canals and negotiating it to full working length may lead to iatrogenic errors such as perforations or instrument separation. This article emphasizes on protocols to be followed on diagnosing and treating PCO and challenges that are to be encountered while managing these cases.Thorough knowledge of tooth morphology, skill, patience, use of appropriate instruments and materials are essential to successfully manage such cases. -
Guideline on Management of Acute Dental Trauma
rEfErence manual v 32 / No 6 10 / 11 Guideline on Management of Acute Dental Trauma Originating Council Council on Clinical Affairs Review Council Council on Clinical Affairs Adopted 2001 Revised 2004, 2007, 2010 Purpose violence, and sports.7-10 All sporting activities have an asso- The American Academy of Pediatric Dentistry (AAPD) ciated risk of orofacial injuries due to falls, collisions, and intends these guidelines to define, describe appearances, and contact with hard surfaces.11 The AAPD encourages the use set forth objectives for general management of acute trau- of protective gear, including mouthguards, which help dis- matic dental injuries rather than recommend specific treat- tribute forces of impact, thereby reducing the risk of severe ment procedures that have been presented in considerably injury.13,14 more detail in text-books and the dental/medical literature. Dental injuries could have improved outcomes if the public were aware of first-aid measures and the need to seek Methods immediate treatment.14-17 Because optimal treatment results This guideline is an update of the previous document re- follow immediate assessment and care,18 dentists have an vised in 2007. It is based on a review of the current dental ethical obligation to ensure that reasonable arrangements and medical literature related to dental trauma. An elec- for emergency dental care are available.19 The history, cir- tronic search was conducted using the following parameters: cumstances of the injury, pattern of trauma, and behavior Terms: “teeth”, “trauma”, “permanent teeth”, and “primary of the child and/or caregiver are important in distin- teeth”; Field: all fields; Limits: within the last 10 years; guishing nonabusive injuries from abuse.20 humans; English. -
Veterinary Dentist at Work (VDAW) Index Chronological March 1996 Through Sept 2020
Veterinary Dentist At Work (VDAW) Index Chronological March 1996 through Sept 2020 1996 Vol. 13 No. 1 • Impacted mandibular canine tooth in a dog • Compound odontoma of a maxillary fourth premolar tooth in a dog 1996 Vol. 13 No. 2 • Incisor teeth radiographs in horses • Subcoronal tooth fracture in a dog 1996 Vol. 13 No. 3 • Orthodontic treatment – care is needed during removal of appliances • CT scans of canine molar teeth and salivary glands. 1996 Vol. 13 No. 4 • Endodontic treatment of a sun bear 1997 Vol. 14 No. 1 • Occupational hazard – dermatitis caused by benzoyl peroxide in chemical cured acrylics. 1997 Vol. 14 No. 2 • Interdental wiring and stainless steel arch repair of avulsed mandibular and maxillary incisors in a racehorse. Paul Orsini 1997 Vol. 14 No. 3 • Anomalous extra canine tooth in a cat. Frank Verstraete 1997 Vol. 14 No. 4 • Complex odontoma with necrosis in a dog. Frank Verstraete 1998 Vol. 15 No. 1 • Dental anomaly in a horse – supernumerary cheek tooth. Zack Matzkin • Cat with malpositioned canine tooth tongue trapping. Andries Van Foreest • Dog with lingual papillomatosis. Jeffrey Rhody 1 1998 Vol. 15 No. 2 • Yorkshire terrier with severe periodontal disease. William Rosenblad • Alveolar osteitis post extraction mandibular canine in a cat. Suzy Aller 1998 Vol. 15 No. 3 • Root abscess in an aardvark. 1998 Vol. 15 No. 4 • Fabrication of a veterinary dental teaching model. Ronald Southerland 1999 Vol. 16 No. 1 • Fabrication of a labial maxillary arch bar. Peak, Lobprise, Wiggs • Labial avulsion repair in a cat and a dog. Zlatko Pavlica • TMJ luxation in a rabbit. -
Pediatric Orbital Fractures
Review Article 9 Pediatric Orbital Fractures Adam J. Oppenheimer, MD1 Laura A. Monson, MD1 Steven R. Buchman, MD1 1 Section of Plastic Surgery, Department of Surgery, University of Address for correspondence and reprint requests Steven R. Buchman, Michigan Hospitals, Ann Arbor, Michigan MD, Section of Plastic Surgery, Department of Surgery, University of Michigan Health System, 2130 Taubman Center, SPC 5340, Craniomaxillofac Trauma Reconstruction 2013;6:9–20 1500 E. Medical Center Drive, Ann Arbor, MI 48109-5340 (e-mail: [email protected]). Abstract It is wise to recall the dictum “children are not small adults” when managing pediatric Keywords orbital fractures. In a child, the craniofacial skeleton undergoes significant changes in ► orbit size, shape, and proportion as it grows into maturity. Accordingly, the craniomaxillo- ► pediatric facial surgeon must select an appropriate treatment strategy that considers both the ► trauma nature of the injury and the child’s stage of growth. The following review will discuss the ► enophthalmos management of pediatric orbital fractures, with an emphasis on clinically oriented ► entrapment anatomy and development. Pediatric orbital fractures occur in discreet patterns, based on 12 years of age. During mixed dentition, the cuspid teeth are the characteristic developmental anatomy of the craniofacial immediately beneath the orbit: hence the term eye tooth in skeleton at the time of injury. To fully understand pediatric dental parlance. It is not until age 12 that the maxillary sinus orbital trauma, the craniomaxillofacial surgeon must first be expands, in concert with eruption of the permanent denti- aware of the anatomical and developmental changes that tion. At 16 years of age, the maxillary sinus reaches adult size occur in the pediatric skull. -
The Impact of Sport Training on Oral Health in Athletes
dentistry journal Review The Impact of Sport Training on Oral Health in Athletes Domenico Tripodi, Alessia Cosi, Domenico Fulco and Simonetta D’Ercole * Department of Medical, Oral and Biotechnological Sciences, University “G. D’Annunzio” of Chieti-Pescara, Via dei Vestini 31, 66100 Chieti, Italy; [email protected] (D.T.); [email protected] (A.C.); [email protected] (D.F.) * Correspondence: [email protected] Abstract: Athletes’ oral health appears to be poor in numerous sport activities and different diseases can limit athletic skills, both during training and during competitions. Sport activities can be considered a risk factor, among athletes from different sports, for the onset of oral diseases, such as caries with an incidence between 15% and 70%, dental trauma 14–70%, dental erosion 36%, pericoronitis 5–39% and periodontal disease up to 15%. The numerous diseases are related to the variations that involve the ecological factors of the oral cavity such as salivary pH, flow rate, buffering capability, total bacterial count, cariogenic bacterial load and values of secretory Immunoglobulin A. The decrease in the production of S-IgA and the association with an important intraoral growth of pathogenic bacteria leads us to consider the training an “open window” for exposure to oral cavity diseases. Sports dentistry focuses attention on the prevention and treatment of oral pathologies and injuries. Oral health promotion strategies are needed in the sports environment. To prevent the onset of oral diseases, the sports dentist can recommend the use of a custom-made mouthguard, an oral device with a triple function that improves the health and performance of athletes. -
Sideline Emergency Management
Lecture 13 June 15, 2018 6/15/2018 Sideline Emergency Management Benjamin Oshlag, MD, CAQSM Assistant Professor of Emergency Medicine Assistant Professor of Sports Medicine Columbia University Medical Center Disclosures Nothing to disclose ©AllinaHealthSystems 1 Lecture 13 June 15, 2018 31 New Orleans Criteria • Single center, 1429 patients from 1997-99 • CT needed if patient meets one of the following: –Headache –Vomiting –Age >60 –Drug or alcohol intoxication –Persistent anterograde amnesia –Visible trauma above the clavicle –Seizure • 6.5% of patients (93/1429) had intracranial injuries, 0.4% (6/1429) required neurosurgical intervention • 100% sensitive for intracranial injuries, 25% specific 32 Canadian Head CT Rule • Prospective cohort study at 10 sites in Canada (N=3121) (2001) • Apply only to: –GCS 13-15 –Amnesia or disorientation to the head injury event or +LOC –Injury within 24 hours •Exclusion –Age <16 –Oral anticoagulants –Seizure after injury –Minimal head injury (no LOC, disorientation, or amnesia) –Obvious skull injury/fracture –Acute neurologic deficit –Unstable vitals –Pregnant ©AllinaHealthSystems 16 Lecture 13 June 15, 2018 33 Canadian Head CT Rule • High Risk Criteria (need for NSG intervention) –GCS <15 at 2 hours post injury –Suspected open or depressed skull fracture –Any sign of basilar skull fracture > Hemotympanum, racoon eyes, Battle’s sign, CSF oto/rhinorrhea –>= 2 episodes of vomiting –Age >= 65 –100% sensitivity 34 Canadian Head CT Rule • Medium Risk Criteria (positive CT that usually requires admission) –Retrograde -
University of Oslo Faculty of Dentistry
UNIVERSITY OF OSLO FACULTY OF DENTISTRY Department of Endodontics Postgraduate Program in Endodontics Case Book Trude Handal Autumn Semester 2014 1 Table of contents Endodontic treatment guidelines ............................................................................................... 1 Non-surgical cases Case 1 ............................................................................................................................................................... 6 Endodontic treatment of the mandibular right second molar with irreversible pulpitis and cracked tooth syndrome Case 2 ............................................................................................................................................................ 12 Endodontic treatment of the mandibular right second molar with irreversible pulpitits and iatrogenic furcal perforation Case 3 ............................................................................................................................................................ 17 Endodontic treatment of a mandibular right first incisor in a patient with DiGeorge Syndrome (velo-cardio-facial-syndrome) Case 4 ............................................................................................................................................................ 22 Endodontic retreatment of the maxillary right second molar Case 5 ........................................................................................................................................................... -
1 – Pathogenesis of Pulp and Periapical Diseases
1 Pathogenesis of Pulp and Periapical Diseases CHRISTINE SEDGLEY, RENATO SILVA, AND ASHRAF F. FOUAD CHAPTER OUTLINE Histology and Physiology of Normal Dental Pulp, 1 Normal Pulp, 11 Etiology of Pulpal and Periapical Diseases, 2 Reversible Pulpitis, 11 Microbiology of Root Canal Infections, 5 Irreversible Pulpitis, 11 Endodontic Infections Are Biofilm Infections, 5 Pulp Necrosis, 12 The Microbiome of Endodontic Infections, 6 Clinical Classification of Periapical (Apical) Conditions, 13 Pulpal Diseases, 8 Nonendodontic Pathosis, 15 LEARNING OBJECTIVES After reading this chapter, the student should be able to: 6. Describe the histopathological diagnoses of periapical lesions of 1. Describe the histology and physiology of the normal dental pulpal origin. pulp. 7. Identify clinical signs and symptoms of acute apical periodon- 2. Identify etiologic factors causing pulp inflammation. titis, chronic apical periodontitis, acute and chronic apical 3. Describe the routes of entry of microorganisms to the pulp and abscesses, and condensing osteitis. periapical tissues. 8. Discuss the role of residual microorganisms and host response 4. Classify pulpal diseases and their clinical features. in the outcome of endodontic treatment. 5. Describe the clinical consequences of the spread of pulpal 9. Describe the steps involved in repair of periapical pathosis after inflammation into periapical tissues. successful root canal treatment. palisading layer that lines the walls of the pulp space, and their Histology and Physiology of Normal Dental tubules extend about two thirds of the length of the dentinal Pulp tubules. The tubules are larger at a young age and eventually become more sclerotic as the peritubular dentin becomes thicker. The dental pulp is a unique connective tissue with vascular, lym- The odontoblasts are primarily involved in production of mineral- phatic, and nervous elements that originates from neural crest ized dentin.