1. Drugs That May Prove Useful for Treatment of Mucositis in Patients
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Another Family with the 'Habsburg Jaw'
J Med Genet: first published as 10.1136/jmg.25.12.838 on 1 December 1988. Downloaded from Journal of Medical Genetics 1988, 25, 838-842 Another family with the 'Habsburg jaw' E M THOMPSON* AND R M WINTERt From *the Department of Paediatric Genetics, Institute of Child Health, 30 Guilford Street, London WCIN IEH; and tthe Kennedy-Galton Centre, Clinical Research Centre, Northwick Park Hospital, Watford Road, Harrow, Middlesex HAI 3HJ. SUMMARY We report a three generation family with similar facial characteristics to those of the Royal Habsburgs, including mandibular prognathism, thickened lower lip, prominent, often misshapen nose, flat malar areas, and mildly everted lower eyelids. One child had craniosyn- ostosis which may be part of the syndrome. The Habsburgs, one of Europe's foremost royal nine successive generations of the family' (fig 1). families, are famous not only for the duration of Although it was transmitted as an autosomal their reign and brilliance of their leadership, but also dominant trait, males were more severely affected because they represent one of the few examples of than females. Examination of the abundant portraits Mendelian inheritance of facial characteristics. This of the family shows, in addition to prognathism, a has been referred to as the 'Habsburg jaw' to thick, everted lower lip, a large, often misshar -n describe the prognathic mandible which was seen in nose with a prominent dorsal hump, a tendenc) to flattening of the malar areas, and mild eversior of Received for publication 9 December 1987. copyright. Revised version accepted for publication 2 March 1988. http://jmg.bmj.com/ on October 3, 2021 by guest. -
Glossary for Narrative Writing
Periodontal Assessment and Treatment Planning Gingival description Color: o pink o erythematous o cyanotic o racial pigmentation o metallic pigmentation o uniformity Contour: o recession o clefts o enlarged papillae o cratered papillae o blunted papillae o highly rolled o bulbous o knife-edged o scalloped o stippled Consistency: o firm o edematous o hyperplastic o fibrotic Band of gingiva: o amount o quality o location o treatability Bleeding tendency: o sulcus base, lining o gingival margins Suppuration Sinus tract formation Pocket depths Pseudopockets Frena Pain Other pathology Dental Description Defective restorations: o overhangs o open contacts o poor contours Fractured cusps 1 ww.links2success.biz [email protected] 914-303-6464 Caries Deposits: o Type . plaque . calculus . stain . matera alba o Location . supragingival . subgingival o Severity . mild . moderate . severe Wear facets Percussion sensitivity Tooth vitality Attrition, erosion, abrasion Occlusal plane level Occlusion findings Furcations Mobility Fremitus Radiographic findings Film dates Crown:root ratio Amount of bone loss o horizontal; vertical o localized; generalized Root length and shape Overhangs Bulbous crowns Fenestrations Dehiscences Tooth resorption Retained root tips Impacted teeth Root proximities Tilted teeth Radiolucencies/opacities Etiologic factors Local: o plaque o calculus o overhangs 2 ww.links2success.biz [email protected] 914-303-6464 o orthodontic apparatus o open margins o open contacts o improper -
Basal Cell Adenoma of Zygomatic Salivary Gland in a Young Dog – First Case Report in Mozambique
RPCV (2015) 110 (595-596) 229-232 Basal cell adenoma of zygomatic salivary gland in a young dog – First case report in Mozambique Adenoma das células basais da glândula salivar zigomática em cão jovem – Primeiro relato de caso em Moçambique Ivan F. Charas dos Santos*1,2, José M.M. Cardoso1, Giovanna C. Brombini3 Bruna Brancalion3 1Departamento de Cirurgia, Faculdade de Veterinária, Universidade Eduardo Mondlane, Maputo, Moçambique 2Pós-doutorando (Bolsista FAPESP), Departamento de Cirurgia e Anestesiologia Veterinária, Faculdade de Medicina Veterinária e Zootecnia (FMVZ), Universidade Estadual Paulista (UNESP), Botucatu, São Paulo, Brasil. 3Faculdade de Medicina Veterinária e Zootecnia (FMVZ), Universidade Estadual Paulista (UNESP),Botucatu, São Paulo, Brasil. Summary: Basal cell adenoma of zygomatic salivary gland Introduction was described in a 1.2 years old Rottweiler dog with swelling of right zygomatic region tissue. Clinical signs were related to Salivary glands diseases in small animals include anorexia, slight pain on either opening of the mouth. Complete blood count, serum biochemistry, urinalysis, thoracic radio- mucocele, salivary gland fistula, sialadenitis, sialad- graphic examination; and transabdominal ultrasound showed enosis, sialolithiasis and less neoplasia (Spangler and no alteration. The findings of cytology examination were con- Culbertson, 1991; Johnson, 2008). Primary tumours sistent with benign tumour and surgical treatment was elected. of salivary glands are rare in dogs and not common- The histopathologic examinations were consistent with basal ly reported in small animals. The incidence is about cell adenoma of zygomatic salivary gland. Seven days after the surgery no alteration was observed. One year later, the dog re- 0.17% in dogs with age between 10 and 12 years turned to check up and confirmed that the dog was healthy and old (Spangler and Culbertson, 1991; Hammer et al., free of clinical and laboratorial signs of tumour recurrence or 2001; Head and Else, 2002). -
A Primary Parotid Mucosa-Associated Lymphoid Tissue Non-Hodgkin Lymphoma in a Patient with Sjogren Syndrome
Open Access Case Report DOI: 10.7759/cureus.15679 A Primary Parotid Mucosa-Associated Lymphoid Tissue Non-Hodgkin Lymphoma in a Patient With Sjogren Syndrome Michael R. Povlow 1 , Mitchell Streiff 2 , Sunthosh Madireddi 1 , Couger Jaramillo 3 1. Department of Radiology, Brooke Army Medical Center, San Antonio, USA 2. Department of Radiology, Ponce Health Sciences University, Ponce, USA 3. Department of Pathology, Brooke Army Medical Center, San Antonio, USA Corresponding author: Michael R. Povlow, [email protected] Abstract The salivary gland tumors are rare entities and the majority of these are benign. However, there are some entities such as prior neck radiation, certain infections, and systemic diseases which should raise the clinical suspicion for a malignant lesion. Patients with Sjogren syndrome are at increased risk for a salivary gland neoplasm, specifically non-Hodgkin lymphoma. While clinical findings play an important role in the initial workup, imaging plays a critical role in the diagnosis and management. This case describes a patient with Sjogren syndrome who presented with a left face mass where imaging was able to confidently diagnose her with a suspicious parotid neoplasm with lymphoma as the favored diagnosis. After histological evaluation, she was diagnosed with primary parotid mucosa-associated lymphoid tissue (MALT) non-Hodgkin lymphoma after which she went on to non-operative management. Categories: Otolaryngology, Pathology, Radiology Keywords: parotid tumor, non-hodgkin’s lymphomas, salivary gland neoplasm, mucosa-associated lymphoid tissue (malt), head and neck neoplasms, head and neck radiology, sjogren's Introduction The salivary gland tumors are rare, accounting for only 6-8% of all head and neck tumors annually in the United States [1]. -
Tutankhamun's Dentition: the Pharaoh and His Teeth
Brazilian Dental Journal (2015) 26(6): 701-704 ISSN 0103-6440 http://dx.doi.org/10.1590/0103-6440201300431 1Department of Oral and Maxillofacial Tutankhamun’s Dentition: Surgery, University Hospital of Leipzig, Leipzig, Germany The Pharaoh and his Teeth 2Institute of Egyptology/Egyptian Museum Georg Steindorff, University of Leipzig, Leipzig, Germany 3Department of Orthodontics, University Hospital of Greifswald, Greifswald, Germany Niels Christian Pausch1, Franziska Naether2, Karl Friedrich Krey3 Correspondence: Dr. Niels Christian Pausch, Liebigstraße 12, 04103 Leipzig, Germany. Tel: +49- 341-97-21160. e-mail: niels. [email protected] Tutankhamun was a Pharaoh of the 18th Dynasty (New Kingdom) in ancient Egypt. Medical and radiological investigations of his skull revealed details about the jaw and teeth status of the mummy. Regarding the jaw relation, a maxillary prognathism, a mandibular retrognathism and micrognathism have been discussed previously. A cephalometric analysis was performed using a lateral skull X-ray and a review of the literature regarding Key Words: Tutankhamun’s King Tutankhamun´s mummy. The results imply diagnosis of mandibular retrognathism. dentition, cephalometric analysis, Furthermore, third molar retention and an incomplete, single cleft palate are present. mandibular retrognathism Introduction also been discussed (11). In 1922, the British Egyptologist Howard Carter found the undisturbed mummy of King Tutankhamun. The Case Report spectacular discovery enabled scientists of the following In the evaluation of Tutankhamun’s dentition and jaw decades to analyze the Pharaoh's remains. The mummy alignment, contemporary face reconstructions and coeval underwent multiple autopsies. Until now, little was artistic images can be of further use. However, the ancient published about the jaw and dentition of the King. -
Oralmedicine
116 Test 98.2 ORAL MEDICINE Developmental Mandibular Salivary Gland Defect The Importance of Clinical Evaluation developmental mandibular salivary gland defect (also known as static A bone cyst, static bone defect, Stafne bone cavity, latent bone cyst, latent bone defect, idiopathic bone cavity, developmen- tal submandibular gland defect of the mandible, aberrant salivary gland defect in the mandible, and lingual mandibular bone Sako Ohanesian, concavity) is a deep, well-defined depression DDS in the lingual surface of the posterior body of the mandible. More precisely, the most common location is within the submandibu- lar gland fossa and often close to the inferi- or border of the mandible. In developmental bone defects investigated surgically, an aberrant lobe of the submandibular gland extends into the bony depression. First recognized by Dr. Edward Stafne in 1942, numerous cases of developmental mandibular salivary gland defect have since been reported, and the lesion should not be considered rare.1 In a study of 4963 pan- Most authorities now agree that this entity is a congenital defect, although it has rarely been observed in children and its precise anatomic nature is still uncertain. oramic images of adult patients, 18 cases of Figure 1. CT slices/panoramic views showing a well-defined radiolucent lesion in the right mandible. salivary gland depression were found by Karmiol and Walsh2, an incidence of nearly 0.4%. Most authorities now agree that this The margins of the radiolucent defect are around an extension of salivary tissue. This entity is a congenital defect, although it has well-defined by a dense radiopaque line. -
Mandibular Prognathism with Unilateral Crossbite—Treatment
message board Mandibular Prognathism with Unilateral Crossbite—Treatment Not Going Well Townie “3MOrtho” wonders how to treat a young boy with a slew of problems such as mandibular prognathism and a mild protrusion of the mandibular incisors 3Mortho Member Since: 11/18/07 Introduction: Post: 1 of 11 The patient is a 10-year-old boy with mandibular prognathism, unilateral crossbite, spacing in the lower arch, minimal crowding in the upper arch, skeletal Class III, and mild protrusion of mandibular incisors. The treatment was started with Delaire mask and RPE. At the end of this treatment (9 months) he had a tete-a-tete bite in the front and the upper arch was successfully expanded. Treatment was continued with Class III and open bite elastics, but the patient is not compliant in wearing the elastics. His second year in treatment, there is deviation of the mandible to the right and the bite cannot be closed with elastics. How would you proceed with the treatment? Would you extract in this case? n 14 DECEMBER 2017 // orthotown.com message board 10/4/2017 Fenrisúlfr Member Since: 02/25/09 What was the rationale for the early intervention/Phase 1? Any discussion re: surgery? Given his Post: 2 of 11 age, and pending a shift, the Class III is likely to significantly worsen with continued mand. growth. A prudent option may be to correct the transverse completely, alleviate any slides and then remove appliances. Once there has been cessation of growth, he can then be evaluated for surgical correction. n 10/4/2017 Shwan Member Since: 08/02/10 There is skeletal mandibular asymmetry that will worsen with time. -
Restriction in Infants
Introduction Ankyloglossia is a congenital anomaly characterized by presence of a hypertrophic lingual frenulum which is short and attached to the very tip of the tongue, limiting its normal movements. Clinically the patient cannot protrude the tongue past the incisal edge of the gingiva and the tongue becomes heart shaped when attempted to protrude. The restricted movements of the tongue can result in problems with breast feeding, lactation, speech disorders and other oral motor disorders like problems with swallowing and licking.1,2 The clinical significance of this anomaly and symptoms produced and the best method of management have been the subject of debate for some time. A review of the relation between the above problems and ankyloglossia is presented. Review Tongue is an accessory organ of importance in mastication, deglutition and speech; many authors describe its stimulatory influence on the development of the dental arches.3 At birth, the tongue unconfined by the teeth, extends outward between the maxillary and mandibular occlusal gum pads. The “infantile swallow” with jaws parted and tongue placed between the jaws is replaced by the adult swallow at the age of two and half years of age. At the start of the normal adult swallow, the lips are closed, the teeth are brought into full occlusion, and the tip is raised and placed against the anterior portion of the palate. The respiratory opening, the nasal cavity, and the anterior portion of the mouth are all sealed off as the tongue, in a sweeping and undulating motion, sweeps backward the mass of chewed food.3,4 The tongue is always short at birth and the tip of the tongue is yet incompletely developed. -
Monomorphic Adenoma: a Diagnosis Or a Misnomer? a Review of Literature on Terminologies, Features and Differential Diagnosis of Basal Cell Adenoma
Acta Scientific DENTAL SCIENCES (ISSN: 2581-4893) Volume 5 Issue 1 January 2021 Research Article Monomorphic Adenoma: A Diagnosis or a Misnomer? A Review of Literature on Terminologies, Features and Differential Diagnosis of Basal Cell Adenoma Swati Gupta1, Ramakant Gupta2* and Manju Gupta3 Received: December 01, 2020 1Senior Consultant, Oral and Maxillofacial Pathology, Dr. Jatinder Gupta’s Gupta Published: Clinic and Opticals, Haryana, India © All rights are reserved by Ramakant 2Head and Consultant, Department of Dental Services, Dr. Jatinder Gupta’s Gupta December 29, 2020 Gupta., et al. Clinic and Opticals, Haryana, India 3Director and Clinic coordinator, Dr. Jatinder Gupta’s Gupta Clinic and Opticals, Haryana, India *Corresponding Author: Ramakant Gupta, Head and Consultant, Department of Dental Services, Dr. Jatinder Gupta’s Gupta Clinic and Opticals, Haryana, India. Abstract Basal cell adenoma, previously was termed by few authors as Monomorphic adenoma. The term “Monomorphic adenoma” was originally proposed for any benign epithelial salivary gland tumour other than benign mixed tumors. Monomorphic adenoma includ- salivary gland tumours the term “monomorphic adenoma” is not included and basal cell adenoma is considered as a separate entity. ed tumours such as Warthins tumour, basal cell adenoma and canalicular adenoma. However, in the new 2005 WHO classification of diagnosis and differentiation of basal cell adenoma from other tumours. This paper intends to discuss the controversies regarding the terminology and classification of monomorphic adenoma along with Keywords: Adenoid Cystic Carcinoma; Basal Cell Adenoma; Basal Cell Carcinoma; Canalicular Adenoma; Epithelial Salivary Gland Tumour; Monomorphic Adenoma; WHO Classification Abbreviations to controversies. The term “monomorphic adenoma” was original- ly proposed for any benign epithelial salivary gland tumour other ACC: Adenoid Cystic Carcinoma; BCA: Basal Cell Adenoma; BCC: than benign mixed tumours by Rouch in 1970 [7]. -
Features of Reactive White Lesions of the Oral Mucosa
Head and Neck Pathology (2019) 13:16–24 https://doi.org/10.1007/s12105-018-0986-3 SPECIAL ISSUE: COLORS AND TEXTURES, A REVIEW OF ORAL MUCOSAL ENTITIES Frictional Keratosis, Contact Keratosis and Smokeless Tobacco Keratosis: Features of Reactive White Lesions of the Oral Mucosa Susan Müller1 Received: 21 September 2018 / Accepted: 2 November 2018 / Published online: 22 January 2019 © Springer Science+Business Media, LLC, part of Springer Nature 2019 Abstract White lesions of the oral cavity are quite common and can have a variety of etiologies, both benign and malignant. Although the vast majority of publications focus on leukoplakia and other potentially malignant lesions, most oral lesions that appear white are benign. This review will focus exclusively on reactive white oral lesions. Included in the discussion are frictional keratoses, irritant contact stomatitis, and smokeless tobacco keratoses. Leukoedema and hereditary genodermatoses that may enter in the clinical differential diagnoses of frictional keratoses including white sponge nevus and hereditary benign intraepithelial dyskeratosis will be reviewed. Many products can result in contact stomatitis. Dentrifice-related stomatitis, contact reactions to amalgam and cinnamon can cause keratotic lesions. Each of these lesions have microscopic findings that can assist in patient management. Keywords Leukoplakia · Frictional keratosis · Smokeless tobacco keratosis · Stomatitis · Leukoedema · Cinnamon Introduction white lesions including infective and non-infective causes will be discussed -
Peripheral Giant Cell Reparative Granuloma of Maxilla in a Patient with Aggressive Periodontitis
Peripheral Giant Cell Reparative Granuloma of Maxilla in a Patient with Aggressive Periodontitis E Cayci1, B Kan2, E Guzeldemir-Akcakanat1, B Muezzinoglu3 1Department of Periodontology, Kocaeli University, Faculty of Dentistry, Kocaeli, Turkey. 2Department of Oral and Maxillofacial Surgery, Kocaeli University, Faculty of Dentistry, Kocaeli, Turkey. 3Department of Pathology, Kocaeli University, School of Medicine, Kocaeli, Turkey. Abstract Peripheral giant cell reparative granuloma is a reactive and rare lesion of oral cavity with unknown etiology which is derived from periosteum and periodontal ligament and occurs frequently in young adults. Inflammation or trauma is underlying causative factor of reactive proliferation. In the present case report, a 35 year-old male with aggressive periodontitis and peripheral giant cell reparative granuloma is presented. The patient applied to our clinic with a complaining about a big nodule at his palate. The lesion was pedunculated and localized at his right maxilla between #16 and #17 which arose from distal aspect of #16, and the surface of the lesion was hyperkeratotic and the lesion was measured 22 x 30 mm at the largest diameter. He also had severe generalized aggressive periodontitis and hypertension. Amoxicillin clavulanate 625 mg, three times a day, metronidazole 500 mg three times a day and 0.2% chlorhexidine digluconate oral rinse, twice a day for a week, were prescribed to the patient. Then, scaling and root planing were performed along with systemic antibiotic treatment and he scheduled for surgery. The lesion was excised completely and #16 was extracted. After the healing period, periodontal surgery was planned for the treatment of aggressive periodontitis. Obtained tissue specimen was sent for histopathological examination. -
Iii Bds Oral Pathology and Microbiology
III BDS ORAL PATHOLOGY AND MICROBIOLOGY Theory: 120 Hours ORAL PATHOLOGY MUST KNOW 1. Benign and Malignant Tumours of the Oral Cavity (30 hrs) a. Benign tumours of epithelial tissue origin - Papilloma, Keratoacanthoma, Nevus b. Premalignant lesions and conditions: - Definition, classification - Epithelial dysplasia - Leukoplakia, Carcinoma in-situ, Erythroplakia, Palatal changes associated with reverse smoking, Oral submucous fibrosis c. Malignant tumours of epithelial tissue origin - Basal Cell Carcinoma, Epidermoid Carcinoma (Including TNM staging), Verrucous carcinoma, Malignant Melanoma. d. Benign tumours of connective tissue origin : - Fibroma, Giant cell Fibroma, Peripheral and Central Ossifying Fibroma, Lipoma, Haemangioma (different types). Lymphangioma, Chondroma, Osteoma, Osteoid Osteoma, Benign Osteoblastoma, Tori and Multiple Exostoses. e. Tumour like lesions of connective tissue origin : - Peripheral & Central giant cell granuloma, Pyogenic granuloma, Peripheral ossifying fibroma f. Malignant Tumours of Connective tissue origin : - Fibrosarcoma, Chondrosarcoma, Kaposi's Sarcoma Ewing's sarcoma, Osteosarcoma Hodgkin's and Non Hodgkin's L ymphoma, Burkitt's Lymphoma, Multiple Myeloma, Solitary Plasma cell Myeloma. g. Benign Tumours of Muscle tissue origin : - Leiomyoma, Rhabdomyoma, Congenital Epulis of newborn, Granular Cell tumor. h. Benign and malignant tumours of Nerve Tissue Origin - Neurofibroma & Neurofibromatosis-1, Schwannoma, Traumatic Neuroma, Melanotic Neuroectodermal tumour of infancy, Malignant schwannoma. i. Metastatic