Analysis of Lymphatic Drainage in Various Forms of Leg Edema Using Two Compartment Lymphoscintigraphy

Total Page:16

File Type:pdf, Size:1020Kb

Analysis of Lymphatic Drainage in Various Forms of Leg Edema Using Two Compartment Lymphoscintigraphy 43 Lymphology 31 (1998) 43-55 ANALYSIS OF LYMPHATIC DRAINAGE IN VARIOUS FORMS OF LEG EDEMA USING TWO COMPARTMENT LYMPHOSCINTIGRAPHY P. Brautigam, E. FOldi, I. Schaiper, T. Krause, W. Vanscheidt, E. Moser Abteilung Nuklearmedizin, Radiol. University Klinik Freiburg (PB,TK,EM), FOldiklinik, Fachklinik ftir lymphologische Erkrankungen, Hinterzarten (EF), Zentrum ftir Nuklearmedizin, Paracelsus- Klinik Osnabrtick (IS), and Universitats-Hautklinik, Freiburg (WV), Germany ABSTRACT was reduced. Patients with lipedema (obesity) scintigraphically showed no alteration in The anatomical and functional status lymphatic transport. of the epifascial and subfascial lymphatic This study demonstrates that lymphatic compartments was analyzed using two drainage is notably affected (except in obesity compartment lymphoscintigraphy in five termed lipedema) in various edemas of the leg. groups of patients (total 55) with various Lymphatic drainage varied depending on the forms of edema of the lower extremities. specific compartment and the pathophysiologic Digital whole body scintigraphy enabled mechanism accounting for the edema. Two semiquantitative estimation of radio tracer compartment lymphoscintigraphy is a valuable transport with comparison of lymphatic diagnostic tool for accurate assessment of leg drainage between those individuals without edema of known and unknown origin. (normal) and those with leg edema by calculating the uptake of the radiopharma- The lymphatic system is deranged in ceutical transported to regional lymph nodes. various forms of leg edema of both lymphatic A visual assessment of the lymphatic drainage and non-lymphatic origin including venous- pathways of the legs was also performed. lymphedema, cyclic idiopathic edema, and In patients with cyclic idiopathic edema, lipedema (obesity) (1,2). As no discrete an accelerated rate of lymphatic transport differences exist on clinical grounds among was detected (high lymph volume overload or the various edema syndromes, differential dynamic insufficiency). In those with venous diagnosis and identification of the main (phlebo )edemas, high volume lymphatic pathophysiologic factors may be difficult or overload (dynamic insufficiency) of the impossible (1). epifascial compartment was scintigraphically Cyclic idiopathic edema of women entails detected by increased tracer uptake in regional much more than manifestations of the nodes. In patients with deep femoral venous premenstrual syndrome. Because of increased occlusion (post-thrombotic syndrome), permeability of the blood capillaries, both subfasciallymphatic transport was uniformly plasma proteins and great quantities of fluid markedly reduced (safety valve lymphatic (6-8 liters) may accumulate in the inter- insufficiency). On the other hand, in the stitium within a few days. As a result, the epifascial compartment, lymph transport was lymphatic system is overloaded (high volume accelerated. In those patients with recurrent insufficiency) with anasarca and in rare or extensive skin ulceration, lymph transport instances, even pulmonary edema (2). Permission granted for single print for individual use. Reproduction not permitted without permission of Journal LYMPHOLOGY . 44 In phleboedema, the venous and capillary Group 1: Eighteen patients (10 female, hydrostatic pressure are increased and the 8 male; age 19-48 years) had localized demand for lymphatic drainage increases (2). malignant melanoma of the skin proximal to Over time, the lymphatic system becomes the umbilicus. Accordingly, involvement of functionally insufficient, the lymph transport the lymphatic drainage of the lower extremity capacity decreases, and edema ensues was deemed unlikely. Two compartment (termed safety valve lymphatic insufficiency) lymphoscintigraphy was carried out and the (2). In more chronic venous insufficiency, results were used to define normal values. lymph flow is also impaired (lymphedema). In lipedema or obesity, the pathophysiology Group 2: Ten women (age 21-41 years) of lymphatic drainage is less clear. Stallworth had cyclic idiopathic edema likely from et al (3) claims that the lymphatics, arteries, increased microvascular permeability (8). and veins are unaltered. Herpertz (4), on the Fluid retention consisted of swelling of the other hand, suggests a slight mechanical face, breast, abdomen, and legs which drainage blockage to lymph capillaries, worsened as the day advanced. A diurnal precollectors, and collectors secondary to weight variation of more than 1.4 kg daily increased pressure of the excess fatty tissue. was considered abnormal. A modified FOidi et al (2) emphasizes a combination of Streeten test (9) was also used and was contributions to leg enlargement including considered positive if the renal excretion of lipedema + lymphedema and lipedema + an oral fluid load (20 mllkg body weight) was cyclic idiopathic edema. In the clinical pre- less than 60% after 4 hours of orthostasis. sentation of patients with lipedema (pressure sensitive edema and a supramalleolar fat Group 3: Seven patients (4 male, 3 collar), both edema of the top of the foot and female; age 36-64 years) with primary venous a positive Stemmer's thickened skin fold sign varicosities of the greater saphenous vein (signs of lymphedema) are present as well. (phleboedema) (stage IV according to Hach) Two compartment lymphoscintigraphy (10) underwent lymphoscintigraphy to is a useful method to quantify lymphatic exclude occult lymphedema before excision drainage in the epifascial and subfascial and ligation of varicose veins. Functionally compartment of the legs (5-7). This study and morphologically, the femoral (deep) analyzed the lymphatic drainage in the venous system was intact according to previously mentioned forms of leg edema as Doppler sonography and phlebography. well as in deep venous occlusion (post- thrombotic syndrome) using two compart- Group 4: Eleven patients (6 male, 5 ment lymphoscintigraphy. Of special interest female; age 45-68 years) had deep vein was the contribution of this scintiscan thrombosis (from 1-19 years) (post- technique in the differential diagnosis in leg thrombotic syndrome) before lymphoscinti- edema of uncertain origin. graphy. Clinical complications included dermatoliposclerosis and recurrent skin ulcerations. MATERIAL AND METHODS Group 5: Five of 9 women (age 23-37 Patients years) showed considerable fat changes of the upper and lower legs (lipedema). In the other Between 1990 and 1996, 55 patients four patients, "edema" was seen only in the clinically separated into five groups were upper leg. Stemmer's skin fold sign was subjected to two compartment absent in all patients and lipedema had been lymphoscintigraphy. present for over 5 years. Permission granted for single print for individual use. Reproduction not permitted without permission of Journal LYMPHOLOGY . 45 Fig. 1. Injection of the radio tracer into the lateral aspect of the side of the foot at a depth of 1.5 cm ensures drainage by the sUbfasciallymphatics. Lymphoscintigraphy To examine the epifascial compartment, a second study using identical lymphatic Subjasciai compartment: 4x20 MBq 99m stimulation and acquisition was performed Tc of labeled human albumin nanocolloid was after disappearance of residual tracer activity injected into the dorsolateral muscles of the (at least 2 days later) but the radiopharma- sole of the foot (Fig. 1). Standardized stimu- ceutical was injected subcutaneously between lation of lymph flow followed using bicycle the first and third toes. ergometry at a low level of intensity (25 Watt) Lymphatic function was assessed by in 2 phases. After 20 minutes the first whole analyzing the uptake of the radiopharma- body scintigram was performed using a single ceutical into the lymph nodes. For this head gamma camera (Diacam®, Siemens purpose, regions of interest (ROI) were Company). After another 30 minutes of drawn, one over the inguinal and parailiac bicycle ergometry and a recovery phase, a lymph nodes (ROIl)' and a second over the second scintigram was obtained at 2 hours. injection site and the lymph vessels (R0I2). Permission granted for single print for individual use. Reproduction not permitted without permission of Journal LYMPHOLOGY . 46 50 50 50 50 50 •• 40 40 40 40 40 ••• •• • 1i. • • • ...';30 30 • 30 • 30 30 :l Q. ::::I • • Gi .:-. .., • • • • • g 20 • 20 20 • 20 20 • 0 •••• • •• '6.. • • • •••••• a:: :• •• .-.... : ........ • 10 - -.::.- .. 10 10 10 10 - •••••.-. • • • .:.• 0 0 0 0 0 Cyclic Postthrombo- Lipedema Controls idiopathic Phlebedema tic Syndrome Edema Fig. 2. Lymph node radiotracer uptake (%) for each patient in the epifascial compartment. Percent uptake was calculated as RESULTS (R0I1)/{R0I1 + R0I2) x 100. Lymphatic transport was also visually The radiotracer uptake values for each evaluated. Reduced or absent radiotracer patient are shown in Fig. 2 (epifascial activity in regional lymph nodes represented compartment) and Fig. 3 (subfascial impaired lymphatic drainage. The compartment). In cyclic idiopathic edema appearance of 99m Tc nanocolloid in the and lipedema, the uptake values are shown proximal portion of the thoracic duct was for both legs. In phleboedema and deep interpreted as physiologic lymph pooling venous occlusion (post-thrombotic syndrome), before entry into the venous system and only the uptake values for the affected thought to represent accelerated lymphatic (edematous) side are shown. The median, the transport (high lymph volume overload or 5th and the 95th percentiles of each patient dynamic
Recommended publications
  • Lower Extremity Deep Venous Thrombosis
    SECTION 5 Vascular System CHAPTER 34 Lower Extremity Deep Venous Thrombosis Ariel L. Shiloh KEY POINTS • Providers can accurately detect lower extremity deep venous thrombosis with point-of- care ultrasound after limited training. • Compression ultrasound exams are as accurate as traditional duplex and triplex vascular ultrasound exams. • Compression ultrasound exam at only two sites, the common femoral vein and popliteal vein, permits rapid and accurate assessment of deep venous thrombosis. Background care providers can perform lower extremity compression ultrasonography exams rapidly Venous thromboembolic disease (VTE) is a and with high diagnostic accuracy to detect common cause of morbidity and mortality in DVT. 7–13 A meta-analysis of 16 studies showed hospitalized patients and is especially preva- that point-of-care ultrasound can accurately lent in critically ill patients.1–3 Approximately diagnose lower extremity DVTs with a pooled 70% to 90% of patients with an identified source sensitivity of 96% and specificity of 97%.14 of pulmonary embolism (PE) have a proxi- Traditional vascular studies, the duplex mal lower extremity deep venous thrombosis and triplex exams, use a combination of (DVT). Conversely, 40% to 50% of patients two-dimensional (2D) imaging with compres- with a proximal DVT have a concurrent pul- sion along with the use of color and/or spectral monary embolism at presentation, and simi- Doppler ultrasound. More recent studies have larly, in only 50% of patients presenting with a demonstrated that 2D compression ultrasound PE can a DVT be found.4–6 exams alone yield similar accuracy as tradi- Point-of-care ultrasound is readily available tional duplex or triplex vascular studies.9,11,15–17 as a diagnostic tool for VTE.
    [Show full text]
  • Difference Between Vein and Venule Key Difference - Vein Vs Venule
    Difference Between Vein and Venule www.differencebetween.com Key Difference - Vein vs Venule The veins are the blood vessels that carry blood towards the heart. It always has a low blood pressure. Except the pulmonary and the umbilical veins, all the remaining veins carry the deoxygenated blood. On the contrary, the arteries carry blood away from the heart. The veins have valves in them to prevent the black flow of blood and to maintain unidirectional blood flow. They are less muscular, larger and are located closer to the skin. Venules are very smaller veins. They are the ones that collect blood from the capillaries. The collected blood will be directed to the larger and medium veins where the blood is transported towards the heart again. Many venules unite to form larger veins. The key difference between Vein and Venule is, the vein is a larger blood vessel that carries blood towards the heart while, the venule is a smaller minute blood vessel that drains blood from capillaries to the veins. What is a Vein? The veins are larger blood vessels present in throughout the body. The main function of veins is to carry oxygen-poor blood back into the heart. Veins are classified into some categories such as, superficial veins, pulmonary veins, deep veins, perforator veins, communicating veins and systematic veins. The wall of the vein is thinner and less elastic than the wall of an artery. The walls of the veins consist three layers of tissues which are named as tunica externa, tunica media, and tunica intima. Veins also have larger and irregular lumen.
    [Show full text]
  • Peripheral Vascular Disease (PVD) Fact Sheet
    FACT SHEET FOR PATIENTS AND FAMILIES Peripheral Vascular Disease (PVD) What is peripheral vascular disease? Vascular disease is disease of the blood vessels (arteries and veins). Peripheral vascular disease (PVD) affects The heart receives blood, the areas that are “peripheral,” or outside your heart. sends it to The most common types of PVD are: the lungs to get oxygen, • Carotid artery disease affects the arteries and pumps that carry blood to your brain. It occurs when it back out. one or more arteries are narrowed or blocked by plaque, a fatty substance that builds up inside artery walls. Carotid artery disease can increase Veins carry Arteries carry your risk of stroke. It can also cause transient blood to your oxygen-rich [TRANZ-ee-ent] ischemic [iss-KEE-mik] attacks (TIAs). heart to pick blood from up oxygen. your heart TIAs are temporary changes in brain function to the rest of that are sometimes called “mini-strokes.” your body. • Peripheral arterial disease (PAD) often affects the arteries to your legs and feet. It is also caused by Healthy blood vessels provide oxygen plaque buildup, and can for every part of your body. cause pain that feels like a dull cramp or heavy tiredness in your hips or legs when • Venous insufficiency affects the veins, usually you exercise or climb stairs. in your legs or feet. Your veins have valves that This pain is sometimes Damaged Healthy keepvalve blood fromvalve flowing backward as it moves called claudication. If PAD toward your heart. If the valves stop working, blood worsens, it can cause cold Plaque can build backs up in your body, usually in your legs.
    [Show full text]
  • Microlymphatic Surgery for the Treatment of Iatrogenic Lymphedema
    Microlymphatic Surgery for the Treatment of Iatrogenic Lymphedema Corinne Becker, MDa, Julie V. Vasile, MDb,*, Joshua L. Levine, MDb, Bernardo N. Batista, MDa, Rebecca M. Studinger, MDb, Constance M. Chen, MDb, Marc Riquet, MDc KEYWORDS Lymphedema Treatment Autologous lymph node transplantation (ALNT) Microsurgical vascularized lymph node transfer Iatrogenic Secondary Brachial plexus neuropathy Infection KEY POINTS Autologous lymph node transplant or microsurgical vascularized lymph node transfer (ALNT) is a surgical treatment option for lymphedema, which brings vascularized, VEGF-C producing tissue into the previously operated field to promote lymphangiogenesis and bridge the distal obstructed lymphatic system with the proximal lymphatic system. Additionally, lymph nodes with important immunologic function are brought into the fibrotic and damaged tissue. ALNT can cure lymphedema, reduce the risk of infection and cellulitis, and improve brachial plexus neuropathies. ALNT can also be combined with breast reconstruction flaps to be an elegant treatment for a breast cancer patient. OVERVIEW: NATURE OF THE PROBLEM Clinically, patients develop firm subcutaneous tissue, progressing to overgrowth and fibrosis. Lymphedema is a result of disruption to the Lymphedema is a common chronic and progres- lymphatic transport system, leading to accumula- sive condition that can occur after cancer treat- tion of protein-rich lymph fluid in the interstitial ment. The reported incidence of lymphedema space. The accumulation of edematous fluid mani- varies because of varying methods of assess- fests as soft and pitting edema seen in early ment,1–3 the long follow-up required for diagnosing lymphedema. Progression to nonpitting and irre- lymphedema, and the lack of patient education versible enlargement of the extremity is thought regarding lymphedema.4 In one 20-year follow-up to be the result of 2 mechanisms: of patients with breast cancer treated with mastec- 1.
    [Show full text]
  • Anatomy of the Large Blood Vessels-Veins
    Anatomy of the large blood vessels-Veins Cardiovascular Block - Lecture 4 Color index: !"#$%&'(& !( "')*+, ,)-.*, $()/ Don’t forget to check the Editing File !( 0*"')*+, ,)-.*, $()/ 1$ ($&*, 23&%' -(0$%"'&-$(4 *3#)'('&-$( Objectives: ● Define veins, and understand the general principles of venous system. ● Describe the superior & inferior Vena Cava and their tributaries. ● List major veins and their tributaries in the body. ● Describe the Portal Vein. ● Describe the Portocaval Anastomosis Veins ◇ Veins are blood vessels that bring blood back to the heart. ◇ All veins carry deoxygenated blood. with the exception of the pulmonary veins(to the left atrium) and umbilical vein(umbilical vein during fetal development). Vein can be classified in two ways based on Location Circulation ◇ Superficial veins: close to the surface of the body ◇ Veins of the systemic circulation: NO corresponding arteries Superior and Inferior vena cava with their tributaries ◇ Deep veins: found deeper in the body ◇ Veins of the portal circulation: With corresponding arteries Portal vein Superior Vena Cava ◇Formed by the union of the right and left Brachiocephalic veins. ◇Brachiocephalic veins are formed by the union of internal jugular and subclavian veins. Drains venous blood from : ◇ Head & neck ◇ Thoracic wall ◇ Upper limbs It Passes downward and enter the right atrium. Receives azygos vein on its posterior aspect just before it enters the heart. Veins of Head & Neck Superficial veins Deep vein External jugular vein Anterior Jugular Vein Internal Jugular Vein Begins just behind the angle of mandible It begins in the upper part of the neck by - It descends in the neck along with the by union of posterior auricular vein the union of the submental veins.
    [Show full text]
  • Incidence of Deep Venous Thrombosis and Stratification of Risk Groups in A
    ORIGINAL ARTICLE Incidence of deep venous thrombosis and stratification of risk groups in a university hospital vascular surgery unit Incidência de trombose venosa profunda e estratificação dos grupos de risco em serviço de cirurgia vascular de hospital universitário 1 1 1 2 Alberto Okuhara *, Túlio Pinho Navarro , Ricardo Jayme Procópio , José Oyama Moura de Leite Abstract Background: There is a knowledge gap with relation to the true incidence of deep vein thrombosis among patients undergoing vascular surgery procedures in Brazil. This study is designed to support the implementation of a surveillance system to control the quality of venous thromboembolism prophylaxis in our country. Investigations in specific institutions have determined the true incidence of deep vein thrombosis and identified risk groups, to enable measures to be taken to ensure adequate prophylaxis and treatment to prevent the condition. Objective: To study the incidence of deep venous thrombosis in patients admitted to hospital for non-venous vascular surgery procedures and stratify them into risk groups. Method: This was a cross-sectional observational study that evaluated 202 patients from a university hospital vascular surgery clinic between March 2011 and July 2012. The incidence of deep venous thrombosis was determined using vascular ultrasound examinations and the Caprini scale. Results: The mean incidence of deep venous thrombosis in vascular surgery patients was 8.5%. The frequency distribution of patients by venous thromboembolism risk groups was as follows: 8.4% were considered low risk, 17.3% moderate risk, 29.7% high risk and 44.6% were classified as very high risk. Conclusion: The incidence of deep venous thrombosis in vascular surgery patients was 8.5%, which is similar to figures reported in the international literature.
    [Show full text]
  • M. H. RATZLAFF: the Superficial Lymphatic System of the Cat 151
    M. H. RATZLAFF: The Superficial Lymphatic System of the Cat 151 Summary Four examples of severe chylous lymph effusions into serous cavities are reported. In each case there was an associated lymphocytopenia. This resembled and confirmed the findings noted in experimental lymph drainage from cannulated thoracic ducts in which the subject invariably devdops lymphocytopenia as the lymph is permitted to drain. Each of these patients had com­ munications between the lymph structures and the serous cavities. In two instances actual leakage of the lymphography contrrult material was demonstrated. The performance of repeated thoracenteses and paracenteses in the presenc~ of communications between the lymph structures and serous cavities added to the effect of converting the. situation to one similar to thoracic duct drainage .The progressive immaturity of the lymphocytes which was noted in two patients lead to the problem of differentiating them from malignant cells. The explanation lay in the known progressive immaturity of lymphocytes which appear when lymph drainage persists. Thankful acknowledgement is made for permission to study patients from the services of Drs. H. J. Carroll, ]. Croco, and H. Sporn. The graphs were prepared in the Department of Medical Illustration and Photography, Dowristate Medical Center, Mr. Saturnino Viloapaz, illustrator. References I Beebe, D. S., C. A. Hubay, L. Persky: Thoracic duct 4 Iverson, ]. G.: Phytohemagglutinin rcspon•e of re­ urctcral shunt: A method for dccrcasingi circulating circulating and nonrecirculating rat lymphocytes. Exp. lymphocytes. Surg. Forum 18 (1967), 541-543 Cell Res. 56 (1969), 219-223 2 Gesner, B. M., J. L. Gowans: The output of lympho­ 5 Tilney, N.
    [Show full text]
  • What Is Dvt? Deep Vein Thrombosis (DVT) Occurs When an Abnormal Blood Clot Forms in a Large Vein
    What is DVt? Deep vein thrombosis (DVT) occurs when an abnormal blood clot forms in a large vein. These clots usually develop in the lower leg, thigh, or pelvis, but can also occur in other large veins in the body. If you develop DVT and it is diagnosed correctly and quickly, it can be treated. However, many people do not know if they are at risk, don’t know the symptoms, and delay seeing a healthcare professional if they do have symptoms. CAn DVt hAppen to me? Anyone may be at risk for DVT but the more risk factors you have, the greater your chances are of developing DVT. Knowing your risk factors can help you prevent DVt: n Hospitalization for a medical illness n Recent major surgery or injury n Personal history of a clotting disorder or previous DVT n Increasing age this is serious n Cancer and cancer treatments n Pregnancy and the first 6 weeks after delivery n Hormone replacement therapy or birth control products n Family history of DVT n Extended bed rest n Obesity n Smoking n Prolonged sitting when traveling (longer than 6 to 8 hours) DVt symptoms AnD signs: the following are the most common and usually occur in the affected limb: n Recent swelling of the limb n Unexplained pain or tenderness n Skin that may be warm to the touch n Redness of the skin Since the symptoms of DVT can be similar to other conditions, like a pulled muscle, this often leads to a delay in diagnosis. Some people with DVT may have no symptoms at all.
    [Show full text]
  • What You Need to Know About Deep Vein Thrombosis (DVT)
    What You Need to Know About Deep Vein Thrombosis What is a Deep Vein Thrombosis? A deep vein thrombosis (DVT) is a blood clot that forms in the veins in the body (usually in the leg or pelvis). What causes a Deep Vein Thrombosis? Deep vein thrombosis (DVT) sometimes occurs for no apparent reason. However, certain factors can increase the chance of developing a DVT: • Inactivity • Hospital stays and surgery • Damage to your blood vessel from an injury or trauma • Medical and genetic conditions • Pregnancy • Taking estrogen-based medicine such as hormonal birth control or hormone replacement therapy • Overweight or obese • Family history of DVT • Older age What are the symptoms of DVT? Only about half of the people who have a DVT have signs and symptoms. These signs and symptoms of a deep vein clot include: • Pain or tenderness, often starting in the calf. • Swelling, including the ankle & foot. • Warmth and redness of the area or a noticeable discoloration Vascular Surgery -1- How is a DVT diagnosed? Your doctor will ask you questions about your symptoms and if your symptoms suggest that a blood clot is likely, you could have one or all of the following tests: • Blood test for a D-dimer: this test measures the level of a compound released when blood clots are dissolving. A high level may mean you have Deep Vein Thrombosis (DVT). • Imaging studies: o Ultrasound – This is the most common test for diagnosing deep vein blood clots. This test uses sound waves to create pictures of blood flowing through the arteries and veins in the affected leg.
    [Show full text]
  • Acr–Aium–Spr–Sru Practice Parameter for the Performance of Peripheral Venous Ultrasound Examination
    The American College of Radiology, with more than 30,000 members, is the principal organization of radiologists, radiation oncologists, and clinical medical physicists in the United States. The College is a nonprofit professional society whose primary purposes are to advance the science of radiology, improve radiologic services to the patient, study the socioeconomic aspects of the practice of radiology, and encourage continuing education for radiologists, radiation oncologists, medical physicists, and persons practicing in allied professional fields. The American College of Radiology will periodically define new practice parameters and technical standards for radiologic practice to help advance the science of radiology and to improve the quality of service to patients throughout the United States. Existing practice parameters and technical standards will be reviewed for revision or renewal, as appropriate, on their fifth anniversary or sooner, if indicated. Each practice parameter and technical standard, representing a policy statement by the College, has undergone a thorough consensus process in which it has been subjected to extensive review and approval. The practice parameters and technical standards recognize that the safe and effective use of diagnostic and therapeutic radiology requires specific training, skills, and techniques, as described in each document. Reproduction or modification of the published practice parameter and technical standard by those entities not providing these services is not authorized. Revised 2019 (Resolution 29)* ACR–AIUM–SPR–SRU PRACTICE PARAMETER FOR THE PERFORMANCE OF PERIPHERAL VENOUS ULTRASOUND EXAMINATION PREAMBLE This document is an educational tool designed to assist practitioners in providing appropriate radiologic care for patients. Practice Parameters and Technical Standards are not inflexible rules or requirements of practice and are not intended, nor should they be used, to establish a legal standard of care1.
    [Show full text]
  • Lower Limb Deep Venous Flow in Patients with Peripheral Vascular Disease
    View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Elsevier - Publisher Connector Lower limb deep venous flow in patients with peripheral vascular disease Gabor Libertiny, FRCS, and Linda Hands, MS, FRCS, Oxford, United Kingdom Purpose: A prospective controlled study was undertaken to determine how peripheral vascular disease (PVD) influences flow in the deep veins of the leg. Methods: Eighty-nine patients with peripheral vascular disease and 35 age-matched con- trol subjects were studied. The popliteal vein diameter and flow velocity were measured at rest by means of color duplex ultrasound scanning, and these measurements were compared with the ankle-brachial pressure index. For 23 subjects, measurements were also performed during reactive hyperemia and then repeated after venous return from the foot was prevented by an ankle cuff. Results: There was a significant correlation between the ankle-brachial pressure index and the popliteal vein diameter (r = 0.35, P < .001) but a negative correlation between the ankle-brachial pressure index and venous flow velocity among patients with PVD (r = –0.24, P = .002). In PVD patients the diameter decreased further in reactive hyper- emia, whereas it increased in control subjects (P < .001). Preventing venous return from the foot in PVD patients led to diameter increase at rest and abolished the reduction in diameter caused by reactive hyperemia. Despite the reduction in diameter during reac- tive hyperemia, flow velocity increased less in patients with PVD than it did in control subjects (P = .01). Conclusion: Chronic tissue ischemia results in constriction of the popliteal vein.
    [Show full text]
  • Deep Vein Thrombosis (DVT) and Pulmonary Embolism (PE)
    How can it be prevented? You can take steps to prevent deep vein thrombosis (DVT) and pulmonary embolism (PE). If you're at risk for these conditions: • See your doctor for regular checkups. • Take all medicines as your doctor prescribes. • Get out of bed and move around as soon as possible after surgery or illness (as your doctor recommends). Moving around lowers your chance of developing a blood clot. References: • Exercise your lower leg muscles during Deep Vein Thrombosis: MedlinePlus. (n.d.). long trips. Walking helps prevent blood Retrieved October 18, 2016, from clots from forming. https://medlineplus.gov/deepveinthrombos is.html If you've had DVT or PE before, you can help prevent future blood clots. Follow the steps What Are the Signs and Symptoms of Deep above and: Vein Thrombosis? - NHLBI, NIH. (n.d.). Retrieved October 18, 2016, from • Take all medicines that your doctor http://www.nhlbi.nih.gov/health/health- prescribes to prevent or treat blood clots topics/topics/dvt/signs • Follow up with your doctor for tests and treatment Who Is at Risk for Deep Vein Thrombosis? - • Use compression stockings as your DEEP NHLBI, NIH. (n.d.). Retrieved October 18, doctor directs to prevent leg swelling 2016, from http://www.nhlbi.nih.gov/health/health- VEIN topics/topics/dvt/atrisk THROMBOSIS How Can Deep Vein Thrombosis Be Prevented? - NHLBI, NIH. (n.d.). Retrieved October 18, 2016, from (DVT) http://www.nhlbi.nih.gov/health/health- topics/topics/dvt/prevention How Is Deep Vein Thrombosis Treated? - NHLBI, NIH. (n.d.). Retrieved October 18, 2016, from http://www.nhlbi.nih.gov/health/health- topics/topics/dvt/treatment Trinity Surgery Center What is deep vein Who is at risk? What are the thrombosis (DVT)? The risk factors for deep vein thrombosis symptoms? (DVT) include: Only about half of the people who have DVT A blood clot that forms in a vein deep in the • A history of DVT.
    [Show full text]