Temporomandibular Joint Disorders Policy Number: MP-016 Last Review Date: 02/21/2019 Effective Date: 04/01/2019 Policy
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Temporomandibular Joint Dysfunction Corporate Medical Policy Policy
Temporomandibular Joint Dysfunction Corporate Medical Policy File Name: Temporomandibular Joint Dysfunction File Code: UM.SURG.07 Origination: 06/2011 Last Review: 07/2019 Next Review: 07/2020 Effective Date: 10/01/2019 Description/Summary Temporomandibular joint (TMJ) dysfunction refers to a group of disorders characterized by pain in the TMJ and surrounding tissues. Initial conservative therapy is generally recommended; there are also a variety of nonsurgical and surgical treatment possibilities for patients whose symptoms persist. Policy Coding Information Click the links below for attachments, coding tables & instructions. Attachment I - CPT® Code Table & Instructions Attachment II - ICD-10-CM Diagnosis of TMJ When a service may be considered medically necessary • The following diagnostic procedures may be considered medically necessary in the diagnosis of temporomandibular joint (TMJ) dysfunction: - Diagnostic x-ray, tomograms, and arthrograms; - Computed tomography (CT) scan or magnetic resonance imaging (MRI) (in general, CT scans and MRIs are reserved for presurgical evaluations); - Cephalograms (x-rays of jaws and skull); - Pantograms (x-rays of maxilla and mandible). When a service is considered investigational • The following diagnostic procedures are considered investigational in the diagnosis of TMJ dysfunction: - Electromyography (EMG), including surface EMG; Page 1 of 17 Medical Policy Number: UM.SURG.07 - Kinesiography; - Thermography; - Neuromuscular junction testing; - Somatosensory testing; - Transcranial or lateral -
Applied Anatomy of the Temporomandibular Joint
Applied anatomy of the temporomandibular joint CHAPTER CONTENTS process which, together with the temporal process of the zygo- Bones . e198 matic bone, forms the zygomatic arch (Fig. 1). The midline fusion of the left and right mandibular bodies Joint .capsule .and .ligaments . e198 provides a connection between the two temporomandibular Intra-articular .meniscus . e198 joints, so that movement in one joint always influences the opposite one. Nociceptive .innervation . e199 Muscles .and .tendons . e199 Joint capsule and ligaments Biomechanical .aspects . e200 Forward movement of the mandible . e200 The joint capsule is wide and loose on the upper aspect around Opening and closing the mouth. e200 the mandibular fossa. Distally, it diminishes in a funnel shaped Grinding movements . e200 manner to become attached to the mandibular neck (Fig. 2). Nerves .and .blood .vessels . e200 Its laxity prevents rupture even after dislocation. Laterally and medially, a local reinforcement of the joint capsule is found. The lateral collateral ligament courses from The temporomandibular joint (TMJ) is sited at the base of the the zygomatic arch obliquely downwards and backwards skull and formed by parts of the mandible and the temporal towards the posterior rim of the mandibular neck, lateral to bone, separated by an intra-articular meniscus. It is a synovial the outer aspect of the capsule. At its posterior aspect, it is in joint capable of both hinge (rotation) and sliding (translatory) close relation to the joint capsule and prevents the joint from movements. Like other synovial joints, it may be affected by opening widely. Medially, the joint capsule is locally reinforced internal derangement, inflammatory arthritis, arthrosis, and by the medial collateral ligament. -
Innervation of the Temporomandibular Joint Can Be Discussed It Is Necessary First to Describe Its Embryology, Gfoss Anatomy and Microscopic Appe¿Ìrance
à8.ì 'R? INNERVATION OF THE TEMPOROMAI\DIBULAR J AN EXPERIMENTAL AMMAL MODEL USING AUSTRALIAN MERINO STIEEP ABDOLGHAFAR TAHMASEBI-SARVESTANI' B. Sc, M. Sc Thesis submitted for the degree of DOCTOR OF PHILOSOPHY In The Department of Anatomical Sciences The University of Adelaide (Faculty of Medicine)' Adelaide, South Australia, 5005 April, L997 tfüs tñesisis [elicatelø nl wtfe Aggñleñ ø¡tlour g4.arzi"e tfr.re e c friûfren Ía fiera ñ, fo zic ñ atú fi l-1 ACKNOWLEDGMENTS I am greatly indebted to my supervisors Dr. Ray Tedman and Professor Alastair Goss who first inrroduced me to this freld of study and providing me with the opportunity to carry out this work. I wish to thank them for their constant interest and guidance throughout the course of this study. I am also indebted to the scholarship committee of the Shiraz Medical Science University and Ministry of Health and Medical Education, Iran for gânting me a 4 year scholarship to study at the Universiry of Adelaide. I thank professor Goss and the Japanese Surgical Research team for their expertise in surgical animal models, and Professor July Polak and Dr Mika Hukkanen, Royal postgraduate Medical School London University for their expertise in immunohistochemistry and for providing some of the antisera used in the neuropeptide studies. I would also like to thank Professor Ian Gibbins, Department of Anatomy and Histology of the Flinders Medical Centre for, without the use of his laboratories, materials, and expertise, the double and triple labelling parts of the immunocytochemical work would not have occurred. I also orwe many thanks to Susan Matthew, a senior laboratory officer for her skilful technical assistance in double and triple immunocytochemistry. -
Diagnosis and Treatment of Temporomandibular Disorders ROBERT L
Diagnosis and Treatment of Temporomandibular Disorders ROBERT L. GAUER, MD, and MICHAEL J. SEMIDEY, DMD, Womack Army Medical Center, Fort Bragg, North Carolina Temporomandibular disorders (TMD) are a heterogeneous group of musculoskeletal and neuromuscular conditions involving the temporomandibular joint complex, and surrounding musculature and osseous components. TMD affects up to 15% of adults, with a peak incidence at 20 to 40 years of age. TMD is classified asintra-articular or extra- articular. Common symptoms include jaw pain or dysfunction, earache, headache, and facial pain. The etiology of TMD is multifactorial and includes biologic, environmental, social, emotional, and cognitive triggers. Diagnosis is most often based on history and physical examination. Diagnostic imaging may be beneficial when malocclusion or intra-articular abnormalities are suspected. Most patients improve with a combination of noninvasive therapies, including patient education, self-care, cognitive behavior therapy, pharmacotherapy, physical therapy, and occlusal devices. Nonsteroidal anti-inflammatory drugs and muscle relaxants are recommended initially, and benzodiazepines or antidepressants may be added for chronic cases. Referral to an oral and maxillofacial surgeon is indicated for refrac- tory cases. (Am Fam Physician. 2015;91(6):378-386. Copyright © 2015 American Academy of Family Physicians.) More online he temporomandibular joint (TMJ) emotional, and cognitive triggers. Factors at http://www. is formed by the mandibular con- consistently associated with TMD include aafp.org/afp. dyle inserting into the mandibular other pain conditions (e.g., chronic head- CME This clinical content fossa of the temporal bone. Muscles aches), fibromyalgia, autoimmune disor- conforms to AAFP criteria Tof mastication are primarily responsible for ders, sleep apnea, and psychiatric illness.1,3 for continuing medical education (CME). -
CIHI - Canadian Institute for Health Information CEDIS - the Canadian Emergency Department Information System Committee
CIHI - Canadian Institute for Health Information CEDIS - The Canadian Emergency Department Information System Committee ICIS - Institut canadien d'information sur la santé SIGDUC - le comité des systèmes d'informations de gestion des départements d'urgence canadiens Bernard Unger1, Marc Afilalo1, Jean François Boivin2, Michael Bullard3, Eric Grafstein4, Michael Schull5, Eddy Lang1, Antoinette Colacone1, Nathalie Soucy1, Xiaoqing Xue1, Eli Segal3 1 Emergency Multidisciplinary Research Unit, SMBD-Jewish General Hospital, McGill University, Montreal 2 Centre for Clinical Epidemiology and Community Studies, SMBD-Jewish General Hospital, McGill University, Montreal 3 University of Alberta Hospital, University of Alberta, Edmonton, Alberta 4 St. Paul's Hospital, Vancouver, University of British Columbia, Vancouver, British Columbia 5 Sunnybrook Health Sciences Centre and Institute for Clinical Evaluative Sciences, University of Toronto, Toronto, Ontario with/avec Canadian Institute for Health Information - Classifications & Terminologies and Clinical Administrative Databases (CAD) teams Institut canadien d'information sur la santé - les équipes de classifications & terminologies & des bases de données clinico-administratives (BDCA) Alberta Children's Hospital: Belanger F, Millar K BC Children's Hospital: Clarke M, Colbourne M, Haughton D, Hung G, Whitehouse S Canadian Pediatric Society Cape Breton Regional Hospital: Currie T Children's Hospital of Eastern Ontario: Farion K CH de l'Université de Montréal: Desaulniers P, Boulet M, Charbonneau L, Laurens JP, Jourdenais E CH de l'Université de Québec-Pavillon CHUL: Bernier D, Germain V, Guimont C, Nazair P, Turgeon R Colchester Regional Hospital: Howlett M Credit Valley Hospital, Mississauga: Humniski AM, Letovsky E, Scampoli N CSSS Gatineau: Folot MH, Forest G, Michaud MN, Pham Dinh M, Sibille P Foothills Medical Centre, Calgary: Wertzler B Greater Niagara General Hospital: Turineck D Grey Nuns Comm. -
Diagnosing and Treating Trigeminal Neuralgia in General Dentistry
general practice feature Chasing Pain Diagnosing and Treating Trigeminal Neuralgia in General Dentistry by Steven Olmos, DDS, DABCP, DABCDSM, DABDSM, DAAPM, FAAOP, FAACP, FICCMO, FADI, FIAO As dentists, we know quite a bit about tooth and gum pain, but when it comes to chronic facial pain and neuropathic pain, our dental school education leaves us unprepared. The objective of this article is to explain the differences between men and women with chronic orofacial pain and the relationship to proper functional breathing, using a case study as demonstration. 34 JANUARY 2016 // dentaltown.com general practice feature the United States, nearly half research published in Chest 2015 demonstrates that of all adults lived with chronic respiratory-effort-related arousal may be the most pain in 2011. Of 353,000 adults likely cause (nasal obstruction or mouth breath- 11 aged 18 years or older who were ing). Rising C02 (hypercapnia) in a patient with a surveyed by Gallup-Health- sleep-breathing disorder (including mouth breath- ways, 47 percent reported having at least one of ing) specifically stimulates the superficial masseter three types of chronic pain: neck or back pain, muscles to contract.12 knee or leg pain, or recurring pain.2 Identifying the structural area of obstruction A study published in The Journal of the Amer- (Four Points of Obstruction; Fig. 1) of the air- ican Dental Association October 2015 stated: way will insure the most effective treatment for a “One in six patients visiting a general dentist had sleep-breathing disorder and effectively reduce the experienced orofacial pain during the last year. -
MRI-Based Assessment of Masticatory Muscle Changes in TMD Patients After Whiplash Injury
Journal of Clinical Medicine Article MRI-Based Assessment of Masticatory Muscle Changes in TMD Patients after Whiplash Injury Yeon-Hee Lee 1,* , Kyung Mi Lee 2 and Q-Schick Auh 1 1 Department of Orofacial Pain and Oral Medicine, Kyung Hee University Dental Hospital, #613 Hoegi-dong, Dongdaemun-gu, Seoul 02447, Korea; [email protected] 2 Department of Radiology, Kyung Hee University College of Medicine, Kyung Hee University Hospital, #26 Kyunghee-daero, Dongdaemun-gu, Seoul 02447, Korea; [email protected] * Correspondence: [email protected]; Tel.: +82-2-958-9409; Fax: +82-2-968-0588 Abstract: Objective: to investigate the change in volume and signal in the masticatory muscles and temporomandibular joint (TMJ) of patients with temporomandibular disorder (TMD) after whiplash injury, based on magnetic resonance imaging (MRI), and to correlate them with other clinical parameters. Methods: ninety patients (64 women, 26 men; mean age: 39.36 ± 15.40 years), including 45 patients with symptoms of TMD after whiplash injury (wTMD), and 45 age- and sex- matched controls with TMD due to idiopathic causes (iTMD) were included. TMD was diagnosed using the study diagnostic criteria for TMD Axis I, and MRI findings of the TMJ and masticatory muscles were investigated. To evaluate the severity of TMD pain and muscle tenderness, we used a visual analog scale (VAS), palpation index (PI), and neck PI. Results: TMD indexes, including VAS, PI, and neck PI were significantly higher in the wTMD group. In the wTMD group, muscle tenderness was highest in the masseter muscle (71.1%), and muscle tenderness in the temporalis (60.0%), lateral pterygoid muscle (LPM) (22.2%), and medial pterygoid muscle (15.6%) was significantly more frequent than that in the iTMD group (all p < 0.05). -
Orofacial Pain Caused by Trigeminal Neuralgia And/Or Temporomandibular Joint Disorder
PERIODICUM BIOLOGORUM UDC 57:61 VOL. 115, No 2, 185–189, 2013 CODEN PDBIAD ISSN 0031-5362 Original scientific paper Orofacial pain caused by trigeminal neuralgia and/or temporomandibular joint disorder Abstract TOMISLAV BADEL1 IVANA SAVI] PAVI^IN2 Background and Purpose: The purpose was to evaluate an accurate VANJA BA[I] KES3 method of differentiating between temporomandibular joint (TMJ) dis- IRIS ZAVOREO3 4 order and trigeminal neuralgia (TN) in the sample of patients from a DIJANA ZADRAVEC subspecialist dental practice. JOSIPA KERN5 Patients and Methods: Patients (n=239, mean age 39.3 years, 83.3% 1 Department of Removable Prosthodontics, female) were examined for clinical symptoms and signs of orofacial pain of School of Dental Medicine, non-dental origin. The study included 12 female patients (group G-1; mean University of Zagreb, Gunduli}eva 5, 10000 Zagreb, Croatia age 60.3 years) with determined co-morbidity of TMJ disorder and TN, and 17 patients (group G-2; mean age 53.8 years, 64.7% female) with only TN 2Department of Dental Anthropology, confirmed and the TMJ disorder ruled out. The TMJ diagnosis by means of School of Dental Medicine magnetic resonance imaging (MRI) was confirmed. Pain intensity was University of Zagreb, Gunduli}eva 5, 10000 Zagreb, Croatia rated on a visual-analogue scale (VAS with range 0–10) and maximal mouth opening capacity (mm) measured by gauge. 3 Department of Neurology, Clinical Hospital Results: TMJ pain on the VAS scale for G-1 patients amounted to 6.91. Centre “Sisters of Charity”University of Zagreb, Vinogradska cesta 29, TN related pain symptoms on the VAS scale for G-1 patients amounted to 10000 Zagreb, Croatia 9.0±1.6 and for G-2 patients 8.1±2.7. -
Workcover NSW 2012 Claims Estimation
Minimum Weekly Compensation Estimate for Certain Injuries Extract from Workcover NSW Claims Estimation Manual - Dec 2012 Minimum Bodily Location And Injury Type Estimate GROUP 1: HEAD (includes cranium, eye, ear, mouth, nose and face) Fracture of skull (without brain injury) 6 weeks Fracture of jaw (without dislocation) 6 weeks Fracture-dislocation of jaw 12 weeks Concussion 1 week Serious head injuries (including closed/open head and brain injuries, severe facial Refer to Chapter J injuries involving face, nose and/or ear) Eye Major burn/thermal injury 26 weeks Moderate thermal or chemical burn 6 weeks Foreign body (corneal) and abrasions 2 weeks Foreign body (intraocular) 6 weeks Conjunctivitis/chemical irritation 1 week Contusions/bruising 1 week Retinal detachment 6 weeks Ear Perforated ear drum 2 weeks GROUP 2: NECK Whiplash associated disorder (WAD) without radicular pain 4 weeks Whiplash associated disorder (WAD) with radicular pain 12 weeks Contusion/bruising/sprains 4 weeks Fracture: to vertebral body 12 weeks to spinous or transverse process 6 weeks Fracture – dislocation 26 weeks Fracture with spinal cord injury Refer to Chapter J/Q GROUP 3: TRUNK (includes upper/lower back, chest, abdomen and pelvic region) Acute or recurrent back pain (non-radicular) 4 weeks Radicular back pain 12 weeks Fracture: of vertebral body 12 weeks of transverse or spinous process 6 weeks of sacrum 4 weeks of coccyx 4 weeks Contusion/bruising (upper/lower back) 4 weeks Chest/thorax: Closed rib fracture 4 weeks Fracture with complications (eg: pneumothorax) -
Giant Cell Arteritis Misdiagnosed As Temporomandibular Disorder: a Case Report and Review of the Literature
360_Reiter.qxp 10/14/09 3:17 PM Page 360 Giant Cell Arteritis Misdiagnosed as Temporomandibular Disorder: A Case Report and Review of the Literature Shoshana Reiter, DMD Giant cell arteritis (GCA) is a systemic vasculitis involving medium Teacher and large-sized arteries, most commonly the extracranial branches Department of Oral Rehabilitation of the carotid artery. Early diagnosis and treatment are essential to avoid severe complications. This article reports on a GCA case Ephraim Winocur, DMD and discusses how the orofacial manifestations of GCA can lead to Lecturer misdiagnosis of GCA as temporomandibular disorder. GCA Department of Oral Rehabilitation should be included in the differential diagnosis of orofacial pain in Carole Goldsmith, DMD the elderly based on the knowledge of related signs and symptoms, Instructor mainly jaw claudication, hard end-feel limitation of range of Department of Oral Rehabilitation motion, and temporal headache. J OROFAC PAIN 2009;23:360–365 Alona Emodi-Perlman, DMD Key words: Giant cell arteritis, jaw claudication, Teacher temporomandibular disorders, trismus Department of Oral Rehabilitation Meir Gorsky, DMD Professor Department of Oral Pathology and Oral iant cell arteritis (GCA) is a systemic vasculitis involving Medicine the large and medium-sized vessels, particularly the extracranial branches of the carotid artery. It is more com- The Maurice and Gabriela Goldschleger G School of Dental Medicine mon in women (M:F ratio 2:5) and usually affects patients older 1 Tel Aviv University, Israel than 50 years with an increased risk with age. The highest preva- lence of GCA has been reported in Scandinavian populations and Correspondence to: in those with a strong Scandinavian ethnic background.2 Dr. -
Anatomic Relationship Between Trigeminal Nerve and Temporomandibular Joint
Eur opean Rev iew for Med ical and Pharmacol ogical Sci ences 2008; 12: 15-18 Anatomic relationship between trigeminal nerve and temporomandibular joint F. PAPARO, F.M.G. FATONE, V. RAMIERI, P. CASCONE Cattedra di Chirurgia Maxillo-Facciale, Università degli Studi “La Sapienza” – Roma Abstract. – Temporomandibular joint ship with TMD. Nevertheless, other common disorders (TMD) is a collective term used to complains with uncertain pathogenesis are of - describe pathologic conditions involving tem - ten associated with TMD, such as earache, poromandibular joint (TMJ), masticatory mus - cles and associated structures. headache tinnitus and atypical trigeminal 2,3 Common related complaints include local symptoms . pain, limited mouth opening and TMJ noises Particularly, TMD trigeminal related symp - whereas symptoms often associated to TMD toms are complex, difficult to treat and have a with debated pathogenesis enclose earache, great impact on everyday life of the affected pa - headaches, tinnitus and trigeminal-like symp - tients thus suggesting a compound multimodal toms such as atypical orofacial pain. and multidisciplinary treatment. In particular, TMD trigeminal associated 4 symptoms are intricate, difficult to treat and James B. Costen , in 1934, was the first to exert a great impact on everyday life of the pa - depict a syndrome related to TMD in enclosing tients thus invoking a complex multidiscipli - all toghtether signs and symptoms such as per - nary treatment. ceived hearing loss, stuffy sensation in the ears, In this paper, the authors analyze the pain in the ear region, tinnitus, dizziness, si - anatomic and topographic relationships be - nusitis-like pain, burning sensation in the throat tween the mandibular branch of the trigeminal nerve and the medial aspect of the TMJ cap - and tongue, headache, trismus as well as tran - sule in 8 fresh adult cadavers thus resuming a sient paresthesia in the region of the mandibu - pathologic relationship between atypical lar nerve. -
Diagnostic Treatment Protocols Regulation Interpretative Guide
Alberta Office of the Superintendent of Insurance Diagnostic Treatment Protocols Regulation Interpretative Guide Last updated: August 23, 2018 Alberta Treasury Board and Finance Superintendent of Insurance Financial Sector Regulation and Policy (FSRP) Room 402, Terrace Building 9515 - 107 Street Edmonton, Alberta, T5K 2C3 Phone 780-427-8322 • Fax 780-420-0752 For toll-free service in Alberta, call 310-0000, then enter the number. For more information, visit insurance.alberta.ca T able of Contents Table of Contents ........................................................................ 1 General Information .................................................................... 3 Introduction ..................................................................................................... 3 Scope of the Diagnosis and Treatment Protocols ......................................... 4 Definitions .................................................................................... 5 For the purposes of this Guide ....................................................................... 5 Claims and Billing ....................................................................... 6 Privacy Statement ....................................................................... 9 For Health Care Practitioners ......................................................................... 9 For More Information ..................................................................................... 9 Sprains and Strains ..................................................................