Sleep Self-Care
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Participant Guide
Participant Guide Get Enough Sleep Session Focus Getting enough sleep can help you prevent or delay type 2 diabetes. This session we will talk about: z Why sleep matters z Some challenges of getting enough sleep and ways to cope with them You will also make a new action plan! Tips: ✓ Go to bed and get up at the same time each day. This helps your body get on a schedule. ✓ Follow a bedtime routine that helps you wind down. Participant Guide: Get Enough Sleep 2 Jenny’s Story Jenny is at risk for type 2 diabetes. Her doctor asks her if she gets at least 7 hours of sleep each night. Jenny laughs. “Are you serious?” she asks. “I’m lucky if I get 5 hours.” Jenny usually doesn’t have much trouble falling asleep. But she often has to use the bathroom in the early morning. This gets her thinking about all the things she needs to do the next day. Plus, her husband’s breathing is loud. Both of these things make it hard for Jenny to fall back to sleep. She often lies awake for hours. These days, Jenny drinks less water and avoids caffeine in the evening. She makes a list of things to do the next day. Then she sets it aside. Jenny rarely needs to get up to use the bathroom during the night. If she does, she breathes deeply to help her get back to sleep. She also runs a fan to cover up the sound of her husband’s breathing. Jenny is closer to getting 7 hours of sleep a night. -
Sleep Apnea Sleep Apnea
Health and Safety Guidelines 1 Sleep Apnea Sleep Apnea Normally while sleeping, air is moved at a regular rhythm through the throat and in and out the lungs. When someone has sleep apnea, air movement becomes decreased or stops altogether. Sleep apnea can affect long term health. Types of sleep apnea: 1. Obstructive sleep apnea (narrowing or closure of the throat during sleep) which is seen most commonly, and, 2. Central sleep apnea (the brain is causing a change in breathing control and rhythm) Obstructive sleep apnea (OSA) About 25% of all adults are at risk for sleep apnea of some degree. Men are more commonly affected than women. Other risk factors include: 1. Middle and older age 2. Being overweight 3. Having a small mouth and throat Down syndrome Because of soft tissue and skeletal alterations that lead to upper airway obstruction, people with Down syndrome have an increased risk of obstructive sleep apnea. Statistics show that obstructive sleep apnea occurs in at least 30 to 75% of people with Down syndrome, including those who are not obese. In over half of person’s with Down syndrome whose parents reported no sleep problems, sleep studies showed abnormal results. Sleep apnea causing lowered oxygen levels often contributes to mental impairment. How does obstructive sleep apnea occur? The throat is surrounded by muscles that are active controlling the airway during talking, swallowing and breathing. During sleep, these muscles are much less active. They can fall back into the throat, causing narrowing. In most people this doesn’t affect breathing. However in some the narrowing can cause snoring. -
The Effects of Time of Day and Chronotype on Cognitive and Physical Performance in Healthy Volunteers Facer-Childs, Elise; Boiling, Sophie; Balanos, George
CORE Metadata, citation and similar papers at core.ac.uk Provided by University of Birmingham Research Portal The effects of time of day and chronotype on cognitive and physical performance in healthy volunteers Facer-Childs, Elise; Boiling, Sophie; Balanos, George DOI: 10.1186/s40798-018-0162-z License: Creative Commons: Attribution (CC BY) Document Version Publisher's PDF, also known as Version of record Citation for published version (Harvard): Facer-Childs, E, Boiling, S & Balanos, G 2018, 'The effects of time of day and chronotype on cognitive and physical performance in healthy volunteers', Sports Medicine - Open, vol. 4, no. 1, 47. https://doi.org/10.1186/s40798-018-0162-z Link to publication on Research at Birmingham portal Publisher Rights Statement: Checked for eligibility 09/11/2018 Sports Medicine - Open20184:47 https://doi.org/10.1186/s40798-018-0162-z General rights Unless a licence is specified above, all rights (including copyright and moral rights) in this document are retained by the authors and/or the copyright holders. The express permission of the copyright holder must be obtained for any use of this material other than for purposes permitted by law. •Users may freely distribute the URL that is used to identify this publication. •Users may download and/or print one copy of the publication from the University of Birmingham research portal for the purpose of private study or non-commercial research. •User may use extracts from the document in line with the concept of ‘fair dealing’ under the Copyright, Designs and Patents Act 1988 (?) •Users may not further distribute the material nor use it for the purposes of commercial gain. -
INSOMNIA Authors: Dr
INSOMNIA Authors: Dr. Ashna Bowry, Dr. Karl Iglar Created Dec 2010 Overview Insomnia is commonly reported due to daytime fatigue, physical discomfort and psychological distress1. Although highly prevalent and can lead to physical and mental health problems as well as social, occupational and economic repercussions, poor sleep quality is often overlooked. The prevalence of insomnia is estimated to be 13.4% in Canada and 6-10% worldwide. 2 Definitions Acute insomnia: new onset of difficulty initiating or maintaining sleep, with the presence of an identifiable trigger lasting < 4 wks. duration;Chronic insomnia: symptoms lasting >4 weeks; Primary insomnia: a conditioned state of hyperarousal that inhibits the sleep process. Not usually associated with a comorbidity and often lasts < 1 month; Secondary insomnia: associated with primary sleep disorders (C: Circadian rhythm, A: sleep apnea, L: restless legs), medications, medical or psychiatric conditions (mood disorders and substance abuse) – see table 1; Sleep-onset latency: the time it takes to fall asleep. Normal is < 30 minutes8 Diagnostic Considerations Consider diagnosis of insomnia for patients with difficulty falling asleep, staying asleep or non-refreshing sleep in a person who has the opportunity to sleep 7-8 hours per night. Clinically relevant if effects daytime functionality (daytime dysfunction, fatigue, poor concentration and irritability.) 1,3 Sleep history is essential and should include: recent life changes, alcohol/drug use, sleep and wakefulness patterns, effect on functioning. -
Towards a Passive BCI to Induce Lucid Dream Morgane Hamon, Emma Chabani, Philippe Giraudeau
Towards a Passive BCI to Induce Lucid Dream Morgane Hamon, Emma Chabani, Philippe Giraudeau To cite this version: Morgane Hamon, Emma Chabani, Philippe Giraudeau. Towards a Passive BCI to Induce Lucid Dream. Journée Jeunes Chercheurs en Interfaces Cerveau-Ordinateur et Neurofeedback 2019 (JJC- ICON ’19), Mar 2019, Villeneuve d’Ascq, France. hal-02108903 HAL Id: hal-02108903 https://hal.archives-ouvertes.fr/hal-02108903 Submitted on 29 Apr 2019 HAL is a multi-disciplinary open access L’archive ouverte pluridisciplinaire HAL, est archive for the deposit and dissemination of sci- destinée au dépôt et à la diffusion de documents entific research documents, whether they are pub- scientifiques de niveau recherche, publiés ou non, lished or not. The documents may come from émanant des établissements d’enseignement et de teaching and research institutions in France or recherche français ou étrangers, des laboratoires abroad, or from public or private research centers. publics ou privés. TOWARDS A PASSIVE BCI TO INDUCE LUCID DREAM JJC-ICON ’19 Morgane Hamon Emma Chabani Philippe Giraudeau Ullo Institut du Cerveau et de la Moelle épinière Inria La Rochelle, France Paris, France Bordeaux, France [email protected] [email protected] [email protected] April 29, 2019 ABSTRACT Lucid dreaming (LD) is a phenomenon during which the person is aware that he/she dreaming and is able to control the dream content. Studies have shown that only 20% of people can experience lucid dreams on a regular basis. However, LD frequency can be increased through induction techniques. External stimulation technique relies on the ability to integrate external information into the dream content. -
MEDICAL CHALLENGES: EFFECTS of TRAVEL FATIGUE and JET LAG
MEDICAL CHALLENGES: EFFECTS of TRAVEL FATIGUE and JET LAG Prof Christa Janse van Rensburg MBChB, MSc, MMed, MD FACSM, FFIMS Sports Medicine & SEMLI, University of Pretoria Consultant Rheumatologist, Eugene Marais Hospital South Africa IOC Advanced Team Physician Course TOKYO, 01-03 JULY 2019 INTRODUCTION @ChristaJVR #IOCATPC2019 No disclosures A) CIRCADIAN SYSTEM ~24h self-sustained variation in physical, mental & behavioural function • Functions endogenously: ✓autonomous ✓rhythm maintained in absence of external cues • Circadian timing: Master Clock: Suprachiasmatic nucleus (SCN) in HT Peripheral Clocks: in mostly every cell of all tissue REGULATION • Complex series of interlocking transcriptional feedback loops (clock genes that code for clock proteins) • Various stimuli = zeitgebers or "time givers” ✓ Light/dark cycle most NB advance or delay phase • Rhythmical change phase response curve (PRC) - Amount of phase shift depends on zeitgeber: - timing - intensity - duration - frequency - Synergistic or antagonistic action of zeitgebers i.e. interact in ‘push’ or ‘pull’ on circadian rhythm phase • Inter-individual variations - Chronotype differences (morning lark vs. night owl) The Nobel Prize in Physiology or Medicine 2017 - Genetic variances of circadian-related phenotypes was awarded jointly to Rosbash M, Young MW, Hall JC for their pioneering discovery that our [Period (PER1, PER2, PER3); Cryptochrome (CRY1,CRY2) circadian clocks are regulated by a small group genes] of genes working at individual cell level. CIRCADIAN RHYTHM DESYNCHRONISATION -
Obstructive Sleep Apnea in Adults? NORMAL AIRWAY OBSTRUCTED AIRWAY
American Thoracic Society PATIENT EDUCATION | INFORMATION SERIES What Is Obstructive Sleep Apnea in Adults? NORMAL AIRWAY OBSTRUCTED AIRWAY Obstructive sleep apnea (OSA) is a common problem that affects a person’s breathing during sleep. A person with OSA has times during sleep in which air cannot flow normally into the lungs. The block in CPAP DEVICE airflow (obstruction) is usually caused by the collapse of the soft tissues in the back of the throat (upper airway) and tongue during sleep. Apnea means not breathing. In OSA, you may stop ■■ Gasping breathing for short periods of time. Even when you are or choking trying to breathe, there may be little or no airflow into sounds. the lungs. These pauses in airflow (obstructive apneas) ■■ Breathing pauses observed by someone watching can occur off and on during sleep, and cause you to you sleep. wake up from a sound sleep. Frequent apneas can cause ■■ Sudden or jerky body movements. many problems. With time, if not treated, serious health ■■ Restless tossing and turning. problems may develop. ■■ Frequent awakenings from sleep. OSA is more common in men, women after menopause CLIP AND COPY AND CLIP Common symptoms you may have while awake: and people who are over the age of 65. OSA can also ■■ Wake up feeling like you have not had enough sleep, occur in children. Also see ATS Patient Information Series even after sleeping many hours. fact sheet on OSA in Children. People who are at higher ■■ Morning headache. risk of developing sleep apnea include those with: ■■ Dry or sore throat in the morning from breathing ■■ enlarged tonsils and/or adenoids through your mouth during sleep. -
Brain Computer Interface for Sleep Apnea Detection
Special Issue - 2020 International Journal of Engineering Research & Technology (IJERT) ISSN: 2278-0181 NCAIT - 2020 Conference Proceedings Brain Computer Interface for Sleep Apnea Detection Poorvitha H R1, Aishwarya K Gowda2, Kavya K B2, Nayana R2 Vivekananda Institute of Technology Gudimavu, Kumbalagodu Post, Kengeri Hobli, Bengaluru – 560 074 Abstract - A brain-computer interface (BCI) is the varied phases of sleep and may be a crucial step in a interface method that someone to deliver instructions and shot to assist doctor within the designation and medical messages to a automated gadget, via way of means of care of connected sleep obstruction. using person mind activity. An electroencephalogram (EEG), mounted BCI became related with a synthetic gadget to command a home automation application. It provides an alternative to built-in interface and control. It is a digital surroundings shape that enters the body’s everyday prepared pathways, which might be neuromuscular output passage. Sleep apnea is personification of sickness which occurs during sleep, which impacts the human fitness via way of means of blockage in respiratory for positive period of time. The prognosis is critical that's viable via way of means of the perceiving of apnea episodes the use of electroencephalogram (EEG) reports. EEG is an adjustable decomposition for the identifying of apnea events using EEG signals. EEG plays an essential role in identifying the sleep apnea by recognizing and producing the brain neurons activities. The EEG sign dataset is filtered, the purified EEG sign is subjected for sub-band separation and 5 frequency bands inclusive of Gamma, Beta, Alpha, Theta, and Delta. -
What%Is%Idiopathic%Hypersomnia?%
What%is%Idiopathic%Hypersomnia?% Idiopathic*hypersomnia*is*sleeping*too*much*(hypersomnia)*without*an*obvious*cause.*It*is*different* from*narcolepsy,*because*idiopathic*hypersomnia*does*not*involve*suddenly*falling*asleep*or*losing* muscle*control*due*to*strong*emotions*(cataplexy).* Causes* The*usual*approach*is*to*consider*other*potential*causes*of*excessive*daytime*sleepiness.* Other*sleep*disorders*that*may*cause*daytime*sleepiness*include:* •Isolated*sleep*paralysis* •Narcolepsy* •Obstructive*sleep*apnea* •Restless*leg*syndrome* Other*causes*of*excessive*sleepiness*include:* •Atypical*depression* •Certain*medications* •Drug*and*alcohol*use* •Low*thyroid*function*(hypothyroidism)* •Previous*head*injury* Symptoms* Symptoms*often*develop*slowly*during*adolescence*or*young*adulthood.*They*include:* •Daytime*naps*that*do*not*relieve*drowsiness* •Difficulty*waking*from*a*long*sleep*KK*may*feel*confused*or*disoriented* •Increased*need*for*sleep*during*the*day*KK*even*while*at*work,*or*during*a*meal*or*conversation* •Increased*sleep*time*KK*up*to*14*K*18*hours*per*day* Other*symptoms*may*include:* •Anxiety* •Feeling*irritated* •Loss*of*appetite* •Low*energy* •Restlessness* •Slow*thinking*or*speech* •Trouble*remembering* Cataplexy*KK*suddenly*falling*asleep*or*losing*muscle*control*KK*which*is*part*of*narcolepsy,*is*NOT*a* symptom*of*idiopathic*hypersomnia.* Exams*and*Tests* The*health*care*provider*will*take*a*detailed*sleep*history.*Tests*may*include:* •MultipleKsleep*latency*test* •Sleep*study*(polysomnography,*to*identify*other*sleep*disorders)* -
Insomnia-1111 30/11/11 4:37 PM Page 1
SHF-Insomnia-1111 30/11/11 4:37 PM Page 1 Insomnia Important Things to Know About Insomnia • Around 1 in 3 people have at least mild insomnia. • Many poor sleepers have developed poor sleep habits. • For specialist help, cognitive behavioural therapy for insomnia is best • In the long run, taking sleeping pills isn’t effective. • There are sleep specialists, clinics and on-line programs that can help. What is insomnia? How common is insomnia? Insomnia is said to be present when you regularly find it Most people have experience insomnia symptoms at hard to fall asleep or stay asleep. It has several patterns. some time of their lives. At any given time around 10% You may have trouble getting to sleep initially. Or even if of people have at least mild insomnia. you can fall asleep, you might not be able to stay asleep for as long as you would like. Also you may wake up Who is at risk? during the night and not be able to go back to sleep for a long time. Many people have two of the above problems, Older people with poor health have a higher risk. Also or even all three. Because of these, you might feel tired women have twice the rates compared to men. This may during the day. be related to higher rates of anxiety and depression, which can be associated with insomnia. Shift workers What causes insomnia? have a higher risk too. Insomnia has many causes which can include: How does it affect people? • Some medicines and drugs, e.g. -
Non-24-Hour Sleep-Wake Disorder Revisited – a Case Study
CASE REPORT published: 29 February 2016 doi: 10.3389/fneur.2016.00017 Non-24-Hour Sleep-Wake Disorder Revisited – a Case study Corrado Garbazza1,2† , Vivien Bromundt3† , Anne Eckert2,4 , Daniel P. Brunner5 , Fides Meier2,4 , Sandra Hackethal6 and Christian Cajochen1,2* 1 Centre for Chronobiology, Psychiatric Hospital of the University of Basel, Basel, Switzerland, 2 Transfaculty Research Platform Molecular and Cognitive Neurosciences, University of Basel, Basel, Switzerland, 3 Sleep-Wake-Epilepsy-Centre, Department of Neurology, Inselspital, Bern University Hospital, Bern, Switzerland, 4 Neurobiology Laboratory for Brain Aging and Mental Health, Psychiatric Hospital of the University of Basel, Basel, Switzerland, 5 Center for Sleep Medicine, Hirslanden Clinic Zurich, Zurich, Switzerland, 6 Charité – Universitaetsmedizin Berlin, Berlin, Germany The human sleep-wake cycle is governed by two major factors: a homeostatic hourglass process (process S), which rises linearly during the day, and a circadian process C, which determines the timing of sleep in a ~24-h rhythm in accordance to the external Edited by: Ahmed S. BaHammam, light–dark (LD) cycle. While both individual processes are fairly well characterized, the King Saud University, Saudi Arabia exact nature of their interaction remains unclear. The circadian rhythm is generated by Reviewed by: the suprachiasmatic nucleus (“master clock”) of the anterior hypothalamus, through Axel Steiger, cell-autonomous feedback loops of DNA transcription and translation. While the phase Max Planck Institute of Psychiatry, Germany length (tau) of the cycle is relatively stable and genetically determined, the phase of Timo Partonen, the clock is reset by external stimuli (“zeitgebers”), the most important being the LD National Institute for Health and Welfare, Finland cycle. -
Adult NREM Parasomnias: an Update
Review Adult NREM Parasomnias: An Update Maria Hrozanova 1, Ian Morrison 2 and Renata L Riha 3,* 1 Department of Neuromedicine and Movement Science, Norwegian University of Science and Technology, N-7491 Trondheim, Norway; [email protected] 2 Department of Neurology, Ninewells Hospital and Medical School, DD1 9SY Dundee, UK; [email protected] 3 Department of Sleep Medicine, Royal Infirmary of Edinburgh, EH16 4SA Edinburgh, UK * Correspondence: [email protected] or [email protected]; Tel.: +44-013-242-3872 Received: 23 August 2018; Accepted: 15 November 2018; Published: 23 November 2018 Abstract: Our understanding of non-rapid eye movement (NREM) parasomnias has improved considerably over the last two decades, with research that characterises and explores the causes of these disorders. However, our understanding is far from complete. The aim of this paper is to provide an updated review focusing on adult NREM parasomnias and highlighting new areas in NREM parasomnia research from the recent literature. We outline the prevalence, clinical characteristics, role of onset, pathophysiology, role of predisposing, priming and precipitating factors, diagnostic criteria, treatment options and medico-legal implications of adult NREM parasomnias. Keywords: NREM parasomnias; slow-wave sleep disorders; parasomnias; adult; arousal disorders; review 1. Introduction Non-rapid eye movement (NREM) parasomnias constitute a category of sleep disorders characterised by abnormal behaviours and physiological events primarily arising from N3sleep [1–3] and occuring outside of conscious awareness. Due to their specific association with slow wave sleep (SWS), NREM parasomnias are also termed ‘SWS disorders’. Behaviours such as confusional arousals, sleepwalking, sleep eating (also called sleep-related eating disorder, or SRED), night terrors, sexualised behaviour in sleep (also called sexsomnia) and sleep-related violence are NREM parasomnias that arise from N3 sleep.