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Crush Injuries Pathophysiology and Current Treatment Michael Sahjian, RN, BSN, CFRN, CCRN, NREMT-P; Michael Frakes, APRN, CCNS, CCRN, CFRN, NREMT-P
LWW/AENJ LWWJ331-02 April 23, 2007 13:50 Char Count= 0 Advanced Emergency Nursing Journal Vol. 29, No. 2, pp. 145–150 Copyright c 2007 Wolters Kluwer Health | Lippincott Williams & Wilkins Crush Injuries Pathophysiology and Current Treatment Michael Sahjian, RN, BSN, CFRN, CCRN, NREMT-P; Michael Frakes, APRN, CCNS, CCRN, CFRN, NREMT-P Abstract Crush syndrome, or traumatic rhabdomyolysis, is an uncommon traumatic injury that can lead to mismanagement or delayed treatment. Although rhabdomyolysis can result from many causes, this article reviews the risk factors, symptoms, and best practice treatments to optimize patient outcomes, as they relate to crush injuries. Key words: crush syndrome, traumatic rhabdomyolysis RUSH SYNDROME, also known as ology, pathophysiology, diagnosis, and early traumatic rhabdomyolysis, was first re- management of crush syndrome. Cported in 1910 by German authors who described symptoms including muscle EPIDEMIOLOGY pain, weakness, and brown-colored urine in soldiers rescued after being buried in struc- Crush injuries may result in permanent dis- tural debris (Gonzalez, 2005). Crush syn- ability or death; therefore, early recognition drome was not well defined until the 1940s and aggressive treatment are necessary to when nephrologists Bywaters and Beal pro- improve outcomes. There are many known vided descriptions of victims trapped by mechanisms inducing rhabdomyolysis includ- their extremities during the London Blitz ing crush injuries, electrocution, burns, com- who presented with shock, swollen extrem- partment syndrome, and any other pathology ities, tea-colored urine, and subsequent re- that results in muscle damage. Victims of nat- nal failure (Better & Stein, 1990; Fernan- ural disasters, including earthquakes, are re- dez, Hung, Bruno, Galea, & Chiang, 2005; ported as having up to a 20% incidence of Gonzalez, 2005; Malinoski, Slater, & Mullins, crush injuries, as do 40% of those surviving to 2004). -
Thoracic Gunshot Wound: a Tanmoy Ganguly1, 1 Report of 3 Cases and Review of Sandeep Kumar Kar , Chaitali Sen1, Management Chiranjib Bhattacharya2, Manasij Mitra3
2015 iMedPub Journals Journal of Universal Surgery http://www.imedpub.com Vol. 3 No. 1:2 ISSN 2254-6758 Thoracic Gunshot Wound: A Tanmoy Ganguly1, 1 Report of 3 Cases and Review of Sandeep Kumar Kar , Chaitali Sen1, Management Chiranjib Bhattacharya2, Manasij Mitra3, 1 Department of Cardiac Anesthesiology, Abstract Institute of Postgraduate Medical Thoracic gunshot injury may have variable presentation and the treatment plan Education and Research, Kolkata, India differs. The risk of injury to heart, major blood vessels and the lungs should be 2 Department of Anesthesiology, Institute evaluated in every patient with rapid clinical examination and basic monitoring and of Postgraduate Medical Education and surgery should be considered as early as possible whenever indicated. The authors Research, Kolkata, India present three cases of thoracic gunshot injury with three different presentations, 3 Krisanganj Medical College, Institute of one with vascular injury, one with parenchymal injury and one case with fortunately Postgraduate Medical Education and no life threatening internal injury. The first case, a 52 year male patient presented Research, Kolkata, India with thoracic gunshot with hemothorax and the bullet trajectory passed very near to the vital structures without injuring them. The second case presented with 2 hours history of thoracic gunshot wound with severe hemodynamic instability. Corresponding author: Sandeep Kumar Surgical exploration revealed an arterial bleeding from within the left lung. The Kar, Assistant Professor third case presented with post gunshot open pneumothorax. All three cases managed successfully with resuscitation and thoracotomy. Preoperative on table fluoroscopy was used for localisation of bullet. [email protected] Keywords: Horacic trauma, Gunshot injury, Traumatic pneumothorax, Emergency thoracotomy, Fluoroscopy. -
Femoral Shaft Fracture Fixation and Chest Injury After Polytrauma
This is an enhanced PDF from The Journal of Bone and Joint Surgery The PDF of the article you requested follows this cover page. Femoral Shaft Fracture Fixation and Chest Injury After Polytrauma Lawrence B. Bone and Peter Giannoudis J Bone Joint Surg Am. 2011;93:311-317. doi:10.2106/JBJS.J.00334 This information is current as of January 25, 2011 Reprints and Permissions Click here to order reprints or request permission to use material from this article, or locate the article citation on jbjs.org and click on the [Reprints and Permissions] link. Publisher Information The Journal of Bone and Joint Surgery 20 Pickering Street, Needham, MA 02492-3157 www.jbjs.org 311 COPYRIGHT Ó 2011 BY THE JOURNAL OF BONE AND JOINT SURGERY,INCORPORATED Current Concepts Review Femoral Shaft Fracture Fixation and Chest Injury After Polytrauma By Lawrence B. Bone, MD, and Peter Giannoudis, MD, FRCS Thirty years ago, the standard of care for the multiply injured tients with multiple injuries, defined as an ISS of ‡18, and patient with fractures was placement of the fractured limb in a patients with essentially an isolated femoral fracture and an splint or skeletal traction, until the patient was considered stable ISS of <18. Pulmonary complications consisting of ARDS, enough to undergo surgery for fracture fixation1. This led to a pulmonary dysfunction, fat emboli, pulmonary emboli, and number of complications2, such as adult respiratory distress pneumonia were present in 38% (fourteen) of thirty-seven syndrome (ARDS), infection, pneumonia, malunion, non- patients in the late fixation/multiple injuries group and 4% union, and death, particularly when the patient had a high (two) of forty-six in the early fixation/multiple injuries group; Injury Severity Score (ISS)3. -
An Update on the Management of Severe Crush Injury to the Forearm and Hand
An Update on the Management of Severe Crush Injury to the Forearm and Hand a, Francisco del Piñal, MD, Dr. Med. * KEYWORDS Crush syndrome Hand Compartimental syndrome Free flap Hand revascularization Microsurgery Forzen hand KEY POINTS Microsurgery changes the prognosis of crush hand syndrome. Radical debridement should be followed by rigid (vascularized) bony restoration. Bringing vascularized gliding tissue allows active motion to be restored. Finally, the mangement of the chronic injury is discussed. INTRODUCTION the distal forearm, wrist, or metacarpal area and fingers separately. Severe crush injuries to the hand and fingers often carry an unavoidably bad prognosis, resulting in stiff, crooked, and painful hands or fingers. In ACUTE CRUSH TO THE DISTAL FOREARM, follow-up, osteoporosis is often times seen on ra- WRIST, AND METACARPAL AREA OF THE diographs. A shiny appearance of the skin and HAND complaints of vague pain may lead the surgeon Clinical Presentations and Pathophysiology to consider a diagnosis of reflex sympathetic dys- Two striking features after a severe crush injury are trophy,1 to offer some “explanation” of the gloomy prognosis that a crush injury predicates. Primary 1. The affected joints tend to stiffen and the or secondary amputations are the common end affected tendons tend to stick. options of treatment. 2. The undamaged structures distal to the area of In the authors’ experience, the prompt and pre- injury usually get involved. cise application of microsurgical techniques can The trauma appears to have a “contagious” ef- help alter the often dismal prognosis held by those fect that spreads distally, similar to a fire spreading suffering from severe crush injuries. -
ISR/PFC Crush Injury Clinical Practice Guideline
All articles published in the Journal of Special Operations Medicine are protected by United States copyright law and may not be reproduced, distributed, transmitted, displayed, or otherwise published without the prior written permission of Breakaway Media, LLC. Contact [email protected]. An Ongoing Series Management of Crush Syndrome Under Prolonged Field Care Thomas Walters, PhD; Douglas Powell, MD; Andrew Penny, NREMT-P; Ian Stewart, MD; Kevin Chung, MD; Sean Keenan, MD; Stacy Shackelford, MD Introduction to the Prolonged Field Care beyond the initial evaluation and treatment of casual- Prehospital Clinical Practice Guideline Series ties in a PFC operational environment. This and fu- ture CPGs are aimed at serious clinical problems seen Sean Keenan, MD less frequently (e.g., crush injury, burns) or where fur- ther advanced practice recommendations are required THIS FIRST CLINICAL PRACTICE GUIDELINE (CPG) (e.g., pain and sedation recommendations beyond was produced through a collaboration of the SOMA TCCC recommendations, traumatic brain injury). Prolonged Field Care Working Group (PFCWG) and the Joint Trauma System (JTS) at the U.S. Army Insti- We hope that this collaboration of experienced op- tute of Surgical Research (USAISR) in San Antonio. Of erational practitioners and true subject matter ex- note, this effort is the result from requests for informa- perts, operating under the guidance set forth in past tion and guidance through the PFC website (PFCare.org) JTS CPG editorial standards, will bring practical and and from the Joint Special Operations Medical Training applicable clinical recommendations to the advanced Center instructors located at Fort Bragg, North Carolina. practice first responders and Role 1 providers in the field. -
With Crush Injury Syndrome
Crush Syndrome Made Simple Malta & McConnelsville Fire Department Division of Emergency Medical Service Objectives Recognize the differences between Crush Injury and Crush Syndrome Understand the interventions performed when treating someone with Crush Syndrome Assessing the Crush Injury victim S&S of crush injuries Treatment of crush injury Malta & McConnelsville Fire Department Division of Emergency Medical Service INJURY SYNDROME • Cell Disruption/ • Systemic effects injury at the point of when muscle is impact. RELEASED from Compression • Occurs < 1 hour • Occurs after cells have been under pressure >4 hours* • Suspect Syndrome with lightening strikes Malta & McConnelsville Fire Department Division of Emergency Medical Service CRUSHING MECHANISM OF INJURY • Building and Structure Collapse • Bomb Concussions • MVAs’ and Farm Accidents • Assault with blunt weapon Malta & McConnelsville Fire Department Division of Emergency Medical Service AKA: COMPRESSION SYNDROME First described by Dr. Minami in 1940 Malta & McConnelsville Fire Department Division of Emergency Medical Service INVOLVED ANATOMY Upper Arms Upper Legs Thorax and Buttocks Malta & McConnelsville Fire Department Division of Emergency Medical Service Crush Injuries Crush injuries occur when a crushing force is applied to a body area. Sometimes they are associated with internal organ rupture, major fractures, and hemorrhagic shock. Early aggressive treatment of patients suspected of having a crush injury is crucial. Along with the severity of soft tissue damage and fractures, a major concern of a severe crush injury is the duration of the compression/entrapment. Malta & McConnelsville Fire Department Division of Emergency Medical Service Crush Injuries Prolonged compression of a body region or limb may lead to a dangerous syndrome that can become fatal. Crush Syndrome is difficult to diagnose and treat in the pre-hospital setting because of the many complex variables involved. -
Assessment, Management and Decision Making in the Treatment of Polytrauma Patients with Head Injuries
Compartment Syndrome Andrew H. Schmidt, M.D. Professor, Dept. of Orthopedic Surgery, Univ. of Minnesota Chief, Department of Orthopaedic Surgery Hennepin County Medical Center April 2016 Disclosure Information Andrew H. Schmidt, M.D. Conflicts of Commitment/ Effort Board of Directors: OTA Critical Issues Committee: AOA Editorial Board: J Knee Surgery, J Orthopaedic Trauma Medical Director, Director Clinical Research: Hennepin County Med Ctr. Disclosure of Financial Relationships Royalties: Thieme, Inc.; Smith & Nephew, Inc. Consultant: Medtronic, Inc.; DGIMed; Acumed; St. Jude Medical (spouse) Stock: Conventus Orthopaedics; Twin Star Medical; Twin Star ECS; Epien; International Spine & Orthopedic Institute, Epix Disclosure of Off-Label and/or investigative Uses I will not discuss off label use and/or investigational use in my presentation. Objectives • Review Pathophysiology of Acute Compartment Syndrome • Review Current Diagnosis and Treatment – Risk Factors – Clinical Findings – Discuss role and technique of compartment pressure monitoring. Pathophysiology of Compartment Syndrome Pressure Inflexible Fascia Injured Muscle Vascular Consequences of Elevated Intracompartment Pressure: A-V Gradient Theory Pa (High) Pv (Low) artery arteriole capillary venule vein Local Blood Pa - Pv Flow = R Matsen, 1980 Increased interstitial pressure Pa (High) Tissue ischemia artery arteriole capillary venule vein Lysis of cell walls Release of osmotically active cellular contents into interstitial fluid Increased interstitial pressure More cellular -
Approach to the Trauma Patient Will Help Reduce Errors
The Approach To Trauma Author Credentials Written by: Nicholas E. Kman, MD, The Ohio State University Updated by: Creagh Boulger, MD, and Benjamin M. Ostro, MD, The Ohio State University Last Update: March 2019 Case Study “We have a motor vehicle accident 5 minutes out per EMS report.” 47-year-old male unrestrained driver ejected 15 feet from car arrives via EMS. Vital Signs: BP: 100/40, RR: 28, HR: 110. He was initially combative at the scene but now difficult to arouse. He does not open his eyes, withdrawals only to pain, and makes gurgling sounds. EMS placed a c-collar and backboard, but could not start an IV. What do you do? Objectives Upon completion of this self-study module, you should be able to: ● Describe a focused rapid assessment of the trauma patient using an organized primary and secondary survey. ● Discuss the components of the primary survey. ● Discuss possible pathology that can occur in each domain of the primary survey and recommend treatment/stabilization measures. ● Describe how to stabilize a trauma patient and prioritize resuscitative measures. ● Discuss the secondary survey with particular attention to head/central nervous system (CNS), cervical spine, chest, abdominal, and musculoskeletal trauma. ● Discuss appropriate labs and diagnostic testing in caring for a trauma patient. ● Describe appropriate disposition of a trauma patient. Introduction Nearly 10% of all deaths in the world are caused by injury. Trauma is the number one cause of death in persons 1-50 years of age and results in significant life years lost. According to the National Trauma Data Bank, falls were the leading cause of trauma followed by motor vehicle collisions (MVCs) and firearm related injuries with an overall mortality rate of 4.39% in 2016. -
Traumatic Deceleration Injury of the Thoracic Aorta
ECHO ROUNDS Section Editor: Edmund Kenneth Kerut, M.D. Traumatic Deceleration Injury of the Thoracic Aorta Edmund Kenneth Kerut, M.D.,∗,∗∗ Glenn Kelley, M.D.,† Vivian Carina Falco, M.D.,† Ty Ovella, M.D.,‡ Lisa Diethelm, M.D.,‡ and Frederick Helmcke, M.D.† ∗Departments of Pharmacology and Physiology, Louisiana State University Health Sciences Center, New Orleans, Louisiana, ∗∗Heart Clinic of Louisiana, Marrero, Louisiana, †Division of Cardiology, Department of Medicine, Louisiana State University Health Sciences Center, New Orleans, Louisiana, and ‡Department of Radiology, Louisiana State University Health Sciences Center, New Orleans, Louisiana (ECHOCARDIOGRAPHY, Volume 22, September 2005) deceleration, trauma, transection, aorta, transesophageal echocardiography, multislice computed tomography A 42-year-old intoxicated male pedestrian to surgery, where a spiral, subtotal, subadventi- was struck by a motor vehicle and “thrown” 45 tial aortic disruption (see discussion) involving feet. The patient did not lose consciousness. He three-fourths of the circumference of the aorta had no specific localized pain, and was initially was found.1,2 The tear was successfully resected noted to have only minor limb abrasions. He and replaced with a composite thoracic graft. was then admitted to the trauma center for ob- Blunt traumatic chest injury (deceleration in- servation. A screening CT was performed using jury) most often occurs from a steering wheel in- a16row multidetector CT scanner. It revealed jury or from a fall from a height. This may result a mediastinal hematoma, and also an aortic in myocardial contusion, traumatic ventricular pseudoaneurysm with an intraluminal defect septal defect, tricuspid or mitral valve trauma, at the level of the aortic isthmus (Fig. -
Crush Injury Management
Crush Injury Management In the Underground Environment Background • 1910 - Messina Earthquake • WW2 - Air Raid Shelters fell on people crushing limbs - First time called Crush Syndrome • Granville Rail Disaster - Sydney Australia • Chain Valley Bay Colliery fatality 2011 What is it? Definition: Crush Injury • Injury that occurs because of pressure from a heavy object onto a body part • Squeezing of a body part between two objects Definition: Crush Syndrome The shock-like state following release of a limb or limbs, trunk and pelvis after a prolonged period of compression Crush Syndrome Basic Science • Muscle groups are covered by a tough membrane (fascia) that does not readily expand • Damage to these muscle groups cause swelling and/or bleeding; due to inelasticity of fascia, swelling occurs inward resulting in compressive force • Compressive force leads to vascular compromise with collapse of blood vessels, nerves and muscle cells • Without a steady supply of oxygen and nutrients, nerve and muscle cells die in a matter of hours • Problem is local to a limb or body area Traumatic • Crush syndrome - loss of blood to supply muscle tissue rhabdomyolysis toxins produced from muscle metabolism without oxygen as well as normal intracellular contents • Muscles can withstand approx. 4 hours without blood flow before cell death occurs • Toxins may continue to leak into body for as long as 60 hours after release of crush injury • The major problem is not recognising the potential for its existence, then removing the compressive force prior to arrival -
Incidence of Asymptomatic Pulmonary Embolism in Moderately to Severely Injured Trauma Patients David J
The Journal of TRAUMA Injury, Infection, and Critical Care Incidence of Asymptomatic Pulmonary Embolism in Moderately to Severely Injured Trauma Patients David J. Schultz, MD, Karen J. Brasel, MD, MPH, Lacey Washington, MD, Lawrence R. Goodman, MD, Robert R. Quickel, MD, Randolph J. Lipchik, MD, Todd Clever, BS, and John Weigelt, MD Background: Chest computed tomo- sessed using an anatomic scoring system. patients receiving pharmacologic prophy- graphic (CT) scanning is used frequently Patients not receiving anticoagulation laxis had a PE. to evaluate symptomatic patients for pul- were followed. Conclusion: Asymptomatic PE occur monary embolus (PE). The incidence of Results: Twenty-two of 90 patients in 24% of moderately to severely injured PE diagnosed by helical CT scanning in had a PE. Four had major clot burden, patients. Age, head, chest, and lower ex- asymptomatic patients is unknown. including one patient with a saddle embo- tremity injury are associated with an in- Methods: Asymptomatic trauma pa- lus. Risk factors for asymptomatic PE in- creased risk. Standard thromboembolic tients with an Injury Severity Score > 9 clude age (odds ratio [OR], 1.04), head prophylaxis is not reliably protective. were studied with contrast-enhanced heli- injury (OR, 6.78), chest injury (OR, 4.51), Key Words: Computed tomographic cal CT images of the chest, pelvis, and lower extremity injury (OR, 5.03), and scanning, Pulmonary embolus, Head in- lower extremities. Clot burden was as- transfusion (OR, 3.42). Thirty percent of jury, Chest injury, Transfusion. J Trauma. 2004;56:727–733. hromboembolic complications are common in the uate respiratory symptoms in these patients. -
Modern Management of Traumatic Hemothorax
rauma & f T T o re l a t a m n r e u n o t J Mahoozi, et al., J Trauma Treat 2016, 5:3 Journal of Trauma & Treatment DOI: 10.4172/2167-1222.1000326 ISSN: 2167-1222 Review Article Open Access Modern Management of Traumatic Hemothorax Hamid Reza Mahoozi, Jan Volmerig and Erich Hecker* Thoraxzentrum Ruhrgebiet, Department of Thoracic Surgery, Evangelisches Krankenhaus, Herne, Germany *Corresponding author: Erich Hecker, Thoraxzentrum Ruhrgebiet, Department of Thoracic Surgery, Evangelisches Krankenhaus, Herne, Germany, Tel: 0232349892212; Fax: 0232349892229; E-mail: [email protected] Rec date: Jun 28, 2016; Acc date: Aug 17, 2016; Pub date: Aug 19, 2016 Copyright: © 2016 Mahoozi HR. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Abstract Hemothorax is defined as a bleeding into pleural cavity. Hemothorax is a frequent manifestation of blunt chest trauma. Some authors suggested a hematocrit value more than 50% for differentiation of a hemothorax from a sanguineous pleural effusion. Hemothorax is also often associated with penetrating chest injury or chest wall blunt chest wall trauma with skeletal injury. Much less common, it may be related to pleural diseases, induced iatrogenic or develop spontaneously. In the vast majority of blunt and penetrating trauma cases, hemothoraces can be managed by relatively simple means in the course of care. Keywords: Traumatic hemothorax; Internal chest wall; Cardiac Hemodynamic response injury; Clinical manifestation; Blunt chest-wall injuries; Blunt As above mentioned the hemodynamic response is a multifactorial intrathoracic injuries; Penetrating thoracic trauma response and depends on severity of hemothorax according to its classification.