Neonatal Orthopaedics
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Extrinsic Factors Influencing Fetal Deformations and Intrauterine
Hindawi Publishing Corporation Journal of Pregnancy Volume 2012, Article ID 750485, 11 pages doi:10.1155/2012/750485 Review Article Extrinsic Factors Influencing Fetal Deformations and Intrauterine Growth Restriction Wendy Moh, 1 John M. Graham Jr.,2 Isha Wadhawan,2 and Pedro A. Sanchez-Lara1 1 Center for Craniofacial Molecular Biology, Ostrow School of Dentistry and Children’s Hospital Los Angeles, Keck School of Medicine of the University of Southern California, 4650 Sunset Boulevard, MS 90, Los Angeles, CA 90027, USA 2 Cedars-Sinai Medical Center, Medical Genetics Institute and David Geffen School of Medicine at UCLA, 8700 Beverly Boulevard, PACT Suite 400, Los Angeles, CA 90048, USA Correspondence should be addressed to Pedro A. Sanchez-Lara, [email protected] Received 24 March 2012; Revised 4 June 2012; Accepted 4 June 2012 Academic Editor: Sinuhe Hahn Copyright © 2012 Wendy Moh et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. The causes of intrauterine growth restriction (IUGR) are multifactorial with both intrinsic and extrinsic influences. While many studies focus on the intrinsic pathological causes, the possible long-term consequences resulting from extrinsic intrauterine physiological constraints merit additional consideration and further investigation. Infants with IUGR can exhibit early symmetric or late asymmetric growth abnormality patterns depending on the fetal stage of development, of which the latter is most common occurring in 70–80% of growth-restricted infants. Deformation is the consequence of extrinsic biomechanical factors interfering with normal growth, functioning, or positioning of the fetus in utero, typically arising during late gestation. -
Souvenir Programme, This Information Is Correct
1 SUBSPECIALTY INTEREST GROUP MEETINGS At time of printing this souvenir programme, this information is correct. Should there be any last minute changes, please refer to the signage outside each meeting room or enquire at the secretariat counter. SUBSPECIALTY INTEREST GROUP MEETING DATE TIME MEETINGS ROOM ASEAN OA Education Committee 21st May 2015 0830 - 1700hrs Kelantan Room Meeting Hand Subspecialty Interest Group 22nd May 2015 1030 - 1200hrs Pahang Room Foot & Ankle Subspecialty Interest Negeri Sembilan 22nd May 2015 1030 - 1200hrs Group Room Spine Subspecialty Interest Group 22nd May 2015 1130 - 1200hrs Johore Room Negeri Sembilan Paediatrics Subspecialty Interest Group 22nd May 2015 1400 - 1530hrs Room ASEAN OA Council Meeting 22nd May 2015 1400 - 1730hrs Kelantan Room Arthroplasty Subspecialty Interest 22nd May 2015 1600 - 1730hrs Pahang Room Group Negeri Sembilan Trauma Subspecialty Interest Group 22nd May 2015 1600 - 1730hrs Room Oncology Subspecialty Interest Group 23rd May 2015 1030 - 1200hrs Penang Room Sports Subspecialty Interest Group 23rd May 2015 1400 - 1530hrs Johore Room MOA Annual General Meeting 23rd May 2014 1600 - 1730hrs Johore Room 23rd May 2015 0800 - 1730hrs APOA Council Meeting Kelantan Room 24th May 2015 0800 - 1500hrs 2 INDEX PAGE NO. DESCRIPTION 2 Subspecialty Interest Group Meetings Malaysian Orthopaedic Association Office Bearers 4 45th Malaysian Orthopaedic Association Annual Scientific Meeting 2015 Organising Committee Welcome Message from President of Malaysian Orthopaedic Association and 5 Organising -
A Brief Evaluation and Image Formation of Pediatrics Nutritional Forum in Opinion Sector Disouja Wills* Nutritonal Sciences, Christian Universita Degli Studo, Italy
d Pediatr Wills, Matern Pediatr Nutr 2016, 2:2 an ic l N a u n t DOI: 10.4172/2472-1182.1000113 r r e i t t i o Maternal and Pediatric a n M ISSN: 2472-1182 Nutrition ShortResearch Commentary Article OpenOpen Access Access A Brief Evaluation and Image formation of Pediatrics Nutritional Forum in Opinion Sector Disouja Wills* Nutritonal Sciences, Christian Universita degli studo, Italy Abstract Severe most and one of the main global threat is Nutritional disorders to backward countries, with respect to this issue WHO involved and trying to overcome this issue with the Co-ordination of INF and BNF. International Nutrition Foundation and British Nutrition Foundation, development in weight gain through proper nutrition and proper immune mechanism in the kids is their main role to eradicate and overcome nutritional problems in world. Keywords: INF; BNF; Malnutrition; Merasmus; Rickets; Weight loss; Precautions to Avoid Nutrition Deficiency in Paediatric health issue Paediatrics Introduction Respective disease having respective deficiency dis order but in the case of nutritional diseases. Proper nutrition is the only thing to cure In the mankind a respective one health and weight gain is fully nutritional disorders. Providing sufficient diet like fish, meat, egg, milk based on perfect nutritional intake which he is having daily, poor diet to malnourished kids and consuming beef, fish liver oil, sheep meat, will show the improper impact and injury to the some of the systems boiled eggs from the age of 3 itself (Tables 1 and 2). in the body, total health also in some times. Blindness, Scurvy, Rickets will be caused by nutritional deficiency disorders only, mainly in kids. -
Basic Biomechanics
Posture analysis • A quick evaluation of structure and function • Doctor views patient from behind (P-A) and from the side (lateral) • References points of patient’s anatomy relative to plumb (a position of balance) 9/3/2013 1 Posture analysis • Lateral View – Knees (anterior, posterior, plumb, genu recurvatum) – Trochanter (anterior, posterior, plumb) – Pelvis (anterior, posterior, neutral pelvic tilt) – Lumbar lordosis (hypo-, hyper-, normal) – Mid-axillary line (anterior, posterior, plumb) – Thoracic kyphosis (hyp-, hyper- normal) – Acromion (anterior, posterior, plumb) – Scapulae (protracted, retracted, normal) – Cervical lordosis (hypo-, hyper-, normal) – External auditory meatus (anterior, posterior, plumb) – Occiput (extended, neutral, flexed) 9/3/2013 2 Posture analysis • Posterior – Anterior View – Feet (pronation, supination, normal) – Achilles tendon (bowed in/out, normal) – Knees (genu valga/vera, normal - internal/external rotation) – Popliteal crease heights (low, high, level) – Trochanter heights (low, high, level) – Iliac crest heights (low on the right/left, normal) – Lumbar scoliosis (right/left, or no signs of) – Thoracic scoliosis (right/left, or no signs of) – Shoulder level (low on the right/left, or normal) – Cervical scoliosis (right/left, or no signs of) – Cervical position (rotation, tilt, neutral) – Mastoid (low on the right/left, or normal) 9/3/2013 3 …..poor postures 9/3/2013 4 Functional Anatomy of the Spine • The vertebral curvatures – Cervical Curve • Anterior convex curve (lordosis) develop in infancy -
ICD-9CM Coding Achilles Bursitis Or Tendinitis 726.71 Adhesive
ICD-9CM CODING OF COMMON CHIROPRACTIC CONDITIONS CONDITION ICD-9CM coding Achilles bursitis or tendinitis 726.71 Adhesive capsulitis of shoulder 726 Anklyosing Spondylitis (spine only) 720 Anterior cruciate ligament (old disruption) 717.83 Bicipital tenosynovitis 726.12 Boutonniere deformity 736.21 Brachial neuritis or radiculitis 723.4 Bunion 727.1 Bursitis of knee 726.6 Calcaneal spur 726.73 Carpal tunnel syndrome 354 Cervical radiculitis 723.4 Cervical spondylosis with myelopathy 721.1 Cervical spondylosis without myelopathy 721 Cervicalgia 723.1 Chondromalacia of patella 717.7 Claw hand (acquired) 736.06 Claw toe (acquired) 735.5 Coccygodynia 724.79 Coxa plana 732.1 Coxa valga (acquired) 736.31 Coxa vara (acquired) 736.32 de Quervain's disease 727.04 Degeneration of cervical intervertebral disc 722.4 Degeneration of thoracic or lumbar intervertebral disc 722.5 Disc Degeneration (Cervical with myelopathy) 722.71 Disc Degeneration (Lumbar with myelopathy) 722.73 Disc Degeneration (Thoracic) 722.51 Disc Degeneration (Cervical) 722.4 Disc Degeneration (Lumbar) 722.52 Disc Degeneration (Thoracic with myelopathy) 722.72 Disc displacement without myelopathy (Thoracic) 722.11 Disc displacement of without myelopathy (Cervical ) 722 Disc displacement without myelopathy (Lumbar) 722.1 Dupuytren's contracture 728.6 Entrapment syndromes 354.0-355.9 Epicondylitis (Lateral) 726.32 Epicondylitis (Medial) 726.31 Flat foot-Pes planus (acquired) 734 Fracture (Lumbar) 805.4 Ganglion of tendon sheath 727.42 Genu recurvatum (acquired) 736.5 Genu valgum -
Necrotizing Enterocolitis in a Newborn Following Intravenous Immunoglobulin Treatment for Haemolytic Disease
CASE REPORT Necrotizing Enterocolitis in a Newborn Following Intravenous Immunoglobulin Treatment for Haemolytic Disease Semra Kara1, Hulya Ulu-ozkan2, Yavuz Yilmaz2, Fatma Inci Arikan3, Ugur Dilmen4 and Yildiz Dallar Bilge3 ABSTRACT ABO iso-immunization is the most frequent haemolytic disease of the newborn. Treatment depends on the total serum bilirubin level, which may increase very rapidly in the first 48 hours of life in cases of haemolytic disease of the newborn. Phototherapy and, in severe cases, exchange transfusion are used to prevent hyperbilirubinaemic encephalopathy. Intravenous immunoglobulins (IVIG) are used to reduce exchange transfusion. Herein, we present a female newborn who was admitted to the NICU because of ABO immune haemolytic disease. After two courses of 1 g/kg of IVIG infusion, she developed necrotizing enterocolitis (NEC). Administration of IVIG to newborns with significant hyperbilirubinaemia due to ABO haemolytic disease should be cautiously administered and followed for complications. Key Words: Necrotizing enterocolitis. Hyperbilirubinaemia. Newborn. Intravenous immunoglobulins. Iso-immunization. INTRODUCTION important adverse reactions,6 one of which is described Blood group incompatibilities are most frequent and hereby. severe conditions causing hyperbilirubinaemia in the neonatal period.1 Phototherapy and in severe cases CASE REPORT exchange transfusion are used to prevent kernicterus A female newborn was admitted to our neonatal unit and reduce perinatal mortality. Exchange transfusion because of jaundice on the tenth hours after being born. is not free of severe complications such as thrombo- The baby was born by uncomplicated vaginal delivery to cytopenia, apnoea, pulmonary haemorrhage, haemodyna- a healthy 31-year-old mother who was fourth gravida mic instability, septicaemia and necrotizing enterocolitis and second para. -
Lista De Doenças Raras E Sinónimos: Listadas Por Ordem Alfabética
Março 2016 Lista de doenças raras e sinónimos: Listadas por ordem alfabética www.orpha.net www.orphadata.org METODOLOGIA Recolha de dados A Orphanet fornece uma compressiva enciclopédia de Com o aparecimento de novos conhecimentos doenças raras na Europa, publicada bianualmente em científicos, a enciclopédia de doenças raras da Orphanet formato de lista. é actualizada por adição/actualização regular de novas As doenças raras registadas na Orphanet são definidas doenças através de duas fontes não exclusivas: fontes em dois âmbitos: documentais e/ou aconselhamento especializado. O conhecimento científico é monitorizado através: Qualquer entidade é definida pela sua homogeneidade clinica, independentemente da Uma análise, duas vezes por mês, de um sua etiologia ou do número de genes causadores conjunto definido de revistas científicas identificados; internacionais revistas por pares, que abrangem a diversidade das especialidades médicas A raridade é definida de acordo com a representadas na Orphanet; legislação europeia definindo um limite de prevalência não superior a 5 pessoas afectadas Um algoritmo de pesquisa mensal na Medline: por cada 10.000 (Regulação (EC) Nº141/200 do (nosologia[Titulo] OU classificação[Titulo] OU Parlamento Europeu e do conselho de 16 de nomenclatura[Titulo] ou terminologia[Titulo]) dezembro de 1999 em medicamentos órfãos, E (doença rara* OU Síndrome* OU http://ec.europa.eu/health/files/eudralex/vol- desordem*); 1/reg_2000_141/reg_2000_141_en.pdf). As doenças raras registadas foram descritas na literatura Pesquisas específicas na Medline de acordo científica internacional (artigos revistos por pares) com com as solicitações dos especialistas, dos pelo menos dois casos que confirmam que os sinais utilizadores da base de dados ou das clínicos não são associados fortuitamente. -
SUBGALEAL HEMATOMA Sarah Meyers MS4 Ilse Castro-Aragon MD CASE HISTORY
SUBGALEAL HEMATOMA Sarah Meyers MS4 Ilse Castro-Aragon MD CASE HISTORY Ex-FT (37w6d) male infant born by low transverse C-section for arrest of descent and chorioamnionitis to a 34-year-old G2P1 mother. The infant had 1- and 5-minute APGAR scores of 9 and 9, weighed 3.625 kg (54th %ile), and had a head circumference of 34.5 cm (30th %ile). Following a challenging delivery of the head during C/s, the infant was noted to have left-sided parietal and occipital bogginess, and an ultrasound was ordered due to concern for subgaleal hematoma. PEDIATRIC HEAD ULTRASOUND: SUBGALEAL HEMATOMA Superficial pediatric head ultrasound showing moderately echogenic fluid collection (green arrow), superficial to the periosteum (blue arrow), crossing the sagittal suture (red arrow). Findings on U/S consistent with large parieto-occipital subgaleal hematoma. PEDIATRIC HEAD ULTRASOUND: SUBGALEAL HEMATOMA Superficial pediatric head ultrasound showing moderately echogenic fluid collection (green arrow), consistent with large parieto-occipital subgaleal hematoma. CLINICAL FOLLOW UP - Subgaleal hematoma was confirmed on ultrasound and the infant was transferred from the newborn nursery to the NICU for close monitoring, including hourly head circumferences and repeat hematocrit measurements - Serial head circumferences remained stable around 34 cm and hematocrit remained stable between 39 and 41 throughout hospital course - The infant was subsequently treated with phototherapy for hyperbilirubinemia, thought to be secondary to resorption of the SGH IN A NUTSHELL: -
Differential Diagnosis of Oromandibular Limb Hypogenesis Syndromes Ole Junga,B, Ralf Smeetsb, Henning Hankenb, Reinhard E
Biomed Pap Med Fac Univ Palacky Olomouc Czech Repub. 2016 Jun; 160(2):310-315. A patient with Charlie M Syndrome: Differential diagnosis of Oromandibular Limb Hypogenesis Syndromes Ole Junga,b, Ralf Smeetsb, Henning Hankenb, Reinhard E. Friedrichb, Max Heilandb, Amir Tagnihaa, Brian Labowa Aim. In order to provide adequate treatment to a patient with a subtype of Oromandibular Limb Hypogenesis Syndromes (OLHS), this study aimed to review and to analyze the current literature and treatment options of OLHS. Methods. Literature review in PubMed and Sciencedirect. Due to the small number of results, all available references were analyzed precisely. Results. Cases of OLHS are formerly rare and often incomplete. There are various classifications available, which, however, often seem confusing and are of little practical relevance. Furthermore, we present a complete case report of a patient with Charlie M syndrome, a type IV (Chicarilli)/ V (Hall) OLHS malformation. We also describe embryologic pathogenesis and differential diagnoses. Conclusion. As a result of our literature review, we recommend an adjusted classification for OLHS. Key words: Oromandibular Limb Hypogenesis Syndromes (OLHS), Charlie M Syndrome, Oromandibular and limb hypogenesis malformations (OLHM) Received: August 1, 2015; Accepted with revision: April 8, 2016; Available online: April 27, 2016 http://dx.doi.org/10.5507/bp.2016.020 aDepartment of Plastic and Oral Surgery, Children´s Hospital Boston, Harvard Medical School, Boston, USA bDepartment of Oral and Maxillofacial Surgery, University Medical Center Hamburg, Hamburg, Germany Corresponding author: Ole Jung, e-mail: [email protected] INTRODUCTION CASE REPORT Oromandibular Limb Hypogenesis Syndromes A twenty-three-year-old male with severe oroman- (OLHS) describe a group of heterogeneous malforma- dibular and limb deformities presented for mandibular tions of the face and body. -
Research Article
z Available online at http://www.journalcra.com INTERNATIONAL JOURNAL OF CURRENT RESEARCH International Journal of Current Research Vol. 10, Issue, 07, pp.71222-71228, July, 2018 ISSN: 0975-833X RESEARCH ARTICLE THE TONGUE SPEAKS A LOT OF HEALTH. 1,*Dr. Firdous Shaikh, 2Dr. Sonia Sodhi, 3Dr Zeenat Fatema Farooqui and 4Dr. Lata Kale 1PG Student, Department of Oral Medicine and Radiology, CSMSS Dental College and Hospital, Aurangabad 2Professor, Department of Oral Medicine and Radiology, CSMSS Dental College and Hospital, Aurangabad 3Fatema Farooqui, Chief Medical Officer, Sri Ram Homeopathic Clinic and Research Center, Solapur 4Professor and Head, Department of Oral Medicine and Radiology, CSMSS Dental College and Hospital, Aurangabad ARTICLE INFO ABSTRACT Article History: Multifunctional organ of the human body without a bone yet strong is the tongue. It mainly consists Received 26th April, 2018 of the functional portion of muscle mass, mucosa, fat and the specialized tissue of taste i.e. the Received in revised form papillae. Diseases may either result from internal/ systemic causes of extrinsic causes like trauma, 14th May, 2018 infection, etc. A new method for classification has been proposed in this review for diseases of Accepted 09th June, 2018 tongue. This review mainly focuses on encompassing almost each aspect that the body reflects via its th Published online 30 July, 2018 mirror in mouth, the tongue. Key Words: Tongue, Diseases of Tongue, Discoloration of Tongue, Oral health, Hairy Tongue. Copyright © 2018, Firdous Shaikh et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. -
Genetic Causes of Congenital Malformation in India
International Journal of Human Genetics ISSN: 0972-3757 (Print) (Online) Journal homepage: http://www.tandfonline.com/loi/rhug20 Genetic Causes of Congenital Malformation in India Geeta Talukder & Archana Sharma To cite this article: Geeta Talukder & Archana Sharma (2006) Genetic Causes of Congenital Malformation in India, International Journal of Human Genetics, 6:1, 15-25, DOI: 10.1080/09723757.2006.11885942 To link to this article: https://doi.org/10.1080/09723757.2006.11885942 Published online: 04 Sep 2017. Submit your article to this journal Article views: 2 View related articles Full Terms & Conditions of access and use can be found at http://www.tandfonline.com/action/journalInformation?journalCode=rhug20 © Kamla-Raj 2006 Int J Hum Genet, 6(1): 15-25 (2006) Genetic Causes of Congenital Malformation in India Geeta Talukder1 and Archana Sharma2 1. Vivekananda Institute of Medical Sciences, 99 Sarat Bose Road, Kolkata 700 026, West Bengal, India E-mail: geetatalukdar @hotmail.com 2. CAS in Cell & Chromosome Research, Department of Botany, University College of Science, 35 Ballygunj Circular Road, Kolkata 700 019, West Bengal, India KEYWORDS Congenital malformations; neonates; stillbirths; prenatal detection; prevention ABSTRACT Congenital malformations are a major cause of death of neonates in India where prenatal detection and treatment are not adequate in many hospitals and health centers. Incidence is specially high in stillbirths. It is not realized that genetic causes - chromosomal, single gene and polygenic - are the main causes of many congenital defects and early detection and prevention should be essential to make the small family norm a success. INTRODUCTION Recently Patel and Adhia (2005) detected major malformations in 7.92% of 17653 births and Phenotypic changes of genetic diseases at were able to attribute chromosomal cause to birth include congenital malformations in 4%,polygenic to 45.1% and total genetic chromosomes and single gene defects. -
Congenital Transverse Defects of Limbs and Digits* ('Intrauterine Amputation') by H
Arch Dis Child: first published as 10.1136/adc.37.193.263 on 1 June 1962. Downloaded from CONGENITAL TRANSVERSE DEFECTS OF LIMBS AND DIGITS* ('INTRAUTERINE AMPUTATION') BY H. G. KOHLER From the Department ofPathology, Birmingham Maternity Hospital (RECEIVED FOR PUBLICATION OCTOBER 23, 1961) Intrauterine amputation of the extremities is an Thomas Bartholin of Copenhagen (1616-1680) uncommon and peculiar congenital deformity which is said to be the first to mention a congenitally is characterized by the absence of one or more distal deficient extremity. He lived at the very threshold limb portions. Termination of the proximal part of the modern scientific era and belonged to a family is usually abrupt and bears no apparent relation to well known for their contributions to medicine and anatomical boundaries. The term 'amputation' biology: Thomas Bartholin's son, Caspar Secundus, suggests separation, by mechanical force, of a limb was the first to describe the vestibulo-vaginal glands. already formed rather than a failure of develop- Thomas was a man of great learning and wide ment, but such a view of the pathogenesis of this interests (Rhodes, 1957). His textbook on anatomy abnormality is far from universally accepted. It was translated into English and used widely for a would probably be better to use a neutral term such long time. Bartholin's descriptions of monsters, as 'transverse defects', but for the conservatism of however, tend to be more imaginative than factual medical nomenclature. (Hendry and Kohler, 1956). The essay which copyright. Circular grooves around the digits or limbs, and contains a reference to limb defects bears the title similar soft tissue defects, also known as ring con- 'Gravidarum Imaginatio' and there, in passing, he strictions, are seen usually in association with 'intra- tells us of a deformed male infant with only one uterine amputation', but also on their own.