27 Cardiovascular Disorders
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
The Acute and Long-Term Ocular Effects of Accelerated Hypertension: a Clinical and Electrophysiological Study
THE ACUTE AND LONG-TERM OCULAR EFFECTS OF ACCELERATED HYPERTENSION: A CLINICAL AND ELECTROPHYSIOLOGICAL STUDY 1 2 2 3 3 L2 S. J. TALKS , , P. GOOD , C. G. CLOUGH , D. G. BEEVERS and P. M. DODSON Birmingham SUMMARY the invention of the ophthalmoscope in 1851 the Thirty-four patients with accelerated hypertension were causes of this disturbance could be described? clinically examined. The visual evoked potential (VEP) Leibreich,4 in 1859, was the first to describe and electroretinogram (ERG) were recorded: acutely 'albuminuric retinitis' in Bright's disease. However, in 12 patients, being repeated in 7 patients up to 6 it was not until 1914, after the invention of the Riva months later. In the remaining 22 patients these tests Rocci sphygmomanometer at the end of the nine were performed 2-4 years after presentation. Visual teenth century, that Volhard and Fahr6 related the acuity was 5.6/12 in 22 of 68 (32 %) eyes at presentation retinopathy to arterial hypertension. and 5.6/12 in 10 of 58 (19%) eyes at follow-up. The It was then realised that the fundal appearance cause of severest loss of vision appeared to be anterior was associated with the severity of the hypertension ischaemic optic neuropathy, found in 3 cases. During and with the prognosis of the patient. In 1939 Keith the acute stage 11 patients (92%) had abnormal VEPs et aC drew up a four-group grading system. Grade 3 and all had abnormal ERGs. The group mean PI00 (more recently called accelerated hypertensionS) latency, of the 7 patients (14 eyes) seen acutely and consisted of 'angiospastic retinitis', 'characterised followed up at 6 months, was 123.8 ms with significant especially by oedema, cotton wool patches, and recovery of latency (p<0.005) to 110.9 ms. -
MOC PORT/DOCK Practice Core Modules Content Outline
MOC PORT/DOCK Practice Core Modules Content Outline 1 Cataract and Anterior Segment (10%) 1.1 Basic Anatomical and Scientific Aspects 1.1.1 The Normal Lens 1.1.1.1 Anatomy 1.2 Assessment Techniques 1.2.1 History-Taking and Preoperative Examination of Ocular Structures 1.2.1.1 Take patient history 1.2.1.2 Examine ocular structures 1.2.1.3 Signs and symptoms 1.2.1.4 Indications for surgery 1.2.1.6 External examination 1.2.1.7 Slit-lamp examination 1.2.1.8 Fundus evaluation 1.2.1.9 Impact on visual functioning and quality of life 1.2.2 Measurement of Visual and Macular Function 1.2.2.1 Visual acuity testing 1.2.2.2 Test visual acuity 1.2.2.5 Visual field testing 1.2.2.6 Test visual field 1.2.2.7 Cataract's effect on visual acuity 1.2.2.8 Estimate cataract's effect on visual acuity 1.2.2.10 Measure visual (and macular) function 1.2.4.3 Evaluate glare disability: subjective and objective 1.3 Clinical Disease Conditions and Manifestations 1.3.1 Types of Cataract 1.3.1.1 Identify types of cataracts 1.3.2 Anterior Segment Disorders 1.3.2.1 Diagnose anterior segment disorders 1.3.2.2 Cataract associated with uveitis 1.3.2.3 Diabetes mellitus and cataract formation 1.3.2.4 Lens-induced glaucoma 1.3.2.4.1 Lens particle 1.3.2.4.2 Phacolytic 1.3.2.4.3 Phacomorphic Copyright and Use Policy for ABO Content Outlines © 2017 – American Board of Ophthalmology. -
Ophthalmology July 10-11, 2018 Bangkok, Thailand
J Clin Exp Ophthalmol 2018, Volume 9 conferenceseries.com DOI: 10.4172/2155-9570-C4-088 3rd International Conference on Ophthalmology July 10-11, 2018 Bangkok, Thailand Stage of Hypertensive Retinopathy among Patients who undergone Cataract Surgery in Zamboanga City Medical Center Jerne Kaz Niels B. Paber Dr.evangelista st, sta. Catalina Background: Individuals who are not known hypertensive are noted to have blurring of vision as an initial presentation. Preventable co- morbidity such as hypertension is essential in saving sight in patients with cataract. Objective: To determine the prevalence of hypertension and stage of hypertensive retinopathy among individuals who undergone cataract surgery and to identify the association between the stage of hypertension and the risk factors for hypertension and the stage of hypertensive retinopathy. Methods: This prospective study included 203 individuals. All of the participants were noted to have mature cataract surgery done and was noted to have followed up at Tzu Chi Eye center from July 1 2017 to March 30, 2018. The nature, significance and procedure of the study were explained to every identified respondent. There was only one ophthalmologist who saw the participants who enrolled in the study. Once they understood the study, a written informed consent was taken. They were asked to answer questions provided by the researcher and their laboratory results were recorded. A follow up after 2 weeks was done in order to determine the stage of retinopathy of the patients. Demographic variables, hypertensive retinopathy, history of hypertension, medication usage, compliance, ECG changes, proteinuria, creatinine, and cardiomegaly on chest x-ray, radiographic identification of Atheromatous aorta and fundoscopic examination were analyzed. -
Multimodal Imaging of Cotton Wool Spots in Branch Retinal Vein Occlusion
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Bern Open Repository and Information System (BORIS) Original Paper Ophthalmic Res 2015;54:48–56 Received: April 7, 2015 DOI: 10.1159/000430843 Accepted: April 20, 2015 Published online: June 12, 2015 Multimodal Imaging of Cotton Wool Spots in Branch Retinal Vein Occlusion a, b, e a, b, f a, b Marion R. Munk Gerlinde Matt Magdalena Baratsits a, b c a, d a, b Roman Dunavoelgyi Wolfgang Huf Alessio Montuoro Wolf Buehl a, b a, b Ursula Schmidt-Erfurth Stefan Sacu on behalf of the Macula Study Group a b c Department of Ophthalmology, Medical University of Vienna, Center for Medical Physics and Biomedical d e Engineering and Vienna Reading Center, Medical University of Vienna, Vienna , Austria; Department of f Ophthalmology, Inselspital, University Hospital Bern, Bern , and Department of Ophthalmology, University Hospital Zurich, Zurich , Switzerland Key Words er than on CF (0.26 ± 0.17 mm 2 vs. 0.13 ± 0.1 mm 2 , p < 0.0001). Ischemia · Cotton wool spots · Cystoid bodies · Argon The CWS area on SD-OCT and surrounding pathology such laser treatment · Retinal vein occlusion · Branch retinal as intraretinal cysts, avascular zones and intraretinal hemor- vein occlusion · Hypoperfusion · Optical coherence rhage were predictive for how long CWS remained visible tomography · Fluorescein angiography · Axoplasmic (r 2 = 0.497, p < 0.002). Conclusions: The lifetime and presen- debris · Multimodal imaging · Acute macular tation of CWS in BRVO seem comparable to other diseases. neuroretinopathy SD-OCT shows a higher sensitivity for detecting CWS com- pared to CF. -
Ocular Co-Morbidity in Patients with Refractive Errors in Nigeria
N igerian Journal of Ophthalmology 2011; 19(1): 19-24 Ocular Co-morbidity in Patients with Refractive Errors in Nigeria BO Adegbehingbe FWA CS, O Adeoye FWA CS, BA Adewara M BBS Ophthalmology Unit, Faculty of Clinical Sciences, Obafemi Awolowo University, Ile-Ife 1-4 ABSTRACT a high proportion of patients attend ing ophthalm ic clinics. Refractive errors can be easily d iagnosed , m easured and Purpose: To d eterm ine the pattern and prevalence of other corrected w ith spectacles or other refractive corrections to ocular problem s seen in patients w ith refractive errors attain normal vision. H ow ever, non correction or inad equate in a N igerian teaching hospital. correction of refractive errors becom es a m ajor cause of low Methods: A retrospective hospital-based review of all vision and even blind ness. Globally, there are 8 m illion consecutive patients w ho presented w ith signs and people w ho are blind and 153 m illion w ith visual sym ptom s of refractive errors at the Obafem i Aw olow o im pairm ent (presenting visual acuity <6/ 18 in the better University Teaching H ospitals Com plex betw een 1st eye) d ue to uncorrected refractive errors; this exclud es January 2007 and 31st August 2007. Patients w ho had a presbyopia.5 Poor visual outcom e in patients w ith refractive d iagnosis of refractive error and su bsequently had errors could be in part d ue to other associated ocular d etailed eye exam ination w ere includ ed in this stud y. -
Hypertensive Retinopathycourse # 40048
Hypertensive Retinopathy Course # 40048 Instructor: Amiee Ho OD Section: Systemic Disease COPE Course ID: 5119251192----SDSDSDSD Expiration Date: October 10, 2019 Qualified Credits: 1.00 credits --- $39.00 COURSE DESCRIPTION: This course focuses on the clinical features, diagnosis and management of hypertensive retinopathy. Additionally, some background and statistics on systemic hypertension is also presented. LEARNING OBJECTIVES: • Know the retinal signs of hypertension • Understand importance of checking blood pressure and knowing when to refer immediately • Understand general background and current statistics of systemic hypertension • Know how to manage and co-manage these types of patients • Understand early detection and proper referrals can save lives Hello, I am Dr. Aimee Ho, and welcome to Pacific University’s online CE course on Hypertensive Retinopathy. For this course, we will be reviewing systemic hypertension, ocular manifestations of hypertension, and the management of these patients. Without further ado, let’s get started. I would like to start with presenting a case. This is a 45 Table 1 year old Caucasian male. He presents for his 1 st eye exam back in 2005. He complains of blurry vision at near without an Rx. His ocular history and medical history is unremarkable, and he’s not on any medications. For his occupation, he reports being an attorney in a very high stress environment. BCVA is 20/20 in the right eye, and 20/20 in the left. Pupils, EOM’s and confrontation visual fields were all unremarkable at that visit. The anterior segment was also unremarkable. Table 2 After dilation, looking at the posterior segment, the optic nerves looked healthy with pink rims, distinct margins, and a small C/D ratio. -
Hypertension and the Eye
rnib.org.uk/gp [email protected] Hypertension and the eye This factsheet was produced under a collaboration between the UK Vision Strategy, RNIB, and the Royal College of General Practitioners. Key learning points • Hypertensive retinopathy is a clinical diagnosis made when characteristic fundus findings are seen in a patient with or who has had systemic arterial hypertension. • Mild hypertensive retinal features are seen commonly and are of limited relevance, advanced changes represent important signs of accelerated hypertension. • The main complications from hypertension are retinal artery and retinal vein occlusions, and these cause considerable visual morbidity. • Treatment of hypertension may resolve ocular features, but does not improve established vision loss. • Other vascular risk factors eg hyperglycaemia, dyslipidaemia, smoking and abnormal circulation compound the risk and effect of hypertension in the eye. Evidence base: common eye diseases are commoner if the patient is hypertensive • Cataract: In a meta-analysis of 25 studies across the world, risk of cataract was found to be increased in populations with hypertension independent of glycaemic risk, obesity or lipids [1]. We don’t know how generalisable this finding is. • Glaucoma: Nocturnal hypotension is also found to be associated with progression of visual field defects in glaucoma. • Late stage AMD: Some population studies show increased incidence with high © 2016 RNIB Reg charity nos 226227, SC039316 rnib.org.uk/gp [email protected] systolic BP [2], others show incidence associated with the metabolic syndrome but not specifically with hypertension [3]. • Other: Incidental retinal detachment in non-myopic eyes has been found to be more common [4] but again we don’t know how generalisable this is. -
Cotton Wool Spots the Moral of the Story Brown Et Al
Examining the Retina Assess the optic nerve Clinical Decisions in Retina What is the cup to disc ratio Is there good coloration and perfusion Mark T. Dunbar, O.D., F.A.A.O. Is it flat Choroidal or scleral crescent Mark Dunbar: Disclosure Examining the Retina Consultant for Allergan Pharmn What is the caliber of the retinal vessels Optometry Advisory Board for: Make sure you look and consciously take not of what the caliber is Allergan Carl Zeiss Meditec Narrowing of the vessels requires checking Alcon Nutritional the blood pressure Advisory Board Mark Dunbar does not own stock in any of the above companies Normal A/V ratio is 2/3, ¾ ArticDx What about the arterial light reflex? Examining the Retina Examining the Retina Don’t forget to look at the anterior vitreous The Macula Needs to be done on every dilated patient Is there a foveal light reflex (FLR)? Done at the slit lamp, looking posterior to Is it flat? the lens Is there any fluid, hemorrhage, or exudate Retroillumination may help if you suspect Presence of drusen vitreous cell RPE mottling More on this later Examining the Retina PVD The peripheral retina 65% of individuals > 65 have PVD It has to be done through a dilated pupil More common in women Don’t substitute imaging for indirect ophthalmoscopy More common following intraocular surgery Use Imaging as a compliment, but not substitute More common following Be systematic in your examination inflammation You should be able to see ora on “all” gazes More common in aphakes It’s all about technique -
Diabetic Retinopathy by the Numbers a Guide to Following and Educating Patients LACRIMAL OCCLUSION ■ Who Face This Threatening Diagnosis, P
REVIEW OF OPTOMETRY Earn 2 CE Credits: Can You Identify These Vitreous Anomalies?, p. 94 ■ VOL. 153 NO. 6 June 15, 2016 www.reviewofoptometry.com ■ JUNE 15, 2016 ■ ANNUAL 7th Annual Retina Report RETINA REPORT Diabetic Retinopathy By the Numbers ■ A guide to following and educating patients LACRIMAL OCCLUSION who face this threatening diagnosis, p. 36 • Food for Thought: Diet, Genetics and AMD, p. 48 • The Dilation Dilemma p. 54 • Managing Patients with ■ VITREOUS ANOMALIES Hypertensive Crisis, p. 58 ■ VIRAL CONJUNCTIVITIS 125 YEARS Plug the Drain with Lacrimal Occlusion, p. 70 | Claim Victory Over Viral Conjunctivitis, p. 78 001_ro0616_fc.indd 1 6/9/16 9:57 AM We call it our fundamental business truth: 1 20/200 Your success is essential to Our success. When your practice grows and flourishes, so does ours. At Johnson & Johnson Vision Care, we have operated according to this simple, but profound, principle for over 30 years. And it is the inspiration and 2 20/100 energy behind every year of our 3 decades of Global leadership in the contact lens marketplace. But history tells only a small part of the story. It is what we will do together in partnership over 3 20/70 the next 3 decades that will truly change the very meaning of vision care. Our commitment to innovation is unwavering. 4 20/50 But at the center of all of the R & D eff orts—be they technological, educational, or commercial in nature— is our commitment to advancing the eye care profession. 5 20/40 We thank you for your partnership and look forward to the continued journey together. -
Welcome to Williamson Eyecare Your Vision Source
Welcome to Williamson Eyecare your Vision Source Please complete the following forms in its entirety. Last Name_________________________________ First Name__________________________________ MI__________ Address_____________________________________ City_______________________ State_______ Zip_____________ Date of Birth_____________________ Age_______ Social Security #_______________________ Marital Status______ Home Phone_______________________________________ Cell Phone_______________________________________ E-Mail ____________________________________________________________________________________________ Please list BOTH vision and medical insurances and bring all insurance cards with you to your appointment. MEDICAL INSURANCE Ins. Co. Name: Secondary Ins. Co. Name: Insured’s Name: Insured’s Name: Identification #: Identification #: Group#: Group#: Insured’s DOB: Insured’s DOB: Patient Relation to insured: Patient Relation to insured: VISION INSURANCE Ins. Co. Name: Insured’s DOB: Insured’s Name: Insured’s SS#: Identification #: Patient Relationship to Insured: Primary Care Physician ____________________________________________Phone:_____________________________ Please list any medications that you are currently taking below (drug name and dosage) ______________________ ______________________ ______________________ ______________________ ______________________ ______________________ ______________________ ______________________ Drug Allergies______________________________________________________________________________________ Consent -
What Is Hypertensive Retinopathy?
What is hypertensive retinopathy? Hypertension, or high blood pressure, can be a spontaneous (primary) condition, but is usually associated with kidney disease or hyperthyroidism. We always recommend a complete blood count (CBC), serum profile / panel, urinalysis and a thyroid (T4) level, if it has not already been recently performed. It is very common to diagnose hypertension only after changes in vision have occurred, because while this medical condition is serious, it is not painful and may not cause any other changes in the pet's behavior. Hypertensive retinopathy, which can include small to extensive areas of retinal detachments and hemorrhage, is often controllable with oral medications. However, systemic hypertension is rarely curable and lifelong treatment is necessary. Eyes with complete (total) retinal detachments, and especially those with significant hemorrhage, tend to be associated with a poorer visual prognosis. Any changes in vision may be permanent. It is important to control blood pressure as much as possible due to the damage that can be caused to the heart and kidneys, as well as the retinas. Regular monitoring of blood pressure and blood testing with your family veterinarian is crucial for optimal long-term management of this condition. An aged cat, blind, with a systemic hypertension and a secondary hypertensive retinopathy: note the complete dilation of the pupil (we can almost not see his yellow irises). Picture of the fundus (retina) of the same cat: note Picture of the fundus (retina) of the same cat after 10 the important hemorrhages. days of medical treatment for systemic hypertension: note that most of the hemorrhages have resolved. -
Non-Diabetic Retinal Vascular Disease”
“Non-Diabetic Retinal Vascular Disease” Brad Sutton, O.D., F.A.A.O. Clinical Professor IU School of Optometry Indianapolis Eye Care Center Retinal Vascular Disease No financial disclosures Hypoperfusion Syndrome Occurs when the eye lacks blood perfusion secondary to carotid artery blockage or ophthalmic artery blockage. Terminology debate: venous stasis retinopathy vs. hypoperfusion syndrome Why is venous stasis retinopathy a poor term for this condition? Hypoperfusion Syndrome Patient may complain of dull, chronic ache in the affected eye Photostress issues / dazzle TIA symptoms may or may not be present (amaurosis fugax) Possible bruit / decreased pulse strength in carotid Hypoperfusion Syndrome Bruit at 30-85% blockage; swishing sound Bell vs. diaphragm Definitive diagnosis requires carotid imaging Hypoperfusion Syndrome Peripheral dot / blot hemorrhages Dilated veins Relatively spares the posterior pole Ocular Ischemic Syndrome With ocular NVD / NVE / NVI ischemic Iritis syndrome same Sluggish pupil findings plus……….. Conjunctival congestion Corneal Edema 80% unilateral / 20% bilateral Ocular Ischemic Syndrome Rare! Only 10% of eyes with 70+% blocked carotids 60% CF or worse VA by one year: 82% if NVI is present Teichopsia: colored afterimages after viewing lights More likely in patients with increased homocysteine and CRP Ocular Ischemic Syndrome When presented with Question about TIA these ocular findings……. Check carotids Arrange for carotid testing (Doppler has limits) ESR C-reactive protein CBC Ocular Ischemic Syndrome Treatment: Systemic management (diet, drugs, surgery) PRP / cryotherapy? Avastin / Lucentis / Eyelea? Five year mortality rate of 40% Sickle Cell Retinopathy Hemoglobinopathy affecting mostly AA (8% in US carry trait, .15% have SS dis.) 60,000 in US: 250,000 born yearly w-wide Malaria and natural selection (sickle trait carriers and sickle cell patients are resistant to malaria) AC, SA, SS, Sthal, SC.