Copyrighted Material

Total Page:16

File Type:pdf, Size:1020Kb

Copyrighted Material INDEX A American hemorrhagic fevers: Antibiotic action; Drug resistance; Α-dystroglycan, 301 Argentine, 16–17, 35, 279–280; Immune system; specifi c disease Abrus precatorius, 689 Bolivian, 277, 280, 287–288; Antiviral agents: AZT (zidovudine), Acid-fast bacteria, 212 Brazilian, 277, 281; overview of, 13, 32, 38, 48, 340–341, 355, 356; Active Bacterial Core (ABCs) 274, 274–279, 282–286; treatment, blocking synthesis of bacterial surveillance, 240 prevention, surveillance of, 275, RNA, 47; categories of HIV, 356; Active immunization, 465 287–289; Venezuelan, 277, 280–281 dengue fever, DHF, and DSS, 327– Acute glomerulonephritis, 119, 121, 126 Amoeba histolytica, 34 328; description of, 32; HAART, Acute Q fever, 15 Anaplasma phagocytophilum, 36, 76, 77, 48, 356–357, 382; hepatitis C virus Adaptive immunity, 40–42 84, 85, 86, 89, 90, 640–641, 645 (HCV), 391, 402–403; HHV-8 Adenocarcinoma of the lower Anaplasmosis, 552 and KS treatment using, 380–382; esophagus, 167–168 Andes virus, 442 SARS, 466–467. See also Interferons Adenovirus infection, 646–647 Anemia, 176, 528 Arenaviridae, 300 Aedes aegypti mosquito, 318, 322, Angiotensin-converting enzyme 2 Argentine hemorrhagic fever, 16–17, 328–329 (ACE-2), 463–464 35, 279–280 Aedes albopictus mosquito, 318, 322–323 Anopheles mosquitoes, 35, 525, 526, 537 Artemisinin, 535 Age-related immune system defects, Anthrax, 15, 670, 674–676 “Asian Flu” pandemic (type H2N2) 639–640, 642 Antibiotic action, 226, 228–229, 235– [1957], 413 Agglutination, 44 237. See also Antimicrobial agents; Aspergillus fungi, 648, 649, 650–651 Agroterrorism, 692–693 Drug-resistant bacteria AST (aspartate aminotransferase), 304 Aichi virus, 15 Antibodies: fi ve classes of, 35, 36, Attaching and effacing lesions, 159 AIDS (acquired immune defi ciency 43–44; IgA (immunoglobulin A), Attenuated microbes, 40 syndrome): AZT (zidovudine) 35, 36, 44, 151–152, 175, 350, Avian bornaviruses, 15 antiviral for treating, 32, 38, 340– 639; IgD (immunoglobulin D), Avian infl uenza (H5N1), 410, 411, 341, 355, 356; Cryptosporidium parvum 44; IgE (immunoglobulin E), 43; 418–421, 425–426, 469 and, 566, 568, 570–576, 581, 655; IgG (immunoglobulin G), 44, 126, AZT (zidovudine), 32, 38, 48, 340– description of, 14, 336, 338–339; 151–152, 175, 327, 379, 488, 557; 341, 355, 356 diseases associated with, 341–344, IgM (immunoglobulin M), 44, 67, 366, 367–368, 369, 371–372, 382; 327, 379, 513, 548, 557, 575, 639; B treatment, prevention, surveillance limitations of, 47–48; passed by B lymphocytes, 41, 44, 233–234 of, 48, 337, 355–360. See also HIV breastfeeding or placenta, 350, 639. Β-lactamase, 229, 230, 233 (human immunodefi ciencyCOPYRIGHTED virus); See also Immune system MATERIALΒ-lactamase-producing Escherichia coli, Viral infections Antibody-dependent enhancement 233–234 AIDS dementia, 344 (ADE), 324–326 Babesia species: animal diseases AIDS-associated malignancies, 343 Antigen-presenting cells, 42 caused by, 551–552; B. bovis, 549; AIDS-related complex (ARC), 341 Antigenic drift, 417 B. burgdorferi, 561; B. divergens, 548, Akodon azarae, 279 Antigenic shift, 417 550, 554; B. microti, 548, 550, 554, Alper’s syndrome, 517, 610, 617 Antigenic variation, 417 561; description of, 549, 553–554; Alzheimer’s disease, 625, 627 Antimicrobial agents: Anaplasma diagnosis of, 558; hosts of, 553; Amastigote, 593 phagocytophilum actions blocked by, 84; immune response to, 548, 557; life Amblyomma americanum (lone star tick), description of, 29, 46–48; modes and cycle of, 555–556; transmission of, 65, 81 mechanisms of, 226, 235–236. See also 554–555, 556–557 bbindex.inddindex.indd 772323 221/02/111/02/11 112:102:10 PPMM 724 INDEX Babesiosis: Babesia causation agent endocarditis, 102, 105–106; Oroya Borrelia japonica, 63 of, 548, 549, 550–559; description fever, 100, 101, 111, 112; overview Borellia lonestari, 63 of, 14, 549, 550–552; diagnosis of, of, 98, 109–110; treatment, Borrelia miyamoto, 63 558; history of outbreaks, 549–550; prevention, surveillance of, 98–99, Botulinum toxin, 690–692 immune response to, 548, 557; 111–113; trench fever (quintana Bovine spongiform encephalopathy symptoms of, 548; as tickborne fever), 100, 102, 104, 110, 111 (BSE), 613, 615–616, 618–619, 620. disease, 552; treatment, prevention, Bartonella species: B. bacilliformis, 106; B. See also “Mad cow disease” surveillance of, 549, 558–561 elizabethae, 106, 109; B. henselae, 36, Brazilian hemorrhagic fever, 277, 281 Bacillary angiomatosis (BA), 99, 100, 102, 103–104, 106, 107, Breakbone fever, 319. See also Dengue 100–101, 102, 105, 111 110, 111, 645; B. quintana, 35, 106, fever (DF) Bacillary peliosis hepatis (BP), 105, 111 108–109, 110, 645; HIV-positive Brucellosis, 678–680 Bacillus anthracis, 31, 674, 694 persons and, 343; overview of, 14, 98 Buboes, 672 Bacillus brevis, 47 Basophilic stippling, 101 Bubonic plague (Black death): spread Bacteria: acid-fast, 212; bacteriophages Basophils, 43 and symptoms of, 15–17, 670–672; (viruses) infecting, 31, 149; Baylisascaris, 15 Yersinia pestis causative agent of, 5, description of, 33, 228, 612; Bayou virus, 442 36, 671–674, 694 “fl esh-eating bacteria,” 124; Bcl-2, 376–377 Bunyaviridae family, 439–440 genetic diversity of, 38–39; morula Benign tertian malaria, 532 Burkholderia cepacia, 643 (microcolony) of, 88; obligate Beta-hemolytic growth (GAS Burkholderia mallei, 683–684 aerobes, 212–213; VBNC (viable but bacteria), 133 Burkholderia pseudomallei, 683–684 not able to be cultured), 156. See also Bilirubin, 528 Drug-resistant bacteria; specifi c bacteria Binary fi ssion, 33 C Bacterial infections: Bartonella Biofi lms, 187 CA-MRSA (community-associated infections, 36, 98–113; as Biological and Toxin Weapons MRSA), 231, 232, 240–241 bioterrorism agents, 671–684; Convention (1973), 671 Calomys laucha, 279, 280 digestive tract infections, 644; Biosafety level 4 (BSL-4) laboratory, Calomys musculinus, 279 Escherichia coli O157:H7 strains, 284, 304, 447, 685 Campylobacter enteritis, 15 139–155; group A streptococci Bioweapons: bacterial, viral, and Campylobacter jejuni, 642 (GAS), 14, 118–135; Helicobacter toxins as, 671–692; categories A, Campylobacter pyloridis, 164. See also pylori, ulcers, and cancer, 14, 33, B, and C, 669–670; characteristics Helicobacter pylori 162–176; human ehrlichiosis, 13, of, 669; history of, 668, 670–671; Campylobacter species, 10, 143, 162 76–78, 87–93; immunosuppression protective vaccines against, 694– Candida albicans, 342, 354, 642 resulting from, 640–641; 695; public health responses to, 668, Candida dubliniensis, 15 Legionnaires’ disease and Pontiac 693–694; threat of agroterrorism, Candida species, 648–649, 651 fever, 14, 35, 182–200; Lyme 692–693 Carrión’s disease, 99–100, disease, 13, 33, 35, 57–71, 551, 552; Black Creek Canal virus, 442 101–102, 111 other types of, 644–646; pulmonary Black death. See Bubonic plague Case Western Reserve tuberculosis, 14, 206–220; (Black death) University, 627 respiratory tract infections, 643– Blackwater fever, 529 Caseation, 210 644. See also Drug-resistant bacteria; Blastocystis hominis, 643, 654 Castleman’s disease, 366, 373 Infectious diseases Blood Borne Virus Transmission Risk Cat-scratch disease (CSD), 36, 99, 100, Bacterial resistance genes, 228, Assessment Questionnaire (WHO), 404 102, 103–104, 111 237–238 Blood transfusion HIV Category A HIV infection, 341 Bacteriophages, 31, 149, 228 transmission, 350 Category B HIV infection, 341 Barrett esophagus, 167–168 Bloodstream trypomastigote, 592 Category C HIV infection, 341 Barrier clothing protection, 266, 451 Bluetongue virus, 15 Cavitation, 210 Barrier nursing, 306–307 Bocavirus, 15 CD4 T helper cells: adaptive Bartonella infections: B. henselae Bolivian hemorrhagic fever, 277, 280, immunity and, 41, 42; dendritic (cat-scratch disease), 36, 99, 100, 287–288 cells aiding in infection of, 354–355; 102, 103–104, 106, 107, 110, 111, Bolomys obscurus, 280 drugs that suppress the, 641; innate 645; bacillary angiomatosis (BA), Borrelia afzelii, 63 immune system and, 42–43; STSS 100–101, 102, 105, 111; bacillary Borellia burgdorferi, 33, 35, 57, 58, resulting from overstimulated, peliosis hepatis (BP), 105, 111; 63–66, 78, 551 119, 120, 121, 127; superantigens bartonellosis (Carrión’s disease), Borrelia garinii, 63 stimulating activity of, 130. See also 99–100, 101–102, 111; Borrelia hispanica relapsing fever, 15 specifi c diseases bbindex.inddindex.indd 772424 221/02/111/02/11 112:102:10 PPMM INDEX 725 CD8 T killer cells: adaptive immunity Clostridium diffi cile, 143, 642, 643, 644, DEET, 58, 69–70, 490–493, 560 and, 41–42; drugs that suppress the, 645–646 Defi nitive host, 556 641; HHV-8 genes used to escape Clostridium perfringes, 124 Dematiaceous fungi, 653 death by, 378–379; innate immune Clostridium tetani, 692 Dendritic cells, 354–355 system and, 43; West Nile virus and, Coccidioides immitis, 652–653 Dengue fever (DF): causation of, 321– 488, 489. See also specifi c diseases Collectins, 639 324; description and history of, 10, CDC (Centers for Disease Control Colorado tick fever, 552 14, 314–321, 324–327; treatment, and Prevention): antitrypanosomal Common variable immunodefi ciency, prevention, surveillance of, 327– medications availability through, 640 331. See also Breakbone fever 599; Arbonet Surveillance System Community-associated MRSA (CA- Dengue hemorrhagic fever (DHF): for West Nile virus by, 493–494; MRSA), 231, 232, 240–241 causation of, 321–324; description Emerging Infections Program Complement cascade, 44 and outbreaks of, 314–321, 324–327; Network of the, 240–241; Emerging Congenital neutropenia, 640 treatment, prevention, surveillance Infectious Diseases by, 15; hepatitis Conjugation, 33, 228 of, 327–331 C surveillance by, 404–405; HIV/ Consumption. See Tuberculosis (TB) Dengue shock syndrome (DSS): AIDS Surveillance Report for 2006 by, Coronaviruses, 461–464, 465 causation of, 314–315, 321–324; 359; Legionella antisera developed by, Corticosteroids, 467, 641 description of, 314–316, 319–321, 195; Legionella longbeachae incidences Coxiella burnetii, 680–681 324–327; treatment, prevention, reported by the, 190; Legionnaires’ Coxsackievirus, 15 surveillance of, 327–331 disease reported to, 200; measures Creutzfeldt-Jakob disease (CJD): Dengue virus, 321–324 to control SARS in the U.S.
Recommended publications
  • Invasive Group a Streptococcal Disease Communicable Disease Control Unit
    Public Health and Primary Health Care Communicable Disease Control 4th Floor, 300 Carlton St, Winnipeg, MB R3B 3M9 T 204 788-6737 F 204 948-2040 www.manitoba.ca November, 2015 Re: Streptococcal Invasive Disease (Group A) Reporting and Case Investigation Reporting of Streptococcal invasive disease (Group A) (Streptococcus pyogenes) is as follows: Laboratory: All specimens isolated from sterile sites (refer to list below) that are positive for S. pyogenes are reportable to the Public Health Surveillance Unit by secure fax (204-948-3044). Health Care Professional: Probable (clinical) cases of Streptococcal invasive disease (Group A) are reportable to the Public Health Surveillance Unit using the Clinical Notification of Reportable Diseases and Conditions form (http://www.gov.mb.ca/health/publichealth/cdc/protocol/form13.pdf) ONLY if a positive lab result is not anticipated (e.g., poor or no specimen taken, person has recovered). Cooperation in Public Health investigation (when required) is appreciated. Regional Public Health or First Nations Inuit Health Branch (FNIHB): Cases will be referred to Regional Public Health or FNIHB. Completion and return of the Communicable Disease Control Investigation Form is generally not required, unless otherwise directed by a Medical Officer of Health. Sincerely, “Original Signed By” “Original Signed By” Richard Baydack, PhD Carla Ens, PhD Director, Communicable Disease Control Director, Epidemiology & Surveillance Public Health and Primary Health Care Public Health and Primary Health Care Manitoba Health, Healthy Living and Seniors Manitoba Health, Healthy Living and Seniors The sterile and non-sterile sites listed below represent commonly sampled body sites for the purposes of diagnosis, but the list is not exhaustive.
    [Show full text]
  • Toxic Shock-Like Syndrome Associated with Necrotizing Streptococcus Pyogenes Infection
    Henry Ford Hospital Medical Journal Volume 37 Number 2 Article 5 6-1989 Toxic Shock-like Syndrome Associated with Necrotizing Streptococcus Pyogenes Infection Thomas J. Connolly Donald J. Pavelka Eugene F. Lanspa Thomas L. Connolly Follow this and additional works at: https://scholarlycommons.henryford.com/hfhmedjournal Part of the Life Sciences Commons, Medical Specialties Commons, and the Public Health Commons Recommended Citation Connolly, Thomas J.; Pavelka, Donald J.; Lanspa, Eugene F.; and Connolly, Thomas L. (1989) "Toxic Shock- like Syndrome Associated with Necrotizing Streptococcus Pyogenes Infection," Henry Ford Hospital Medical Journal : Vol. 37 : No. 2 , 69-72. Available at: https://scholarlycommons.henryford.com/hfhmedjournal/vol37/iss2/5 This Article is brought to you for free and open access by Henry Ford Health System Scholarly Commons. It has been accepted for inclusion in Henry Ford Hospital Medical Journal by an authorized editor of Henry Ford Health System Scholarly Commons. Toxic Shock-like Syndrome Associated with Necrotizing Streptococcus Pyogenes Infection Thomas J. Connolly,* Donald J. Pavelka, MD,^ Eugene F. Lanspa, MD, and Thomas L. Connolly, MD' Two patients with toxic shock-like syndrome are presented. Bolh patients had necrotizing cellulitis due to Streptococcus pyogenes, and both patients required extensive surgical debridement. The association of Streptococcus pyogenes infection and toxic shock-like syndrome is discussed. (Henry Ford Hosp MedJ 1989:37:69-72) ince 1978, toxin-producing strains of Staphylococcus brought to the emergency room where a physical examination revealed S aureus have been implicated as the cause of the toxic shock a temperature of 40.9°C (I05.6°F), blood pressure of 98/72 mm Hg, syndrome (TSS), which is characterized by fever and rash and respiration of 36 breaths/min, and a pulse of 72 beats/min.
    [Show full text]
  • VENEREAL DISEASES in ETHIOPIA Survey and Recommendations THORSTEIN GUTHE, M.D., M.P.H
    Bull. World Hlth Org. 1949, 2, 85-137 10 VENEREAL DISEASES IN ETHIOPIA Survey and Recommendations THORSTEIN GUTHE, M.D., M.P.H. Section on Venereal Diseases World Health Organization Page 1. Prevalent diseases . 87 1.1 Historical .............. 87 1.2 Distribution.............. 88 2. Syphilis and related infections . 89 2.1 Spread factors . 89 2.2 Nature of syphilis . 91 2.3 Extent of syphilis problem . 98 2.4 Other considerations . 110 3. Treatment methods and medicaments . 114 3.1 Ancient methods of treatment . 114 3.2 Therapy and drugs . 115 4. Public-health organization. 116 4.1 Hospital facilities . 117 4.2 Laboratory facilities . 120 4.3 Personnel .............. 121 4.4 Organizational structure . 122 4.5 Legislation.............. 124 5. Recommendations for a venereal-disease programme . 124 5.1 General measures. ........... 125 5.2 Personnel, organization and administration . 126 5.3 Collection of data . 127 5.4 Diagnostic and laboratory facilities . 129 5.5 Treatment facilities . 130 5.6 Case-finding, treatment and follow-up . 131 5.7 Budget. ......... ... 134 6. Summary and conclusions . 134 References . 136 In spite of considerable handicaps, valuable developments in health took place in Ethiopia during the last two decades. This work was abruptly arrested by the war, and the fresh start necessary on the liberation of the country emphasized that much health work still remains to be done. A realistic approach to certain disease-problems and the necessity for compe- tent outside assistance to tackle such problems form the basis for future work. The accomplishments of the Ethiopian Government in the limited time since the war bode well for the future.
    [Show full text]
  • Can Leptospirosis Be Treated Without Any Kind of Medication?
    ISSN: 2573-9565 Research Article Journal of Clinical Review & Case Reports Can Leptospirosis Be Treated Without Any Kind of Medication? Huang W L* *Corresponding author Huang Wei Ling, Rua Homero Pacheco Alves, 1929, Franca, Sao Paulo, Infectologist, general practitioner, nutrition doctor, acupuncturist, 14400-010, Brazil, Tel: (+55 16) 3721-2437; E-mail: [email protected] pain management, Medical Acupuncture and Pain Management Clinic, Franca, Sao Paulo, Brazil Submitted: 16 Apr 2018; Accepted: 23 Apr 2018; Published: 10 May 2018 Abstract Introduction: Leptospirosis is an acute infectious disease caused by pathogenic Leptospira. Spread in a variety of ways, though the digestive tract infection is the main route of infection. As the disease pathogen final position in the kidney, the urine has an important role in the proliferation of the disease spreading [1]. Purpose: The purpose of this study was to show if leptospirosis can be treated without any kind of medication. The methodology used was the presentation of one case report of a woman presenting three days of generalized pain all over her body, especially in her muscles, mainly the calves of her legs, fever, headache and trembling. A blood exam was asked, as well as serology and acupuncture to relieve her symptoms. Findings: she recovered very well after five sessions of Acupuncture once a day. A month later, she came back with the results of her serology: it was positive leptospirosis. Conclusion: In this case, leptospirosis was cured without the use any kind of medication, being acupuncture a good therapeutic option, reducing the necessity of the patient’s admittance into a hospital, minimizing the costs of the treatmentand restoring the patient to a normal life very quickly.
    [Show full text]
  • WO 2014/134709 Al 12 September 2014 (12.09.2014) P O P C T
    (12) INTERNATIONAL APPLICATION PUBLISHED UNDER THE PATENT COOPERATION TREATY (PCT) (19) World Intellectual Property Organization International Bureau (10) International Publication Number (43) International Publication Date WO 2014/134709 Al 12 September 2014 (12.09.2014) P O P C T (51) International Patent Classification: (81) Designated States (unless otherwise indicated, for every A61K 31/05 (2006.01) A61P 31/02 (2006.01) kind of national protection available): AE, AG, AL, AM, AO, AT, AU, AZ, BA, BB, BG, BH, BN, BR, BW, BY, (21) International Application Number: BZ, CA, CH, CL, CN, CO, CR, CU, CZ, DE, DK, DM, PCT/CA20 14/000 174 DO, DZ, EC, EE, EG, ES, FI, GB, GD, GE, GH, GM, GT, (22) International Filing Date: HN, HR, HU, ID, IL, IN, IR, IS, JP, KE, KG, KN, KP, KR, 4 March 2014 (04.03.2014) KZ, LA, LC, LK, LR, LS, LT, LU, LY, MA, MD, ME, MG, MK, MN, MW, MX, MY, MZ, NA, NG, NI, NO, NZ, (25) Filing Language: English OM, PA, PE, PG, PH, PL, PT, QA, RO, RS, RU, RW, SA, (26) Publication Language: English SC, SD, SE, SG, SK, SL, SM, ST, SV, SY, TH, TJ, TM, TN, TR, TT, TZ, UA, UG, US, UZ, VC, VN, ZA, ZM, (30) Priority Data: ZW. 13/790,91 1 8 March 2013 (08.03.2013) US (84) Designated States (unless otherwise indicated, for every (71) Applicant: LABORATOIRE M2 [CA/CA]; 4005-A, rue kind of regional protection available): ARIPO (BW, GH, de la Garlock, Sherbrooke, Quebec J1L 1W9 (CA). GM, KE, LR, LS, MW, MZ, NA, RW, SD, SL, SZ, TZ, UG, ZM, ZW), Eurasian (AM, AZ, BY, KG, KZ, RU, TJ, (72) Inventors: LEMIRE, Gaetan; 6505, rue de la fougere, TM), European (AL, AT, BE, BG, CH, CY, CZ, DE, DK, Sherbrooke, Quebec JIN 3W3 (CA).
    [Show full text]
  • 2012 Case Definitions Infectious Disease
    Arizona Department of Health Services Case Definitions for Reportable Communicable Morbidities 2012 TABLE OF CONTENTS Definition of Terms Used in Case Classification .......................................................................................................... 6 Definition of Bi-national Case ............................................................................................................................................. 7 ------------------------------------------------------------------------------------------------------- ............................................... 7 AMEBIASIS ............................................................................................................................................................................. 8 ANTHRAX (β) ......................................................................................................................................................................... 9 ASEPTIC MENINGITIS (viral) ......................................................................................................................................... 11 BASIDIOBOLOMYCOSIS ................................................................................................................................................. 12 BOTULISM, FOODBORNE (β) ....................................................................................................................................... 13 BOTULISM, INFANT (β) ...................................................................................................................................................
    [Show full text]
  • Scarlet Fever Fact Sheet
    Scarlet Fever Fact Sheet Scarlet fever is a rash illness caused by a bacterium called Group A Streptococcus (GAS) The disease most commonly occurs with GAS pharyngitis (“strep throat”) [See also Strep Throat fact sheet]. Scarlet fever can occur at any age, but it is most frequent among school-aged children. Symptoms usually start 1 to 5 days after exposure and include: . Sandpaper-like rash, most often on the neck, chest, elbows, and on inner surfaces of the thighs . High fever . Sore throat . Red tongue . Tender and swollen neck glands . Sometimes nausea and vomiting Scarlet fever is usually spread from person to person by direct contact The strep bacterium is found in the nose and/or throat of persons with strep throat, and can be spread to the next person through the air with sneezing or coughing. People with scarlet fever can spread the disease to others until 24 hours after treatment. Treatment of scarlet fever is important Persons with scarlet fever can be treated with antibiotics. Treatment is important to prevent serious complications such as rheumatic fever and kidney disease. Infected children should be excluded from child care or school until 24 hours after starting treatment. Scarlet fever and strep throat can be prevented . Cover the mouth when coughing or sneezing. Wash hands after wiping or blowing nose, coughing, and sneezing. Wash hands before preparing food. See your doctor if you or your child have symptoms of scarlet fever. Maryland Department of Health Infectious Disease Epidemiology and Outbreak Response Bureau Prevention and Health Promotion Administration Web: http://health.maryland.gov February 2013 .
    [Show full text]
  • Strep Throat and Scarlet Fever N
    n Strep Throat and Scarlet Fever n After a few days, the rash begins to fade. The skin usually Strep throat is caused by infection with the bac- begins to peel, as it often does after a sunburn. teria Streptococcus. In addition to sore throat, swollen glands, and other symptoms, some chil- What are some possible dren develop a rash. When this rash is present, the infection is called scarlet fever. If your child complications of scarlet fever? has a strep infection, he or she will need antibio- Strep infection has some potentially serious complica- tics to treat it and to prevent rheumatic fever, tions. With proper treatment, most of these can be pre- which can be serious. vented. Complications include: Strep infection may cause an abscess (localized area of pus) in the throat. What are strep throat and scarlet Rheumatic fever. This disease develops a few weeks after fever? the original strep infection. It is felt to be caused by our immune system. It can be serious and can cause fever, heart Most sore throats are caused by virus, but strep throat is inflammation, arthritis, and other symptoms. Your child caused by the bacteria Group A Strepococcus. Treatment can be left with heart problems called rheumatic disease. of this infection with antibiotics may help your child feel Rheumatic fever is uncommon now and can be prevented better and prevent rheumatic fever. by treating the strep infection properly with antibiotics. Some children with strep throat or strep infections else- where may develop a rash. When that occurs, the infection Acute glomerulonephritis.
    [Show full text]
  • WO 2013/042140 A4 28 March 2013 (28.03.2013) P O P C T
    (12) INTERNATIONAL APPLICATION PUBLISHED UNDER THE PATENT COOPERATION TREATY (PCT) (19) World Intellectual Property Organization International Bureau (10) International Publication Number (43) International Publication Date WO 2013/042140 A4 28 March 2013 (28.03.2013) P O P C T (51) International Patent Classification: NO, NZ, OM, PA, PE, PG, PH, PL, PT, QA, RO, RS, RU, A61K 31/197 (2006.01) A61K 45/06 (2006.01) RW, SC, SD, SE, SG, SK, SL, SM, ST, SV, SY, TH, TJ, A61K 31/60 (2006.01) A61P 31/00 (2006.01) TM, TN, TR, TT, TZ, UA, UG, US, UZ, VC, VN, ZA, A61K 33/22 (2006.01) ZM, ZW. (21) International Application Number: (84) Designated States (unless otherwise indicated, for every PCT/IN20 12/000634 kind of regional protection available): ARIPO (BW, GH, GM, KE, LR, LS, MW, MZ, NA, RW, SD, SL, SZ, TZ, (22) International Filing Date UG, ZM, ZW), Eurasian (AM, AZ, BY, KG, KZ, RU, TJ, 24 September 2012 (24.09.2012) TM), European (AL, AT, BE, BG, CH, CY, CZ, DE, DK, (25) Filing Language: English EE, ES, FI, FR, GB, GR, HR, HU, IE, IS, IT, LT, LU, LV, MC, MK, MT, NL, NO, PL, PT, RO, RS, SE, SI, SK, SM, (26) Publication Language: English TR), OAPI (BF, BJ, CF, CG, CI, CM, GA, GN, GQ, GW, (30) Priority Data: ML, MR, NE, SN, TD, TG). 2792/DEL/201 1 23 September 201 1 (23.09.201 1) IN Declarations under Rule 4.17 : (72) Inventor; and — of inventorship (Rule 4.17(iv)) (71) Applicant : CHAUDHARY, Manu [IN/IN]; 51-52, In dustrial Area Phase- 1, Panchkula 1341 13 (IN).
    [Show full text]
  • Rat Bite Fever Due to Streptobacillus Moniliformis a CASE TREATED by PENICILLIN by F
    View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by PubMed Central Rat Bite Fever Due to Streptobacillus Moniliformis A CASE TREATED BY PENICILLIN By F. F. KANE, M.D., M.R.C.P.I., D.P.H. Medical Superintendent, Purdysburn Fever Hospital, Belfast IT is unlikely that rat-bite fever will rver become a public health problem in this country, so the justification for publishing the following case lies rather in its rarity, its interesting course and investigation, and in the response to Penicillin. PRESENT CASE. The patient, D. G., born on 1st January, 1929, is the second child in a family of three sons and one daughter of well-to-do parents. There is nothing of import- ance in the family history or the previous history of the boy. Before his present illness he was in good health, was about 5 feet 9j inches in height, and weighed, in his clothes, about 101 stone. The family are city dwellers. On the afternoon of 18th March, 1944, whilst hiking in a party along a country lane about fifteen miles from Belfast city centre, he was bitten over the terminal phalanx of his right index finger by a rat, which held on until pulled off and killed. The rat was described as looking old and sickly. The wound bled slightly at the time, but with ordinary domestic dressings it healed within a few days. Without missing a day from school and feeling normally well in the interval, the boy became sharply ill at lunch-time on 31st March, i.e., thirteen days after the bite.
    [Show full text]
  • Emerging Infectious Diseases Objectives What
    12/2/2015 EMERGING INFECTIOUS DISEASES What could be emerging in North Dakota? TRACY K. MILLER, PHD, MPH STATE EPIDEMIOLOGIST [email protected] OBJECTIVES 1. Identify new or re-emerging infections 2. Identify ways outside agencies can help the health department monitor for disease 3. Determine what education needs are available. 2 WHAT ARE "EMERGING" INFECTIOUS DISEASES? Infectious diseases whose incidence in humans has increased in the past two decades or threatens to increase in the near future have been defined as "emerging." These diseases, which respect no national boundaries, include: • New infections resulting from changes or evolution of existing organisms • Known infections spreading to new geographic areas or populations • Previously unrecognized infections appearing in areas undergoing ecologic transformation 3 1 12/2/2015 WHAT ARE RE-EMERGING INFECTIOUS DISEASES? Any condition, usually an infection, that had decreased in incidence in the global population and was brought under control through effective health care policy and improved living conditions, reached a nadir, and, more recently, began to resurge as a health problem due to changes in the health status of a susceptible population. 4 REPORTABLE CONDITIONS 5 CDC’S LIST OF EIDS • malaria • drug-resistant infections (antimicrobial resistance) • Marburg hemorrhagic fever • bovine spongiform encephalopathy (Mad cow disease) & variant Creutzfeldt-Jakob disease (vCJD) • measles • campylobacteriosis • meningitis • Chagas disease • monkeypox • cholera • MRSA (Methicillin Resistant
    [Show full text]
  • Report June 2
    COMMUNICABLE DISEASES • ISSN 1361 - 1887 Provisional Summary 2003 Northern Ireland Edition Vol 12 No 13 COMMUNICABLE DISEASES ISSN 1361 - 1887 Introduction This report summarises the main trends in communicable disease in Northern Ireland during 2003. It is primarily based on laboratory reports forward to CDSC (NI) and information supplied by Consultants in Communicable Disease Control. This is a more detailed annual summary than in previous years and replaces our annual report. The data for 2003 should be regarded as provisional to allow for late reporting of results and further typing of organisms. CDSC (NI) is extremely grateful to colleagues in Trusts and Boards for providing timely data and information on a wide range of infections and communicable disease issues. This summary can also be downloaded from our website http://www.cdscni.org.uk Contributing Laboratories Information Altnagelvin Mater Editorial Team: CDSC (NI) Antrim Musgrave Park Belfast City Hospital Belfast City Regional Mycology Dr Brian Smyth Lisburn Road, Belfast, BT9 7AB Belvoir Park Regional Virus Audrey Lynch N.Ireland Causeway Royal Victoria Dr Julie McCarroll Telephone: 028 9026 3765 Craigavon Tyrone County Dr Hilary Kennedy Fax: 028 9026 3511 Daisyhill Ulster Ruth Fox Email: [email protected] Erne Julie Boucher COMMUNICABLE DISEASES: Provisional Summary 1 Contents 1 Gastrointestinal Infections 3 Foodborne and Gastrointestinal outbreaks: 2003 2 Imported Infections 9 3 Human Brucellosis in Northern Ireland, 2003 13 4 Enhanced Surveillance of Influenza in Northern Ireland 15 5 Enhanced Surveillance of Meningococcal Disease 19 6 Enhanced Surveillance of Tuberculosis 23 7 Legionella Infections 27 8 Hepatitis 29 9 HIV and AIDS 33 10 Syphilis Outbreak in Northern Ireland 2001-2003 37 11 Childhood Vaccination Programme 41 Appendix 1 – Trends in Specific Reported Pathogens Appendix 2 – Notifications of Infections Diseases COMMUNICABLE DISEASES: Provisional Summary 2 1 Gastrointestinal infections Notifications of food poisoning increased steadily from1991 to 2000.
    [Show full text]