DISORDERS of AUDITORY PROCESSING: EVIDENCE for MODULARITY in AUDITION Michael R
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Prosopagnosia by B
J. Neurol. Neurosurg. Psychiat., 1959, 22, 124. PROSOPAGNOSIA BY B. BORNSTEIN and D. P. KIDRON From the Department of Neurology, Beilinson Hospital, Petah Tiqva, Israel "And what is the nature of this knowledge or recollection? I mean to ask, Whether a person, who having seen or heard or in any way perceived anything, knows not only that, but has a conception of something else which is the subject, not of the same but of some other kind of knowledge, may not be fairly said to recollect that of which he has the conception?" "And when the recollection is derived from like things, then another consideration is sure to arise, which is, Whether the likeness in any degree falls short or not of that which is recollected?" "The Philosophy of Plato " Phaedo (the Jowett translation). Does visual agnosia exist in a partial or isolated bances in sensation time, in adaptation time, in form, in which certain qualities only are affected, visual acuity, and in brightness discrimination. as opposed to generalized visual agnosia? Many Ettlinger (1956) rejected Bay's contentions. After workers cast doubt on this concept, maintaining analysing 30 cases of head injury, he showed that that partial visual agnosia is no more than a com- some patients had neither field nor perceptual bination of defects in vision, memory, and orienta- defects, others had field but not perceptual defects, tion, appearing together. and only in eight of the 30 patients were field and The clinical elucidation of partial visual agnosia perceptual defects found together. It is true that is likely to be affected by the patient's intellectual visual agnosia is frequently associated with homony- capacity, his mental state at the time of examination, mous hemianopsia, but despite this there are cases and his ability to cooperate without being influenced of hemianopsia without gnostic defects. -
Hearing Screening Training Manual REVISED 12/2018
Hearing Screening Training Manual REVISED 12/2018 Minnesota Department of Health (MDH) Community and Family Health Division Maternal and Child Health Section 1 2 For more information, contact Minnesota Department of Health Maternal Child Health Section 85 E 7th Place St. Paul, MN 55164-0882 651-201-3760 [email protected] www.health.state.mn.us Upon request, this material will be made available in an alternative format such as large print, Braille or audio recording. 3 Revisions made to this manual are based on: Guidelines for Hearing Screening After the Newborn Period to Kindergarten Age http://www.improveehdi.org/mn/library/files/afternewbornperiodguidelines.pdf American Academy of Audiology, Childhood Screening Guidelines http://www.cdc.gov/ncbddd/hearingloss/documents/AAA_Childhood%20Hearing%2 0Guidelines_2011.pdf American Academy of Pediatrics (AAP), Hearing Assessment in Children: Recommendations Beyond Neonatal Screening http://pediatrics.aappublications.org/content/124/4/1252 4 Contents Introduction .................................................................................................................... 7 Audience ..................................................................................................................... 7 Purpose ....................................................................................................................... 7 Overview of hearing and hearing loss ............................................................................ 9 Sound, hearing, and hearing -
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The Evolution of Language: Towards Gestural Hypotheses DIS/CONTINUITIES TORUŃ STUDIES IN LANGUAGE, LITERATURE AND CULTURE Edited by Mirosława Buchholtz Advisory Board Leszek Berezowski (Wrocław University) Annick Duperray (University of Provence) Dorota Guttfeld (Nicolaus Copernicus University) Grzegorz Koneczniak (Nicolaus Copernicus University) Piotr Skrzypczak (Nicolaus Copernicus University) Jordan Zlatev (Lund University) Vol. 20 DIS/CONTINUITIES Przemysław ywiczy ski / Sławomir Wacewicz TORUŃ STUDIES IN LANGUAGE, LITERATURE AND CULTURE Ż ń Edited by Mirosława Buchholtz Advisory Board Leszek Berezowski (Wrocław University) Annick Duperray (University of Provence) Dorota Guttfeld (Nicolaus Copernicus University) Grzegorz Koneczniak (Nicolaus Copernicus University) The Evolution of Language: Piotr Skrzypczak (Nicolaus Copernicus University) Jordan Zlatev (Lund University) Towards Gestural Hypotheses Vol. 20 Bibliographic Information published by the Deutsche Nationalbibliothek The Deutsche Nationalbibliothek lists this publication in the Deutsche Nationalbibliografie; detailed bibliographic data is available in the internet at http://dnb.d-nb.de. The translation, publication and editing of this book was financed by a grant from the Polish Ministry of Science and Higher Education of the Republic of Poland within the programme Uniwersalia 2.1 (ID: 347247, Reg. no. 21H 16 0049 84) as a part of the National Programme for the Development of the Humanities. This publication reflects the views only of the authors, and the Ministry cannot be held responsible for any use which may be made of the information contained therein. Translators: Marek Placi ski, Monika Boruta Supervision and proofreading: John Kearns Cover illustration: © ńMateusz Pawlik Printed by CPI books GmbH, Leck ISSN 2193-4207 ISBN 978-3-631-79022-9 (Print) E-ISBN 978-3-631-79393-0 (E-PDF) E-ISBN 978-3-631-79394-7 (EPUB) E-ISBN 978-3-631-79395-4 (MOBI) DOI 10.3726/b15805 Open Access: This work is licensed under a Creative Commons Attribution Non Commercial No Derivatives 4.0 unported license. -
Neurovascular Anatomy (1): Anterior Circulation Anatomy
Neurovascular Anatomy (1): Anterior Circulation Anatomy Natthapon Rattanathamsakul, MD. December 14th, 2017 Contents: Neurovascular Anatomy Arterial supply of the brain . Anterior circulation . Posterior circulation Arterial supply of the spinal cord Venous system of the brain Neurovascular Anatomy (1): Anatomy of the Anterior Circulation Carotid artery system Ophthalmic artery Arterial circle of Willis Arterial territories of the cerebrum Cerebral Vasculature • Anterior circulation: Internal carotid artery • Posterior circulation: Vertebrobasilar system • All originates at the arch of aorta Flemming KD, Jones LK. Mayo Clinic neurology board review: Basic science and psychiatry for initial certification. 2015 Common Carotid Artery • Carotid bifurcation at the level of C3-4 vertebra or superior border of thyroid cartilage External carotid artery Supply the head & neck, except for the brain the eyes Internal carotid artery • Supply the brain the eyes • Enter the skull via the carotid canal Netter FH. Atlas of human anatomy, 6th ed. 2014 Angiographic Correlation Uflacker R. Atlas of vascular anatomy: an angiographic approach, 2007 External Carotid Artery External carotid artery • Superior thyroid artery • Lingual artery • Facial artery • Ascending pharyngeal artery • Posterior auricular artery • Occipital artery • Maxillary artery • Superficial temporal artery • Middle meningeal artery – epidural hemorrhage Netter FH. Atlas of human anatomy, 6th ed. 2014 Middle meningeal artery Epidural hematoma http://www.jrlawfirm.com/library/subdural-epidural-hematoma -
The Importance of Modality Specificity in Diagnosing Central Auditory Processing Disorder
Point/Counterpoint Article The Importance of Modality Specificity in Diagnosing Central Auditory Processing Disorder Anthony T. Cacace Albany Medical College, Albany, NY Dennis J. McFarland Wadsworth Laboratories, New York State Health Department, Albany Purpose: This article argues for the use of processes from more generalized dysfunction; modality specificity as a unifying framework by it lacked discriminant validity and resulted in which to conceptualize and diagnose central an incomplete assessment. Consequently, any auditory processing disorder (CAPD). The intent hypothetical model resulting from incomplete is to generate dialogue and critical discussion assessments or potential therapies that are in this area of study. based on indeterminate diagnoses are them- Method: Research in the cognitive, behavioral, selves questionable, and caution should be and neural sciences that relates to the concept used in their application. of modality specificity was reviewed and Conclusions: Improving specificity of diag- synthesized. nosis is an imperative core issue to the area Results: Modality specificity has a long of CAPD. Without specificity, the concept has history as an organizing construct within a little explanatory power. Because of serious diverse collection of mainstream scientific flaws in concept and design, the unimodal disciplines. The principle of modality specificity inclusive framework should be abandoned in was contrasted with the unimodal inclusive favor of a more valid approach that uses framework, which holds that auditory tests modality specificity. alone are sufficient to make the CAPD diag- nosis. Evidence from a large body of data demonstrated that the unimodal framework Key Words: central auditory processing was unable to delineate modality-specific disorder, modality specificity ew discoveries, technical innovations, and novel review process is not perfect, it has stood the test of ideas are some of the driving forces behind the time and remains an important element in establishing N advancement of science. -
Vestibular Neuritis, Labyrinthitis, and a Few Comments Regarding Sudden Sensorineural Hearing Loss Marcello Cherchi
Vestibular neuritis, labyrinthitis, and a few comments regarding sudden sensorineural hearing loss Marcello Cherchi §1: What are these diseases, how are they related, and what is their cause? §1.1: What is vestibular neuritis? Vestibular neuritis, also called vestibular neuronitis, was originally described by Margaret Ruth Dix and Charles Skinner Hallpike in 1952 (Dix and Hallpike 1952). It is currently suspected to be an inflammatory-mediated insult (damage) to the balance-related nerve (vestibular nerve) between the ear and the brain that manifests with abrupt-onset, severe dizziness that lasts days to weeks, and occasionally recurs. Although vestibular neuritis is usually regarded as a process affecting the vestibular nerve itself, damage restricted to the vestibule (balance components of the inner ear) would manifest clinically in a similar way, and might be termed “vestibulitis,” although that term is seldom applied (Izraeli, Rachmel et al. 1989). Thus, distinguishing between “vestibular neuritis” (inflammation of the vestibular nerve) and “vestibulitis” (inflammation of the balance-related components of the inner ear) would be difficult. §1.2: What is labyrinthitis? Labyrinthitis is currently suspected to be due to an inflammatory-mediated insult (damage) to both the “hearing component” (the cochlea) and the “balance component” (the semicircular canals and otolith organs) of the inner ear (labyrinth) itself. Labyrinthitis is sometimes also termed “vertigo with sudden hearing loss” (Pogson, Taylor et al. 2016, Kim, Choi et al. 2018) – and we will discuss sudden hearing loss further in a moment. Labyrinthitis usually manifests with severe dizziness (similar to vestibular neuritis) accompanied by ear symptoms on one side (typically hearing loss and tinnitus). -
Sudden Bilateral Simultaneous Deafness with Vertigo As a Sole Manifestation of Vertebrobasilar Insufficiency H Lee, H a Yi, R W Baloh
539 J Neurol Neurosurg Psychiatry: first published as 10.1136/jnnp.74.4.539 on 1 April 2003. Downloaded from SHORT REPORT Sudden bilateral simultaneous deafness with vertigo as a sole manifestation of vertebrobasilar insufficiency H Lee, H A Yi, R W Baloh ............................................................................................................................. J Neurol Neurosurg Psychiatry 2003;74:539–541 CASE REPORT A 68 year old woman presented with bilateral sudden A 68 year old woman with long standing non-insulin depend- simultaneous hearing loss and transient spontaneous ent diabetes mellitus and hypertension suddenly developed vertigo as a sole manifestation of vertebrobasilar vertigo, vomiting, and bilateral tinnitus. Five minutes later, insufficiency. Extensive investigation to exclude other she noticed bilateral hearing loss. She had difficulty hearing causes was unremarkable. Magnetic resonance imaging her husband speak. She did not have dysarthria, numbness, of the brain, including diffusion images, showed no abnor- weakness, visual distortion, or incoordination. The hearing malities. A magnetic resonance angiogram showed severe loss persisted, but vertigo resolved over a few hours. Two days stenosis of the middle third of the basilar artery. A pure previously, she had had two episodes of transient vertigo and tone audiogram showed moderate sensorineural-type hearing loss involving the left ear that lasted no more than a hearing loss bilaterally. The localisation and mechanism of few minutes without any accompanying neurological symp- an isolated cochleovestibular dysfunction are discussed. toms. The patient had no previous history of hearing difficulty. There was no prior history of temporal bone fracture, menin- gitis, autoimmune disease, or exposure to ototoxic drugs. On admission, she had persistent bilateral hearing loss and ertebrobasilar insufficiency (VBI) can cause sudden non-specific dizziness with lightheadedness. -
Bilateral Sudden Hearing Difficulty Caused by Bilateral Thalamic Infarction
JCN Open Access LETTER TO THE EDITOR pISSN 1738-6586 / eISSN 2005-5013 / J Clin Neurol 2016 Bilateral Sudden Hearing Difficulty Caused by Bilateral Thalamic Infarction Jun-Hyung Lee Dear Editor, Sang-Soon Park Sudden-onset bilateral hearing difficulty has various possible causes, including infectious Jin-Young Ahn diseases of the inner ear, ototoxic medications, and Meniere’s disease.1,2 However, there have Jae-Hyeok Heo been only rare reports of vertebrobasilar arterial infarction that extensively invades the Department of Neurology, brainstem, or bilateral middle cerebral artery infarction that simultaneously invades both Seoul Medical Center, Seoul, Korea auditory cortexes.3-5 Herein we describe a case of bilateral sudden hearing difficulty due to cerebral infarction of the bilateral medial geniculate bodies. A 44-year-old male patient was admitted to Seoul Medical Center due to a 17-day history of sudden-onset hearing difficulty. About 1 year previously he had visited another hospital due to acute left-side paresthesia, and was diagnosed with and treated for diabetic neuropa- thy. A neurological examination revealed normal muscle strength in the bilateral upper and lower extremities, but paresthesia on his left side (both in the limbs and trunk) and hypes- thesia on the right side of the face. A brain MRI scan showed a chronic cerebral infarction at the right thalamic-midbrain junction and a subacute cerebral infarction at the left tha- lamic-midbrain junction (Fig. 1A, B, and C). An otolaryngological examination revealed chronic otitis media without structural abnormalities. His pure-tone audiogram indicated severe sensorineural hearing loss in both ears (Fig. -
Central Auditory Function Evaluation to Diagnose Central Auditory Processing Disorder Product Applicability
bmchp.org | 888-566-0008 wellsense.org | 877-957-1300 Medical Policy Central Auditory Function Evaluation to Diagnose Central Auditory Processing Disorder Policy Number: OCA 3.82 Version Number: 17 Version Effective Date: 01/01/21 + Product Applicability All Plan Products Well Sense Health Plan ∆ Boston Medical Center HealthNet Plan ∆ Well Sense Health Plan MassHealth ACO MassHealth MCO Qualified Health Plans/ConnectorCare/Employer Choice Direct Senior Care Options Notes: + Disclaimer and audit information is located at the end of this document. ∆ As required by state regulations and/or benefit coverage, different Plan definitions are specified for BMC HealthNet Plan products and Well Sense Health Plan products. See the applicable Definitions section for the Plan member. Policy Summary The evaluation of central auditory functioning to diagnose central auditory processing disorder (CAPD) is considered medically necessary for an adult or pediatric member when applicable Plan criteria are met. The evaluation is medically necessary when it is not part of a member’s individualized education plan (IEP) when one is appropriate for the member, it is a covered benefit for the member, and the Plan’s medical criteria are met (as specified in the Medical Policy Statement section of this Plan policy). Prior authorization is required for Plan covered services. See the member’s applicable benefit document available at www.bmchp.org for a member enrolled in a BMC HealthNet Plan product or Central Auditory Function Evaluation to Diagnose Central Auditory Processing Disorder + Plan refers to Boston Medical Center Health Plan, Inc. and its affiliates and subsidiaries offering health coverage plans to enrolled members. -
THE CLINICAL ASSESSMENT of the PATIENT with EARLY DEMENTIA S Cooper, J D W Greene V15
J Neurol Neurosurg Psychiatry: first published as 10.1136/jnnp.2005.081133 on 16 November 2005. Downloaded from THE CLINICAL ASSESSMENT OF THE PATIENT WITH EARLY DEMENTIA S Cooper, J D W Greene v15 J Neurol Neurosurg Psychiatry 2005;76(Suppl V):v15–v24. doi: 10.1136/jnnp.2005.081133 ementia is a clinical state characterised by a loss of function in at least two cognitive domains. When making a diagnosis of dementia, features to look for include memory Dimpairment and at least one of the following: aphasia, apraxia, agnosia and/or disturbances in executive functioning. To be significant the impairments should be severe enough to cause problems with social and occupational functioning and the decline must have occurred from a previously higher level. It is important to exclude delirium when considering such a diagnosis. When approaching the patient with a possible dementia, taking a careful history is paramount. Clues to the nature and aetiology of the disorder are often found following careful consultation with the patient and carer. A focused cognitive and physical examination is useful and the presence of specific features may aid in diagnosis. Certain investigations are mandatory and additional tests are recommended if the history and examination indicate particular aetiologies. It is useful when assessing a patient with cognitive impairment in the clinic to consider the following straightforward questions: c Is the patient demented? c If so, does the loss of function conform to a characteristic pattern? c Does the pattern of dementia conform to a particular pattern? c What is the likely disease process responsible for the dementia? An understanding of cognitive function and its anatomical correlates is necessary in order to ascertain which brain areas are affected. -
Cognitive Communication Disorders: a Clinician's Guide
A Presentation to the Mississippi Speech-Language-Hearing Association - 2018 Cognitive Communication Disorders: A Clinician’s Guide Scott S. Rubin, Ph.D. Associate Professor LSU Health Sciences Center New Orleans & Adjunct Professor Department of Psychology Tulane University Disclosure Statement: † Speaker is receiving both travel support and honorarium from conference organization. ‡There are no other disclosures to be made 2018 Cognitive Communication Disorders: A Clinician’s Guide OUTLINE 1. Hemispheric and system specific cognitive functions related to communication disorders. 2. Evaluation techniques for cognitive disorders. 3. Functional treatment issues/tasks focused on targeted cognitive communication deficits and patient needs. Getting right into it.. What a great area of cortex! Gotta love it. • Pre-frontal lobe (cortex) IS the center of our Executive System. Just think about impacts on communication! Functions include: − Thought – i.e., the highest levels of thought! • 1 The voice in your head… or voices… that you use to think consciously: Of course – the internal voice(s) do involve language. • Continued - Thought − We “verbally mediate” (consciously) what we are processing and thus what we do. − Prior to the inner voice, we have higher levels of pre-conscious thought: Language of the mind. − An unbelievable amount of activation; processing, planning, analyses, use of a wide variety of pre-conscious systems (centers), and integration of information. Far more than you can imagine. Source art above: mylifeingodsgarden.com/2017/06/17/voices-in-my-head/ • continued – Thought • Integration of information − Not just knowing a fact, but how different facts and concepts interact • Sorry students, but academic and clinical faculty see that some students do well memorizing terms and their individual concepts, however sometimes they can’t put it all together, see how various systems, functions, and/or disorders interact – to form the big picture. -
NEUROLOGY in TABLE.Pdf
ZAPORIZHZHIA STATE MEDICAL UNIVERSITY DEPARTMENT OF NEUROLOGY DISEASES NEUROLOGY IN TABLE (General neurology) for practical employments to the students of the IV course of medical faculty Zaporizhzhia, 2015 2 It is approved on meeting of the Central methodical advice Zaporozhye state medical university (the protocol № 6, 20.05.2015) and is recommended for use in scholastic process. Authors: doctor of the medical sciences, professor Kozyolkin O.A. candidate of the medical sciences, assistant professor Vizir I.V. candidate of the medical sciences, assistant professor Sikorskaya M.V. Kozyolkin O. A. Neurology in table (General neurology) : for practical employments to the students of the IV course of medical faculty / O. A. Kozyolkin, I. V. Vizir, M. V. Sikorskaya. – Zaporizhzhia : [ZSMU], 2015. – 94 p. 3 CONTENTS 1. Sensitive function …………………………………………………………………….4 2. Reflex-motor function of the nervous system. Syndromes of movement disorders ……………………………………………………………………………….10 3. The extrapyramidal system and syndromes of its lesion …………………………...21 4. The cerebellum and it’s pathology ………………………………………………….27 5. Pathology of vegetative nervous system ……………………………………………34 6. Cranial nerves and syndromes of its lesion …………………………………………44 7. The brain cortex. Disturbances of higher cerebral function ………………………..65 8. Disturbances of consciousness ……………………………………………………...71 9. Cerebrospinal fluid. Meningealand hypertensive syndromes ………………………75 10. Additional methods in neurology ………………………………………………….82 STUDY DESING PATIENT BY A PHYSICIAN NEUROLOGIST