DOCSLIB.ORG

MW S Rationales Files/Rash Rationale.Pdf

Total Page：16

File Type：pdf, Size：1020Kb

Download full-text PDF Read full-text
MW S Rationales Files/Rash Rationale.Pdf A RATIONALE FOR RASHES A RATIONALE FOR RASHES Assoc. Prof. Warwick Carter MB.BS; FRACGP; FAMA A guide to the diagnosis of skin rashes, patches, itches and damage. 1 A RATIONALE FOR RASHES CONTENTS Introduction SECTION ONE Dermatological Diagnostic Flow Chart A flow chart that leads the user through various symptoms and signs to possible diagnoses. SECTION TWO Diagnostic Algorithm for Rashes Symptoms and signs involving the skin and the conditions that may be responsible SECTION THREE Dermatological Conditions The symptoms, signs, investigation and treatment of medical conditions that may cause an alteration in the skin. Appendices Syndromes that may cause Skin Lesions Sun Exposure Skin Abnormalities 2 A RATIONALE FOR RASHES INTRODUCTION This book is designed for both the medical student and the doctor who is not a specialist in dermatology. It will take the user through a logical rationale in order to diagnose, and then treat, virtually every rash or skin condition likely to be encountered outside a specialist practice. There are two ways to reach a diagnosis, using the flow chart in Section One, or the Diagnostic Algorithms in Section Two. In Section One, a flow chart will guide the user through the presenting symptoms and signs of most rashes to a selection of possible diagnoses. As an alternative, the algorithms in Section Two will indicate the diagnoses possible with a variety of dermatological presenting symptoms. Once a diagnosis has, or number of differential diagnoses have been made, a detailed explanation of the various dermatological diagnoses can be found in the largest part of the book, Section Three. This has been written in a style that should be easy to understand by even junior medical students, with technical terms explained in each monograph, but should still be useful to the non-specialist doctor. The symptoms, signs, investigations and treatment of a very wide range of conditions are explained, along with pictures of the more common conditions. I trust that you will find it useful. Warwick Carter Brisbane OTHER BOOKS IN THIS SERIES A Rationale for the Brain A Rationale for Eyes A Rationale for the Abdomen A Rationale for the Chest 3 A RATIONALE FOR RASHES Section ONE DERMATOLOGICAL DIAGNOSTIC FLOW CHART 4 A RATIONALE FOR RASHES 5 A RATIONALE FOR RASHES Section Two DIAGNOSTIC ALGORITHMS FOR RASHES 6 A RATIONALE FOR RASHES Acne DIAGNOSTIC Acne vulgaris (common form) Papular acne (inflamed papules) ALGORITHMS Comedonal acne (less inflammation) Cystic acne (inflamed nodules, scarring) Steroid acne (secondary to steroid therapy) FOR RASHES Acne medicamentosa (due to drugs or cosmetics) Acne mechanica (due to friction from straps, etc.) Rosacea (telangiectasia, central face papules) Symptoms and signs Chloracne (contact with oils and chemicals) Pseudofolliculitis barbae (ingrown hair) involving the skin Acne keloidalis (keloid scar from traumatised acne) and the conditions that Pituitary tumour may be responsible Syndromes Adrenogenital syn. (amenorrhoea, rough skin) Cushing syn. (ecchymoses, obese, hirsute) Polycystic ovarian syn. (late onset, persistent) Premenstrual tension syn. (mastalgia, headache) See also Pustules FORMAT Alopecia Presenting Symptom Loss of hair (Alternate Name) Baldness Explanation of terminology Male pattern baldness (genetic) System or other group of symptoms Virilisation Diagnoses that may present with this symptom Testosterone secreting tumour [alternate name of diagnosis] (other symptoms of Alopecia totalis each diagnosis, or a discussion of the diagnosis) Chemotherapy Other entries to consider Diffuse hair loss Idiopathic (thinning of hair, either sex) Clinical Sign Any severe illness Sign (Alternate Name) [Abbreviation] Rapid weight loss Exp: An explanation of the sign, with its Shock or stress (eg. bereavement, surgery) methodology described in sufficient detail to Telogen effluvium (after stress) enable the practitioner to perform the test. Hypothyroidism (fatigue, dry skin, cold intolerance) Int: The interpretation of the sign. Hyperthyroidism (sweating, heat intolerance) (+) The diseases, syndromes etc. that SLE (butterfly rash, arthritis, nephritis) should be considered if the test is positive Postpartum (++) The interpretation of an exaggerated or Hypopituitarism grossly positive test Postmenopause (–) Ditto for a negative test result Diabetes mellitus (polydipsia, polyuria) (AB) Ditto for an abnormal test result Renal failure Phys: The pathophysiology of the sign to enable Protein deficiency (eg. malnutrition, vegetarian) its significance to be better understood Addison's disease (groin and axilla only) Other entries to consider Hypervitaminosis A (anorexia, weight loss, rashes) Irradiation Iron or zinc deficiency Ectodermal dysplasia Dubowitz syn. (reduced growth, ptosis) Fröhlich syn. (thin skin, low libido, obese) 7 A RATIONALE FOR RASHES Hallermann-Streiff syn. (dwarf, cataracts) Bleeding in Skin Langer-Giedion syn. (bullous nose, exostoses) See Ecchymosis; Purpura and Petechiae Loose Anagen syn. Drugs (eg. cancer therapy, anticoagulants, vitamin A, lithium, beta-blockers, oral contraceptives) Patchy hair loss Blisters Alopecia areata See Acne; Bullous Rash; Pustules; Vescicles Any severe illness Fungal scalp infection Discoid lupus erythematosus Bouchard's Nodes Traction (hair style that pulls on one area of scalp) Bony prominences at the dorsal margins of proximal Trichotillomania (recurrent trauma) interphalangeal joints Syphilis (variable symptoms) Severe osteoarthritis See also Heberden's Nodes Anal Itch See Pruritus Ani Bruising, Excess Thrombocytopenia (bleeding time increased) Idiopathic purpura Annular Rash Hereditary disorders of coagulation Rash consisting of circular lesion(s) (eg.haemophilia, von Willebrand's, Christmas Tinea cruris, corporis or capitis (red, scaly edge, disease) pruritis) Bone marrow suppression Pityriasis rosea (herald patch, trunk, scaly centre) Ionising radiation (eg. X-rays, gamma rays) Cutaneous larva migrans (hookworm infestation, Systemic viral infections itchy, track) Leukaemia (abnormal white cell count) Pigmented purpuic dermatosis (venous Typhus (fever, malaise) insufficiency, pepper spots peripheral on lesion) Subacute bacterial endocarditis Granuloma annulare (faint, flesh colour, limbs) Insect and snake bites Cutaneous lupus (chronic relapsing, trunk, Following massive blood transfusions confluent) Renal failure Hepatic failure Polycythaemia (rubra) vera Scurvy (inflamed and bleeding gums) Areolar Pigmentation Cushing syn. (moon face, obese, amenorrhoea) Darkening of areola and nipple AIDS (splenomegaly, fever, cachexia) Present or past pregnancy Defibrination syn. (see Syndromes) Sex hormone therapy Painful bruising syn. (female, paraesthesiae) Familial, Drugs (eg. steroids, arsenic, quinine, aspirin, Racial warfarin, chlorothiazide) See also Ecchymoses Auspitz's Sign Exp: When white scale is removed from a plaque Bullous Rash of scale covered dermatitis on the shins, a Large fluid filled blisters bleeding area results Impetigo (crusts, vesicles, pruritic) Int: (+) Psoriasis Herpes zoster (severe pain, dermatome Phys: In psoriasis, the plaque has a microcapillary distribution) circulation that is disrupted by its removal Herpes simplex (pain) Cellulitis (red, hot, fever) Toxic epidermal necrolysis Bald Contact dermatitis (erythema, itch) See Alopecia Drug eruptions Insect and arachnid bites 1 A RATIONALE FOR RASHES Erythema multiforme (target lesions, erythema, Circular Rash extensor surfaces) See Annular Rash Pemphigus (normal skin surrounds, relapsing crops) Pemphigoid (large, tense, elderly) Dermatitis herpetiformis (vesicles, papules, erythema) Circumoral Pallor Pompholyx (soles and palms, tense blisters) Exp: Relatively white area around mouth Porphyria cutanea tarda Int: (+) Fever of any cause (eg. scarlet fever) Epidermolysis bullosa Phys: Dilatation of superficial blood vessels in Lichen planus looser skin further away from mouth causes Burns (heat or irradiation) darkening and reddening of that area (rather than blanching of circumoral tissue) Butterfly Rash Coilonychia Erythematous, scaly rash spreading across both See Koilonychia cheeks and meeting on the nasal bridge Systemic lupus erythematosus (SLE) Photodermatitis Discoid lupus Cullen's Sign Atopic dermatitis Exp: Spontaneous umbilical bruising Serum sickness Int: (+) Ruptured ectopic pregnancy, carcinoma of pancreas, haemorrhagic pancreatitis, other causes of haemoperitoneum Café-au-Lait Spots Phys: Tracking of free intraperitoneal blood to Light brown spots on skin umbilicus Von Recklinghausen's disease of multiple neurofibromata Pityriasis versicolor Tuberous sclerosis Darierʼs Sign See also Pigmentation of Skin, Excess Exp: Rubbing a finger or blunt object firmly over a skin lesion causes oedema and erythema around the lesion Int: (+) Mastocytosis Chip Sign Phys: Pressure releases histamine from increased Exp: Skin blotches covered with fine, number of mast cells in lesion nonadherent scabs that are easily removed by a fingernail or blunt edge Int: (+) Pityriasis versicolor Phys: Superficial fungal infection Dark Skin See Chloasma; Face Pigmented; Pigmentation of Mouth; Pigmentation of Skin, Excess Chloasma Yellow-brown spotty skin pigmentation on upper cheeks and forehead Pregnancy Depigmented Skin Oral contraceptives Racial and familial Sun exposure Pityriasis versicolor (reddish, scaling) Syphilis Pityriasis alba (eczematous, asymptomatic) mMlaria, Vitiligo (dead white, well demarcated patches) Tuberculosis (TB) Halo naevus Cirrhosis Postinflammatory
Load more

Recommended publications
  • International Journal of Scientific Research
    ORIGINAL RESEARCH PAPER Volume-9 | Issue-1 | January-2020 | PRINT ISSN No. 2277 - 8179 | DOI : 10.36106/ijsr INTERNATIONAL JOURNAL OF SCIENTIFIC RESEARCH TYPES AND VARIANTS OF ACNE Dermatology Shailee Patel ABSTRACT Acne occur when pores of skin are blocked with oil, dead skin, or bacteria. It can occur when excessive oil is produced by follicles, bacteria build up in pores, and dead skin cells accumulate in pores. All these problem contribute in development of pimple. Acne are majorly seen among teenagers but they can also occur in adults. There are varying from of acne, and their varying treatment. KEYWORDS 1.INTRODUCTION ulcerative colitis and Crohn's disease and syndromes, such as Acne is linked to the change in hormone level during puberty. Acne is a synovitis, acne, pustulosis, hyperostosis, and osteitis (SAPHO) and disorder that is seen worldwide. Acne is a disease of the teenagers but pyogenic arthritis, pyoderma gangrenosum, and acne (PAPA) can be seen even in newborn children and also adults. Age and gender syndromes. also play a very important role in onset of acne. Acne most commonly occur between the ages of 10-13 years. Girls have an earlier onset 3.4 Occupational Acne which easily contribute to the onset of puberty in girls than in boys. The Occupational acne is defined as development of acne-like lesions after disease severity in more in boys during the late adolescence. Acne exposure to occupational agents in persons not prone to develop acne mostly develops on areas of skin that have abundant oil glands, like the and who have not had acne before engaging in the said occupation.
    [Show full text]
  • Revista6vol88ingles Layout 1 1/2/14 11:39 AM Página 1039 Página AM 11:39 1/2/14 1 Revista6vol88ingles Layout Revista6vol88ingles Layout 1 1/2/14 11:39 AM Página 1040
    Revista6Vol88ingles_Layout 1 1/2/14 11:39 AM Página 1039 ICONOGRAPHY 1039 s Drug-induced acne and rose pearl: similarities* Acne medicamentosa e a pérola rosa: semelhanças Rubens Pontello Junior 1 Rogerio Nabor Kondo 2 DOI: http://dx.doi.org/10.1590/abd1806-4841.20132586 Abstract: Drug-induced acne is a common skin condition whose classic symptoms can be similar to a rose pearl, as in the case of a male patient presenting with this condition after excessive use of a cream containing corticos- teroids. Keywords: Acneiform eruptions; Drug eruptions; Skin diseases Resumo: A acne medicamentosa é uma dermatose comum, que pode apresentar no seu quadro clássico seme- lhanças à pérola rosa, como no caso apresentado de um paciente do sexo masculino cujo quadro surgiu após uso intempestivo de creme contendo corticoesteróide. Palavras-chave: Dermatopatias; Erupção por droga; Erupções acneiformes Drug-induced acne, or drug-induced acneiform Discontinuation of the drug leads to remission eruption, is an adverse effect of a series of systemic of symptoms. Antihistamines are recommended in drugs, such as corticosteroids, lithium, vitamin B12, case of pruritus, and oral antibiotics are recommend- thyroid hormones, halogen compounds (iodine, ed in case of secondary infection with pustules or 2,3 bromine, fluorine, and chlorine), antibiotics (tetracy- impetiginization. cline and streptomycin), antituberculosis drugs We can observe the usual aspect of a papular (INH), lithium carbonate, antiepileptic drugs (pheno- follicular eruption and, on closer look, a small papule barbital and hydantoin derivatives), cyclosporin A, carefully surmounted by a pustule, which might be a antimycotics, gold salts, isotretinoin, clofazimine, epi- possible evolution into a vesiculopustule, as cited in dermal growth factor receptor inhibitors (cetuximab, the literature, demonstrating the inexorable aspect of gefitinib, and erlotinib), and interferon-beta.1,2 Usually, drug-induced acne (Figure 1).
    [Show full text]
  • AKNE (Acne Vulgaris )
    AKNE (acne vulgaris) - PRISTUP U OOM - prevalencija kod adolescenata do 95%, najčešće 13 - 15 g., ranije kod djevojčica, nakon 20. g. se obično povlače, no mogu potrajati i nakon 30. g. (češće kod Ž), teži oblici češće kod M - kronična upala kože, zahvaća pilosebacealnu jedinicu (dlaka sa ovojnicama, mišić podizač dlake i lojna žlijezda), najčešće na licu (99%), rjeđe leđa (60%) te prsa (15%) ETIOLOGIJA - nasljeđe, hormoni i bakterije - nasljeđivanje AD (veličina lojnica i osjetljivost na podražaje → luče više loja → glavni preduvjet za razvoj akni) - pubertet - poremećen odnos spolnih hormona (↑androgena ili ↓estrogena) → hipertrofija i hiperfunkcija lojnica → više loja → folikularna hiperkeratoza → pilosebacealne jedinice začepe se lojem i odljuštenim keratinocitima → komedon - otvoreni (otvor folikula širok → crna točkica) ili zatvoreni (otvor nije vidljiv, sitne polukuglaste bjelkaste tvorbe) - endokrinološka i gin obrada kod žena samo u slučaju drugih znakova hiperandrogenizma (hirzutizam, poremečaj ciklusa, neplodnost.. → PCOS) ili preuranjenog puberteta - komedoni se ponekad koloniziraju normalnim anaerobom kože Propionibacterium acnes → upala → papula, pustula, apscedirajući čvorovi KLINIČKI OBLICI: 1) ACNE COMEDONICA - blagi oblik - promjene opipljive, promjer 1-3 mm - otvoreni i zatvoreni komedoni - može biti prisutna i max 1 papula 2) ACNE PAPULOPUSTULOSA - umjereno težak oblik akni - papule i pustule 2-5 mm, pustule površnije, papule kod dublje upale (češće leđa i sternalno) - BLAGI oblik → komedoni, do max 5 papula/pustula
    [Show full text]
  • Acneiform Dermatoses
    Dermatology 1998;196:102–107 G. Plewig T. Jansen Acneiform Dermatoses Department of Dermatology, Ludwig Maximilians University of Munich, Germany Key Words Abstract Acneiform dermatoses Acneiform dermatoses are follicular eruptions. The initial lesion is inflamma- Drug-induced acne tory, usually a papule or pustule. Comedones are later secondary lesions, a Bodybuilding acne sequel to encapsulation and healing of the primary abscess. The earliest histo- Gram-negative folliculitis logical event is spongiosis, followed by a break in the follicular epithelium. The Acne necrotica spilled follicular contents provokes a nonspecific lymphocytic and neutrophilic Acne aestivalis infiltrate. Acneiform eruptions are almost always drug induced. Important clues are sudden onset within days, widespread involvement, unusual locations (fore- arm, buttocks), occurrence beyond acne age, monomorphous lesions, sometimes signs of systemic drug toxicity with fever and malaise, clearing of inflammatory lesions after the drug is stopped, sometimes leaving secondary comedones. Other cutaneous eruptions that may superficially resemble acne vulgaris but that are not thought to be related to it etiologically are due to infection (e.g. gram- negative folliculitis) or unknown causes (e.g. acne necrotica or acne aestivalis). oooooooooooooooooooo Introduction matory (acne medicamentosa) [1]. The diagnosis of an ac- neiform eruption should be considered if the lesions are The term ‘acneiform’ refers to conditions which super- seen in an unusual localization for acne, e.g. involvement of ficially resemble acne vulgaris but are not thought to be re- distal parts of the extremities (table 1). In contrast to acne lated to it etiologically. vulgaris, which always begins with faulty keratinization Acneiform eruptions are follicular reactions beginning in the infundibula (microcomedones), comedones are usu- with an inflammatory lesion, usually a papule or pustule.
    [Show full text]
  • Drug-Induced Acneform Eruptions: Definitions and Causes Saira Momin, DO; Aaron Peterson, DO; James Q
    REVIEW Drug-Induced Acneform Eruptions: Definitions and Causes Saira Momin, DO; Aaron Peterson, DO; James Q. Del Rosso, DO Several drugs are capable of producing eruptions that may simulate acne vulgaris, clinically, histologi- cally, or both. These include corticosteroids, epidermal growth factor receptor inhibitors, cyclosporine, anabolic steroids, danazol, anticonvulsants, amineptine, lithium, isotretinoin, antituberculosis drugs, quinidine, azathioprine, infliximab, and testosterone. In some cases, the eruption is clinically and his- tologically similar to acne vulgaris; in other cases, the eruption is clinically suggestive of acne vulgaris without histologic information, and in still others, despite some clinical resemblance, histology is not consistent with acne vulgaris.COS DERM rugs are a relatively common cause of involvement; and clearing of lesions when the drug eruptions that may resemble acne clini- is discontinued.1 cally, histologically,Do or both.Not With acne Copy vulgaris, the primary lesion is com- CORTICOSTEROIDS edonal, secondary to ductal hypercor- It has been well documented that high levels of systemic Dnification, with inflammation leading to formation of corticosteroid exposure may induce or exacerbate acne, papules and pustules. In drug-induced acne eruptions, as evidenced by common occurrence in patients with the initial lesion has been reported to be inflammatory Cushing disease.2 Systemic corticosteroid therapy, and, with comedones occurring secondarily. In some cases in some cases, exposure to inhaled or topical corticoste- where biopsies were obtained, the earliest histologic roids are recognized to induce monomorphic acneform observation is spongiosis, followed by lymphocytic and lesions.2-4 Corticosteroid-induced acne consists predomi- neutrophilic infiltrate. Important clues to drug-induced nantly of inflammatory papules and pustules that are acne are unusual lesion distribution; monomorphic small and uniform in size (monomorphic), with few or lesions; occurrence beyond the usual age distribution no comedones.
    [Show full text]
  • Download Your Ultimate Guide to Treat Acne
    Your Ultimate Guide to treating acne FIND OUT The many factors that may be impacting your acne, the types of acne and treatments and products available for managing this common condition. Beat the blackheads Can blackheads be managed at home with JUST skincare? Skip to page xx to find out Contents PAGE NUMBER What is acne? Types of acne Types of acne lesions Grades of acne Causes of acne Common skincare mistakes What skincare should you use? The best treatments for treating acne (INC IV DRIPS, CLEARSKIN, PEELS, NEEDLING, CLEARLIFT, INFINI) Maintaining acne-prone skin Solutions for acne scarring W W W . E D G B A S T O N W E L L N E S S . C O . U K What is acne? Acne is the most common skin disease in adolescence and is just as problematic amongst adults. Acne causes skin various skin lesions and inflammation on the face and body, can appear in waves or as a constant manifestation. More than 90% of the global world population are said to be affected by acne at some point in their lives. Acne is most commonly found on the face, back & chest About 60% of people with adult acne turn to non-prescription treatments to improve the condition. Depending on a number of factors, acne can also cause skin scarring and have a negative psychological impact too. Research tells us that acne can impact our self esteem and image, and increase risk of depression and anxiety. W W W . E D G B A S T O N W E L L N E S S .
    [Show full text]
  • Clinical Dermatology
    CLINICAL DERMATOLOGY A Manual of Differential Diagnosis Third Edition By Stanferd L. Kusch, MD Compliments of: www.taropharma.com Copyright © 1979 (original edition) by Stanferd L. Kusch, MD Second Edition 1987 Third Edition 2003 All rights reserved. No part of the contents of this book may be reproduced or transmitted in any form or by any means, including photocopying, without the written permission of the copyright owner. NOTICE Medicine is an ever-changing science. As new research and clinical experience broaden our knowledge, changes in treatment and drug therapy are required. The author and the publisher of this work have checked with sources believed to be reliable in their efforts to pro- vide information that is complete and generally in accord with the standards accepted at the time of publication. However, in view of the possibility of human error or changes in medical sciences, neither the author nor the publisher nor any other party who has been involved in the preparation or publication of this work warrants that the information contained herein is in every respect accurate or com- plete, and they disclaim all responsibility for any errors or omissions or for the results obtained from use of the information contained in this work. Readers are encouraged to confirm the information here- in with other sources. For example and in particular, readers are advised to check the product information sheet included in the pack- age of each drug they plan to administer to be certain that the infor- mation contained in this work is accurate and that changes have not been made in the recommended dose or in the contraindications for administration.
    [Show full text]
  • Acneiform Dermatoses
    View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Universität München: Elektronischen Publikationen Dermatology 1998;196:102–107 G. Plewig T. Jansen Acneiform Dermatoses Department of Dermatology, Ludwig Maximilians University of Munich, Germany Key Words Abstract Acneiform dermatoses Acneiform dermatoses are follicular eruptions. The initial lesion is inflamma- Drug-induced acne tory, usually a papule or pustule. Comedones are later secondary lesions, a Bodybuilding acne sequel to encapsulation and healing of the primary abscess. The earliest histo- Gram-negative folliculitis logical event is spongiosis, followed by a break in the follicular epithelium. The Acne necrotica spilled follicular contents provokes a nonspecific lymphocytic and neutrophilic Acne aestivalis infiltrate. Acneiform eruptions are almost always drug induced. Important clues are sudden onset within days, widespread involvement, unusual locations (fore- arm, buttocks), occurrence beyond acne age, monomorphous lesions, sometimes signs of systemic drug toxicity with fever and malaise, clearing of inflammatory lesions after the drug is stopped, sometimes leaving secondary comedones. Other cutaneous eruptions that may superficially resemble acne vulgaris but that are not thought to be related to it etiologically are due to infection (e.g. gram- negative folliculitis) or unknown causes (e.g. acne necrotica or acne aestivalis). oooooooooooooooooooo Introduction matory (acne medicamentosa) [1]. The diagnosis of an ac- neiform eruption should be considered if the lesions are The term ‘acneiform’ refers to conditions which super- seen in an unusual localization for acne, e.g. involvement of ficially resemble acne vulgaris but are not thought to be re- distal parts of the extremities (table 1).
    [Show full text]
  • Program Mounts Bay Road, Perth, Western Australia 49Th Annual Scientific Meeting Perth Convention Andexhibitioncentre, Saturday 14May to Tuesday 17May 2016
    PROGRAM 49TH ANNUAL SCIENTIFIC MEETING PERTH CONVENTION AND EXHIBITION CENTRE, MOUNTS BAY ROAD, PERTH, WESTERN AUSTRALIA SATURDAY 14 MAY TO TUESDAY 17 MAY 2016 AUSTRALIAN DERMATOLOGY NURSES’ ASSOCIATION 15TH NATIONAL CONFERENCE SATURDAY 14 MAY AND SUNDAY 15 MAY 2016 AUSTRALASIAN SOCIETY OF COSMETIC DERMATOLOGISTS INCLUDES: 3RD ANNUAL MEETING SUNDAY 15 MAY TO TUESDAY 17 MAY 2016 www.dermcoll.edu.au PERTH _ AUSTRALIA’S SUNNIEST CAPITAL CITY WELCOMES YOU TO THE AUSTRALASIAN COLLEGE OF DERMATOLOGISTS 49TH ANNUAL SCIENTIFIC MEETING WELCOME FROM THE PRESIDENT Welcome to Perth, Western Australia for the 49th Annual Scientific Meeting of the Australasian College of Dermatologists. Enclosed in this brochure is the program and other important information for the meeting along with the programs for the meetings of the Australian Dermatology Nurses’ Association and the Australasian Society of Cosmetic Dermatologists. The local organising committee was this year headed by Dr Daniel Hewitt, FACD, and supported by the College’s Scientific Meetings Steering Committee. The Western Australia Faculty welcomes Fellows, trainees, students and other medical practitioners to the meeting. 2 I am excited to welcome our overseas speakers: Professor John McGrath, Professor of Molecular Dermatology and Head of the Genetic Skin Disease Unit at St John’s Institute of Dermatology, King’s College London, United Kingdom. Professor Peter Mortimer, Professor of Dermatological Medicine at St George’s, University of London, United Kingdom. Professor Dirk Elston, Professor and Chairman of the Department of Dermatology and Dermatologic Surgery at the Medical University of South Carolina, Charleston, United States of America. Professor Clark Otley, Chair of the Department of Dermatology and Professor of Dermatology at Mayo Clinic in Rochester, Minnesota, United States of America.
    [Show full text]
  • Acne Related to Dietary Supplements
    Volume 26 Number 8| Aug 2020| Dermatology Online Journal || Commentary 26(8):2 Acne related to dietary supplements Dina H Zamil1 BS, Ariadna Perez-Sanchez2 MD, Rajani Katta3 MD Affiliations: 1Baylor College of Medicine, Houston, Texas, USA, 2University of Texas Health Science Center at San Antonio, San Antonio, Texas, USA, 3McGovern Medical School at University of Texas Health Houston, Houston, Texas, USA Corresponding Author: Rajani Katta MD, 6800 West Loop South, Suite 180, Bellaire, TX 77401, Tel: 281-501-3150, Email: [email protected] Drugs that may induce acne include corticosteroids, Abstract anabolic-androgenic steroids, hormonal drugs, Multiple prescription medications may cause or lithium, antituberculosis medications, and drugs that aggravate acne. A number of dietary supplements contain halogens, specifically iodides and bromides have also been linked to acne, including those [1-7]. There are also reports of acne potentially containing vitamins B6/B12, iodine, and whey, as well induced by cancer therapies, immunosuppressants, as “muscle building supplements” that may be and autoimmune disease medications [7]. contaminated with anabolic-androgenic steroids (AAS). Acne linked to dietary supplements generally Multiple dietary supplements have also been linked resolves following supplement discontinuation. to acne. A number of case reports and series have Lesions associated with high-dose vitamin B6 and described the onset of acne with certain dietary B12 supplements have been described as supplements, with resolution following monomorphic and although pathogenesis is discontinuation of supplement use. These include unknown, a number of hypotheses have been vitamins and dietary ingredients that may seem proposed. Iodine-related acne may be related to the innocuous from the standpoint of the consumer or use of kelp supplements and has been reported as monomorphic, inflammatory pustules on the face physician.
    [Show full text]
  • WO 2012/125941 Al 20 September 2012 (20.09.2012) P O P C T
    (12) INTERNATIONAL APPLICATION PUBLISHED UNDER THE PATENT COOPERATION TREATY (PCT) (19) World Intellectual Property Organization International Bureau (10) International Publication Number (43) International Publication Date WO 2012/125941 Al 20 September 2012 (20.09.2012) P O P C T (51) International Patent Classification: (US). JU, William, D. [US/US]; 435 Bernardsville Road, A 59/22 (2006.01) Mendham, NJ 07945 (US). KELLOGG, Scott, C. [US/US]; PO Box 1773, Mattapoisett, MA 02739 (US). (21) International Application Number: PROUTY, Stephen, M. [US/US]; 236 Windsor Way, PCT/US20 12/029475 Doylestown, PA 18901 (US). SCHWEIGER, Eric (22) International Filing Date: [US/US]; 166 East 34th Street, #10B, New York, NY 16 March 2012 (16.03.2012) 10016 (US). LEDERMAN, Seth [US/US]; 200 E. 72nd Street - 35k, New York, NY 10021 (US). OSBAKKEN, English (25) Filing Language: Mary [US/US]; 29 W. Walnut Lane, Suite 300, Phil (26) Publication Language: English adelphia, PA 19144 (US). SCHINAZI, Alan, D. [US/US]; 129 Butler Avenue, Providence, RI 02906 (US). (30) Priority Data: 61/453,895 17 March 201 1 (17.03.201 1) US (74) Agents: CORUZZI, Laura, A. et al; Jones Day, 222 East 61/453,902 17 March 201 1 (17.03.201 1) US 41st Street, New York, NY 10017-6702 (US). 61/453,848 17 March 201 1 (17.03.201 1) US (81) Designated States (unless otherwise indicated, for every 61/453,746 17 March 201 1 (17.03.201 1) US kind of national protection available): AE, AG, AL, AM, 61/5 13,906 1 August 201 1 (01.08.201 1) US AO, AT, AU, AZ, BA, BB, BG, BH, BR, BW, BY, BZ, 61/525,589 19 August 201 1 (19.08.201 1) US CA, CH, CL, CN, CO, CR, CU, CZ, DE, DK, DM, DO, 61/534,156 13 September 201 1 (13.09.201 1) US DZ, EC, EE, EG, ES, FI, GB, GD, GE, GH, GM, GT, HN, 61/590,937 26 January 2012 (26.01.2012) US HR, HU, ID, IL, IN, IS, JP, KE, KG, KM, KN, KP, KR, (71) Applicant (for all designated States except US): FOL- KZ, LA, LC, LK, LR, LS, LT, LU, LY, MA, MD, ME, LICA, INC.
    [Show full text]
  • 4 Haartalgdruesen SS 09.Pdf
    Klinik für Dermatologie und Allergologie Philipps-Universität Marburg Seminarreihe Dermatologie Erkrankungen der Anhangsgebilde: - Haare insbes. Alopezien - Talgdrüsen insbes. Acne, Rosacea, Rhinophym SS – 2009 Haarzyklus Anagen = Wachstumsphase 3 - 6 Jahre 1/3 mm /d Katagen = Übergangsphase wenige Tage Telogen = Ruhephase 2 - 4 Monate 100 Kolbenhaare /d Haarerkrankungen Begriffsdefinitionen Hypotrichose, Atrichie Î angeborene Verringerung der Haardichte / vollständigesFehlen der Haare Haarerkrankungen Begriffsdefinitionen Hypotrichose, Atrichie Î angeborene Verringerung der Haardichte / vollständigesFehlen der Haare Effluvium Î Haarausfall (d.h. der Vorgang) Anageneffluvium Î während der Wachstumsphase Telogeneffluvium Î während der Ruhephase (Kolbenhaare) Haarerkrankungen Begriffsdefinitionen Hypotrichose, Atrichie Î angeborene Verringerung der Haardichte / vollständigesFehlen der Haare Effluvium Î Haarausfall (d.h. der Vorgang) Anageneffluvium Î während der Wachstumsphase Telogeneffluvium Î während der Ruhephase (Kolbenhaare) Alopezie Î erworbener Verlust der Behaarung (d.h. der Zustand) Alopezie ● diffuse Alopezie: - gleichmäßiger Befall - Musterbildung ("patterned") ● umschriebene Alopezien - herdförmig Alopezie ● vernarbend: irreversibel ● nichtvernarbend: oft reversibel Nichtvernarbende Alopezie, Beispiele ● Androgenetische Alopezie ● Alopecia areata ● Chemisch / medikamentös - Schwermetalle: Thallium, Arsen, Blei etc. - Thyreostatika: Thiouracil, Carbamizol - Antikoagulanzien: Cumarin, Heparin - Retinoide - Zytostatika - u.a.:
    [Show full text]