Tsix
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- Differentiation-Dependent Requirement of Tsix Long Non-Coding RNA in Imprinted X-Chromosome Inactivation
- Characterization of Rex1 Role in Tsix Regulation Abstract Tsix Activation
- X Chromosome Dosage Compensation
- HIV-1 Natural Antisense Transcription and Its Role in Viral Persistence
- In Silico and in Vitro Analysis of Lncrna XIST Reveals a Panel of Possible Lung Cancer Regulators and a Five-Gene Diagnostic Signature
- X Inactivation: Tsix and Xist As Yin and Yang Susanna K
- Origin and Evolution of X Chromosome Inactivation
- Tsix Transcription Across the Xist Gene Alters Chromatin Conformation Without Affecting Xist Transcription: Implications for X-Chromosome Inactivation
- Roles of Long Noncoding Rnas in X-Chromosome Inactivation
- Releasing the Break on X Chromosome Inactivation: Rnf12/RLIM Targets REX1 for Degradation
- Dosage Compensation Mechanisms: Evolution Dosage Compensation Mechanisms: Evolution
- Mechanistic Concepts in X Inactivation Underlying Dosage Compensation in Mammals
- Pluripotency Factor Binding and Tsix Expression Act Synergistically To
- REX1 Is the Critical Target of RNF12 in Imprinted X Chromosome Inactivation in Mice
- Implications for Tsix Function
- Identification of TSIX, Encoding an RNA Antisense to Human XIST
- New Insights Into X-Chromosome Reactivation During Reprogramming to Pluripotency
- A Functional Role for Tsix Transcription in Blocking Xist RNA Accumulation but Not in X-Chromosome Choice