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SOCS3
Quantitative Modelling Explains Distinct STAT1 and STAT3
SOCS3 Antibody A
Japanese Encephalitis Virus Upregulates the Expression of SOCS3 in Mouse Brain and Raw264.7 Cells
Suppression of Cytokine Signaling by SOCS3: Characterization of the Mode of Inhibition and the Basis of Its Specificity
Expression of SOCS1 and SOCS3 Genes Is Differentially Regulated in Breast Cancer Cells in Response to Proinflammatory Cytokine and Growth Factor Signals
Potential Implications for SOCS in Chronic Wound Healing
Opposing Roles of STAT1 and STAT3 in IL-21 Function in + CD4 T Cells
Negative Regulation of Cytokine Signaling in Immunity
The Role of Suppressor of Cytokine Signaling 1 and 3 in Human Cytomegalovirus Replication
PCM1-JAK2 Activates SOCS2 and SOCS3 Via STAT5
STAT3 Is a Master Regulator of Epithelial Identity and KRAS-Driven Tumorigenesis
Interleukin-6: a Constitutive Modulator of Glycoprotein 130, Neuroinflammatory and Cell Survival Signaling in Retina Franklin D
STAT3 Activation in Response to IL-6 Is Prolonged by the Binding of IL-6 Receptor to EGF Receptor
Deficient SOCS3 and SHP-1 Expression in Psoriatic T Cells Karsten W
The Role of SOCS3 in the Regulation of T-Cell Activation and IL-7Rα Expression
Active Suppressors of Cytokine Signaling 3 (SOCS3)
SOCS2 and SOCS3 in Macrophage Polarization M1 Macrophages Are Pro-Inflammatory, Whereas M2 Macrophages Are Involved in Tissue Repair
Toxoplasma Gondii Glycoprotein 130 Hypermorphs to IL-6 Mediates The
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Understanding the Dynamic Regulation of SOCS3 – Nikoletta Kalenderoglou – November 2019
CD4+ T Cell Help Creates Memory CD8+ T Cells with Innate and Help-Independent Recall Capacities
Hepatocyte Growth Control by SOCS1 and SOCS3
Studies on the Role of Socs3 and Stat3 in Development and Function of the Immune System
SOCS1 and SOCS3 Target IRF7 Degradation to Suppress TLR7-Mediated Type I IFN Production of Human Plasmacytoid Dendritic Cells
Suppressor of Cytokine Signaling (SOCS)1 and SOCS3 Stimulation During Experimental Cytomegalovirus Retinitis: Virologic, Immunologic, Or Pathologic Mechanisms
Loss of SOCS3 in the Liver Promotes Fibrosis by Enhancing STAT3
Inducible Gene That Suppresses Gp130-Mediated Cardiac Myocyte Hypertrophy and Survival Pathways
Downregulated SOCS1 Expression Activates the JAK1/STAT1 Pathway and Promotes Polarization of Macrophages Into M1 Type
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STAT3 Activation in Response to IL-6 Is Prolonged by the Binding of IL-6 Receptor to EGF Receptor
Adenovirus-Mediated SOCS3 Gene Transfer Inhibits the Growth and Enhances the Radiosensitivity of Human Non-Small Cell Lung Cancer Cells
SOCS3 and SOCS6 Are Required for the Risperidone-Mediated Inhibition of Insulin and Leptin Signaling in Neuroblastoma Cells
434.Full.Pdf
The Effects of SOCS1 and SOCS3 Peptide Mimetics on Macrophage Phagocytosis of Malignant Cells
Endothelial SOCS3 Maintains Homeostasis and Promotes Survival in Endotoxemic Mice
Polarization and Inflammation SOCS3 Deficiency Promotes M1 Macrophage
Tyrosine-Phosphorylated SOCS3 Negatively Regulates Cellular